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Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
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Basavarajeeyam is an important text for ayurvedic physician belonging to andhra pradehs. It is a popular compendium in various parts of our country as well as in andhra pradesh. The content of the text was presented in sanskrit and telugu language (Bilingual). One of the most famous book in ayurvedic pharmaceutics and therapeutics. This book contains 25 chapters called as prakaranas. Many rasaoushadis were explained, pioneer of dhatu druti, nadi pareeksha, mutra pareeksha etc. Belongs to the period of 15-16 century. New diseases like upadamsha, phiranga rogas are explained.
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
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Amiodarone Hydrochloride Injection Taj Pharma SmPC
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Amiodarone Hydrochloride
Injection 150mg/3ml,
450mg/9ml
1. Name of the medicinal product
Amiodarone hydrochloride Injection
150mg/3ml Taj Pharma.
Amiodarone hydrochloride Injection
450mg/9ml Taj Pharma.
2. Qualitative and quantitative
composition
a) Amiodarone hydrochloride Injection
150mg/3ml
Each ml contains:
Amiodarone Hydrochloride 50mg
Polysorbate 80 100mg
Benzyl alcohol 20.2mg
Water for injection q.s
a) Amiodarone hydrochloride Injection
450mg/9ml
Each ml contains:
Amiodarone Hydrochloride 50mg
Polysorbate 80 100mg
Benzyl alcohol 20.2mg
Water for injection q.s
For excipients, see 6.1
3. Pharmaceutical form
Solution for injection.
4. Clinical particulars
4.1 Therapeutic indications
Treatment should be initiated and normally
monitored only under hospital or specialist
supervision. Amiodarone hydrochloride
Intravenous is indicated only for the
treatment of severe rhythm disorders not
responding to other therapies or when other
treatments cannot be used.
Tachyarrhythmias associated with Wolff-
Parkinson-White syndrome.
All types of tachyarrhythmias including
supraventricular, nodal and ventricular
tachycardias; atrial flutter and fibrillation;
ventricular fibrillation; when other drugs
cannot be used.
Amiodarone hydrochloride Intravenous can
be used where a rapid response is required
or where oral administration is not possible.
4.2 Posology and method of
administration
Amiodarone hydrochloride Intravenous
should only be used when facilities exist for
cardiac monitoring, defibrillation, and
cardiac pacing.
Amiodarone hydrochloride Intravenous may
be used prior to DC cardioversion.
The standard recommended dose is 5mg/kg
bodyweight given by intravenous infusion
over a period of 20 minutes to 2 hours. This
should be administered as a dilute solution
in 250 ml 5% dextrose. This may be
followed by repeat infusion up to 1200 mg
(approximately 15 mg/kg bodyweight) in up
to 500 ml 5% dextrose per 24 hours, the rate
of infusion being adjusted on the basis of
clinical response (see section 4.4).
In extreme clinical emergency the drug may,
at the discretion of the clinician, be given as
a slow injection of 150 – 300 mg in 10 – 20
ml 5% dextrose over a minimum of 3
minutes. This should not be repeated for at
least 15 minutes. Patients treated in this way
with Amiodarone hydrochloride Intravenous
must be closely monitored, e.g. in an
intensive care unit (see section 4.4).
Changeover from Intravenous to Oral
therapy
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As soon as an adequate response has been
obtained, oral therapy should be initiated
concomitantly at the usual loading dose (i.e.
200 mg three times a day). Amiodarone
hydrochloride Intravenous should then be
phased out gradually.
Paediatric population
The safety and efficacy of amiodarone in
children has not been established.
Currently available data are described in
sections 5.1 and 5.2.
Due to the presence of benzyl alcohol,
intravenous amiodarone is contraindicated in
neonates, infants and children up to 3 years
old.
Elderly
As with all patients it is important that the
minimum effective dose is used. Whilst
there is no evidence that dosage
requirements are different for this group of
patients they may be more susceptible to
bradycardia and conduction defects if too
high a dose is employed. Particular attention
should be paid to monitoring thyroid
function (see sections 4.3, 4.4 and 4.8).
Cardiopulmonary resuscitation
The recommended dose for ventricular
fibrillations/pulseless ventricular tachycardia
resistant to defibrillation is 300 mg (or 5
mg/kg body-weight) diluted in 20 ml 5%
dextrose and rapidly injected. An additional
150 mg (or 2.5 mg/kg body-weight) IV dose
may be considered if ventricular fibrillation
persists.
See section 6.2 for information on
incompatibilities.
4.3 Contraindications
• Sinus bradycardia and sino-atrial heart
block. In patients with severe conduction
disturbances (high grade AV block,
bifascicular or trifascicular block) or sinus
node disease, Amiodarone hydrochloride
should be used only in conjunction with a
pacemaker.
• Evidence or history of thyroid dysfunction.
Thyroid function tests should be performed
where appropriate prior to therapy in all
patients.
• Severe respiratory failure, circulatory
collapse, or severe arterial hypotension;
hypotension, heart failure and
cardiomyopathy are also contra-indications
when using Amiodarone hydrochloride
Intravenous as a bolus injection.
• Known hypersensitivity to iodine or to
amiodarone, or to any of the excipients.
(One ampoule contains approximately 56mg
iodine).
• The combination of Amiodarone
hydrochloride with drugs which may induce
torsades de pointes is contra-indicated (see
section 4.5).
• Due to the content of benzyl alcohol,
Amiodarone hydrochloride Intravenous is
contraindicated in newborns or premature
neonates, infants and children up to 3 years
old.
• Pregnancy - except in exceptional
circumstances (see section 4.6)
• Lactation (see section 4.6)
All these above contra-indications do not
apply to the use of amiodarone for
cardiopulmonary resuscitation of shock
resistant ventricular fibrillation.
4.4 Special warnings and precautions for
use
Amiodarone injection contains benzyl
alcohol (20 mg/ml). Benzyl alcohol may
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cause toxic reactions and allergic reactions
in infants and children up to 3 years old.
The administration of medications
containing benzyl alcohol to newborns or
premature neonates has been associated with
a fatal “Gasping Syndrome” (symptoms
include a striking onset of gasping
syndrome, hypotension, bradycardia and
cardio-vascular collapse). As benzyl alcohol
may cross the placenta, solution for injection
should be used with caution in pregnancy.
Amiodarone hydrochloride Intravenous
should only be used in a special care unit
under continuous monitoring (ECG and
blood pressure).
IV infusion is preferred to bolus due to the
haemodynamic effects sometimes associated
with rapid injection (see section 4.8).
Circulatory collapse may be precipitated by
too rapid administration or overdosage
(atropine has been used successfully in such
patients presenting with bradycardia).
Do not mix other preparations in the same
syringe. Do not inject other preparations in
the same line. If Amiodarone hydrochloride
should be continued, this should be via
intravenous infusion (see section 4.2).
Repeated or continuous infusion via
peripheral veins may lead to injection site
reactions (see section 4.8). When repeated or
continuous infusion is anticipated,
administration by a central venous catheter
is recommended.
When given by infusion Amiodarone
hydrochloride may reduce drop size and, if
appropriate, adjustments should be made to
the rate of infusion.
Anaesthesia (see section 4.5):
Before surgery, the anaesthetist should be
informed that the patient is taking
amiodarone.
Cardiac disorders:
Caution should be exercised in patients with
hypotension and decompensated
cardiomyopathy and severe heart failure
(also see section 4.3).
Amiodarone has a low pro-arrhythmic
effect. Onsets of new arrhythmias or
worsening of treated arrhythmias,
sometimes fatal, have been reported. It is
important, but difficult to differentiate a lack
of efficacy of the drug from a proarrhythmic
effect, whether or not this is associated with
a worsening of the cardiac condition.
Proarrhythmic effects generally occur in the
context of QT prolongation factors such as
drug interactions and/or electrolytic
disorders (see sections 4.5 and 4.8). Despite
QT interval prolongation, amiodarone
exhibits a low torsadogenic activity.
Too high a dosage may lead to severe
bradycardia and to conduction disturbances
with the appearance of an idioventricular
rhythm, particularly in elderly patients or
during digitalis therapy. In these
circumstances, Amiodarone hydrochloride
treatment should be withdrawn. If necessary,
beta-adreno-stimulants or glucagon may be
given. Because of the long half-life of
amiodarone, if bradycardia is severe and
symptomatic the insertion of a pacemaker
should be considered.
The pharmacological action of amiodarone
induces ECG changes: QT prolongation
(related to prolonged repolarisation) with the
possible development of U-waves and
deformed T-waves; these changes do not
reflect toxicity.
Severe bradycardia (see section 4.5):
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Cases of severe, potentially life-threatening
bradycardia and heart block have been
observed when amiodarone is used in
combination with sofosbuvir in combination
with another hepatitis C virus (HCV) direct
acting antiviral (DAA), such as daclatasvir,
simeprevir, or ledipasvir. Therefore,
coadministration of these agents with
amiodarone is not recommended.
If concomitant use with amiodarone cannot
be avoided, it is recommended that patients
are closely monitored when initiating
sofosbuvir in combination with other DAAs.
Patients who are identified as being at high
risk of bradyarrhythmia should be
continuously monitored for at least 48 hours
in an appropriate clinical setting after
initiation of the concomitant treatment with
sofosbuvir.
Due to the long half-life of amiodarone,
appropriate monitoring should also be
carried out for patients who have
discontinued amiodarone within the past few
months and are to be initiated on sofosbuvir
alone or in combination with other direct
DAAs.
Patients receiving these hepatitis C
medicines with amiodarone, with or without
other medicines that lower heart rate, should
be warned of the symptoms of bradycardia
and heart block and should be advised to
seek urgent medical advice if they
experience them.
Primary graft dysfunction (PGD) post
cardiac transplant:
In retrospective studies, amiodarone use in
the transplant recipient prior to heart
transplant has been associated with an
increased risk of PGD.
PGD is a life-threatening complication of
heart transplantation the presents as a left,
right or biventricular dysfunction occurring
within the first 24 hours of transplant
surgery for which there is no identifiable
secondary cause (see section 4.8). Severe
PGD may be irreversible.
For patients who are on the heart transplant
waiting list, consideration should be given to
use an alternative antiarrhythmic drug as
early as possible before transplant.
Endocrine disorders (see section 4.8):
Amiodarone IV may induce
hyperthyroidism, particularly in patients
with a personal history of thyroid disorders
or patients who are taking/have previously
taken oral amiodarone. Serum usTSH level
should be measured when thyroid
dysfunction is suspected.
Amiodarone contains iodine and thus may
interfere with radio-iodine uptake. However,
thyroid function tests (free-T3, free-T4,
usTSH) remain interpretable. Amiodarone
inhibits peripheral conversion of
levothyroxine (T4) to triiodothyronine (T3)
and may cause isolated biochemical changes
(increase in serum free-T4, free-T3 being
slightly decreased or even normal) in
clinically euthyroid patients. There is no
reason in such cases to discontinue
amiodarone treatment if there is no clinical
or further biological (usTSH) evidence of
thyroid disease.
Respiratory, thoracic and mediastinal
disorders (see section 4.8):
Onset of dyspnoea or non-productive cough
may be related to pulmonary toxicity such as
interstitial pneumonitis. Very rare cases of
interstitial pneumonitis have been reported
with intravenous amiodarone. When the
diagnosis is suspected, a chest X-ray should
be performed. Amiodarone therapy should
be re-evaluated since interstitial pneumonitis
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is generally reversible following early
withdrawal of amiodarone, and
corticosteroid therapy should be
considered (see section 4.8). Clinical
symptoms often resolve within a few weeks
followed by slower radiological and lung
function improvement. Some patients can
deteriorate despite discontinuing
Amiodarone hydrochloride. Fatal cases of
pulmonary toxicity have been reported.
Very rare cases of severe respiratory
complications, sometimes fatal, have been
observed usually in the period immediately
following surgery (adult acute respiratory
distress syndrome); a possible interaction
with a high oxygen concentration may be
implicated (see sections 4.5 and 4.8).
Hepatobiliary disorders (see section 4.8):
Severe hepatocellular insufficiency may
occur within the first 24 hours of IV
amiodarone and may sometimes be fatal.
Close monitoring of transaminases is
therefore recommended as soon as
amiodarone is started.
Severe bullous reactions:
Life-threatening or even fatal cutaneous
reactions Stevens-Johnson syndrome (SJS),
Toxic Epidermal Necrolysis (TEN) (see
section 4.8). If symptoms or signs of SJS,
TEN (e.g. progressive skin rash often with
blisters or mucosal lesions) are present
amiodarone treatment should be
discontinued immediately.
Eye disorders (see section 4.8):
If blurred or decreased vision occurs,
complete ophthalmologic examination
including fundoscopy should be promptly
performed. Appearance of optic neuropathy
and/or optic neuritis requires amiodarone
withdrawal due to the potential progression
to blindness.
Drug interactions (see section 4.5):
Concomitant use of amiodarone with the
following drugs is not recommended; beta-
blockers, heart rate lowering calcium
channel inhibitors (verapamil, diltiazem),
stimulant laxative agents which may cause
hypokalaemia.
Increased plasma levels of flecainide have
been reported with co-administration of
amiodarone. The flecainide dose should be
reduced accordingly, and the patient closely
monitored.
4.5 Interaction with other medicinal
products and other forms of interaction
Drugs inducing “Torsade de Pointes” or
prolonging the QT interval
Some of the more important drugs that
interact with amiodarone include warfarin,
digoxin, phenytoin and any drug which
prolongs the QT interval.
Combined therapy with the following drugs
which prolong the QT interval is contra-
indicated (see section 4.3) due to the
increased risk of torsade de pointes; for
example:
• Class Ia anti-arrhythmic drugs e.g.
quinidine, procainamide, disopyramide
• Class III anti-arrhythmic drugs e.g. sotalol,
bretylium
• intravenous erythromycin, co-trimoxazole
or pentamidine injection
• some anti-psychotics e.g. chlorpromazine,
thioridazine, fluphenazine, pimozide,
haloperidol, amisulpiride and sertindole
• lithium and tricyclic anti-depressants e.g.
doxepin, maprotiline, amitriptyline
• certain antihistamines e.g. terfenadine,
astemizole, mizolastine
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• anti-malarials e.g. quinine, mefloquine,
chloroquine, halofantrine
• moxifloxacin
Fluoroquinolones
There have been rare reports of QTc interval
prolongation, with or without torsade de
pointes, in patients taking amiodarone with
fluoroquinolones. Concomitant use of
amiodarone with fluoroquinolones should be
avoided (concomitant use with moxifloxacin
is contra-indicated, see above).
Drugs lowering heart rate, causing
automaticity or conduction disorders.
Combined therapy with the following drugs
is not recommended:
• Beta blockers and certain calcium channel
inhibitors (diltiazem, verapamil);
potentiation of negative chronotropic
properties and conduction slowing effects
may occur.
• Stimulant laxatives, which may cause
hypokalaemia thus increasing the risk of
torsade de pointes; other types of laxatives
should be used.
Caution should be exercised over combined
therapy with the following drugs which may
also cause hypokalaemia and/or
hypomagnesaemia, e.g. diuretics, systemic
corticosteroids, tetracosactide, intravenous
amphotericin B.
In cases of hypokalaemia, corrective action
should be taken, and QT interval monitored.
In case of torsade de pointes antiarrhythmic
agents should not be given; pacing may be
instituted, and IV magnesium may be used.
General anaesthesia
Caution is advised in patients undergoing
general anaesthesia or receiving high dose
oxygen therapy.
Potentially severe complications have been
reported in patients taking amiodarone
undergoing general anaesthesia: bradycardia
unresponsive to atropine, hypotension,
disturbances of conduction, decreased
cardiac output.
Very rare cases of severe respiratory
complications (adult acute respiratory
distress syndrome), sometimes fatal, have
been observed usually in the period
immediately following surgery. A possible
interaction with a high oxygen concentration
may be implicated.
Effect of Amiodarone hydrochloride on
other medicinal products
Amiodarone and/or its metabolite,
desethylamiodarone, inhibit CYP1A1,
CYP1A2, CYP3A4, CYP2C9, CYP2D6 and
P-glycoprotein and may increase exposure
of their substrates.
Due to the long half-life of amiodarone,
interactions may be observed for several
months after discontinuation of amiodarone.
PgP Substrates
Amiodarone is a P-gp inhibitor. Co
administration with P-gp substrates is
expected to result in an increase in their
exposure.
Digoxin
Administration of Amiodarone
hydrochloride to a patient already receiving
digoxin will bring about an increase in the
plasma digoxin concentration and thus
precipitate symptoms and signs associated
with high digoxin levels; disturbances in
automaticity (excessive bradycardia), a
synergistic effect on heart rate and
atrioventricular conduction may occur.
Clinical, ECG and biological monitoring is
recommended to observe for signs of
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digitalis toxicity and digoxin dosage should
be halved.
Dabigatran
Caution should be exercised when
amiodarone is co administered with
dabigatran due to the risk of bleeding. It
may be necessary to adjust the dosage of
dabigatran as per its label.
CYP2C9 substrates:
Amiodarone raises the plasma
concentrations of CYP 2C9 substrates such
as oral anticoagulants (warfarin) and
phenytoin by inhibition of the cytochrome
P450 2C9.
Warfarin
The dose of warfarin should be reduced
accordingly. More frequent monitoring of
prothrombin time both during and after
amiodarone treatment is recommended.
Phenytoin
Phenytoin dosage should be reduced if signs
of overdosage appear, and plasma levels
may be measured.
CYP2D6 substrates
Flecainide
Given that flecainide is mainly metabolised
by CYP 2D6, by inhibiting this isoenzyme,
amiodarone may increase flecainide plasma
levels; it is advised to reduce the flecainide
dose by 50% and to monitor the patient
closely for adverse effects. Monitoring of
flecainide plasma levels is strongly
recommended in such circumstances.
CYP P450 3A4 substrates
When drugs are co-administered with
amiodarone, an inhibitor of CYP 3A4, this
may result in a higher level of their plasma
concentrations, which may lead to a possible
increase in their toxicity:
• Ciclosporin: plasma levels of ciclosporin
may increase as much as 2-fold when used
in combination. A reduction in the dose of
ciclosporin may be necessary to maintain the
plasma concentration within the therapeutic
range.
• Statins: the risk of muscular toxicity (e.g.
rhabdomyolysis) is increased by
concomitant administration of amiodarone
with statins metabolised by CYP 3A4 such
as simvastatin, atorvastatin and lovastatin. It
is recommended to use a statin not
metabolised by CYP 3A4 when given with
amiodarone.
• Other drugs metabolised by cytochrome
P450 3A4: examples of such drugs are
lidocaine, tacrolimus, sildenafil, fentanyl,
midazolam, triazolam, dihydroergotamine
and ergotamine and colchine.
Interaction with substrates of other CYP 450
isoenzymes
In vitro studies show that amiodarone also
has the potential to inhibit CYP1A2,
CYP2C19 and CYP2D6 through its main
metabolite. When co-administered,
amiodarone would be expected to increase
the plasma concentration of drugs whose
metabolism is dependent upon CYP1A2,
CYP2C19 and CYP2D6.
Effect of other products on Amiodarone
hydrochloride
CYP3A4 inhibitors and CYP2C8 inhibitors
may have a potential to inhibit amiodarone
metabolism and to increase its exposure.
It is recommended to avoid CYP 3A4
inhibitors (e.g. grapefruit juice and certain
medicinal products) during treatment with
amiodarone.
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Grapefruit juice inhibits cytochrome P450
3A4 and may increase the plasma
concentration of amiodarone. Grapefruit
juice should be avoided during treatment
with oral amiodarone.
Other drug interactions with amiodarone
(see section 4.4)
Coadministration of amiodarone with
sofosbuvir in combination with another
HCV direct acting antiviral (such as
daclatasvir, simeprevir, or ledipasvir) is not
recommended as it may lead to serious
symptomatic bradycardia. The mechanism
for this bradycardia effect is unknown.
If coadministration cannot be avoided,
cardiac monitoring is recommended (see
section 4.4).
4.6 Pregnancy and lactation
Pregnancy
There are insufficient data on the use of
amiodarone during pregnancy in humans to
judge any possible toxicity. However, in
view of its effect on the foetal thyroid gland,
amiodarone is contraindicated during
pregnancy, except in exceptional
circumstances.
Lactation
Amiodarone is excreted into the breast milk
in significant quantities and breast-feeding is
contra-indicated.
4.7 Effects on ability to drive and use
machines
Not relevant
4.8 Undesirable effects
The following adverse reactions are
classified by system organ class and ranked
under heading of frequency using the
following convention: very common (≥
10%), common (≥ 1% and < 10%);
uncommon (≥ 0.1% and < 1%); rare (≥
0.01% and < 0.1%), very rare (< 0.01%), not
known (cannot be estimated from the
available data).
Blood and lymphatic system disorders:
In patients taking amiodarone there have
been incidental findings of bone marrow
granulomas. The clinical significance of this
is unknown.
Frequency not known: neutropenia,
agranulocytosis
Cardiac disorders:
Common: bradycardia, generally moderate.
Very rare:
• marked bradycardia, sinus arrest requiring
discontinuation of amiodarone, especially in
patients with sinus node dysfunction and/or
in elderly patients.
• onset of worsening of arrhythmia,
sometimes followed by cardiac arrest (see
sections 4.4 and 4.5).
Frequency not known: Torsade de pointes
(see 4.4 and 5.1).
Eye disorders:
Frequency not known: optic
neuropathy/neuritis that may progress to
blindness (see section 4.4).
Endocrine disorders:
Very rare: Syndrome of inappropriate
antidiuretic hormone secretion (SIADH)
Frequency not known: hyperthyroidism (see
section 4.4).
Gastrointestinal disorders:
Very rare: nausea
Pancreatitis/ acute pancreatitis
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General disorders and administration site
conditions:
Common: injection site reactions such as
pain, erythema, oedema, necrosis,
extravasation, infiltration, inflammation,
induration, thrombophlebitis, phlebitis,
cellulitis, infection, pigmentation changes.
Hepatobiliary disorders:
Very rare:
• isolated increase in serum transaminases,
which is usually moderate (1.5 – 3 times
normal range) at the beginning of therapy.
They may return to normal with dose
reduction or even spontaneously.
• acute liver disorders with high serum
transaminases and/or jaundice, including
hepatic failure, sometimes fatal (see section
4.4).
Immune system disorders:
Very rare: anaphylactic shock.
Frequency not known: angioneurotic
oedema (Quincke's Oedema)
Musculoskeletal and connective tissue
disorders
Frequency not known: Back pain
Nervous system disorders:
Very rare: benign intra-cranial hypertension
(pseudo tumor cerebri), headache.
Respiratory, thoracic and mediastinal
disorders:
Very rare:
• interstitial pneumonitis or fibrosis,
sometimes fatal (see section 4.4).
• severe respiratory complications (adult
acute respiratory distress syndrome),
sometimes fatal (see sections 4.4 and 4.5).
• bronchospasm and/or apnoea in case of
severe respiratory failure, and especially in
asthmatic patients.
Psychiatric disorders
Frequency not known: Confusional
state/delirium
Reproductive system and breast disorders
Frequency not known: libido decreased
Skin and subcutaneous tissue disorders:
Common: eczema
Very rare: sweating
Frequency not known: urticaria, severe skin
reactions sometimes fatal including toxic
epidermal necrolysis/Stevens-Johnson
syndrome, bullous dermatitis and Drug
Reaction with Eosinophilia and Systematic
Symptoms.
Vascular disorders:
Common: decrease in blood pressure,
usually moderate and transient. Cases of
hypotension or collapse have been reported
following overdosage or a too rapid
injection.
Very rare: hot flushes
Injury, poisoning and procedural
complaints:
Not known: primary graft dysfunction post
cardiac transplant (see section 4.4).
Reporting of suspected adverse reactions
Reporting suspected adverse reactions after
authorisation of the medicinal product is
important.
4.9 Overdose
There is no information regarding
overdosage with intravenous amiodarone.
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Little information is available regarding
acute overdosage with oral amiodarone. Few
cases of sinus bradycardia, heart block,
attacks of ventricular tachycardia, torsades
de pointes, circulatory failure and hepatic
injury have been reported.
In the event of overdose, treatment should
be symptomatic, in addition to general
supportive measures. The patient should be
monitored and if bradycardia occurs beta-
adrenostimulants or glucagon may be given.
Spontaneously resolving attacks of
ventricular tachycardia may also occur. Due
to the pharmacokinetics of amiodarone,
adequate and prolonged surveillance of the
patient, particularly cardiac status, is
recommended.
Neither amiodarone nor its metabolites are
dialysable.
5. Pharmacological properties
5.1 Pharmacodynamic properties
Cordarone is a product for the treatment of
tachyarrhythmias and has complex
pharmacological actions. Its effects are anti-
adrenergic (partial alpha and beta blocker).
It has haemodynamic effects (increased
blood flow and systemic/coronary
vasodilation). The drug reduces myocardial
oxygen consumption and has been shown to
have a sparing effect of rat myocardial ATP
utilisation, with decreased oxidative
processes. Amiodarone inhibits the
metabolic and biochemical effects of
catecholamines on the heart and inhibits
Na+
and K+
activated ATPase.
No controlled paediatric studies have been
undertaken.
In published studies the safety of
amiodarone was evaluated in 1118
paediatric patients with various arrhythmias.
The following doses were used in paediatric
clinical trials.
Oral
- Loading dose: 10 – 20 mg/kg/day for 7 –
10 days (or 500 mg/m2
/day if expressed per
square meter).
- Maintenance dose: the minimum effective
dosage should be used; according to
individual response, it may range between 5
– 10 mg/kg/day (or 250 mg/m2
/day if
expressed per square meter).
Intravenous
- Loading dose: 5 mg/kg body weight over
20 minutes to 2 hours.
- Maintenance dose: 10 – 15 mg/kg/day
from few hours to several days.
If needed, oral therapy may be initiated
concomitantly at the usual loading dose.
5.2 Pharmacokinetic properties
Amiodarone is metabolised mainly by
CYP3A4, and also by CYP2C8, however the
pharmacokinetics of amiodarone are unusual
and complex, and have not been completely
elucidated.
Absorption following oral administration is
variable and may be prolonged, with
enterohepatic cycling. The major metabolite
is desethylamiodarone. Amiodarone is
highly protein bound (> 95%). Renal
excretion is minimal and faecal excretion is
the major route. A study in both healthy
volunteers and patients after intravenous
administration of amiodarone reported that
the calculated volumes of distribution and
total blood clearance using a two-
compartment open model were similar for
both groups. Elimination of amiodarone
after intravenous injection appeared to be
biexponential with a distribution phase
lasting about 4 hours. The very high volume
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of distribution combined with a relatively
low apparent volume for the central
compartment suggests extensive tissue
distribution. A bolus IV injection of 400mg
gave a terminal T½ of approximately 11
hours.
Amiodarone and its metabolite,
desethylamiodarone, exhibit a potential in
vitro to inhibit CYP1A1, CYP1A2,
CYP2C9, CYP2C19, CYP2D6, CYP3A4,
CYP2A6, CYP2B6 and CYP 2C8.
Amiodarone and desethylamiodarone have
also a potential to inhibit some transporters
such as P-gp and organic cation transporter
(OCT2) (One study shows a 1.1% increase
in concentration of creatinine (an OCT 2
substrate). In vivo data describe amiodarone
interactions on CYP3A4, CYP2C9,
CYP2D6 and P-gp substrates.
No controlled paediatric studies have been
undertaken. In the limited published data
available in paediatric patients, there were
no differences noted compared to adults.
5.3 Preclinical safety data
In a 2-years carcinogenicity study in rats,
amiodarone caused an increase in thyroid
follicular tumours (adenomas and/or
carcinomas) in both sexes at clinically
relevant exposures. Since mutagenicity
findings were negative, an epigenic rather
than genotoxic mechanism is proposed for
this type of tumour induction. In the mouse,
carcinomas were not observed, but a dose-
dependent thyroid follicular hyperplasia was
seen. These effects on the thyroid in rats and
mice are most likely due to effects of
amiodarone on the synthesis and/or release
of thyroid gland hormones. The relevance of
these findings to man is low.
6. Pharmaceutical particulars
6.1 List of excipients
Benzyl alcohol,
Polysorbate and
Water for Injections.
6.2 Incompatibilities
Amiodarone hydrochloride Intravenous is
incompatible with saline and should be
administered solely in 5% dextrose solution.
Amiodarone hydrochloride Intravenous,
diluted with 5% dextrose solution to a
concentration of less than 0.6 mg/ml, is
unstable. Solutions containing less than 2
ampoules Amiodarone hydrochloride
Intravenous in 500 ml dextrose 5% are
unstable and should not be used.
The use of administration equipment or
devices containing plasticizers such as
DEHP (di-2-ethylhexylphthalate) in the
presence of amiodarone may result in
leaching out of DEHP. In order to minimise
patient exposure to DEHP, the final
amiodarone dilution for infusion should
preferably be administered through non
DEHP-containing sets.
6.3 Shelf life
24 months.
6.4 Special precautions for storage
Do not store above 25°C. Store in the
original container.
6.5 Nature and contents of container
Amiodarone hydrochloride 150 mg/3 ml
Solution for Injection is supplied in boxes
containing 6 or 10 glass ampoules.
Not all pack sizes may be marketed.
6.6 Special precautions for disposal and
other handling
Refer to 4.2 above.
7.Manufactured in India by:
TAJ PHARMACEUTICALS LTD.
Mumbai, India
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at:Gujarat, INDIA.
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