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This presentation covers very selected and important points regarding the corticosterois and short explanation of the UV and IR spectra of the drug Prednisolone.
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This presentation covers very selected and important points regarding the corticosterois and short explanation of the UV and IR spectra of the drug Prednisolone.
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Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
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RX
AMODIAQUINE
HYDROCHLORIDE
TABLETS
150MG
1.NAME OF THE MEDICINAL PRODUCT
Amodiaquine hydrochloride tablets.
2. QUALITATIVE AND QUANTITATIVE
COMPOSITION
Each tablet contains 150 mg amodiaquine
(equivalent to 196mg of amodiaquine
hydrochloride).
For a full list of excipients, see section 6.1.
3. PHARMACEUTICAL FORM
Film-coated round tablets with yellow cores.
The tablets should not be divided.
4. CLINICAL PARTICULARS
4.1 Therapeutic indications
Amodiaquine hydrochloride tablets is indicated
for the treatment of uncomplicated cases of
malaria.
When prescribing amodiaquine for malaria
caused by Plasmodium falciparum, the
following should be considered:
• Resistance of Plasmodium falciparum to 4-
aminoquinolines (e.g. amodiaquine,
chloroquine) is widespread. Therefore it is
recommended to use amodiaquine in
combination with another antimalarial,
preferably an artemisinin derivative. Treatment
of P.falciparum malaria with amodiaquine alone
should be used only under exceptional
circumstances (e.g. unavailability of appropriate
combination partners)
• Amodiaquine is effective against some
chloroquine-resistant strains of Plasmodium
falciparum, although there is cross-resistance. In
areas with high rates of chloroquineresistant P.
falciparum strains, the remaining activity of
amodiaquine in the specific region should be
known before starting treatment.
The most recent official guidelines on the
appropriate use of antimalarial agents and local
information on the prevalence of resistance to
antimalarial drugs must be taken into
consideration for deciding on the
appropriateness of therapy with Amodiaquine
hydrochloride tablets.
4.2 Posology and method of administration
For oral use.
Artemisinin-based Combination Therapy (ACT)
Dosage in combination therapy is 10 mg/kg/day
(range 7.5 to 15 mg/kg/day).
Age Amodiaquine 150 mg (number of
tablets)
Day 1 Day 2 Day 3
1-6 years 150 mg (1) 150 mg (1) 150 mg (1)
7-13 years 300 mg (2) 300 mg (2) 300 mg (2)
Over 13
years
600 mg (4) 600 mg (4) 600 mg (4)
Children
Amodiaquine hydrochloride tablets should not
be used in children less than 1 year of age, as
appropriate dose adjustments cannot be achieved
with this formulation.
Renal/hepatic impairment:
No data are available on dosing in hepatically or
renally impaired patients (see section 4.4).
Monotherapy (not recommended, see section
4.1)
Adults and adolescents (> 40 kg):
Dosage should be adapted according to body
weight:
The total dose for the duration of therapy should
be 35 mg/kg bodyweight (BW) of amodiaquine
base administered within three days:
Morning Evening
Day 1 7.5 mg /kg BW 7.5 mg /kg BW
Day 2 5 mg /kg BW 5 mg /kg BW
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Day 3 5 mg /kg BW 5 mg /kg BW
Method of administration
The tablets should be swallowed with water and
taken after a meal.
Should vomiting occur within half an hour after
dosing, a repeated dose of Amodiaquine
hydrochloride tabletsis to be taken. In case of
further vomiting, treatment for severe malaria
should be considered.
4.3 Contraindications
Hypersensitivity to active substances or to any
of the excipients,
History of liver injury during treatment with
amodiaquine,
Previous haematological event during
treatment with amodiaquine,
Retinopathy (in case of frequent treatment).
Amodiaquine hydrochloride tablets must not be
used for malaria prophylaxis, since it may result
in agranulocytosis and severe hepatotoxicity (see
section 4.4).
4.4 Special Warnings and precautions for use
Amodiaquine hydrochloride tablets has not been
evaluated for the treatment of complicated
malaria and is therefore not recommended.
It is not known, whether the toxicity of
amodiaquine, observed with prophylactic use
(i.e. agranulocytosis, hepatotoxicity), may also
develop after repeated cycles of curative
treatment.
Amodiaquine hydrochloride tablets has not been
studied specifically in patients with
thalassaemia, sickle cell anaemia or G6PD-
deficiency. Since haemolysis and acute renal
failure have been reported in a few patients with
glucose 6-phosphate dehydrogenase deficiency
receiving chloroquine, this should also
considered when using amodiaquine.
Renal/hepatic impairment
In the absence of specific clinical studies,
caution should be exercised in patients with
renal or hepatic impairment.
Because amodiaquine may concentrate in liver,
the drug should be used with caution in patients
with hepatic disease or alcoholism, and in
patients receiving hepatotoxic drugs.
Since chloroquine, a 4-aminoquinoline, has been
reported to provoke seizures, amodiaquine
should be used with caution in patients with a
history of epilepsy.
Symptoms suggestive of the following diseases
should be carefully monitored:
Hepatitis, pre-icteric phase and especially
when jaundice has developed,
Agranulocytosis (as suggested, for instance, by
a clinical condition including fever and/or
tonsillitis and/or mouth ulcers).
When these symptoms develop or exacerbate
during the course of therapy with Amodiaquine
hydrochloride tablets, laboratory tests for liver
function and/or blood cell counts should be
performed at once. In such cases, continuation of
treatment with amodiaquine increases the risk of
death.
Cardiovascular effects have been reported with
other amino-4-quinoline derivatives during high
dose treatment. There is no evidence that an
overdose of amodiaquine causes any of the
lifethreatening cardiovascular complications
often seen after an overdose of chloroquine.
However, by chemical class analogy, caution
should be exercised with patients who have
recently taken other antimalarial drug with
cardiovascular side effects (quinine, quinidine,
halofantrine, piperaquine, lumefantrine,
mefloquine) or those who are under treatment
with cardiovascular drugs or other drugs with
the potential to prolong the QT interval (see
section 4.9).
Caution is also advised when amodiaquine is
combined with drugs known to inhibit CYP 2C8
isoenzyme (see sections 4.5 and 5.2).
Co-administration of Amodiaquine
hydrochloride tablets and efavirenz should be
avoided, since this combination has been noted
to cause marked hepatotoxicity
4.5 Interaction with other medicinal products
and other forms of interaction
Single dose data have shown that the co-
administration of artesunate and amodiaquine
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leads to a 47% decrease in the Cmax of the
active metabolite dihydroartemisinin, and a 17%
decrease of its AUC, relative to what is seen
when artesunate is administered alone. If
Amodiaquine hydrochloride tablets is used in
the presence of amodiaquine resistance, this
might further compromise the antimalarial
activity of this artemisinin-based combination
(see also sections 4.1, 4.4 and 5.1).
Antacid-containing products, e.g. magnesium
trisilicate and kaolin, are known to decrease the
gastrointestinal absorption of amodiaquine when
administered simultaneously.
The incidence of hepatic and haematological
adverse events is higher when amodiaquine is
combined with other drugs with potential for
liver and/or haematological toxicity.
Interactions with drugs used for treatment of
HIV and/or tuberculosis may occur, though little
clinical data is available. Prescribers should be
vigilant for adverse events potentially related to
such interactions, including liver toxicity and
neutropenia. HIV-infected patients receiving
zidovudine or efavirenz should, if possible,
avoid amodiaquine-containing regimens.
In the absence of clinical data, Amodiaquine
hydrochloride tablets is not recommended when
administered concomitantly with drugs known to
inhibit the liver enzymes cytochrome (CYP)
2C8 (e.g. trimethoprim, ketoconazole, ritonavir,
saquinavir, lopinavir, gemfibrozil, montelukast)
(see section 5.2).
Though no pharmacokinetic interactions have
been documented, amodiaquine and
desethylamodiaquine inhibit CYP 2D6 in vitro
and may cause clinically significant interactions
with some β-blockers, antidepressants, and
antipsychotic drugs. Caution should be exercised
when co-administration of these agents with
Amodiaquine hydrochloride tablets is deemed
necessary.
A statistically significant decrease in
dihydroartemisinin (DHA), the main active
metabolite of artesunate, occurs with
concomitant use of artesunate and amodiaquine
(Cmax decreased 47%, AUC0-inf decreased
17%) (see section 5.2).
4.6 Fertility, Pregnancy and lactation
Pregnancy
Malaria is known to be particularly hazardous
during pregnancy. The benefits and risks of
therapy with Amodiaquine hydrochloride tablets
to mother and foetus must be assessed by the
prescriber.
The safety of amodiaquine in pregnant women
has not been conclusively established, although
many years of experience with the drug have not
indicated any major teratogenicity.
During 1st trimester of pregnancy, Amodiaquine
hydrochloride tablets should not be used unless
clearly necessary e.g. if treatment is life-saving
for the mother, and if another antimalarial is not
suitable or not tolerated.
During 2nd or 3rd trimesters of pregnancy,
Amodiaquine hydrochloride tablets may be used
with caution, only if other antimalarials are
unsuitable.
Lactation
The amounts of antimalarials in breast milk are
small. Therefore, lactating women can receive
Amodiaquine hydrochloride tablets for malaria
treatment.
4.7 Effects on ability to drive and use
machines
No studies on the effects on the ability to drive
and use machines have been performed. Patients
receiving amodiaquine should be warned that
undesirable effects such as dizziness and visual
disorders may occur, in which case they should
not drive or use machines.
4.8 Undesirable Effects
The adverse events are listed below by body
system, organ class and absolute frequency.
They are not based on adequately sized
randomized controlled trials, but on published
literature data generated mostly during post-
approval use. Since frequency estimates are
highly variable across the studies, no frequencies
are given for these events. When frequency
estimates are available they are defined as
follows: very common (≥1/10), common
(≥1/100, <1/10), uncommon (≥1/1000,<1/100),
rare (≥1/10,000,<1/1000), very rare (≤1/10,000).
For some of these events, it is unclear whether
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they are related to amodiaquine or occur as a
result of the underlying disease process.
The following adverse reactions have been
reported with amodiaquine, especially at higher
doses and/or during prolonged treatment:
Blood and lymphatic system disorders
leucopenia and neutropenia (agranulocytosis).
Nervous system disorders
Very common: weakness, headache, dizziness
Rare: neuromyopathy
Gastrointestinal disorders
Very common: anorexia, nausea, vomiting,
abdominal pain, diarrhoea
Eye disorders
transient accommodation disorders, corneal
opacifications regressive once treatment is
stopped
Very rare: irreversible retinopathy justifying
ophthalmologist attention
Hepato-biliary disorders:
severe and sometimes fatal hepatitis.
Development of hepatic disorders may be
delayed.
Skin and subcutaneous disorders:
slate-gray pigmentation, notably affecting the
fingers and mucous membranes Common:
pruritus
4.9 Overdose
In cases of suspected overdose, the patient
should be urgently transferred to a specialized
unit where appropriate monitoring and
symptomatic and supportive therapy should be
applied.
The dangerous dose of amodiaquine cannot be
stated precisely because of the low number of
known cases; by analogy with chloroquine, it
can be estimated at around 2 grams as a single
administration in adults
Symptoms: headache, dizziness, visual
disorders, cardiovascular collapse and
convulsions, followed by early respiratory and
cardiac arrest.
5. PHARMACOLOGICAL PROPERTIES
5.1 Pharmacodynamic properties
Category: Antimalarial.
Amodiaquine is a synthetic 4-aminoquinoline
antimalarial. Its activity is characterized by a
schizonticidal action on Plasmodium falciparum,
Plasmodium vivax, Plasmodium ovale and
Plasmodium malaria by destroying
intraerythrocytic forms.
The mechanism of action of 4-aminoquinoline
derivatives against plasmodium is not yet
completely known. It is nonetheless accepted
that these derivatives, one of which is
amodiaquine, penetrate the infected red blood
cells and prevent the parasite from polymerizing
haeme into an insoluble product called
haemozoin, leading to parasite death.
Strains of Plasmodium falciparum resistant to 4-
aminoquinolines (chloroquine, amodiaquine) are
present in many areas, and their geographical
distribution is constantly changing. However,
amodiaquine remains active against some
chloroquine-resistant P. falciparum strains.
5.2 Pharmacokinetic properties
Absorption
After oral administration in healthy subjects,
amodiaquine is quickly absorbed and
biotransformed into its main active form,
desethylamodiaquine. The absolute
bioavailability of amodiaquine is not known.
Following single dose administration of three
Amodiaquine Hydrochloride Tablets (i.e. 450
mg amodiaquine base) in healthy volunteers, the
mean (±SD) amodiaquine Cmax value was
30.3±12.6 ng/ml and the corresponding value for
AUC0-t was 309.4±76.0 ng.h/ml. The median
(±SD) amodiaquine tmax value was 0.91±1.13h.
Distribution
The volume of distribution of amodiaquine is
estimated at 20 to 40 l/kg.
Desethylamodiaquine, the main metabolite of
amodiaquine, is assumed to be the main active
form after oral administration. It is mainly found
in blood, at much higher concentrations than
unchanged amodiaquine. Its concentration in
whole blood is 4-6 times higher than in plasma.
Metabolism
The hepatic first pass metabolism of
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amodiaquine is high, with formation of the
active metabolite, desethylamodiaquine,
presumably via the CYP2C8 isoenzyme. Further
metabolism includes oxidation and
glucuronoconjugation.
Elimination
Amodiaquine is eliminated principally through
biotransformation with only around 2% excreted
unchanged in urine. Desethylamodiaquine is
slowly eliminated with a terminal half-life of 9-
18 days.
Food effect
When a fixed dose combination of
amodiaquine/artesunate was taken with a high
fat meal in healthy volunteers, the Cmax and
AUC(0-t) of the active metabolite
desethylamodiaquine (DeAQ) increased 18%
and 12% respectively, compared to fasting.
5.3 Preclinical safety data
General toxicity
Single dose toxicity studies reported a LD50
(mouse intraperitoneal) of 225mg/kg; LD50
(mouse oral) of 550mg/kg and a LD0 (mouse
intraperitoneal) of 137 mg/kg. Histopathological
changes (pigmentation) were seen in the heart at
30 mg/kg/day in rats. The statistically significant
effects seen in vitro on ion channels in the heart
at 0.1 µM in the hERG current (expressed in
Human Embryonic Kidney cells) as well as the
increase in QRS complex and QT interval
durations at concentrations higher than 0.1µM in
the isolated rabbit Purkinje fibres appeared to be
due to a non-specific multi-ion channel
blockade. Transient prolongation of QT interval
duration were observed at 30 mg/kg
administered orally. This dose level corresponds
to approximately 2-fold the maximum
recommended therapeutic dose. At the dose
level of 100 mg/kg administered orally (about
6.7 fold the maximum recommended therapeutic
dose), also slight respiratory depressant and
natriuretic effects were noted.
Pigmentations were also seen in liver, kidney
and thyroid glands in rats as well as in kidneys,
liver and lymph nodes in dogs (at doses of 25
mg/kg/day). Also an increase in haemosiderosis
in the spleen and bone marrow as well as thymus
lymphoid depletion were observed.
Genotoxicity
In vitro (Ames test) and in vivo tests (sister
chromatid exchange and chromosome aberration
tests) showed that amodiaquine, like
chloroquine, has both, a mutagenic and a
clastogenic potential.
Carcinogenicity
No studies on the carcinogenic potential of
amodiaquine have been conducted.
Reproductive toxicity
No data on toxicity on the reproductive system
and embryofetal development is available for
amodiaquine alone. The combination of
amodiaquine and artesunate did not demonstrate
any particular effects on fertility or associated
parameters. In the peri-postnatal study, the
offspring from the F1 generation did not show
any effect on sexual development, and despite
an early slowing of bodyweight increases with
some effect on testicular and epididymal
weights, no sequelae were noted on reproductive
capacity.
6. PHARMACEUTICAL PARTICULARS
6.1 List of excipients
Tablet core
Dextrin
Hydroxypropyl cellulose
Magnesium stearate
Microcrystalline cellulose
Polysorbate 80
Sodium Starch Glycolate
Starch (corn)
Film coating
Methacrylic acid copolymer
6.2 Incompatibilities
Not applicable.
6.3 Shelf life
3 years
6.4 Special precautions for storage
Store below 30°C. Store in the original
container.
6.5 Nature and contents of container
PVC / Aluminium blister cards containing 2 x 6
tablets, packed in cardboard boxes.
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6.6 Special precautions for disposal and other
handling
Any unused product or waste material should be
disposed of in accordance with local
requirements.
7. MANUFACTURER:
Manufactured in India by:
TAJ PHARMACEUTICALS LTD.
Mumbai, India
At: 615, GIDC, Kerala, Bavla, Dist. Ahmedabad
438225, Gujarat, INDIA