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PERIODONTAL POCK
ET
DEFINITION:
The periodontal pocket is defined as a pathologically d
eepened gingiva sulcus. Deepening of gingiva sulcus may oc
cur by coronal movement of the gingiva margin, apical displa
cement of gingiva attachment or combination of above.
GINGIVA POCKET PERIODONTAL POCKET

Also known as pseudo pocket or rel
ative pocket or false pocket

Seen in the gingivitis

Formed by the gingiva enlargement
without extraction of the underlying p
eriodontal tissues.

The sulcus is deepened because of
increased bulk of gingiva

Also known as absolute or tr
ue pocket

Seen in periodontitis

Occurs with destruction of th
e supporting periodontal tiss
ues and loosening and exfoli
ation of the teeth.
CLASSIFICATION 1:
POCKET
Gingival pocket Periodontal pocket
Suprabony pocket Infrabony pocket
Suprabony pocket
Two types of Periodon
tal Pocket
Infrabony pocket
Gingival Pocket
SUPRABONY POCKET INFRABONY POCKET

Also known as Supracrestal or Supra
alveolar pocket.

Also known as Subcrestal or Intraalv
eolar pocket.

Bottom of the pocket is coronal to t
he underlying alveolar bone.

Bottom of the pocket is apical to th
e crest of the alveolar bone.

Lateral wall consist of the soft tissue
alone

Lateral wall consist of the soft tissue
and bone.

Pattern of destruction of bone is ho
rizontal

Pattern of destruction of bone is ver
tical
CLASSIFICATION 2:
According to the involved tooth surfaces
Involve one surface Involve more than
one surface
Originating on one tooth surface an
d twisting around the tooth to involv
e one or more additional surfaces (
But open into oral cavity on the surf
ace of its origin).
POCKET
Simple pocket Compound pocket Complex or Spiral pocket
PATHOGENESIS OF POCKET FORMATION
Presence of bacterial plaque on tooth surface
Marginal gingiva become inflamed
Gingiva sulcus deepens due to edematous enlargement of gingiva
Gingiva pocket
Anareobic organisms tend to colonise the subgingiva plaque
(Spirochaetes and motile rods)
(Due to an aerobic environment created in the pocket)
Large number of PMN leykocytes and macrophages migrates to th
e gingiva tissue in response to bacterial challenge
CLINICAL FEATURES
CLINICAL FEATURES CAUSES
1.

Bluish red discoloration of the gin
giva wall of pocket.

Flaccidity

A smooth, shiny surface

Pitting on pressure

Due to circulatory stagnation

Due to destruction of gingiva fibres

Due to atrophy of the epithelium and
edema

Due to edema and degeneration
3.

Bleeding on probing

Due to

increased vascularity

thinning and degeneration of the
epithelium

the proximity of the engorged
vessels to the inner surface.
4.

Probing is generally painful

Due to ulceration of the inner aspect
of the pocket wall.
5.

Pus may be present 
Due to suppurative inflammation
OTHER CLINICAL FEATURES
 Thickened marginal gingiva
 Loss of stippling
 Tooth mobility and diastema formation
HISTOPATHOLOGY
[I] Soft tissue wall/lateral wall
Epithelium:
Shows
1. Epithelial cells proliferate into the underlying connective tissu
es forming deep rete pegs
2. Micro ulcerations develops on soft tissue wall
3. Pocket epithelial is infiltrated by PMN’s and edematous fluid f
rom inflammed connective tissues.
4. Bacterial invasion in intercellular space of epithelium (eg. Gra
m negative organism)
Degenerative changes
Proliferative changes
CONNECTIVE TISSUE
1. Edematous.
2. Densely infiltrated with plasmecells (80%), lymphocyte
s and PMN leykocytes.
3. Vascularity 
4. B.V. dilated and engorged
5. Area of necrosis and degeneration
6. Suppuration is commonly seen
CONTENTS OF POCKET
1. Micro organisms
2. Bacterial products (enzymes and endotoxins)
3. GCF
4. Remnants of food
5. Salivary mycin
6. Desquamated epithelial cells
7. Leukocytes
8. Purulent exudates may be present .
Eg. deep pocket may have little or no pus and shallow po
cket may have extensive pus formation so pus is not an in
dication of the depth of the pocket.
[II] Root surface wall of the pocket
Root surface forms the medial wall of the pocket.
The root surface that gets expose to the oral environment
, as a result of periodontal attachment loss, undergoes follo
wing changes.
Structural changes Chemical changes Cytotonic changes
Structural changes
Exposure of cementum to the oral environment
Areas of demineralization
Root caries (Yellowish or light brown patch)
Soft and lethargy on probing
Patient feels severe sensitivity to thermal changes and sweets
Pulp exposure may occur in severe forms
Five zones can be seen at the bottom of the pocket
Also kn
own as
Plaque
free zo
ne
DIAGNOSIS/DETECTION OF POCKETS
1.Careful exploration with a periodontal probe – accurate method.
2.Radiograph:
supra
bony Pockets are not detected by radiographic examination because
pocket is a soft tissue change.
Disadvantages of radiograph:
 Radiograph indicates areas of bone loss where pocket may be su
spected, they do not show pocket presence or depth.
 Radiograph show no difference before or after pocket elimination
unless bone has been modified
Note: Gutta Percha points or Calibrated Silver points can be us
ed with radiograph to assist in determining the level of attach
ment of periodontal pocket.
POCKET PROBING
Two different pocket depths
Biologic or histologic depth
Distance between gingiva mar
gin and base of the pocket
Clinical or probing depth
Distance to which a probe pen
etrates into the pocket
Pocket depth versus level of attachment:
Pocket depth: Distance between base of the pocket and gingiv
a margins
Level of attachment loss: Distance between base of the pock
et and a fixed point on the crown such as the CET.
Level of attachment l
oss Pocket depth
PROBING TECHNIQUES
1.
BLEEDING ON PROBING
1. If gingiva is inflamed and the pocket epithelium is atrophic or ulcer
ated.
2. To test for bleeding after probing, the probe is carefully introduced
to the bottom of the pocket and gently moved laterally along the po
cket wall.
3. Bleeding may appear immediately after removal of the probe or ma
y be delayed a few seconds.
4. Depending on the severity of inflammation, bleeding can vary from
a tenuous red line along the gingiva sulcus to profuse bleeding.
PROBING AROUND IMPLANTS
Periimplantitis:
Periimplantitis can create pockets around implants
Plastic probe should be used instead of the usual steel probes
used for the natural dentition.
To prevent the scratching of the implant surface.
المحاضره 5 د  اسماء.pdf periodontal tissues

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المحاضره 5 د اسماء.pdf periodontal tissues

  • 2. DEFINITION: The periodontal pocket is defined as a pathologically d eepened gingiva sulcus. Deepening of gingiva sulcus may oc cur by coronal movement of the gingiva margin, apical displa cement of gingiva attachment or combination of above.
  • 3. GINGIVA POCKET PERIODONTAL POCKET  Also known as pseudo pocket or rel ative pocket or false pocket  Seen in the gingivitis  Formed by the gingiva enlargement without extraction of the underlying p eriodontal tissues.  The sulcus is deepened because of increased bulk of gingiva  Also known as absolute or tr ue pocket  Seen in periodontitis  Occurs with destruction of th e supporting periodontal tiss ues and loosening and exfoli ation of the teeth. CLASSIFICATION 1: POCKET Gingival pocket Periodontal pocket Suprabony pocket Infrabony pocket
  • 4. Suprabony pocket Two types of Periodon tal Pocket Infrabony pocket Gingival Pocket
  • 5. SUPRABONY POCKET INFRABONY POCKET  Also known as Supracrestal or Supra alveolar pocket.  Also known as Subcrestal or Intraalv eolar pocket.  Bottom of the pocket is coronal to t he underlying alveolar bone.  Bottom of the pocket is apical to th e crest of the alveolar bone.  Lateral wall consist of the soft tissue alone  Lateral wall consist of the soft tissue and bone.  Pattern of destruction of bone is ho rizontal  Pattern of destruction of bone is ver tical
  • 6. CLASSIFICATION 2: According to the involved tooth surfaces Involve one surface Involve more than one surface Originating on one tooth surface an d twisting around the tooth to involv e one or more additional surfaces ( But open into oral cavity on the surf ace of its origin). POCKET Simple pocket Compound pocket Complex or Spiral pocket
  • 7. PATHOGENESIS OF POCKET FORMATION Presence of bacterial plaque on tooth surface Marginal gingiva become inflamed Gingiva sulcus deepens due to edematous enlargement of gingiva Gingiva pocket Anareobic organisms tend to colonise the subgingiva plaque (Spirochaetes and motile rods) (Due to an aerobic environment created in the pocket) Large number of PMN leykocytes and macrophages migrates to th e gingiva tissue in response to bacterial challenge
  • 9. CLINICAL FEATURES CAUSES 1.  Bluish red discoloration of the gin giva wall of pocket.  Flaccidity  A smooth, shiny surface  Pitting on pressure  Due to circulatory stagnation  Due to destruction of gingiva fibres  Due to atrophy of the epithelium and edema  Due to edema and degeneration 3.  Bleeding on probing  Due to  increased vascularity  thinning and degeneration of the epithelium  the proximity of the engorged vessels to the inner surface. 4.  Probing is generally painful  Due to ulceration of the inner aspect of the pocket wall. 5.  Pus may be present  Due to suppurative inflammation
  • 10. OTHER CLINICAL FEATURES  Thickened marginal gingiva  Loss of stippling  Tooth mobility and diastema formation
  • 11. HISTOPATHOLOGY [I] Soft tissue wall/lateral wall Epithelium: Shows 1. Epithelial cells proliferate into the underlying connective tissu es forming deep rete pegs 2. Micro ulcerations develops on soft tissue wall 3. Pocket epithelial is infiltrated by PMN’s and edematous fluid f rom inflammed connective tissues. 4. Bacterial invasion in intercellular space of epithelium (eg. Gra m negative organism) Degenerative changes Proliferative changes
  • 12. CONNECTIVE TISSUE 1. Edematous. 2. Densely infiltrated with plasmecells (80%), lymphocyte s and PMN leykocytes. 3. Vascularity  4. B.V. dilated and engorged 5. Area of necrosis and degeneration 6. Suppuration is commonly seen
  • 13. CONTENTS OF POCKET 1. Micro organisms 2. Bacterial products (enzymes and endotoxins) 3. GCF 4. Remnants of food 5. Salivary mycin 6. Desquamated epithelial cells 7. Leukocytes 8. Purulent exudates may be present . Eg. deep pocket may have little or no pus and shallow po cket may have extensive pus formation so pus is not an in dication of the depth of the pocket.
  • 14. [II] Root surface wall of the pocket Root surface forms the medial wall of the pocket. The root surface that gets expose to the oral environment , as a result of periodontal attachment loss, undergoes follo wing changes. Structural changes Chemical changes Cytotonic changes
  • 15. Structural changes Exposure of cementum to the oral environment Areas of demineralization Root caries (Yellowish or light brown patch) Soft and lethargy on probing Patient feels severe sensitivity to thermal changes and sweets Pulp exposure may occur in severe forms
  • 16. Five zones can be seen at the bottom of the pocket Also kn own as Plaque free zo ne
  • 17. DIAGNOSIS/DETECTION OF POCKETS 1.Careful exploration with a periodontal probe – accurate method. 2.Radiograph: supra bony Pockets are not detected by radiographic examination because pocket is a soft tissue change. Disadvantages of radiograph:  Radiograph indicates areas of bone loss where pocket may be su spected, they do not show pocket presence or depth.  Radiograph show no difference before or after pocket elimination unless bone has been modified Note: Gutta Percha points or Calibrated Silver points can be us ed with radiograph to assist in determining the level of attach ment of periodontal pocket.
  • 18. POCKET PROBING Two different pocket depths Biologic or histologic depth Distance between gingiva mar gin and base of the pocket Clinical or probing depth Distance to which a probe pen etrates into the pocket
  • 19. Pocket depth versus level of attachment: Pocket depth: Distance between base of the pocket and gingiv a margins Level of attachment loss: Distance between base of the pock et and a fixed point on the crown such as the CET. Level of attachment l oss Pocket depth
  • 21. BLEEDING ON PROBING 1. If gingiva is inflamed and the pocket epithelium is atrophic or ulcer ated. 2. To test for bleeding after probing, the probe is carefully introduced to the bottom of the pocket and gently moved laterally along the po cket wall. 3. Bleeding may appear immediately after removal of the probe or ma y be delayed a few seconds. 4. Depending on the severity of inflammation, bleeding can vary from a tenuous red line along the gingiva sulcus to profuse bleeding. PROBING AROUND IMPLANTS Periimplantitis: Periimplantitis can create pockets around implants Plastic probe should be used instead of the usual steel probes used for the natural dentition. To prevent the scratching of the implant surface.