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Shock & Haemorrhage
Dr. Md. Nazmus Sakib
Junior Consultant
Email-sakibs23@gmail.com
After any complication the surgeon
may feel worry and concern for the
patient (91.5%), guilt (64.6%),
anxiety (68.3%) and disappointment
(63.4%).
Shock
• Shock is a systemic state of low tissue
perfusion that is in adequate for normal
cellular respiration.
Classification
• Haemorrahgic/ hypovolumic shock
• Cardiogenic shock
• Obstructive shock
• Distributive shock
• Endocrine shock
• Haemorrhagic shock- haemorrhage, dehydration ,
diarrhoea, third spacing of fluid.
• Cardiogenic shock-myocardial infarction, cardiac
dysrhythmia, blunt myocardial injury.
• Obstructive shock- decrease preload. Cardiac
temponade, tension pneumothorax, pulmonary
embolism.
• Distributive shock-septic shock, anaphylaxis,
spinal cord injury.
– Vasodilatation
– Low systemic vascular resistance
– Histamin release
• Endocrine shock- combintion of cardiogenic,
hypovolumic , distributive shock. Adrenal
insufficiency
Effect of organ failure
• Cardiac- cardiovascular failure
• Lung- acute respiratory distress syndrome
• Kidney-acute renal insufficiency
• Liver –liver failure and coagulopathy
• Brain- cerebral swelling and dysfunction
Compensated
shock
Decompensated
shock
• Mild shock
• Moderate shock
• Severe shock
Haemorrhage
Revealed haemorrhage
Concealed haemorrhage
 Surgical haemorrhage
 Non surgical haemorrhage
Primary haemorrhage
Reactionary haemorrhage (within 24 hours)
Secondary haemorrhage (7-14 days)
• Class I : <15%
• Class II : 15-20%
• Class III : 30-40%
• Class IV : >40%
Causes of reactionary
haemorrhage???
• Within 24 hours
• Dislodgement of clot
• Normalization of blood pressure
• Vasodilation
• Slippage of ligature
Monitoring for patient in shock
Minimum
• ECG
• Pulse oximetry
• Blood pressure
• Urine output
Additional modalities
• Central venous pressure
• Invasive blood pressure
• Cardiac output
• Base deficit and serum
lactate
Indication of blood transfusion
• Acute blood loss, to replace circulating volume and
maintain oxygen delivery;
• Perioperative anaemia, to ensure adequate oxygen
delivery during the perioperative phase;
• Symptomatic chronic anaemia, without haemorrhage
or impending surgery.
• Autologous transfusion
Complication of blood transfusion
• Single transfusion
• Incompatibility hemolytic transfusion reaction
• Febrile transfusion reaction
• Allergic reaction
• Infection (bac, hepatitis, HIV, malaria)
• Air embolism
• Thrombophlebitis
• Transfusion related acute lung injury
Massive transfusion
• Coagulopathy
• Hypocalcaemia
• Hyperkalaemia
• Hyperkalaemia
• hypothermia
Blood substitute
• Biomimeic- hemoglobin based
• Abiotic – perfluoro carbon based
3rd year lecture Haemorrhage and shock.pptx

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3rd year lecture Haemorrhage and shock.pptx

  • 1. Shock & Haemorrhage Dr. Md. Nazmus Sakib Junior Consultant Email-sakibs23@gmail.com
  • 2. After any complication the surgeon may feel worry and concern for the patient (91.5%), guilt (64.6%), anxiety (68.3%) and disappointment (63.4%).
  • 3. Shock • Shock is a systemic state of low tissue perfusion that is in adequate for normal cellular respiration.
  • 4.
  • 5. Classification • Haemorrahgic/ hypovolumic shock • Cardiogenic shock • Obstructive shock • Distributive shock • Endocrine shock
  • 6. • Haemorrhagic shock- haemorrhage, dehydration , diarrhoea, third spacing of fluid. • Cardiogenic shock-myocardial infarction, cardiac dysrhythmia, blunt myocardial injury. • Obstructive shock- decrease preload. Cardiac temponade, tension pneumothorax, pulmonary embolism.
  • 7.
  • 8.
  • 9. • Distributive shock-septic shock, anaphylaxis, spinal cord injury. – Vasodilatation – Low systemic vascular resistance – Histamin release
  • 10. • Endocrine shock- combintion of cardiogenic, hypovolumic , distributive shock. Adrenal insufficiency
  • 11. Effect of organ failure • Cardiac- cardiovascular failure • Lung- acute respiratory distress syndrome • Kidney-acute renal insufficiency • Liver –liver failure and coagulopathy • Brain- cerebral swelling and dysfunction
  • 13. • Mild shock • Moderate shock • Severe shock
  • 14.
  • 15.
  • 16. Haemorrhage Revealed haemorrhage Concealed haemorrhage  Surgical haemorrhage  Non surgical haemorrhage Primary haemorrhage Reactionary haemorrhage (within 24 hours) Secondary haemorrhage (7-14 days)
  • 17. • Class I : <15% • Class II : 15-20% • Class III : 30-40% • Class IV : >40%
  • 18. Causes of reactionary haemorrhage??? • Within 24 hours • Dislodgement of clot • Normalization of blood pressure • Vasodilation • Slippage of ligature
  • 19. Monitoring for patient in shock Minimum • ECG • Pulse oximetry • Blood pressure • Urine output Additional modalities • Central venous pressure • Invasive blood pressure • Cardiac output • Base deficit and serum lactate
  • 20. Indication of blood transfusion • Acute blood loss, to replace circulating volume and maintain oxygen delivery; • Perioperative anaemia, to ensure adequate oxygen delivery during the perioperative phase; • Symptomatic chronic anaemia, without haemorrhage or impending surgery.
  • 21.
  • 23. Complication of blood transfusion • Single transfusion • Incompatibility hemolytic transfusion reaction • Febrile transfusion reaction • Allergic reaction • Infection (bac, hepatitis, HIV, malaria) • Air embolism • Thrombophlebitis • Transfusion related acute lung injury
  • 24. Massive transfusion • Coagulopathy • Hypocalcaemia • Hyperkalaemia • Hyperkalaemia • hypothermia
  • 25. Blood substitute • Biomimeic- hemoglobin based • Abiotic – perfluoro carbon based