This is a lot but since they all related to each other I ha.pdf

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This is a lot, but since they all related to each other I had to post as one question, please answer all. Individuals who are homozygous for allele #2 have sickle-shaped RBCs 24 hours a day. However, individuals who are heterozygous for the gene have sickle-shaped RBCs only when the RBCs become "overworked" or "stressed"; otherwise, heterozygotes have normal-shaped RBCs. Examples of things that may cause a heterozygote's RBCs to become sickle-shaped include: (1) strenuous exercise, (2) exposure to high altitudes, and (3) exposure to pathogens that infect RBCs. The shaded areas in the map below represent parts of the world where the Anopheles mosquito is most prevalent. This is the mosquito that transmits the malaria parasite (which specifically infects RBCs ). It turns out that this parasite has a very difficult time infecting an RBC if it's sickle-shaped. (b) Suppose that, thousands of years ago, everyone living in North America was homozygous for Allele #1. If a baby was born that carried a mutation (one that converted Allele #1 to Allele #2), do you believe that this baby would have an advantage or disadvantage as compared to others on North America? Explain your answer. Relate your answer to the information given above.(c) Now suppose that, thousands of years ago, everyone living in the Middle East and South Asia was homozygous for Allele #1. If a baby was born that carried a mutation (one that converted Allele #1 to Allele #2), do you believe that this baby would have an advantage or disadvantage as compared to others in the Middle East and South Asia? Explain your answer.(d) Based on your answers to parts b&c, you can hypothesize that, TODAY, the sickle-cell allele (a.k.a. Allele #2) is more commonly found in people who have... (BOLD the correct answer) North American ancestry or Middle Eastern/South Asian ancestry (e) Explain your answer to part d. (f) Consider individuals who live in Europe and have 100% European ancestry. Is it possible for any of these individuals to have the sickle cell allele? Why or why not?.

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5. The pedigree below wis from a tare kidney disease: a) What is the most likely mode of inheritance for this disease? Explain your reasons. b) If person \#1 and \#2 have offspring, what is the probability that the first child will have kidney disease? c) If person HI:and H2 have 2 offspring, what is the probability that neither will have kidney disease? 6. For each pedigree below, indicate the most likely mode of inheritance (i.e,, autosomal or sexlinked, dominant or recessive) and the probability of the individuals indicated by the question marks being affected. Assume that the traits are rare in the general population and that the pedigrees show complete penetrance for the traits. 7. For the pedigree below determine the most likely mode of inheritance (i.e,, autosomal or sex-linked, dominant or recessive) and the probability of the individuals indicated by the question marks being affected. Assume that the traits are rare in the general population and that the pedigrees show complete penetrance for the traits .

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Darwin\'s 4 postulates are basic rules for when a trait is likely to evolve by natural selection. Please apply Darwin\'s postulates to evolution of antibiotic resistance in a Bacillus bacteria. Why was the lack of understanding of the mechanism of heredity such a significant problem for Darwin? Why was the age of the universe a challenge to Evolutionary thinking in Darwin\'s time? Why is it not a challenge now? The vertebrate eye is sometimes given as an example of irreducible complexity. How is our emerging understanding of eyes in a variety of animals giving us insight into how simple eyes or partial eyes may have been selectively advantageous in early animals? Solution Answer: darwin\'s 4 postulates of evolution are: 1. Individual variation (phenotypes amongst individuals vary) 2. In every generation, the proportion of progeny that survives is less than the number of progeny produced. 3. Survival and reproduction of individuals not random. With the current prevailing conditions, individuals with some phenotypes produce more offspring, find more mates, survive better and are naturally selected. 4. Some phenotypic variants passed on to offspring (heritable genetic information is passed on). Thus, more fit phenotypes are better represented in the next generation. therefore, microbacterial resistance of Bacillus tuberculosis is a classic example of darwinian selection. This is not a natural process, but a man-made situation superimposed on nature; there is perhaps no better example of the Darwinian notions of selection and survival. 1. the bacterial populations are individually diverse. 2. the number of bacterial conies surviving is less than the total colonies produced. 3. survival is the result of many years of unremitting selection pressure from human applications of antibiotics, via underuse, overuse, and misuse. 4. the surviving genotypes/traits are transffered to the progeny by conjugation, transfusion and transformation. part 2) Lack of understanding of heredity mechanism was a huge problem for Derwin as he could not give conclusive evidence of natural selection. and may skeptics continued to question his theory like Hugo de Vries, William Bateson till around the 1920s. After which the mechanisms of genetics started coming into fore. part 3) Age of universe was a fierce challenge for the success of theory of evolution a) the age of earth then accepted was around 6000 years as estimated by readings of bibl by Bishop Ussher. b) Darwin proposed the age of earth to be several hundred million years so that were sufficient for subtle canges in variation to happen for successful evolution c) William Thompson argues that based on thermodynamics and the source od radiation (which was mainly chemical and gravitational) the age of earth could never be more than 10s of million years which was too less for the theory of evolution to sustain. Hence age of universe was a major sore point for Darwin and his Theory of evolution. Part 4) now it is not a .

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Please give an explaination fir those quesions! 2 3 4 5 1. Aman with red.geen colour blindness is marned to a girl with normal colwir vision. What is the probaliliny that his childron will be coluur blind? 2. A boy has Dachenne muscular dyatrophy. His malemal uncle had died of museular dystrophy at the age of 21 years. What is the prohatility that this chilfs sibs will be affected? Assignments \#3 rocosive or S -kinked dominant and X -linked recesive inheritance. (Nole that a potigrte may be coempable with muve thas ane type of inheritance.) Assignnents \#4 Examine the pedieree frum a funily with a ernetic disease and anvv the questiens below: a. Does this pedigree isdicate autosonal deuninant, teoessive of set-link type of inheritance? Give reasons for your choice. b. Assuaning that B and b are the normal and mataut alleies respectively what would be the enenotypes of the individuals :II.1, 11 2 and III .3? c. Individual II. 3 requested genctic counselling. What is the probubility that her child woald be affected. Fxplain why. Assignments \#5 Susan's grandfather was deaf, and passed down a hercditary form of deafhess within Susan's family as shown in Figure. A. Is this mutation most likely to be dominant of recessive?? B. Is it carried on an autosome of a sex cluomosome? Why? C. A complete SNP analysis has been done for all of the 11 grandchildren (4 affected, and 7 unaffected) In comparing these 11 SNP result, how long a haplotype block would you expect to find around the critical \#ene? How might you deleet it? Assignments \#1 Distinguish between the following genetic terms: A. Gene and allele. B. Homozygous and heterozygous. C. Genotype and phenotype. D. Dominant and recessive. 1. A man with red-green colour blindness is married to a girl with normal colour vision. What is the probability that his children will be colour blind? 2. A boy has Duchenne muscular dystrophy. His maternal uncle had died of muscular dystrophy at the age of 21 years. What is the probability that this child's sibs will be affected? Explain, giving reasons, whether the following pedigrees are compatible with autosomal dominant, autosomal recessive or X -linked dominant and X -linked recessive inheritance. (Note that a pedigree may be compatible with more than one type of inheritance.) a. b. c. a. b. c. Assignments \#4 Examine the pedigree from a family with a genetic disease and ansv the questions below: a. Does this pedigree indicate autosomal dominant, recessive or sex-link type of inheritance? Give reasons for your choice. b. Assuming that B and b are the normal and mutant alleles respectively what would be the genotypes of the individuals: II.I, II.2 and III.3 ? c. Individual II. 3 requested genetic counselling. What is the probability that her child would be affected. Explain why. Susan's grandfather was deaf, and passed down a hereditary form of deafness within Susan's family as shown in Figure. A. Is this mutation most likely to be dominant or recessive? B. I.

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A researcher wanted to determine if a sample of DNA belonged to an animal, or plant, or bacteria, or fungi, or unknown organism. What gene would provide evidence that the DNa came from an animal cell? gene for synthesis of energy molecule ATP (B) gene for encoding the starch cellulose gene for production of the protein collagen (D) gene for production of peptidoglycan for cell wall Question 2 Starfish (an echinoderm) and jellyfish both have radial symmetry, but humans are more closely related to the starfish than jellyfish are. What evidence supports this? Humans have radial symmetry during most of their embryonic development Starfish undergo similar embryonic stages as humans during developmet (blastula, gastrula), and show bilateral symmetry in the larval stage, but later develop radial symmetry as adults. Jellyfish are always radially symmetrical. Jellyfish have bilateral symmetry, and also have triploblastic larvae Both starfish and humans have diploblastic development during embryonic stages..

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2 3 4 5 Assignments \#1 Distinguish between the following genetic terms: A. Gene and allele. B. Homozygous and heterozygous. C. Genotype and phenotype. D. Dominant and recessive. 1. A man with red-green colour blindness is married to a girl with normal colour vision. What is the probability that his children will be colour blind? 2. A boy has Duchenne muscular dystrophy. His maternal uncle had died of muscular dystrophy at the age of 21 years. What is the probability that this child's sibs will be affected? Explain, giving reasons, whether the following pedigrees are compatible with autosomal dominant, autosomal recessive or X-linked dominant and X -linked recessive inheritance. (Note that a pedigree may be compatible with more than one type of inheritance.) a. b. c. a. b. c. Assignments \#4 Examine the pedigree from a family with a genetic disease and ansv the questions below: a. Does this pedigree indicate autosomal dominant, recessive or sex-link type of inheritance? Give reasons for your choice. b. Assuming that B and b are the normal and mutant alleles respectively what would be the genotypes of the individuals: II.I, II.2 and III.3? c. Individual II.3 requested genetic counselling. What is the probability that her child would be affected. Explain why. Susan's grandfather was deaf, and passed down a hereditary form of deafness within Susan's family as shown in Figure. A. Is this mutation most likely to be dominant or recessive? B. Is it carried on an autosome or a sex chromosome? Why? C. A complete SNP analysis has been done for all of the 11 grandchildren (4 affected, and 7 unaffected). In comparing these 11 SNP results, how long a haplotype block would you expect to find around the critical gene? How might you detect it? .

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In the pedigree below, persons with a hatched marking are afflicted by the same rare, autosomal recessive disease. The couple III-2 og III-3 are expecting a child and wishes to know the risk for their child to develop the dissease. a) What is the probability that IV-1 is affected by the disease based on the pedigree? A polymorph marker locus with two co dominant alleles A1 and A2 is found so close to the disease locus that no crossing over can be assumed to occur. The marker does not cause the disease but can be used as a tracker to follow the disease allele in different families. Genotypes for the marker locus are given for a number of persons in the pedigree including the IV-1 fetus. b) What are the genotypes of individuals I-1 and I-2 for the two loci? In your answer, include how the alleles of the disease locus (e.g. B and b) and the marker locus (A1 and A2) are positioned on the chromosomes e.g. -> (e.g. A1b/A2B, A1B/A2b, A2b/A2b, etc.). c) What is the probability that IV-1 will get the disease when the A-locus is taken into account? d) What is the probability that IV-1 will be a heterozygote (carrier) given the information above? e) Would the probability/answer to question c) change if the A-locus was linked less tightly to the disease locus, so that there was a few centiMorgan between them? How would it change and why? IVI II III ? IV A2A2.

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Title-Investigating the effect of counterfeit permits on the Cross-Border Road Transport Agency, Johannesburg-Harare Route Background to the Problem Cross-border travel between South Africa and its neighbours has increased significantly over the years due to economic development in the South African Development Community (SADC) region. As a result, the demand for public passenger transport has continued to increase leading to the significant growth of the public passenger transport business. Due to the increase in cross- border passenger transport traffic, the South African government regulates the market in order to ensure that there is orderliness in the industry. The regulation of both passenger and freight road transport is conducted by agencies on behalf department of transport. This is done through regulatory frameworks put in place to ensure that cross-border passenger transport complies with safety and environmental standards. Thus, passenger transport operators must obtain permits in order to operate in a specific region. However, there are several operators that have not been complying with this requirement. Instead, they have been using counterfeit permits, making it difficult to determine market saturation or otherwise. 1. Discuss how data will be analysed. 2.The study is qualitative - refer to the specific type of analysis that will be used (for example, content analysis, thematic analysis, narrative analysis or grounded theory). Also describe how the data will be transcribed..

TitleInvestigating the effect of counterfeit permits on the.pdf

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Tketici talebindeki kk deiiklikler aadaki nedenlerle ve.pdf

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Titanic Project Management Blunders Links to an external s.pdf

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This is my table met Mry anlea Nole isch studein las diru ev.pdf

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