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Foot drop
Foot drop is the inability to lift the front part of the foot. It can be caused by injuries or conditions that damage the common peroneal nerve. Symptoms include difficulty lifting the foot and dragging the toes. Treatment depends on the underlying cause but may include bracing, nerve stimulation, tendon transfers, or joint fusions. The goal is to improve mobility and gait.
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Foot drop
- 1. FOOT DROP Dr.A.Supraja PG IIYEAR Gandhi Medical College
- 2. Inability to activelydorsiflex and evert the foot Definition
- 4. I. Traumatic: • Tendoninjuries to dorsiflexors of foot • Neurogenic A)At or below the level of common peroneal nerve Direct injuries: incised and penetrating injuries Fracture and dislocations: Fracture of lateral condyle of tibia Fracture/ dislocation of head/neck of fibula Dislocation of knee compound fracture of upper 1/3rd of tibia 4 Causes of Foot Drop
- 5. Iatrogenic : Hightibial skeletal traction Tight plaster around knee High tibial osteotomy Total knee replacement B) Above the level of common peroneal nerve Fracture of shaft of femur Posterior dislocation of hip Deep intra muscular injection PIVD Spina bifida If any cerebral tumors and space occupying lesions of CNS 5
- 6. II. infective • leprosy •Poliomyelitis Guillain-Barré Syndrome • Syphilis III. Metabolic: • Diabetes mellitus • Beri beri • Alcoholic neuritis IV. Exogenous toxin: • Lead • Arsenic • Mercury 6
- 8. COURSE OF THECOMMON PERONEAL NERVE
- 9. COURSE OF THECOMMON PERONEAL NERVE
- 10. • Fasiculi ofthe peroneal nerve - larger and have less connective tissue • Fewer autonomic fibers, so in any injury, motor and sensory fibers bear the brunt of the trauma. • More superficial course, especially at the fibular neck • Adheres closely to the periosteum of the proximal fibula Vulnerability of Peroneal Nerve
- 11. • Cutaneous sensationis impaired over the lateral aspect of the lower leg and ankle and dorsum of the foot. • Reduced dorsiflexion and eversion of the foot and of toe extension – The patients will compensate by having a steppage gait. • N.B Inversion and plantar flexion are normal. Signs
- 12. • Progressive weaknessof the peronei and tibialis anterior muscles which result in foot drop. peroneus longus , tibialis anterior and the extensor digitorum wasting • N.B The paresis results in ankle weakness and predispose to ankle sprains
- 13. • Difficulty inlifting the foot. • Dragging the foot on the floor as one walks. • Slapping the foot down with each step. • Raising thigh while walking(stepping gait) • Pain , weakness or numbness in the foot. SYMPTOMS
- 14. Types of footdrop • Type I – High above the level of fibular head deep peroneal nerve • Type II- Low below the level of fibular head superficial peroneal nerve
- 16. • High lesion: total foot drop • Unable to dorsiflex and invert foot • Able to do eversion • Wasting of ant group of muscles • Loss of sensation over the 1st web space 16 Clinical features of Type 1 foot drop
- 17. • Low lesion: incomplete foot drop • Unable to do eversion • Able to do dorsiflexion and inversion of the foot • Wasting of outer half of leg • Sensation lost over outer leg and foot 17 Clinical features of type 2 foot drop
- 18. • Gait offoot drop gait is high stepping gait • The patients lift the knee high and slaps the foot to the ground on advancing to the involved side 18 Gait of Foot Drop
- 21. X-Ray Post-Traumatic - tibia/injuryfibulaand ankle - any bony. Anatomic dysfunction (eg. Charcot joint) • Ultrasonography If bleeding is suspected in a patient with a hip or knee prosthesis • Magnetic Resonance Neurography Tumor or a compressive mass lesion to the peroneal nerve DIAGNOSIS
- 22. – This studycan confirm the type of neuropathy, establish the site of the lesion, estimate extent of injury, and provide a prognosis. – Sequential studies are useful to monitor recovery of acute lesions. Electromyelogram
- 23. • Depends onthe underlying cause. • If cause is successfully treated foot drop may improve or even disappear. • Medical treatment - painful paresthesia amitriptyline nortriptyline pregabalin TREATMENT
- 24. • Assistive andadaptive devices and equipment. – Canes, crutches, or walkers may be used to help prevent falling, normalize gait patterns, or unload a painful weight-bearing limb. • Electrical Stimulation. – Transcutaneous electrical nerve stimulation (TENS) for the reduction or obliteration of pain.
- 25. • Positioning. – Correctpositioning of limb • Protective Devices and Equipment eg splints, orthoses • Cryotherapy, massage
- 26. • Conservative treatment: shows high incidence of recovery • Splintage – splint knee in 20° of flexion and ankle in 90° for night time • In day time, walking is allowed by using ‘foot- drop appliance’ • Varieties of foot drop appliances: i) dynamic-spring shoe ii) static- back stop shoe 26 Treatment
- 27. • Ankle footorthotics (AFO) -support the foot with light-weight leg braces and shoe inserts • Exercises -strengthen the muscle, help to maintain range of motion (ROM) and improve gait • Electrical Functional Stimulations -electrically stimulate the peroneal nerve 27
- 29. Stimulating the nerve(peroneal nerve) improves foot drop especially if it caused by a stroke. Nerve Stimulation
- 30.
- 31. • Surgery –done if conservative management fails • Repairs or decompression of a damaged nerve, fusion of the foot and ankle joint or transfers tendons from stronger leg muscles 31
- 33. SURGICAL MANAGEMENT Points tobe considered age i. Mobility of joints ii. Availability of muscles and tendons for transfer A. Soft tissue and muscle contractures B. Bony changes
- 34. TENDON TRANSFER SURGERIES •Objectives: • Provide active motor power – paralysed muscle • Eliminate deforming effect of muscle- antagonist paralysed • Improve stability- muscle balance
- 35. PRINCIPLES • Muscle • transferred-strong—good/better •Nerve and blood supply-not impaired • Free end of tendon- • close to insertion of paralysed muscle • Retained in its own sheath/ another tendon • Routed direct in line bet muscle origin and new insertion • Contractures near the joint on which muscle acts- released • Agonists> antagonists • Tendon =range of excursion=one reinforcing /replacing
- 36. BARR’S TECHNIQUE (anterior transferof tibialis posterior) • Classic- 2/3 cuniform; 2/3 metatarsal • modified - cuboid • ( tendon passed through ant interossois membrane) • cast: • long leg calcaneovalgus-foot • >3weeks- B/K- • foot-N ;ankle- DF • >6WEEKS- remove cast- • Rehabilitation • 6 months- Double bar • foot drop brace with an • outside T STRAP
- 37. OBER’S TECHNIQUE • (tendon passed through ant comparment of leg) • Classic- 2nd metatarsal • HATTS modified obers- medial cuneiform • Post op = barrs
- 39. KAUFER’S PROCEDURE ( splittransfer of tibialis post tendon) • Incision- • 1st- curvilinear- navicular tuberosity • Extend-inf and post to medial malleolus • Proximal- post midline over tendocalcaneus • Tibialis Post –split longitudinally-plantar and dorsal • Tendocalneus lengthening • 2nd- tip of lateral malleolus-base of 5th metatarsal • Peronius brevis- pass tendon carrier –proximally –just post to lat malleolus- t.post tendon- foot in corrected position- • t.post sutured to brevis under tension. • Post op: • long leg cast- 2months • short leg cast – 2 months
- 41. SRINIVASAN TECHNIQUE ( twotailed transfer of tibialis post) • Position- supine – • passive dorsiflexion of ankle- knee extension • 1st-Short transverse incision- navicular-tibialis post endon exposed • Flexar retinaculum split • 2nd-Medial aspect of L/3rd of leg-incision-FDL retracted- tibialis post hooked up-split • 3rd& 4th -2 curvilinear – dorsum of foot- • medial- tendon of EHL-LOWER SLIP(anderson tunneler) • Lateral- tendon of EDL- UPPER SLIP- laced up
- 43. • Post op: •B/K- foot in 70 deg dorsiflexion • >3weeks- non-weight bearing reeducative exercises • Bivalve POP cast- crutches • 7th post op week- weight bearing
- 44. TENDO ACHILLIS LENGTHENING WHITESTECHNIQUE: (open) Posteromedial incision- expose tendocalcaneus Long cast- knee extension ;ankle-dorsiflexion 1ST post op- weight bearing Knee extension – 3weeks Short leg- 3 weeks AFO- ankle in neutral dorsiflexion
- 46. • HAUSER TECHNIQUE: •Posteromedial incision- expose tendocalcaneus • Plantaris tendon- incised-beneficial • Cast- mid thigh to toe knee-full flexion; ankle-neutral dorsiflexion skin blanching- little equinus (1st cast change-N) >6WEEKS- AFO
- 47. PERCUTANEOUS LENGTHENING OF TENDOCALCANEUS •Position: prone knee-E; ankle- DF 3 partial tenotomies Heel-varus-2 incisions medially valgus- 2 incisions laterally After suregery Mx = White technique
- 49. Semiopen sliding tenotomyof tendocalcaneus • Position-prone • 2 long incision 2cm along the tendocalcaneus • Plantaris tendon- tenotomy • Post op-=Hauser technique
- 51.
- 52. LAMBRINUDI ARTHRODESIS • Indiactions:Isolatedfixed equinus deformity > 10 years • CI: flail foot hip& knee instability requiring brace Lateral x-ray- ant subluxation of talus- 2 stage plantar arthodesis Complications-1. ankle instability 2.residual varus/valgus due to muscle imbalance 3.Pseudoarthrosis of talonavicular joint
- 54. • S/R –10-14 days • Short leg cast- x –ray satisfactory • Weight bearing- > 6weeks • Short leg walking cast- fusion complete(3mon)
- 55. TRIPLE ARTHODESIS • Mosteffective ; age > 12 years • Subtalar-calcaneocuboid-talonavicular joints • Indications: weakness and deformity of subtalar & mid tarsal joints stable& static realignment remove deforming forces arrest progression of deformity Eliminate pain eliminate use of short leg brace/ provide sufficient correction for fitting long leg brace obtain near normal correction of foot After Rx: walk with crutches / walker
- 59. CAMPBELLS post boneblock • Permits lengthening of tendocalcaneus & ankylosing both ankle and subtalar joints • Incision: medial and parallel to TA • FHL retracted to capsule of ankle joint is exposed • Post part of talus and articular surfaces of ankle and subtalar joints- excised • Ilium- large bony bridge across the ankle and subtalar joints • post op: ankle- plaster cast- A/K- foot at right angle >4 weeks- boot cast ( snugly fitting) full weight bearing delayed > 8-12 weeks cast immobilisation- until fusion walking on irregular surfaces difficult
- 61. ANKLE ARTHODESIS • BARR& BONE • Severe paralytic equinus deformity in adults • Subcutaneous plantar fasciotomy- lenthening of TA – ankle arthodesis