This document summarizes soil nutrients in a tabular form, including their roles, deficiency and toxicity symptoms, concentration in plants, discoverers, and year of discovery. It discusses the following nutrients: nitrogen, phosphorus, potassium, calcium, magnesium, sulfur, copper, iron, zinc, manganese, chlorine, molybdenum, boron, and nickel. It also provides keys to identifying deficiency symptoms, including whether older or younger leaves are affected first and whether symptoms are generalized or localized. Mobile nutrients affect older leaves while immobile nutrients affect young, growing tissues. The document references several sources and concludes with brief discussions of nutrient-related plant diseases.
Asian American Pacific Islander Month DDSD 2024.pptx
Soil nutreints
1. RANI LAKSHMI BAI CENTRAL AGRICULTURAL UNIVERSITY, JHANSI,
UTTAR PRADESH
ASSIGNMENT TOPIC:
SUMMARISATION OF SOIL
NUTRIENTSINTOTABULARFORM
SUBMITTED TO:
Dr. SUSHEEL KUMAR SINGH
SUBMITTED BY:
SHALINI SHUKLA (Ag/063/17)
2. ROLE DEFICIENCY
SYMPTOMS
TOXICITY
SYMPTOMS
ABSORBE
D AS/IN
THE
FORM OF
CONCENTRATIO
N IN THE PLANT/
AVERAGE CONC.
IN PLANTS
DISCOVE
RER
YEAR
OF
DISCO
VERY
N Component of
amino acids,
proteins,
nucleic acids,
porphyrins,
flavins, purines
and
pyrimidines,
enzymes etc.
Improves
quality of leafy
vegetables and
fodders
Improves
protein quality
of food grains
Early flowering
Lower protein
content
Overall yellow
green leaves
instead of dark
green
Yellowing and
dropping of
lower leaves
Delayed maturity
Increases
succulency
Lodging
Abortion of
flowers
NO3
-
NH4
+
1-5% (10000-
50000ppm)
1.5%
Theodore
de
Saussure
1804
P Development
of sugars, ATP
and nucleic
acids.
Energy transfer
Nutrient
movement
within plants
Transfer of
genetic
characteristics
from one
generation to
next
Bluish green to
Purplish
discoloration
(due to
accumulation of
sugars which
favors synthesis
of anthocyanin)
Delayed
maturity
Leaf curling
Root rot
diseases
Burning of leaf
tips
Reduce Fe, Mn,
Zn uptake
H2PO4
-
HPO4
--
0.1-0.4% (1000-
4000ppm)
0.2%
C.
Sprengel
1839
K Enzyme
activation,
photosynthesis
Protein
formation
Sugar transport
Crop quality
Disease
resistance
Stomata
opening and
closing
Hidden hunger
Chlorosis
followed by
scorching and
browning of
tips
Chlorosis from
margin towards
base and
necrosis
Rolling of leaf
edge
Leaf burn
(scorching) at
margins
Excessive
tillering in small
crops
Reduced Stalk
strength, disease
resistance and
winter hardiness
sugar
accumulation in
tubers affected
seeds and fruits-
shriveled
Reduce uptake of
Mg and Ca
K+
1-5%
1.0%
C.
Sprengel
1839
3. Ca Cell wall
construction
Growth of
meristems
Functioning of
root tips
Maintain the
chromosome
structure
Less than
0.1%(1000ppm)
Distorted young
leaves and turn
dark green
Withering of
leaf tips
Poor
germination
Cupping,
crinkling of
leaves
Ca+2
0.2-1%
0.5%
C.
Sprengel
1839
Mg Chlorophyll
Enzyme
activation
Uptake and
transport of P
Movement of
sugars
Cofactor for
ATP
production
Interveinal
chlorosis
(Leaf margins-
yellow or
reddish-purple
Midrib green)
Older leaf turn
yellow at edge
leaving a green
arrowhead
shape.
Grass tetany in
animals (wheat
forage)
Stiff and brittle
leaf in tuber
crops
Mg+2
0.1-0.4%
0.2%
C.
Sprengel
1839
S Constituent of
amino acids
and proteins
Metabolic
activities of
vitamins like,
biotin, thiamine
and coenzyme
A
Oil quality (oil
seed crops)
Resemble with
N and Mo
deficiency
No
characteristic
spot or stripes.
Plant-spindly
and small
Stem- thin
Puckering in
older leaves
with inward
raised areas
between veins
Orange- reddish
tints on older
foliage
SO4
-2
0.1-0.4%
0.1%
Sachs and
Knop
1860
Cu Chlorophyll
synthesis
Respiration
Protein
synthesis
SILVERY
LEAF
White tips,
reclamation
disease,
necrosis, leaf
distortion, die-
back
Melanosis
(brown
discoloration)
Male flower’s
sterility
Prone to
diseases
especially ergot
Similar to Zn-
reduced
reproductive
efficiency in
cattle.
20-100ppm
Displace Fe
Cu+2
5-30 ppm
6ppm
A.L.
Sommer,
C.P.
Lipman
and G.
McKinne
y
1931
4. Fe Plant
respiration
Chlorophyll
synthesis
Nucleic acid
and metabolism
Enzyme
activation
Interveinal
chlorosis with
sharp distinction
between veins
and chlorotic
areas in young
leaves
Tiny brown spots
appear on lower
leaves of rice
(from tip to base)
Entire leaf turn
purplish brown
Fe+2
100-500ppm
100ppm
E Gris 1843
Zn Growth
hormone
production
Internode
elongation
Translocation
and transport
of P
Less than
15ppm
Stripping
Mottling
Severe- gray-
white leaves
(prematurely
fall and die)
Poor flowering
and seed set
Patchy
appearance
Reduce
reproductive
efficiency in
cattle.
Terminal leaves
– rosetted.
Occurs on very
saline soils,
Fe deficiency,
Leaves- dark
green, chlorosis
Reduction in root
growth and leaf
expansion
Zn+2
27-150ppm
20ppm
A.L.
Sommer,
C.P.
Lipman
1926
Mn Role in TCA
cycle
Constituent of
superoxide-
dismutase
Component of
water splitting
enzyme in PS-
II
Less than 25
ppm
Interveinal
chlorosis(dicots)
but no sharp
distinction
between veins
and chlorotic
areas
FRIZZLE
TOP (palm
fronds are
deformed and
stunted)
Dead spots and
patches
(monocots)
Blackish-brown
or red spots on
older leaves
Tillering is
limited
Stunting of
plants
Occurs in
lowland rice
Mn+2
20-300ppm
20ppm
J.S
McHargu
e
1922
Cl Osmoregulation
Co-factor in
Mn containing
water splitting
enzyme ofPS-
II
Improve
nutritional
quality in
vegetables
Wilting at leaf
margins
Curling of
leaflets
Highly
branched root
symptoms
Similar to Mn
deficiency
Cl-
100-500ppm
100ppm
T.C.
Broyer,
A.B.
Carlton,
C.M.
Johnson,
P.R. Stout
1954
Mo Enzyme
activity
Biological
nitrogen
fixation (BNF)
Protein
biosynthesis
Pollen viability
Anther
formation
Chlorotic
mottling
between veins
Scorched,
rolled, cupped
leaves
Leaves- thick
and brittle and
wither(leaving
only midrib)
Toxic to
livestock
MoO4
-2
0.1-2ppm
0.1ppm
D.I.
Arnon,
P.R. Stout
1939
5. B Cell wall
formation and
reproductive
tissue
Imparts drought
tolerance
Pollen
germination
Ion uptake
Transport of K+
Discoloration
and death of
terminal bud
Dark brown,
irregular
lesions (leaf
necrosis)
Whitish leaf
spot at leaf base
Low sugar
accumulation
Root rot
Shortened
internode
(bushy/rosette)
Chlorosis
followed by
necrosis (from
leaf tip and
margin towards
midrib)
Large, dark
brown elliptical
spots
H3BO3
H2BO3
-
HBO3
-2
BO3
-3
10-200ppm
20ppm
K.
Waringto
n
1923
Ni Seed
germination
N metabolism
in legumes
component in
urease
Decrease in
amino acid
content,
Accumulation
of nitrates.
Fe deficiency Ni+2
0.1-10ppm
0.1ppm
P.H.
Brown,
R.M.
Welch,
E.E. Cary
1987
BRIEF KEY TO NUTRIENT DEFICIENCY SYMPTOMS(FINCK,1992)
NUTRIENT DEFICIENCY SYMPTOMS
SYMPTOMS APPEARING FIRST ON OLDER LEAVES
N Chlorosis starting from leaf tips
P Reddish color on green leaves or stem
K Necrosis on leaf margins
Mg Chlorosis between veins
Mn Brownish, greyish, whitish spots (cereals)
SYMPTOMS APPEARING FIRST ON YOUNGER LEAVES
S Mottled yellow-green leaves with yellowish veins
Fe Mottled yellow-green leaves with green veins
Mn Brownish black spots (legumes, potatoes)
Cu Youngest leaf has white tip
B Youngest leaf is brownish or dead (sugar beet)
6. APPROXIMATE CONCENTRATION OF NUTRIENTS IN MATURE LEAF TISSUES OF VARIOUS CROP PLANTS
NUTRIENT DEFICIENT SUFFICIENT/NORMAL TOXIC
N 1-5
P 0.1-0.4
K 1-5
Ca 0.2-1.0
Mg 0.1-0.4
S 0.1-0.4
Fe <50 100-500 >500
Mn 15-25 20-300 300-500
Zn 10-20 27-150 100-400
Cu 2-5 5-30 200-100
B 5-30 10-20 50-200
Mo 0.03-0.15 0.1-2 >100
Cl <100 100-500 500-1000
Ni <0.1
DISEASES ASSOCIATED WITH NUTRIENT DEFICIENCY
NUTRIENT DIFICIENCY DISEASES
Ca Blossom End Rot in tomatoes
Bitter pit in apples
Mg Grass tetany
Fe Green mottling of citrus
Mn Grey speck of oats
Speckled yellow of sugar beet
Marsh spots of peas
White spot of wheat
Brown spot of barley
Pahala blight of sugar cane
Frenching of Tung trees
Mo Whiptail of cauliflower
Zn Khaira in rice
White bud of maize
Mottle leaf (little leaf) or Frenching of citrus
Little leaf of cotton
K Cotton rust
Little leaf spot of alfalfa
B Black tip of mango
Heart rot of sugar beet
Hard fruit of citrus
Die back of apple
Cu Dieback of citrus
Wither tip of apple
7. KEY POINTS FOR IDENTIFICATION OF DEFICIENCY SYMPTOMS
MOBILE NUTRIENTS
OLDER OR LOWER LEAVES AFFECTED NO
YES
EFFECTS MOSTLY GENERALISED; PLANTS DARK OR
LIGHT GREEN
NO
YES
EFFECTS MOSTLY LOCALISED; CHLOROSIS WITH OR
WITHOUT SPOTTING
PLANTSDARK GREEN, OFTEN DEVELOPING PURPLE
OR RED COLOR
YES
PHOSPHORUS
NO
PLANTS LIGHT GREEN WITH LEAVES LIGHT GREEN OR YELLOW;
NO NECROTIC SPOTTING
YES
CHLOROSIS WITH INTERVEINAL CHLOROSIS; LEAVES
SOMETIMES RED OR WITH DEAD SPOTS
YES
MAGNESIUM
NO
NO INTERVEINAL CHLOROSIS; CHLOROTIC
AREAS WITH BURNING OF LEAF MARGINS;
SPOTTING SOMETIMES ALONG LEAF MARGINS
YES
NO
POTASSIUM
NO INTERVEINAL CHLOROSIS, DISTINCT NECROTIC AND
CHLOROTIC LESIONS WITH ABRUPT BOUNDRY BETWEEN
DEAD AND LIVE TISSUE
YES
CHLORIDE
YES
NITROGEN
NO
PLANTS LIGHT GREEN; NECROTIC SPOTTING ON LEAVES;
PALE LEAVESSOMETIMES SCORCHED, CUPPED OR ROLLED
YES
MOLYBDENUM
8. IMMOBILE NUTRIENTS
NEWER OR YOUNGER LEAVES AFFECTED;
SYMPTOMS LOCALIZED
YES
GROWING POINT DIES
NO
YES
YOUNG LEAVES OF TERMINAL BUD
BECOME LIGHT GREEN AT BASES;
LEAVES BECOME TWISTED AND BRITTLE
AND FIEBACK AT GROWING POINT;
CHLORSIS OF YOUNG LEAVES
NO
YES
BORON
YOUNG LEAVES OF TERMINAL BUDS
TYPICALLY HOOKED AT FIRST, FINALLY
TURNING BROWN AND DYING BACK
YES
CALCIUM
REMAINS ALIVE
YES
NO
YOUNG LEAVES WITH INTERVEINAL CHLOROSISCHLOROSIS
WITHOUT
INTERVEINAL
CHLOROSIS
YES
YOUNG LEAVES LIGHT GREEN;
TYPICALLY, NO CHLOROTIC
SPOTTING OR STRIPING
SULPHUR
YES
NO
CHLOROSIS OF YOUNG LEAVES, TIPS APPEAR
WITHERED AND WILL EVENTUALLY DIE.
YES
COPPER
YES
SHARP DISTINCTION BETWEEN
VEINS AND CHLOROTIC AREAS
YES
IRON
NO
NO SHARP DISTINCTION
BETWEEN VEINS AND CHLOROTIC
AREAS; SPOTTY APPEARANCE
MANGANESE
YES
MIDDLE LEAVES WITH
INTERVEINAL CHLOROSIS;
STUNTED GROWTH
ZINC
YES
NO
9. REFERENCES
FUNDAMENTALS OF SOIL SCIENCE- INDIAN SOCIETY OF SOIL SCIENCE
PRINCIPLES OF AGRONOMY- REDDY AND REDDY
Landresouces.montana.edu (nutrient management module no.9 Montana State University)
https://missouribotanicalgarden.org
https://extension.arizona.edu
https://omexcanada.com