2. Postgraduate Orthopaedics
FRCS(Tr&Orth) Revision Course
Newcastle Upon Tyne 16-21 March 2015
•
Professor Deiary Kader
Consultant Orthopaedic & Trauma Surgeon
Knee Surgeon
Nuffield Hospital Newcastle
NGMV Charity
3. Classification of knee
Stabilizers
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Lateral Complex
IT Tract
LCL
Popliteus
Biceps Femoris
Central Complex
ACL
PCL
Med Menx
Lat Menx
Medial Complex
MCL
Postromedial
Capsule
Semi-Memb
Pes anserinus
4. PCL
Three components:
AL Bundle: Long and thick part, 2X the size of PMB
Tightens in flexion
PM Bundle: Tight in extension
Meniscofemoral ligaments: mechanically very strong
Anterior: Humphrey’s ligament
Posterior: Wrisberg’s ligament
10. PCL
✦ The strongest ligament in the knee
✦ It is “a central stabilizer”
✦ Originates from a broad crescent-shaped area MFC
✦ Inserts centrally posteriorly 1–1.5cm below articular
surface of the tibia
✦ Average length of 38 mm and diameter of 13 mm
✦ PCL and quadriceps are dynamic partners in
stabilizing the knee in the sagittal plane
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12. Mechanism of injury
3% of all knee injuries
Dashboard Injury at 90° is the most common
Falling on a flexed knee with foot in plantar flexion
Forced hyperextension (>30º) is associated with
multi-ligament injury
High association with fracture femur
13. Presentation
Acute isolated PCL injury is commonly missed
Present with very little pain in the knee without hemarthrosis
There may be only bruising at the popliteal fossa.
Chronic PCL injury on the other hand may present with pain in
the medial compartment or anterior knee pain.
14. In isolation, it often causes little long-term
instability. However, it may lead to medial or
PFJ pain (OA) at a later date.
More troublesome in soccer players due to
difficulty in deceleration.
Presentation 2
15. Diagnosis 2
Clinical
Posterior drawer test at 90° and 30°
Quadriceps active drawer test. Flexion 60°
Posterior sag sign (step-off)
Posterolateral rotatory instability (Dial test prone)
External rotation recurvatum test
16. Diagnosis 1
MRI & PCL
Clinical examination is more reliable than MRI scan
The PCL may be dysfunctional despite normal MRI
Kneeling stress x-ray
Measure the degree of translation
PostGrad Orth Deiary Kader
17. Grading of PCL instability
Normal tibia step-off is 10 mm at 90° flexion
Instability could be mild, moderate or severe
Grade I instability is when there is a 5-mm step-off
Grade II instability is when there is no step-off (flush)
Grade III instability is when there is –5 mm step-off
There is a high association between Grade III PCL
injury and posterolateral corner injury.
18. Treatment
Treat acute, isolated PCL injury conservatively
Extension brace with calf support
(Posterior Tibial Support, PTS Brace) until the
pain subsides (4-6 weeks) with quadriceps
rehabilitation
Start early passive motion only in prone position to
maintain anterior tibia translation.
19. Surgical reconstruction
Indications
Acute combined injuries
Acute bony avulsion
Symptomatic chronic PCL injuries that failed
rehabilitation.
There is no difference in clinical outcome between
single and double bundle PCL reconstruction.
21. Complications
Immediate
Neurovascular injury popliteal vessels
Infection
Technical error → tunnel placement, graft tensioning, insecure
fixation
Delayed
Loss of motion
Avascular necrosis (medial femoral condyle)
Recurrent or persistent laxity (common) when a combined injury is
not adequately addressed
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22. What are the structures in the
Posterolateral Complex of the
Knee?
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23. Posterolateral Complex
Components:
– Biceps, ITB, Popliteus, Popliteofibular
ligament, arcuate ligament, LCL
Function
– Resists External and Varus rotation
Mechanism of Injury
– Direct blow to anteromedial tibia
– Hyperextension/varus
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24. The Posterolateral Corner
(PLC)
Primary stabilisers of external tibial
rotation at all knee flexion angles
Secondary restraints to anterior and
posterior translation
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25. The Posterolateral Corner
Resist Ext Rotation of Tibia
The LCL is a cord like structure 5-7 cm in lengthS
Primary static restraint to varus opening of the knee
Secondary restraint to posterolateral rotation
The popliteus is a static and dynamic external rotation stabiliser.
The popletiofibular ligament acts as
a primary restraint to external rotation of
the tibia on the femur at 30º of flexion
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26. The Posterolateral Corner
(PLC)
Isolated PLC sectioning produce a maximal
Average increase of 13° of tibial ER at 30° of knee flexion
Average increase of 5.3° of tibial ER at 90°
Isolated PCL sectioning has no effect on external tibial rotation
Combined injury to the PCL and PLC leads to ER of 20.9° at
90° of knee flexion
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30. 30
Fib
Pop
Extension
The popliteus tendon inserted
10 mm distal
5 mm posterior to the lateral epicondyle
The LCL inserted
2 mm proximal
4 mm posterior to the lateral epicondyle
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34. Posterolateral Complex
Injury--Treatment
Partial
– Grade I & II Instability with a good end point
– Nonsurgical Treatment
– 1-3 week immobilisation in extension
Complete Acute
– Primary repair best
– Augment with allo/auto graft
Complete Chronic
– Reconstruct Popliteus and LCL
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35. PLC Reconstruction
The reconstruction can be:-
✴Fibula based such as modified Larson’s technique or
✴Combined tibia and fibula based such as LaPrade’s
anatomical reconstruction.
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37. The principles of surgery
Early repair/ Recon (within 3 weeks) of torn and detached ligaments,
tendons and capsule in acute injuries. A combination of early repair and
reconstruction has been shown to provide better results.
Late reconstruction of the two or three of the main stabilisers of the
posterolateral corner of the knee i.e. the lateral collateral ligament,
Popliteus tendon, and popliteofibular ligament in chronic cases.
Combined ACL/PCL and PLC injury must be treated by reconstruction of all injured
ligaments. Isolated ACL or PCL reconstruction without addressing the PLC will
ultimately fail.
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38. Knee dislocation
Any triple-ligament knee injury constitutes a frank
dislocation. This is relatively rare but a severe and
potentially limb-threatening injury.
High-energy injury such as RTA
Sporting accident
May be missed on initial assessment.
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39. Vascular Injuries
Previously it was thought there was a 50%
incidence of vascular compromise Now 3.3-18%
20%–30% incidence of nerve injury.
Fracture incidence may be as high as 60%.
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40.
41. Classification
Classified on the basis on tibial displacement in respect to the femur
Closed or open
High or low energy
Dislocation or subluxation
Neurovascular involvement
Anterior (common: 30-50% of dislocations, associated with intimal tears)
Posterior; also medial, lateral (highest rate of peroneal nerve injury) and
rotatory (usually irreducible) or combined
Hyperextension leads to anterior dislocation
Dashboard injury leads to posterior dislocation
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42. Examination
Valgus and varus laxity
Anteroposterior translation
Recurvatum
>10º hyperextension suggests ACL injury
>30º hyperextension indicates PCL injury
Rotation indicates MCL and LCL injury
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43. Management
Surgical emergency
Deal with life-threatening injuries first
Circulation in A&E
Serial examination for 48 hours.
Ankle brachial Index (ABI)
ABI <0.9 is suggestive of significant arterial injury
Involve the vascular surgeon
Radiography before manipulation
– (assess direction and associated fracture)
Reduction as soon as possible in the emergency/operating Room
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44. Management
Immobilization in an extension knee splint
Check radiograph to confirm congruity, if not, consider
external fixator
Conservative management out of favour
Early surgical reconstruction and/or repair, is currently
recommended by the Knee Dislocation Study Group
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45. Management
Surgery as soon as the vascular surgeon allows
Most ACL/PCL/MCL can be treated with bracing the MCL followed by
combined ACL/PCL reconstruction once range of movement is restarted,
usually after 6 weeks.
ACL/PCL/posterolateral corner can be treated by repairing the
posterolateral corner acutely (within three weeks) and delayed ACL/PCL
reconstruction 8 weeks later. Or all in One
Open dislocation, fracture dislocation and vascular compromise require
staged procedures.
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