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CAUSES OF LOW VISION
IN ADULTS
Presenter:-Harshali Jadhav
Moderator :-KUNAL KISHOR
Definition
Low vision:- According to WHO
A person with low vision is are who has
impairment of visual function even after
treatment and/or standard refractive correction
and has an a acuity of less than 6/18 to light
perception or visual field less than 10* from the
point of fixation, but who uses or its potentially
able to use vision for the planning execution of
the task.
Introduction
The number of with low vision is increasing with
ageing of the population
Visual loss in adult is associated with many factors,
activity, limitation and lower quality of life
CAUSES OF
LOW VISION IN
ADULT
AGE RELATED MACULAR DEGENERATION
It is an acquired retinal disorder
A degenerative change in retinal pigmented
epithelium
Subsequent degeneration of the over line rods and
cones
It results in progressive irreversible loss of central
vision
Due to fibrous scarring or geographic atrophy of the
macular area
Risk Factors …
Hyperopic
Increase in cup disc ratio
Elevated serum cholesterol & hypertension
Smoking and sunlight exposure
VISUAL ACUITY :-
V/A May vary with extent of degeneration with dry
state ARMD
V/A can range from 20/20 to 20/400
Wet state ARMD V/A shows worse than 20/400
VISUAL FIELD:-
Demonstrate a central or Para central Scotoma with
normal peripheral findings
Most patient response well to magnification at
distance and near
Non-optical systems filters, tint , and sun lenses for
improve contrast and avoided glare and photophobia
CATARACT
It’s the opacification of crystaline lens
The types includes anterior and posterior subcapsular
, cortical, equatorial and nuclear
ETIOLOGY:-
Traumatic
Metabolic
Toxic
Pharmacological
Age related
Inflammatory
Hereditory
VISUAL ACUITY:-
V/A varies greatly dividing on the degree and location
of opacification
Acuity range from normal to severe vision
impairment
VISUAL FIELD:-
Central and peripheral visual field testing generally
source a relative generalize depression without focal
defects
LOW VISION MANAGEMENT:-
Direct lighting is generally for near point
activities
Glare can be reduce by use of Filters tint and sun
lenses
Patients response to magnification at distance
and near which may postpond the need of cataract
surgery
Non-optical system such as large print reading
material, bold line writing paper , typo scope .
DIABETIC RETINOPATHY
It’s a highly specific vascular complication
Independent Diabetic mellitus and Non -
Independent Diabetic Meliates
It is one of the leading cause of blindness
VISUAL ACUITY:-
V/A ranges from 20/20 to total blindness
VISUAL FIELD:-
In profilative disease retinal schemic loser scar and
retinal detachment can cause corresponding field loss
LOW VISION MANAGEMENT:-
Refractive error should be checked during at least to
different visits
Use of contact lens to correct refractive error
Corning photochromatic filter and sunlens that block
blue wavelength may improve contrast and eliminate
glare and photophobia
Direct illumination for near task
A flash light is helpful for poor night vision patient
Eccentric viewing technique along with magnification
for distance and near
Non-optical device such as glucose monitor and
insulin syringe aid are helpful for the patient.
MULTIPLE SCLEROSIS
It is demilinating nerve fibers have on impaired
ability to conduct impulses at
physiological frequencies
VISUAL ACUITY:-
V/A ranges from normal to severely impaired
depending on the extend of ocular manifestation
VISUAL FIELD:-
Several pattern of visual field loss occurs of patients
with optic neuritis secondary to multiple sclerosis
Central and ceco-central scotomas may be present
LOW VISION MANAGEMENT:-
Magnification device for near and distance are
beneficial for patient with central vision loss
Spectacle mounted device
Corning photo chromatic filter, Tints and sunglasses
help to eliminate glare
Non-optical device such as direct lighting
MYOPIC DEGENERATION
It occurs due to accessive stretching of posterior
segment of the eye
It associated with increasing axial length
VISUAL ACUITY:-
V/A may be corrected by 20/20
High myopia can results in variety of visual field
defect
In more advanced state sub-retinal neo-vascular
membrane formation may occur causing further in
decrease in central acuity
LOW VISION MANAGEMENT:-
Optical correction of refractive error with
conventional spectacles or contact lenses usually
improve V/A
Direct illumination for near
Cont……..
When using spectacle lenses a small round frame,
high index lenses and anti reflection coating will help
to reduce peripheral distortion
For poor night vision flashlight is helpful
RETINAL DETACHMENT
It results from accumulation of fluid between the
sensory retina and retinal pigment membrane
VISUAL ACUITY:-
V/A can be severely reduced , if there is involvement
of macular area
VISUAL FIELD :-
Field defect develops corresponding to the sight of
retina detachment
LOW VISION MANAGEMENT:-
Using sunglasses and tint can be eliminate glare and
photophobia
Direct illumination for near point task
Due to affected central vision a new correction may
be indicated
Cont………
If bilateral temporal detachment occurs presence may
be considered for improve mobility along with
orientation and mobility services
GLAUCOMA
It is an optic neuropathy cause by reduce blood flow
to the optic nerve resulting in peripheral visual field
loss progressing to central vision loss with severe
ETIOLOGY
Family history
Advanced age
Systemic vascular disease
Blunt ocular trauma
Pigment dispersion
Psuedo-exploxation
Rubio sis iriditis
Use of steroid drugs
Glaucoma in fellow eye
VISUAL ACUITY
Central v/a is generally un affected until the end stage
of disease
VISUAL FIELD
Early glaucomatous visual field defects includes Para
central Scotoma , nasal field loss from this area occur
as disease worse
LOW VISION MANAGEMENT:-
 Electronic Magnification system such as closed circuit
television ( C.C.TV) increased contrast and brightness
 Corning photo chromatic filter may reducing glare
and improving contrast
Minus lenses and reversible telescope helps the
Patient with intact central acuity and peripheral visual
field loss
To enhance visual field
Prisms or mirror system may stimulate peripheral
awareness
Create Orientation and mobility in end stage visual
field loss of galucoma patient
 long cane travel can beneficial in end stage disease
Use of flash light in night travel
THANK YOU…

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Causes of low vision in adult

  • 1. CAUSES OF LOW VISION IN ADULTS Presenter:-Harshali Jadhav Moderator :-KUNAL KISHOR
  • 2. Definition Low vision:- According to WHO A person with low vision is are who has impairment of visual function even after treatment and/or standard refractive correction and has an a acuity of less than 6/18 to light perception or visual field less than 10* from the point of fixation, but who uses or its potentially able to use vision for the planning execution of the task.
  • 3. Introduction The number of with low vision is increasing with ageing of the population Visual loss in adult is associated with many factors, activity, limitation and lower quality of life
  • 5. AGE RELATED MACULAR DEGENERATION It is an acquired retinal disorder A degenerative change in retinal pigmented epithelium
  • 6. Subsequent degeneration of the over line rods and cones It results in progressive irreversible loss of central vision Due to fibrous scarring or geographic atrophy of the macular area
  • 7. Risk Factors … Hyperopic Increase in cup disc ratio Elevated serum cholesterol & hypertension Smoking and sunlight exposure
  • 8. VISUAL ACUITY :- V/A May vary with extent of degeneration with dry state ARMD V/A can range from 20/20 to 20/400 Wet state ARMD V/A shows worse than 20/400 VISUAL FIELD:- Demonstrate a central or Para central Scotoma with normal peripheral findings
  • 9. Most patient response well to magnification at distance and near Non-optical systems filters, tint , and sun lenses for improve contrast and avoided glare and photophobia
  • 10. CATARACT It’s the opacification of crystaline lens The types includes anterior and posterior subcapsular , cortical, equatorial and nuclear
  • 12. VISUAL ACUITY:- V/A varies greatly dividing on the degree and location of opacification Acuity range from normal to severe vision impairment VISUAL FIELD:- Central and peripheral visual field testing generally source a relative generalize depression without focal defects
  • 13. LOW VISION MANAGEMENT:- Direct lighting is generally for near point activities Glare can be reduce by use of Filters tint and sun lenses Patients response to magnification at distance and near which may postpond the need of cataract surgery
  • 14. Non-optical system such as large print reading material, bold line writing paper , typo scope .
  • 15. DIABETIC RETINOPATHY It’s a highly specific vascular complication Independent Diabetic mellitus and Non - Independent Diabetic Meliates It is one of the leading cause of blindness
  • 16. VISUAL ACUITY:- V/A ranges from 20/20 to total blindness VISUAL FIELD:- In profilative disease retinal schemic loser scar and retinal detachment can cause corresponding field loss
  • 17. LOW VISION MANAGEMENT:- Refractive error should be checked during at least to different visits Use of contact lens to correct refractive error Corning photochromatic filter and sunlens that block blue wavelength may improve contrast and eliminate glare and photophobia
  • 18. Direct illumination for near task A flash light is helpful for poor night vision patient
  • 19. Eccentric viewing technique along with magnification for distance and near Non-optical device such as glucose monitor and insulin syringe aid are helpful for the patient.
  • 20. MULTIPLE SCLEROSIS It is demilinating nerve fibers have on impaired ability to conduct impulses at physiological frequencies
  • 21. VISUAL ACUITY:- V/A ranges from normal to severely impaired depending on the extend of ocular manifestation VISUAL FIELD:- Several pattern of visual field loss occurs of patients with optic neuritis secondary to multiple sclerosis Central and ceco-central scotomas may be present
  • 22. LOW VISION MANAGEMENT:- Magnification device for near and distance are beneficial for patient with central vision loss Spectacle mounted device Corning photo chromatic filter, Tints and sunglasses help to eliminate glare Non-optical device such as direct lighting
  • 23. MYOPIC DEGENERATION It occurs due to accessive stretching of posterior segment of the eye It associated with increasing axial length
  • 24. VISUAL ACUITY:- V/A may be corrected by 20/20 High myopia can results in variety of visual field defect In more advanced state sub-retinal neo-vascular membrane formation may occur causing further in decrease in central acuity
  • 25. LOW VISION MANAGEMENT:- Optical correction of refractive error with conventional spectacles or contact lenses usually improve V/A Direct illumination for near
  • 26. Cont…….. When using spectacle lenses a small round frame, high index lenses and anti reflection coating will help to reduce peripheral distortion For poor night vision flashlight is helpful
  • 27. RETINAL DETACHMENT It results from accumulation of fluid between the sensory retina and retinal pigment membrane
  • 28. VISUAL ACUITY:- V/A can be severely reduced , if there is involvement of macular area VISUAL FIELD :- Field defect develops corresponding to the sight of retina detachment
  • 29. LOW VISION MANAGEMENT:- Using sunglasses and tint can be eliminate glare and photophobia Direct illumination for near point task Due to affected central vision a new correction may be indicated
  • 30. Cont……… If bilateral temporal detachment occurs presence may be considered for improve mobility along with orientation and mobility services
  • 31. GLAUCOMA It is an optic neuropathy cause by reduce blood flow to the optic nerve resulting in peripheral visual field loss progressing to central vision loss with severe
  • 32. ETIOLOGY Family history Advanced age Systemic vascular disease Blunt ocular trauma Pigment dispersion Psuedo-exploxation Rubio sis iriditis Use of steroid drugs Glaucoma in fellow eye
  • 33. VISUAL ACUITY Central v/a is generally un affected until the end stage of disease VISUAL FIELD Early glaucomatous visual field defects includes Para central Scotoma , nasal field loss from this area occur as disease worse
  • 34. LOW VISION MANAGEMENT:-  Electronic Magnification system such as closed circuit television ( C.C.TV) increased contrast and brightness  Corning photo chromatic filter may reducing glare and improving contrast
  • 35. Minus lenses and reversible telescope helps the Patient with intact central acuity and peripheral visual field loss
  • 36. To enhance visual field Prisms or mirror system may stimulate peripheral awareness Create Orientation and mobility in end stage visual field loss of galucoma patient  long cane travel can beneficial in end stage disease Use of flash light in night travel