ES UNA EXPOCISION DE DIABETES,PTOG ,SM,COMPLICACIONES-ORGANIZADA POR MI.EN INGLES,CON LOS MAS MINUCIOSOS DETALLES,

2. Kg 1.75cmNot to exceed 75 mg total

4. CONDITIONS TO BE MET

5. GLYCOSYLATED HEMOGLOBIN CRYTICAL RISK INCREASED RISK HIIGH RISK MILK RISK LOW RISK

6. GLYCOSYLATED HEMOGLOBIN

7. WHAT IS THE BEST SCREENING TEST FOR DM?

8. QUESTIONAIRE

10. WHO IS SHE SHOULD HAVE A SCREENING TEST FOR DM? Family diabetic first degree relativesLatest from rural and urbanDMG obstetric historyWith hypertension and other risk factorImpaired glucose priorDiagnosis of metabolic syndrome

12. 50%

13. 62%

14. PPPD

15. Glucemia Fasting

18. Symptoms:The problems found in metabolic syndrome include:

19. regulation of glucocorticoid and receptor levels—impact on the metabolic syndrome

21. Dawn Phenomenon: Morning Hyperglycemia secondary toelevated bloodglucoselevels in thelasthours of themorning (whenthesungoesdown) Somogy Phenomenon: Morning Hyperglycemia secondarytohypoglycemia in themorning (3-5 am)

22. Dawn Phenomenon: Morning Hyperglycemia secondary toelevated bloodglucoselevels in thelasthours of themorning (whenthesungoesdown)Somogy Phenomenon: Morning Hyperglycemia secondarytohypoglycemia in themorning (3-5 am)

23. Hypoglycemia is defined as blood sugar less than… REMISSION OR HONEYMOON Acute onsetRemissionintensificationTotal diabetes

24. SEVERE ACUTE COMPLICATIONS OF T2DM

27. Sudden reduction of steroid dose

28. Altered ability of patient to report symptoms

30. adrenergic symptoms sweat tremor pallor neuroglupenicos symptoms blurredordouble vision alteration of behavior headache

32. ETIOLOGY Causes of hypoglycemia can be classified into three groups: 1. Medications or toxins. 2. Disorders associated with fasting hypoglycemia. 3.Disorders associated postprandial hypoglycemia.

33. TREATMENT OF HYPOGLYCEMIA An initial bolus, 20-50 ml 50% dextrose, should be given immediately, followed by infusion of D5W to maintain blood sugar above 100 mg/dL.

36. SEVERE HYPERGLYCAEMIC FREQUENT IN DM 1 FREQUENT IN DM 2 ↓ 2% IN DM 2 POSSIBLE IN DM1 poorly treated

37. Diagnostic Criteria for DKA and HHS

38. CRITERIO ADA PARA CA

39. CLINICAL PITURE Role of 2 Hormones Lack of insulin stimulates hyperglycemia Lack of insulin stimulates fat & muscle breakdown Increase in fatty acids to liver stimulates ketosis Fluid loss stimulates hyperosmolarity Pathophysiology of DKA Role of Insulin

40. Hypoglycemia is defined as blood sugar less than… Pathophysiology Counter-Regulatory Hormones : .,

41. STANDARDS FOR ADA HDCA

42. Stressful precipitating event that results in increased catecholamines, cortisol, glucagon.

43. MANEJO S Y M P T O M S O F D K A / H H S Polydypsia Fatigue Polyuria Obtundation Abdominal Pain Confusion Blurred vision Nausea/Vomiting

48. ANTEPARTUM FETAL EVALUATION

49. ultrasoundantepartum fetal 1.. Ultrasound is a valuable tool in evaluating fetal growth, estimating fetal weight, and detecting hydramnios and malformations.

50. 2.. While the false-negative rate with maternal monitoring of fetal activity is low (~1 percent), the false-positive rate may be as high as 60 percent. Maternal hypoglycemia, while generally believed to be associated with decreased fetal movement, may actually stimulate fetal activity.

52. remains the preferred method to assess antepartum fetal well-being in the patient with diabetes mellitus

53. If the NST is nonreactive, a biophysical profile (BPP) or contraction stress test is then performed .