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EVALUATION OF CLINICAL 
CHARACTERISTICS AND METHODS 
OF ANESTHESIES RELATING TO 
SEVERE BRONCHOSPASM 
IN SURGERY ENT 
PRESENTATION: DR.Nguyen Phu Van 
ENT Hospital National
Introduction 
 Bronchospasm is caused by the spasmodic contraction of 
the bronchial smooth muscle. 
 Occur during periods of anesthesia 
 Incidence: 1,7‰ in patients without a history of asthma 
8 ‰ in patients with a history of asthma 
30% in patients with severe asthma 
 Incidence decreases with age 
Pedersen,ActaAnaesthesiol Scand2001
Introduction 
 Risk of bronchospasm increase10 times when a 
respiratory infection. 
- Patients with ENT infections : flu, allergic rhinitis… 
- Tracheal stenosis, dyspnea, accumulation of sputum, 
pneumonia … 
Olsson, ActaAnaesthesiol Scand 2007 
 Severe bronchospasm may be at risk of hypoxic brain, 
cause brain damage or brain death. 
 In cases of severe bronchospasm : 
70% death 
18% hypoxic brain 
Cheney, Anesthesiology 1991
For these reasons that we do this research aims : 
1.Evaluate the clinical characteristics related to severe 
bronchospasm in ENT surgery. 
2.Methods of anesthesia-related bronchospasm 
3.Evaluate the results of treatment of bronchospasm 
severe.
Obstructing air exhaled 
Gas trapping 
Excessive alveolar stretch 
alveolar increased pressure 
Literature review 
Pneumothorax alveolar rupture
Obstructing air exhaled 
gas trapping 
Excessive alveolar stretch 
Reduced lung compliance 
alveolar increased pressure 
Reduced 
circulation 
back to the heart 
Disorders of 
blood-gas distribution Increased pulmonary 
artery pressure 
literature review 
Pneumothorax alveolar rupture
Obstructing air exhaled 
gas trapping 
Excessive alveolar stretch 
Reduced lung compliance 
alveolar increased pressure 
Mobilization of auxiliary 
respiratory muscles 
Increased 
respiratory activity 
Increased production of CO2 
Reduced 
circulation 
back to the heart 
Disorders of 
blood-gas distribution 
Hypotension 
Hypoxemia 
Acute heart failure 
Increased pulmonary 
artery pressure 
Increased right ventricular load 
Increased oxygen consumption 
literature review 
Pneumothorax alveolar rupture
Sonde, tube 
Mechanism of bronchospasm 
General anesthesia 
Central nervous system 
Bronchial 
smooth 
muscle 
Local anesthesia 
The stimulation of surgery 
Stimulate, traction, 
The vagus nerve 
Bronchial 
parasympathetic 
ganglion 
rub Epithelium trachea 
The gases, 
exudates, blood 
or bronchi 
Increased bronchial 
response 
BRONCHOSPASM
DEEP ANAESTHESIA 
Sonde, tube 
Mechanism of bronchospasm 
General anesthesia 
Central nervous system 
Bronchial 
smooth 
muscle 
Local anesthesia 
The stimulation of surgery 
Stimulate, traction, 
The vagus nerve 
Bronchial 
parasympathetic 
ganglion 
rub Epithelium trachea 
The gases, 
exudates, blood 
or bronchi 
Increased bronchial 
response 
BRONCHOSPASM
Management of bronchospasm 
Stop all stimulation on patients 
Ventilate the patient with 100% oxygen 
Assess whether anaphylaxis is not? Yes Management of 
anaphylaxis 
Deep anesthesia Not 
Normotensive: 
Propofol IV 
Sevofluran 
Hypotension: 
Ketamine IV 
Optimal ventilation 
Control of the airway 
by intubation 
pressure control ventilation 
with prolonged exhalation 
time : I/E= 1/3, 1/4 
Manual ventilation 
with 100% oxygen 
Use bronchodilators 
Salbutamol spray 
Intravenous 
bronchodilator : 
Salbutamol, 
Diaphyline 
If shock or use of 
bronchodilators failure: 
Adrenalin IV 
Solu-medrol IV
Research Methodology 
- Study design : 
Retrospective, descriptive, cross-sectional. At the 
Department of Anesthesiology and Resuscitation 
Emergency Department, ENT Hospital National, within 15 
years, the period from January 1999 to January 2014. 
- Subjects of study : 
The patient had been diagnosed with severe bronchospasm
RESULTS 
Clinical characteristics and type of anesthesia 
Characteristics 
Age <18 years old: 1 18-60years old: 13 > 60years old : 3 
Sex Males: 12 Females: 5 
Period Intraoperative: 15 Intubation: 1 Extubation: 1 
Diseases need 
surgery 
Tracheal stenosis: 13 Sinusitis: 1 Neck abscess: 1 Open glottis 
paralysis: 2 
Type of 
anesthesia 
Local anesthesia: 15 General anesthesia:2 
Cause Stimulation of surgery: 14 Reflux: 2 Drug reactions: 1
Symptoms Signal Quantity Ratio % 
Subjective symptom 
Intense dyspnea 17 100% 
Tachypnea 3 
Bradypnea 15 88,2% 
Stiffened body 12 70,5% 
Objective 
symptom 
Auscultation 
Murmur decreased alveolar 17 100% 
Wheezing 13 76,4% 
Lung silence 4 
Perception 
Vague 12 41,2% 
Tumbledown 5 29,4% 
Heart rate 
Tachycardia 10 58,8% 
Bradycardia 7 41,2% 
Blood pressure 
Normotensive 2 
Hypotension 15 88,2% 
SpO2 
>90% 2 47% 
50-90% 10 
<50% 5 29,4% 
Pneumothorax 
Unilateral 3 
Bilateral 12 70,5%
Results of treatment 
Emergency Management 
General anesthesia, using bronchodilators, support 100% oxygen 
through the tube (17/17), tracheostomy set canuyl 14/17 patients, 3/17 
patients intubation.. 
After the emergency management 
Pleural drainage and continuous suction, 15/17 patients. 
Sedation, pressure control ventilation, antibiotics, anti-inflammatory 
Outcome 
•12/17 patients recover completely and do not leave sequelae 
•2/17 patients were treated early and properly position the principle has 
recovered immediately. 
•3/17 patients died.
DISCUSSION 
Clinical characteristics and type of anesthesia 
-13/17 (76.5%) tracheal stenosis scarring need to tracheostomy for 
airway control under local anesthesia. 
-According to Nathalie Schutz, the incidence rate of bronchospasm met 
80% of the patients with airway obstruction and surgery without 
anesthesia. 
-Study on 31 patients who had surgical tracheostomy under local 
anesthesia by the author Rohail Ahmed, Muhammad Faheem Malik, the 
2 patients with pneumothorax, 2 patients with cardiac arrest due to 
bronchospasm.
The stage and cause of bronchospasm 
The stages R N Westhorpe Our 
Induction 48 1 
Perioperative 66 15 
Postoperative 11 1 
Total 125 17 
Causes 
Stimulates airway 23 15 
Drug reactions 8 1 
Early extubation 6 1 
Other causes 5 0 
Total 42 17
Results of treatment 
-2/15 patients received early treatment were stable and 
without complications 
- 70% (12/17 patients) were pneumothorax were treated 
for pleural drainage continuous suction with negative 
pressure, pressure control ventilation, sedative, 
analgesic, antibiotic, anti-inflammatory and has stable 
after 5- 7 days. 
-3/17 patients died.
CONCLUSIONS 
- Complications severe bronchospasm in ENT surgery met 
the majority of patients with tracheal stenosis scarring 
require surgery tracheostomy under local anesthesia 
(accounting for 76.5%). 
- 88.2% (15/17) patients with severe bronchospasm were 
pneumothorax and 3/17 patients died due to delays and 
improper management principles. 
- 2/17 patients received timely and proper management 
principles, has recovered completely without complications.
Thanks for your attention!

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EVALUATION OF BRONCHOSPASM MANAGEMENT IN ENT SURGERY

  • 1. EVALUATION OF CLINICAL CHARACTERISTICS AND METHODS OF ANESTHESIES RELATING TO SEVERE BRONCHOSPASM IN SURGERY ENT PRESENTATION: DR.Nguyen Phu Van ENT Hospital National
  • 2. Introduction  Bronchospasm is caused by the spasmodic contraction of the bronchial smooth muscle.  Occur during periods of anesthesia  Incidence: 1,7‰ in patients without a history of asthma 8 ‰ in patients with a history of asthma 30% in patients with severe asthma  Incidence decreases with age Pedersen,ActaAnaesthesiol Scand2001
  • 3. Introduction  Risk of bronchospasm increase10 times when a respiratory infection. - Patients with ENT infections : flu, allergic rhinitis… - Tracheal stenosis, dyspnea, accumulation of sputum, pneumonia … Olsson, ActaAnaesthesiol Scand 2007  Severe bronchospasm may be at risk of hypoxic brain, cause brain damage or brain death.  In cases of severe bronchospasm : 70% death 18% hypoxic brain Cheney, Anesthesiology 1991
  • 4. For these reasons that we do this research aims : 1.Evaluate the clinical characteristics related to severe bronchospasm in ENT surgery. 2.Methods of anesthesia-related bronchospasm 3.Evaluate the results of treatment of bronchospasm severe.
  • 5. Obstructing air exhaled Gas trapping Excessive alveolar stretch alveolar increased pressure Literature review Pneumothorax alveolar rupture
  • 6. Obstructing air exhaled gas trapping Excessive alveolar stretch Reduced lung compliance alveolar increased pressure Reduced circulation back to the heart Disorders of blood-gas distribution Increased pulmonary artery pressure literature review Pneumothorax alveolar rupture
  • 7. Obstructing air exhaled gas trapping Excessive alveolar stretch Reduced lung compliance alveolar increased pressure Mobilization of auxiliary respiratory muscles Increased respiratory activity Increased production of CO2 Reduced circulation back to the heart Disorders of blood-gas distribution Hypotension Hypoxemia Acute heart failure Increased pulmonary artery pressure Increased right ventricular load Increased oxygen consumption literature review Pneumothorax alveolar rupture
  • 8. Sonde, tube Mechanism of bronchospasm General anesthesia Central nervous system Bronchial smooth muscle Local anesthesia The stimulation of surgery Stimulate, traction, The vagus nerve Bronchial parasympathetic ganglion rub Epithelium trachea The gases, exudates, blood or bronchi Increased bronchial response BRONCHOSPASM
  • 9. DEEP ANAESTHESIA Sonde, tube Mechanism of bronchospasm General anesthesia Central nervous system Bronchial smooth muscle Local anesthesia The stimulation of surgery Stimulate, traction, The vagus nerve Bronchial parasympathetic ganglion rub Epithelium trachea The gases, exudates, blood or bronchi Increased bronchial response BRONCHOSPASM
  • 10. Management of bronchospasm Stop all stimulation on patients Ventilate the patient with 100% oxygen Assess whether anaphylaxis is not? Yes Management of anaphylaxis Deep anesthesia Not Normotensive: Propofol IV Sevofluran Hypotension: Ketamine IV Optimal ventilation Control of the airway by intubation pressure control ventilation with prolonged exhalation time : I/E= 1/3, 1/4 Manual ventilation with 100% oxygen Use bronchodilators Salbutamol spray Intravenous bronchodilator : Salbutamol, Diaphyline If shock or use of bronchodilators failure: Adrenalin IV Solu-medrol IV
  • 11. Research Methodology - Study design : Retrospective, descriptive, cross-sectional. At the Department of Anesthesiology and Resuscitation Emergency Department, ENT Hospital National, within 15 years, the period from January 1999 to January 2014. - Subjects of study : The patient had been diagnosed with severe bronchospasm
  • 12. RESULTS Clinical characteristics and type of anesthesia Characteristics Age <18 years old: 1 18-60years old: 13 > 60years old : 3 Sex Males: 12 Females: 5 Period Intraoperative: 15 Intubation: 1 Extubation: 1 Diseases need surgery Tracheal stenosis: 13 Sinusitis: 1 Neck abscess: 1 Open glottis paralysis: 2 Type of anesthesia Local anesthesia: 15 General anesthesia:2 Cause Stimulation of surgery: 14 Reflux: 2 Drug reactions: 1
  • 13. Symptoms Signal Quantity Ratio % Subjective symptom Intense dyspnea 17 100% Tachypnea 3 Bradypnea 15 88,2% Stiffened body 12 70,5% Objective symptom Auscultation Murmur decreased alveolar 17 100% Wheezing 13 76,4% Lung silence 4 Perception Vague 12 41,2% Tumbledown 5 29,4% Heart rate Tachycardia 10 58,8% Bradycardia 7 41,2% Blood pressure Normotensive 2 Hypotension 15 88,2% SpO2 >90% 2 47% 50-90% 10 <50% 5 29,4% Pneumothorax Unilateral 3 Bilateral 12 70,5%
  • 14. Results of treatment Emergency Management General anesthesia, using bronchodilators, support 100% oxygen through the tube (17/17), tracheostomy set canuyl 14/17 patients, 3/17 patients intubation.. After the emergency management Pleural drainage and continuous suction, 15/17 patients. Sedation, pressure control ventilation, antibiotics, anti-inflammatory Outcome •12/17 patients recover completely and do not leave sequelae •2/17 patients were treated early and properly position the principle has recovered immediately. •3/17 patients died.
  • 15. DISCUSSION Clinical characteristics and type of anesthesia -13/17 (76.5%) tracheal stenosis scarring need to tracheostomy for airway control under local anesthesia. -According to Nathalie Schutz, the incidence rate of bronchospasm met 80% of the patients with airway obstruction and surgery without anesthesia. -Study on 31 patients who had surgical tracheostomy under local anesthesia by the author Rohail Ahmed, Muhammad Faheem Malik, the 2 patients with pneumothorax, 2 patients with cardiac arrest due to bronchospasm.
  • 16. The stage and cause of bronchospasm The stages R N Westhorpe Our Induction 48 1 Perioperative 66 15 Postoperative 11 1 Total 125 17 Causes Stimulates airway 23 15 Drug reactions 8 1 Early extubation 6 1 Other causes 5 0 Total 42 17
  • 17. Results of treatment -2/15 patients received early treatment were stable and without complications - 70% (12/17 patients) were pneumothorax were treated for pleural drainage continuous suction with negative pressure, pressure control ventilation, sedative, analgesic, antibiotic, anti-inflammatory and has stable after 5- 7 days. -3/17 patients died.
  • 18. CONCLUSIONS - Complications severe bronchospasm in ENT surgery met the majority of patients with tracheal stenosis scarring require surgery tracheostomy under local anesthesia (accounting for 76.5%). - 88.2% (15/17) patients with severe bronchospasm were pneumothorax and 3/17 patients died due to delays and improper management principles. - 2/17 patients received timely and proper management principles, has recovered completely without complications.
  • 19. Thanks for your attention!