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Pacemaker for beginners
KITA yosuke
Iizuka Hospital
Objectives
 Review basic pacemaker terminology
and function
 Discuss diagnosis and management of
pacemaker emergencies
Historical Perspective
 Electrical cardiac pacing for the
management of brady-arrhythmias was
first described in 1952
 Permanent transvenous pacing devices
were first introduced in the early 1960’s
Pacemaker Components
 Pulse Generator
 Electronic Circuitry
 Lead system
Pulse Generator
 Lithium-iodine cell is the current
standard battery
 Advantages:
 Long life – 4 to 10 years
 Output voltage decreases gradually with
time making sudden battery failure unlikely
Electronic Circuitry
 Determines the function of the
pacemaker itself
 Utilizes a standard nomenclature for
describing pacemakers
Pacemaker Nomenclature
I II III IV V
Chamber
Paced
Chamber
Sensed
Response to
Sensing
Rate Modulation,
Programmability
Anti-
tachycardia
Features
A=Atrium A=Atrium T=Triggered P=Simple P=Pacing
V=Ventricle V=Ventricle I=Inhibited M=Multi-
programmable
S=Shock
D=Dual D=Dual D=Dual R=Rate Adaptive D=Dual
O=None O=None O=None C=Communicating
O=None
Lead Systems
 Endocardial leads which are inserted
using a subclavian vein approach
 Actively fixed to the endocardium using
screws or tines
 Unipolar or bipolar leads
Electrocardiogram During
Cardiac Pacing
 Pacemaker has two main functions:
 Sense intrinsic cardiac electrical activity
 Electrically stimulate the heart
 VVI- senses intrinsic cardiac activity in
the ventricle and when a preset interval
of time with no ventricular activity
occurs it depolarizes the right ventricle
causing ventricular contraction
Pacer spike
Electrocardiogram
 Dual chamber pacer is more complicated
because the pacer has the ability to both
sense and pace either the atrium or the
ventricle
 Possible to have only atrial, only ventricular
or both atrial and ventricular pacing
 DDD pacer is a common example of this
Atrial Spike Ventricular Spike
AV Pacing Ventricular Pacing
Magnet Placement
 The EKG technician should perform a 12 lead
cardiogram and then a rhythm strip with a
magnet over the pacer
 Often a very poorly understood concept by
the non-cardiologist
 Does not inactivate the pacer as is commonly
believed
 Activate a lead switch present in the
pacemaker which converts the pacer to a
asynchronous or fixed-rate pacing mode
 Inhibits the sensing function of a pacemaker
Class I Indications For
Permanent Pacing
 Third degree AV block associated with:
 Symptomatic bradycardia
 Symptomatic bradycardia secondary to
drugs required for dysrhythmia
management
 Asystole > 3 seconds or escape rate < 40
 After catheter ablation of the AV node
 Post-op AV block not expected to resolve
 Neuromuscular disease with AV block
Indications
 Symptomatic bradycardia from second
degree AV block
 Bifascicular or trifascicular block with
intermittent third degree or type II second
degree block
 Sinus node dysfunction with symptomatic
bradycardia
 Recurrent syncope caused by carotid sinus
stimulation
Indications
 Post myocardial infarction with any of:
 Persistent second degree AV block with
bilateral bundle branch block or third
degree AV block
 Transient second or third degree AV block
and bundle branch block
 Symptomatic, persistent second or third
degree AV block
Infections
 Pacemaker insertion is a surgical
procedure:
 1% risk for bacteremia
 2% risk for wound or pocket infection
 Usually occur soon after pacer insertion
 Presence of a foreign body complicates
management
Infection
 Cellulitis or pocket infection:
 Tenderness and redness over the pacemaker
itself
 Avoid performing a needle aspiration – damage
the pacer
 Bacteremia: Staphylococcus
 aureus and Staphylococcus epi 60-70% of the
time
 Empiric antibiotics should include vancomycin
pending culture
Infection
 Consult the pacemaker physician
 Draw blood cultures
 Give appropriate antibiotics
 Frequently the pacer and lead system
need to be removed
Case 1
 67 year old male presents to the
emergency room 12 hours after
insertion of a pacemaker complaining
of left sided chest pain and shortness of
breath
 PR96, RR 33, BP 125/85, Oxygen
saturation 88% RA
 CXR as shown
Pneumothorax
 Occurs during cannulation of the
subclavian vien
 Incidence - ?? Cardiologist dependent
 Treatment:
 Asymptomatic or small – observation
 Symptomatic or large – tube thoracostomy
 Notify the pacemaker physician
Case 2
 72 year old male presents to the
emergency room after a fall, tripped
over a bath mat, no LOC
 Shortened and rotated left leg
 Past history – pacemaker, hypertension
 Nurse does an routine pre-op CXR and
EKG
Septal Perforation
 Usually identified at the time of pacer
insertion but leads can displace after
insertion
 Can occur with transvenous pacer
insertion
 Keys diagnosis are a RBBB pattern on
EKG and a pacer lead displaced to the
apex of the heart on CXR
Septal Perforation
 Management:
 Notify the pacer service
 Pacer wire has to be removed but not
emergently
 Small VSD which heals spontaneously
Conclusions
 Pacemakers are becoming more common
everyday
 We need to understand basic pacing
terminology and modes to treat patients
effectively.
 Most pacer malfunctions are due to failure to
sense, failure to capture, over-sensing, or in-
appropriate rate
 Standard ACLS protocols apply to all
unstable patients with pacemakers.

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pacer.ppt

  • 1. Pacemaker for beginners KITA yosuke Iizuka Hospital
  • 2. Objectives  Review basic pacemaker terminology and function  Discuss diagnosis and management of pacemaker emergencies
  • 3. Historical Perspective  Electrical cardiac pacing for the management of brady-arrhythmias was first described in 1952  Permanent transvenous pacing devices were first introduced in the early 1960’s
  • 4. Pacemaker Components  Pulse Generator  Electronic Circuitry  Lead system
  • 5. Pulse Generator  Lithium-iodine cell is the current standard battery  Advantages:  Long life – 4 to 10 years  Output voltage decreases gradually with time making sudden battery failure unlikely
  • 6. Electronic Circuitry  Determines the function of the pacemaker itself  Utilizes a standard nomenclature for describing pacemakers
  • 7. Pacemaker Nomenclature I II III IV V Chamber Paced Chamber Sensed Response to Sensing Rate Modulation, Programmability Anti- tachycardia Features A=Atrium A=Atrium T=Triggered P=Simple P=Pacing V=Ventricle V=Ventricle I=Inhibited M=Multi- programmable S=Shock D=Dual D=Dual D=Dual R=Rate Adaptive D=Dual O=None O=None O=None C=Communicating O=None
  • 8. Lead Systems  Endocardial leads which are inserted using a subclavian vein approach  Actively fixed to the endocardium using screws or tines  Unipolar or bipolar leads
  • 9.
  • 10.
  • 11. Electrocardiogram During Cardiac Pacing  Pacemaker has two main functions:  Sense intrinsic cardiac electrical activity  Electrically stimulate the heart  VVI- senses intrinsic cardiac activity in the ventricle and when a preset interval of time with no ventricular activity occurs it depolarizes the right ventricle causing ventricular contraction
  • 13. Electrocardiogram  Dual chamber pacer is more complicated because the pacer has the ability to both sense and pace either the atrium or the ventricle  Possible to have only atrial, only ventricular or both atrial and ventricular pacing  DDD pacer is a common example of this
  • 16. Magnet Placement  The EKG technician should perform a 12 lead cardiogram and then a rhythm strip with a magnet over the pacer  Often a very poorly understood concept by the non-cardiologist  Does not inactivate the pacer as is commonly believed  Activate a lead switch present in the pacemaker which converts the pacer to a asynchronous or fixed-rate pacing mode  Inhibits the sensing function of a pacemaker
  • 17.
  • 18. Class I Indications For Permanent Pacing  Third degree AV block associated with:  Symptomatic bradycardia  Symptomatic bradycardia secondary to drugs required for dysrhythmia management  Asystole > 3 seconds or escape rate < 40  After catheter ablation of the AV node  Post-op AV block not expected to resolve  Neuromuscular disease with AV block
  • 19. Indications  Symptomatic bradycardia from second degree AV block  Bifascicular or trifascicular block with intermittent third degree or type II second degree block  Sinus node dysfunction with symptomatic bradycardia  Recurrent syncope caused by carotid sinus stimulation
  • 20. Indications  Post myocardial infarction with any of:  Persistent second degree AV block with bilateral bundle branch block or third degree AV block  Transient second or third degree AV block and bundle branch block  Symptomatic, persistent second or third degree AV block
  • 21. Infections  Pacemaker insertion is a surgical procedure:  1% risk for bacteremia  2% risk for wound or pocket infection  Usually occur soon after pacer insertion  Presence of a foreign body complicates management
  • 22. Infection  Cellulitis or pocket infection:  Tenderness and redness over the pacemaker itself  Avoid performing a needle aspiration – damage the pacer  Bacteremia: Staphylococcus  aureus and Staphylococcus epi 60-70% of the time  Empiric antibiotics should include vancomycin pending culture
  • 23. Infection  Consult the pacemaker physician  Draw blood cultures  Give appropriate antibiotics  Frequently the pacer and lead system need to be removed
  • 24. Case 1  67 year old male presents to the emergency room 12 hours after insertion of a pacemaker complaining of left sided chest pain and shortness of breath  PR96, RR 33, BP 125/85, Oxygen saturation 88% RA  CXR as shown
  • 25.
  • 26. Pneumothorax  Occurs during cannulation of the subclavian vien  Incidence - ?? Cardiologist dependent  Treatment:  Asymptomatic or small – observation  Symptomatic or large – tube thoracostomy  Notify the pacemaker physician
  • 27. Case 2  72 year old male presents to the emergency room after a fall, tripped over a bath mat, no LOC  Shortened and rotated left leg  Past history – pacemaker, hypertension  Nurse does an routine pre-op CXR and EKG
  • 28.
  • 29.
  • 30. Septal Perforation  Usually identified at the time of pacer insertion but leads can displace after insertion  Can occur with transvenous pacer insertion  Keys diagnosis are a RBBB pattern on EKG and a pacer lead displaced to the apex of the heart on CXR
  • 31. Septal Perforation  Management:  Notify the pacer service  Pacer wire has to be removed but not emergently  Small VSD which heals spontaneously
  • 32. Conclusions  Pacemakers are becoming more common everyday  We need to understand basic pacing terminology and modes to treat patients effectively.  Most pacer malfunctions are due to failure to sense, failure to capture, over-sensing, or in- appropriate rate  Standard ACLS protocols apply to all unstable patients with pacemakers.