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Common chest X-rays
and common ECGs
Maryam Majid Al Ezairej
Collage of medicine
RAKMHSU
ECG
Electrocardiogram
Tracing of heart Tracing of heart’s electrical activity s electrical activity
12-leads ECG
 Check
 Name
 DoB
 time and date
 Indication
 Any previous or subsequent ECGs
 Is it part of a serial ECG sequence? In which case it may be numbered
rate
 HR of 60-100 per minute is normal
 HR > 100 = tachycardia
 HR < 60 = bradycardia
Rhythm
 Sinus
 Originating from SA node
 P wave before every QRS
 P wave in same direction as QRS
intervals
 PR
 0.20 sec (3-5)
 QRS
 0.08 – 0.10 sec (2.5)
 QT
 450 ms in men, 460 ms in women
 Based on sex / heart rate
 Half the R-R interval with normal HR
axis
 Left axis deviation:
 Right axis deviation
Chamber enlargement
atria
ventricles
RVH
Heart blocks
First degree AV block
 PR is fixed and longer than 0.2 sec
second degree block type 1 (Wenckebach)
second degree block type 2
3rd degree AV block
SVT
 Reentrant arrhythmia at AV node that is spontaneous in onset
 May have neck fullness, hypotension and/or polyuria due to ANP
 Narrow QRS with tachycardia
 First line is vagal maneuvers
 Second line is adenosine or verapamil
 For chronic SVT, class 1A or 1C or amiodarone or sotalol work well
 Ablation will cure it too, but we usually do this only in young patients
Narrow complex, regular; retrograde P
waves, rate <220
VT
 Impulse is initiated from the ventricle itself
 Wide QRS, Rate is 140-250
 If unstable DC cardiovert
 If not, IV Amiodarone and/or DCCV
 Consider procainamide
 Nonsustained ventricular tachycardia needs no treatment
Ventricular fibrillation
 Most common in acute MI, also drug overdose, anesthesia, hypothermia &
electric shock can precipitate
 Absence of ventricular complexes
 Usually terminal event
 Use Amiodarone if refractory to DCCV.
Atrial flutter
 Atrial activity of 240-320 with sawtooth pattern. Usually a 2:1 conduction
pattern; if it is 3:1 or higher, there is AV node damage
 Treatment is to slow AV node conduction with amiodarone, propafenone or
sotalol
 DC cardiovert if <48 hours or unstable
 You can also ablate the reentry pathway within the atrium between the
tricuspid and the IVC.
hypokalemia
U waves
Can also see PVCs, ST depression, small T waves
hyperkalemia
Tall, narrow and symmetric T waves
Differential Diagnosis of Tachycardia
Myocardial infarction
CHEST XRAY
1-R Atrium
2-R Ventricle
3-Apex of L Ventricle
4-Superior Vena Cava
5-Inferior Vena Cava
6. Tricuspid Valve
7-Pulmonary Valve
8-Pulmonary Trunk
9. R PA 10. L PA
consildations
The lung is said to be consolidated when the
alveoli and small airways are filled with
dense material.
This dense material may consist of:
Pus (pneumonia)
Fluid (pulmonary edema)
Blood (pulmonary hemorrhage)
Cells (cancer)
Atelectasis
Almost always associated with a linear
increased density due to volume loss
Indirect indications of volume loss include
vascular crowding or mediastinal shift toward
the collapse
Possible observance of hilar elevation with an
upper lobe collapse, or a hilar depression
with a lower lobe collapse
Pleural effusion
On an upright film, an effusion will cause blunting on the lateral costophrenic
sulcus and, if large enough, on the posterior costophrenic sulcus.
 Approximately 200 ml of fluid are needed to detect an effusion in a PA film,
while approximately 75 ml of fluid would be visible in the lateral view
In the AP film, an effusion will appear as a graded haze that is denser at the base
A lateral decubitus film is helpful in confirming an effusion as the fluid will
collect on the dependent side
A patient with bilateral pleural effusions.
Note the concave menisci blunting both posterior
costophrenic angles.
Pneumothorax
 Appears in the chest radiograph as air without lung markings
 In a PA film it is usually seen in the apices since the air rises to the least
dependent part of the chest
 The air is typically found peripheral to the white line of the visceral pleura
 Best demonstrated by an expiration film
pulmonary edema
There are two basic types of pulmonary edema:
 Cardiogenic pulmonary edema caused by increased hydrostatic pulmonary
capillary pressure
 Noncardiogenic pulmonary edema caused by either altered capillary
membrane permeability or decreased plasma oncotic pressure
Emphysema
Common features seen on the chest radiograph include:
 Hyperinflation with flattening of the diaphragms
 Increased retrosternal space
 Bullae
 Enlargement of PA/RV (cor pulmonale)
Hyperlucent lung fields
Multiple blebs
Avascular zones
Prominent pulmonary arteries
Lung tumours
 A lung mass will typically present as a lesion with sharp margins and a
homogenous appearance, in contrast to the diffuse appearance of an
infiltrate.
Pneumonia
Typical findings on the chest radiograph include:
 Airspace opacity
 Lobar consolidation
 Interstitial opacities
thank you 

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Common chest x rays and common ecgs

  • 1. Common chest X-rays and common ECGs Maryam Majid Al Ezairej Collage of medicine RAKMHSU
  • 2. ECG
  • 3. Electrocardiogram Tracing of heart Tracing of heart’s electrical activity s electrical activity
  • 5.
  • 6.  Check  Name  DoB  time and date  Indication  Any previous or subsequent ECGs  Is it part of a serial ECG sequence? In which case it may be numbered
  • 7.
  • 8.
  • 9. rate  HR of 60-100 per minute is normal  HR > 100 = tachycardia  HR < 60 = bradycardia
  • 10. Rhythm  Sinus  Originating from SA node  P wave before every QRS  P wave in same direction as QRS
  • 11. intervals  PR  0.20 sec (3-5)  QRS  0.08 – 0.10 sec (2.5)  QT  450 ms in men, 460 ms in women  Based on sex / heart rate  Half the R-R interval with normal HR
  • 12. axis
  • 13.  Left axis deviation:
  • 14.  Right axis deviation
  • 16. atria
  • 17.
  • 19.
  • 20. RVH
  • 22.
  • 23. First degree AV block  PR is fixed and longer than 0.2 sec
  • 24. second degree block type 1 (Wenckebach)
  • 26.
  • 27. 3rd degree AV block
  • 28.
  • 29.
  • 30.
  • 31.
  • 32.
  • 33. SVT  Reentrant arrhythmia at AV node that is spontaneous in onset  May have neck fullness, hypotension and/or polyuria due to ANP  Narrow QRS with tachycardia  First line is vagal maneuvers  Second line is adenosine or verapamil  For chronic SVT, class 1A or 1C or amiodarone or sotalol work well  Ablation will cure it too, but we usually do this only in young patients
  • 34. Narrow complex, regular; retrograde P waves, rate <220
  • 35. VT  Impulse is initiated from the ventricle itself  Wide QRS, Rate is 140-250  If unstable DC cardiovert  If not, IV Amiodarone and/or DCCV  Consider procainamide  Nonsustained ventricular tachycardia needs no treatment
  • 36. Ventricular fibrillation  Most common in acute MI, also drug overdose, anesthesia, hypothermia & electric shock can precipitate  Absence of ventricular complexes  Usually terminal event  Use Amiodarone if refractory to DCCV.
  • 37. Atrial flutter  Atrial activity of 240-320 with sawtooth pattern. Usually a 2:1 conduction pattern; if it is 3:1 or higher, there is AV node damage  Treatment is to slow AV node conduction with amiodarone, propafenone or sotalol  DC cardiovert if <48 hours or unstable  You can also ablate the reentry pathway within the atrium between the tricuspid and the IVC.
  • 38.
  • 39.
  • 40.
  • 41. hypokalemia U waves Can also see PVCs, ST depression, small T waves
  • 42. hyperkalemia Tall, narrow and symmetric T waves
  • 45.
  • 46.
  • 47.
  • 49.
  • 50.
  • 51. 1-R Atrium 2-R Ventricle 3-Apex of L Ventricle 4-Superior Vena Cava 5-Inferior Vena Cava 6. Tricuspid Valve 7-Pulmonary Valve 8-Pulmonary Trunk 9. R PA 10. L PA
  • 52.
  • 53. consildations The lung is said to be consolidated when the alveoli and small airways are filled with dense material. This dense material may consist of: Pus (pneumonia) Fluid (pulmonary edema) Blood (pulmonary hemorrhage) Cells (cancer)
  • 54. Atelectasis Almost always associated with a linear increased density due to volume loss Indirect indications of volume loss include vascular crowding or mediastinal shift toward the collapse Possible observance of hilar elevation with an upper lobe collapse, or a hilar depression with a lower lobe collapse
  • 55.
  • 56. Pleural effusion On an upright film, an effusion will cause blunting on the lateral costophrenic sulcus and, if large enough, on the posterior costophrenic sulcus.  Approximately 200 ml of fluid are needed to detect an effusion in a PA film, while approximately 75 ml of fluid would be visible in the lateral view In the AP film, an effusion will appear as a graded haze that is denser at the base A lateral decubitus film is helpful in confirming an effusion as the fluid will collect on the dependent side
  • 57. A patient with bilateral pleural effusions. Note the concave menisci blunting both posterior costophrenic angles.
  • 58. Pneumothorax  Appears in the chest radiograph as air without lung markings  In a PA film it is usually seen in the apices since the air rises to the least dependent part of the chest  The air is typically found peripheral to the white line of the visceral pleura  Best demonstrated by an expiration film
  • 59.
  • 60.
  • 61.
  • 62. pulmonary edema There are two basic types of pulmonary edema:  Cardiogenic pulmonary edema caused by increased hydrostatic pulmonary capillary pressure  Noncardiogenic pulmonary edema caused by either altered capillary membrane permeability or decreased plasma oncotic pressure
  • 63.
  • 64.
  • 65. Emphysema Common features seen on the chest radiograph include:  Hyperinflation with flattening of the diaphragms  Increased retrosternal space  Bullae  Enlargement of PA/RV (cor pulmonale)
  • 66. Hyperlucent lung fields Multiple blebs Avascular zones Prominent pulmonary arteries
  • 67.
  • 68.
  • 69.
  • 70. Lung tumours  A lung mass will typically present as a lesion with sharp margins and a homogenous appearance, in contrast to the diffuse appearance of an infiltrate.
  • 71.
  • 72. Pneumonia Typical findings on the chest radiograph include:  Airspace opacity  Lobar consolidation  Interstitial opacities
  • 73.

Editor's Notes

  1. Qrs
  2. Complete incomplete?
  3. Atelectasis Left Lung Homogenous density left hemithorax Mediastinal shift to left Left hemithorax smaller Diaphragm and heart silhouette are not identifiable
  4. Atelectasis Right Upper Lobe Density in the right upper lung field Transverse fissure pulled up Right hilum pulled up Smaller right lung Smaller right hemithorax
  5. gravity
  6. bullae (lucent, air-containing spaces that have no vessels and therefore are not perfused)  In smokers with known emphysema the upper lung zones are commonly more involved than the lower lobes. This situation is reversed in patients with alpha-1 anti-trypsin deficiency, where the lower lobes are affected.
  7. Lung mass Round or oval Sharp margin Homogenous No respect for anatomy
  8. Pneumonia is airspace disease and consolidation.  The air spaces are filled with bacteria or other microorganisms and pus. Pneumonia is NOT associated with volume loss.  What differentiates it from a mass? Masses are generally more well-defined.
  9. Consolidation Right Upper Lobe Density in right upper lung field Lobar density Loss of ascending aorta silhouette No shift of mediastinum Transverse fissure not significantly shifted Air bronchogram