This document provides an overview of common chest x-rays and electrocardiograms (ECGs). It discusses how to read ECGs, including assessing heart rate, rhythm, intervals, axis, and signs of chamber enlargement or blocks. Common arrhythmias like SVT, VT, atrial flutter are described. Chest x-ray findings for conditions like consolidation, atelectasis, pleural effusion, pneumothorax, pulmonary edema, emphysema, lung tumors and pneumonia are also reviewed. The document aims to equip readers with the basics of interpreting these common diagnostic imaging tests.
6. Check
Name
DoB
time and date
Indication
Any previous or subsequent ECGs
Is it part of a serial ECG sequence? In which case it may be numbered
7.
8.
9. rate
HR of 60-100 per minute is normal
HR > 100 = tachycardia
HR < 60 = bradycardia
11. intervals
PR
0.20 sec (3-5)
QRS
0.08 – 0.10 sec (2.5)
QT
450 ms in men, 460 ms in women
Based on sex / heart rate
Half the R-R interval with normal HR
33. SVT
Reentrant arrhythmia at AV node that is spontaneous in onset
May have neck fullness, hypotension and/or polyuria due to ANP
Narrow QRS with tachycardia
First line is vagal maneuvers
Second line is adenosine or verapamil
For chronic SVT, class 1A or 1C or amiodarone or sotalol work well
Ablation will cure it too, but we usually do this only in young patients
35. VT
Impulse is initiated from the ventricle itself
Wide QRS, Rate is 140-250
If unstable DC cardiovert
If not, IV Amiodarone and/or DCCV
Consider procainamide
Nonsustained ventricular tachycardia needs no treatment
36. Ventricular fibrillation
Most common in acute MI, also drug overdose, anesthesia, hypothermia &
electric shock can precipitate
Absence of ventricular complexes
Usually terminal event
Use Amiodarone if refractory to DCCV.
37. Atrial flutter
Atrial activity of 240-320 with sawtooth pattern. Usually a 2:1 conduction
pattern; if it is 3:1 or higher, there is AV node damage
Treatment is to slow AV node conduction with amiodarone, propafenone or
sotalol
DC cardiovert if <48 hours or unstable
You can also ablate the reentry pathway within the atrium between the
tricuspid and the IVC.
51. 1-R Atrium
2-R Ventricle
3-Apex of L Ventricle
4-Superior Vena Cava
5-Inferior Vena Cava
6. Tricuspid Valve
7-Pulmonary Valve
8-Pulmonary Trunk
9. R PA 10. L PA
52.
53. consildations
The lung is said to be consolidated when the
alveoli and small airways are filled with
dense material.
This dense material may consist of:
Pus (pneumonia)
Fluid (pulmonary edema)
Blood (pulmonary hemorrhage)
Cells (cancer)
54. Atelectasis
Almost always associated with a linear
increased density due to volume loss
Indirect indications of volume loss include
vascular crowding or mediastinal shift toward
the collapse
Possible observance of hilar elevation with an
upper lobe collapse, or a hilar depression
with a lower lobe collapse
55.
56. Pleural effusion
On an upright film, an effusion will cause blunting on the lateral costophrenic
sulcus and, if large enough, on the posterior costophrenic sulcus.
Approximately 200 ml of fluid are needed to detect an effusion in a PA film,
while approximately 75 ml of fluid would be visible in the lateral view
In the AP film, an effusion will appear as a graded haze that is denser at the base
A lateral decubitus film is helpful in confirming an effusion as the fluid will
collect on the dependent side
57. A patient with bilateral pleural effusions.
Note the concave menisci blunting both posterior
costophrenic angles.
58. Pneumothorax
Appears in the chest radiograph as air without lung markings
In a PA film it is usually seen in the apices since the air rises to the least
dependent part of the chest
The air is typically found peripheral to the white line of the visceral pleura
Best demonstrated by an expiration film
59.
60.
61.
62. pulmonary edema
There are two basic types of pulmonary edema:
Cardiogenic pulmonary edema caused by increased hydrostatic pulmonary
capillary pressure
Noncardiogenic pulmonary edema caused by either altered capillary
membrane permeability or decreased plasma oncotic pressure
63.
64.
65. Emphysema
Common features seen on the chest radiograph include:
Hyperinflation with flattening of the diaphragms
Increased retrosternal space
Bullae
Enlargement of PA/RV (cor pulmonale)
70. Lung tumours
A lung mass will typically present as a lesion with sharp margins and a
homogenous appearance, in contrast to the diffuse appearance of an
infiltrate.
71.
72. Pneumonia
Typical findings on the chest radiograph include:
Airspace opacity
Lobar consolidation
Interstitial opacities
Atelectasis Left Lung
Homogenous density left hemithorax
Mediastinal shift to left
Left hemithorax smaller
Diaphragm and heart silhouette are not identifiable
Atelectasis Right Upper Lobe
Density in the right upper lung field
Transverse fissure pulled up
Right hilum pulled up
Smaller right lung
Smaller right hemithorax
gravity
bullae (lucent, air-containing spaces that have no vessels and therefore are not perfused)
In smokers with known emphysema the upper lung zones are commonly more involved than the lower lobes. This situation is reversed in patients with alpha-1 anti-trypsin deficiency, where the lower lobes are affected.
Lung mass
Round or oval
Sharp margin
Homogenous
No respect for anatomy
Pneumonia is airspace disease and consolidation. The air spaces are filled with bacteria or other microorganisms and pus.
Pneumonia is NOT associated with volume loss.
What differentiates it from a mass? Masses are generally more well-defined.
Consolidation Right Upper Lobe
Density in right upper lung field
Lobar density
Loss of ascending aorta silhouette
No shift of mediastinum
Transverse fissure not significantly shifted
Air bronchogram