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Tramadol 200mg is a leading medicament that is prescribed to treat moderate to severe pain. It comes under the category of centrally acting analgesic. This drug recommended treating both chronic as well as acute pain. It is prescribed in the medical condition of fibromyalgia, motor neuron disease, restless legs syndrome, rheumatoid arthritis and other chronic pain. Generic Tramadol Hydrochloride being the active ingredient of this drug lessens the intensity of the pain by binding it to the µ-opioid
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Trasturel 150 mg injection consist a targeted cancer drug known as Trastuzumab, which is Pharmacologically grouped as humanized monoclonal antibody produced by recombinant DNA technology, it helps to reduce the rate of reactivation & mortality
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Piperaquine tetraphosphate 320 mg and dihydroartemisinin 40 mg film coated ta...Taj Pharma
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Tolterodine Tartrate2mg and 4mg Prolonged-release Capsules (Dezrol La) Taj Ph...TajPharmaQC
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Tasigna 150mg capsules | cancer treatmentsharonpaula
Inquire price of Tasigna 150mg Capsules composition of Nilotinib 150mg Capsules by Novartis Pharma, Blueberry pharmaceuticals is Worldwide Suppliers, Traders & wholesale exporter industry
Paracetamol and ibuprofen 500 mg and 150 mg film coated tablets smpc taj pha...Taj Pharma
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ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
Prix Galien International 2024 Forum ProgramLevi Shapiro
June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
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Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Phone Us ❤85270-49040❤ #ℂall #gIRLS In Surat By Surat @ℂall @Girls Hotel With...
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RX
TRIMETHOPRIM 80 MG
AND
SULFAMETHOXAZOLE
400 MG TABLETS
1.NAME OF THE MEDICINAL PRODUCT
Trimethoprim 80mg and sulfamethoxazole
400mg tablets
2. QUALITATIVE AND QUANTITATIVE
COMPOSITION
Each tablet contains: 80mg Trimethoprim BP
and 400mg Sulfamethoxazole BP.
Excipients:
For a full list of excipients, see section 6.1
3. PHARMACEUTICAL FORM
White uncoated tablets.
White, circular, biconvex uncoated tablets
4. CLINICAL PARTICULARS
4.1 Therapeutic indications
Co-trimoxazole is indicated in adults and
children (>12 to <18 years old) and adults (>18
years old).
Co-trimoxazole tablets are indicated for the
treatment of the following infections when
owing to sensitive organisms (see section 5.1):
- Treatment and prevention of Pneumocystis
jiroveci pneumonitis or 'PJP'.
- Treatment and prophylaxis of toxoplasmosis
- Treatment of nocardiosis.
The following infections may be treated with co-
trimoxazole where there is bacterial evidence of
sensitivity to co-trimoxazole and good reason to
prefer the combination of antibiotics in co-
trimoxazole to a single antibiotic:
- Acute uncomplicated urinary tract infection
- Acute otitis media
- Acute exacerbation of chronic bronchitis
Consideration should be given to official
guidance on the appropriate use of antibacterial
agents.
4.2 Posology and method of administration
Posology
General Dosage Recommendations
Where dosage is expressed as "tablets" this
refers to the adult tablet, i.e. 80 mg
Trimethoprim BP and 400 mg Sulfamethoxazole
BP. If other formulations are to be used
appropriate adjustment should be made.
Standard dosage recommendations for acute
infections
Adults (>18 years old):
Children over 12 years old (>12 to <18 years
old):
The standard dosage for children is equivalent to
approximately 6 mg trimethoprim and 30 mg
sulfamethoxazole per kg body weight per day,
given in two equally divided doses. The
schedules for children are according to the
child's age and provided in the table below:
Treatment should be continued until the patient
has been symptom free for two days; the
majority will require treatment for at least 5
days. If clinical improvement is not evident after
7 days therapy, the patient should be reassessed.
As an alternative to Standard Dosage for acute
uncomplicated lower urinary tract infections,
short-term therapy of 1 to 3 days duration has
been shown to be effective.
Elderly patients:
STANDARD DOSAGE
Age Tablets
>18 years old 2 tablets every 12 hours
Age Tablets
>12 to <18 years old 2 tablets every 12 hours
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See Special Warnings and Precautions for Use
(Section 4.4). Unless otherwise specified
standard dosage applies.
Impaired hepatic function:
No data are available relating to dosage in
patients with impaired hepatic function.
Impaired renal function:
Dosage recommendation:
Children (>12 to <18 years old) and adults
(>18 years old):
Creatinine
Clearance
(ml/min)
Recommended Dosage
>30 2 tablets every 12 hours
15 to 30 1 tablets every 12 hours
<15 Not recommended
No information available for children aged 12
years and under with renal failure. See section
5.2 for the pharmacokinetics in the paediatric
population with normal renal function of both
components of Co-Trimoxazole, TMP and SMZ.
Measurements of plasma concentration of
sulfamethoxazole at intervals of 2 to 3 days are
recommended in samples obtained 12 hours
after administration of Co-Trimoxazole. If the
concentration of total sulfamethoxazole exceeds
150 microgram/ml then treatment should be
interrupted until the value falls below 120
microgram/ml.
Pneumocystis jirovecii pneumonitis
Treatment - Children (>12 to <18 years old)
and adults (>18 years old):
A higher dosage is recommended, using 20 mg
trimethoprim and 100 mg sulfamethoxazole per
kg of body weight per day in two or more
divided doses for two weeks. The aim is to
obtain peak plasma or serum levels of
trimethoprim of greater than or equal to 5
microgram/ml (verified in patients receiving 1-
hour infusions of intravenous Co-Trimoxazole).
(See 4.8 Undesirable Effects).
Prevention - Adults (>18 years old):
The following dose schedules may be used:
160 mg trimethoprim/800 mg sulfamethoxazole
daily 7 days per week.
160 mg trimethoprim/800 mg sulfamethoxazole
three times per week on alternate days.
320 mg trimethoprim/1600 mg
sulfamethoxazole per day in two divided doses
three times per week on alternate days.
Prevention - Children (>12 to <18 years old):
The standard dosage for children is equivalent to
approximately 6 mg trimethoprim and 30 mg
sulfamethoxazole per kg body weight per day,
given in two equally divided doses. The
following dose schedules may be used for the
duration of the period at risk:
The daily dose given on a treatment day
approximates to 150 mg trimethoprim/m2
/day
and 750 mg sulfamethoxazole/m2
/day. The total
daily dose should not exceed 320 mg
trimethoprim and 1600 mg sulfamethoxazole.
Nocardiosis - Adults (>18 years old):
There is no consensus on the most appropriate
dosage. Adult doses of 6 to 8 tablets daily for up
to 3 months have been used.
Toxoplasmosis:
There is no consensus on the most appropriate
dosage for the treatment or prophylaxis of this
condition. The decision should be based on
clinical experience. For prophylaxis, however,
the dosages suggested for prevention
of Pneumocystis jirovecii pneumonitis may be
appropriate.
Age Tablets
>12 to <18 years
old
2 tablets every 12 hours, seven
days per week
>12 to <18 years
old
2 tablets every 12 hours, three
times per week on alternative days
>12 to <18 years
old
2 tablets every 12 hours, three
times per week on consecutive
days
>12 to <18 years
old
4 tablets once a day, three times
per week on consecutive days
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Method of administration:
Oral.
It may be preferable to take Co-Trimoxazole
with some food or drink to minimise the
possibility of gastrointestinal disturbances.
4.3 Contraindications
• Hypersensitivity to the active substance,
sulphonamides, trimethoprim, co-trimoxazole or
to any of the excipients listed in section 6.1.
• Contra-indicated in patients showing marked
severe liver parenchymal damage.
• Contra-indicated in severe renal insufficiency
where repeated measurements of the plasma
concentration cannot be performed.
• Co-Trimoxazole should not be given to infants
during the first 6 weeks of life.
• Co-Trimoxazole should not be given to
patients with a history of drug-induced immune
thrombocytopenia with use of trimethoprim
and/or sulphonamides.
• Co-Trimoxazole should not be given to
patients with acute porphyria.
4.4 Special Warnings and precautions for use
Fatalities, although very rare, have occurred due
to severe reactions including Stevens-Johnson
syndrome, toxic epidermal necrolysis, fulminant
hepatic necrosis, agranulocytosis, aplastic
anaemia, other blood dyscrasias and
hypersensitivity of the respiratory tract.
- Life-threatening cutaneous reactions Stevens-
Johnson syndrome (SJS) and toxic epidermal
necrolysis (TEN) and drug reaction with
eosinophilia and systemic symptoms (DRESS)
have been reported with the use of co-
trimoxazole.
- Patients should be advised of the signs and
symptoms and monitored closely for skin
reactions. The highest risk for occurrence of
SJS, TEN or DRESS is within the first weeks of
treatment.
- If symptoms or signs of SJS, TEN or DRESS
(e.g. progressive skin rash often with blisters or
mucosal lesions) are present, co-trimoxazole
treatment should be discontinued (see 4.8
Undesirable Effects).
- The best results in managing SJS, TEN and
DRESS come from early diagnosis and
immediate discontinuation of any suspect drug.
Early withdrawal is associated with a better
prognosis.
- If the patient has developed SJS or TEN with
the use of co-trimoxazole, co-trimoxazole must
not be re-started in this patient at any time.
Particular care is always advisable when treating
elderly patients because, as a group, they are
more susceptible to adverse reactions and more
likely to suffer serious effects as a result
particularly when complicating conditions exist,
e.g. impaired kidney and/or liver function and/or
concomitant use of other drugs.
For patients with known renal impairment
special measures should be adopted (see section
4.2).
An adequate urinary output should be
maintained at all times. Evidence of
crystalluria in vivo is rare, although sulfonamide
crystals have been noted in cooled urine from
treated patients. In patients suffering from
malnutrition the risk may be increased.
Regular monthly blood counts are advisable
when co-trimoxazole is given for long periods,
or to folate deficient patients or to the elderly;
since there exists a possibility of asymptomatic
changes in haematological laboratory indices
due to lack of available folate. Supplementation
with folinic acid may be considered during
treatment but this should be initiated with
caution due to possible interference with
antimicrobial efficacy (see section 4.5).
In glucose-6-phosphate dehydrogenase (G-6-
PD) deficient patients haemolysis may occur.
Co-trimoxazole should be given with caution to
patients with severe allergy or bronchial asthma.
Co-trimoxazole should not be used in the
treatment of streptococcal pharyngitis due to
Group A beta-haemolytic streptococci;
eradication of these organisms from the
oropharynx is less effective than with penicillin.
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Trimethoprim has been noted to impair
phenylalanine metabolism but this is of no
significance in phenylketonuric patients on
appropriate dietary restriction.
The administration of co-trimoxazole to patients
known or suspected to be at risk of acute
porphyria should be avoided. Both trimethoprim
and sulfonamides (although not specifically
sulfamethoxazole) have been associated with
clinical exacerbation of porphyria.
Close monitoring of serum potassium is
warranted in patients at risk of hyperkalaemia.
Co-Trimoxazole has been associated with
metabolic acidosis when other possible
underlying causes have been excluded. Close
monitoring is always advisable when metabolic
acidosis is suspected.
Except under careful supervision co-trimoxazole
should not be given to patients with serious
haematological disorders (see 4.8 Undesirable
Effects). Co-trimoxazole has been given to
patients receiving cytotoxic therapy with little or
no additional effect on the bone marrow or
peripheral blood.
The combination of antibiotics in co-trimoxazole
should only be used where, in the judgement of
the physician, the benefits of treatment outweigh
any possible risks; consideration should be given
to the use of a single effective antibacterial
agent.
4.5 Interaction with other medicinal products
and other forms of interaction
Interaction with laboratory tests: trimethoprim
may interfere with the estimation of
serum/plasma creatinine when the alkaline
picrate reaction is used. This may result in
overestimation of serum/plasma creatinine of the
order of 10%. The creatinine clearance is
reduced: the renal tubular secretion of creatinine
is decreased from 23% to 9% whilst the
glomerular filtration remains unchanged.
Zidovudine: in some situations, concomitant
treatment with zidovudine may increase the risk
of haematological adverse reactions to co-
trimoxazole. If concomitant treatment is
necessary, consideration should be given to
monitoring of haematological parameters.
Cyclosporin: reversible deterioration in renal
function has been observed in patients treated
with co-trimoxazole and cyclosporin following
renal transplantation.
Rifampicin: concurrent use of rifampicin and
Co-Trimoxazole results in a shortening of the
plasma half-life of trimethoprim after a period of
about one week. This is not thought to be of
clinical significance.
When trimethoprim is administered
simultaneously with drugs that form cations at
physiological pH, and are also partly excreted by
active renal secretion (e.g. procainamide,
amantadine), there is the possibility of
competitive inhibition of this process which may
lead to an increase in plasma concentration of
one or both of the drugs.
Diuretics (thiazides): in elderly patients
concurrently receiving diuretics, mainly
thiazides, there appears to be an increased risk of
thrombocytopenia with or without purpura.
Pyrimethamine: occasional reports suggest that
patients receiving pyrimethamine at doses in
excess of 25 mg weekly may develop
megaloblastic anaemia should co- trimoxazole
be prescribed concurrently.
Warfarin: co-trimoxazole has been shown to
potentiate the anticoagulant activity of warfarin
via stereo-selective inhibition of its metabolism.
Sulfamethoxazole may displace warfarin from
plasma-albumin protein-binding sites in vitro.
Careful control of the anticoagulant therapy
during treatment with Co-Trimoxazole is
advisable.
Phenytoin: co-trimoxazole prolongs the half-life
of phenytoin and if co-administered could result
in excessive phenytoin effect. Close monitoring
of the patient's condition and serum phenytoin
levels are advisable.
Digoxin: concomitant use of trimethoprim with
digoxin has been shown to increase plasma
digoxin levels in a proportion of elderly patients.
Methotrexate: co-trimoxazole may increase the
free plasma levels of methotrexate. If Co-
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Trimoxazole is considered appropriate therapy
in patients receiving other anti- folate drugs such
as methotrexate, a folate supplement should be
considered (see section 4.4).
Trimethoprim interferes with assays for serum
methotrexate when dihydrofolate reductase
from Lactobacillus casei is used in the assay. No
interference occurs if methotrexate is measured
by radioimmuno assay.
Lamivudine: administration of trimethoprim
/sulfamethoxazole 160 mg/800 mg (co-
trimoxazole) causes a 40% increase in
lamivudine exposure because of the
trimethoprim component. Lamivudine has no
effect on the pharmacokinetics of trimethoprim
or sulfamethoxazole.
Interaction with sulphonylurea
hypoglycaemic agents is uncommon but
potentiation has been reported.
Hyperkalaemia: caution should be exercised in
patients taking any other drugs that can cause
hyperkalaemia, for example ACE inhibitors,
angiotensin receptor blockers and potassium-
sparing diuretics such as spironolactone.
Concomitant use of trimethoprim-
sulfamethoxazole (co-trimoxazole) may result in
clinically relevant hyperkalaemia.
Repaglinide: trimethoprim may increase the
exposure of repaglinide which may result in
hypoglycaemia.
Folinic acid: folinic acid supplementation has
been shown to interfere with the antimicrobial
efficacy of trimethoprim-sulfamethoxazole. This
has been observed in Pneumocystis
jirovecii pneumonia prophylaxis and treatment.
Contraceptives: oral contraceptive failures have
been reported with antibiotics. The mechanism
of this effect has not been elucidated. Women on
treatment with antibiotics should temporarily use
a barrier method in addition to the oral
contraceptive, or choose another method of
contraception.
Azathioprine: There are conflicting clinical
reports of interactions between azathioprine and
trimethoprim-sulfamethoxazole, resulting in
serious haematological abnormalities.
4.6 Fertility, Pregnancy and lactation
Pregnancy
Trimethoprim and sulfamethoxazole cross the
placenta and their safety in pregnant women has
not been established. Case-control studies have
shown that there may be an association between
exposure to folate antagonists and birth defects
in humans.
Trimethoprim is a folate antagonist and, in
animal studies, both agents have been shown to
cause foetal abnormalities (see section 5.3).
Co-trimoxazole should not be used in
pregnancy, particularly in the first trimester,
unless clearly necessary. Folate supplementation
should be considered if co-trimoxazole is used in
pregnancy.
Sulfamethoxazole competes with bilirubin for
binding to plasma albumin. As significantly
maternally derived drug levels persist for several
days in the newborn, there may be a risk of
precipitating or exacerbating neonatal
hyperbilirubinaemia, with an associated
theoretical risk of kernicterus, when co-
trimoxazole is administered to the mother near
the time of delivery. This theoretical risk is
particularly relevant in infants at increased risk
of hyperbilirubinaemia, such as those who are
preterm and those with glucose-6-phosphate
dehydrogenase deficiency.
Breast-feeding
The components of co-trimoxazole
(trimethoprim and sulfamethoxazole) are
excreted in breast milk. Administration of co-
trimoxazole should be avoided in late pregnancy
and in lactating mothers where the mother or
infant has, or is at particular risk of developing,
hyperbilirubinaemia. Additionally,
administration of co-trimoxazole should be
avoided in infants younger than eight weeks in
view of the predisposition of young infants to
hyperbilirubinaemia.
4.7 Effects on ability to drive and use
machines
There have been no studies to investigate the
effect of co-trimoxazole on driving performance
or the ability to operate machinery. Further a
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detrimental effect on such activities cannot be
predicted from the pharmacology of the drug.
Nevertheless the clinical status of the patient and
the adverse events profile of co-trimoxazole
should be borne in mind when considering the
patients ability to operate machinery.
4.8 Undesirable Effects
The frequency categories associated with the
adverse events below are estimates. For most
events, suitable data for estimating incidence
were not available. In addition, adverse events
may vary in their incidence depending on the
indication.
Data from large published clinical trials were
used to determine the frequency of very
common to rare adverse events. Very rare
adverse events were primarily determined from
post-marketing experience data and therefore
refer to reporting rate rather than a "true"
frequency.
The following convention has been used for the
classification of adverse events in terms of
frequency: Very common ≥1/10, common
≥1/100 and <1/10, uncommon ≥1/1000 and
<1/100, rare ≥1/10,000 and <1/1000, very rare
<1/10,000, not known - cannot be estimated
from the available data.
System Organ
Class
Frequency Side effects
Infections and
infestations
Common Overgrowth fungal.
Very rare Pseudomembranous colitis
Blood and
lymphatic system
disorders
Very rare Leukopenia, neutropenia,
thrombocytopenia,
agranulocytosis, anaemia
megaloblastic, aplastic
anaemia, haemolytic anaemia,
methaemoglobinaemia,
eosinophilia, purpura,
haemolysis in certain
susceptible G-6-PD deficient
patients.
Immune system
disorders
Very rare Serum sickness, anaphylactic
reaction, allergic myocarditis,
hypersensitivity vasculitis
resembling Henoch-Schoenlein
purpura, periarteritis nodosa,
systemic lupus erythematosus.
Severe hypersensitivity
reactions associated with
PJP*, rash, pyrexia,
neutropenia,
thrombocytopenia, hepatic
enzyme increased,
hyperkalaemia,
hyponatraemia,
rhabdomyolysis.
Metabolism and
nutrition
disorders
Very common Hyperkalaemia.
Very rare Hypoglycaemia,
hyponatraemia, decreased
appetite, metabolic acidosis
Psychiatric
disorders
Very rare Depression, hallucination.
Not known Psychotic disorder.
Nervous system
disorders
Common Headache.
Very rare Meningitis aseptic *,
convulsions, neuropathy
peripheral, ataxia, dizziness.
Ear and labrynth
disorders
Very rare Vertigo, tinnitus
Eye disorders Very rare Uveitis
Respiratory,
thoracic and
mediastinal
disorders
Very rare Cough *, dyspnoea*, lung
infiltration*.
Gastrointestinal
disorders
Common Nausea, diarrhoea.
Uncommon Vomiting.
Very rare Glossitis, stomatitis,
pancreatitis.
Hepatobiliary
disorders
Very rare Transaminases increased,
blood bilirubin increased,
cholestatic jaundice, hepatic
necrosis
Skin and
subcutaneous
tissue disorders*
Common Rash
Very rare Photosensitivity reaction,
angiodema, dermatitis
exfoliative, fixed drug eruption,
erythema multiforme, Stevens-
Johnson syndrome (SJS) *,
toxic epidermal necrolysis
(TEN) *.
Not known Drug reaction with eosinophilia
and systemic symptoms
(DRESS)
Musculoskeletal
and connective
tissue disorders
Very rare Arthralgia, myalgia.
Renal and
urinary disorders
Very rare Renal impairment (sometimes
reported as renal failure),
tubulointerstitial nephritis and
uveitis syndrome, renal tubular
acidosis
* see description of selected adverse reactions
Description of selected adverse reactions
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Aseptic meningitis
Aseptic meningitis was rapidly reversible on
withdrawal of the drug, but recurred in a number
of cases on re-exposure to either co-trimoxazole
or to trimethoprim alone.
The majority of haematological changes are
mild and reversible when treatment is stopped.
Most of the changes cause no clinical symptoms
although they may become severe in isolated
cases, especially in the elderly, in those with
hepatic or renal dysfunction or in those with
poor folate status. Fatalities have been recorded
in at-risk patients and these patients should be
observed carefully (see 4.3 Contra-indications).
Close supervision is recommended when co-
trimoxazole is used in elderly patients or in
patients taking high doses of co-trimoxazole as
these patients may be more susceptible to
hyperkalaemia and hyponatraemia.
Pulmonary hypersensitivity reactions
Cough, dyspnoea and lung infiltration may be
early indicators of respiratory hypersensitivity
which, while very rare, has been fatal.
Hepatobiliary disorders
Jaundice cholestatic and hepatic necrosis may be
fatal.
Severe cutaneous adverse reactions (SCARs)
Stevens-Johnson syndrome (SJS) and toxic
epidermal necrolysis (TEN) have been reported
(see section 4.4).
Effects associated with Pneumocystis jirovecii
Pneumonitis (PJP) management.
Very rare: Severe hypersensitivity reactions,
rash, pyrexia, neutropenia, thrombocytopenia,
hepatic enzyme increased, hyperkalaemia,
hyponatraemia, rhabdomyolysis.
At the high dosages used for PJP management
severe hypersensitivity reactions have been
reported, necessitating cessation of therapy. If
signs of bone marrow depression occur, the
patient should be given calcium folinate
supplementation (5-10 mg/day). Severe
hypersensitivity reactions have been reported in
PJP patients on re-exposure to co-trimoxazole,
sometimes after a dosage interval of a few days.
Rhabdomyolysis has been reported in HIV
positive patients receiving trimethoprim-
sulfamethoxazole for prophylaxis or treatment of
PJP.
Reporting of suspected adverse reactions
Reporting suspected adverse reactions after
authorisation of the medicinal product is
important. It allows continued monitoring of the
benefit/risk balance of the medicinal product.
4.9 Overdose
Symptoms
Nausea, vomiting, dizziness and confusion are
likely signs/symptoms of overdosage. Bone
marrow depression has been reported in acute
trimethoprim overdosage.
Treatment
If vomiting has not occurred, induction of
vomiting may be desirable. Gastric lavage may
be useful, though absorption from the
gastrointestinal tract is normally very rapid and
complete within approximately two hours. This
may not be the case in gross overdosage.
Dependant on the status of renal function
administration of fluids is recommended if urine
output is low.
Both trimethoprim and active sulfamethoxazole
are moderately dialysable by haemodialysis.
Peritoneal dialysis is not effective.
5. PHARMACOLOGICAL PROPERTIES
5.1 Pharmacodynamic properties
Pharmacotherapeutic group: Combinations of
sulfonamides and trimethoprim, incl.
derivatives;
Mechanism of Action
Co-trimoxazole is an antibacterial drug
composed of two active principles,
sulfamethoxazole and trimethoprim.
Sulfamethoxazole is a competitive inhibitor of
dihydropteroate synthetase enzyme.
Sulfamethoxazole competitively inhibits the
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utilisation of para-aminobenzoic acid (PABA) in
the synthesis of dihydrofolate by the bacterial
cell resulting in bacteriostasis. Trimethoprim
reversibly inhibits bacterial dihydrofolate
reductase (DHFR), an enzyme active in the
folate metabolic pathway converting
dihydrofolate to tetrahydrofolate.Depending on
the conditions the effect may be bactericidal.
Thus trimethoprim and sulfamethoxazole block
two consecutive steps in the biosynthesis of
purines and therefore nucleic acids essential to
many bacteria. This action produces marked
potentiation of activity in vitro between the two
agents.
Trimethoprim binds to plasmodial DHFR but
less tightly than to the bacterial enzyme. Its
affinity for mammalian DHFR is some 50,000
times less than for the corresponding bacterial
enzyme.
Mechanism of resistance
In vitro studies have shown that bacterial
resistance can develop more slowly with both
sulfamethoxazole and trimethoprim in
combination that with either sulfamethoxazole
or trimethoprim alone.
Resistance to sulfamethoxazole may occur by
different mechanisms. Bacterial mutations cause
an increase the concentration of PABA and
thereby out-compete with sulfamethoxazole
resulting in a reduction of the inhibitory effect
on dihydropteroate synthetase enzyme. Another
resistance mechanism is plasmid-mediated and
results from production of an altered
dihydropteroate synthetase enzyme, with
reduced affinity for sulfamethoxazole compared
to the wild-type enzyme.
Resistance to trimethoprim occurs through a
plasmid-mediated mutation which results in
production of an altered dihydrofolate reductase
enzyme having a reduced affinity for
trimethoprim compared to the wild-type
enzyme.
Trimethoprim binds to plasmodial DHFR but
less tightly than to bacterial enzyme. Its affinity
for mammalian DHFR is some 50,000 times less
than for the corresponding bacterial enzyme.
Many common pathogenic bacteria are
susceptible in vitro to trimethoprim and
sulfamethoxazole at concentrations well below
those reached in blood, tissue fluids and urine
after the administration of recommended doses.
In common with other antibiotics, however, in
vitro activity does not necessarily imply that
clinical efficacy has been demonstrated and it
must be noted that satisfactory susceptibility
testing is achieved only with recommended
media free from inhibitory substances,
especially thymidine and thymine.
Susceptability testing breakpoints
EUCAST
Enterobacteriaceae: S≤ 2 R> 4
S. maltophilia: S≤ 4 R> 4
Acinetobacter: S≤ 2 R> 4
Staphylococcus: S≤ 2 R> 4
Enterococcus: S≤ 0.032 R> 1
Streptococcus ABCG: S≤ 1 R> 2
Streptococcus pneumoniae: S≤ 1 R> 2
Hemophilus influenza: S≤ 0.5 R> 1
Moraxella catarrhalis: S≤0.5 R >1
Psuedomonas aeruginosa and other non-
enterobacteriaceae: S≤ 2* R> 4*
S = susceptible, R = resistant. *These are CLSI
breakpoints since no EUCAST breakpoints are
currently available for these organisms.
Trimethoprim: sulfamethoxazole in the ratio
1:19. Breakpoints are expressed as trimethoprim
concentration.
Antibacterial Spectrum
The prevalence of resistance may vary
geographically and with time for selected
species and local information on resistance is
desirable, particularly when treating severe
infections. As necessary, expert advice should
be sought when the local prevalence of
resistance is such that the utility of the agent in
at least some types of infections is questionable.
This information gives only an approximate
guidance on probabilities whether
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microorganisms will be susceptible to
trimethoprim/sulfamethoxazole or not.
Trimethoprim/sulfamethoxazole susceptibility
against a number of bacteria are shown in the
table below:
Commonly susceptible species:
Gram-positive aerobes:
Staphylococcus aureus
Staphylococcus saprophyticus
Streptococcus pyogenes
Gram-negative aerobes:
Enterobacter cloacae
Haemophilus influenzae
Klebsiella oxytoca
Moraxella catarrhalis
Salmonella
spp.
Stenotrophomonas maltophilia
Yersinia spp.
Species for which acquired resistance may be a
problem:
Gram-positive aerobes:
Enterococcus faecalis
Enterococcus faecium
Nocardia
spp.
Staphylococcus epidermidis
Streptococcus pneumoniae
Gram-negative aerobes:
Citrobacter spp.
Enterobacter aerogenes
Escherichia coli
Klebsiella pneumoniae
Klebsiella pneumonia
Proteus mirabilis
Proteus vulgaris
Providencia spp.
Serratia marcesans
Inherently resistant organisms:
Gram-negative aerobes:
Pseudomonas aeruginosa
Shigella spp.
Vibrio cholera
5.2 Pharmacokinetic properties
Absorption
After oral administration trimethoprim and
sulfamethoxazole are rapidly and nearly
completely absorbed. The presence of food does
not appear to delay absorption. Peak levels in the
blood occur between one and four hours after
ingestion and the level attained is dose related.
Effective levels persist in the blood for up to 24
hours after a therapeutic dose. Steady state
levels in adults are reached after dosing for 2-3
days. Neither component has an appreciable
effect on the concentrations achieved in the
blood by the other.
Distribution
Approximately 50% of trimethoprim in the
plasma is protein bound.
Tissue levels of trimethoprim are generally
higher than corresponding plasma levels, the
lungs and kidneys showing especially high
concentrations. Trimethoprim concentrations
exceed those in plasma in the case of bile,
prostatic fluid and tissue, saliva, sputum and
vaginal secretions. Levels in the aqueous humor,
breast milk, cerebrospinal fluid, middle ear fluid,
synovial fluid and tissue (intestinal) fluid are
adequate for antibacterial activity. Trimethoprim
passes into amniotic fluid and foetal tissues
reaching concentrations approximating those of
maternal serum.
Approximately 66% of sulfamethoxazole in the
plasma is protein bound. The concentration of
active sulfamethoxazole in amniotic fluid,
aqueous humour, bile, cerebrospinal fluid,
middle ear fluid, sputum, synovial fluid and
tissue (interstitial) fluids is of the order of 20 to
50% of the plasma concentration.
Biotransformation
Renal excretion of intact sulfamethoxazole
accounts for 15-30% of the dose. This drug is
more extensively metabolised than
trimethoprim, via acetylation, oxidation or
glucuronidation. Over a 72 hour period,
approximately 85% of the dose can be accounted
for in the urine as unchanged drug plus the
major (N4-acetylated) metabolite.
Elimination
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The half-life of trimethoprim in man is in the
range 8.6 to 17 hours in the presence of normal
renal function. It is increased by a factor of 1.5
to 3.0 when the creatinine clearance is less than
10 ml/minute. There appears to be no significant
difference in elderly patients compared with
young patients.
The principal route of excretion of trimethoprim
is renal and approximately 50% of the dose is
excreted in the urine within 24 hours as
unchanged drug. Several metabolites have been
identified in the urine. Urinary concentrations of
trimethoprim vary widely.
The half-life of sulfamethoxazole in man is
approximately 9 to 11 hours in the presence of
normal renal function.
There is no change in the half-life of active
sulfamethoxazole with a reduction in renal
function but there is prolongation of the half-life
of the major, acetylated metabolite when the
creatinine clearance is below 25 ml /minute.
The principal route of excretion of
sulfamethoxazole is renal; between 15% and
30% of the dose recovered in the urine is in the
active form.
The pharmacokinetics in the paediatric
population with normal renal function of both
components of Co-Trimoxazole, TMP and SMZ
are age dependent. Elimination of TMP-SMZ is
reduced in neonates, during the first two months
of life, thereafter both TMP and SMZ show a
higher elimination with a higher body clearance
and a shorter elimination half-life. The
differences are most prominent in young infants
(> 1.7 months up to 24 months) and decrease
with increasing age, as compared to young
children (1 year up to 3.6 years), children (7.5
years and < 10 years) and adults (see section
4.2).
Trimethoprim is a weak base with a pKa of 7.4.
It is lipophilic. Tissue levels of trimethoprim are
generally higher than corresponding plasma
levels, the lungs and kidneys showing especially
high concentrations. Trimethoprim
concentrations exceed those in plasma in the
case of bile, prostatic fluid and tissue, saliva,
sputum and vaginal secretions. Levels in the
aqueous humor, breast milk, cerebrospinal fluid,
middle ear fluid, synovial fluid and tissue
(intestinal) fluid are adequate for antibacterial
activity. Trimethoprim passes into amniotic fluid
and foetal tissues reaching concentrations
approximating those of maternal serum.
In elderly patients there is a reduced renal
clearance of sulfamethoxazole.
Special patient population
Renal impairment
The elimination half-life of trimethoprim is
increased by a factor of 1.5-3.0 when the
creatinine clearance is less than 10 mL/minute.
When the creatinine clearance falls below 30
mL/min the dosage of Co-Trimoxazole should
be reduced (see section 4.2).
Hepatic impairment
Caution should be exercised when treating
patients with severe hepatic parenchymal
damage as there may be changes in the
absorption and biotransformation of
trimethoprim and sulfamethoxazole.
Elderly patients
In elderly patients, a slight reduction in renal
clearance of sulfamethoxazole but not
trimethoprim has been observed.
Paediatric population
See special dosage regimen (see section 4.2).
5.3 Preclinical safety data
Reproductive toxicology: At doses in excess of
recommended human therapeutic dose,
trimethoprim and sulfamethoxazole have been
reported to cause cleft palate and other foetal
abnormalities in rats, findings typical of a folate
antagonist. Effects with trimethoprim were
preventable by administration of dietary folate.
In rabbits, foetal loss was seen at doses of
trimethoprim in excess of human therapeutic
doses.
6. PHARMACEUTICAL PARTICULARS
6.1 List of excipients
Also contains: docusate sodium, magnesium
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stearate, maize starch, silica, sodium lauryl
sulfate, stearic acid, industrial methylated spirit.
6.2 Incompatibilities
None known.
6.3 Shelf life
Shelf-life
Three years from the date of manufacture.
Shelf-life after dilution/reconstitution
Not applicable.
Shelf-life after first opening
Not applicable.
6.4 Special precautions for storage
Store below 25°C in a dry place.
Protect from light.
6.5 Nature and contents of container
The product containers are rigid injection
moulded polypropylene or injection blow-
moulded polyethylene tablet containers with
polyfoam wad or polyethylene ullage filler and
snap-on polyethylene lids; in case any supply
difficulties should arise the alternative is amber
glass bottles with screw caps and polyfoam wad
or cotton wool. An alternative closure for
polyethylene containers is a polypropylene, twist
on, push down and twist off child-resistant,
tamper-evident lid.
The product may also be supplied in blister
packs in cartons:
a) Carton: Printed carton manufactured from
white folding box board.
b) Blister pack: (i) 250µm white rigid PVC. (ii)
Surface printed 20µm hard temper aluminium
foil with 5-7g/M² PVC and PVdC compatible
heat seal lacquer on the reverse side.
Pack size: 5's, 7's, 10's, 14's, 15's, 20's, 21's, 28's,
30's, 35's, 56's, 100's, 250's, 500's, 1000's.
Product may also be supplied in bulk packs, for
reassembly purposes only, in polybags contained
in tins, skillets or polybuckets filled with
suitable cushioning material.
Maximum size of bulk packs: 20,000.
6.6 Special precautions for disposal and
other handling
Not applicable.
7. MANUFACTURER:
Manufactured in India by:
TAJ PHARMACEUTICALS LTD.
Mumbai, India
At: 615, GIDC, Kerala, Bavla, Dist. Ahmedabad
438225, Gujarat, INDIA
This leaflet was last revised in October 2019