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Aprepitant 80mg, 125 mg hard capsules smpc taj pharmaceuticalsTaj Pharma
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Sunitinib Capsules 50mg Taj Pharma: Uses, Side Effects, Interactions, Pictures, Warnings, Sunitinib Dosage & Rx Info | Sunitinib Uses, Side Effects Sunitinib: Indications, Side Effects, Warnings, Sunitinib -Drug Information –Taj Pharma, Sunitinib dose Taj pharmaceuticals Sunitinib interactions, Taj Pharmaceutical Sunitinib contraindications, Sunitinib price, Sunitinib Taj Pharma Sunitinib SmPC-Taj Pharma Stay connected to all updated on Sunitinib Taj Pharmaceuticals Mumbai. Patient Information Leaflets, SmPC.
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Simvastatin 20 mg film coated tablets smpc- taj pharmaceuticalsTaj Pharma
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Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
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Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
New Drug Discovery and Development .....NEHA GUPTA
The "New Drug Discovery and Development" process involves the identification, design, testing, and manufacturing of novel pharmaceutical compounds with the aim of introducing new and improved treatments for various medical conditions. This comprehensive endeavor encompasses various stages, including target identification, preclinical studies, clinical trials, regulatory approval, and post-market surveillance. It involves multidisciplinary collaboration among scientists, researchers, clinicians, regulatory experts, and pharmaceutical companies to bring innovative therapies to market and address unmet medical needs.
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Pyrimethamine 25 mg tablets smpc taj pharmaceuticals
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RX
PYRIMETHAMINE
TABLETS
25 MG
1.NAME OF THE MEDICINAL PRODUCT
Pyrimethamine 25 mg Tablets.
2. QUALITATIVE AND QUANTITATIVE
COMPOSITION
Each tablet contains 25 mg of pyrimethamine.
For the full list of excipients, see section 6.1.
3. PHARMACEUTICAL FORM
Tablet.
Each tablet is white and round
4. CLINICAL PARTICULARS
4.1 Therapeutic indications
For the treatment of:
Toxoplasmosis, including ocular infections,
proven foetal infection following maternal
infection during pregnancy, and toxoplasmosis
in immune-deficient patients (for the treatment
of toxoplasmosis Pyrimethamine must always be
used in combination with a synergistic agent e.g.
sulphadiazine).
Treatment is not normally required for
asymtomatic or mild toxoplasma infection.
4.2 Posology and method of administration
Posology
Toxoplasmosis (including ocular infections)
Pyrimethamine should be given concurrently
with sulphadiazine or another appropriate
antibiotic.
In the treatment of toxoplasmosis, all patients
receiving Pyrimethamine should be given a
folinic acid supplement (calcium folinate) to
reduce the risk of bone marrow depression (see
section 4.4).
Pyrimethamine treatment should generally be
given for 3 to 6 weeks and not less than six
weeks in immunosuppressed patients. If further
therapy is indicated, a period of two weeks
should elapse between treatments.
There have been no dose response studies of
pyrimethamine in the treatment of
toxoplasmosis. The following recommendations
are therefore for guidance only.
Adults
A loading dose of Pyrimethamine 100 mg
should be given for the first 1 to 2 days,
followed by 25 mg to 50 mg daily. This should
be given together with 2 g to 4 g of
sulphadiazine daily in divided doses.
• Foetal toxoplasmosis during pregnancy
Pyrimethamine 50 mg every 12 hours for 2 days,
followed by 50 mg daily. This should be given
together with an initial dose of sulfadiazine 75
mg/kg, followed by 50 mg/kg every 12 hours (to
a maximum of 4 g daily) (see section 4.4 and
section 4.6).
Immune-deficient adults and adolescents
Guidelines for the treatment of opportunistic
infections in HIV-infected adults and
adolescents consider pyrimethamine plus
sulfadiazine to be the initial therapy of choice
for Toxoplasma gondii encephalitis and
recommend the following doses, based on body-
weight, be given for at least 6 weeks:
- less than 60 kg - pyrimethamine 200 mg orally,
followed by 50 mg daily plus sulfadiazine 1 g
orally every 6 hours
- 60 kg or more - pyrimethamine 200 mg orally,
followed by 75 mg daily plus sulfadiazine 1.5 g
orally every 6 hours.
Paediatric Population
Children over 6 years
A loading dose of Pyrimethamine 100 mg
should be given for the first 1 to 2 days,
followed by 25 mg to 50 mg daily. This should
be given together with 2 g to 4 g of
sulphadiazine daily in divided doses.
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Children aged 5 to 6 years
An initial dose of Pyrimethamine 2 mg/kg
bodyweight (to a maximum of 50 mg) followed
by 1 mg/kg bodyweight/day (to a maximum of
25 mg); combined with sulphadiazine 150
mg/kg bodyweight (maximum 2 g) daily in four
divided doses.
Immune-deficient children
Dosage regimens for immune-deficient children
are not defined.
Children under 5 years
There is insufficient data to provide specific
dose recommendations in children. This
formulation is not suitable for children under 5
years.
Elderly
There is no definitive information on the effect
of Pyrimethamine on elderly individuals. It is
theoretically possible that elderly patients might
be more susceptible to folate depression
associated with the daily administration of
Pyrimethamine in the treatment of
toxoplasmosis, and supplementation of folinic
acid is therefore essential (see section 4.2).
Patients with renal impairment
Pyrimethamine should be given with caution to
patients with renal impairment. Since
Pyrimethamine is co-administered with a
sulphonamide care should be taken to avoid
accumulation of the sulphonamide in patients
with renal impairment (see section 4.4).
Patients with hepatic impairment
Pyrimethamine should be given with caution to
patients with hepatic impairment. There are no
general recommendations for dosage reductions
for liver-impaired states but consideration
should be given to dose adjustments for
individual cases (see section 4.4).
Method of administration
For oral administration.
4.3 Contraindications
Pyrimethamine is contraindicated in:
Hypersensitivity to pyrimethamine or to any of
the excipients of this medicinal product.
Pyrimethamine should not generally be used
during the first trimester of pregnancy (see
section 4.6).
Since Pyrimethamine is to be taken in
conjunction with another drug for the indications
listed, the relevant prescribing information for
the synergistic agent should also be considered.
Breast-feeding should be avoided during
toxoplasmosis treatment. (See section 4.6).
4.4 Special Warnings and precautions for use
Depression of haematopoesis
Daily therapeutic doses of Pyrimethamine have
been shown to depress haematopoesis in 25% to
50% of patients. The likelihood of inducing
leucopenia, anaemia or thrombocytopenia is
reduced by concurrent administration of calcium
folinate. Pancytopenia, responsive to folate, has
been reported in patients with probable pre-
existing folate deficiency. Fatalities have
occurred in the absence of folate treatment.
Prevention of haematological toxicity
During pregnancy and in other conditions
predisposing to folate deficiency, a folate
supplement should be given. The co-
administration of a folate supplement is
necessary for treatment of toxoplasmosis (see
section 4.2). Full blood counts should be carried
out weekly during therapy and for a further two
weeks after treatment is stopped. In
immunosuppressed patients, full blood counts
should be carried out twice weekly. Should signs
of folate deficiency develop, treatment must be
discontinued and high doses of calcium folinate
administered. Calcium folinate should be used
because folic acid does not correct folate
deficiency due to dihydrofolate reductase
inhibitors.
Pyrimethamine may exacerbate folate deficiency
in subjects predisposed to this condition through
disease or malnutrition. Accordingly, a calcium
folinate supplement should be given to such
individuals. In patients with megaloblastic
anaemia due to folate deficiency the risks versus
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benefits of administering Pyrimethamine require
careful consideration.
Seizures
Caution should be exercised in administering
Pyrimethamine to patients with a history of
seizures; large loading doses should be avoided
in such patients (see section 4.8).
Risk of crystalluria
When a sulphonamide is given an adequate fluid
intake should be ensured to minimise the risk of
crystalluria.
Precautions applicable to sulphonamides
Since Pyrimethamine is administered with a
sulphonamide for the conditions indicated the
general precautions applicable to sulphonamides
should be observed.
Renal impairment
The kidney is not the major route of excretion of
pyrimethamine and excretion is not significantly
altered in patients with renal failure. There are,
however, no substantial data on the use of
Pyrimethamine in patients with renal
impairment, therefore Pyrimethamine should be
given with caution. Since Pyrimethamine is co-
administered with a sulphonamide, care should
be taken to avoid accumulation of the
sulphonamide in renally impaired patients.
Hepatic impairment
The liver is the main route for metabolism of
pyrimethamine. Data on the use of
Pyrimethamine in patients with liver disease are
limited. Pyrimethamine should be given with
caution to patients with hepatic impairment.
There are no general recommendations for
dosage reductions for liver-impaired states but
consideration should be given to dose
adjustment for individual cases.
Lactose
This medicinal product contains lactose. Patients
with rare hereditary problems of galactose
intolerance, the Lapp lactase deficiency or
glucose-galactose malabsorption should not take
this medicine.
4.5 Interaction with other medicinal products
and other forms of interaction
Folate inhibitors, agents associated with
myelosuppression
Pyrimethamine, by its mode of action, may
further depress folate metabolism in patients
receiving treatment with other folate inhibitors,
or agents associated with myelosuppression,
including cotrimoxazole, trimethoprim,
proguanil, zidovudine, or cytostatic agents (e.g.
methotrexate).
Cases of fatal bone marrow aplasia have been
associated with the administration of
daunorubicin, cytosine arabinoside and
pyrimethamine to individuals suffering from
acute myeloid leukaemia.
Megaloblastic anaemia has been reported
occasionally in individuals who took
pyrimethamine concurrently with a
trimethoprim/sulphonamide combination.
Methotrexate
Convulsions have occurred after concurrent
administration of methotrexate and
pyrimethamine to children with central nervous
system leukaemia.
Other antimalarial drugs
Seizures have occasionally been reported when
pyrimethamine was used in combination with
other antimalarial drugs.
Lorazepam
The concurrent administration of lorazepam and
Pyrimethamine may induce hepatotoxicity.
Antacid salts, kaolin
In vitro data suggest that antacid salts and the
anti-diarrhoeal agent kaolin reduce the
absorption of pyrimethamine.
Highly protein bound compounds
The high protein binding exhibited by
pyrimethamine may prevent protein binding by
other compounds (eg. quinine or warfarin). This
could affect the efficacy or toxicity of the
concomitant drug depending on the levels of
unbound drug.
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4.6 Fertility, Pregnancy and lactation
Pregnancy
Pyrimethamine should not be used during the
first trimester of pregnancy unless the benefits
outweigh the risk. Pyrimethamine has been
shown to be teratogenic in animal studies. The
risks associated with the administration of
Pyrimethamine must be balanced against the
dangers of abortion or foetal malformation due
to the infection.
Treatment with Pyrimethamine and sulfadiazine
during pregnancy is indicated in the presence of
confirmed placental or foetal infection or when
the mother is at risk of serious sequelae.
However, in view of the theoretical risk of foetal
abnormality arising from the use of
Pyrimethamine in early pregnancy, its use in
combination therapy should be restricted to the
second and third trimesters.
Pregnant women receiving Pyrimethamine must
be given a concurrent folinic acid supplement.
Breastfeeding
Pyrimethamine enters human breast milk. It has
been estimated that over a 9-day period an
average weight infant would receive about 45%
of the dose ingested by the mother. In view of
the high doses of pyrimethamine and concurrent
sulphonamides needed in toxoplasmosis
treatment, breast feeding should be avoided for
the duration of treatment.
Fertility
There are no relevant data available
4.7 Effects on ability to drive and use
machines
No studies on the effects on the ability to drive
and use machines have been performed. Some
patients may experience dizziness or
convulsions, therefore, caution is recommended
(see section 4.8).
4.8 Undesirable Effects
Since a concurrent sulphonamide is to be taken
with pyrimethamine for the indications listed,
the relevant prescribing information for the
sulphonamide should be consulted for
sulphonamide-associated adverse events.
It is important to note that the frequency
categories assigned for each adverse event
below are only estimates as suitable data for
accurately calculating incidence were not
available. Adverse events may vary in their
incidence according to the indication and the
possible contribution of concomitant
sulphonamides to the occurrence of these events
is unknown. In addition some events may be
related to the underlying disease.
Adverse drug reactions (ADRs) are listed below
by MedDRA system organ class and by
frequency.
Frequencies are defined as:
very common ≥1/10,
common ≥1/100 and <1/10,
uncommon ≥1/1000 and <1/100,
rare ≥1/10,000 and <1/1000,
very rare <1/10,000.
Blood and lymphatic system disorders
Very
common:
Anaemia
Common: Leucopenia, thromboctopenia
Very rare: Pancytopenia
Nervous system disorders
Very
common:
Headache
Common: Dizziness
Very rare: Convulsions (see section 4.4 and
section 4.7)
Respiratory, thoracic and mediastinal Disorders
Very rare: Pneumonia with cellular and
eosinophilic pulmonary infiltration
(observed when pyrimethamine was
administered once weekly in
association with sulfadoxine)
Gastrointestinal disorders
Very
common:
Vomiting, nausea, diarrhoea
Very rare: Colic, buccal ulceration
Skin and subcutaneous tissue disorders
Very
common:
Rash
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Uncommon: Abnormal skin pigmentation
Very rare: Dermatitis
General disorders and administrative site conditions
Uncommon: Fever
Reporting of suspected adverse reactions
Reporting suspected adverse reactions after
authorisation of the medicinal product is
important. It allows continued monitoring of the
benefit/risk balance of the medicinal product.
4.9 Overdose
Symptoms
Vomiting and convulsions occur in cases of
severe, acute overdoses. Ataxia, tremor and
respiratory depression can also occur. There
have been isolated cases with fatal outcomes
following acute overdose of pyrimethamine.
Chronic excess doses can result in bone marrow
depression (e.g. megaloblastic anaemia,
leucopenia, thrombocytopenia) resulting from
folic acid deficiency.
Management
Routine supportive treatment, including
maintenance of a clear airway and control of
convulsions.
Adequate fluids should be given to ensure
optimal diuresis.
To counteract possible folate deficiency,
calcium folinate should be given until signs of
toxicity have subsided. There may a delay of 7
to 10 days before the full leucopenic side effects
become evident, therefore calcium folinate
therapy should be continued for the period at
risk.
Further management should be as clinically
indicated or as recommended by the national
poisons centre, where available.
5. PHARMACOLOGICAL PROPERTIES
5.1 Pharmacodynamic properties
Pyrimethamine is an antiparasitic agent.
Pharmacotherapeutic group:
diaminopyrimidines.
Mechanism of Action
The antiparasitic action of pyrimethamine is due
to its specific activity on folic acid metabolism
in the Plasmodium and Toxoplasma parasites. In
this respect it competitively inhibits the
dihydrofolate reductase enzyme with an affinity
far greater for the protozoal than for the human
enzyme.
5.2 Pharmacokinetic properties
Absorption
Pyrimethamine is almost completely absorbed
from the gastrointestinal tract. Peak plasma
concentrations generally occur 2 to 4 hours after
a dose and can vary widely between individuals;
concentrations ranging from 260 to 1411 ng/ml
after daily oral doses of 25 mg.
Distribution
The volume of distribution for pyrimethamine is
approximately 2L/kg. In patients with HIV
infection, population pharmacokinetic analysis
has indicated that the mean volume of
distribution (corrected for bioavailability) is
246+/-64L.
About 80 to 90% of the pyrimethamine is bound
to plasma proteins.
Pyrimethamine is mainly concentrated in the
kidneys, lungs, liver, and spleen. In AIDS
patients given daily dose of pyrimethamine,
concentrations of about one-fifth of those in the
serum occur in cerebrospinal fluid.
Pyrimethamine crosses the placenta. It is
distributed into breast milk
Elimination
Pyrimethamine is predominantly metabolised by
the liver. The mean elimination half-life is 85
hours. Pyrimethamine is slowly excreted in
urine. In AIDS patients, the total clearance is
1.28+/-0.41L/h resulting in an elimination half
life of 139+/-34h. Data are lacking on the nature
of the metabolites of pyrimethamine, their
route/rate of formation and elimination in man
and any pharmacological activity, particularly
after prolonged daily dosing.
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Multiple dose studies indicate that steady state is
achieved in 12 to 20 days with daily dosing. It is
theoretically possible that metabolic pathways
might be saturable, leading to excessive
accumulation of the drug in some patients.
However, it has been demonstrated that plasma
levels are approximately proportional to dose at
steady state so this appears unlikely. Genetic
variation in the exposure to pyrimethamine has
been reported but these data are unsubstantiated.
Some studies in patients with AIDS have
indicated shorter half lives than those noted
above: these are very likely to be a consequence
of inappropriate sampling and analytical
techniques. However, if there are patients in
whom the half-life is particularly short, steady
state therapeutic levels might be inadequate.
5.3 Preclinical safety data
Mutagenicity
In microbial tests, pyrimethamine was found to
be non-mutagenic in the Ames Salmonella assay
whereas DNA damage was seen in the
Escherichia coli repair assay. Further in vitro
data indicate that pyrimethamine induces
mutagenic activity in mouse lymphoma cells in
the absence, but not in the presence of metabolic
activation.
Pyrimethamine also showed clastogenic activity
in mammalian lymphocytes in the absence of
metabolic activation.
Following intraperitoneal administration,
pyrimethamine has been shown to induce
chromosomal damage in male rodent germ cells
although studies in somatic cells (micronucleus
tests) are either negative or inconclusive. Studies
following oral administration of pyrimethamine
in rodents showed negative results in female
germ cells and in male and female bone
marrow/peripheral blood cells.
Carcinogenicity
A study in mice (dosed with either 500 or 1000
ppm pyrimethamine in the diet for 5 days per
week, for 78 weeks) showed no evidence of
carcinogenicity in females. Survival in the male
mice did not allow for an assessment of
carcinogenicity in this sex.
A similar study in rats dosed at 200 or 400 ppm
pyrimethamine showed no evidence of
carcinogenicity.
Teratogenicity
No changes in early development were seen in
embryos from 15 mice given a single intra-
gastric dose of pyrimethamine (50 mg/kg
bodyweight) on the first day of gestation.
However development of mouse and rat
embryos in culture was severely hindered by
pyrimethamine in a dose-dependent manner.
Pyrimethamine was teratogenic in rodents and in
the Gottingen minipig in a dose-dependent
manner.
Other studies in rats dosed at either 1 mg/kg or
10 mg/kg bodyweight showed some inhibition
of developmental processes but no teratological
effects.
Pyrimethamine was not teratogenic in rabbits at
dose levels up to 100 mg/kg bodyweight/day
administered on days 6 to 18 of pregnancy.
Pyrimethamine markedly reduced early stage
cell division in rabbit embryos but implantation
and foetal development were normal.
Fertility
A study in rats dosed with 5 mg/kg
bodyweight/day for 6 weeks resulted in reduced
sperm concentrations and testis weights, but
there were no effects on fertility. Reversible
arrest of spermatogenesis was shown in a study
on mice dosed with 200 mg/kg/day for 50 days.
However, this dose is far in excess of human
therapeutic doses.
6. PHARMACEUTICAL PARTICULARS
6.1 List of excipients
Lactose Monohydrate
Maize Starch
Hydrolysed Starch
Docusate sodium
Magnesium stearate
6.2 Incompatibilities
Not applicable.
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6.3 Shelf life
5 years
6.4 Special precautions for storage
Store below 30°C.
Store in the original container.
6.5 Nature and contents of container
PVC/PVdC/aluminium foil blister pack or
PVC/PVdC/child-resistant aluminium foil blister
pack
Pack size: 30 tablets
6.6 Special precautions for disposal and other
handling
Not applicable
7. MANUFACTURER:
Manufactured in India by:
TAJ PHARMACEUTICALS LTD.
Mumbai, India
At: 615, GIDC, Kerala, Bavla, Dist. Ahmedabad
438225, Gujarat, INDIA