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Trimethoprim 80mg and sulfamethoxazole 400mg tablets smpc taj pharmaceuticalsTaj Pharma
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Olmesartan 20mg/40mg Tablets USP Taj Pharma: Uses, Side Effects, Interactions, Pictures, Warnings, Olmesartan Dosage & Rx Info | Olmesartan Uses, Side Effects Vecuronium bromide: Indications, Side Effects, Warnings, Olmesartan-Drug Information –Taj Pharma, Olmesartan dose Taj pharmaceuticals Olmesartan interactions, Taj Pharmaceutical Olmesartan contraindications, Olmesartan price, Olmesartan Taj Pharma Olmesartan SmPC-Taj Pharma Stay connected to all updated on Olmesartan Taj Pharmaceuticals Mumbai. Patient Information Leaflets, SmPC.
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Fenofibrate 160 mg tablets smpc taj pharmaceuticalsTaj Pharma
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These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
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June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
- ADVANCES IN CARDIOLOGY: A NEW PARADIGM IS COMING
- WOMEN’S HEALTH: FERTILITY PRESERVATION
- WHAT’S NEW IN THE TREATMENT OF INFECTIOUS,
ONCOLOGICAL AND INFLAMMATORY SKIN DISEASES?
- ARTIFICIAL INTELLIGENCE AND ETHICS
- GENE THERAPY
- BEYOND BORDERS: GLOBAL INITIATIVES FOR DEMOCRATIZING LIFE SCIENCE TECHNOLOGIES AND PROMOTING ACCESS TO HEALTHCARE
- ETHICAL CHALLENGES IN LIFE SCIENCES
- Prix Galien International Awards Ceremony
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Report Back from SGO 2024: What’s the Latest in Cervical Cancer?bkling
Are you curious about what’s new in cervical cancer research or unsure what the findings mean? Join Dr. Emily Ko, a gynecologic oncologist at Penn Medicine, to learn about the latest updates from the Society of Gynecologic Oncology (SGO) 2024 Annual Meeting on Women’s Cancer. Dr. Ko will discuss what the research presented at the conference means for you and answer your questions about the new developments.
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
NVBDCP.pptx Nation vector borne disease control programSapna Thakur
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
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Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
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Hemodialysis: Chapter 3, Dialysis Water Unit - Dr.Gawad
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RX
METOCLOPRAMIDE
HYDROCHLORIDE
TABLETS
10MG
1.NAME OF THE MEDICINAL PRODUCT
Metoclopramide Hydrochloride 10 mg tablets
2. QUALITATIVE AND QUANTITATIVE
COMPOSITION
Each tablet contains
Metoclopramide Hydrochloride equivalent to 10
mg anhydrous Metoclopramide
HydrochlorideExcipients: 101.24 mg lactose
monohydrate
For a full list of excipients, see section 6.1.
3. PHARMACEUTICAL FORM
Tablet
White to off-white, round, biconvex tablets with
a scoreline on the other side.
The tablet can be divided into equal halves.
4. CLINICAL PARTICULARS
4.1 Therapeutic indications
Adult population
Metoclopramide is indicated in adults for:
- Prevention of delayed chemotherapy induced
nausea and vomiting (CINV)
- Prevention of radiotherapy induced nausea and
vomiting (RINV).
- Symptomatic treatment of nausea and
vomiting, including acute migraine induced
nausea and vomiting. Metoclopramide can be
used in combination with oral analgesics to
improve the absorption of analgesics in acute
migraine.
Paediatric population
Metoclopramide is indicated in children (aged 1-
18 years) for:
- Prevention of delayed chemotherapy induced
nausea and vomiting (CINV) as a second line
option
4.2 Posology and method of administration
Posology
Adult population
The recommended single dose is 10 mg,
repeated up to three times daily.
The maximum recommended daily dose is 30
mg or 0.5mg/kg body weight.
The maximum recommended treatment duration
is 5 days.
Prevention of delayed chemotherapy induced
nausea and vomiting (CINV) (paediatric patients
aged 1-18 years)
The recommended dose is 0.1 to 0.15 mg/kg
body weight, repeated up to three times daily by
oral route. The maximum dose in 24 hours is 0.5
mg/kg body weight.
Dosing table
The maximum treatment duration is 5 days for
prevention of delayed chemotherapy induced
nausea and vomiting (CINV).
Tablets are not suitable for use in children
weighing less than 30 kg.
Other pharmaceutical forms/strengths may be
more appropriate for administration to this
population.
Method of administration:
A minimal interval of 6 hours between two
administrations is to be respected, even in case
Age Body
Weight
Dose Frequency
1-3 years 10-14 kg 1 mg Up to 3 times daily
3-5 years 15-19 kg 2 mg Up to 3 times daily
5-9 years 20-29 kg 2.5 mg Up to 3 times daily
9-18 years 30-60 kg 5 mg Up to 3 times daily
15-18 years Over 60kg 10 mg Up to 3 times daily
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of vomiting or rejection of the dose (see section
4.4).
Special population
Elderly
In elderly patients a dose reduction should be
considered, based on renal and hepatic function
and overall frailty.
Renal impairment:
In patients with end stage renal disease
(Creatinine clearance ≤ 15 ml/min), the daily
dose should be reduced by 75%.
In patients with moderate to severe renal
impairment (Creatinine clearance 15-60
ml/min), the dose should be reduced by 50%
(see section 5.2).
Hepatic impairment:
In patients with severe hepatic impairment, the
dose should be reduced by 50% (see section
5.2).
Paediatric population
Metoclopramide is contraindicated in children
aged less than 1 year (see section 4.3).
4.3 Contraindications
• Hypersensitivity to the active substance or any
of the excipients listed in 6.1.
• Gastrointestinal haemorrhage, mechanical
obstruction or gastrointestinal perforation for
which the stimulation of gastrointestinal motility
constitutes a risk.
• A history of neuroleptic or metoclopramide-
induced tardive dyskinesia.
• Epilepsy (increased crises frequency and
intensity)
• Parkinson's disease
• Confirmed or suspected phaeochromocytoma
due to the risk of severe hypertension episode.
• Combination with levodopa or dopaminergic
agonists (see section 4.5).
• Known history of methaemoglobinaemia with
metoclopramide or of NADH cytochrome-b5
deficiency.
• Use in children less than 1 year of age due to
an increased risk of extrapyramidal disorders
(see section 4.4)
4.4 Special Warnings and precautions for use
Special warnings
Neurological Disorders
Extrapyramidal disorders may occur, particular
in children and young adults, and/or when high
doses are used.
These reactions occur usually at the beginning of
the treatment and can occur after a single
administration. Metoclopramide should be
discontinued immediately in the event of
extrapyramidal symptoms. These effects are
generally completely reversible after treatment
discontinuation, but may require a symptomatic
treatment (benzodiazepines in children and/or
anticholinergic anti-Parkinsonian medicinal
products in adults).
The time interval of at least 6 hours specified in
the section 4.2 should be respected between each
metoclopramide administration, even in case of
vomiting and rejection of the dose, in order to
avoid overdose.
Prolonged treatment with metoclopramide may
cause tardive dyskinesia, potentially irreversible,
especially in the elderly. Treatment should not
exceed 3 months because of the risk of tardive
dyskinesia (see section 4.8). Treatment must be
discontinued if clinical signs of tardive
dyskinesia appear.
Neuroleptic malignant syndrome has been
reported with metoclopramide in combination
with neuroleptics as well as with
metoclopramide monotherapy (see section 4.8).
Metoclopramide should be discontinued
immediately in the event of symptoms of
neuroleptic malignant syndrome and appropriate
treatment should be initiated.
Special care should be exercised in patients with
underlying neurological conditions and in
patients being treated with other centrally-acting
drugs (see section 4.3)
Symptoms of Parkinson's disease may also be
exacerbated by metoclopramide.
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Methaemoglobinemia
Methemoglobinemia which could be related to
NADH cytochrome b5 reductase deficiency has
been reported. In such cases, metoclopramide
should be immediately and permanently
discontinued and appropriate measures initiated
(such as treatment with methylene blue).
Cardiac Disorders
There have been reports of serious
cardiovascular undesirable effects including
cases of circulatory collapse, severe bradycardia,
cardiac arrest and QT prolongation following
dministration of metoclopramide by injection,
particularly via the intravenous route (see
section 4.8).
Special care should be taken when administering
metoclopramide, particularly via the intravenous
route to the elderly population, to patients with
cardiac conduction disturbances (including QT
prolongation), patients with uncorrected
electrolyte imbalance, bradycardia and those
taking other drugs known to prolong QT
interval.
Intravenous doses should be administered as a
slow bolus (at least over 3 minutes) in order to
reduce the risk of adverse effects (e.g.
hypotension, akathisia).
Renal and Hepatic Impairment
In patients with renal impairment or with severe
hepatic impairment, a dose reduction is
recommended (see section 4.2).
Metoclopramide Hydrochloride tablets contain
lactose. Patients with rare hereditary problems of
galactose intolerance, the Lapp lactase
deficiency or glucose-galactose malabsorption
should not use this medicine.
4.5 Interaction with other medicinal
products and other forms of interaction
Contraindicated combination
Levodopa or dopaminergic agonists and
metoclopramide have a mutual antagonism (see
section 4.3).
Combination to be avoided
Alcohol potentiates the sedative effect of
metoclopramide.
Combination to be taken into account
Due to the prokinetic effect of metoclopramide,
the absorption of certain drugs may be modified.
Anticholinergics and morphine derivatives
Anticholinergics and morphine derivatives may
have both a mutual antagonism with
metoclopramide on the digestive tract motility.
Central nervous system depressants (morphine
derivatives, anxiolytics, sedative H1
antihistamines, sedative antidepressants,
barbiturates, clonidine and related)
Sedative effects of Central Nervous System
depressants and metoclopramide are potentiated.
Neuroleptics
Metoclopramide may have an additive effect
with other neuroleptics on the occurrence of
extrapyramidal disorders.
Serotonergic drugs
The use of metoclopramide with serotonergic
drugs such as SSRIs may increase the risk of
serotonin syndrome.
Digoxin
Metoclopramide may decrease digoxin
bioavailability. Careful monitoring of digoxin
plasma concentration is required.
Cyclosporine
Metoclopramide increases cyclosporine
bioavailability (Cmax by 46% and exposure by
22%). Careful monitoring of cyclosporine
plasma concentration is required. The clinical
consequence is uncertain.
Mivacurium and suxamethonium
Metoclopramide injection may prolong the
duration of neuromuscular block (through
inhibition of plasma cholinesterase).
Strong CYP2D6 inhibitors
Metoclopramide exposure levels are increased
when co-administered with strong CYP2D6
inhibitors such as fluoxetine and paroxetine.
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Although the clinical significance is uncertain,
patients should be monitored for adverse
reactions.
4.6 Fertility, Pregnancy and lactation
Pregnancy
A large amount of data on pregnant women
(more than 1000 exposed outcomes) indicates no
malformative toxicity nor foetotoxicity.
Metoclopramide can be used during pregnancy
if clinically needed. Due to pharmacological
properties (as other neuroleptics), in case of
metoclopramide administration at the end of
pregnancy, extrapyramidal syndrome in
newborn cannot be excluded. Metoclopramide
should be avoided at the end of pregnancy. If
metoclopramide is used, neonatal monitoring
should be undertaken.
Breastfeeding
Metoclopramide is excreted in breast milk at low
level. Adverse reactions in the breast-fed baby
cannot be excluded. Therefore metoclopramide
is not recommended during breastfeeding.
Discontinuation of metoclopramide in
breastfeeding women should be considered.
4.7 Effects on ability to drive and use
machines
Metoclopramide may cause drowsiness,
dizziness, dyskinesia and dystonias which could
affect the vision and also interfere with the
ability to drive and operate machinery.
4.8 Undesirable Effects
Adverse reactions listed by System Organ Class.
Frequencies are defined using the following
convention: very common (≥1/10), common
(≥1/100, <1/10), uncommon (≥1/1000, <1/100),
rare (≥1/10000, <1/1000), very rare (<1/10000),
not known (cannot be estimated from the
available data).
Systeme
Organ
Class
Frequency Adverse reactions
Blood and lymphatic system disorders
Not known Methaemoglobinaemia,
which could be related to
NADH cytochrome b5
reductase deficiency,
particularly in neonates (see
section 4.4)
Sulfhaemoglobinaemia,
mainly with concomitant
administration of high doses
of sulphur-releasing
medicinal products
Cardiac disorders
Uncommon Bradycardia, particularly
with intravenous
Formulation
Not known Cardiac arrest, occurring
shortly after injectable use,
and which can be subsequent
to bradycardia (see section
4.4);
Atrioventricular block, Sinus
arrest particularly with
intravenous formulation;
Electrocardiogram QT
prolonged; Torsade de
Pointes;
Endocrine disorders*
Uncommon Amenorrhoea,
Hyperprolactinaemia,
Rare Galactorrhoea
Not known Gynaecomastia
Gastrointestinal disorders
Common Diarrhoea
General disorders and administration site conditions
Common Asthenia
Immune
system
disorders
Uncommon Hypersensitivity
Not known Anaphylactic reaction
(including anaphylactic
shock particularly with
intravenous formulation
Nervous
system
disorders
Nervous
system
disorders
Very
Common
Somnolence
Common Extrapyramidal disorders
(particularly in children and
young adults and/or when
the recommended dose is
exceeded, even following
administration of a single
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dose of the drug) (see
section 4.4), Parkinsonism,
Akathisia
Uncommon Dystonia, Dyskinesia,
Depressed level of
Consciousness
Rare Convulsion especially in
epileptic patients
Not known Tardive dyskinesia which
may be persistent, during or
after prolonged treatment,
particularly in elderly
patients (see section 4.4),
Neuroleptic malignant
syndrome (see section 4.4)
Psychiatric disorders
Common Depression
Uncommon Hallucination
Rare Confusional state
Vascular disorder
Common Hypotension, particularly
with intravenous
Formulation
Not known Shock, syncope after
injectable use Acute
hypertension in patients with
phaeochromocytoma (see
section 4.3), Transient
increase in blood pressure
* Endocrine disorders during prolonged
treatment in relation with hyperprolactinaemia
(amenorrhoea, galactorrhoea, gynaecomastia).
The following reactions, sometimes associated,
occur more frequently when high doses are used:
- Extrapyramidal symptoms: acute dystonia and
dyskinesia, parkinsonian syndrome, akathisia,
even following administration of a single dose of
the medicinal product, particularly in children
and young adults (see section 4.4).
- Drowsiness, decreased level of consciousness,
confusion, hallucination.
Reporting of suspected adverse reactions
Reporting suspected adverse reactions after
authorisation of the medicinal product is
important. It allows continued monitoring of the
benefit/risk balance of the medicinal product.
4.9 Overdose
Symptoms
Extrapyramidal disorders, drowsiness, decreased
level of consciousness, confusion, hallucination,
and cardio-respiratory arrest may occur.
Management
In case of extrapyramidal symptoms related or
not to overdose, the treatment is only
symptomatic (benzodiazepines in children
and/or anticholinergic anti-parkinsonian
medicinal products in adults).
A symptomatic treatment and a continuous
monitoring of the cardiovascular and respiratory
functions should be carried out according to
clinical status.
5. PHARMACOLOGICAL PROPERTIES
5.1 Pharmacodynamic properties
Metoclopramide is a substituted benzamide. It is
used among other things because of its anti-
emetic properties. The anti-emetic effect is the
result of two mechanisms of action involving the
central nervous system:
• antagonism of the dopaminergic D2 receptors
in the chemoreceptor trigger zone and in the
vomiting centre of the medulla which is affected
in apomorphine-induced vomiting;
• antagonism of the serotoninergic 5HT3
receptors and agonist effect on the 5HT4
receptors which are affected in chemotherapy-
induced vomiting.
In addition to the central action, metoclopramide
has a stimulant effect on gastrointestinal motility
via a peripheral mechanism of action. There is
an antidopaminergic effect and potentiation of
the effect of acetylcholine. This causes
accelerated emptying of the stomach and there is
an increase in the pressure exerted by the lower
oesophageal sphincter. Metoclopramide has no
effect on gastric secretions.
5.2 Pharmacokinetic properties
Following oral administration the relative
bioavailability compared with intravenous
administration is 60 to 100%. Peak plasma
concentrations are reached within 0.5 to 2 hours.
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The distribution volume is 2-3 l/kg; 13-22% is
bound to plasma proteins. Metoclopramide is
excreted primarily in the urine, both in
unchanged form and in sulfate or glucuronide
conjugate form. The principal metabolite is an
N-4 sulphur conjugate.
The plasma elimination half-life is 5 to 6 hours,
irrespective of the route of administration.
Special patient populations
Renal impairment
The clearance of metoclopramide is reduced by
up to 70% in patients with severe renal
impairment, while the plasma elimination half-
life is increased (approximately 10 hours for a
creatinine clearance of 10-50 mL/minute and 15
hours for a creatinine clearance <10
mL/minute).
Hepatic impairment
In patients with cirrhosis of the liver,
accumulation of metoclopramide has been
observed, associated with a 50% reduction in
plasma clearance.
5.3 Preclinical safety data
No abnormalities have been found in animal
studies to indicate a safety risk in humans. This
is based on data from pharmacological studies
relating to safety, and data on toxicity following
repeated administration, genotoxicity,
carcinogenicity and reproductive toxicity.
6. PHARMACEUTICAL PARTICULARS
6.1 List of excipients
The tablet contains the following excipients:
Lactose monohydrate.
Pre-gelatinised starch.
Maize starch.
Anhydrous colloidal silica.
Magnesium stearate.
6.2 Incompatibilities
No particulars.
6.3 Shelf life
The shelf life is 2 years in
PVC/PVdC/aluminium blister strips.
6.4 Special precautions for storage
Store below 30 °C.
6.5 Nature and contents of container
Metoclopramide Hydrochloride Tablets are
packaged in PVC/PVdC/aluminium blister
strips. The carton contains 20, 24, 28, 30, 40, 50,
60, 84, 100, 500 tablets.
6.6 Special precautions for disposal and other
handling
No special requirements.
7. MANUFACTURER:
Manufactured in India by:
TAJ PHARMACEUTICALS LTD.
Mumbai, India
At: 615, GIDC, Kerala, Bavla, Dist. Ahmedabad
438225, Gujarat, INDIA
This leaflet was last revised in October 2019