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Irinotecan hydrochloride 20 mgml, concentrate for solution for infusion smpc ...Taj Pharma
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How to reduce fever, pain and inflammation in toddlers?avanlimedia
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Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
Acute scrotum is a general term referring to an emergency condition affecting the contents or the wall of the scrotum.
There are a number of conditions that present acutely, predominantly with pain and/or swelling
A careful and detailed history and examination, and in some cases, investigations allow differentiation between these diagnoses. A prompt diagnosis is essential as the patient may require urgent surgical intervention
Testicular torsion refers to twisting of the spermatic cord, causing ischaemia of the testicle.
Testicular torsion results from inadequate fixation of the testis to the tunica vaginalis producing ischemia from reduced arterial inflow and venous outflow obstruction.
The prevalence of testicular torsion in adult patients hospitalized with acute scrotal pain is approximately 25 to 50 percent
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Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
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micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
New Drug Discovery and Development .....NEHA GUPTA
The "New Drug Discovery and Development" process involves the identification, design, testing, and manufacturing of novel pharmaceutical compounds with the aim of introducing new and improved treatments for various medical conditions. This comprehensive endeavor encompasses various stages, including target identification, preclinical studies, clinical trials, regulatory approval, and post-market surveillance. It involves multidisciplinary collaboration among scientists, researchers, clinicians, regulatory experts, and pharmaceutical companies to bring innovative therapies to market and address unmet medical needs.
These lecture slides, by Dr Sidra Arshad, offer a quick overview of physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar leads (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Couples presenting to the infertility clinic- Do they really have infertility...Sujoy Dasgupta
Dr Sujoy Dasgupta presented the study on "Couples presenting to the infertility clinic- Do they really have infertility? – The unexplored stories of non-consummation" in the 13th Congress of the Asia Pacific Initiative on Reproduction (ASPIRE 2024) at Manila on 24 May, 2024.
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Hemodialysis: Chapter 3, Dialysis Water Unit - Dr.Gawad
Ibuprofen 400 mg film coated tablets smpc- taj pharmaceuticals
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RX
IBUPROFEN FILM
COATED
TABLETS
400MG
1.NAME OF THE MEDICINAL PRODUCT
Ibuprofen 400 mg film-coated tablets
2. QUALITATIVE AND QUANTITATIVE
COMPOSITION
Each film-coated tablet contains 400 mg
ibuprofen
Excipient with known effect: each tablet
contains 47 mg lactose.
For the full list of excipients, see section 6.1.
3. PHARMACEUTICAL FORM
Film-coated tablet
Ibuprofen 400 mg film-coated tablets: pink
coloured, round, approximately 13 mm in
diameter, biconvex film coated tablets
4. CLINICAL PARTICULARS
4.1 Therapeutic indications
In adults and children over 12 years (>40 kg):
Short term treatment of fever and pain of mild to
moderate intensity, including dysmenorrhea.
Long term symptomatic treatment of pain and
inflammation in chronic inflammatory rheumatic
diseases.
4.2 Posology and method of administration
Posology
The treatment should start with the lowest dose
anticipated to be effective, which can
subsequently be adjusted, depending on the
therapeutic response and any undesirable effects.
In long-term treatment a low maintenance dose
should be the aim.
Undesirable effects may be minimised by using
the lowest effective dose for the shortest
duration necessary to control symptoms (see
section 4.4).
Rheumatic diseases
- initial treatment:2x400 mg tablets, three times
daily, i.e. 2400 mg per day
- maintenance treatment : 1x400 mg tablets,
three to four times daily, i.e. 1200 to 1600 mg
per day.
An interval of at least 4-6 hours should be
allowed between doses. The total daily dose
must not exceed 2400mg in divided doses. Some
patients can be maintained on 600 – 1200mg
daily.
Juvenile Rheumatoid Arthritis
For Juvenile Rheumatoid Arthritis, the
recommended dose is 20mg-30mg/kg body
weight daily in 3 to 4 divided doses up to a
maximum of 40mg/kg body weight daily in
severe cases. In children weighing less than 40
kg, use of a different strength or a liquid
formulation of ibuprofen should be considered
to aid accurate dosing.
Short term symptomatic treatment of fever and
pain of mild to moderate intensity:
Adults and adolescents over 12 years (>40 kg)
One 400 mg tablet one to four times daily, as
needed. An interval of at least 4-6 hours should
be allowed between doses. Single doses
exceeding 400 mg have not been shown to have
any additional analgesic effect. The 400 mg
tablet should only be used for more intense pain
or fever that are not relieved by a 200 mg
ibuprofen tablet.
Dysmenorrhoea
One 400 mg tablet one to three times daily, as
needed. An interval of at least 4-6 hours should
be allowed between doses. Treatment is started
at the first sign of menstruation problems.
Elderly
The elderly are at increased risk of serious
consequences of adverse reactions. If an NSAID
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is considered necessary, the lowest effective
dose should be used and for the shortest possible
duration. The patient should be monitored
regularly for GI bleeding during NSAID
therapy. If renal or hepatic function is impaired,
dosage should be assessed individually.
Renal impairment
Caution should be taken with ibuprofen dosage
in patients with renal impairment. The dosage
should be assessed individually. The dose
should be kept as low as possible and renal
function should be monitored (see sections 4.3,
4.4 and 5.2).
Hepatic impairment
Caution should be taken with dosage in patients
with hepatic impairment. The dosage should be
assessed individually and the dose should be
kept as low as possible (see sections 4.3, 4.4 and
5.2)
Method of administration
For oral use.
It is recommended that patients with sensitive
stomachs take ibuprofen tablet with food. If
taken shortly after eating, the onset of action of
ibuprofen tablet may be delayed. To be taken
preferably with or after food, with plenty of
fluid. Ibuprofen tablets should be swallowed
whole and not chewed, broken, crushed or
sucked on to avoid oral discomfort and throat
irritation.
4.3 Contraindications
Hypersensitivity to the active substance or to
any of the excipients listed in section 6.1.
Active gastric or duodenal ulcer or a history of
recurrent gastrointestinal ulcer/bleeding (two or
more clear episodes of demonstrable ulceration
or bleeding).
Severe hepatic failure.
Severe heart failure (NYHA Class IV) or
coronary heart disease.
Severe renal failure (glomerular filtration below
30 mL/min).
Conditions involving an increased tendency to
bleeding.
Gastrointestinal bleeding or perforation in
connection with previous treatment with
NSAIDs.
The third trimester of pregnancy (see section
4.6).
Because of cross-reactions, ibuprofen should not
be given to patients who have developed
hypersensitivity reactions, including symptoms
of asthma, rhinitis or urticaria after taking
acetylsalicylic acid or other NSAIDs.
In patients with cerebrovascular or other acute
bleeding
Hematologic diseases (e.g. hemorrhagic
diathesis, hematopoetic disorder)
In patients with severe dehydration (caused by
vomiting, diarrhoea or insufficient fluid intake),
Colitis ulcerosa
4.4 Special Warnings and precautions for use
General precautions
Undesirable effects may be minimised by using
the lowest effective dose for the shortest
duration necessary to control symptoms (see
section 4.2, and gastrointestinal and
cardiovascular effects below).
As with other NSAIDs, ibuprofen may mask the
signs of infection.
There is some evidence that drugs which inhibit
cyclo-oxygenase/ prostaglandin synthesis may
cause impairment of female fertility by an effect
on ovulation. This is reversible on withdrawal of
treatment.
Ibuprofen tablets contain lactose monohydrate
and should not be given to patients with rare
hereditary problems of galactose intolerance,
Lapp lactase deficiency or glucose-galactose
malabsorption.
Prolonged use of any type of painkiller for
headaches can make them worse. If this situation
is experienced or suspected, medical advice
should be obtained and treatment should be
discontinued. The diagnosis of medication
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overuse headache (MOH) should be suspected in
patients who have frequent or daily headaches
despite (or because of) the regular use of
headache medications.
Cardiovascular effects
Appropriate monitoring and advice are required
for patients with a history of hypertension and/or
mild to moderate heart failure as fluid retention
and oedema have been reported in association
with NSAID therapy. NSAIDs might decrease
the effect of diuretics and other antihypertensive
agents (see section 4.5).
Clinical studies suggest that use of ibuprofen,
particularly at a high dose (2400 mg/ day) may
be associated with a small increased risk of
arterial thrombotic events (for example
myocardial infarction or stroke). Overall,
epidemiological studies do not suggest that low
dose ibuprofen (e.g. ≤ 1200 mg/day) is
associated with an increased risk of arterial
thrombotic events.
Patients with uncontrolled hypertension,
congestive heart failure (NYHA II-III),
established ischaemic heart disease, peripheral
arterial disease, and/or cerebrovascular disease
should only be treated with ibuprofen after
careful consideration and high doses (2400
mg/day) should be avoided.
Careful consideration should also be exercised
before initiating long-term treatment of patients
with risk factors for cardiovascular events (e.g.
hypertension, hyperlipidaemia, diabetes
mellitus, smoking), particularly if high doses of
ibuprofen (2400 mg/day) are required.
Gastrointestinal bleeding, ulceration and
perforation
There is a strong link between the dose and
severe gastrointestinal bleeding. The
concomitant administration of ibuprofen and
other NSAIDs, including selective
cyclooxygenase-2 (COX-2) inhibitors should be
avoided.
Elderly patients are at greater risk of
experiencing undesirable effects when treated
with an NSAID, especially gastrointestinal
bleeding and perforation, which may be fatal.
Potentially fatal gastrointestinal bleeding,
ulceration and perforation have been reported in
connection with treatment with all types of
NSAID and have occurred at any time during
treatment, with or without warning symptoms or
previous episodes of severe gastrointestinal
events.
The risk of gastrointestinal bleeding, ulceration
or perforation is higher at increased doses of
NSAIDs in patients with a history of ulcer,
especially if complicated with bleeding or
perforation (see section 4.3), and in the elderly.
Patients with the above-mentioned risk factors
should commence treatment at the lowest
possible dose.
Treatment with mucosa-protective drugs (e.g.
misoprostol or proton pump inhibitors) should
be considered for these patients as well as for
patients on low doses of a acetylsalicylic acid or
other drugs that may increase the risk of
undesirable gastrointestinal effects (see below
and section 4.5).
Patients with a history of gastrointestinal
reactions, particularly elderly patients, should be
told to watch out for any unusual abdominal
symptoms (especially gastrointestinal bleeding),
particularly at the start of the treatment and, if
such symptoms occur, to seek medical help.
Caution should be exercised in patients
receiving concomitant medication which could
increase the risk of ulceration or bleeding, such
as oral corticosteroids, anticoagulants such as
warfarin, selective serotonin re-uptake inhibitors
or antiplatelet drugs such as acetylsalicylic acid
(see section 4.5).
Treatment with ibuprofen should be withdrawn
if the patient suffers from gastrointestinal
bleeding or ulceration.
NSAIDs should be given with care to patients
with a history of gastrointestinal disease, e.g.
ulcerative colitis and Crohn's disease, as these
conditions may be exacerbated (see section 4.8).
Renal effects
Caution should be exercised with regard to
dehydrated patients. There is a risk of renal
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impairment in dehydrated children and
adolescents.
As with other NSAIDs, the long-term
administration of ibuprofen has resulted in
papillary necrosis and other pathological
changes in the kidney. Renal toxicity has also
been seen in patients in whom renal
prostaglandins have a compensatory role in the
maintenance of normal renal perfusion. In these
patients the administration of an NSAID may
cause a dose-dependent reduction in
prostaglandin formation and, secondarily, in
renal blood flow, which may cause kidney
failure. Those who are at greatest risk of this are
patients with renal impairment, heart failure,
hepatic dysfunction, the elderly and patients on
diuretics or ACE inhibitors. Discontinuation of
NSAID therapy is usually followed by recovery
to the pre-treatment state.
Ibuprofen can cause fluid and sodium and
potassium retention in patients who have never
suffered from renal disorders due to its effects
on renal perfusion. This can cause oedema or
heart failure or hypertension in predisposed
subjects.
In general, regular use of analgesics, especially
combinations of various analgesic agents, has
the potential to cause permanent renal damage
including the risk of renal failure (analgesics
nephropathy).
Haematological Effects
Ibuprofen can inhibit platelet aggregation,
resulting in prolongation of bleeding time.
Hence, careful monitoring of patients with
coagulation disorders or taking anticoagulants is
recommended.
Respiratory disorders
Caution is required if ibuprofen is administered
to patients suffering from, or with a previous
history of, bronchial asthma, chronic rhinitis or
allergic disease since ibuprofen have been
reported to cause bronchospasm, urticaria or
angioedema in such patients.
Dermatological effects
Severe skin reactions, some with a fatal
outcome, such as exfoliative dermatitis, Stevens-
Johnson syndrome and toxic epidermal
necrolysis, have been reported very rarely in
connection with the use of NSAIDs (see section
4.8). The risk of such reactions occurring is
greatest at the start of the treatment, the majority
of cases occurring during the first month.
Treatment with ibuprofen should be withdrawn
at the first signs of skin rash, mucosal lesions or
other signs of hypersensitivity and if visual
disorders or persistent signs of hepatic
dysfunction appear.
SLE and mixed connective tissue disease
In patients with systemic lupus erythematosus
(SLE) and mixed connective tissue disorders
there may be an increased risk of aseptic
meningitis (see below and section 4.8).
Aseptic meningitis
Aseptic meningitis has been observed on rare
occasions in patients on ibuprofen therapy.
Although it is probably more likely to occur in
patients with systemic lupus erythematosus and
related connective tissue diseases, it has been
reported in patients who do not have an
underlying chronic disease.
Infections and infestations
Exceptionally, varicella can be at the origin of
serious cutaneous and soft tissues infectious
complications.
To date, the contributing role of NSAIDs in the
worsening of these infections cannot be ruled
out. Thus, it is advisable to avoid use of
ibuprofen in case of varicella.
Hypersensitivity reactions
Analgesics, antipyretics and non-steroidal anti-
inflammatory drugs (NSAIDs) can cause
potentially serious hypersensitivity reactions,
including anaphylactic reactions, even in
subjects with no previous exposure to this type
of drug. The risk of hypersensitivity after
ibuprofen intake appears to be higher in patients
who have previously exhibited hypersensitivity
to other analgesic agents, antipyretics, NSAIDs
and in patients with bronchial hyper-
responsiveness (asthma), hay fever, nasal polyps
or chronic obstructive pulmonary disease or
previous episodes of angioedema (see sections
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4.3 and 4.8). The allergic reactions may present
as asthma attacks (so-called analgesic asthma),
Quincke's oedema or hives.
Severe hypersensitivity reactions (e.g.
anaphylactic shock) have been rarely reported.
The treatment with ibuprofen should be
immediately withdrawn at the first sign of
hypersensitivity reaction.
Reduced cardiac, renal and hepatic function
In patients with renal, cardiac, or hepatic
impairment, caution is required since the use of
NSAIDs may result in deterioration of renal
function. This risk is further increased in
patients taking combinations of different
analgesic agents regularly. The lowest effective
dose for the shortest period of time and periodic
monitoring of clinical and laboratory
parameters, especially in case of prolonged
treatment, is recommended in patients with
renal, cardiac, or hepatic impairment (see
section 4.3).
Medication overuse headache
Prolonged use of any type of painkiller for
headaches can make them worse. If this situation
is experienced or suspected, medical advice
should be obtained and treatment should be
discontinued. The diagnosis of medication
overuse headache (MOH) should be suspected in
patients who have frequent or daily headaches
despite (or because of) the regular use of
headache medications.
Patients with gastrointestinal problems, SLE,
haematological or coagulation disorders and
asthma should be treated with care and be
closely monitored during NSAID treatment,
since their condition may be exacerbated by the
NSAID.
4.5 Interaction with other medicinal products
and other forms of interaction
The following combinations with Ibuprofen
should be avoided:
The dicumarol group: NSAIDs may increase the
effect of anticoagulants such as warfarin
Experimental studies show that ibuprofen
reinforces the effects of warfarin on bleeding
time. NSAIDs and the dicumarol group are
metabolised by the same enzyme, CYP2C9.
Anti-platelet agent: NSAIDs should not be
combined with antiplatelet agents such as
ticlopidine due to the additive inhibition of the
platelet function (see below).
Methotrexate: NSAIDs inhibit the tubular
secretion of methotrexate and some metabolic
interaction with reduced clearance of
methotrexate may also occur as a result.
Accordingly, in high-dose treatment with
methotrexate one should always avoid
prescribing NSAIDs (see below).
Acetylsalicylic acid: Concomitant administration
of ibuprofen and acetylsalicylic acid is not
generally recommended because of the potential
of increased adverse effects.
Experimental data suggest that ibuprofen may
competitively inhibit the effect of low dose
acetylsalicylic acid on platelet aggregation when
they are dosed concomitantly. Although there
are uncertainties regarding extrapolation of these
data to the clinical situation, the possibility that
regular, long-term use of ibuprofen may reduce
the cardioprotective effect of low-dose
acetylsalicylic acid cannot be excluded. No
clinically relevant effect is considered to be
likely for occasional ibuprofen use (see section
5.1).
Cardiac glycosides: NSAIDs can exacerbate
heart failure, reduce glomerular filtration and
increase plasma cardiac glycoside (e.g. digoxin)
levels.
Mifepristone: A decrease in the efficacy of the
medicinal product can theoretically occur due to
the antiprostaglandin properties of non-steroidal
anti-inflammatory drugs (NSAIDs) including
acetylsalicylic acid. Limited evidence suggests
that co-administration of NSAIDs on the day of
prostaglandin administration does not adversely
influence the effects of mifepristone or the
prostaglandin on cervical ripening or uterine
contractility and does not reduce the clinical
efficacy of medical termination of pregnancy
Sulfonylureas: NSAIDs may potentiate the
effects of sulfonylurea medications. There are
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rare reports of hypoglycaemia in patients on
sulfonylurea medications receiving ibuprofen.
Zidovudine: There is evidence of an increased
risk of haemarthroses and haematoma in HIV(+)
haemophiliacs receiving concurrent treatment
with zidovudine and ibuprofen.
The following combinations with Ibuprofen may
require dose adjustment:
Antihypertensives: NSAIDs can reduce the
effect of diuretics and other antihypertensive
agents.
Aminoglycosides: NSAIDs may reduce the
excretion of aminoglycosides. Children: Care
should be taken during concomitant treatment
with ibuprofen and aminoglycosides.
Lithium: Ibuprofen reduces the renal clearance
of lithium, as a result of which serum lithium
levels may rise. The combination should be
avoided unless frequent checks of serum lithium
can be carried out and a possible reduction in the
dose of lithium made.
ACE inhibitors and angiotensin-II antagonists:
There is an increased risk of acute renal failure,
usually reversible, in patients with renal
impairment (e.g. dehydrated and/or elderly
patients) when treatment with ACE inhibitors or
angiotensin-II antagonists is given at the same
time as NSAIDs, including selective
cyclooxygenase-2 inhibitors. The combination
should, therefore, be given with care to patients
with renal impairment, especially elderly
patients. Patients should be adequately hydrated
and a check of renal function should be
considered after the initiation of combination
treatment and at regular intervals during
treatment (see section 4.4).
Beta-blockers: NSAIDs counteract the
antihypertensive effect of beta-adrenoceptor
blocking drugs.
Selective serotonin re-uptake inhibitors (SSRIs):
SSRIs and NSAIDs each entail an increased risk
of bleeding, e.g. from the gastrointestinal tract.
This risk is increased by combination therapy.
The mechanism may possibly be linked to
reduced uptake of serotonin in the platelets (see
section 4.4).
Cyclosporine: The concomitant administration
of NSAIDs and cyclosporine is thought to be
capable of increasing the risk of nephrotoxicity
due to decreased synthesis of prostacyclin in the
kidney. Accordingly, in the event of
combination treatment, renal function must be
monitored closely.
Captopril: Experimental studies indicate that
ibuprofen counteracts the effect of captopril on
sodium excretion.
Colestyramine: The concomitant administration
of ibuprofen and colestyramine retards and
reduces (by 25%) the absorption of ibuprofen.
These drugs should be given at an interval of at
least 2 hours.
Thiazides, thiazide-related preparations and
loop diuretics: NSAIDs can counteract the
diuretic effect of furosemide and bumetanide,
possibly through inhibition of prostaglandin
synthesis. They can also counteract the
antihypertensive effect of thiazides.
Tacrolimus: Concomitant administration of
NSAIDs and tacrolimus is thought to be capable
of increasing the risk of nephrotoxicity due to
decreased synthesis of prostacyclin in the
kidney. Accordingly, in the event of
combination treatment, renal function should be
monitored closely.
Methotrexate: The risk of a potential interaction
between an NSAID and methotrexate should
also be taken into account in connection with
low-dose treatment with methotrexate,
especially in patients with renal impairment.
Whenever combination treatment is given, renal
function should be monitored. Caution should be
exercised if both an NSAID and methotrexate
are given within 24 hours, as the plasma levels
of methotrexate can increase, resulting in
increased toxicity (see above).
Corticosteroids: Concomitant treatment gives
rise to an increased risk of gastrointestinal
ulceration or bleeding.
Antiplatelet drugs: Increased risk of
gastrointestinal bleeding (see above).
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Quinolone antibiotics: Animal data indicate that
NSAIDs can increase the risk of convulsions
associated with quinolone antibiotics. Patients
taking NSAIDs and quinolones may have an
increased risk of developing convulsions.
Other analgesics and cyclooxygenase-2 selective
inhibitors: Avoid concomitant use of two or
more NSAIDs, including Cox-2 inhibitors, as
this may increase the risk of adverse effects (see
section 4.4).
Herbal extracts: Ginkgo biloba may potentiate
the risk of bleeding with NSAIDs.
CYP2C9 Inhibitors: Concomitant administration
of ibuprofen with CYP2C9 inhibitors may
increase the exposure to ibuprofen (CYP2C9
substrate). In a study with voriconazole and
fluconazole (CYP2C9 inhibitors) an increased
S(+)-ibuprofen exposure by approximately 80 to
100% has been shown. Reduction of the
ibuprofen dose should be considered when
potent CYP2C9 inhibitors are administered
concomitantly, particularly when high-dose
ibuprofen is administered with either
voriconazole or fluconazole.
Ritonavir: It is a possible increase in the
concentration of NSAIDs.
Probenecid: It slows the excretion of NSAIDs,
with possible increase of their plasma
concentrations.
Pemetrexed: An interaction with pemetrexed as
there is an increased risk of its toxicity by
decreased renal clearance. In patients with an
impaired renal function displaying a creatinine
clearance between 45-80 ml/min, this
combination is to be avoided. In patients with
normal renal function, a precaution for use is
sufficient based on laboratory tests of the renal
function.
Interaction studies have only been performed on
adults.
4.6 Fertility, Pregnancy and lactation
Pregnancy
Inhibition of prostaglandin synthesis may
adversely affect the pregnancy and/or
embryo/foetal development. Data from
epidemiological studies suggest an increased
risk of miscarriage and of cardiac malformation
gastroschisis after the use of a prostaglandin
synthesis inhibitor in early pregnancy. The
absolute risk of cardiovascular malformation
was increased from less than 1% up to
approximately 1.5%. The risk is believed to
increase with dose and duration of therapy. In
animals the administration of a prostaglandin
synthesis inhibitor has been shown to result in
increased pre- and post-implantation losses and
embryo/foetal lethality. In addition, increased
incidences of various malformations, including
cardiovascular, has also been reported in animals
given a prostaglandin synthesis inhibitor during
the organogenetic period. During the first and
second trimesters of pregnancy, ibuprofen
should not be given unless clearly necessary. If
ibuprofen is used by a woman attempting to
conceive or during the first and second trimester,
the dose should be kept as low and duration of
treatment as short as possible.
During the third trimester, all prostaglandin
synthesis inhibitors may expose the foetus to:
- Cardiopulmonary toxicity (with premature
closure of the ductus arteriosus and pulmonary
hypertension),
- Renal dysfunction, which may progress to
renal failure with oligohydramnios.
The mother and the neonate, at the end of
pregnancy, to:
- Prolongation of bleeding time,
- Inhibition of uterine contractions, resulting in
delayed or prolonged labour.
Consequently ibuprofen is contraindicated
during the last trimester of pregnancy.
Breast-feeding
Ibuprofen is excreted in breast milk, but with
therapeutic doses during short term treatment the
risk for influence on infant seems unlikely. If,
however, longer treatment is prescribed, early
weaning should be considered.
Fertility
The use of ibuprofen may impair fertility and is
not recommended in women attempting to
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conceive. In women who have difficulties
conceiving or who are undergoing investigation
of infertility, withdrawal of ibuprofen should be
considered.
4.7 Effects on ability to drive and use
machines
Following treatment with ibuprofen, the reaction
time of certain patients may be affected. This
should be taken into account where increased
vigilance is required. Undesirable effects such as
dizziness, drowsiness, fatigue and visual
disturbances are possible after taking NSAIDs.
If affected, patients should not drive or operate
machinery.
4.8 Undesirable Effects
The undesirable effects are mainly associated
with the pharmacological effect of ibuprofen on
prostaglandin synthesis. The most common
effects are dyspepsia and diarrhoea, which are
estimated to occur in about 10–30% of treated
patients.
Adverse events at least possibly related to
ibuprofen are displayed by MedDRA frequency
convention and system organ class database. The
following frequency groupings are used: Very
common (≥1/10), Common (≥1/100 to <1/10),
Uncommon (≥1/1000 to <1/100), Rare
(≥1/10000 to <1/1000), Very rare (<1/10,000)
and Not known (cannot be estimated from the
available data).
System organ class Frequency Adverse reaction
Infections and
infestations
Uncommon Rhinitis
Rare Aseptic meningitis
Blood and lymphatic
system disorders
Uncommon Leukopenia,
thrombocytopenia,
agranulocytosis,
neutropenia, aplastic
anaemia and haemolytic
anaemia
Immune system
disorders
Rare Anaphylactic reaction
Psychiatric disorders Uncommon Insomnia, anxiety
Rare Depression, confusional
state
Nervous system
disorders
Common Headache, dizziness
Uncommon Paraesthesia, somnolence
Rare Optic neuritis
Eye disorders Uncommon Visual impairment
Rare Toxic optic neuropathy
Ear and labyrinth
disorders
Uncommon Hearing impaired
Rare Tinnitus, vertigo
Respiratory, thoracic
and mediastinal
disorders
Uncommon Asthma, bronchospasm,
dyspnoea
Gastrointestinal
disorders
Common Dyspepsia, diarrhoea,
nausea, vomiting,
abdominal pain, flatulence,
constipation, melaena,
haematemesis,
gastrointestinal
haemorrhage
Uncommon Gastritis, duodenal ulcer,
gastric ulcer, mouth
ulceration, gastrointestinal
perforation
Very rare Pancreatitis
Not known Exacerbation of Colitis and
Crohn's disease
Hepatobiliary
disorders
Uncommon Hepatitis, jaundice,
abnormal hepatic function
Rare Liver injury
Very rare Hepatic failure
Skin and
subcutaneous tissue
disorders
Common Rash
Uncommon Urticaria, pruritus, purpura,
angioedema,
photosensitivity reaction
Very rare Severe forms of skin
reactions (e.g. erythema
multiforme, bullous
reactions, including
Stevens-Johnson
syndrome and toxic
epidermal necrolysis)
Not known Drug reaction with
eosinophilia and systemic
symptoms (DRESS
syndrome)
Renal and urinary
disorders
Uncommon Nephrotoxity in various
forms e.g. tubulointerstitial
nephritis, nephrotic
syndrome and renal failure
General disorders
and administration
site conditions
Common Fatigue
Rare Oedema
Cardiac disorders Not known Cardiac failure, myocardial
infarction (also see section
4.4)
Vascular disorders Not known Hypertension
Cardiac disorders and vascular disorders:
Oedema, hypertension and heart failure have
been reported in association with NSAID
treatment. Clinical studies suggest that use of
ibuprofen, particularly at a high dose (2400
mg/day) may be associated with a small
increased risk of arterial thrombotic events such
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as myocardial infarction or stroke (see section
4.4).
Gastrointestinal disorders: The most commonly
observed adverse events are gastrointestinal in
nature. Peptic ulcers, perforation or GI bleeding,
sometimes fatal, particularly in the elderly, may
occur (see section 4.4).
Immune system disorders: Hypersensitivity
reactions have been reported following treatment
with NSAIDs. These may consist of (a) non-
specific allergic reaction and anaphylaxis, (b)
respiratory tract reactivity comprising asthma,
aggravated asthma, bronchospasm or dyspnoea,
or (c) assorted skin disorders, including rashes
of various types, pruritus, urticaria, purpura,
angioedema and, very rarely, erythema
multiforme, bullous dermatoses (including
Stevens- Johnson syndrome and toxic epidermal
necrolysis).
Infections and infestations: Rhinitis and aseptic
meningitis (especially in patients with existing
autoimmune disorders, such as systemic lupus
erythematosus and mixed connective tissue
disease) with symptoms of stiff neck, headache,
nausea, vomiting, fever or disorientation (see
section 4.4).
Exacerbation of infection-related inflammations
(e.g. development of necrotising fasciitis)
coinciding with the use of NSAIDs has been
described. If signs of an infection occur or get
worse during use of Ibuprofen the patient is
therefore recommended to go to a doctor without
delay.
Skin and subcutaneous tissue disorders: In
exceptional cases, severe skin infections and
soft-tissue complications may occur during a
varicella infection (see also "Infections and
infestations").
Ibuprofen can cause prolongation of bleeding
time through reversible inhibition of platelet
aggregation.
Deterioration of ulcerative colitis and Crohn's
disease have been reported in connection with
NSAID treatment.
Reporting of suspected adverse reactions
Reporting suspected adverse reactions after
authorisation of the medicinal product is
important. It allows continued monitoring of the
benefit/risk balance of the medicinal product.
4.9 Overdose
Toxicity
Risk of symptoms at doses >80–100 mg/kg. At
doses >200 mg/kg there is a risk of severe
symptoms, though with considerable variations
between individuals. A dose of 560 mg/kg in a
child aged 15 months gave severe intoxication,
3.2 g in a 6-year-old mild to moderate
intoxication, 2.8–4 g in a 1½-year-old and 6 g in
a 6-year-old severe intoxication even after
gastric lavage, 8 g in an adult moderate
intoxication and >20 g in an adult very severe
intoxication. 8 g administered to a 16-year-old
affected the kidney and 12 g in combination
with alcohol administered to a teenager resulted
in acute tubular necrosis.
Symptoms
Most patients who have ingested significant
amounts of ibuprofen will manifest symptoms
within 4 to 6 hours.
The predominant symptoms of overdose are
ones from the gastrointestinal tract, e.g. nausea,
abdominal pain and vomiting (possibly blood-
streaked). Central nervous system effects
include headache, tinnitus, confusion and
nystagmus. At high doses loss of consciousness
and convulsions (mainly in children) may occur.
Cardiovascular toxicity, including bradycardia,
tachycardia and hypotension have been reported.
Hypernatraemia, kidney effects and haematuria
may occur. In serious poisoning metabolic
acidosis may occur. In significant overdose,
renal failure and liver damage are possible.
Hypothermia and ARDS have occasionally been
reported.
Treatment
Management should be symptomatic and
supportive as required. Within one hour of
ingestion of a potentially toxic amount, activated
charcoal should be considered. Alternatively, in
adults, gastric lavage should be considered
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within one hour of ingestion of a potentially life-
threatening overdose.
In the event of gastrointestinal problems,
administer antacids. In the event of hypotension,
intravenous fluid and, if required, inotropic
support. Ensure adequate diuresis. Correct acid-
base and electrolyte disorders.
Patients should be observed for at least four
hours after ingestion of potentially toxic
amounts.
5. PHARMACOLOGICAL PROPERTIES
5.1 Pharmacodynamic properties
Pharmacotherapeutic group: Anti-inflammatory
and antirheumatic products, nonsteroids;
propionic acid derivatives.
Mechanism of action
Ibuprofen belongs to the group of non-steroidal
anti-inflammatory drugs (NSAIDs). It contains
the propionic acid derivative p-isobutyl-
hydrothropic acid. Ibuprofen has anti-
inflammatory, analgesic and antipyretic effects.
The anti-phlogistic effect is comparable with
that of acetylsalicylic acid and indometacin. The
pharmacological effect of ibuprofen is probably
associated with its ability to inhibit
prostaglandin synthesis. Ibuprofen prolongs
bleeding time through reversible inhibition of
platelet aggregation.
Clinical efficacy and safety
Experimental data suggest that ibuprofen may
competitively inhibit the effect of low dose
acetylsalicylic acid on platelet aggregation when
they are dosed concomitantly. Some
pharmacodynamic studies show that when single
doses of ibuprofen 400 mg were taken within 8 h
before or within 30 min after immediate release
acetylsalicylic acid dosing (81 mg), a decreased
effect of acetylsalicylic acid on the formation of
thromboxane or platelet aggregation occurred.
Although there are uncertainties regarding
extrapolation of these data to the clinical
situation, the possibility that regular, long-term
use of ibuprofen may reduce the
cardioprotective effect of low-dose
acetylsalicylic acid cannot be excluded. No
clinically relevant effect is considered to be
likely for occasional ibuprofen use (see section
4.5).
Ibuprofen inhibits renal prostaglandin synthesis.
In patients with normal renal function this effect
is of no particular significance. In patients with
chronic renal insufficiency, decompensated heart
or liver insufficiency as well as conditions
involving changes in plasma volume, the
inhibited prostaglandin synthesis can lead to
acute renal insufficiency, fluid retention and
heart failure (see section 4.3).
5.2 Pharmacokinetic properties
Absorption
Ibuprofen is rapidly absorbed from the
gastrointestinal tract with a bioavailability of 80-
90%. Peak serum concentrations occur one to
two hours after administration. If administered
with food, peak serum concentrations are lower
and achieved more slowly than when taken on
an empty stomach Food does not affect
markedly total bioavailability.
Distribution
Ibuprofen is extensively bound to plasma
proteins (99%). Ibuprofen has a small volume of
distribution being about 0.12-0.2 L/kg in adults.
Biotransformation
Ibuprofen is rapidly metabolized in the liver
through cytochrome P450, preferentially
CYP2C9, to two primary inactive metabolites,
2-hydroxyibuprofen and 3-carboxyibuprofen.
Following oral ingestion of the drug, slightly
less than 90% of an oral dose of ibuprofen can
be accounted for in the urine as oxidative
metabolites and their glucuronic conjugates.
Very little ibuprofen is excreted unchanged in
the urine.
Elimination
Excretion by the kidney is both rapid and
complete. The elimination half-life is
approximately 2 hours. The excretion of
ibuprofen is virtually complete 24 hours after the
last dose.
Special populations
Elderly
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Given that no renal impairment exists, there are
only small, clinically insignificant differences in
the pharmacokinetic profile and urinary
excretion between young and elderly.
Children
The systemic exposure of ibuprofen following
weight adjusted therapeutic dosage (5mg/kg to
10 mg/kg bodyweight) in children aged 1 year or
over, appears similar to that in adults.
Children 3 months to 2.5 years appeared to have
a higher volume of distribution (L/kg) and
clearance (L/kg/h) of ibuprofen than did children
>2.5 to 12 years of age.
Renal impairment
For patients with mild renal impairment
increased unbound (S)-ibuprofen, higher AUC
values for (S)-ibuprofen and increased
enantiomeric AUC (S/R) ratios as compared
with healthy controls have been reported.
In end-stage renal disease patients receiving
dialysis the mean free fraction of ibuprofen was
about 3% compared with about 1% in healthy
volunteers. Severe impairment of renal function
may result in accumulation of ibuprofen
metabolites. The significance of this effect is
unknown. The metabolites can be removed by
haemodialysis (see sections 4.2, 4.3 and 4.4).
Hepatic impairment
Alcoholic liver disease with mild to moderate
hepatic impairment did not result in substantially
altered pharmacokinetic parameters.
In cirrhotic patients with moderate hepatic
impairment (Child Pugh's score 6-10) treated
with racemic ibuprofen an average 2-fold
prolongation of the half-life was observed and
the enantiomeric AUC ratio (S/R) was
significantly lower compared to healthy controls
suggesting an impairment of metabolic inversion
of (R)-ibuprofen to the active (S)-enantiomer
(see sections 4.2, 4.3 and 4.4).
5.3 Preclinical safety data
There are no preclinical data of relevance for the
safety assessment, apart from what has already
been taken into account in this summary of
product characteristics.
6. PHARMACEUTICAL PARTICULARS
6.1 List of excipients
Tablet Core
Lactose monohydrate
Maize starch
Croscarmellose sodium
Colloidal anhydrous Silica
Microcrystalline cellulose
Magnesium stearate
Tablet Coating
Hypromellose
Triacetin
Titanium dioxide (E171)
Erythrosine aluminum lake (E127)
6.2 Incompatibilities
Not applicable.
6.3 Shelf life
5 years.
6.4 Special precautions for storage
This medicinal product does not require any
special storage conditions.
6.5 Nature and contents of container
PVC - Aluminium blisters or PVC/PVdC –
Aluminium Blisters
Pack sizes: 10, 12, 14, 20, 21, 24, 28, 30, 40, 42,
48, 50, 60, 84, 100 or 500 film-coated tablets in
carton.
Not all pack sizes may be marketed.
6.6 Special precautions for disposal and other
handling
No special requirements.
7. MANUFACTURER:
Manufactured in India by:
TAJ PHARMACEUTICALS LTD.
Mumbai, India
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At: 615, GIDC, Kerala, Bavla, Dist. Ahmedabad
438225, Gujarat, INDIA
This leaflet was last revised in October 2019