Grant Euroskin UV cancer prevention
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This document summarizes epidemiological evidence that vitamin D, produced by exposure to UVB radiation, helps prevent cancer and other diseases. Ecological studies have found inverse correlations between UVB exposure and cancer rates. Cohort studies have linked higher vitamin D levels to reduced cancer risk. A randomized controlled trial found vitamin D and calcium supplementation reduced total cancer incidence. Studies also suggest vitamin D may help prevent respiratory infections, autoimmune diseases, and metabolic diseases. Maintaining vitamin D levels through moderate sun exposure or supplementation could significantly reduce disease rates and economic burdens.
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2. Specific prevention strategies for common cancers like breast, prostate, lung and colorectal cancer.
3. Recommendations for cancer screening and websites with guidance on screening for early detection.
Vitamin D: Going beyond cell differentiation as a theaputic modality for Acut...
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Chapter 26 chemoprevention of cancer
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Greater awareness of risk factors and early diagnosis can cut cancer toll
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cancer in brief discription,variation.pptx
This document discusses epidemiology and prevention of cancer. It defines cancer and identifies the major categories. Globally, cancer burden has risen significantly with lung, breast, and colorectal cancers among the most common. Key risk factors include tobacco, alcohol, diet, viruses, and genetic factors. Prevention strategies focus on primary prevention through reducing environmental risks, immunization, and health education as well as secondary prevention through early detection programs and cancer registration to facilitate treatment.
jnci.oxfordjournals.org JNCI Articles 1DOI 10.1093jn.docx
jnci.oxfordjournals.org JNCI | Articles 1
DOI: 10.1093/jnci/djq072 © The Author 2010. Published by Oxford University Press. All rights reserved.
For Permissions, please e-mail: [email protected]
In 1990, the World Health Organization (1) recommended people
to eat at least five portions (about 400 g) of fruits and vegetables a
day to prevent cancer and other chronic diseases. However, despite
considerable research in this direction, there is no conclusive
evidence that high intake of fruits and vegetables reduces the risk
of cancer development. Most available studies have concentrated
on one or a few types of cancer, which complicates an overall
assessment of the contribution of fruit and vegetable intakes in
cancer risk. In a systematic review, published in 1997, the World
Cancer Research Fund (2) claimed to have found convincing evi-
dence for a protective effect of high intake of fruits and vegetables
against a number of respiratory and digestive cancers. Most of this
Article
Fruit and Vegetable intake and Overall cancer risk in the
european Prospective investigation into cancer and Nutrition
(ePic)
Paolo Boffetta, Elisabeth Couto, Janine Wichmann, Pietro Ferrari, Dimitrios Trichopoulos, H. Bas Bueno-de-Mesquita,
Fränzel J. B. van Duijnhoven, Frederike L. Büchner, Tim Key, Heiner Boeing, Ute Nöthlings, Jakob Linseisen, Carlos A. Gonzalez,
Kim Overvad, Michael R. S. Nielsen, Anne Tjønneland, Anja Olsen, Françoise Clavel-Chapelon, Marie-Christine Boutron-Ruault,
Sophie Morois, Pagona Lagiou, Androniki Naska, Vassiliki Benetou, Rudolf Kaaks, Sabine Rohrmann, Salvatore Panico, Sabina Sieri,
Paolo Vineis, Domenico Palli, Carla H. van Gils, Petra H. Peeters, Eiliv Lund, Magritt Brustad, Dagrun Engeset, José María Huerta,
Laudina Rodríguez, Maria-José Sánchez, Miren Dorronsoro, Aurelio Barricarte, Göran Hallmans, Ingegerd Johansson, Jonas Manjer,
Emily Sonestedt, Naomi E. Allen, Sheila Bingham, Kay-Tee Khaw, Nadia Slimani, Mazda Jenab, Traci Mouw, Teresa Norat,
Elio Riboli, Antonia Trichopoulou
Manuscript received April 23, 2009; revised February 17, 2010; accepted February 18, 2010.
Correspondence to: Paolo Boffetta, MD, MPH, The Tisch Cancer Institute, Mount Sinai School of Medicine, One Gustave L. Levy Place, New York,
NY 10029 (e-mail: [email protected]).
Background It is widely believed that cancer can be prevented by high intake of fruits and vegetables. However, inconsistent
results from many studies have not been able to conclusively establish an inverse association between fruit and
vegetable intake and overall cancer risk.
Methods We conducted a prospective analysis of the European Prospective Investigation into Cancer and Nutrition (EPIC)
cohort to assess relationships between intake of total fruits, total vegetables, and total fruits and vegetables
combined and cancer risk during 1992–2000. Detailed information on the dietary habit and lifestyle variables of
the cohort was obtained. Cancer incide.
11 271.docx
Genetics plays a key role in colorectal cancer development and certain genes can increase hereditary risk. The patient's symptoms of crampy left side pain and constipation are consistent with diverticulitis, which obesity and inactivity increase risk of. The body responds through autophagy, a process where damaged cells are cleaned to prevent cancer progression, stimulated by immune response genes. Key genes like APC and MUTYH involved in colorectal cancer regulate cell signaling and DNA repair. Other risk factors like smoking, alcohol, and diet can also influence cancer development.
Lyman managementofchemotherapy-induced
Gary H Lyman discusses chemotherapy-induced neutropenia and the use of colony-stimulating factors to manage it. Chemotherapy often causes neutropenia, which can lead to febrile neutropenia and complications that reduce chemotherapy dose intensity. Colony-stimulating factors like G-CSF have been shown in clinical trials to reduce the risk of febrile neutropenia and allow for full dose chemotherapy. Clinical practice guidelines from organizations like EORTC and ASCO recommend using colony-stimulating factors for patients with a high risk (over 20%) of febrile neutropenia or when dose-dense chemotherapy is associated with improved survival.
Pediatric and young_adult_patients_and_oncofertility
This document discusses fertility preservation options for pediatric and young adult cancer patients. It finds that cancer treatments can impair development and cause infertility through damage to the ovaries, uterus, testes or hypothalamic-pituitary axis. Pelvic radiation over 2 Gy, alkylating chemotherapy, and hematopoietic stem cell transplantation carry higher risks. The standard options for fertility preservation are embryo and sperm cryopreservation, though oocyte and ovarian tissue cryopreservation are gaining acceptance. No options exist yet for prepubertal boys, though experimental testicular tissue cryopreservation is available at some centers. All at-risk patients should discuss potential infertility with oncologists before starting treatment.
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Vitamin D: Going beyond cell differentiation as a theaputic modality for Acut...
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