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Eye Emergencies
Dane Horsfall
Overview
 Eye Anatomy/Terminology
 History/Examination
 Red Eye
 Acute visual loss
 Eye Trauma
 Practical
Anatomy
Anatomy
Terminology
 Keratitis = inflammation of
cornea
 Blepharitis = inflammation of
the eyelid
 Iritis = inflammation of Iris
 Uveitis = inflam of uvea,
(middle layer-iris, ciliary body
and choroid)
 Anterior uveitis (most
common) – inflam. iris and
ciliary body aka “Iritis”
 Intermediate uveitis –
inflam. ciliary body
 Posterior uveitis – inflam.
choroid
 Diffuse uveitis - all
History/Examination
 Glasses? Contact Lenses?
 Previous eye
conditions/trauma/surgery/med
s
 Visual Acuity – iphone app
 Snellen chart x/y
 X is distance from chart (ie 6
metres)
 Y is smallest font size read
 Eg Normal 6/6, just top line
6/60
 Vision less than 6/60 count
no. of fingers/hand
movements/light perception
 Pin hole corrects refractory
error to 6/9 or better
 Practical - Measure each others
Examination
 Visual Fields
 Evert eyelids-local
anaesthetic (Amethocaine)
aids thorough eye exam
 Eye movements “H” CN III,
IV, VI palsies, fatigability
(myasthenia)
Examination
 Pan-Ophthalmoscopy: dark, dioptric to zero, pt
focus on corner of room
 Pupils
 Reflex
 Symmetry
 Cornea
 Lens
 Humour
 Retina-Fundoscopy-dilate pupil-Tropicamide
 Can use cobalt blue light with fluorescein
Examination
 Slit Lamp-where is it?
 Lateral canthus at black line
on frame
 Pt to look at examiners R ear
when examining R eye
 Joystick to focus
 Cobalt blue light for
fluorescein-NOT green light
filter. But Fluorescein(yellow)
dye appears green under blue
light
Painful Red Eye
 Case:
 65yo F, 1/52 increasing
R unilateral eye pain
assoc n/v, Dx as
migraine
 o/e
 visual acuity reduced
 hazy cornea
 fixed mid-dilated pupil
 hard eyeball
 ?Dx
Acute Angle Closure
Glaucoma
 Females in 60-70s, esp. Asians/Eskimos, +ve FHx
 defined as
 > 2 of
 ocular pain,
 nausea/vomiting,
 intermittent blurred vision with halos
 and at least 3 of:
 conjunctiva injection
 corneal epithelial oedema = hazy
 mid-dilated non-reactive pupil
 shallower chamber in the presence of occlusion.
 Raised IOP ? Normal
 >21 mmHg can be >60 mmHg
Types
 90% Open Angle
 Chronic onset,
painless
 Insidious eye
damage
 10% Closed angle
 Acute onset
 sudden eye pain,
redness, n/v
 From sudden spike
in IOP
 Eye emergency
Acute Angle Closure
Glaucoma
 Aqueous humor
 produced by ciliary body
(posterior chamber)
 passes thu pupil into ant
chamber drained via trabecular
meshwork and canal of
Schlemm in the angle.
 Contact between the lens and the
iris blocks flow, pressure in
posterior chamber - iris bows
forward closing angle – reduce
drainage
 Precipitated by dilated pupil-
darkness, stress, medications
(anticholinergic, sympathomimetic)
Acute Angle Closure
Glaucoma
 Intra-ocular pressure
measurement: Normal 10-
20mmHg
 Goldman applanation
tonometer: attached to the slit
lamp
 Storz/Schiotz Tonometer
 Tono-Pen handheld electronic
contact tonometer ($3000)
Practical –Tonopen use
 https://www.youtube.com/watch?
v=Xx6FXjBXBLE
Acute Angle Closure
Glaucoma
 Mx Ophthal. referral
 Acetazolamide 500mg IV
 Topical beta-blocker
 Topical steroid
 Analgesics/Anti-emetics/Supine
 Once pressure-induced ischemic paralysis of the iris
resolves around 1 hour post initial Rx then:
 Pilocarpine: a miotic (constricts pupil) – opens angle,
should be administered every 5 mins for 30 mins
 Laser peripheral iridotomy performed 24-48 hours after
IOP is controlled is definitive treatment
Famous Eyes
 Who’s eyes are they?
Painful Red Eye
 Case:
 45yo F with unilateral
red, painful eye
 PHx Crohn’s Disease
 o/e blurred vision,
perilimbal injection,
 Slit lamp
 “floaters/debris in
anterior chamber”
 ?Dx
Acute Anterior Uveitis (Iritis)
 Unilateral, painful red eye, blurred
vision, photophobia, and tearing
 Peri-limbal injection, worse closer to
limbus: (conjunctivitis= worse further
from limbus)
 Visual acuity may be decreased
 Examine anterior chamber with Slit lamp
 Increase in protein content of
aqueous causes an effect known as
“flare”, looks “smokey”
 White or red blood cells may be
observed in the anterior chamber
 Severe cases - inflam. cells
accumulate as sediment in ant.
chamber = Hypopyon
Iritis
 Causes
 50% idiopathic
 Assoc
 CTD (ankylosing spondylitis, inflammatory bowel
disease, Reiter syndrome, psoriatic arthritis,
sarcoidosis)
 Infections: Herpes, syphilis, TB, toxoplasmosis,
histoplasmosis, CMV, Candida
 Trauma
 Mx Referral: steroids and cycloplegics,
antimicrobials.
Painful Red Eye
 Herpes simples –
dendritic ulcers Rx
topical Acyclovir
 Sore eye, reduced
aquity in Contact Lens
wearer
 Bacterial Ulcer
Acanthamoebal ulcer:
amoeba assoc contact
lens Mx urgent Ophthal
ref. ?
admit/antimicrobials
Painful Red Eye - Eyelid
 Chalazion - eyelid cyst inflam. of
blocked meibomian gland -usually
painless and larger. Rx warm
compresses/antis/usually resolve
can inject steroids/surgically
remove
 Stye – infection (staph) of the
sebaceous glands at base of the
eyelashes. Rx warm compress,
pull out eyelash, antis
 Blepharitis – inflam. eyelid can be
infective. Rx warm wet
compress/antis
 Herpes Zoster – vesicular rash,
can cause infection of all parts of
eye. Nasociliary branch
involvement predicts serious
complications: ocular inflam. and
corneal denervation. Mx Opthal
ref, Acyclovir
Painful Red Eye
 Conjunctivitis
 Viral - recent URTI,
clear, watery discharge
 Allergic –pruritus,
clear, watery discharge
 Bacterial – pus, swab,
staph/strep/
gonococcal/chlamydia,
 Rx Chlorsig
Red Eye
 Scleritis:
 Inflam sclera- localized, nodular, or
diffuse
 Vision may be impaired
 Sclera thick, discoloured
 Severe pain
 Assoc with CTD (esp RA) and
Vasculitis
 Mx Analgesia, Ophthal ref
steroids/immunosuppressant
 Pterygium :
 raised yellow, fleshy lesion at
limbus, may be inflamed
 Asymptomatic or redness,
swelling, itching, irritation, blurred
vision
 r/f UV, FHx, Male
 Mx lubricant, sunglasses, refer -
surgery
Famous Eyes
Who’s eyes are they?
Case
 60yo M Sudden, painless
loss of vision L eye,
previous
partial/intermittent loss of
vision over a few days
 PHx IHD, HT, DM
 L eye light perception
only, relative afferent
pupillary defect
 Fundus: pale,
arteries/veins narrowed
 ?Dx
Central Retinal Artery
Occlusion
 Embolism
 Most commonly cholesterol,
cardiac (assoc HT,DM) can be
calcific, bacterial, Giant cell
arteritis
 Amaurosis Fugax : transient
loss of vision lasting seconds
to minutes, can precede
 Mx Urgent ophthal referral
 Decrease intra-ocular pressure
 Acetazolamide/Anterior
chamber paracentesis
 Move clot
 Pulsed ocular compression
 Anticoagulate
 Intra-arterial fibrinolysis
Case – Same Pt, ?Dx
Central Retinal Vein Occlusion
 Sudden painless loss of vision
 R/F: age, HT, DM,
prothrombotic disorders
 Types: Non-ischaemic(less
severe) and Ischaemic
 Signs: Decreased visual
acuity, Relative Afferent
pupillary Defect, abnormal red
reflex
 Fundus haemorrhage (“Stormy
sunset”)
 Mx Ophthal referral
 Anticoag, aspirin
 Surgery incl. Laser
photocoagulation
Case
 32 yo F
 Gradual onset
blurred central
vision
Optic Neuritis
 Vision loss (esp. colour) over hours-days,
pain with eye movements, central scotoma
 Usually unilateral, F 18-45yo may be 1st
presentation of demyelinating disease-MS
 Swollen optic disc
 May have other neurology
 Mx Ophthal referral, IV
IV steroids
Case
 60 yo F with R temporal headache and
blurred vision from R eye
Giant Cell Arteritis
 AKA Arteritic Ischaemic Optic
Neuropathy
 Females, 60’s
 Profound unilateral visual loss
 Check for
 Jaw claudication
 Headache
 Scalp tenderness
 Polymyalgia Rheumatica in 50%
 Fundus: disc oedema
 ESR >60mm/hr
 Rx Ophthal referral,
Prednisolone
Case
 75 yo M post Bungee Jumping
 Falshers/floaters L eye, then curtain-like
visual loss
Retinal Detachment
 Result of retinal hole with
seepage of fluid between
retina and choroid
 R/F age, trauma
 Signs
 flashing lights, floaters
 Vision loss may be filmy,
cloudy, irregular, or curtainlike
 Visual field defects
 Mx Ophthal ref., Repair
 Laser therapy
 Cryotherapy
 Intraocular gas (ie, pneumatic
retinopexy) tamponades retina
 Intraocular repair
Famous Eyes
 Who’s eyes are they?
Eye Trauma
Corneal injuries
 Corneal Abrasion
 Sensation of foreign body, light
sensitivity, tearing
 Local drops (Amethocaine 0.5%)
 Fluorescein with blue light
 Rx Chlorsig (drops/ointment), ?LA
drops – studies showing delayed
healing –case reports, pts abusing
the drops
 Corneal Flash burns
 Arc welding/UV lamp
 Red, painful, tearing
 LA, Fluorescein
 Rx Chlorsig
Corneal foreign body
 Dirt/glass/metal (rust ring)
 Velocity of impact
 Signs of penetration
 Removal
 Local
 25G needle, lateral
approach using slit lamp
 Dental burr for rust ring
(adherent rust ring may
loosen with Chlorsig/patch
for 24hrs as the cornea
heals, may recall pt)
Chemical burns
 Acids: toilet/pool cleaner,
battery fluid
 Alkalis (more harmful): lime,
mortar/plaster, drain cleaner,
oven cleaner, ammonia
 Immediate Mx: LA copious
irrigation with fluid-bag of
N/Saline + Morgan Lens until
pH 7.5, test aquity
 Degree of vascular blanching
(esp at limbus) proportional to
severity of burn
 Chlorsig, Ophthal. referral
Blunt Trauma - Haemorrhage
 Subconjunctival Hemorrhage
 usually benign, if spont. Check
BP/Coags
 If cant see post border ?Orbital #
 Hyphaema: blood in anterior
chamber
 If >1/3 = damage to drainage
angle, risk glaucoma
 Mx shield/patch/semi-
recumbent/rest +/-
sedation/admission
no NSAIDs, Ophthal. Ref.
 Recurrent bleeding in 10% esp
with early mobilization
 Hemorrhage vitreous or retina, can be
accompanied by a retinal detachment.
 Iris damage can result in poor pupil
reactivity = Traumatic mydriasis.
Misleading Neuro signs
 Lens can be damaged or dislocated
and a cataract may develop
Blunt trauma - Orbital blowout
fracture
 Usually inferior wall since weakest
 Signs:
 Diplopia/Ophthalmoplegia from
muscle entrapment. Tethering of
inferior rectus prohibits the upward
movement of the globe.
 Proptosis from swelling or
retrobulbar hemorrhage and later
Enophthalmos from loss of volume
 Infraorbital nerve entrapment- numb
cheek/upper teeth
 Epistaxis
 30% incidence of a ruptured globe in
conjunction with orbital fractures.
(Wilkins RB, Havins WE. Current treatment of blow-out
fractures. Ophthalmology. May 1982;89(5):464-6)
Blowout Fracture
 Mx
 Repair: Indicated if
significant diplopia or
cosmetically unacceptable
enophthalmos. Most
surgeons will wait 10 to 14
days following the trauma to
allow for resolution of the
associated edema and
hemorrhage
 Medical : if no
diplopia/enophthalmos
 o antis/no nose blowing/?
steroids
Ruptured Globe
 May be from blunt or
penetrating trauma
 Occurs at thinnest part:
 Limbus (Visible with slit lamp)
 Insertions of the extra-ocular
muscles (reduced eye
movements, loss red reflex
from vitreous haemorrhage)
 Around the optic nerve
 Signs:
 Pupil : peaked, teardrop-
shaped, or otherwise irregular
 Seidel’s Sign
 Enophthalmos (recession of
the globe within the orbit)
 Exophthalmos from retrobulbar
hemorrhage
Ruptured Globe
 Ix: CT most sensitive
 Mx : Anti-emetics/analgesics/prophylactic
antibiotics/tetanus/fast
 Urgent Ophthal. referral always requires surgical
intervention.
 ? Suxamethonium in open globe injury
controversial, weigh up risk to airway Mx and
theoretical risk of ocular extrusion and ask opthal.
Penetrating Eye Trauma
 Easily missed since may seal over and abnormal signs may
be subtle
 High risk with high velocity eg metal striking metal and glass
 Leave bodies insitu until surgery
 Signs:
 Distorted pupil
 Cataract
 Prolapsed black uveal tissue on the ocular surface
 Vitreous hemorrhage.
 Seidel’s Sign
 Shallow/flat anterior chamber or bubbles in anterior chamber
 Mx as for ruptured globe
Lid Lacerations
 Require Ophthal. ref. if:
 Torn lid margins - must
be closed accurately
 Lacrimal ducts damage
 Any suspicion of a
foreign body or
penetrating eyelid injury
 Mx refer/Tetanus/iv
antis/antiemetics/shield
eye
Retrobulbar haematoma
 R eye relative afferent
pupillary defect
 Reduced Extraocular
movements
 Tonopen - pressure
>40 mmHg.
Orbital compartment
syndrome
 Dx
 pain, decreased vision, diplopia, limited
extraocular movements, proptosis, afferent
pupillary defect
 Raised IOC > 40
 CT
 Irreversible Ischaemic retinal damage > 2hrs
 Lateral Canthotomy – inferior crus
 https://www.youtube.com/watch?
v=MhGQ1ikN93M
Famous Eyes
 Who’s eye’s are they?
Golden Rules
 Always check visual acuity
 Always attempt to open eye early and
examine pupil/acuity etc in trauma
 Beware Dx unilateral conjunctivitis until more
serious disease is excluded
 Don’t start Steroid drops without
ophthalmology r/v
References
 Globe Rupture, J Robson, Feb 16 2007, www.emedicine.com
 Handbook of ocular disease, 2000 - 2001 Jobson Publishing,
www.revoptom.com/handbook/hbhome.htm
 P T Khaw et al, Clinical review “ABC of Eyes- Injury to the eye” BMJ 2004;328:36-
38 (3 January)
 Cameron et al, Textbook of Adult Emergency Medicine, Second Ed, Churchill
Livingston, 2004
 Eye Emergency Manual, NSW Ophthalmology Service, 2007
 Retinal Detachment, G Larkin , Apr 7, 2008 www.emedicine.com
 Acanthamoeba, N Crum-Cianflone, Jun 30 2008, www.emedicine.com
 Facial Trauma, Orbital Floor Fractures (Blowout), A Cohen, Dec 18 2006,
www.emedicine.com
 Glaucoma, Acute Angle-Closure, A Darkeh, Oct 3 2007, www.emedicine.com
 Scleritis, T Gaeta, Apr 14 2008 www.emedicine.com
 Wilkins RB, Havins WE. Current treatment of blow-out
fractures. Ophthalmology. May 1982;89(5):464-6
 Life in the fast lane

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Eye emergencies emc

  • 2. Overview  Eye Anatomy/Terminology  History/Examination  Red Eye  Acute visual loss  Eye Trauma  Practical
  • 5. Terminology  Keratitis = inflammation of cornea  Blepharitis = inflammation of the eyelid  Iritis = inflammation of Iris  Uveitis = inflam of uvea, (middle layer-iris, ciliary body and choroid)  Anterior uveitis (most common) – inflam. iris and ciliary body aka “Iritis”  Intermediate uveitis – inflam. ciliary body  Posterior uveitis – inflam. choroid  Diffuse uveitis - all
  • 6. History/Examination  Glasses? Contact Lenses?  Previous eye conditions/trauma/surgery/med s  Visual Acuity – iphone app  Snellen chart x/y  X is distance from chart (ie 6 metres)  Y is smallest font size read  Eg Normal 6/6, just top line 6/60  Vision less than 6/60 count no. of fingers/hand movements/light perception  Pin hole corrects refractory error to 6/9 or better  Practical - Measure each others
  • 7. Examination  Visual Fields  Evert eyelids-local anaesthetic (Amethocaine) aids thorough eye exam  Eye movements “H” CN III, IV, VI palsies, fatigability (myasthenia)
  • 8. Examination  Pan-Ophthalmoscopy: dark, dioptric to zero, pt focus on corner of room  Pupils  Reflex  Symmetry  Cornea  Lens  Humour  Retina-Fundoscopy-dilate pupil-Tropicamide  Can use cobalt blue light with fluorescein
  • 9. Examination  Slit Lamp-where is it?  Lateral canthus at black line on frame  Pt to look at examiners R ear when examining R eye  Joystick to focus  Cobalt blue light for fluorescein-NOT green light filter. But Fluorescein(yellow) dye appears green under blue light
  • 10. Painful Red Eye  Case:  65yo F, 1/52 increasing R unilateral eye pain assoc n/v, Dx as migraine  o/e  visual acuity reduced  hazy cornea  fixed mid-dilated pupil  hard eyeball  ?Dx
  • 11. Acute Angle Closure Glaucoma  Females in 60-70s, esp. Asians/Eskimos, +ve FHx  defined as  > 2 of  ocular pain,  nausea/vomiting,  intermittent blurred vision with halos  and at least 3 of:  conjunctiva injection  corneal epithelial oedema = hazy  mid-dilated non-reactive pupil  shallower chamber in the presence of occlusion.  Raised IOP ? Normal  >21 mmHg can be >60 mmHg
  • 12. Types  90% Open Angle  Chronic onset, painless  Insidious eye damage  10% Closed angle  Acute onset  sudden eye pain, redness, n/v  From sudden spike in IOP  Eye emergency
  • 13. Acute Angle Closure Glaucoma  Aqueous humor  produced by ciliary body (posterior chamber)  passes thu pupil into ant chamber drained via trabecular meshwork and canal of Schlemm in the angle.  Contact between the lens and the iris blocks flow, pressure in posterior chamber - iris bows forward closing angle – reduce drainage  Precipitated by dilated pupil- darkness, stress, medications (anticholinergic, sympathomimetic)
  • 14. Acute Angle Closure Glaucoma  Intra-ocular pressure measurement: Normal 10- 20mmHg  Goldman applanation tonometer: attached to the slit lamp  Storz/Schiotz Tonometer  Tono-Pen handheld electronic contact tonometer ($3000)
  • 15. Practical –Tonopen use  https://www.youtube.com/watch? v=Xx6FXjBXBLE
  • 16. Acute Angle Closure Glaucoma  Mx Ophthal. referral  Acetazolamide 500mg IV  Topical beta-blocker  Topical steroid  Analgesics/Anti-emetics/Supine  Once pressure-induced ischemic paralysis of the iris resolves around 1 hour post initial Rx then:  Pilocarpine: a miotic (constricts pupil) – opens angle, should be administered every 5 mins for 30 mins  Laser peripheral iridotomy performed 24-48 hours after IOP is controlled is definitive treatment
  • 17. Famous Eyes  Who’s eyes are they?
  • 18. Painful Red Eye  Case:  45yo F with unilateral red, painful eye  PHx Crohn’s Disease  o/e blurred vision, perilimbal injection,  Slit lamp  “floaters/debris in anterior chamber”  ?Dx
  • 19. Acute Anterior Uveitis (Iritis)  Unilateral, painful red eye, blurred vision, photophobia, and tearing  Peri-limbal injection, worse closer to limbus: (conjunctivitis= worse further from limbus)  Visual acuity may be decreased  Examine anterior chamber with Slit lamp  Increase in protein content of aqueous causes an effect known as “flare”, looks “smokey”  White or red blood cells may be observed in the anterior chamber  Severe cases - inflam. cells accumulate as sediment in ant. chamber = Hypopyon
  • 20. Iritis  Causes  50% idiopathic  Assoc  CTD (ankylosing spondylitis, inflammatory bowel disease, Reiter syndrome, psoriatic arthritis, sarcoidosis)  Infections: Herpes, syphilis, TB, toxoplasmosis, histoplasmosis, CMV, Candida  Trauma  Mx Referral: steroids and cycloplegics, antimicrobials.
  • 21. Painful Red Eye  Herpes simples – dendritic ulcers Rx topical Acyclovir  Sore eye, reduced aquity in Contact Lens wearer  Bacterial Ulcer Acanthamoebal ulcer: amoeba assoc contact lens Mx urgent Ophthal ref. ? admit/antimicrobials
  • 22. Painful Red Eye - Eyelid  Chalazion - eyelid cyst inflam. of blocked meibomian gland -usually painless and larger. Rx warm compresses/antis/usually resolve can inject steroids/surgically remove  Stye – infection (staph) of the sebaceous glands at base of the eyelashes. Rx warm compress, pull out eyelash, antis  Blepharitis – inflam. eyelid can be infective. Rx warm wet compress/antis  Herpes Zoster – vesicular rash, can cause infection of all parts of eye. Nasociliary branch involvement predicts serious complications: ocular inflam. and corneal denervation. Mx Opthal ref, Acyclovir
  • 23. Painful Red Eye  Conjunctivitis  Viral - recent URTI, clear, watery discharge  Allergic –pruritus, clear, watery discharge  Bacterial – pus, swab, staph/strep/ gonococcal/chlamydia,  Rx Chlorsig
  • 24. Red Eye  Scleritis:  Inflam sclera- localized, nodular, or diffuse  Vision may be impaired  Sclera thick, discoloured  Severe pain  Assoc with CTD (esp RA) and Vasculitis  Mx Analgesia, Ophthal ref steroids/immunosuppressant  Pterygium :  raised yellow, fleshy lesion at limbus, may be inflamed  Asymptomatic or redness, swelling, itching, irritation, blurred vision  r/f UV, FHx, Male  Mx lubricant, sunglasses, refer - surgery
  • 26. Case  60yo M Sudden, painless loss of vision L eye, previous partial/intermittent loss of vision over a few days  PHx IHD, HT, DM  L eye light perception only, relative afferent pupillary defect  Fundus: pale, arteries/veins narrowed  ?Dx
  • 27. Central Retinal Artery Occlusion  Embolism  Most commonly cholesterol, cardiac (assoc HT,DM) can be calcific, bacterial, Giant cell arteritis  Amaurosis Fugax : transient loss of vision lasting seconds to minutes, can precede  Mx Urgent ophthal referral  Decrease intra-ocular pressure  Acetazolamide/Anterior chamber paracentesis  Move clot  Pulsed ocular compression  Anticoagulate  Intra-arterial fibrinolysis
  • 28. Case – Same Pt, ?Dx
  • 29. Central Retinal Vein Occlusion  Sudden painless loss of vision  R/F: age, HT, DM, prothrombotic disorders  Types: Non-ischaemic(less severe) and Ischaemic  Signs: Decreased visual acuity, Relative Afferent pupillary Defect, abnormal red reflex  Fundus haemorrhage (“Stormy sunset”)  Mx Ophthal referral  Anticoag, aspirin  Surgery incl. Laser photocoagulation
  • 30. Case  32 yo F  Gradual onset blurred central vision
  • 31. Optic Neuritis  Vision loss (esp. colour) over hours-days, pain with eye movements, central scotoma  Usually unilateral, F 18-45yo may be 1st presentation of demyelinating disease-MS  Swollen optic disc  May have other neurology  Mx Ophthal referral, IV IV steroids
  • 32. Case  60 yo F with R temporal headache and blurred vision from R eye
  • 33. Giant Cell Arteritis  AKA Arteritic Ischaemic Optic Neuropathy  Females, 60’s  Profound unilateral visual loss  Check for  Jaw claudication  Headache  Scalp tenderness  Polymyalgia Rheumatica in 50%  Fundus: disc oedema  ESR >60mm/hr  Rx Ophthal referral, Prednisolone
  • 34. Case  75 yo M post Bungee Jumping  Falshers/floaters L eye, then curtain-like visual loss
  • 35. Retinal Detachment  Result of retinal hole with seepage of fluid between retina and choroid  R/F age, trauma  Signs  flashing lights, floaters  Vision loss may be filmy, cloudy, irregular, or curtainlike  Visual field defects  Mx Ophthal ref., Repair  Laser therapy  Cryotherapy  Intraocular gas (ie, pneumatic retinopexy) tamponades retina  Intraocular repair
  • 36. Famous Eyes  Who’s eyes are they?
  • 38. Corneal injuries  Corneal Abrasion  Sensation of foreign body, light sensitivity, tearing  Local drops (Amethocaine 0.5%)  Fluorescein with blue light  Rx Chlorsig (drops/ointment), ?LA drops – studies showing delayed healing –case reports, pts abusing the drops  Corneal Flash burns  Arc welding/UV lamp  Red, painful, tearing  LA, Fluorescein  Rx Chlorsig
  • 39. Corneal foreign body  Dirt/glass/metal (rust ring)  Velocity of impact  Signs of penetration  Removal  Local  25G needle, lateral approach using slit lamp  Dental burr for rust ring (adherent rust ring may loosen with Chlorsig/patch for 24hrs as the cornea heals, may recall pt)
  • 40. Chemical burns  Acids: toilet/pool cleaner, battery fluid  Alkalis (more harmful): lime, mortar/plaster, drain cleaner, oven cleaner, ammonia  Immediate Mx: LA copious irrigation with fluid-bag of N/Saline + Morgan Lens until pH 7.5, test aquity  Degree of vascular blanching (esp at limbus) proportional to severity of burn  Chlorsig, Ophthal. referral
  • 41. Blunt Trauma - Haemorrhage  Subconjunctival Hemorrhage  usually benign, if spont. Check BP/Coags  If cant see post border ?Orbital #  Hyphaema: blood in anterior chamber  If >1/3 = damage to drainage angle, risk glaucoma  Mx shield/patch/semi- recumbent/rest +/- sedation/admission no NSAIDs, Ophthal. Ref.  Recurrent bleeding in 10% esp with early mobilization  Hemorrhage vitreous or retina, can be accompanied by a retinal detachment.  Iris damage can result in poor pupil reactivity = Traumatic mydriasis. Misleading Neuro signs  Lens can be damaged or dislocated and a cataract may develop
  • 42. Blunt trauma - Orbital blowout fracture  Usually inferior wall since weakest  Signs:  Diplopia/Ophthalmoplegia from muscle entrapment. Tethering of inferior rectus prohibits the upward movement of the globe.  Proptosis from swelling or retrobulbar hemorrhage and later Enophthalmos from loss of volume  Infraorbital nerve entrapment- numb cheek/upper teeth  Epistaxis  30% incidence of a ruptured globe in conjunction with orbital fractures. (Wilkins RB, Havins WE. Current treatment of blow-out fractures. Ophthalmology. May 1982;89(5):464-6)
  • 43. Blowout Fracture  Mx  Repair: Indicated if significant diplopia or cosmetically unacceptable enophthalmos. Most surgeons will wait 10 to 14 days following the trauma to allow for resolution of the associated edema and hemorrhage  Medical : if no diplopia/enophthalmos  o antis/no nose blowing/? steroids
  • 44. Ruptured Globe  May be from blunt or penetrating trauma  Occurs at thinnest part:  Limbus (Visible with slit lamp)  Insertions of the extra-ocular muscles (reduced eye movements, loss red reflex from vitreous haemorrhage)  Around the optic nerve  Signs:  Pupil : peaked, teardrop- shaped, or otherwise irregular  Seidel’s Sign  Enophthalmos (recession of the globe within the orbit)  Exophthalmos from retrobulbar hemorrhage
  • 45.
  • 46. Ruptured Globe  Ix: CT most sensitive  Mx : Anti-emetics/analgesics/prophylactic antibiotics/tetanus/fast  Urgent Ophthal. referral always requires surgical intervention.  ? Suxamethonium in open globe injury controversial, weigh up risk to airway Mx and theoretical risk of ocular extrusion and ask opthal.
  • 47. Penetrating Eye Trauma  Easily missed since may seal over and abnormal signs may be subtle  High risk with high velocity eg metal striking metal and glass  Leave bodies insitu until surgery  Signs:  Distorted pupil  Cataract  Prolapsed black uveal tissue on the ocular surface  Vitreous hemorrhage.  Seidel’s Sign  Shallow/flat anterior chamber or bubbles in anterior chamber  Mx as for ruptured globe
  • 48. Lid Lacerations  Require Ophthal. ref. if:  Torn lid margins - must be closed accurately  Lacrimal ducts damage  Any suspicion of a foreign body or penetrating eyelid injury  Mx refer/Tetanus/iv antis/antiemetics/shield eye
  • 49. Retrobulbar haematoma  R eye relative afferent pupillary defect  Reduced Extraocular movements  Tonopen - pressure >40 mmHg.
  • 50. Orbital compartment syndrome  Dx  pain, decreased vision, diplopia, limited extraocular movements, proptosis, afferent pupillary defect  Raised IOC > 40  CT  Irreversible Ischaemic retinal damage > 2hrs  Lateral Canthotomy – inferior crus  https://www.youtube.com/watch? v=MhGQ1ikN93M
  • 51. Famous Eyes  Who’s eye’s are they?
  • 52. Golden Rules  Always check visual acuity  Always attempt to open eye early and examine pupil/acuity etc in trauma  Beware Dx unilateral conjunctivitis until more serious disease is excluded  Don’t start Steroid drops without ophthalmology r/v
  • 53. References  Globe Rupture, J Robson, Feb 16 2007, www.emedicine.com  Handbook of ocular disease, 2000 - 2001 Jobson Publishing, www.revoptom.com/handbook/hbhome.htm  P T Khaw et al, Clinical review “ABC of Eyes- Injury to the eye” BMJ 2004;328:36- 38 (3 January)  Cameron et al, Textbook of Adult Emergency Medicine, Second Ed, Churchill Livingston, 2004  Eye Emergency Manual, NSW Ophthalmology Service, 2007  Retinal Detachment, G Larkin , Apr 7, 2008 www.emedicine.com  Acanthamoeba, N Crum-Cianflone, Jun 30 2008, www.emedicine.com  Facial Trauma, Orbital Floor Fractures (Blowout), A Cohen, Dec 18 2006, www.emedicine.com  Glaucoma, Acute Angle-Closure, A Darkeh, Oct 3 2007, www.emedicine.com  Scleritis, T Gaeta, Apr 14 2008 www.emedicine.com  Wilkins RB, Havins WE. Current treatment of blow-out fractures. Ophthalmology. May 1982;89(5):464-6  Life in the fast lane

Editor's Notes

  1. Equipment: Morgan Lens Ophthalmoscope Fluorescein Eye drops-Amethocaine/Tropicamide Tonopen Ref process at Cabrini
  2. Conjuctiva-Bulbar, Palpebral
  3. palpebral conjunctiva lines the lids
  4. Normal ICP
  5. Bowie-L eye permanently dilated pupil from trauma as child
  6. RELATIVE AFFERENT PUPILLARY DEFECT:L light in R eye, L constricts, light swings to L eye, L dilates since reduced light transmission on L
  7. Presumed glaucoma
  8. Forrest Whitaker-congenital ptosis