Complex VSD closure with Pseudoaneurysm and PFO
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A middle-aged man presented with symptoms of shortness of breath and fatigue. An echocardiogram found an enlarged heart chambers and a complex ventricular septal defect with a pseudoaneurysm. Angiography revealed a 3+ shunt. A transcatheter device was used to close the VSD, with the device tucked just below the aortic valve to cover the pseudoaneurysm, resulting in minimal residual shunting.
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Duplex assessment of venous hemodynamics 3 oct 13th, 2011, morning 20mn2
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Ventricular septal defect DR NIKUNJ .R .SHRKHADA (MBBS,MS GEN SURG DNB CTS SR)
Ventricular septal defect DR NIKUNJ .R .SHRKHADA (MBBS,MS GEN SURG DNB CTS SR)
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Presentation on heart valve devices
This document discusses various percutaneous interventions for structural heart diseases including atrial septal defect (ASD), ventricular septal defect (VSD), patent ductus arteriosus (PDA), sinus of Valsalva aneurysm, and paravalvular leaks. It describes current device closure options like the Amplatzer occluder and the techniques used for percutaneous closure of different defects as well as the advantages over open heart surgery. Complications of device closure are also mentioned.
VSD devices
This document summarizes different devices used for closing ventricular septal defects (VSDs). It describes the common complications of VSD devices which are mostly minor, including embolization, arrhythmias, and conduction defects. Three types of Amplatzer devices are outlined - the muscular VSD device, asymmetric VSD occluder, and perimembranous VSD devices. Sizes and designs of each are provided. Results of post-myocardial infarction VSD closure show high residual leak rates. Finally, it briefly mentions some VSD devices manufactured in China including by Yatai and Lifetech, and introduces the novel NitOcclud VSD coil.
ASD ,VSD elkhatib
This document discusses atrial septal defects (ASDs), which are holes between the left and right atria of the heart that allow blood to pass from the left to the right side. The main types of ASDs are secundum, primum, and sinus venosus defects. If left unrepaired, an ASD can cause increased blood flow to the lungs, enlargement of the right atrium, and inadequate blood flow to the body. Surgical closure or device closure are treatment options to close the defect and restore normal blood flow patterns.
Indicaciones Intervencionismo En Cardiopatias Congenitas
The document provides recommendations for various interventional and surgical procedures for different types of congenital heart defects. It recommends when closure of atrial septal defects (ASDs), ventricular septal defects (VSDs), and patent ductus arteriosus (PDA) should be performed either percutaneously or surgically based on factors like hemodynamics, symptoms, and anatomy. It also provides indications for aortic valve repair/replacement and interventions for lesions affecting the left ventricular outflow tract and pulmonary valve. Recommendations are made based on the severity of obstruction/regurgitation, symptoms, and technical factors.
Vsd device closure
Based on the size of the defect, perimembranous VSDs between 4-18 mm in diameter would be suitable for closure with the Amplatzer VSD occluder. The device size would need to be selected based on the actual defect size as assessed by echocardiography. Adequate rims around the defect are required but specifics on rim measurements are not provided in this document. Other factors such as indications for closure and no contraindications to the percutaneous approach would also need to be evaluated for a particular patient.
ASD and VSD Closure
The document discusses non-surgical interventions for atrial septal defects (ASDs) and ventricular septal defects (VSDs). It describes the anatomy, causes, symptoms, and types of ASDs and VSDs. It provides indications for intervention such as large defect size, heart failure, or failure to thrive. The main non-surgical interventions discussed are percutaneous device closure of ASDs and VSDs, which are preferred to surgery when the anatomy is suitable. The future of interventions may include improved 3D imaging, contrast agents, and stents to repair more complex congenital heart defects.
ASD devices
This document summarizes various devices used to close atrial septal defects (ASDs), including their designs, sizes, advantages, and disadvantages. The most commonly used device is the Amplatzer Septal Occluder, which has a double disc design and is self-expanding. Other devices discussed include the Gore HELEX, Lifetech/Cera, Figulla, Cardioseal/Starflex, and newer bioabsorbable options like the Biotrek. Complication rates of ASD device closure are generally low, below 10%, with embolization and arrhythmias being the most common issues. Larger trials have shown the Amplatzer to be very effective and easy
Ventricular Septal Defects - A Review
Ventricular septal defects (VSDs) are congenital holes in the wall separating the left and right ventricles of the heart. Henri Roger first described VSDs in 1879. VSDs are classified based on their location, with the main types being membranous, muscular, inlet, and outlet. The size of the defect determines the severity, with smaller defects having little impact and larger defects causing significant left-to-right shunting of blood. Symptoms range from none with small defects to heart failure in infants with large defects. Treatment may involve monitoring for closure, surgery, or device closure depending on the size and impact of the defect.
Ventricular septal defect
Ventricular septal defect is an abnormal communication through the septum separating the right and left ventricles. It can be single or multiple, located in different areas of the septum. Most are asymptomatic at birth but may later cause failure to thrive and heart failure as pulmonary resistance decreases. Diagnosis is via echocardiogram. Small VSDs often close spontaneously but large defects require closure to prevent long term effects like pulmonary hypertension. Surgical patch closure is standard treatment for large unclosed VSDs.
VENTRICULAR SEPTAL DEFECT
A 2-year-old child presented with recurrent chest infections, difficulty breathing, and failure to thrive. On examination, precordial bulging and a grade 3/6 pansystolic murmur were present. Investigation revealed pallor and echocardiography showed a ventricular septal defect.
Ventricular septal defects are one of the most common congenital heart defects. They involve an abnormal opening in the muscular or membranous septum separating the left and right ventricles. Most small defects close spontaneously, but larger defects require surgery to prevent pulmonary hypertension.
Ventricular septal defects
Ventricular septal defects (VSDs) are the most common congenital heart defect. They can be classified based on their surgical features, morphology, size, location, and other characteristics. Presentation varies depending on the size of the defect and pulmonary vascular resistance. Small, restrictive defects often have no symptoms, while large defects can cause heart failure in infants due to volume overload. Over time, pulmonary pressures may rise in untreated patients. Complications include aortic valve prolapse, right ventricular outflow tract obstruction, infective endocarditis, and arrhythmias.
Ventricular septal defect (vsd)
Ventricular septal defect (VSD) is a congenital heart defect where there is a hole in the wall separating the two lower chambers of the heart (ventricles). VSDs can range from small to large. Small VSDs often close on their own while large VSDs require medical or surgical treatment. VSDs are diagnosed using tests like echocardiograms, electrocardiograms, and chest x-rays. Treatment may involve medications to manage symptoms or surgery to repair the hole in the ventricle wall. Post-surgery, patients need monitoring and antibiotics to prevent infection.
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Complex VSD closure with Pseudoaneurysm and PFO
- 1. Complex VSD with Pseudoaneurysm in scuba diver Luft MD
- 2. Complex VSD with pseudoaneurysm in a middle aged man. Chamber enlargement noted by echo. Symptoms of DOE and fatigue. 3+ angiographic shunt.
- 3. Very high membraneous VSD by TEE
- 4. Easily crossed with JR4 and glidewire
- 5. Wire snarred in right IJ and externalized; AV rail created
- 6. Delivery of 10mm VSD device (note inferior aspect of pseudoaneurysm)
- 7. LAO RAO
- 8. Device tucked into PA just below AV with minimal residual shunting (angio shows we’re clearly below the aortic valve)
- 9. Final LVG
- 10. Deep diver (over 100ft regularly and would NOT stop) with PFO and >2mm gap at rest and 5mm with valsalva
- 11. PFO closure with 25mm Amplatzer cribiform occluder.
- 14. Final cine in LAO cranial and RAO caudal views