This document provides an overview of common communicable diseases that affect children, including definitions, causative agents, modes of transmission, symptoms, and treatments. Some of the diseases discussed are chickenpox, diphtheria, measles, mumps, pertussis, polio, rubella, scarlet fever, tuberculosis, and scabies. The summary focuses on defining the key aspects of each disease such as transmission method, incubation period, symptoms, and complications. Nursing management strategies are also briefly outlined for some of the conditions.
Influenza is a respiratory infection caused by a virus (germ). Influenza occurs most often during the winter and easily spreads from person to person. Most people who get influenza feel sick for a week or two and recover. In some people, influenza leads to more serious lung infections.
Influenza is a respiratory infection caused by a virus (germ). Influenza occurs most often during the winter and easily spreads from person to person. Most people who get influenza feel sick for a week or two and recover. In some people, influenza leads to more serious lung infections.
Brief and easily understandable description on measles along with images for undergraduate students. this presentation would help in picturising what measles is.
Measles is an acute respiratory viral infection, contagious in nature. It may lead to epidemic if susceptible population is more than 40%. But with very effective vaccine, it can be eliminated
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
- Video recording of this lecture in English language: https://youtu.be/lK81BzxMqdo
- Video recording of this lecture in Arabic language: https://youtu.be/Ve4P0COk9OI
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Flu Vaccine Alert in Bangalore Karnatakaaddon Scans
As flu season approaches, health officials in Bangalore, Karnataka, are urging residents to get their flu vaccinations. The seasonal flu, while common, can lead to severe health complications, particularly for vulnerable populations such as young children, the elderly, and those with underlying health conditions.
Dr. Vidisha Kumari, a leading epidemiologist in Bangalore, emphasizes the importance of getting vaccinated. "The flu vaccine is our best defense against the influenza virus. It not only protects individuals but also helps prevent the spread of the virus in our communities," he says.
This year, the flu season is expected to coincide with a potential increase in other respiratory illnesses. The Karnataka Health Department has launched an awareness campaign highlighting the significance of flu vaccinations. They have set up multiple vaccination centers across Bangalore, making it convenient for residents to receive their shots.
To encourage widespread vaccination, the government is also collaborating with local schools, workplaces, and community centers to facilitate vaccination drives. Special attention is being given to ensuring that the vaccine is accessible to all, including marginalized communities who may have limited access to healthcare.
Residents are reminded that the flu vaccine is safe and effective. Common side effects are mild and may include soreness at the injection site, mild fever, or muscle aches. These side effects are generally short-lived and far less severe than the flu itself.
Healthcare providers are also stressing the importance of continuing COVID-19 precautions. Wearing masks, practicing good hand hygiene, and maintaining social distancing are still crucial, especially in crowded places.
Protect yourself and your loved ones by getting vaccinated. Together, we can help keep Bangalore healthy and safe this flu season. For more information on vaccination centers and schedules, residents can visit the Karnataka Health Department’s official website or follow their social media pages.
Stay informed, stay safe, and get your flu shot today!
4. DEFINITION
•A communicable disease is an illness caused
by a specific infectious agent or its toxic
products through a direct or indirect mode of
transmission of that agent from a reservoir.
Wikipedia
7. Chickenpox
• Agents: varicella-zoster virus
• Source: primary secretions of respiratory tract of
infected persons; to a lesser degree, skin lesions
(scabs not infectious).
• Transmissions: direct contact, droplet spread and
contaminated objects.
• Incubation period: 2-3 weeks, usually 14-16 days.
• Period of communicability: probably 1 day before
eruption of lesions to 6 days
8. • Clinical manifestations:
• Prodromal stage
• Constitutional signs and symptoms
• Distribution: centripetal, spreading to face and proximal
extremities but sparse on distal limbs and less on areas not
exposed to heat
• Management:
• Complications:
9. DIPHTHERIA
• Agent: corynebacterium diphtheria
• Source: discharges from mucous membranes of nose
and nasopharynx, skin and other lesions of infected
person.
• Transmission: direct contact with infected person,
carrier or contaminated articles.
• Incubation period: usually 2-5 days, possibly longer
• Period of communicability: usually 2 week but as long
as 4 week.
11. ERYTHEMA INFECTIOSUM
• Agent: human parvovirus B19(HPV)
• Source: infected persons
• Transmission: possibly respiratory secretions
(droplet infections) and blood
• Incubation period: 4-14 days
• Period of communicability: uncertain but before
onset of symptoms in children with aplastic crisis.
13. MEASLES (RUBEOLA):
• Agent: Myxo virus
• Source: respiratory tract, secretions, blood, and
urine of infected persons.
• Transmission: usually by direct contact with droplets of
infected person.
• Incubation period: 10-20 days
• Period of communicability: from 4 days before to 5 days
after rash appears, but mainly during prodromal stage.
14. • Clinical manifestation:
• Prodromal stage: fever, malaise followed in
24 hours by coryza, cough, conjunctivitis, koplik spot
(small irregular red spots with a minute bluish
white center first seen on the buccal mucosa
opposite the molars) 2 days prior to rash;
symptoms gradually increase in severity.
• Rash: appears 3-4 days after onset of prodromal
stage, begins as a maculopapular eruption on face and gradually
spreads downwards.
16. MUMPS
• Agent: rabula virus
• Source: saliva of infected persons
• Transmission: direct contact with or
droplet spread from an infected person
• Incubation period: 14-21 days
• Period of communicability: most communicable
immediately before and after swelling begins.
18. • PERTUSSIS (WHOOPING COUGH)
• Agent: bordetella pertussis
• Source: discharge from respiratory tract of infected persons
• Transmission: direct contact or droplet spread from infected
person, indirect contact with freshly contaminated articles.
• Incubation period: 5-21 days, usually 10 days.
• Period of communicability: greatest during catarrhal stage
before onset and may extend to 4th week.
19. • Clinical manifestation:
• Catarrhal stage: begins with symptoms of upper respiratory
infection- coryza, sneezing, lacrimation etc. symptoms
continue for 1-2 weeks, when dry hacking cough becomes
severe.
• Paroxysmal stage: short, rapid coughs mostly occurs at
night, followed by sudden inspiration associated with a
high pitched crowing sound or whoop during paroxysms,
cheek becomes flushed or cyanotic, eyes bulge and tongue
protrudes.
• Management
20. POLIOMYELITIS
• Agent: enterovirus, 3 types: type 1-most frequent
cause of paralysis, type 2 -least frequently associated
with paralysis, type 3- second most frequent.
• Source: feces and oro-pharyngeal secretions of
infected persons, especially young children.
• Transmission: direct contact with apparent or
inapparent active infection, spread is via fecal-oral
and pharyngeal-oropharyngeal routes.
21. Virus enters the alimentary tract (alimentary phase)
Virus multiplies and get into the blood stream
Reaches CNS and causes meningeal symptoms and paralysis
• Incubation period: usually 7-14 days
• Period of communicability: The cases are most infectious 7-10
days before and after onset of symptoms. In th efeces the virus is
excreted commonly for 2-3 weeks ,sometimes as long as 3-4
months.
22. • Clinical manifestation:
Inapparent infection: Occurs approximately in 91-96 % of
poliovirus infections .There are no presenting symptoms.
Abortive polimyelitis- the child is asymptomatic during abortive
phase, produces fever in viremic phase.
Non-paralytic polimyelitis- meningeal irritation (headache,
vomiting, neck pain, stiffness), no paralysis.
Paralytic polimyelitis- paralysis involving spinal cord and
sometimes brain stem, muscle pain, tenderness, retention of
urine, constipation, spinal and abdominal muscles are also
involved.
24. RUBELLA (GERMAN MEASLES)
• Agent: RNA virus of togavirus family
• Source: nasopharyngeal secretions of persons with apparent
or inapparent infection, virus also present in blood, stool and
urine.
• Transmission: direct contact and spread via infected person,
droplet infection, through feces or urine.
• Incubation period: 14-21 days
• Period of communicability: 7 days before to about 5 days after
appearance of rash.
25. •Clinical manifestation:
• Fever
• Pain in joints
• Posterior cervical and posterior auricular lymphadenopathy
• Maculo-papular rash (rapid progression from face to extremities)
• Infant borne to mother exposed to rubella- growth retardation,
macula-papular rash, heart murmurs, cataracts, visceromegaly,
microcephaly, mental retardation etc.
26. SCARLET FEVER
• Agent: group A beta hemolytic streptococci virus
• Source: nasopharyngeal secretions of infected persons and
carriers
• Transmission: direct contact with infected person or droplet
spread, ingestion of contaminated milk or other food.
• Incubation period: 2-4 days
• Period of communicability: during incubation period and
clinical illness approximately 10 days.
27. • Clinical manifestation:
• Prodromal stage: abrupt high fever, increased pulse, vomiting,
headache, chills, malaise, abdominal pain.
• Enanthema: enlarged tonsils, edematous, reddened and covered
with patches of exudates, pharynx is edematous and beefy red, red
and swollen papillae, and palate is covered with erythematous
punctate lesions.
• Exanthema: rash appears within 12 hours after prodromal signs,
red pin-head sized punctate lesions rapidly become generalized but
are absent on face, rash is more intense on folds of joints.
28. TUBERCULOSIS
• Agent: mycobacterium tuberculosis
• Source: respiratory secretions of actively infected persons
• Transmission: direct contact with infected persons
• Incubation period: from infection to primary lesion, about
4-6 weeks
• Period of communicability: as long as bacilli are
discharged
29. • Clinical manifestations:
Usually asymptomatic
Demonstration of bacilli in sputum or
gastric aspirates.
In advanced cases, fever, pallor,
weakness, weight loss, cough, hoarseness, and
tachypnea.
Treatment:
30. SCABIES
Agent: Sarcoptes scabiei or Acarus scabiei
Source: actively infected persons
Transmission: direct contact with infected
Persons and contaminated clothes
Site of leisions: Hands and wrist(63%); the extensor aspect of
elbows(10.9%); axillae,buttocks,lower abdomen, feet and ankles.
Control measures: Treat with Benzyl Benzoate (25%) weekly twice
HCH:0.5 -1.5% ,2-3 Days of interval
Tetmosol: thrice a day application
Sulphur ointment: 2.5 to 10% daily for 4 days