CLASS -7
• SYSTEMIC CIRCULATION—VENOUS SYSTEM
CIRCULATION
SHOCK
• FAILURE OF CARDIOVASCULAR SYSTEM TO
DELIVER ENOUGH OXYGEN & NUTRIENTS TO
MEET CELLULAR & METABOLIC NEEDS –LEADING
TO LACTIC ACIDOSIS & CELL DEATH
• RESPONSE TO SHOCK—RAA SYSTEM
ACTIVATION;INCREASE IN ADH ;SYMPATHETIC
STIMULATION
• FEATURES—LOW BP,TACHYCARDIA,WEAKNESS,
COLD CLAMMY SKIN ,MENTAL CONFUSION ,
NAUSEA-VOMITING,LOW URINE OUTPUT
SHOCK -CAUSES
• HYPOVOLEMIC SHOCK—20% Reduction in fluid volume
in body, eg diarrhea, vomiting bleeding burn
• CARDIOGENIC SHOCK—poor heart function following
CCF ,MI
• SEPTIC SHOCK –Due to circulating toxin resulting in
vasodilatation-poor contractility of heart,or massive
inflammatory reaction
• NEUROGENIC SHOCK—INCREASED PARASYMPATHETIC
ACTIVITY—vasovagalshock
• ANAPHYLACTIC SHOCK -Vascular dilatation &shock
EDEMA
• Excess fluid accumulation in tissues
• Superficial /Deep
• Due to
– Increased hydrostatic pressure
– Decreased osmotic pressure
– Impaired lymphatic drainage
– Increased permeability with inflammation
• Ascitis , pleural effusion ,pericardial effusion
• Superficial dependent edema

CIRCULATORY SYSTEM FOR NURSES CLASS 7.pptx

  • 1.
    CLASS -7 • SYSTEMICCIRCULATION—VENOUS SYSTEM
  • 22.
  • 28.
    SHOCK • FAILURE OFCARDIOVASCULAR SYSTEM TO DELIVER ENOUGH OXYGEN & NUTRIENTS TO MEET CELLULAR & METABOLIC NEEDS –LEADING TO LACTIC ACIDOSIS & CELL DEATH • RESPONSE TO SHOCK—RAA SYSTEM ACTIVATION;INCREASE IN ADH ;SYMPATHETIC STIMULATION • FEATURES—LOW BP,TACHYCARDIA,WEAKNESS, COLD CLAMMY SKIN ,MENTAL CONFUSION , NAUSEA-VOMITING,LOW URINE OUTPUT
  • 29.
    SHOCK -CAUSES • HYPOVOLEMICSHOCK—20% Reduction in fluid volume in body, eg diarrhea, vomiting bleeding burn • CARDIOGENIC SHOCK—poor heart function following CCF ,MI • SEPTIC SHOCK –Due to circulating toxin resulting in vasodilatation-poor contractility of heart,or massive inflammatory reaction • NEUROGENIC SHOCK—INCREASED PARASYMPATHETIC ACTIVITY—vasovagalshock • ANAPHYLACTIC SHOCK -Vascular dilatation &shock
  • 30.
    EDEMA • Excess fluidaccumulation in tissues • Superficial /Deep • Due to – Increased hydrostatic pressure – Decreased osmotic pressure – Impaired lymphatic drainage – Increased permeability with inflammation • Ascitis , pleural effusion ,pericardial effusion • Superficial dependent edema