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Presentation onPresentation on
childhood disorderschildhood disorders
Presented by : Parul Prasher
mental health nursing dept
Disorders Usually 1Disorders Usually 1stst
DiagnosedDiagnosed
in Infancy, Childhood, &in Infancy, Childhood, &
AdolescenceAdolescence
Core Concept Of Diagnostic
Group:
Categorized by time of onset
Predominantly disorders of abnormal
development and maturation.
Emphasis of disorders is on the inability of
the individual to attain certain normal
developmental milestones and the
associated functions, capabilities, &
behaviors.
10 DIAGNOSTIC SUBGROUPS10 DIAGNOSTIC SUBGROUPS
(DSM-IV-TR)(DSM-IV-TR)
1) Mental Retardation
2) Learning Disorders
3) Motor Skills Disorders
4) Communication Disorders
5) Pervasive Developmental Disorders
6) Attention Deficitand Disruptive Behavior Disorders
7) Feeding & Eating Disorders ofInfancy & Early Childhood
8) Tic Disorders
9) Elimination Disorders
10) Other Disorders ofInfancy, Childhood, or Adolescence
Mental RetardationMental Retardation
Characteristics:
IQ is significantly below average (<
70)
Accompanied by deficits in adaptive
functioning, e.g. communication,
self-care, home living,
social/interpersonal skills, use of
community resources, self-direction,
academic skills, work, leisure,
health, safety.
Onset and codingOnset and coding
 Onset before age 18 years
 Coding: coded on axis II
 Code based on degree of severity, reflecting level of
intellectual impairment:
◦ Mild Mental Retardation – IQ from 50-55
to 70
◦ Moderate Mental Retardation – IQ from
35-40 to 50-55
◦ Severe Mental Retardation – IQ from 20-
25 to 35-40
◦ Profound Mental Retardation – IQ below
20-25
Mental RetardationMental Retardation
 Prevalence: 1-3% of population; 90% are mild MR
 Course: chronic
 Prognosis: variable, depending on IQ & level of
impairment
 Gender differences: more prevalent for males (1.6 to
1); no gender differences for severe & profound MR
 Causes: genetic; chromosomal (Down syndrome,
Fragile X syndrome, Lesch-Nyhan syndrome);
environmental (deprivation, abuse, neglect); prenatal
(exposure to disease, alcohol, drugs, chemicals, poor
maternal nutrition); perinatal (difficulties during labor &
delivery); postnatal (malnutrition, infections, & head
injuries)
 Treatment: behavioral skills training; communication
training; supported living and employment;
mainstreaming
Causes and TreatmentCauses and Treatment
Causes: genetic; chromosomal (Down
syndrome, Fragile X syndrome, Lesch-
Nyhan syndrome); environmental
(deprivation, abuse, neglect); prenatal
(exposure to disease, alcohol, drugs,
chemicals, poor maternal nutrition);
perinatal (difficulties during labor &
delivery); postnatal (malnutrition,
infections, & head injuries)
Treatment: behavioral skills training;
communication training; supported living
and employment; mainstreaming
LEARNING DISORDERLEARNING DISORDER
Characteristics:
Inadequate development of specific
academic skills, such as reading,
writing, and math.
Specific academic skills are
substantially below expected for
age, intelligence, and education
Significantly interferes with aspects
of life requiring those skills.
Subtypes:
Reading Disorder
Mathematics Disorder
Disorder of Written Expression
Learning Disorder Not Otherwise
Specified
Prevalence:
◦ general population: 5-10%
◦ reading disorders: 5-15%
◦ math disorders: 6%
Racial: more common in black children
Negative outcomes: negative school
experiences; school drop-out; lower employment
rates; lower educational & career goals
Causes: genetics; structural & functional
differences in the brain
Treatment: educational interventions
(processing skills; cognitive skills; behavioral skills)
TIC DISORDER
Tic Disorder: Tourette’sTic Disorder: Tourette’s
DisorderDisorder
Symptoms: characterized by multiple motor tics and
one or more vocal tics (involuntary, sudden, rapid,
nonrhythmic, stereotyped motor movements or
vocalizations), which occur many times a day, nearly
every day, or intermittently for more than a year.
Common motor tics: eye-blinking, eye-rolling,
spitting, flipping/twirling hair, rolling head around,
bending/jumping, skin picking, shrugging/jerking
shoulders, thrusting pelvic movements, tapping
fingers/feet
Common vocal tics: throat clearing, tongue-clicking,
whistling, grunting, humming, hoots, howls,
burps/belches, animal noises, repetition of one’s own
words, repetition of others’ words
Contd .Contd .
 Causes: genetic (32% have relatives with TD); abnormal
metabolism of 5HT & D; brain processing problem (basal
ganglia)
 Prevalence: decreases with age; 5-30 per 10,000 in
childhood; 1-2 per 10,000 in adulthood
 Gender: 2-5x as common for males
 Onset: as early as 2 yrs; average age of onset is 6-7 yrs;
typically develops by age 14
 Course: severity, frequency, and disruptiveness of sx
diminish during adolescence & adulthood
 Treatment: antipsychotics; antihypertensive
medications; SSRI’s; self-monitoring; relaxation training;
habit reversal
ADHDADHD
Attention Deficit/Hyperactivity DisorderAttention Deficit/Hyperactivity Disorder
Includes two major syndromes:
1) Inattention
2) Hyperactivity-Impulsivity
Syndromes may occur independently or
together, but usually some components of
each are present.
Symptoms begin before age 7
Symptoms cause some impairment in 2 or
more settings.
Inattention: 6+ of the following for 6+ months
 Often fails to give close attention to details
 Often makes careless mistakes in school, work,
etc.
 Often has difficulty sustaining attention
 Often doesn’t seem to listen when spoken to
directly
 Often doesn’t follow instructions
 Often fails to finish schoolwork, chores, or work
duties
 Has difficulty organizing tasks & activities
 Avoids or dislikes tasks requiring sustained mental
effort
 Often loses things
 Is easily distracted by extraneous stimuli
 Is forgetful in daily activities
Attention Deficit/Hyperactivity DisorderAttention Deficit/Hyperactivity Disorder
Hyperactivity-Impulsivity 6+ of following for 6+ months
Hyperactivity:
 Fidgets with hands or feet; squirms in seat
 Difficulty staying in seat
 Excessive running, climbing, or restlessness
 Difficulty playing or engaging in leisure activities quietly
 Often “on the go;” acts as if “driven by a motor”
 Often talks excessively
Impulsivity:
 Often blurts out statements
 Impatient; difficulty awaiting turn
 Often interrupts or intrudes on others
Attention Deficit/Hyperactivity DisorderAttention Deficit/Hyperactivity Disorder
 Subtypes:
◦ AD/HD, Predominantly Inattentive Type
◦ AD/HD, Predominantly Hyperactive-Impulsive Type
◦ AD/HD, Combined Type
◦ AD/HD, Not Otherwise Specified
 Onset: 3-4 years old
 Age: 68% have ongoing sx in adulthood; inattentive
subtype is more common in adolescents and adults
 Gender: ratios of males to females range from 2:1 to 9:1;
Combined and Hyperactive Subtypes are much more
common in males than females
 Prevalence: up to 3-7% of school-age children
ADHD: Associated FeaturesADHD: Associated Features
 Academic deficits
 School-related problems
 Peer rejection
 Low frustration tolerance
 Tantrums
 Poor self-esteem
 Mood swings
 Bossiness
 Stubbornness
 Accidents
 Driving difficulties – speeding, accidents
ADHD: Diagnostic ConsiderationsADHD: Diagnostic Considerations
 Difficulty of distinguishing normal activity from
hyperactivity and normal distractibility from attention
deficit distractibility.
 Need to evaluate behavior in terms of what’s normal for
others of same gender, age, developmental level, cultural
background.
 Behaviors must occur in multiple settings.
 Behaviors must cause clinically significant impairment.
 Symptoms must have been present and caused
impairment by age 7.
 Combined and Hyperactive Subtypes are less likely to be
missed.
ADHD: Contributing FactorsADHD: Contributing Factors
Genetics: increased incidence of ADHD &
psychopathology in families & relatives
Prenatal factors: inadequate oxygen; drug
exposure; maternal smoking
Neurotransmitters: inadequate availability of
dopamine; NE, 5HT, GABA also implicated
Brain abnormalities: frontal cortex, basal
ganglia, & cerebellar vermis are smaller
Exposure to toxins: allergens, food additives
Parenting: negative attempts to control their
behavior; intrusive, over-bearing parenting
Attention Deficit/Hyperactivity DisorderAttention Deficit/Hyperactivity Disorder
Treatments:
Medication – stimulants, Strattera (SNRI),
Wellbutrin
Psychoeducation & bibliotherapy
Skills-based training – time management,
organizational skills, study skills, problem-
solving, social skills
CONDUCT DISORDERCONDUCT DISORDER
Conduct DisorderConduct Disorder
 Repetitive, persistent pattern of behavior in
which the basic rights of others or major
societal norms or rules are violated.
 3 or more of the following are present in the
past 12 months, and at least one of the
following is present in the past 6 months.
1) Aggression to people and animals
2) Destruction of property
3) Deceitfulness or theft
4) Serious violations of rules
Conduct DisorderConduct Disorder
1) Aggression to People and
Animals:
◦ Bullying, threats, intimidation
◦ Physical fights
◦ Use of weapons
◦ Physical cruelty to people
◦ Physical cruelty to animals
◦ Mugging, purse snatching, extortion,
armed robbery
◦ Forced sexual activity
Conduct DisorderConduct Disorder
2) Destruction of Property:
◦ Deliberate fire-setting
◦ Deliberate destruction of others’ property
3) Deceitfulness or Theft
◦ Breaking & entering
◦ Lying; conning
◦ Stealing; shoplifting; forgery
4) Serious Violations of Rules
◦ Breaking curfew prior to age 13
◦ School truancy prior to age 13
◦ Running away from home
Conduct DisorderConduct Disorder
Subtypes:
 Conduct Disorder, Childhood Onset – onset of at least
1 criterion prior to age 10
 Conduct Disorder, Adolescent Onset – absence of any
criteria prior to 10
 Conduct Disorder, Unspecified Onset – age of onset is
unknown
Specifiers:
 Mild – few, if any, conduct problems in excess of those
required to make dx; cause only minor harm to others
 Moderate – number of conduct problems and effect on
others are in the intermediate range
 Severe – many conduct problems in excess of those
required to make dx; cause considerable harm to others
Conduct DisorderConduct Disorder
 Etiology: genetics; decreased arousal; low levels of
5HT; neurological deficits
 Prevalence: 2-9% of nonclinical population; up to 1/3-
1/2 of child mental health referrals; 87-91% of
incarcerated juveniles
 Gender Differences: mostly males
 Onset: as early as preschool
 Prognosis: poor; 2/3rds of cases develop into
Antisocial Personality Disorder
 Treatment: parent management training; community-
based interventions (group homes, wilderness programs;
therapeutic boarding schools); CBT (social skills,
problem solving, cognitive restructuring)
Oppositional Defiant DisorderOppositional Defiant Disorder
 Pattern of negativistic, hostile, and defiant behavior for at
least 6 months.
 At least 4 of the following are present:
◦ Often loses temper
◦ Often argues with adults
◦ Often actively defies or refuses to comply with
adults’ requests or rules
◦ Often deliberately annoys others
◦ Often blames others for own mistakes or
misbehavior
◦ Is often touchy or easily annoyed by others
◦ Is often angry or resentful
◦ Is often spiteful or vindictive
Oppositional Defiant DisorderOppositional Defiant Disorder
 Prevalence: 1-6%
 Gender differences: more prevalent for males prior
to puberty; ratio evens out after puberty
 Prognosis: relatively persistent – some of the behaviors
persist into adulthood, others are outgrown; higher
divorce rate, employment difficulties, and drug/alcohol
abuse for those with ODD
 Causes: marital conflict; family discord; inconsistent
parenting; overly lenient or rigid parent; coercive or
aversive parent-child interactions; genetics
 Treatment: parent training; family therapy; behavioral
therapy (anger management, social skills training,
problem solving, frustration tolerance); cognitive
interventions to reduce negativity
Separation Anxiety DisorderSeparation Anxiety Disorder
At least 4 weeks of inappropriate or excessive anxiety
about separation from home or major attachment figures,
as evidenced by at least 3 of the following:
◦ excessive anxiety regarding separation
◦ excessive fears of losing major attachment figures
◦ nightmares involving the theme of separation
◦ refusal to go to school
◦ refusal to be alone or without major attachment
figures
◦ refusal to sleep away from home or attachment
figures
◦ repeated physical complaints when separation occurs
or is anticipated
Onset prior to age 18
Pervasive DevelopmentalPervasive Developmental
DisordersDisorders
Characterized by:
 A broad-based impairment or a loss of
functions expected for child’s age.
 Includes 3 components:
1) Impairment in social
interactions/relationships
2) Impairment in communication/language
3) Restricted, repetitive, and stereotyped
patterns of behavior, interests, and activities
Autistic DisorderAutistic Disorder
 Abnormal functioning in at least one of the
following areas, with onset prior to 3:
1) Social interaction
2) Language and communication
3) Symbolic, imaginative play
 Qualitative impairment in social interaction
and relationship development
 Qualitative impairment in communication,
language, and conversation skills
 Restricted, repetitive, stereotyped patterns of
behavior, interests, activities.
AutismAutism
 Mental retardation: 75-80%; 50% are profoundly or
severely MR; 25% are moderately MR; 25% borderline to
average IQ
 Gender differences: higher IQ – more prevalent among
males; IQ < 35 – more prevalent among females
 Prevalence: 1 in 500 births
 Onset: first apparent in infancy & toddlerhood
 Course: chronic; life-long impairment; 50% never acquire
speech
 Causes: abnormalities in brain structure and function (5HT
synthesis, cerebellum); genetics
 Treatments: intensive behavioral Tx focusing on improving
communication, social and daily living skills and reducing
problem behaviors; early intervention programs; applied
behavior analysis; parent training; mainstreaming for
education; community interventions (supportive living
arrangements & work settings)
Asperger’s DisorderAsperger’s Disorder
Qualitative impairment in social
interaction and relationship
development
Restricted, repetitive, and
stereotyped patterns of behavior,
interests, and activities
But lack any clinically significant
delay in language or cognitive
development
Asperger’s SyndromeAsperger’s Syndrome
What you see:
 Anxious, excessive desire for sameness
 Preoccupation with stereotyped, repetitive activities
 Obsess about objects
 Limited interests
 Can’t relate to others
 Can’t read emotions
 Can’t understand social cues
 Social isolation, socially inept
 Average IQ scores
 Motor clumsiness
 Poor coordination
Asperger’s SyndromeAsperger’s Syndrome
Gender: up to 4x as common for males
Prevalence: up to 5x as common as
Autism
Onset: later onset than Autism
Course: chronic, life-long
Etiology: genetics; brain abnormalities
(limbic system, 5HT & D systems, right
hemisphere)
Asperger’s Syndrome: TreatmentsAsperger’s Syndrome: Treatments
Behavioral treatments/skills building:
interventions targeting problem behaviors,
problem solving, social skills, communication
skills, empathy-building, daily living skills
School-based interventions: mainstreaming;
tutoring; special aides; multiple modalities for
presenting information
Psychotherapy to address accompanying
psychiatric disorders, such as depression and
anxiety
Medications: antidepressants, antipsychotics
Nurses Role In Management OfNurses Role In Management Of
Childhood DisorderChildhood Disorder
 Ensuring the child’s safety and that of others
Stop unsafe behavior.
 Provide close supervision
 Give clear directions about acceptable and
unacceptable behavior.
   Improved role performance
  Give positive feedback for meeting
expectations.
 Manage the environment (e.g., provide a quiet
place free of distractions for task completion).
 Simplifying instructions/directions
  Get child’s full attention.
Contd.Contd.

Break complex tasks into small steps.
  Allow breaks.
          Structured daily routine
          Establish a daily schedule.
     Minimize changes.
          Client/family education and support
          Listen to parent’s feelings and
frustrations.
   Improving role performance
   Simplifying instructions
   Promoting a structured daily routine
   Providing client and family education and
support
Childhood disorders
Childhood disorders

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Childhood disorders

  • 1. Presentation onPresentation on childhood disorderschildhood disorders Presented by : Parul Prasher mental health nursing dept
  • 2. Disorders Usually 1Disorders Usually 1stst DiagnosedDiagnosed in Infancy, Childhood, &in Infancy, Childhood, & AdolescenceAdolescence Core Concept Of Diagnostic Group: Categorized by time of onset Predominantly disorders of abnormal development and maturation. Emphasis of disorders is on the inability of the individual to attain certain normal developmental milestones and the associated functions, capabilities, & behaviors.
  • 3. 10 DIAGNOSTIC SUBGROUPS10 DIAGNOSTIC SUBGROUPS (DSM-IV-TR)(DSM-IV-TR) 1) Mental Retardation 2) Learning Disorders 3) Motor Skills Disorders 4) Communication Disorders 5) Pervasive Developmental Disorders 6) Attention Deficitand Disruptive Behavior Disorders 7) Feeding & Eating Disorders ofInfancy & Early Childhood 8) Tic Disorders 9) Elimination Disorders 10) Other Disorders ofInfancy, Childhood, or Adolescence
  • 4.
  • 5. Mental RetardationMental Retardation Characteristics: IQ is significantly below average (< 70) Accompanied by deficits in adaptive functioning, e.g. communication, self-care, home living, social/interpersonal skills, use of community resources, self-direction, academic skills, work, leisure, health, safety.
  • 6. Onset and codingOnset and coding  Onset before age 18 years  Coding: coded on axis II  Code based on degree of severity, reflecting level of intellectual impairment: ◦ Mild Mental Retardation – IQ from 50-55 to 70 ◦ Moderate Mental Retardation – IQ from 35-40 to 50-55 ◦ Severe Mental Retardation – IQ from 20- 25 to 35-40 ◦ Profound Mental Retardation – IQ below 20-25
  • 7. Mental RetardationMental Retardation  Prevalence: 1-3% of population; 90% are mild MR  Course: chronic  Prognosis: variable, depending on IQ & level of impairment  Gender differences: more prevalent for males (1.6 to 1); no gender differences for severe & profound MR  Causes: genetic; chromosomal (Down syndrome, Fragile X syndrome, Lesch-Nyhan syndrome); environmental (deprivation, abuse, neglect); prenatal (exposure to disease, alcohol, drugs, chemicals, poor maternal nutrition); perinatal (difficulties during labor & delivery); postnatal (malnutrition, infections, & head injuries)  Treatment: behavioral skills training; communication training; supported living and employment; mainstreaming
  • 8. Causes and TreatmentCauses and Treatment Causes: genetic; chromosomal (Down syndrome, Fragile X syndrome, Lesch- Nyhan syndrome); environmental (deprivation, abuse, neglect); prenatal (exposure to disease, alcohol, drugs, chemicals, poor maternal nutrition); perinatal (difficulties during labor & delivery); postnatal (malnutrition, infections, & head injuries) Treatment: behavioral skills training; communication training; supported living and employment; mainstreaming
  • 10.
  • 11. Characteristics: Inadequate development of specific academic skills, such as reading, writing, and math. Specific academic skills are substantially below expected for age, intelligence, and education Significantly interferes with aspects of life requiring those skills. Subtypes: Reading Disorder Mathematics Disorder Disorder of Written Expression Learning Disorder Not Otherwise Specified
  • 12. Prevalence: ◦ general population: 5-10% ◦ reading disorders: 5-15% ◦ math disorders: 6% Racial: more common in black children Negative outcomes: negative school experiences; school drop-out; lower employment rates; lower educational & career goals Causes: genetics; structural & functional differences in the brain Treatment: educational interventions (processing skills; cognitive skills; behavioral skills)
  • 14. Tic Disorder: Tourette’sTic Disorder: Tourette’s DisorderDisorder Symptoms: characterized by multiple motor tics and one or more vocal tics (involuntary, sudden, rapid, nonrhythmic, stereotyped motor movements or vocalizations), which occur many times a day, nearly every day, or intermittently for more than a year. Common motor tics: eye-blinking, eye-rolling, spitting, flipping/twirling hair, rolling head around, bending/jumping, skin picking, shrugging/jerking shoulders, thrusting pelvic movements, tapping fingers/feet Common vocal tics: throat clearing, tongue-clicking, whistling, grunting, humming, hoots, howls, burps/belches, animal noises, repetition of one’s own words, repetition of others’ words
  • 15. Contd .Contd .  Causes: genetic (32% have relatives with TD); abnormal metabolism of 5HT & D; brain processing problem (basal ganglia)  Prevalence: decreases with age; 5-30 per 10,000 in childhood; 1-2 per 10,000 in adulthood  Gender: 2-5x as common for males  Onset: as early as 2 yrs; average age of onset is 6-7 yrs; typically develops by age 14  Course: severity, frequency, and disruptiveness of sx diminish during adolescence & adulthood  Treatment: antipsychotics; antihypertensive medications; SSRI’s; self-monitoring; relaxation training; habit reversal
  • 17. Attention Deficit/Hyperactivity DisorderAttention Deficit/Hyperactivity Disorder Includes two major syndromes: 1) Inattention 2) Hyperactivity-Impulsivity Syndromes may occur independently or together, but usually some components of each are present. Symptoms begin before age 7 Symptoms cause some impairment in 2 or more settings.
  • 18. Inattention: 6+ of the following for 6+ months  Often fails to give close attention to details  Often makes careless mistakes in school, work, etc.  Often has difficulty sustaining attention  Often doesn’t seem to listen when spoken to directly  Often doesn’t follow instructions  Often fails to finish schoolwork, chores, or work duties  Has difficulty organizing tasks & activities  Avoids or dislikes tasks requiring sustained mental effort  Often loses things  Is easily distracted by extraneous stimuli  Is forgetful in daily activities
  • 19. Attention Deficit/Hyperactivity DisorderAttention Deficit/Hyperactivity Disorder Hyperactivity-Impulsivity 6+ of following for 6+ months Hyperactivity:  Fidgets with hands or feet; squirms in seat  Difficulty staying in seat  Excessive running, climbing, or restlessness  Difficulty playing or engaging in leisure activities quietly  Often “on the go;” acts as if “driven by a motor”  Often talks excessively Impulsivity:  Often blurts out statements  Impatient; difficulty awaiting turn  Often interrupts or intrudes on others
  • 20. Attention Deficit/Hyperactivity DisorderAttention Deficit/Hyperactivity Disorder  Subtypes: ◦ AD/HD, Predominantly Inattentive Type ◦ AD/HD, Predominantly Hyperactive-Impulsive Type ◦ AD/HD, Combined Type ◦ AD/HD, Not Otherwise Specified  Onset: 3-4 years old  Age: 68% have ongoing sx in adulthood; inattentive subtype is more common in adolescents and adults  Gender: ratios of males to females range from 2:1 to 9:1; Combined and Hyperactive Subtypes are much more common in males than females  Prevalence: up to 3-7% of school-age children
  • 21. ADHD: Associated FeaturesADHD: Associated Features  Academic deficits  School-related problems  Peer rejection  Low frustration tolerance  Tantrums  Poor self-esteem  Mood swings  Bossiness  Stubbornness  Accidents  Driving difficulties – speeding, accidents
  • 22. ADHD: Diagnostic ConsiderationsADHD: Diagnostic Considerations  Difficulty of distinguishing normal activity from hyperactivity and normal distractibility from attention deficit distractibility.  Need to evaluate behavior in terms of what’s normal for others of same gender, age, developmental level, cultural background.  Behaviors must occur in multiple settings.  Behaviors must cause clinically significant impairment.  Symptoms must have been present and caused impairment by age 7.  Combined and Hyperactive Subtypes are less likely to be missed.
  • 23. ADHD: Contributing FactorsADHD: Contributing Factors Genetics: increased incidence of ADHD & psychopathology in families & relatives Prenatal factors: inadequate oxygen; drug exposure; maternal smoking Neurotransmitters: inadequate availability of dopamine; NE, 5HT, GABA also implicated Brain abnormalities: frontal cortex, basal ganglia, & cerebellar vermis are smaller Exposure to toxins: allergens, food additives Parenting: negative attempts to control their behavior; intrusive, over-bearing parenting
  • 24. Attention Deficit/Hyperactivity DisorderAttention Deficit/Hyperactivity Disorder Treatments: Medication – stimulants, Strattera (SNRI), Wellbutrin Psychoeducation & bibliotherapy Skills-based training – time management, organizational skills, study skills, problem- solving, social skills
  • 25.
  • 27. Conduct DisorderConduct Disorder  Repetitive, persistent pattern of behavior in which the basic rights of others or major societal norms or rules are violated.  3 or more of the following are present in the past 12 months, and at least one of the following is present in the past 6 months. 1) Aggression to people and animals 2) Destruction of property 3) Deceitfulness or theft 4) Serious violations of rules
  • 28. Conduct DisorderConduct Disorder 1) Aggression to People and Animals: ◦ Bullying, threats, intimidation ◦ Physical fights ◦ Use of weapons ◦ Physical cruelty to people ◦ Physical cruelty to animals ◦ Mugging, purse snatching, extortion, armed robbery ◦ Forced sexual activity
  • 29. Conduct DisorderConduct Disorder 2) Destruction of Property: ◦ Deliberate fire-setting ◦ Deliberate destruction of others’ property 3) Deceitfulness or Theft ◦ Breaking & entering ◦ Lying; conning ◦ Stealing; shoplifting; forgery 4) Serious Violations of Rules ◦ Breaking curfew prior to age 13 ◦ School truancy prior to age 13 ◦ Running away from home
  • 30. Conduct DisorderConduct Disorder Subtypes:  Conduct Disorder, Childhood Onset – onset of at least 1 criterion prior to age 10  Conduct Disorder, Adolescent Onset – absence of any criteria prior to 10  Conduct Disorder, Unspecified Onset – age of onset is unknown Specifiers:  Mild – few, if any, conduct problems in excess of those required to make dx; cause only minor harm to others  Moderate – number of conduct problems and effect on others are in the intermediate range  Severe – many conduct problems in excess of those required to make dx; cause considerable harm to others
  • 31. Conduct DisorderConduct Disorder  Etiology: genetics; decreased arousal; low levels of 5HT; neurological deficits  Prevalence: 2-9% of nonclinical population; up to 1/3- 1/2 of child mental health referrals; 87-91% of incarcerated juveniles  Gender Differences: mostly males  Onset: as early as preschool  Prognosis: poor; 2/3rds of cases develop into Antisocial Personality Disorder  Treatment: parent management training; community- based interventions (group homes, wilderness programs; therapeutic boarding schools); CBT (social skills, problem solving, cognitive restructuring)
  • 32.
  • 33. Oppositional Defiant DisorderOppositional Defiant Disorder  Pattern of negativistic, hostile, and defiant behavior for at least 6 months.  At least 4 of the following are present: ◦ Often loses temper ◦ Often argues with adults ◦ Often actively defies or refuses to comply with adults’ requests or rules ◦ Often deliberately annoys others ◦ Often blames others for own mistakes or misbehavior ◦ Is often touchy or easily annoyed by others ◦ Is often angry or resentful ◦ Is often spiteful or vindictive
  • 34. Oppositional Defiant DisorderOppositional Defiant Disorder  Prevalence: 1-6%  Gender differences: more prevalent for males prior to puberty; ratio evens out after puberty  Prognosis: relatively persistent – some of the behaviors persist into adulthood, others are outgrown; higher divorce rate, employment difficulties, and drug/alcohol abuse for those with ODD  Causes: marital conflict; family discord; inconsistent parenting; overly lenient or rigid parent; coercive or aversive parent-child interactions; genetics  Treatment: parent training; family therapy; behavioral therapy (anger management, social skills training, problem solving, frustration tolerance); cognitive interventions to reduce negativity
  • 35.
  • 36.
  • 37.
  • 38. Separation Anxiety DisorderSeparation Anxiety Disorder At least 4 weeks of inappropriate or excessive anxiety about separation from home or major attachment figures, as evidenced by at least 3 of the following: ◦ excessive anxiety regarding separation ◦ excessive fears of losing major attachment figures ◦ nightmares involving the theme of separation ◦ refusal to go to school ◦ refusal to be alone or without major attachment figures ◦ refusal to sleep away from home or attachment figures ◦ repeated physical complaints when separation occurs or is anticipated Onset prior to age 18
  • 39. Pervasive DevelopmentalPervasive Developmental DisordersDisorders Characterized by:  A broad-based impairment or a loss of functions expected for child’s age.  Includes 3 components: 1) Impairment in social interactions/relationships 2) Impairment in communication/language 3) Restricted, repetitive, and stereotyped patterns of behavior, interests, and activities
  • 40.
  • 41. Autistic DisorderAutistic Disorder  Abnormal functioning in at least one of the following areas, with onset prior to 3: 1) Social interaction 2) Language and communication 3) Symbolic, imaginative play  Qualitative impairment in social interaction and relationship development  Qualitative impairment in communication, language, and conversation skills  Restricted, repetitive, stereotyped patterns of behavior, interests, activities.
  • 42. AutismAutism  Mental retardation: 75-80%; 50% are profoundly or severely MR; 25% are moderately MR; 25% borderline to average IQ  Gender differences: higher IQ – more prevalent among males; IQ < 35 – more prevalent among females  Prevalence: 1 in 500 births  Onset: first apparent in infancy & toddlerhood  Course: chronic; life-long impairment; 50% never acquire speech  Causes: abnormalities in brain structure and function (5HT synthesis, cerebellum); genetics  Treatments: intensive behavioral Tx focusing on improving communication, social and daily living skills and reducing problem behaviors; early intervention programs; applied behavior analysis; parent training; mainstreaming for education; community interventions (supportive living arrangements & work settings)
  • 43.
  • 44.
  • 45. Asperger’s DisorderAsperger’s Disorder Qualitative impairment in social interaction and relationship development Restricted, repetitive, and stereotyped patterns of behavior, interests, and activities But lack any clinically significant delay in language or cognitive development
  • 46. Asperger’s SyndromeAsperger’s Syndrome What you see:  Anxious, excessive desire for sameness  Preoccupation with stereotyped, repetitive activities  Obsess about objects  Limited interests  Can’t relate to others  Can’t read emotions  Can’t understand social cues  Social isolation, socially inept  Average IQ scores  Motor clumsiness  Poor coordination
  • 47. Asperger’s SyndromeAsperger’s Syndrome Gender: up to 4x as common for males Prevalence: up to 5x as common as Autism Onset: later onset than Autism Course: chronic, life-long Etiology: genetics; brain abnormalities (limbic system, 5HT & D systems, right hemisphere)
  • 48. Asperger’s Syndrome: TreatmentsAsperger’s Syndrome: Treatments Behavioral treatments/skills building: interventions targeting problem behaviors, problem solving, social skills, communication skills, empathy-building, daily living skills School-based interventions: mainstreaming; tutoring; special aides; multiple modalities for presenting information Psychotherapy to address accompanying psychiatric disorders, such as depression and anxiety Medications: antidepressants, antipsychotics
  • 49.
  • 50. Nurses Role In Management OfNurses Role In Management Of Childhood DisorderChildhood Disorder
  • 51.  Ensuring the child’s safety and that of others Stop unsafe behavior.  Provide close supervision  Give clear directions about acceptable and unacceptable behavior.    Improved role performance   Give positive feedback for meeting expectations.  Manage the environment (e.g., provide a quiet place free of distractions for task completion).  Simplifying instructions/directions   Get child’s full attention.
  • 52. Contd.Contd.  Break complex tasks into small steps.   Allow breaks.           Structured daily routine           Establish a daily schedule.      Minimize changes.           Client/family education and support           Listen to parent’s feelings and frustrations.    Improving role performance    Simplifying instructions    Promoting a structured daily routine    Providing client and family education and support