Cementum is the mineralized tissue covering the roots of teeth. It is less calcified and harder than dentin. Cementum is formed by cementoblasts and is composed of collagen fibers embedded in a hydroxyapatite matrix. Cementum provides anchorage for periodontal ligament fibers and continues to be deposited throughout life, increasing the thickness of roots. The cementoenamel junction marks the boundary between cementum and enamel and can be overlapped, edge-to-edge, or with cementum failing to meet enamel. Cementum functions to anchor teeth, maintain periodontal ligament width, and repair root fractures. Conditions like hypercementosis, ankylosis, and cementicles can
Introduction
A sound knowledge of the anatomy of the periodontium and the surrounding hard and soft structures is essential to determine the scope and possibilities of surgical periodontal procedures and to minimize their risks.
Blood vessels, and nerves located in the vicinity of the periodontal surgical field, are particularly important during various surgical procedures.
Arterial Supply
Common Carotid Artery
Carotid Sinus & Carotid Body
Applied Anatomy of CCA
CAROTID PULSE :
CCA may be compressed against the carotid tubercle of transverse process of C6 vertebra about 4cm above the sternoclavicular joint.
External Carotid Artery
Generally it lies anterior to the Internal Carotid Artery.
It is the chief artery of supply to structures in the front of neck, oral cavity and in the face.
In carotid triangle
Crossed superficially by:
Cervical branch of facial nerve
Hypoglossal nerve
Facial, lingual &superior thyroid vein
Deep to artery lies:
Wall of pharynx
Superior laryngeal nerve
Ascending pharyngeal artery
Above the carotid triangle
ECA lies deep in the substance of parotid gland
Branches
Lingual Artery
Principal artery of tongue.
Arises anteromedially from ECA opposite the tip of greater cornu of hyoid bone.
Divided into three parts by hyoglossus muscle.
Applied anatomy
Sublingual artery injury occurs in premolar & molar region, when sharp instrument or rotating disks slips off a lower molar & injure the floor of mouth.
Sublingual and submental arteries may course anteriorly in close proximity to the lingual plate, and branches of these blood vessels enter accessory foramina along the lingual cortex.
Hofschneider et al (1999)
Inadvertent penetration through the lingual cortical plate into the floor of the mouth while preparing an osteotomy can cause arterial trauma, thereby resulting in development of a sublingual or submandibular hematoma
Flanagan D. et al.2003
Facial Artery
ORIGIN: Arises from the ECA just above the tip of greater cornua of hyoid bone.
COURSE:
Runs upwards in neck as cervical part ;
On face as facial part.
Tortuous course—
In neck allows free movements of pharynx during deglutition,
On face allows free movements of mandible , lips, & cheek during mastication & facial expressions, escapes traction & pressure during movements.
Cervical part :
Cervical part runs upwards on superior constrictor of pharynx deep to the posterior belly of digastric.
It grooves the posterior border of submandibular gland, makes S-bend [2 loops]
1st winding down over submandibular gland &
then up over the base of mandible.
Facial part:
The vessel enters the face by winding around the base of the mandible, and by piercing the deep cervical fascia,at the anteroinferior angle of the masseter muscle, here it can be palpated & is called as anaesthetist’s artery. Using contracted masseter as a landmark, pulse of facia
Carranza 2015, 12th edition, Chapter 20, The Periodontal PocketMostafa Montazeri
Carranza's Clinical Periodontology, 12th edition, Chapter 20, The Periodontal Pocket
The periodontal pocket, which is defined as a pathologically deepened gingival sulcus, is one of the most important clinical features of periodontal disease. ....
cementum /certified fixed orthodontic courses by Indian dental academy Indian dental academy
Welcome to Indian Dental Academy
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and offering a wide range of dental certified courses in different formats.
Indian dental academy has a unique training program & curriculum that provides students with exceptional clinical skills and enabling them to return to their office with high level confidence and start treating patients
State of the art comprehensive training-Faculty of world wide repute &Very affordable.
Indian Dental Academy: will be one of the most relevant and exciting training center with best faculty and flexible training programs for dental professionals who wish to advance in their dental practice,Offers certified courses in Dental implants,Orthodontics,Endodontics,Cosmetic Dentistry, Prosthetic Dentistry, Periodontics and General Dentistry.
Why do these remnants proliferate?
The origin of epithelial odontogenic neoplasms, hamartomas and cysts is inextricably bound up with a discussion of the parent cells of these lesions.
Epithelial Remnants may develop into Cysts, Tumors, and Hamartomas.
DR SWARNEET KAKPURE
THIS SEMINAR COVERS ALL ASPECTS OF GINGIVA,ITS MACROSCOPIC & MICROSCOPIC FEATURES, GINGIVAL CREVICULAR FLUID
,CLINICAL FEATURES ,GINGIVAL FIBRES,ARTERIAL SUPPLY
& NERVE SUPPLY,LYMPHATIC DRAINAGE ALONG WITH
GINGIVAL DISEASES
REFERANCE BOOK- CARANZZA TEXTBOOK OF CLINICAL PERIODONTOLOGY
The cementum is a specialised calcified substance covering the root of the tooth. The cementum is a part of the periodontium that attaches the teeth to the alveolar bone by anchoring the periodontal ligament. This presentation covers the anatomy and pathologies associated with the cementum.
Introduction
A sound knowledge of the anatomy of the periodontium and the surrounding hard and soft structures is essential to determine the scope and possibilities of surgical periodontal procedures and to minimize their risks.
Blood vessels, and nerves located in the vicinity of the periodontal surgical field, are particularly important during various surgical procedures.
Arterial Supply
Common Carotid Artery
Carotid Sinus & Carotid Body
Applied Anatomy of CCA
CAROTID PULSE :
CCA may be compressed against the carotid tubercle of transverse process of C6 vertebra about 4cm above the sternoclavicular joint.
External Carotid Artery
Generally it lies anterior to the Internal Carotid Artery.
It is the chief artery of supply to structures in the front of neck, oral cavity and in the face.
In carotid triangle
Crossed superficially by:
Cervical branch of facial nerve
Hypoglossal nerve
Facial, lingual &superior thyroid vein
Deep to artery lies:
Wall of pharynx
Superior laryngeal nerve
Ascending pharyngeal artery
Above the carotid triangle
ECA lies deep in the substance of parotid gland
Branches
Lingual Artery
Principal artery of tongue.
Arises anteromedially from ECA opposite the tip of greater cornu of hyoid bone.
Divided into three parts by hyoglossus muscle.
Applied anatomy
Sublingual artery injury occurs in premolar & molar region, when sharp instrument or rotating disks slips off a lower molar & injure the floor of mouth.
Sublingual and submental arteries may course anteriorly in close proximity to the lingual plate, and branches of these blood vessels enter accessory foramina along the lingual cortex.
Hofschneider et al (1999)
Inadvertent penetration through the lingual cortical plate into the floor of the mouth while preparing an osteotomy can cause arterial trauma, thereby resulting in development of a sublingual or submandibular hematoma
Flanagan D. et al.2003
Facial Artery
ORIGIN: Arises from the ECA just above the tip of greater cornua of hyoid bone.
COURSE:
Runs upwards in neck as cervical part ;
On face as facial part.
Tortuous course—
In neck allows free movements of pharynx during deglutition,
On face allows free movements of mandible , lips, & cheek during mastication & facial expressions, escapes traction & pressure during movements.
Cervical part :
Cervical part runs upwards on superior constrictor of pharynx deep to the posterior belly of digastric.
It grooves the posterior border of submandibular gland, makes S-bend [2 loops]
1st winding down over submandibular gland &
then up over the base of mandible.
Facial part:
The vessel enters the face by winding around the base of the mandible, and by piercing the deep cervical fascia,at the anteroinferior angle of the masseter muscle, here it can be palpated & is called as anaesthetist’s artery. Using contracted masseter as a landmark, pulse of facia
Carranza 2015, 12th edition, Chapter 20, The Periodontal PocketMostafa Montazeri
Carranza's Clinical Periodontology, 12th edition, Chapter 20, The Periodontal Pocket
The periodontal pocket, which is defined as a pathologically deepened gingival sulcus, is one of the most important clinical features of periodontal disease. ....
cementum /certified fixed orthodontic courses by Indian dental academy Indian dental academy
Welcome to Indian Dental Academy
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and offering a wide range of dental certified courses in different formats.
Indian dental academy has a unique training program & curriculum that provides students with exceptional clinical skills and enabling them to return to their office with high level confidence and start treating patients
State of the art comprehensive training-Faculty of world wide repute &Very affordable.
Indian Dental Academy: will be one of the most relevant and exciting training center with best faculty and flexible training programs for dental professionals who wish to advance in their dental practice,Offers certified courses in Dental implants,Orthodontics,Endodontics,Cosmetic Dentistry, Prosthetic Dentistry, Periodontics and General Dentistry.
Why do these remnants proliferate?
The origin of epithelial odontogenic neoplasms, hamartomas and cysts is inextricably bound up with a discussion of the parent cells of these lesions.
Epithelial Remnants may develop into Cysts, Tumors, and Hamartomas.
DR SWARNEET KAKPURE
THIS SEMINAR COVERS ALL ASPECTS OF GINGIVA,ITS MACROSCOPIC & MICROSCOPIC FEATURES, GINGIVAL CREVICULAR FLUID
,CLINICAL FEATURES ,GINGIVAL FIBRES,ARTERIAL SUPPLY
& NERVE SUPPLY,LYMPHATIC DRAINAGE ALONG WITH
GINGIVAL DISEASES
REFERANCE BOOK- CARANZZA TEXTBOOK OF CLINICAL PERIODONTOLOGY
The cementum is a specialised calcified substance covering the root of the tooth. The cementum is a part of the periodontium that attaches the teeth to the alveolar bone by anchoring the periodontal ligament. This presentation covers the anatomy and pathologies associated with the cementum.
Cementum forms a functional unit which is designed to maintain tooth support, integrity, and protection.
Minor, non-pathological resorption defects on the root surface are generally reversible and heal by reparative cementum formation.
Irreversible damage may occur when the cementum is exposed to the environment of a pocket or oral cavity.
Basic Biology of cementum and cementogenesis ( prof. olfat Gaballah ) OlfatGaballah1
Cementum is a mineralized ectomesenchymal tissue covering the entire root surface of the tooth. One of the main functions of cementum is to anchor the principal collagen
fibers of the periodontal ligament to the root surface, but it also has important adaptive
and reparative functions, playing a crucial role to maintain occlusal relationships and to
protect the integrity of the root surface. Dental cementum is unique in various aspects:
it is avascular and not innervated, does not undergo continuous remodeling like bone,
but continues to grow in thickness throughout the life.
There is accumulating histological evidence that cementum is critical for appropriate
maturation of the periodontium, both during development and as well as that
associated with the regeneration of periodontal tissues.
1. Done By : Dr.Mohamad Ghazi Kassem
2. • Enamel is an Epithelially derived hard, protective covering of teeth • Fully formed enamel is the most highly mineralized extracellular matrix known • It is highly brittle yet exhibits certain degree of resistance to withstand fracture
3. • Enamel is the hardest substance of the body, its hardness is comparable to mild steel. • Average knoop hardness number for enamel is approximately 343 • Surface of enamel is more mineralized and hard than deeper enamel. • Unlike other calcified structures in the body enamel is unique as it is totally acellular.
4. Composition of enamel Enamel consists of approximately 96% of inorganic material and 4% of organic material and water by weight. The organic component forms the matrix and the inorganic component comprises of various minerals. Percentage of dental tissue components by weight
5. The organic matrix of enamel is made from non-collagenous proteins and enzymes. Of the enamel proteins 90% are amelogenins and 10% are nonamelogenins. The different types of nonamelogenins associated with formation of enamel are ameloblastin, enamelin and tuftelin. The primary function of the organic material is to direct the growth of enamel crystals.
6. The inorganic component hydroxyapetite crystals. of enamel is comprised almost entirely of Enamel hydroxyapetite crystals are the largest hydroxyapetite crystals of all the calcified tissues in the body. In addition to hydroxyapetite crystals enamel also contains carbonates and trace elements. These crystals are susceptible to dissolution by acids and hence provides the basis for dental caries. SEM
7. Enamel is translucent and varies in colour from light yellow to whitish It varies in thickness, with maximum over cusps (2.5 mm) to a feather edge at the cervical line Thickness of enamel in primary teeth is nearly half than that in permanent teeth
8. Although enamel is an extremely hard tissue it is partially permeable to some fluids, bacteria and other products of the oral cavity The permeability of enamel is due to the presence of cracks and microscopic spaces on the surface of enamel which allows penetration of fluids The permeability of enamel decreases and hardness increases with age
9. Structure of enamel Rod and interrod enamel The fundamental units of enamel are rods and interrod enamel. The rod and interrod enamel is built from closely packed and long ribbon like hydroxyapetite crystals. The rod is shaped like a cylinder with a wide head portion, a neck and a thinner tail portion . Each rod is formed by four ameloblasts. SEM
10. ameloblasts SEM
11. Rods are formed nearly perpendicular to DEJ and curve slightly towards the cusp tip The follow a wavy course as the traverse from the DEJ to the surface of the crown The length of most rods is much longer than the thickness of enamel SEM
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
Knee anatomy and clinical tests 2024.pdfvimalpl1234
This includes all relevant anatomy and clinical tests compiled from standard textbooks, Campbell,netter etc..It is comprehensive and best suited for orthopaedicians and orthopaedic residents.
ARTIFICIAL INTELLIGENCE IN HEALTHCARE.pdfAnujkumaranit
Artificial intelligence (AI) refers to the simulation of human intelligence processes by machines, especially computer systems. It encompasses tasks such as learning, reasoning, problem-solving, perception, and language understanding. AI technologies are revolutionizing various fields, from healthcare to finance, by enabling machines to perform tasks that typically require human intelligence.
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Ethanol (CH3CH2OH), or beverage alcohol, is a two-carbon alcohol
that is rapidly distributed in the body and brain. Ethanol alters many
neurochemical systems and has rewarding and addictive properties. It
is the oldest recreational drug and likely contributes to more morbidity,
mortality, and public health costs than all illicit drugs combined. The
5th edition of the Diagnostic and Statistical Manual of Mental Disorders
(DSM-5) integrates alcohol abuse and alcohol dependence into a single
disorder called alcohol use disorder (AUD), with mild, moderate,
and severe subclassifications (American Psychiatric Association, 2013).
In the DSM-5, all types of substance abuse and dependence have been
combined into a single substance use disorder (SUD) on a continuum
from mild to severe. A diagnosis of AUD requires that at least two of
the 11 DSM-5 behaviors be present within a 12-month period (mild
AUD: 2–3 criteria; moderate AUD: 4–5 criteria; severe AUD: 6–11 criteria).
The four main behavioral effects of AUD are impaired control over
drinking, negative social consequences, risky use, and altered physiological
effects (tolerance, withdrawal). This chapter presents an overview
of the prevalence and harmful consequences of AUD in the U.S.,
the systemic nature of the disease, neurocircuitry and stages of AUD,
comorbidities, fetal alcohol spectrum disorders, genetic risk factors, and
pharmacotherapies for AUD.
3. DEFINATION
It is a mineralized dental tissue covering the anatomic roots of human teeth
4. CHARACTERISTICS
• Calcified structure whose calcification & hardness is less than dentin
• More permeable than dentin
• Light yellow in colour
• Softer and lighter dentin
• Lack luster and is dark therefore differentiated from enamel
• Less readily resorbed than bone
5. LOCATION
Begins at the cervical portion of the tooth at the cemento enamel junction
& continues to the apex
Radicular cementum ( found on to the root surface )
Coronal cementum ( forms on the enamel covering the crown )
6. THICKNESS
Coronal half of root 16-60 microns
apical third & furcation areas 150-200 microns
An average thickness of 95 microns {20years} & 215 microns {65 years}
Between 11-70 year thickness increases 3 times
Cemental deposition continues through out the life
7. CHEMICAL COMPOSITION
• Organic content & water - 50-55%
• Inorganic content - 45-55%
ORGANIC CONTENT
• Type I collagen fibres 90%
• Type III collagen fibres 5 %
• Proteins
• Polysaccrides
• Trace elements
9. SOURCE OF COLLAGEN FIBRES
• IN CEMENTUM
Extrinsic sharpey’s fibre formed by fibroblasts
derived from PDL’s
run in the same direction pdl principal, perpendicular/obligue to the root surface
Intrinsic fibres of cementum matrix formed by cementoblasts
derived from cementum
run parallel to the root surface & at right angles to the extrinsic fibres
10. CEMENTOGENESIS
It takes place in two phases
Matrix formation
Mineralization
There are three cell types responsible for the cementogenesis
Cementoblasts
Cementocytes
Fibroblasts
All these cells are derived from the ectomesenchymal cells
11. • Pre functional phase
during root development
time range 3.75 years – 7.75 years
• Fuctional phase
commences when tooth reaches the occulusal plane continues through out the life
• MATRIX FORMATION
12. Enamel organ reached its final stage
Inner & outer enamel epithelium
proliferate to form HERS
Continues mitotic cell activity at
apical termination of HERS
Coronoapical growth of double
layer sheath
Most apical portion seprates the
dental papilla from dental follicle
13. Inner & outer cell layers of HERS are surrounded
by basement membrane
Cells from dental papilla in the radicular pulp
differentiate into odontoblasts & forms radicular
mantle dentin
HERS fragment forms a network
Network enables mesenchymal cells to pass between
cells of root
These mesenchymal cells are cementoblasts
15. MINERALIZATION
• Mineralization begins at the depth of pre cementum
• Fine hydroxyapatite crystals are deposited, first between & then within the
collagen fibrils by a process that is identical to the mineralization of bone
tissue
• The width of the precemntum layer is about 3.5 mm
• Linear rate of cementum deposition on single-rooted teeth is about 3
um/year
16.
17.
18. Propose activity
• Adhesion/chemoattractant
Mitogenesis
Differentiation
Mineralization
Molecular factors affected
• Osteopontin, bone sialoprotein, laminin(epithelial
factors), Fibronectin, collage I,II XII, proteoglycans
• Growth harmones,transforming growth factor B, insulin
like growth factor I
• Bone morphogenic protein-3, ameloblastin
• Osteopontin , bone sialoprotein, osteocalcin(controls
mineralization prevents ankyloses) ,proteoglycans,
Collagen type I & XII
19. CLASSIFICATION
• On the basis of
• presence/ absence of cells
• time of formation
• location
• presence/absence of fibres
• Origion of fibres
• Schroeder’s classification
24. CEMENTOENAMEL JUNCTION
• the area where enamel and cementum meet at the cervical region of
the tooth.
• Three different relationships among the enamel and cementum:
• 60% to 65% of the cases the cementum overlaps the enamel
• 30% of the cases edge to edge
• 5% to 10% cementum fails to meet enamel resulting in exposed dentin
25. It is the point at which
cementum and enamel meet.
May be of three types.
In some rare cases, a fourth type of
cemento-enamel junction is seen.
In these rare cases, the enamel overlaps the
cementum.
26. FUNCTIONS OF CEMNTUM
• provide anchorage of tooth to alveolus (sharpeys fibers).
• It assists in maintaining occlusal relation by maintaining a balance between
attrition and eruption.
• It serves to maintain the width of PDL space at the apex.
• Cementum repairs root fracture.
• No resorption under masticatory or orthodontic forces so maintains tooth
integrity and fulfils orthodontic requirement.
27. CEMENTAL RESORPTION
Can occur due to physiologic or pathologic
causes
Local causes: Trauma from occlusion,
orthodontic movement, cysts, tumors.
Systemic causes: Calcium deficiency,
Hypothyroidism, Pagets disease.
• MICROSCOPICALLY: Bay like concavities in the root surface
28. AGE CHANGES IN CEMENTUM
CONTINOUS DEPOSITION
• Forms on roots throughout life
• More apically than cervically
• Reduces root surface concavities thicker layer in root surface
grooves and in furcations.
• Variation in tooth position influence pattern of deposition
29. PATHOLOGICAL CONDITIONS
ASSOCIATED WITH CEMENTUM
ANKYLOSIS
• Fusion of cementum and alveolar bone
withobliterated PDL
• Occurs in teeth with cemental resorption
• After periodontal inflammation, tooth
replantation, occlusal trauma.
• Resorption of root and its gradual replacement by
bone
• Lack physiological mobility, metallic percussion
• No proprioception
30. CEMENTICLES
• Abnormal, calcified bodies in the periodontal
ligament
• Form from remnants of HERS
• Usually ovoid or round
• Size ranges from 0.1- 0.4 mm.
• Classified as Free, Attached or Embedded
• Local trauma
• Appear in increasing numbers in the aging
person
31. HYPERCEMENTOSIS
• Hypercementosis is a nonneoplastic
deposition of excessive
• Cementum that is continuous with
the normal radicular cementum.
LOCAL FACTORS
Abnormal occlusal trauma
Adjacent inflammation
Unopposed teeth [e.g.,
impacted, embedded, without
antagonist)
32. SYSTEMIC FACTORS
• Neoplastic and non neoplastic conditions including benign cementoblastoma,
• cementifying fibroma,
• cemental dysplasia
• Acromegaly and pituitary gigantism
• Paget's disease of bone
• Rheumatic fever
• Thyroid goiter
33. CONCRESCENCE
• Fusion of teeth by fusion of cementum
• After root formation has been completed
• Traumatic injury or crowding of teeth with
• resorption of the interdental bone
• Difficulty in extraction
34. REGRESSIVE ALTERATION OF
TEETH
• Abrasion
• Abrasion is the pathologic wearing of tooth substance through some
• abnormal mechanical process.
• Abrasion usually occurs on the exposed root surfaces of teeth, but under
some circumstances, it may be seen elsewhere on tooth
• Abrasion caused by dentrifrice manifests as a v-shaped or wedge shaped
ditch on the root side of cej in teeth with recession.
35. NEOPLASMS ASSOCIATED WITH
CEMENTUM
• CEMENTOBLASTOMA
The benign cementoblastoma is probably a true neoplasm of functional
cementoblasts which form a large mass of cementum or cementum-like tissue on
the tooth root
• CEMENTIFYING FIBROMA
Resemble focal cemento-osseous dysplasia
• The neoplasm is composed of fibrous tissue that contains a variable mixture of
bony trabeculae, cementum like spherules or both.
• origin of these tumors is odontogenic or from periodontal ligament.
36. • PAGETS DISEASE
Paget’s disease is characterized by enhanced resorption of bone.
Etiology: unknown, viral infection, inflammatory cause, autoimmune, connective
tissue and vascular disorder.
• HYPOPHOSPHATASIA
Hypophosphatasia is a rare metabolic bone disease that is characterized by a
deficiency oftissue-nonspecific alkaline phosphatase.
• One of the first presenting signs ofhypophosphatasia may be the premature lossof
the primary teeth presumably caused by alack of cementum on the root surfaces
37. • HYPERPITUITARISM
Gigantism is the childhood version of growth hormone excess and is
characterized by the general symmetrical overgrowth of the body parts.
• Prognathic mandible, frontal bossing, dental malocclusion, andinterdental
spacing are the other features.
• Intraoral radiograph may show hypercementosis of the roots
38. • Acromegaly is characterized by an acquired progressive somatic
disfigurement, mainly involving the face and extremities, but also many other
organs, that are associated with systemic manifestations.
• Dental radiograph may demonstrate large pulp chambers and excessive
deposition of cementum on the roots