SlideShare a Scribd company logo
SPINAL CORD INJURY
MANOJ M.G
MANOJ M.G.
LECTURER (MEDICAL SURGICAL
NURSING)
DEPARTMENT OF NURSING AND
MIDWIFERY
WOLLEGA UNIVERSITY
DEFINITION
•Spinal cord injury (SCI) is damage to the
spinal cord that results in a loss of function
such as mobility or feeling.
Common locations of spinal cord injuries
•Flexion-extension injuries are commonly
located at C4 - C7.
• T11, T12, and L1 are frequent sites of spinal
cord injury resulting rom falls.
Mechanisms of spinal cord injury.
•Flexion-extension: whiplash(is a non-medical term
describing a range of injuries to the neck caused
by or related to a sudden distortion of the neck,
associated with extension)
• Subluxation: incomplete or partial dislocation.
•Torsion: twisting of the spinal cord.
•Compression.
Mechanism of Injury
5
Different
mechanism
of injury
RISK FACTORS
Gender - Spinal cord injury affects a
disproportionate amount of men
Age – (Young adults and seniors)
- Between ages 16 and 35 / MVA leading cause
- Another peak in people older than 60 / falls leading
cause
People active in sports – High risk athletic
activities include football, rugby, wrestling,
gymnastics, diving, surfing, ice hockey and
downhill skiing
Predisposing conditions - A relatively minor
injury can cause spinal cord injury in people with
conditions that affect their bones or joints, such
as arthritis or osteoporosis
causes
•Road Traffic Accident
•Sports injury
•Fall
•Bullet or stab wound
•Traumatic injury
•Electric shock
•Extreme twisting of the middle of the body
•Landing on the head during a sports injury
•Fall from a great height
Types of Spinal Cord Injury
•Complete Spinal Cord Injuries
• Complete paraplegia is described as
permanent loss of motor and nerve function
at T1 level or below, resulting in loss of
sensation and movement in the legs, bowel,
bladder, and sexual region. Arms and hands
retain normal function.
•Complete loss of motor function and
sensation below the area of injury
•Even in a complete injury, the spinal cord is
almost never completely cut in half. Doctors
use the term "complete" to describe a large
amount of damage to the spinal cord.
•many people with partial spinal cord injuries
are able to experience significant recovery.
While those with complete injuries are not .
Incomplete / Partial spinal cord injury
Spinal cord is able to convey some messages
to or from the brain. Therefore, retain some
sensation and possibly some motor function
below the affected area
• Complete or Partial ?
Incomplete / Partial Spinal Cord Injury
•Central
•Lateral / Brown-Sequard Syndrome
•Anterior
•Posterior
14
The articulated spine. A, Anterior
view. B, Posterior view. C, Right lateral view
•Anterior Cord Syndrome
Damage of front 2/3 of spinal cord, loss
of pain and temperature sensation, and
motor function below level of injury
Light touch (pressure) and position and
vibration sensation preserved
Possible for some people with this injury
to later recover some movement
16
•Central Cord Syndrome
Usually with unbelted Motor Vehicle
Accidents and falls of elderly
Typically results greater weakness in arms vs
lower extremities
Sensory loss varies but more severe in upper
extremities
Control over the bowel and bladder varies and
may be preserved
Possible for some recovery from this type of
injury, usually starting in the legs, gradually
progressing upwards
18
Central cord syndrome
•Brown-Sequard Syndrome/lateral
Usually stab or Gun Shot Wound
Damage is towards one side of the
spinal cord
Ipsilateral (same side as the cord
injury) Impaired or loss of movement,
touch, pressure and vibration (Hemi
paraplegia)
Contralateral (opposite side of cord
injury) loss of pain and temperature
sensation (Hemiparasthesia)
20
•Posterior Cord Syndrome
Damage is towards the back of the
spinal cord
May leave the person with good
muscle power, pain and temperature
sensation
However they may experience
difficulty coordinating movement of
their limbs
22
•Cauda Equina Syndrome:
Due to bony compression or disc
protrusions in lumbar or sacral region
Non specific symptoms – back pain
 - bowel and bladder dysfunction
 - leg numbness and weakness
 - saddle paresthesia(loss of sensation
restricted to the area of buttocks, perineum
and inner surface of the thighs)
Spinal Cord Injuries Causal Categories
Traumatic spinal cord injury may stem
from:
• Sudden, traumatic blow that fractures,
dislocates, crushes or compresses one or
more of vertebrae
• Gunshot or knife wound that penetrates and
cuts your spinal cord
• Additional (secondary) damage usually
occurs over days or weeks because of
bleeding, swelling, inflammation and fluid
accumulation in and around spinal cord
Non-traumatic spinal cord injury may be
caused by :
• Arthritis
• Cancer
• Blood vessel problems or bleeding
• Inflammation or infections
• Disk degeneration of the spine
Primary Spinal Injury
•Result of initial
trauma
•Injury usually
permanent
Secondary Spinal Injury
•Occurs after Spinal cord trauma
•Damage at cellular level
•Necrosis (Cells swell, burst and leak
toxic substances to other cells)
• Apoptosis (Programmed cell death /
cell suicide to prevent bursting)
28
Secondary SCI
29
Secondary SCI
30
PATHOPHYSIOLOGY
Due to the etiological factor
Damage to the cord may be a concussion, contusion, laceration,
compression, or complete, incomplete of the cord.
Cord's response to injury includes hemorrhage, ischemia, and edema
SCI Involves loss of:
Motor function
Sensory function
Reflexes
Control of elimination
CLINICAL MANIFESTATIONS
•Patients with tetraplegia (formerly called
quadriplegia) have damage to the cervical
segments of nerves (C1-C8) in the spinal canal.
Function may be impaired in the upper
extremities, trunk, pelvic organs, and lower
extremities.
•Patients with paraplegia have damage to the
thoracic, lumbar, or sacral segments of nerves
in the spinal cord. The arms are unaffected, but
function may be impaired in the trunk, pelvic
organs, and lower extremities.
•Sacral sensation is intact if there is deep sensation
and sensation at the anal mucocutaneous junction;
sacral motor is intact if the patient has voluntary
contraction of the external anal sphincter with
digital stimulation.
•The Zone of Partial Preservation (ZPP) indicates
areas of partial sensory/motor innervation below
the Level Of Injurie; the ZPP is applicable only to
complete injuries.
•The neurologic level of injury is the lowest neural
level with normal sensory and motor function on
both sides of the body. When describing the level
of involvement, the neurologic level is noted
unless stated specifically that the skeletal level of
involvement, which is the level of greatest
vertebral damage, is being discussed.
•Various syndromes (incomplete injuries) may
characterize the clinical presentation
• Patient's symptoms will mirror the level of the
cord injury.
•There will be total sensory loss and motor
paralysis below level of the injury.
• Cervical spinal cord injuries will produce
quadriplegia--loss of function of all four
extremities.
• Injuries to the thoracic spinal cord below the level
of T1 will produce paraplegia--paralysis of the
lower extremities.
• Loss of bowel and bladder control; usually urinary
retention and bladder distention.
•Loss of sweating and vasomotor tone below
the level of the cord injury.
•Marked reduction of blood pressure due to
loss of peripheral vascular resistance.
•Neck/back pain.
•Priapism--persistent, painful erection of the
penis.
CERVICAL (NECK) INJURIES
•Breathing difficulties
•Loss of normal bowel and bladder control
•Numbness
•Sensory changes
•Spasticity (increased muscle tone)
THORACIC (CHEST LEVEL) INJURIES
•Loss of normal bowel and bladder control
•Numbness
•Sensory changes
•Spasticity (increased muscle tone)
•Weakness, paralysis
LUMBAR SACRAL (LOWER BACK)
INJURIES
•Loss of normal bowel and bladder control (you may have
constipation, leakage, and bladder spasms)
•Numbness
•Pain
•Sensory changes
•Weakness and paralysis
DIAGNOSTIC TESTS
•Complete blood count (e.g. Hb, RBC, WBC)
•Arterial blood gas level
•X ray
•CT scan and MRI
X- rays:
Computerized tomography (CT) scans
Magnetic resonance imaging (MRI):
Myelography:
MANAGEMENT
•Requires a multidisciplinary approach because of
multiple systems involvement and the
psychosocial aspects of catastrophic injuries.
Immediately After Trauma (Less Than 1
Hour)
•Immobilization with rigid cervical collar,
sandbags, and rigid spine board to transport from
the field to acute care facility.
48
Acute Phase (1 to 24 Hours)
•Maintenance of pulmonary and cardiovascular
stability.
•Intubation and mechanical ventilation, if needed.
•Vasopressors to maintain adequate perfusion to
sustain mean arterial BP at 85 to 90 mm Hg.
•Medical stabilization before spinal stabilization and
decompression.
•Spinal cord immobilization use of skeletal
tongs.
•Crutchfield and Vinke tongs( used for
immubilization) require predrilled holes in the
skull under local anesthesia; Gardner-Wells and
Heifitz tongs do not.
•Weight is added to traction gradually to reduce the
vertebral fracture; weight maintained at a level to
ensure vertebral alignment.
•Rigid kinetic turning bed to immobilize patients
with thoracic and lumbar injuries.
•Surgical interventions are considered when the
patient has vertebral instability that may result in
further neurologic damage; an injury that is
incomplete at onset may become complete if
instability exists. The objectives are to remove all
of the bony and soft tissues that are compressing
the spinal cord, thereby minimizing the possibility
of a deteriorating neurologic status, and stabilize
the vertebra surrounding the spinal cord so that
rehabilitation may begin as soon as possible.
•Decompression, typically using the anterior
approach in cervical instances, may be
accomplished by removing the bony structures
and soft tissues (eg, fusion, decompression
laminectomy). Realignment of the soft tissues
and vertebral column is required.
•Stabilization, typically done using the posterior
approach, involves the use of wires, bone grafts,
plates, screws, and other fixation devices to
prevent movement at the damaged bony site (eg,
fusion, Harrington rods). Harrington rods, used
for thoracolumbar SCI, extend approximately
one to three levels above and below the fracture.
•Methylprednisolone sodium succinate( to
improve neurological recovery) should be
administered within 8 hours of injury.
•Bolus 30 mg/kg administered over 15 minutes;
maintenance infusion of 5.4 mg/kg/hr infused for 23
hours
•Additional benefit may be achieved by
administering the maintenance dose for 48 hours.
•Management of neurogenic bladder( dysfunction
caused by neurologic damage, symptoms may
include over flow, urgency, frequency) Foley
catheter
•Pressure ulcer prevention pressure reduction
mattress or kinetic turning frame.
Sub acute Phase (Within 1 Week)
•Halo traction is the primary treatment for cervical
injuries
•ganglioside sodium salt I.V., begun within 72
hours after injury, and continued for 18 to 32 days,
is believed to enhance neuronal regeneration.
•H2-receptor blockers to prevent gastric irritation
and haemorrhage.
•Early mobilization and passive exercise as soon as
patient is surgically and medically stable.
•Hyper alimentation( intravenous supply of
nutrients) to retard negative nitrogen balance.
•Interventions to prevent thromboembolism
(intermediate risk) are based on motor
completeness of injury:
•Motor Incomplete ,compression hose; compression
boots in addition to unfractionated heparin (UH) 5,000
units every 12 hours.
•Motor Complete compression hose; compression boots
in addition to UH with the dosage adjusted to high
normal or low-molecular-weight heparin 30 mg bid.
•If the patient is at high risk (ie, motor complete with
other risk factors such as fractures of lower extremities
or previous DVT), an inferior vena cava filter should be
considered.
Chronic Phase (Beyond 1 Week)
•Harrington rods, used in conjunction with a body
jackets, are used for patients with thoracolumbar
injuries.
•To prevent thrombophlebitis in the chronic phase,
compression boots should be continued for 2
weeks; anticoagulants should also be continued
based on motor completeness of injury:
•Motor Incomplete until discharged from hospital (or 8
weeks
•Motor Complete 8 weeks.
•If the patient is at high risk, anticoagulants should be
continued for 12 weeks or until discharged from
hospital.
•Management of complications may include
treatment of infections with antibiotics; treatment
of respiratory compromise with phrenic nerve
pacing, mechanical ventilation, and other methods;
pressure ulcer treatment; management of
heterotopic ossification (is the presence of bone in
soft tissue where bone normally does not exist)
with calcium chelators( which bind calcium) and
anti-inflammatory agents; drainage of
syringomyelia( cyst in spinal cord); management
of spasticity with oral or intrathecal
antispasmodics, surgical procedure, or spinal cord
stimulation; and management of central
neuropathic pain with anticonvulsants, minor
sedatives, antidepressants, nerve block, or surgical
procedure.
•Spasticity should be managed by:
•Maintaining calm, stress-free environment.
•Allowing ample time for activities such as
positioning and transferring.
•Performing joint ROM exercises with slow,
smooth movements.
•Avoiding temperature extremes.
•Administering muscle relaxants, such as
baclofen (Lioresal) (via pump or orally),
diazepam (Valium), and dantrolene (Dantrium),
as prescribed.
•External sphincterotomy may be used for detrusor-
sphincter dyssynergia (Insufficient relaxation of the
sphincter during a voiding contraction prevents effective
bladder emptying and can lead to high pressures in the
bladder) Other options include urethral stents and balloon
dilatation.
•Clonidine has been used to manage spasticity and
facilitate ambulation in patients with incomplete injuries.
•Resistive inspiratory muscles training shows promise in
promoting respiratory muscle strength and reducing sleep-
induced breathing disturbances in patients with
tetraplegia. Resistive training devices are used to perform
respiratory maneuvers at scheduled times during the day.
•Rehabilitation includes medical and psychosocial support,
physical therapy, urologic evaluation, occupational
therapy, and multiple other interventions to facilitate an
increased level of function and community participation.
Nursing management:
Nursing Assessment
•Assess cardiopulmonary status and vital signs to
help determine degree of autonomic dysfunction,
especially in patients with tetraplegia.
•Determine LOC and cognitive function indicating
Traumatic Brain Injuries or other pathology.
•Perform frequent motor and sensory assessment of
trunk and extremities extent of deficits may
increase due to oedema and haemorrhage. And it
may increase the neurologic deficit and pain.
•Note signs and symptoms of spinal shock, such as
flaccid paralysis, urine retention, absent reflexes.
•Assess bowel and bladder function.
•Assess quality, location, severity of pain.
•Perform psychosocial assessment to evaluate
motivation, support network, financial or other
problems.
•Assess for indicators of powerlessness, including
verbal expression of no control over situation,
depression, nonparticipation, dependence on
others, passivity.
Nursing Diagnoses
•Ineffective Breathing Pattern related to paralysis of
respiratory muscles or diaphragm
•Impaired Physical Mobility related to motor
dysfunction
•Risk for Impaired Skin Integrity related to
immobility and sensory deficit
•Urinary Retention related to neurogenic bladder
•Constipation or Bowel Incontinence related to
neurogenic bowel
•Risk for Injury: autonomic dysreflexia (is a
syndrome in which there is a sudden onset of
excessively high blood pressure) and orthostatic
hypotension ( is defined as a decrease in
systolic blood pressure of 20 mm Hg or a
decrease in diastolic blood pressure of 10 mm Hg
within three minutes of standing when compared
with blood pressure from the sitting or supine
position)
•Powerlessness related to loss of function, long
rehabilitation, depression
•Sexual Dysfunction related to erectile dysfunction
and fertility changes
•Chronic Pain related to neurogenic changes
Nursing Management
Objectives of care:
•Reduce the fracture/dislocation and obtain
immobilization of the spine as soon as possible
to prevent further cord damage.
•Observe for symptoms of progressive
neurological damage.
Patient with cervical spine injury will have
some form of skeletal traction. Maintain
traction and provide nursing care local policy.
Continuously observe patient's breathing pattern.
•Patients with injuries at high levels are at risk for
respiratory failure.
•Observe strength of cough effort.
Be alert for signs of spinal shock ( is the temporary
reduction of or loss of reflexes following a spinal
cord injury (SCI) )and report immediately.
•(Spinal shock represents a sudden loss of continuity
between the spinal cord and higher nerve centers.
•It is characterized by a complete loss of motor,
sensory, reflex, and autonomic activity below the
level of the injury.
•Though temporary, spinal shock may last for several
weeks.
Continuously observe patient for motor and
sensory changes due to cord edema or
hemorrhage, which may further compromise
cord function.
•Test patient's motor ability by asking him/her to
spread fingers, grip your hands, shrug
shoulders, etc.
•Test sensory level by gently pinching the skin at
shoulders and progressing down sides; ascertain
level at which patient can no longer feel pinch.
•Note presence/absence of sweating.
•Carefully record findings in patient's clinical
record; report changes in patient's
motor/sensory level immediately to professional
nurse.
If turning is allowed and patient is not on a turning
frame or turning bed, the patient must be carefully log-
rolled with the spine maintained in alignment.
Surgery, depending upon the injury and pathological
findings, may have to be performed to stabilize the
spine before rehabilitation can begin.
Patient will require passive range of motion exercises.
Assist with active rehabilitation procedures when
patient is stable.
•Program is designed according to neurological deficit.
•Usually involves 6 weeks of gradual mobilization with
brace or cast, depending upon level of injury.
Provide constant encouragement and psychological
support to the patient with a spinal cord injury.
Possible Complications
•Blood pressure changes - can be extreme
(autonomic hyper reflexia)
•Chronic kidney disease
•Complications of immobility:
Deep vein thrombosis
Pulmonary infections
Skin breakdown
•Contractures: A muscle contracture is a
permanent shortening of a muscle or joint
•Increased risk of urinary tract infections
•Loss of bladder control
•Loss of bowel control
•Loss of sensation
•Loss of sexual functioning (male impotence)
•Muscle spasticity
•Paralysis of breathing muscles
•Paralysis (paraplegia, quadriplegia)
•Pressure sores
•Shock
REHABILITATION
•Cognitive Rehabilitation Therapy
•Speech Therapy
•Mental Rehabilitation
•Physical Exercise
•Occupational Therapy
THANK YOU

More Related Content

Similar to 4. CNS Spinal cord injury.pptx

Spinal Injury Trauma.pptx
Spinal Injury Trauma.pptxSpinal Injury Trauma.pptx
Spinal Injury Trauma.pptx
CHANDAN PADHAN
 
Spinal Injury Trauma.pptx
Spinal Injury Trauma.pptxSpinal Injury Trauma.pptx
Spinal Injury Trauma.pptx
CHANDAN PADHAN
 
spinal Trauma.ppt
spinal Trauma.pptspinal Trauma.ppt
spinal Trauma.ppt
mhmodsaad2
 
Traumatic paraplegia & bladder management by dr ashutosh
Traumatic paraplegia & bladder management by dr ashutoshTraumatic paraplegia & bladder management by dr ashutosh
Traumatic paraplegia & bladder management by dr ashutosh
Ashutosh Kumar
 
Traumatic Paraplegia
Traumatic ParaplegiaTraumatic Paraplegia
Traumatic Paraplegia
Sri Harsha Gutta
 
Spinal Cord Injury 1
Spinal Cord Injury 1Spinal Cord Injury 1
Spinal Cord Injury 1
Dr. Dinu Dixon (P.T)
 
Spinal cord injury
Spinal cord injurySpinal cord injury
Spinal cord injury
Zahid Khan
 
Cervical spine injury
Cervical spine injuryCervical spine injury
Cervical spine injury
manu tiwari
 
Spinal cord injury
Spinal cord injurySpinal cord injury
Spinal cord injury
Shweta Sharma
 
Cervical spine trauma and spinal cord injuries by Dr Shamavu Gabriel.pptx
Cervical spine trauma and spinal cord injuries by Dr Shamavu Gabriel.pptxCervical spine trauma and spinal cord injuries by Dr Shamavu Gabriel.pptx
Cervical spine trauma and spinal cord injuries by Dr Shamavu Gabriel.pptx
Gabriel Shamavu
 
management of spinal cord injuries
 management of spinal cord injuries management of spinal cord injuries
management of spinal cord injuries
Balqees Majali
 
Spinal cord injury (SCI)
Spinal cord injury (SCI)Spinal cord injury (SCI)
Spinal cord injury (SCI)
Sachin Dwivedi
 
spinal cord injury
spinal cord injuryspinal cord injury
spinal cord injury
HezekiahAyuba1
 
Spinal cord injury 2012 intern
Spinal cord injury 2012 internSpinal cord injury 2012 intern
Spinal cord injury 2012 intern
Pierre Lopez
 
Spinal cord injuries
Spinal cord injuries Spinal cord injuries
Spinal cord injuries
Mostafa Shakshak
 
Traumatic spinal cord injury
Traumatic spinal cord injuryTraumatic spinal cord injury
Traumatic spinal cord injury
Jebaraj Fletcher
 
Non Traumatic Spinal cord injuries
Non Traumatic Spinal cord injuries   Non Traumatic Spinal cord injuries
Non Traumatic Spinal cord injuries
Dr. Muzahid
 
Spine and extermity injury.pptx
Spine and extermity injury.pptxSpine and extermity injury.pptx
Spine and extermity injury.pptx
kalilinux24
 
Spinal trauma wo anatomy
Spinal trauma wo anatomySpinal trauma wo anatomy
Spinal trauma wo anatomy
haron taufiq
 
Diseases of the spinal cord.pptx
Diseases of the spinal cord.pptxDiseases of the spinal cord.pptx
Diseases of the spinal cord.pptx
ssuser9f80d7
 

Similar to 4. CNS Spinal cord injury.pptx (20)

Spinal Injury Trauma.pptx
Spinal Injury Trauma.pptxSpinal Injury Trauma.pptx
Spinal Injury Trauma.pptx
 
Spinal Injury Trauma.pptx
Spinal Injury Trauma.pptxSpinal Injury Trauma.pptx
Spinal Injury Trauma.pptx
 
spinal Trauma.ppt
spinal Trauma.pptspinal Trauma.ppt
spinal Trauma.ppt
 
Traumatic paraplegia & bladder management by dr ashutosh
Traumatic paraplegia & bladder management by dr ashutoshTraumatic paraplegia & bladder management by dr ashutosh
Traumatic paraplegia & bladder management by dr ashutosh
 
Traumatic Paraplegia
Traumatic ParaplegiaTraumatic Paraplegia
Traumatic Paraplegia
 
Spinal Cord Injury 1
Spinal Cord Injury 1Spinal Cord Injury 1
Spinal Cord Injury 1
 
Spinal cord injury
Spinal cord injurySpinal cord injury
Spinal cord injury
 
Cervical spine injury
Cervical spine injuryCervical spine injury
Cervical spine injury
 
Spinal cord injury
Spinal cord injurySpinal cord injury
Spinal cord injury
 
Cervical spine trauma and spinal cord injuries by Dr Shamavu Gabriel.pptx
Cervical spine trauma and spinal cord injuries by Dr Shamavu Gabriel.pptxCervical spine trauma and spinal cord injuries by Dr Shamavu Gabriel.pptx
Cervical spine trauma and spinal cord injuries by Dr Shamavu Gabriel.pptx
 
management of spinal cord injuries
 management of spinal cord injuries management of spinal cord injuries
management of spinal cord injuries
 
Spinal cord injury (SCI)
Spinal cord injury (SCI)Spinal cord injury (SCI)
Spinal cord injury (SCI)
 
spinal cord injury
spinal cord injuryspinal cord injury
spinal cord injury
 
Spinal cord injury 2012 intern
Spinal cord injury 2012 internSpinal cord injury 2012 intern
Spinal cord injury 2012 intern
 
Spinal cord injuries
Spinal cord injuries Spinal cord injuries
Spinal cord injuries
 
Traumatic spinal cord injury
Traumatic spinal cord injuryTraumatic spinal cord injury
Traumatic spinal cord injury
 
Non Traumatic Spinal cord injuries
Non Traumatic Spinal cord injuries   Non Traumatic Spinal cord injuries
Non Traumatic Spinal cord injuries
 
Spine and extermity injury.pptx
Spine and extermity injury.pptxSpine and extermity injury.pptx
Spine and extermity injury.pptx
 
Spinal trauma wo anatomy
Spinal trauma wo anatomySpinal trauma wo anatomy
Spinal trauma wo anatomy
 
Diseases of the spinal cord.pptx
Diseases of the spinal cord.pptxDiseases of the spinal cord.pptx
Diseases of the spinal cord.pptx
 

More from NimonaAAyele

PE afe.ppt
PE afe.pptPE afe.ppt
PE afe.ppt
NimonaAAyele
 
Infection Control in the Emergency Room presentation.ppt
Infection Control in the Emergency Room presentation.pptInfection Control in the Emergency Room presentation.ppt
Infection Control in the Emergency Room presentation.ppt
NimonaAAyele
 
Acute Ear Problem.pptx
Acute Ear Problem.pptxAcute Ear Problem.pptx
Acute Ear Problem.pptx
NimonaAAyele
 
2023 MSS.ppt
2023 MSS.ppt2023 MSS.ppt
2023 MSS.ppt
NimonaAAyele
 
2023 Chronic Respiratory Problems.pptx
2023 Chronic Respiratory Problems.pptx2023 Chronic Respiratory Problems.pptx
2023 Chronic Respiratory Problems.pptx
NimonaAAyele
 
2023 Gastro intestinal system problems.pptx
2023 Gastro intestinal system problems.pptx2023 Gastro intestinal system problems.pptx
2023 Gastro intestinal system problems.pptx
NimonaAAyele
 
2023 Diabetes Mellitus.pptx
2023 Diabetes Mellitus.pptx2023 Diabetes Mellitus.pptx
2023 Diabetes Mellitus.pptx
NimonaAAyele
 
Gastro intestinal cancer by Azu and Dere (1) (4).pptx
Gastro intestinal cancer by Azu and Dere (1) (4).pptxGastro intestinal cancer by Azu and Dere (1) (4).pptx
Gastro intestinal cancer by Azu and Dere (1) (4).pptx
NimonaAAyele
 
Immunodeficiency dis order [Repaired] FINAL.pptx
Immunodeficiency dis order [Repaired] FINAL.pptxImmunodeficiency dis order [Repaired] FINAL.pptx
Immunodeficiency dis order [Repaired] FINAL.pptx
NimonaAAyele
 
WORKU and YADETA AHN-II Group Assignment.pptx
WORKU and YADETA AHN-II Group Assignment.pptxWORKU and YADETA AHN-II Group Assignment.pptx
WORKU and YADETA AHN-II Group Assignment.pptx
NimonaAAyele
 
teeth disorders seminar presentation.pptx
teeth disorders seminar presentation.pptxteeth disorders seminar presentation.pptx
teeth disorders seminar presentation.pptx
NimonaAAyele
 
Thyroid Disorder.pptx
Thyroid Disorder.pptxThyroid Disorder.pptx
Thyroid Disorder.pptx
NimonaAAyele
 
pitutiary disorder final.pptx
pitutiary disorder  final.pptxpitutiary disorder  final.pptx
pitutiary disorder final.pptx
NimonaAAyele
 
Edited Assignment of Adrenal Disorders (1)(1).pptx
Edited Assignment of Adrenal Disorders (1)(1).pptxEdited Assignment of Adrenal Disorders (1)(1).pptx
Edited Assignment of Adrenal Disorders (1)(1).pptx
NimonaAAyele
 
CHRONIC LIVER DISEASE.pptx
CHRONIC LIVER DISEASE.pptxCHRONIC LIVER DISEASE.pptx
CHRONIC LIVER DISEASE.pptx
NimonaAAyele
 
BONE TUMORS.pptx
BONE TUMORS.pptxBONE TUMORS.pptx
BONE TUMORS.pptx
NimonaAAyele
 
1. Medical Helminthology.ppt
1. Medical Helminthology.ppt1. Medical Helminthology.ppt
1. Medical Helminthology.ppt
NimonaAAyele
 
25 gall bladder and pancrease.pdf
25 gall bladder and pancrease.pdf25 gall bladder and pancrease.pdf
25 gall bladder and pancrease.pdf
NimonaAAyele
 
endocrine lecture.pdf
endocrine lecture.pdfendocrine lecture.pdf
endocrine lecture.pdf
NimonaAAyele
 
8- Seizure disorder.ppt
8- Seizure disorder.ppt8- Seizure disorder.ppt
8- Seizure disorder.ppt
NimonaAAyele
 

More from NimonaAAyele (20)

PE afe.ppt
PE afe.pptPE afe.ppt
PE afe.ppt
 
Infection Control in the Emergency Room presentation.ppt
Infection Control in the Emergency Room presentation.pptInfection Control in the Emergency Room presentation.ppt
Infection Control in the Emergency Room presentation.ppt
 
Acute Ear Problem.pptx
Acute Ear Problem.pptxAcute Ear Problem.pptx
Acute Ear Problem.pptx
 
2023 MSS.ppt
2023 MSS.ppt2023 MSS.ppt
2023 MSS.ppt
 
2023 Chronic Respiratory Problems.pptx
2023 Chronic Respiratory Problems.pptx2023 Chronic Respiratory Problems.pptx
2023 Chronic Respiratory Problems.pptx
 
2023 Gastro intestinal system problems.pptx
2023 Gastro intestinal system problems.pptx2023 Gastro intestinal system problems.pptx
2023 Gastro intestinal system problems.pptx
 
2023 Diabetes Mellitus.pptx
2023 Diabetes Mellitus.pptx2023 Diabetes Mellitus.pptx
2023 Diabetes Mellitus.pptx
 
Gastro intestinal cancer by Azu and Dere (1) (4).pptx
Gastro intestinal cancer by Azu and Dere (1) (4).pptxGastro intestinal cancer by Azu and Dere (1) (4).pptx
Gastro intestinal cancer by Azu and Dere (1) (4).pptx
 
Immunodeficiency dis order [Repaired] FINAL.pptx
Immunodeficiency dis order [Repaired] FINAL.pptxImmunodeficiency dis order [Repaired] FINAL.pptx
Immunodeficiency dis order [Repaired] FINAL.pptx
 
WORKU and YADETA AHN-II Group Assignment.pptx
WORKU and YADETA AHN-II Group Assignment.pptxWORKU and YADETA AHN-II Group Assignment.pptx
WORKU and YADETA AHN-II Group Assignment.pptx
 
teeth disorders seminar presentation.pptx
teeth disorders seminar presentation.pptxteeth disorders seminar presentation.pptx
teeth disorders seminar presentation.pptx
 
Thyroid Disorder.pptx
Thyroid Disorder.pptxThyroid Disorder.pptx
Thyroid Disorder.pptx
 
pitutiary disorder final.pptx
pitutiary disorder  final.pptxpitutiary disorder  final.pptx
pitutiary disorder final.pptx
 
Edited Assignment of Adrenal Disorders (1)(1).pptx
Edited Assignment of Adrenal Disorders (1)(1).pptxEdited Assignment of Adrenal Disorders (1)(1).pptx
Edited Assignment of Adrenal Disorders (1)(1).pptx
 
CHRONIC LIVER DISEASE.pptx
CHRONIC LIVER DISEASE.pptxCHRONIC LIVER DISEASE.pptx
CHRONIC LIVER DISEASE.pptx
 
BONE TUMORS.pptx
BONE TUMORS.pptxBONE TUMORS.pptx
BONE TUMORS.pptx
 
1. Medical Helminthology.ppt
1. Medical Helminthology.ppt1. Medical Helminthology.ppt
1. Medical Helminthology.ppt
 
25 gall bladder and pancrease.pdf
25 gall bladder and pancrease.pdf25 gall bladder and pancrease.pdf
25 gall bladder and pancrease.pdf
 
endocrine lecture.pdf
endocrine lecture.pdfendocrine lecture.pdf
endocrine lecture.pdf
 
8- Seizure disorder.ppt
8- Seizure disorder.ppt8- Seizure disorder.ppt
8- Seizure disorder.ppt
 

Recently uploaded

Promoting Wellbeing - Applied Social Psychology - Psychology SuperNotes
Promoting Wellbeing - Applied Social Psychology - Psychology SuperNotesPromoting Wellbeing - Applied Social Psychology - Psychology SuperNotes
Promoting Wellbeing - Applied Social Psychology - Psychology SuperNotes
PsychoTech Services
 
TEST BANK For An Introduction to Brain and Behavior, 7th Edition by Bryan Kol...
TEST BANK For An Introduction to Brain and Behavior, 7th Edition by Bryan Kol...TEST BANK For An Introduction to Brain and Behavior, 7th Edition by Bryan Kol...
TEST BANK For An Introduction to Brain and Behavior, 7th Edition by Bryan Kol...
rightmanforbloodline
 
Cardiac Assessment for B.sc Nursing Student.pdf
Cardiac Assessment for B.sc Nursing Student.pdfCardiac Assessment for B.sc Nursing Student.pdf
Cardiac Assessment for B.sc Nursing Student.pdf
shivalingatalekar1
 
The Best Ayurvedic Antacid Tablets in India
The Best Ayurvedic Antacid Tablets in IndiaThe Best Ayurvedic Antacid Tablets in India
The Best Ayurvedic Antacid Tablets in India
Swastik Ayurveda
 
Cell Therapy Expansion and Challenges in Autoimmune Disease
Cell Therapy Expansion and Challenges in Autoimmune DiseaseCell Therapy Expansion and Challenges in Autoimmune Disease
Cell Therapy Expansion and Challenges in Autoimmune Disease
Health Advances
 
Chapter 11 Nutrition and Chronic Diseases.pptx
Chapter 11 Nutrition and Chronic Diseases.pptxChapter 11 Nutrition and Chronic Diseases.pptx
Chapter 11 Nutrition and Chronic Diseases.pptx
Earlene McNair
 
CHEMOTHERAPY_RDP_CHAPTER 3_ANTIFUNGAL AGENT.pdf
CHEMOTHERAPY_RDP_CHAPTER 3_ANTIFUNGAL AGENT.pdfCHEMOTHERAPY_RDP_CHAPTER 3_ANTIFUNGAL AGENT.pdf
CHEMOTHERAPY_RDP_CHAPTER 3_ANTIFUNGAL AGENT.pdf
rishi2789
 
CHEMOTHERAPY_RDP_CHAPTER 2 _LEPROSY.pdf1
CHEMOTHERAPY_RDP_CHAPTER 2 _LEPROSY.pdf1CHEMOTHERAPY_RDP_CHAPTER 2 _LEPROSY.pdf1
CHEMOTHERAPY_RDP_CHAPTER 2 _LEPROSY.pdf1
rishi2789
 
share - Lions, tigers, AI and health misinformation, oh my!.pptx
share - Lions, tigers, AI and health misinformation, oh my!.pptxshare - Lions, tigers, AI and health misinformation, oh my!.pptx
share - Lions, tigers, AI and health misinformation, oh my!.pptx
Tina Purnat
 
CBL Seminar 2024_Preliminary Program.pdf
CBL Seminar 2024_Preliminary Program.pdfCBL Seminar 2024_Preliminary Program.pdf
CBL Seminar 2024_Preliminary Program.pdf
suvadeepdas911
 
Abortion PG Seminar Power point presentation
Abortion PG Seminar Power point presentationAbortion PG Seminar Power point presentation
Abortion PG Seminar Power point presentation
AksshayaRajanbabu
 
CHEMOTHERAPY_RDP_CHAPTER 4_ANTI VIRAL DRUGS.pdf
CHEMOTHERAPY_RDP_CHAPTER 4_ANTI VIRAL DRUGS.pdfCHEMOTHERAPY_RDP_CHAPTER 4_ANTI VIRAL DRUGS.pdf
CHEMOTHERAPY_RDP_CHAPTER 4_ANTI VIRAL DRUGS.pdf
rishi2789
 
Does Over-Masturbation Contribute to Chronic Prostatitis.pptx
Does Over-Masturbation Contribute to Chronic Prostatitis.pptxDoes Over-Masturbation Contribute to Chronic Prostatitis.pptx
Does Over-Masturbation Contribute to Chronic Prostatitis.pptx
walterHu5
 
K CỔ TỬ CUNG.pdf tự ghi chép, chữ hơi xấu
K CỔ TỬ CUNG.pdf tự ghi chép, chữ hơi xấuK CỔ TỬ CUNG.pdf tự ghi chép, chữ hơi xấu
K CỔ TỬ CUNG.pdf tự ghi chép, chữ hơi xấu
HongBiThi1
 
CHEMOTHERAPY_RDP_CHAPTER 1_ANTI TB DRUGS.pdf
CHEMOTHERAPY_RDP_CHAPTER 1_ANTI TB DRUGS.pdfCHEMOTHERAPY_RDP_CHAPTER 1_ANTI TB DRUGS.pdf
CHEMOTHERAPY_RDP_CHAPTER 1_ANTI TB DRUGS.pdf
rishi2789
 
TEST BANK For Community Health Nursing A Canadian Perspective, 5th Edition by...
TEST BANK For Community Health Nursing A Canadian Perspective, 5th Edition by...TEST BANK For Community Health Nursing A Canadian Perspective, 5th Edition by...
TEST BANK For Community Health Nursing A Canadian Perspective, 5th Edition by...
Donc Test
 
Complementary feeding in infant IAP PROTOCOLS
Complementary feeding in infant IAP PROTOCOLSComplementary feeding in infant IAP PROTOCOLS
Complementary feeding in infant IAP PROTOCOLS
chiranthgowda16
 
A Classical Text Review on Basavarajeeyam
A Classical Text Review on BasavarajeeyamA Classical Text Review on Basavarajeeyam
A Classical Text Review on Basavarajeeyam
Dr. Jyothirmai Paindla
 
REGULATION FOR COMBINATION PRODUCTS AND MEDICAL DEVICES.pptx
REGULATION FOR COMBINATION PRODUCTS AND MEDICAL DEVICES.pptxREGULATION FOR COMBINATION PRODUCTS AND MEDICAL DEVICES.pptx
REGULATION FOR COMBINATION PRODUCTS AND MEDICAL DEVICES.pptx
LaniyaNasrink
 
Muscles of Mastication by Dr. Rabia Inam Gandapore.pptx
Muscles of Mastication by Dr. Rabia Inam Gandapore.pptxMuscles of Mastication by Dr. Rabia Inam Gandapore.pptx
Muscles of Mastication by Dr. Rabia Inam Gandapore.pptx
Dr. Rabia Inam Gandapore
 

Recently uploaded (20)

Promoting Wellbeing - Applied Social Psychology - Psychology SuperNotes
Promoting Wellbeing - Applied Social Psychology - Psychology SuperNotesPromoting Wellbeing - Applied Social Psychology - Psychology SuperNotes
Promoting Wellbeing - Applied Social Psychology - Psychology SuperNotes
 
TEST BANK For An Introduction to Brain and Behavior, 7th Edition by Bryan Kol...
TEST BANK For An Introduction to Brain and Behavior, 7th Edition by Bryan Kol...TEST BANK For An Introduction to Brain and Behavior, 7th Edition by Bryan Kol...
TEST BANK For An Introduction to Brain and Behavior, 7th Edition by Bryan Kol...
 
Cardiac Assessment for B.sc Nursing Student.pdf
Cardiac Assessment for B.sc Nursing Student.pdfCardiac Assessment for B.sc Nursing Student.pdf
Cardiac Assessment for B.sc Nursing Student.pdf
 
The Best Ayurvedic Antacid Tablets in India
The Best Ayurvedic Antacid Tablets in IndiaThe Best Ayurvedic Antacid Tablets in India
The Best Ayurvedic Antacid Tablets in India
 
Cell Therapy Expansion and Challenges in Autoimmune Disease
Cell Therapy Expansion and Challenges in Autoimmune DiseaseCell Therapy Expansion and Challenges in Autoimmune Disease
Cell Therapy Expansion and Challenges in Autoimmune Disease
 
Chapter 11 Nutrition and Chronic Diseases.pptx
Chapter 11 Nutrition and Chronic Diseases.pptxChapter 11 Nutrition and Chronic Diseases.pptx
Chapter 11 Nutrition and Chronic Diseases.pptx
 
CHEMOTHERAPY_RDP_CHAPTER 3_ANTIFUNGAL AGENT.pdf
CHEMOTHERAPY_RDP_CHAPTER 3_ANTIFUNGAL AGENT.pdfCHEMOTHERAPY_RDP_CHAPTER 3_ANTIFUNGAL AGENT.pdf
CHEMOTHERAPY_RDP_CHAPTER 3_ANTIFUNGAL AGENT.pdf
 
CHEMOTHERAPY_RDP_CHAPTER 2 _LEPROSY.pdf1
CHEMOTHERAPY_RDP_CHAPTER 2 _LEPROSY.pdf1CHEMOTHERAPY_RDP_CHAPTER 2 _LEPROSY.pdf1
CHEMOTHERAPY_RDP_CHAPTER 2 _LEPROSY.pdf1
 
share - Lions, tigers, AI and health misinformation, oh my!.pptx
share - Lions, tigers, AI and health misinformation, oh my!.pptxshare - Lions, tigers, AI and health misinformation, oh my!.pptx
share - Lions, tigers, AI and health misinformation, oh my!.pptx
 
CBL Seminar 2024_Preliminary Program.pdf
CBL Seminar 2024_Preliminary Program.pdfCBL Seminar 2024_Preliminary Program.pdf
CBL Seminar 2024_Preliminary Program.pdf
 
Abortion PG Seminar Power point presentation
Abortion PG Seminar Power point presentationAbortion PG Seminar Power point presentation
Abortion PG Seminar Power point presentation
 
CHEMOTHERAPY_RDP_CHAPTER 4_ANTI VIRAL DRUGS.pdf
CHEMOTHERAPY_RDP_CHAPTER 4_ANTI VIRAL DRUGS.pdfCHEMOTHERAPY_RDP_CHAPTER 4_ANTI VIRAL DRUGS.pdf
CHEMOTHERAPY_RDP_CHAPTER 4_ANTI VIRAL DRUGS.pdf
 
Does Over-Masturbation Contribute to Chronic Prostatitis.pptx
Does Over-Masturbation Contribute to Chronic Prostatitis.pptxDoes Over-Masturbation Contribute to Chronic Prostatitis.pptx
Does Over-Masturbation Contribute to Chronic Prostatitis.pptx
 
K CỔ TỬ CUNG.pdf tự ghi chép, chữ hơi xấu
K CỔ TỬ CUNG.pdf tự ghi chép, chữ hơi xấuK CỔ TỬ CUNG.pdf tự ghi chép, chữ hơi xấu
K CỔ TỬ CUNG.pdf tự ghi chép, chữ hơi xấu
 
CHEMOTHERAPY_RDP_CHAPTER 1_ANTI TB DRUGS.pdf
CHEMOTHERAPY_RDP_CHAPTER 1_ANTI TB DRUGS.pdfCHEMOTHERAPY_RDP_CHAPTER 1_ANTI TB DRUGS.pdf
CHEMOTHERAPY_RDP_CHAPTER 1_ANTI TB DRUGS.pdf
 
TEST BANK For Community Health Nursing A Canadian Perspective, 5th Edition by...
TEST BANK For Community Health Nursing A Canadian Perspective, 5th Edition by...TEST BANK For Community Health Nursing A Canadian Perspective, 5th Edition by...
TEST BANK For Community Health Nursing A Canadian Perspective, 5th Edition by...
 
Complementary feeding in infant IAP PROTOCOLS
Complementary feeding in infant IAP PROTOCOLSComplementary feeding in infant IAP PROTOCOLS
Complementary feeding in infant IAP PROTOCOLS
 
A Classical Text Review on Basavarajeeyam
A Classical Text Review on BasavarajeeyamA Classical Text Review on Basavarajeeyam
A Classical Text Review on Basavarajeeyam
 
REGULATION FOR COMBINATION PRODUCTS AND MEDICAL DEVICES.pptx
REGULATION FOR COMBINATION PRODUCTS AND MEDICAL DEVICES.pptxREGULATION FOR COMBINATION PRODUCTS AND MEDICAL DEVICES.pptx
REGULATION FOR COMBINATION PRODUCTS AND MEDICAL DEVICES.pptx
 
Muscles of Mastication by Dr. Rabia Inam Gandapore.pptx
Muscles of Mastication by Dr. Rabia Inam Gandapore.pptxMuscles of Mastication by Dr. Rabia Inam Gandapore.pptx
Muscles of Mastication by Dr. Rabia Inam Gandapore.pptx
 

4. CNS Spinal cord injury.pptx

  • 1. SPINAL CORD INJURY MANOJ M.G MANOJ M.G. LECTURER (MEDICAL SURGICAL NURSING) DEPARTMENT OF NURSING AND MIDWIFERY WOLLEGA UNIVERSITY
  • 2. DEFINITION •Spinal cord injury (SCI) is damage to the spinal cord that results in a loss of function such as mobility or feeling. Common locations of spinal cord injuries •Flexion-extension injuries are commonly located at C4 - C7. • T11, T12, and L1 are frequent sites of spinal cord injury resulting rom falls.
  • 3.
  • 4. Mechanisms of spinal cord injury. •Flexion-extension: whiplash(is a non-medical term describing a range of injuries to the neck caused by or related to a sudden distortion of the neck, associated with extension) • Subluxation: incomplete or partial dislocation. •Torsion: twisting of the spinal cord. •Compression.
  • 6. RISK FACTORS Gender - Spinal cord injury affects a disproportionate amount of men Age – (Young adults and seniors) - Between ages 16 and 35 / MVA leading cause - Another peak in people older than 60 / falls leading cause People active in sports – High risk athletic activities include football, rugby, wrestling, gymnastics, diving, surfing, ice hockey and downhill skiing Predisposing conditions - A relatively minor injury can cause spinal cord injury in people with conditions that affect their bones or joints, such as arthritis or osteoporosis
  • 8. •Road Traffic Accident •Sports injury •Fall •Bullet or stab wound •Traumatic injury •Electric shock •Extreme twisting of the middle of the body •Landing on the head during a sports injury •Fall from a great height
  • 9.
  • 10. Types of Spinal Cord Injury •Complete Spinal Cord Injuries • Complete paraplegia is described as permanent loss of motor and nerve function at T1 level or below, resulting in loss of sensation and movement in the legs, bowel, bladder, and sexual region. Arms and hands retain normal function. •Complete loss of motor function and sensation below the area of injury
  • 11. •Even in a complete injury, the spinal cord is almost never completely cut in half. Doctors use the term "complete" to describe a large amount of damage to the spinal cord. •many people with partial spinal cord injuries are able to experience significant recovery. While those with complete injuries are not .
  • 12. Incomplete / Partial spinal cord injury Spinal cord is able to convey some messages to or from the brain. Therefore, retain some sensation and possibly some motor function below the affected area
  • 13. • Complete or Partial ?
  • 14. Incomplete / Partial Spinal Cord Injury •Central •Lateral / Brown-Sequard Syndrome •Anterior •Posterior 14
  • 15. The articulated spine. A, Anterior view. B, Posterior view. C, Right lateral view
  • 16. •Anterior Cord Syndrome Damage of front 2/3 of spinal cord, loss of pain and temperature sensation, and motor function below level of injury Light touch (pressure) and position and vibration sensation preserved Possible for some people with this injury to later recover some movement 16
  • 17.
  • 18. •Central Cord Syndrome Usually with unbelted Motor Vehicle Accidents and falls of elderly Typically results greater weakness in arms vs lower extremities Sensory loss varies but more severe in upper extremities Control over the bowel and bladder varies and may be preserved Possible for some recovery from this type of injury, usually starting in the legs, gradually progressing upwards 18
  • 20. •Brown-Sequard Syndrome/lateral Usually stab or Gun Shot Wound Damage is towards one side of the spinal cord Ipsilateral (same side as the cord injury) Impaired or loss of movement, touch, pressure and vibration (Hemi paraplegia) Contralateral (opposite side of cord injury) loss of pain and temperature sensation (Hemiparasthesia) 20
  • 21.
  • 22. •Posterior Cord Syndrome Damage is towards the back of the spinal cord May leave the person with good muscle power, pain and temperature sensation However they may experience difficulty coordinating movement of their limbs 22
  • 23.
  • 24. •Cauda Equina Syndrome: Due to bony compression or disc protrusions in lumbar or sacral region Non specific symptoms – back pain  - bowel and bladder dysfunction  - leg numbness and weakness  - saddle paresthesia(loss of sensation restricted to the area of buttocks, perineum and inner surface of the thighs)
  • 25. Spinal Cord Injuries Causal Categories Traumatic spinal cord injury may stem from: • Sudden, traumatic blow that fractures, dislocates, crushes or compresses one or more of vertebrae • Gunshot or knife wound that penetrates and cuts your spinal cord • Additional (secondary) damage usually occurs over days or weeks because of bleeding, swelling, inflammation and fluid accumulation in and around spinal cord
  • 26. Non-traumatic spinal cord injury may be caused by : • Arthritis • Cancer • Blood vessel problems or bleeding • Inflammation or infections • Disk degeneration of the spine
  • 27. Primary Spinal Injury •Result of initial trauma •Injury usually permanent
  • 28. Secondary Spinal Injury •Occurs after Spinal cord trauma •Damage at cellular level •Necrosis (Cells swell, burst and leak toxic substances to other cells) • Apoptosis (Programmed cell death / cell suicide to prevent bursting) 28
  • 31. PATHOPHYSIOLOGY Due to the etiological factor Damage to the cord may be a concussion, contusion, laceration, compression, or complete, incomplete of the cord. Cord's response to injury includes hemorrhage, ischemia, and edema SCI Involves loss of: Motor function Sensory function Reflexes Control of elimination
  • 32. CLINICAL MANIFESTATIONS •Patients with tetraplegia (formerly called quadriplegia) have damage to the cervical segments of nerves (C1-C8) in the spinal canal. Function may be impaired in the upper extremities, trunk, pelvic organs, and lower extremities. •Patients with paraplegia have damage to the thoracic, lumbar, or sacral segments of nerves in the spinal cord. The arms are unaffected, but function may be impaired in the trunk, pelvic organs, and lower extremities.
  • 33. •Sacral sensation is intact if there is deep sensation and sensation at the anal mucocutaneous junction; sacral motor is intact if the patient has voluntary contraction of the external anal sphincter with digital stimulation. •The Zone of Partial Preservation (ZPP) indicates areas of partial sensory/motor innervation below the Level Of Injurie; the ZPP is applicable only to complete injuries.
  • 34. •The neurologic level of injury is the lowest neural level with normal sensory and motor function on both sides of the body. When describing the level of involvement, the neurologic level is noted unless stated specifically that the skeletal level of involvement, which is the level of greatest vertebral damage, is being discussed. •Various syndromes (incomplete injuries) may characterize the clinical presentation
  • 35. • Patient's symptoms will mirror the level of the cord injury. •There will be total sensory loss and motor paralysis below level of the injury. • Cervical spinal cord injuries will produce quadriplegia--loss of function of all four extremities. • Injuries to the thoracic spinal cord below the level of T1 will produce paraplegia--paralysis of the lower extremities. • Loss of bowel and bladder control; usually urinary retention and bladder distention.
  • 36. •Loss of sweating and vasomotor tone below the level of the cord injury. •Marked reduction of blood pressure due to loss of peripheral vascular resistance. •Neck/back pain. •Priapism--persistent, painful erection of the penis.
  • 37.
  • 38. CERVICAL (NECK) INJURIES •Breathing difficulties •Loss of normal bowel and bladder control •Numbness •Sensory changes •Spasticity (increased muscle tone)
  • 39. THORACIC (CHEST LEVEL) INJURIES •Loss of normal bowel and bladder control •Numbness •Sensory changes •Spasticity (increased muscle tone) •Weakness, paralysis
  • 40. LUMBAR SACRAL (LOWER BACK) INJURIES •Loss of normal bowel and bladder control (you may have constipation, leakage, and bladder spasms) •Numbness •Pain •Sensory changes •Weakness and paralysis
  • 41. DIAGNOSTIC TESTS •Complete blood count (e.g. Hb, RBC, WBC) •Arterial blood gas level •X ray •CT scan and MRI
  • 45.
  • 47. MANAGEMENT •Requires a multidisciplinary approach because of multiple systems involvement and the psychosocial aspects of catastrophic injuries. Immediately After Trauma (Less Than 1 Hour) •Immobilization with rigid cervical collar, sandbags, and rigid spine board to transport from the field to acute care facility.
  • 48. 48
  • 49. Acute Phase (1 to 24 Hours) •Maintenance of pulmonary and cardiovascular stability. •Intubation and mechanical ventilation, if needed. •Vasopressors to maintain adequate perfusion to sustain mean arterial BP at 85 to 90 mm Hg. •Medical stabilization before spinal stabilization and decompression.
  • 50. •Spinal cord immobilization use of skeletal tongs. •Crutchfield and Vinke tongs( used for immubilization) require predrilled holes in the skull under local anesthesia; Gardner-Wells and Heifitz tongs do not. •Weight is added to traction gradually to reduce the vertebral fracture; weight maintained at a level to ensure vertebral alignment.
  • 51. •Rigid kinetic turning bed to immobilize patients with thoracic and lumbar injuries. •Surgical interventions are considered when the patient has vertebral instability that may result in further neurologic damage; an injury that is incomplete at onset may become complete if instability exists. The objectives are to remove all of the bony and soft tissues that are compressing the spinal cord, thereby minimizing the possibility of a deteriorating neurologic status, and stabilize the vertebra surrounding the spinal cord so that rehabilitation may begin as soon as possible.
  • 52. •Decompression, typically using the anterior approach in cervical instances, may be accomplished by removing the bony structures and soft tissues (eg, fusion, decompression laminectomy). Realignment of the soft tissues and vertebral column is required. •Stabilization, typically done using the posterior approach, involves the use of wires, bone grafts, plates, screws, and other fixation devices to prevent movement at the damaged bony site (eg, fusion, Harrington rods). Harrington rods, used for thoracolumbar SCI, extend approximately one to three levels above and below the fracture.
  • 53. •Methylprednisolone sodium succinate( to improve neurological recovery) should be administered within 8 hours of injury. •Bolus 30 mg/kg administered over 15 minutes; maintenance infusion of 5.4 mg/kg/hr infused for 23 hours •Additional benefit may be achieved by administering the maintenance dose for 48 hours. •Management of neurogenic bladder( dysfunction caused by neurologic damage, symptoms may include over flow, urgency, frequency) Foley catheter •Pressure ulcer prevention pressure reduction mattress or kinetic turning frame.
  • 54. Sub acute Phase (Within 1 Week) •Halo traction is the primary treatment for cervical injuries •ganglioside sodium salt I.V., begun within 72 hours after injury, and continued for 18 to 32 days, is believed to enhance neuronal regeneration. •H2-receptor blockers to prevent gastric irritation and haemorrhage. •Early mobilization and passive exercise as soon as patient is surgically and medically stable. •Hyper alimentation( intravenous supply of nutrients) to retard negative nitrogen balance.
  • 55. •Interventions to prevent thromboembolism (intermediate risk) are based on motor completeness of injury: •Motor Incomplete ,compression hose; compression boots in addition to unfractionated heparin (UH) 5,000 units every 12 hours. •Motor Complete compression hose; compression boots in addition to UH with the dosage adjusted to high normal or low-molecular-weight heparin 30 mg bid. •If the patient is at high risk (ie, motor complete with other risk factors such as fractures of lower extremities or previous DVT), an inferior vena cava filter should be considered.
  • 56. Chronic Phase (Beyond 1 Week) •Harrington rods, used in conjunction with a body jackets, are used for patients with thoracolumbar injuries. •To prevent thrombophlebitis in the chronic phase, compression boots should be continued for 2 weeks; anticoagulants should also be continued based on motor completeness of injury: •Motor Incomplete until discharged from hospital (or 8 weeks •Motor Complete 8 weeks. •If the patient is at high risk, anticoagulants should be continued for 12 weeks or until discharged from hospital.
  • 57. •Management of complications may include treatment of infections with antibiotics; treatment of respiratory compromise with phrenic nerve pacing, mechanical ventilation, and other methods; pressure ulcer treatment; management of heterotopic ossification (is the presence of bone in soft tissue where bone normally does not exist) with calcium chelators( which bind calcium) and anti-inflammatory agents; drainage of syringomyelia( cyst in spinal cord); management of spasticity with oral or intrathecal antispasmodics, surgical procedure, or spinal cord stimulation; and management of central neuropathic pain with anticonvulsants, minor sedatives, antidepressants, nerve block, or surgical procedure.
  • 58. •Spasticity should be managed by: •Maintaining calm, stress-free environment. •Allowing ample time for activities such as positioning and transferring. •Performing joint ROM exercises with slow, smooth movements. •Avoiding temperature extremes. •Administering muscle relaxants, such as baclofen (Lioresal) (via pump or orally), diazepam (Valium), and dantrolene (Dantrium), as prescribed.
  • 59. •External sphincterotomy may be used for detrusor- sphincter dyssynergia (Insufficient relaxation of the sphincter during a voiding contraction prevents effective bladder emptying and can lead to high pressures in the bladder) Other options include urethral stents and balloon dilatation. •Clonidine has been used to manage spasticity and facilitate ambulation in patients with incomplete injuries. •Resistive inspiratory muscles training shows promise in promoting respiratory muscle strength and reducing sleep- induced breathing disturbances in patients with tetraplegia. Resistive training devices are used to perform respiratory maneuvers at scheduled times during the day. •Rehabilitation includes medical and psychosocial support, physical therapy, urologic evaluation, occupational therapy, and multiple other interventions to facilitate an increased level of function and community participation.
  • 61. Nursing Assessment •Assess cardiopulmonary status and vital signs to help determine degree of autonomic dysfunction, especially in patients with tetraplegia. •Determine LOC and cognitive function indicating Traumatic Brain Injuries or other pathology. •Perform frequent motor and sensory assessment of trunk and extremities extent of deficits may increase due to oedema and haemorrhage. And it may increase the neurologic deficit and pain.
  • 62. •Note signs and symptoms of spinal shock, such as flaccid paralysis, urine retention, absent reflexes. •Assess bowel and bladder function. •Assess quality, location, severity of pain. •Perform psychosocial assessment to evaluate motivation, support network, financial or other problems. •Assess for indicators of powerlessness, including verbal expression of no control over situation, depression, nonparticipation, dependence on others, passivity.
  • 63. Nursing Diagnoses •Ineffective Breathing Pattern related to paralysis of respiratory muscles or diaphragm •Impaired Physical Mobility related to motor dysfunction •Risk for Impaired Skin Integrity related to immobility and sensory deficit •Urinary Retention related to neurogenic bladder •Constipation or Bowel Incontinence related to neurogenic bowel
  • 64. •Risk for Injury: autonomic dysreflexia (is a syndrome in which there is a sudden onset of excessively high blood pressure) and orthostatic hypotension ( is defined as a decrease in systolic blood pressure of 20 mm Hg or a decrease in diastolic blood pressure of 10 mm Hg within three minutes of standing when compared with blood pressure from the sitting or supine position) •Powerlessness related to loss of function, long rehabilitation, depression •Sexual Dysfunction related to erectile dysfunction and fertility changes •Chronic Pain related to neurogenic changes
  • 65. Nursing Management Objectives of care: •Reduce the fracture/dislocation and obtain immobilization of the spine as soon as possible to prevent further cord damage. •Observe for symptoms of progressive neurological damage. Patient with cervical spine injury will have some form of skeletal traction. Maintain traction and provide nursing care local policy.
  • 66. Continuously observe patient's breathing pattern. •Patients with injuries at high levels are at risk for respiratory failure. •Observe strength of cough effort. Be alert for signs of spinal shock ( is the temporary reduction of or loss of reflexes following a spinal cord injury (SCI) )and report immediately. •(Spinal shock represents a sudden loss of continuity between the spinal cord and higher nerve centers. •It is characterized by a complete loss of motor, sensory, reflex, and autonomic activity below the level of the injury. •Though temporary, spinal shock may last for several weeks.
  • 67. Continuously observe patient for motor and sensory changes due to cord edema or hemorrhage, which may further compromise cord function. •Test patient's motor ability by asking him/her to spread fingers, grip your hands, shrug shoulders, etc. •Test sensory level by gently pinching the skin at shoulders and progressing down sides; ascertain level at which patient can no longer feel pinch. •Note presence/absence of sweating. •Carefully record findings in patient's clinical record; report changes in patient's motor/sensory level immediately to professional nurse.
  • 68. If turning is allowed and patient is not on a turning frame or turning bed, the patient must be carefully log- rolled with the spine maintained in alignment. Surgery, depending upon the injury and pathological findings, may have to be performed to stabilize the spine before rehabilitation can begin. Patient will require passive range of motion exercises. Assist with active rehabilitation procedures when patient is stable. •Program is designed according to neurological deficit. •Usually involves 6 weeks of gradual mobilization with brace or cast, depending upon level of injury. Provide constant encouragement and psychological support to the patient with a spinal cord injury.
  • 69. Possible Complications •Blood pressure changes - can be extreme (autonomic hyper reflexia) •Chronic kidney disease •Complications of immobility: Deep vein thrombosis Pulmonary infections Skin breakdown •Contractures: A muscle contracture is a permanent shortening of a muscle or joint
  • 70. •Increased risk of urinary tract infections •Loss of bladder control •Loss of bowel control •Loss of sensation •Loss of sexual functioning (male impotence) •Muscle spasticity •Paralysis of breathing muscles •Paralysis (paraplegia, quadriplegia) •Pressure sores •Shock
  • 71. REHABILITATION •Cognitive Rehabilitation Therapy •Speech Therapy •Mental Rehabilitation •Physical Exercise •Occupational Therapy