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20
DAVID SUTTON
DAVID SUTTON PICTURES
DR. Muhammad Bin Zulfiqar
PGR-FCPS III SIMS/SHL
• Fig. 20.1 Compression paddle. The patient lies
prone on the paddle and the balloon is
inflated to compress overlying small-bowel
loops during fluoroscopy.
• Fig. 20.2 : Small Bowel Enema.
• Fig. 20.3 Normal ileostomy enema.
• Fig. 20.4 Non-ionic, water-soluble follow-
through performed using iohexol in a
postoperative patient.
• Fig. 20.5 Plain
abdominal film
reveals a dilated
jejunal loop in this
patient with
obstruction secondary
to an internal hernia
(note residual
contrast in the
appendix from recent
barium enema).
• Fig. 20.6 CT shows unequivocal small bowel
obstruction.
• Fig. 20.7 Multislice CT with reconstruction shows no
mass at the transition point between dilated (curve
arrow) and undilated (straight arrow) small bowel.
Diagnosis: adhesions, confirmed at subsequent
laparotomy.
• Fig. 20.8 Barium follow-through in a patient with
adhesions. There is an abrupt transition point from
dilated to undilated small bowel in this patient with
obstruction to the afferent limb of an ileoanal pouch.
• Fig. 20.9 Scleroderma.
• Fig. 20.10 Primary visceral myopathy. Note
the characteristic, massively dilated duodenal
loop (arrow).
• Fig. 20.11 Crohn's disease. Compression view
reveals an intense mucosal granularity,
caused by villous oedema.
• Fig. 20.12 Crohn's disease. Fold thickening.
• Fig. 20.13 Crohn's disease. Compression of an
ileal loop reveals several aphthous ulcers (one
of which is arrowed). Also note the
background granularity caused by villous
oedema.
• Fig. 20.14 Advanced Crohn's disease
evidenced by several, long 'cobblestone‘
segments with intervening dilatation.
• Fig. 20.15 Advanced Crohn's disease with
several characteristic pseudodiverticula
(arrows).
• Fig. 20.16 Crohn's disease. Neoterminal ileal
recurrence at right hemicolectomy site.
• Fig. 20.17 Crohn's disease. CT reveals a
parastomal hernia when the patient is in the
right lateral position.
• Fig. 20.18 Crohn's disease. Normal Kock
pouch.
• Fig. 20.19 Crohn's disease. Ultrasound reveals
gross mural thickening in an ileal loop.
• Fig. 20.20 Crohn's disease. CT shows the
extent of terminal ileal thickening (arrows).
• Fig. 20.21 Crohn's disease. Fat suppressed T2-
weighed MR scan shows thickened ileal loops
(curved arrows) and also reveals a parastomal
abscess (straight arrow).
• Fig. 20.22 Benign stromal tumour. (A) Barium
follow-through reveals an intraluminal mass
(arrow) on compression. (B) The tumour is
also visible on CT (arrow).
• Fig. 20.23 Small bowel adenocarcinoma
(between arrows) complicating Muir-Torre
syndrome.
• Fig. 20.24 Lymphoma. Diffuse fold thickening
and nodularity.
• Fig. 20.25 Lymphoma. CT reveals a well-
demarcated soft-tissue mass.
• Fig. 20.26 CT reveals a desmoplastic reaction
in a patient with carcinoid tumour.
• Fig. 20.27 CT reveals a large pelvic soft-tissue
mass that proved to be recurrent stromal
tumour.
Fig. 20.28 Duodenal adenomas (some of which
are arrowed) complicating familial
adenomatous polyposis.
• Fig. 20.29 Familial adenomatous polyposis.
T2 -weighted MR image of a mesenteric
desmoid tumour (arrows).
• Fig. 20.30 Barium follow-through reveals an
ileal hamartoma (arrow) in Peutz-Jeghers
syndrome.
• Fig. 20.31 Barium follow-through in a patient
with extensive radiation enteritis reveals
strictures, dilatation and a 'picket-fence'
appearance (arrows).
• Fig. 20.32 Gross intramural jejunal
haemorrhage revealed by CT in a young man
taking oral anticoagulants.
• Fig. 20.33 Small-bowel thickening, causing a
'target' sign, in a young woman with Henoch-
Schönlein purpura (arrows).
• Fig. 20.34 Barium follow-through reveals a
large Meckel's diverticulum (arrows).
• Fig. 20.35 Terminal ileum nodular lymphoid
hyperplasia.
• Fig. 20.36 Plain film showing pneumatosis
intestinalis evidenced by (arrows). innumerable
air-filled cysts.
• Fig. 20.37 Peritoneal attachments and potential spaces
when viewed from the front (A) and side (B); (A) also
demonstrates likely pathways for pathological spread.
• Fig. 20.37 Peritoneal attachments and
potential spaces when viewed from the front
(A) and side (B); (A) also demonstrates likely
pathways for pathological spread.
• Fig. 20.38 CT reveals deposits on the liver
surface (arrow) in this patient with ovarian
carcinoma (note splenic ascites).
• Fig. 20.39 Contrast-enhanced CT reveals
plaques of high-attenuation peritoneal
deposits in a patient with disseminated
colorectal adenocarcinoma.
• Fig. 20.40 CT reveals the liver scalloping
typical of pseudomyxoma.
• Fig. 20.41 Barium follow-through shows
distal ileal encasement in mesenteric
panniculitis.
20 DAVID SUTTON PICTURES THE SMALL BOWEL AND PERITONEAL CAVITY

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20 DAVID SUTTON PICTURES THE SMALL BOWEL AND PERITONEAL CAVITY

  • 2. DAVID SUTTON PICTURES DR. Muhammad Bin Zulfiqar PGR-FCPS III SIMS/SHL
  • 3. • Fig. 20.1 Compression paddle. The patient lies prone on the paddle and the balloon is inflated to compress overlying small-bowel loops during fluoroscopy.
  • 4. • Fig. 20.2 : Small Bowel Enema.
  • 5. • Fig. 20.3 Normal ileostomy enema.
  • 6. • Fig. 20.4 Non-ionic, water-soluble follow- through performed using iohexol in a postoperative patient.
  • 7. • Fig. 20.5 Plain abdominal film reveals a dilated jejunal loop in this patient with obstruction secondary to an internal hernia (note residual contrast in the appendix from recent barium enema).
  • 8. • Fig. 20.6 CT shows unequivocal small bowel obstruction.
  • 9. • Fig. 20.7 Multislice CT with reconstruction shows no mass at the transition point between dilated (curve arrow) and undilated (straight arrow) small bowel. Diagnosis: adhesions, confirmed at subsequent laparotomy.
  • 10. • Fig. 20.8 Barium follow-through in a patient with adhesions. There is an abrupt transition point from dilated to undilated small bowel in this patient with obstruction to the afferent limb of an ileoanal pouch.
  • 11. • Fig. 20.9 Scleroderma.
  • 12. • Fig. 20.10 Primary visceral myopathy. Note the characteristic, massively dilated duodenal loop (arrow).
  • 13. • Fig. 20.11 Crohn's disease. Compression view reveals an intense mucosal granularity, caused by villous oedema.
  • 14. • Fig. 20.12 Crohn's disease. Fold thickening.
  • 15. • Fig. 20.13 Crohn's disease. Compression of an ileal loop reveals several aphthous ulcers (one of which is arrowed). Also note the background granularity caused by villous oedema.
  • 16. • Fig. 20.14 Advanced Crohn's disease evidenced by several, long 'cobblestone‘ segments with intervening dilatation.
  • 17. • Fig. 20.15 Advanced Crohn's disease with several characteristic pseudodiverticula (arrows).
  • 18. • Fig. 20.16 Crohn's disease. Neoterminal ileal recurrence at right hemicolectomy site.
  • 19. • Fig. 20.17 Crohn's disease. CT reveals a parastomal hernia when the patient is in the right lateral position.
  • 20. • Fig. 20.18 Crohn's disease. Normal Kock pouch.
  • 21. • Fig. 20.19 Crohn's disease. Ultrasound reveals gross mural thickening in an ileal loop.
  • 22. • Fig. 20.20 Crohn's disease. CT shows the extent of terminal ileal thickening (arrows).
  • 23. • Fig. 20.21 Crohn's disease. Fat suppressed T2- weighed MR scan shows thickened ileal loops (curved arrows) and also reveals a parastomal abscess (straight arrow).
  • 24. • Fig. 20.22 Benign stromal tumour. (A) Barium follow-through reveals an intraluminal mass (arrow) on compression. (B) The tumour is also visible on CT (arrow).
  • 25. • Fig. 20.23 Small bowel adenocarcinoma (between arrows) complicating Muir-Torre syndrome.
  • 26. • Fig. 20.24 Lymphoma. Diffuse fold thickening and nodularity.
  • 27. • Fig. 20.25 Lymphoma. CT reveals a well- demarcated soft-tissue mass.
  • 28. • Fig. 20.26 CT reveals a desmoplastic reaction in a patient with carcinoid tumour.
  • 29. • Fig. 20.27 CT reveals a large pelvic soft-tissue mass that proved to be recurrent stromal tumour.
  • 30. Fig. 20.28 Duodenal adenomas (some of which are arrowed) complicating familial adenomatous polyposis.
  • 31. • Fig. 20.29 Familial adenomatous polyposis. T2 -weighted MR image of a mesenteric desmoid tumour (arrows).
  • 32. • Fig. 20.30 Barium follow-through reveals an ileal hamartoma (arrow) in Peutz-Jeghers syndrome.
  • 33. • Fig. 20.31 Barium follow-through in a patient with extensive radiation enteritis reveals strictures, dilatation and a 'picket-fence' appearance (arrows).
  • 34. • Fig. 20.32 Gross intramural jejunal haemorrhage revealed by CT in a young man taking oral anticoagulants.
  • 35. • Fig. 20.33 Small-bowel thickening, causing a 'target' sign, in a young woman with Henoch- Schönlein purpura (arrows).
  • 36. • Fig. 20.34 Barium follow-through reveals a large Meckel's diverticulum (arrows).
  • 37. • Fig. 20.35 Terminal ileum nodular lymphoid hyperplasia.
  • 38. • Fig. 20.36 Plain film showing pneumatosis intestinalis evidenced by (arrows). innumerable air-filled cysts.
  • 39. • Fig. 20.37 Peritoneal attachments and potential spaces when viewed from the front (A) and side (B); (A) also demonstrates likely pathways for pathological spread.
  • 40. • Fig. 20.37 Peritoneal attachments and potential spaces when viewed from the front (A) and side (B); (A) also demonstrates likely pathways for pathological spread.
  • 41. • Fig. 20.38 CT reveals deposits on the liver surface (arrow) in this patient with ovarian carcinoma (note splenic ascites).
  • 42. • Fig. 20.39 Contrast-enhanced CT reveals plaques of high-attenuation peritoneal deposits in a patient with disseminated colorectal adenocarcinoma.
  • 43. • Fig. 20.40 CT reveals the liver scalloping typical of pseudomyxoma.
  • 44. • Fig. 20.41 Barium follow-through shows distal ileal encasement in mesenteric panniculitis.