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 Definition
 Theories of dentin hypersensitivity
 Etiology
 Clinical features
 Diagnosis
 Management
 Treatment strategies
 According to Holland et al it is characterized
by short, sharp pain arising from exposed
dentin in response to stimuli typically thermal,
chemical, osmotic or tactile : and which cannot
be described as any other form of dental
pathology.
Direct Innervation Theory
Hydrodynamic Theory
Odontoblastic receptor
Theory
 States that direct mechanical stimulation
of exposed nerve endings at the DEJ is
responsible for dentinal hypersensitivity.
 Proposes that odontoblasts themselves
act as neural receptors and relay the
signal to the nerve terminals.
Pain
Stimulation of baroreceptors
(nerve receptor sensitive to pressure)
Fluid flow in exposed open dentinal tubules
Stimulus
(thermal, tactile, osmotic etc)
I. Loss of enamel
II. Gingival recession
III. Attrition
IV. erosion
V. abrasion
VI. Tooth malposition
VII. Periodontal diseases
VIII. Patient habits
 Pain
 -it is typically rapid in onset, sharp and of short
duration.
 -it may vary from mild to severe in intensity
 Sensitivity
 External stimuli include:
i. Thermal stimuli >hot/cold food and beverages
>cold blast of air
Osmotic stimuli: >sweet food
Acidic stimuli: >citrus fruits, acidic
beverages
Mechanical stimuli: >toothbrush, dental
instruments
 Complete patient history:
signs and symptoms.
intensity,
duration,
any dietary changes.
Clinical examination:
> visual assessment
> pocket depth
> use the 2way syringe to check the patient’s response to
air
Removal of
etiological factors:
•Improper brushing
habits,
•Poor oral hygiene,
gingival recession
Patient education
•About diet mod.,
plaque control,
proper brushing
techniques.
1. Desensitizing the nerve:
using toothpaste containing potassium nitrate
Salts/ions:
 0.4% Stannous fluoride
 2% sodium fluoride
 calcium carbonate
 strontium chloride
Protein precipitates:
 casein phosphopeptide attached to amorphous
calcium phosphate (CPP-ACP)
 silver nitrate
 zinc chloride
4. Crown placement
5. Periodontal grafts
6. Lasers
3.Dentin adhesives: GIC, composite
 Strudevant’s art and science of operative
dentistry
 Carranza’s clinical periodontology
Dentinal hypersensitivity

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Dentinal hypersensitivity

  • 1.
  • 2.  Definition  Theories of dentin hypersensitivity  Etiology  Clinical features  Diagnosis  Management  Treatment strategies
  • 3.  According to Holland et al it is characterized by short, sharp pain arising from exposed dentin in response to stimuli typically thermal, chemical, osmotic or tactile : and which cannot be described as any other form of dental pathology.
  • 4. Direct Innervation Theory Hydrodynamic Theory Odontoblastic receptor Theory
  • 5.  States that direct mechanical stimulation of exposed nerve endings at the DEJ is responsible for dentinal hypersensitivity.
  • 6.  Proposes that odontoblasts themselves act as neural receptors and relay the signal to the nerve terminals.
  • 7.
  • 8. Pain Stimulation of baroreceptors (nerve receptor sensitive to pressure) Fluid flow in exposed open dentinal tubules Stimulus (thermal, tactile, osmotic etc)
  • 9. I. Loss of enamel II. Gingival recession III. Attrition IV. erosion V. abrasion VI. Tooth malposition VII. Periodontal diseases VIII. Patient habits
  • 10.  Pain  -it is typically rapid in onset, sharp and of short duration.  -it may vary from mild to severe in intensity  Sensitivity  External stimuli include: i. Thermal stimuli >hot/cold food and beverages >cold blast of air
  • 11. Osmotic stimuli: >sweet food Acidic stimuli: >citrus fruits, acidic beverages Mechanical stimuli: >toothbrush, dental instruments
  • 12.  Complete patient history: signs and symptoms. intensity, duration, any dietary changes. Clinical examination: > visual assessment > pocket depth > use the 2way syringe to check the patient’s response to air
  • 13. Removal of etiological factors: •Improper brushing habits, •Poor oral hygiene, gingival recession Patient education •About diet mod., plaque control, proper brushing techniques.
  • 14. 1. Desensitizing the nerve: using toothpaste containing potassium nitrate
  • 15. Salts/ions:  0.4% Stannous fluoride  2% sodium fluoride  calcium carbonate  strontium chloride Protein precipitates:  casein phosphopeptide attached to amorphous calcium phosphate (CPP-ACP)  silver nitrate  zinc chloride
  • 16. 4. Crown placement 5. Periodontal grafts 6. Lasers 3.Dentin adhesives: GIC, composite
  • 17.  Strudevant’s art and science of operative dentistry  Carranza’s clinical periodontology