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Endodontic
Diagnosis
Dr. Hadil Abdallah Altilbani
BDS Santiago de Compostela University Spain.
MSc. University of Valencia Spain.
Department of Endodontics University of Palestine .
Diagnostic
Terminology
Approved by the American
Association of Endodontists
and the American Board of
Endodontics
1. Normal Pulp is a clinical
diagnostic category in which the pulp is
Symptom-free and Normally Responsive To
Pulp Testing.
Although the pulp may not be histologically
normal, a “clinically” normal pulp results in a
mild or transient response to thermal cold
testing, lasting No More Than One To Two
Seconds after the stimulus is removed.
One cannot arrive at a probable diagnosis
without comparing the tooth in question
with Adjacent And Contralateral Teeth.
It is best to test the adjacent teeth and contralateral teeth first
so that the patient is familiar with the experience of a normal
response to cold.
Pulpal Diagnoses
Control Teeth
Prior to performing any test, the
clinician should select “Control Teeth”
This calibrates the test and provides a
baseline with which to compare the
patient's response
Control teeth should be similar to the
suspect tooth As referred pain cannot
cross the midlline, it may be preferable
to select control teeth on the
contralateral side The first application
of the test is most significant
2. Reversible Pulpitis is based upon subjective and objective
findings indicating that the inflammation should resolve and
the pulp return to normal following appropriate management
of the etiology.
Discomfort is experienced when a stimulus, such as cold or sweet, is applied,
and goes away within a couple of seconds after the stimulus is removed.
Typical etiologies may include exposed dentin (dentinal sensitivity),
caries or deep restorations. There are no significant radiographic changes in
the periapical region of the suspect tooth, and the pain experienced is not
spontaneous.
Following the management of the etiology (e.g., caries removal plus
restoration or covering the exposed dentin), the tooth requires further
evaluation to determine whether the reversible pulpitis has returned to a
normal status.
Although dentinal sensitivity is not an inflammatory process, per se, all of the
symptoms of this entity mimic those of a reversible pulpitis.
3. Symptomatic Irreversible Pulpitis is based on subjective
and objective findings that the vital inflamed pulp is incapable
of healing and that root canal treatment is indicated.
Characteristics may include sharp pain upon thermal stimulus, lingering pain
(often 30 seconds or longer after stimulus removal), spontaneity
(unprovoked pain) and referred pain.
Sometimes, the pain may be accentuated by postural changes, such as lying
down or bending over.
In addition, over-the-counter analgesics are typically ineffective.
Common etiologies may include deep caries, extensive restorations, or
fractures exposing the pulpal tissues.
Teeth with symptomatic irreversible pulpitis may be Difficult To Diagnose
because the inflammation has not yet reached the periapical tissues, thus
resulting in no pain or discomfort to percussion.
In such cases, the patient’s dental history and thermal testing are the primary
tools for assessing pulpal status.
4. Asymptomatic
Irreversible Pulpitis is a
clinical diagnosis based on
subjective and objective findings
indicating that the vital inflamed
pulp is incapable of healing and
that root canal treatment is
indicated.
These cases have no clinical
symptoms and usually respond
normally to thermal testing, but
may have had trauma or deep
caries that would likely result in
exposure following removal.
5. Pulp Necrosis Is A Clinical Diagnostic
Category Indicating Death Of The Dental Pulp,
Necessitating Root Canal Treatment.
The Pulp Is Nonresponsive To Pulp Testing And
Is Asymptomatic.
Pulp Necrosis By Itself Does Not Cause Apical
Periodontitis (E.G., Pain To Percussion Or
Radiographic Evidence Of Osseous Breakdown)
Unless The Canal Is Infected.
Some Teeth May Be Nonresponsive To Pulp
Testing Because Of Calcification Or Recent
History Of Trauma, Or It May Simply Be That
The Tooth Is Not Responding.
As Previously Stated, This Is Why All Testing
Must Be Of A Comparative Nature (E.G., The
Patient May Not Respond To Thermal Testing
On Any Teeth).
6. Previously Treated is a clinical diagnostic category
indicating that the tooth has been endodontically treated and
the canals are obturated with various filling materials (other
than intracanal medicaments).
The tooth typically does not respond to thermal or electric
pulp testing.
7. Previously Initiated Therapy is a clinical diagnostic
category indicating that the tooth has been previously treated
by partial endodontic therapy, such as a pulpotomy or
pulpectomy.
Depending on the level of therapy, the tooth may or may not
respond to pulp testing modalities.
APICAL
DIAGNOSES
1. Normal Apical
Tissues are not sensitive to
percussion or palpation
testing and, radiographically,
the lamina dura surrounding
the root is intact and the
periodontal ligament space is
uniform.
As with pulp testing,
comparative testing for
percussion and palpation
should always begin with
normal teeth as a baseline for
the patient.
2. Symptomatic Apical
Periodontitis represents
inflammation, usually of the apical
periodontium, producing clinical
symptoms involving a painful response
to biting and/or percussion or
palpation.
This may or may not be accompanied
by radiographic changes (i.e., depending
upon the stage of the disease, there
may be normal width of the periodontal
ligament or there may be periapical
radiolucency).
Severe pain to percussion and/or palpation is highly indicative of a
degenerating pulp, and root canal treatment is needed.
3. Asymptomatic
Apical Periodontitis is
inflammation and
destruction of the apical
periodontium that is of
pulpal origin.
It appears as an apical
radiolucency and does not
present clinical symptoms
(e.g., there is no pain on
percussion or palpation).
4. Chronic Apical
Abscess is an inflammatory
reaction to pulpal infection and
necrosis characterized by gradual
onset, little or no discomfort, and an
intermittent discharge of pus through
an associated sinus tract.
Radiographically, there are typically
signs of osseous destruction, such as a
radiolucency.
To identify the source of a draining
sinus tract when present, a gutta-
percha cone is carefully placed
through the Stoma or opening until it
stops and a radiograph is taken.
5. Acute Apical Abscess is
an inflammatory reaction to
pulpal infection and necrosis
characterized by rapid onset,
spontaneous pain, extreme
tenderness of the tooth to
pressure, pus formation, and
swelling of associated
tissues.
There may be no
radiographic signs of
destruction and the patient
often experiences malaise,
fever and lymphadenopathy.
6. Condensing
Osteitis is a diffuse,
radiopaque lesion
representing a localized
bony reaction to a low-
grade inflammatory
stimulus usually seen at
the apex of the tooth.
DIAGNOSTIC
CASE
EXAMPLES
This mandibular right
first molar had been
hypersensitive to
cold and sweets
over the past few
months, but the
symptoms have
subsided.
Now there is no
response to thermal
testing, but there is
tenderness to
biting and pain
upon percussion.
Radiographically, there
are diffuse
radiopacities around
the root apices.
Diagnosis: Pulp necrosis;
symptomatic apical periodontitis
with condensing osteitis.
Nonsurgical endodontic treatment
is indicated, followed by a build-up
and crown.
Over time the condensing osteitis
should regress partially or totally.
Following the placement of
a full gold crown on the
maxillary right second
molar, the patient
complained of sensitivity to
both hot and cold liquids;
now the discomfort is
spontaneous.
Upon application of a pulp
vitality refrigerant spray on
this tooth (i.e., thermal
testing), the patient
experienced pain, and,
upon removal of the
stimulus, the discomfort
lingered for 12 seconds.
Responses to both
percussion and palpation
were normal;
radiographically, there was
no evidence of osseous
changes.
Diagnosis: Symptomatic
irreversible pulpitis; normal
apical tissues.
Nonsurgical endodontic treatment is
indicated; access is to be repaired with a
permanent restoration.
Note that the maxillary
second premolar has
severe distal caries.
Following evaluation,
the tooth was
diagnosed with
symptomatic
irreversible pulpitis
(hypersensitive to cold,
lingering eight
seconds), and
symptomatic apical
periodontitis (pain to
percussion).
The maxillary left first molar has occlusal-mesial caries and the patient has been complaining
of sensitivity to sweets and cold liquids.
There is no discomfort to biting or percussion.
The tooth is hyperresponsive to thermal (cold) testing, with no lingering pain.
Diagnosis: reversible pulpitis; normal
apical tissues.
Treatment would be excavation of the
caries, followed by placement of a
permanent restoration.
If the pulp is exposed, treatment would
be nonsurgical endodontic treatment,
followed by a permanent restoration,
such as a crown.
Mandibular right lateral incisor has an
apical radiolucency that was discovered
during a routine examination.
There was a history of trauma more
than 10 years ago and the tooth was
slightly discolored.
The tooth did not respond to Endo-Ice® or
to the EPT; the adjacent teeth responded
normally to pulp testing.
There was no tenderness to percussion or
palpation in the region.
Diagnosis: pulp necrosis;
asymptomatic apical
periodontitis.
Treatment is nonsurgical endodontic
treatment followed by bleaching and
permanent restoration.
The mandibular left first molar
demonstrates a relatively large
apical radiolucency encompassing
both the mesial and distal roots,
along with furcation involvement.
Periodontal probing depths were
all within normal limits.
The tooth did not respond to
thermal (cold) testing, and both
percussion and palpation elicited
normal responses.
There was a draining sinus
tract on the midfacial of the
attached gingiva, which was
traced with a gutta-percha
cone.
There was recurrent caries
around the distal margin of the
crown.
Diagnosis: pulp necrosis; chronic apical
abscess.
Treatment is crown removal, nonsurgical
endodontic treatment and placement of a
new crown.
The maxillary left first
molar was
endodontically
treated more than 10
years ago.
The patient is
complaining of pain
to biting over the
past three months.
There appear to be
apical radiolucencies
around all three
roots. The tooth was
tender to both
percussion and use of
a fracture-detecting
device.
Diagnosis: previously
treated; symptomatic apical
periodontitis.
Treatment is nonsurgical
endodontic retreatment, followed
by permanent restoration of the
access cavity.
This maxillary left lateral incisor
exhibits an apical radiolucency.
There is no history of pain and
the tooth is asymptomatic.
There is no response to thermal
(cold) testing or electric pulp
testing, whereas the adjacent
teeth respond normally to both
tests.
There is no tenderness to percussion or
palpation.
Diagnosis: pulp necrosis;
asymptomatic apical periodontitis.
Treatment is nonsurgical
endodontic therapy and placement
of a permanent restoration.
Diagnostic Terminology.pdf

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Diagnostic Terminology.pdf

  • 1. Endodontic Diagnosis Dr. Hadil Abdallah Altilbani BDS Santiago de Compostela University Spain. MSc. University of Valencia Spain. Department of Endodontics University of Palestine .
  • 2. Diagnostic Terminology Approved by the American Association of Endodontists and the American Board of Endodontics
  • 3. 1. Normal Pulp is a clinical diagnostic category in which the pulp is Symptom-free and Normally Responsive To Pulp Testing. Although the pulp may not be histologically normal, a “clinically” normal pulp results in a mild or transient response to thermal cold testing, lasting No More Than One To Two Seconds after the stimulus is removed. One cannot arrive at a probable diagnosis without comparing the tooth in question with Adjacent And Contralateral Teeth. It is best to test the adjacent teeth and contralateral teeth first so that the patient is familiar with the experience of a normal response to cold. Pulpal Diagnoses
  • 4.
  • 5. Control Teeth Prior to performing any test, the clinician should select “Control Teeth” This calibrates the test and provides a baseline with which to compare the patient's response Control teeth should be similar to the suspect tooth As referred pain cannot cross the midlline, it may be preferable to select control teeth on the contralateral side The first application of the test is most significant
  • 6. 2. Reversible Pulpitis is based upon subjective and objective findings indicating that the inflammation should resolve and the pulp return to normal following appropriate management of the etiology. Discomfort is experienced when a stimulus, such as cold or sweet, is applied, and goes away within a couple of seconds after the stimulus is removed. Typical etiologies may include exposed dentin (dentinal sensitivity), caries or deep restorations. There are no significant radiographic changes in the periapical region of the suspect tooth, and the pain experienced is not spontaneous. Following the management of the etiology (e.g., caries removal plus restoration or covering the exposed dentin), the tooth requires further evaluation to determine whether the reversible pulpitis has returned to a normal status. Although dentinal sensitivity is not an inflammatory process, per se, all of the symptoms of this entity mimic those of a reversible pulpitis.
  • 7. 3. Symptomatic Irreversible Pulpitis is based on subjective and objective findings that the vital inflamed pulp is incapable of healing and that root canal treatment is indicated. Characteristics may include sharp pain upon thermal stimulus, lingering pain (often 30 seconds or longer after stimulus removal), spontaneity (unprovoked pain) and referred pain. Sometimes, the pain may be accentuated by postural changes, such as lying down or bending over. In addition, over-the-counter analgesics are typically ineffective. Common etiologies may include deep caries, extensive restorations, or fractures exposing the pulpal tissues. Teeth with symptomatic irreversible pulpitis may be Difficult To Diagnose because the inflammation has not yet reached the periapical tissues, thus resulting in no pain or discomfort to percussion. In such cases, the patient’s dental history and thermal testing are the primary tools for assessing pulpal status.
  • 8. 4. Asymptomatic Irreversible Pulpitis is a clinical diagnosis based on subjective and objective findings indicating that the vital inflamed pulp is incapable of healing and that root canal treatment is indicated. These cases have no clinical symptoms and usually respond normally to thermal testing, but may have had trauma or deep caries that would likely result in exposure following removal.
  • 9. 5. Pulp Necrosis Is A Clinical Diagnostic Category Indicating Death Of The Dental Pulp, Necessitating Root Canal Treatment. The Pulp Is Nonresponsive To Pulp Testing And Is Asymptomatic. Pulp Necrosis By Itself Does Not Cause Apical Periodontitis (E.G., Pain To Percussion Or Radiographic Evidence Of Osseous Breakdown) Unless The Canal Is Infected. Some Teeth May Be Nonresponsive To Pulp Testing Because Of Calcification Or Recent History Of Trauma, Or It May Simply Be That The Tooth Is Not Responding. As Previously Stated, This Is Why All Testing Must Be Of A Comparative Nature (E.G., The Patient May Not Respond To Thermal Testing On Any Teeth).
  • 10. 6. Previously Treated is a clinical diagnostic category indicating that the tooth has been endodontically treated and the canals are obturated with various filling materials (other than intracanal medicaments). The tooth typically does not respond to thermal or electric pulp testing. 7. Previously Initiated Therapy is a clinical diagnostic category indicating that the tooth has been previously treated by partial endodontic therapy, such as a pulpotomy or pulpectomy. Depending on the level of therapy, the tooth may or may not respond to pulp testing modalities.
  • 12. 1. Normal Apical Tissues are not sensitive to percussion or palpation testing and, radiographically, the lamina dura surrounding the root is intact and the periodontal ligament space is uniform. As with pulp testing, comparative testing for percussion and palpation should always begin with normal teeth as a baseline for the patient.
  • 13. 2. Symptomatic Apical Periodontitis represents inflammation, usually of the apical periodontium, producing clinical symptoms involving a painful response to biting and/or percussion or palpation. This may or may not be accompanied by radiographic changes (i.e., depending upon the stage of the disease, there may be normal width of the periodontal ligament or there may be periapical radiolucency). Severe pain to percussion and/or palpation is highly indicative of a degenerating pulp, and root canal treatment is needed.
  • 14. 3. Asymptomatic Apical Periodontitis is inflammation and destruction of the apical periodontium that is of pulpal origin. It appears as an apical radiolucency and does not present clinical symptoms (e.g., there is no pain on percussion or palpation).
  • 15. 4. Chronic Apical Abscess is an inflammatory reaction to pulpal infection and necrosis characterized by gradual onset, little or no discomfort, and an intermittent discharge of pus through an associated sinus tract. Radiographically, there are typically signs of osseous destruction, such as a radiolucency. To identify the source of a draining sinus tract when present, a gutta- percha cone is carefully placed through the Stoma or opening until it stops and a radiograph is taken.
  • 16. 5. Acute Apical Abscess is an inflammatory reaction to pulpal infection and necrosis characterized by rapid onset, spontaneous pain, extreme tenderness of the tooth to pressure, pus formation, and swelling of associated tissues. There may be no radiographic signs of destruction and the patient often experiences malaise, fever and lymphadenopathy.
  • 17. 6. Condensing Osteitis is a diffuse, radiopaque lesion representing a localized bony reaction to a low- grade inflammatory stimulus usually seen at the apex of the tooth.
  • 19. This mandibular right first molar had been hypersensitive to cold and sweets over the past few months, but the symptoms have subsided. Now there is no response to thermal testing, but there is tenderness to biting and pain upon percussion. Radiographically, there are diffuse radiopacities around the root apices.
  • 20. Diagnosis: Pulp necrosis; symptomatic apical periodontitis with condensing osteitis. Nonsurgical endodontic treatment is indicated, followed by a build-up and crown. Over time the condensing osteitis should regress partially or totally.
  • 21. Following the placement of a full gold crown on the maxillary right second molar, the patient complained of sensitivity to both hot and cold liquids; now the discomfort is spontaneous. Upon application of a pulp vitality refrigerant spray on this tooth (i.e., thermal testing), the patient experienced pain, and, upon removal of the stimulus, the discomfort lingered for 12 seconds. Responses to both percussion and palpation were normal; radiographically, there was no evidence of osseous changes.
  • 22. Diagnosis: Symptomatic irreversible pulpitis; normal apical tissues. Nonsurgical endodontic treatment is indicated; access is to be repaired with a permanent restoration.
  • 23. Note that the maxillary second premolar has severe distal caries. Following evaluation, the tooth was diagnosed with symptomatic irreversible pulpitis (hypersensitive to cold, lingering eight seconds), and symptomatic apical periodontitis (pain to percussion).
  • 24. The maxillary left first molar has occlusal-mesial caries and the patient has been complaining of sensitivity to sweets and cold liquids. There is no discomfort to biting or percussion. The tooth is hyperresponsive to thermal (cold) testing, with no lingering pain.
  • 25. Diagnosis: reversible pulpitis; normal apical tissues. Treatment would be excavation of the caries, followed by placement of a permanent restoration. If the pulp is exposed, treatment would be nonsurgical endodontic treatment, followed by a permanent restoration, such as a crown.
  • 26. Mandibular right lateral incisor has an apical radiolucency that was discovered during a routine examination. There was a history of trauma more than 10 years ago and the tooth was slightly discolored. The tooth did not respond to Endo-Ice® or to the EPT; the adjacent teeth responded normally to pulp testing. There was no tenderness to percussion or palpation in the region.
  • 27. Diagnosis: pulp necrosis; asymptomatic apical periodontitis. Treatment is nonsurgical endodontic treatment followed by bleaching and permanent restoration.
  • 28. The mandibular left first molar demonstrates a relatively large apical radiolucency encompassing both the mesial and distal roots, along with furcation involvement. Periodontal probing depths were all within normal limits. The tooth did not respond to thermal (cold) testing, and both percussion and palpation elicited normal responses. There was a draining sinus tract on the midfacial of the attached gingiva, which was traced with a gutta-percha cone. There was recurrent caries around the distal margin of the crown.
  • 29. Diagnosis: pulp necrosis; chronic apical abscess. Treatment is crown removal, nonsurgical endodontic treatment and placement of a new crown.
  • 30. The maxillary left first molar was endodontically treated more than 10 years ago. The patient is complaining of pain to biting over the past three months. There appear to be apical radiolucencies around all three roots. The tooth was tender to both percussion and use of a fracture-detecting device.
  • 31. Diagnosis: previously treated; symptomatic apical periodontitis. Treatment is nonsurgical endodontic retreatment, followed by permanent restoration of the access cavity.
  • 32. This maxillary left lateral incisor exhibits an apical radiolucency. There is no history of pain and the tooth is asymptomatic. There is no response to thermal (cold) testing or electric pulp testing, whereas the adjacent teeth respond normally to both tests. There is no tenderness to percussion or palpation.
  • 33. Diagnosis: pulp necrosis; asymptomatic apical periodontitis. Treatment is nonsurgical endodontic therapy and placement of a permanent restoration.