The document discusses renal stones (nephrolithiasis), including their definition, etiology, risk factors, pathophysiology, types, clinical manifestations, diagnostic studies, management, nursing management, prevention, and questions. Renal stones are formed by crystallization of substances in the urine, and can be caused by metabolic, lifestyle, or genetic factors. Common types include calcium oxalate, calcium phosphate, uric acid, cystine, and struvite stones. Treatment may involve medical management, extracorporeal shock wave lithotripsy (ESWL), or surgical procedures like ureteroscopy. Nursing focuses on pain management, education, and preventing infection and recurrence.
4. DEFINITION
• Nephrolithiasis refers to renal stone
disease; urolithiasis refers to the
presence of stones in the urinary
system. Stones, or calculi, are formed
in the urinary tract from the kidney
to bladder by the crystallization of
substances excreted in the urine
•
Edited by:- MR. ROMAN BAJRANG
8. RISK FACTORS
HIGH MINERAL
CONTENT IN DRINKING
WATER
DIETARY INTAKE
UTI & H/O FEMALE
GENITAL MUTILATION
PROLONGED
INDWELLING
CATHETERISATION
NEUROGENIC BLADDER
Editedby:-MR.ROMANBAJRANG
9. PATHOPHYSIOLOGY
• Slow urine flow, resulting in
supersaturation of the urine
with the particular element
that first become crystallized
and later become stone
23. COMPLIMENTARY THERAPY
•Hypnosis, imagery, therapeutic
or healing touch, acupuncture
and breathing techniques
•Positioning the client to
comfortable position aids in pain
reduction
24. OTHER TECHNIQUES
• Avoiding over
hydration and
under hydration
• Strain the urine
• Send any strained
stone to laboratory
to aid in preventive
treatment in the
future
29. SPECIFIC APPROACHES
URINARY
STONE
CHARACTERISTICS PREDISPOSING
FACTORS
THERAPEUTIC MEASURES
Cystine
1-2 %
Genetic
autosomal
recessive
defect,defective
absorption of gi
cystine from gi
tract and kidney
excess
concentrations
causing stone
formation
Acid urine Increase hydration
Give α pencillamine and
tiopronin to prevent cystine
crystallization
Potassium citrate to
alkaline urine
37. NURSING DIAGNOSIS
• Acute pain related to irritation
and spasm from stone
movement in the urinary tract as
manifested by complaints of
pain, facial grimacing,
restlessness
38. • Anxiety related to uncertain
outcome and lack of
knowledge regarding possible
surgery as manifested by
expressions
NURSING DIAGNOSIS
39. • Ineffective therapeutic regimen
management related to lack of
knowledge as manifested by
repeated questions
NURSING DIAGNOSIS
40. • Impaired urinary elimination
related to trauma or blockage of
ureters or urethra as manifested
by decreased urinary output and
bloody urine
NURSING DIAGNOSIS
41. • Risk for infection related to
introduction of bacteria
following manipulations of the
urinary tract and obstructed
urinary blood flow
NURSING DIAGNOSIS
42. PREVENTION
• Avoid protein intake; usually protein is
restricted to 60g/day to decrease urinary
excretion of calcium and uric acid.
• A sodium intake of 3 to 4 g/day is
recommended.Table salt and high-sodium
foods should be reduced, because sodium
competes with calcium for reabsorption
in the kidneys.
43. • Low-calcium diets are not generally
recommended,except for true absorptive
hypercalciuria. Evidence shows thatlimiting
calcium, especially in women, can lead to
osteoporosis and does not prevent renal
stones.
• Avoid intake of oxalate-containing foods
(eg,spinach,strawberries,rhubarb,tea,peanuts,
wheat bran).
PREVENTION
44. • During the day,drink fluids (ideally water)
every1 to 2 hours.
• Drink two glasses of water at bedtime
and an additional glass at each nighttime
awakening to prevent urine from
becoming too concentrated during the
night.
PREVENTION
45. • Avoid activities leading to sudden
increases in environmental temperatures
that may cause excessive sweating and
dehydration.
• Contact your primary health care
provider at the first sign of a urinary
tract infection
PREVENTION