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Dr Mukhilesh R M.S
Salient features of Head Injury

Most common cause of death
following road traffic
accidents.
Outcome of traumatic brain
injury
not only personal loss
neuro-reahabilitaion
long term nursing care
supportive care
Pathophysiology of brain Injury
Cerebral autoregulation
normal blood flow – 55ml/100g/min
mean arterial pressure 50-150 mmHg
Monroker Kelle Doctrine ????
Primary vs Secondary brain Injury

Primary brain Injury
At the time of impact
Brainstem and hemispheric contusions
Diffuse axonal injury
Cortical lacerations
Primary vs Secondary brain Injury

Secondary brain Injury

sometime after the impact
often preventable
causes
Hypoxia PO2<8kPa
Hypotension SBP<90mmhg
Raised ICP >20mmhg
Pyrexia
Cerebral Perfusion Pressure <65mmhg
Metabolic Disturbances
Classification Of Head Injury
Blunt vs Penetrating
Morphological
Skull Fractures
Vault –
open / closed
linear / communited
depressed / non depressed
Base of skull fracture
Intracranial Hematoma
EDH / SDH/ SAH/ ICH
Clinical Features

Rule out multisystem injury esp spinal injury
Rule out non accidental causes of collapse
syncope
aneurysmal SAH
hypoglycemia
electrolyte imbalance
medications and drug abuse
Clinical Features

A – Airway
B- Breathing
C- Circulation
D- Disability assesment
Pupils and GCS
Glasgow Coma Scale - GCS
Racoon Eyes

Battle’s
Sign
Dilated
Pupil
NICE guideline for computed tomography (CT)

GCS <13
Focal Neurological deficit
Suspected open, depressed or basal skull fracture
Seizure
Vomiting >1 episode
Urgent CT even if none of the above
Age>65
coagulopathy
antegrade amnesia >30 min
Extradural hematoma

Neurosurgical emergency

Associated with skull fracture
Tearing of meningeal artery

PTERION – middle meningeal
Can also be dural venous bleed

LUCID INTERVAL
Extradural hematoma

LENTIFORM
SHAPE

MASS EFFECT
Management

Burr Hole evacuation of hematoma
Subdural Hematoma
disruption of cortical
vessel or brain laceration

significant primary brain
injury
impaired conscious level
diffuse and concave
appearance in CT

Poor prognosis
CONVEX SHAPE
Clinical Features

Small hematomas with
little mass effect –
conservative
management
Surgery inappropriate
best GCS
pupillary reactivity
age
anticoagulant drugs
Subarachnoid hemorrhage

Aneurysms and trauma
Traumatic SAH –
conservative Rx
Chronic SDH

Elderly / anticoagulant
Tear in small bridging veins and samll ASDH – silent
Hematoma breaks down – mass effect
Headache / ICT increased/ focal deficits
Rx – evacuation via Burr Hole
Cerebral Contusions
Coup injury

Contre coup
injury
ICP
Cerebral perfusion pressure
= MAP- ICP

CPP to be maintained
>65mmhg
ICP >20 mmhg – poor
outcome
ICP monitoring – ventricular
or parenchymal
ICP

Sedation

Diuretics
Thermoregulation – pyrexia increases brain
metabolism
Barbiturates – thiopentones
Seizure control

Decompressive craniectomy
Skull fractures

Vault fractures

elevation of depressed segments
wound debridement
Base of skull fractures
CSf rhinorhoea or otorrhoea
anterior fossa dural repair
Long term sequelae

Neurorehabilitation
Pyschological support
Seizures
TRAUMATIC BRIN INJURY IS A PREVENTABLE CAUSE
Head injury

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Head injury