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Joint imaging
By
Dr. Firas Abdullah
Aims of our lecture:
🠶Radiological signs of joint disease
🠶Diagnosis of arthritis
🠶Different types of arthritis
🠶Other joint pathology
🠶MRI of knee and shoulder
Plain film signs of joint disease
Joint space narrowing: due to destruction of articular
cartilage. Itoccurs in practically all forms of joint
disease, except avascular necrosis.
Soft tissue swelling: a feature of inflammatory, and
particularly infective arthritis. Also can be seen in
gouty tophi.
Osteoporosis: painful conditions and underuse of the
bones. E.g. rheumatoid and tuberculous arthritis.
Articular erosions: destruction of the articular cortex and
the adjacent trabecular bone
Causes:
1 Inflammatory overgrowth of the synovium (pannus)
• Rheumatoid arthritis , commonest
• Juvenile rheumatoid arthritis (Still’s disease)
• Psoriasis
• Reiter’sdisease
• Ankylosing spondylitis
• Tuberculosis.
2 Deposition of urate crystalsin gout.
3 Infection: pyogenic arthritis and tuberculosis.
4Repeated hemorrhage in hemophilia
5- Neoplastic, e.g. synovial sarcoma.
Osteophytes, subchondral sclerosis and cysts:
Features of osteoarthritis. A characteristic increase
in the density of subchondral bone isseen in
avascular necrosis
Alteration in the shape of the joint:slipped
epiphysis, developmental dysplasia of the hip,
osteochondritis dissecans and avascular necrosis in
its later stages.
Joint erosion
Diagnosis of arthritis
1. Whether one or more than one joint involved? e.g.
rheumatoid arthritis, infections and synovial tumours.
2. Which joints are involved?
 Rheumatoid arthritis
 Psoriatic arthritis
 Gout characteristically
 Osteoarthritis
 Neuropathic arthritis
3. Isa known disease present? e.g. haemophilia or
diabetes.
Rheumatoid arthritis
A polyarthritis caused by inflammatory overgrowth of synovium
known as pannus.
The earliest change is periarticular soft tissue swelling and
osteoporosis.
Joint space narrowing.
Initially small bony erosions, at the joint margins. Later, extensive
erosions
Metatarso-or metacarpophalangeal joints, proximal
interphalangeal joints and on the styloid process of the ulna.
Advance changes: Ulnar deviation. Arthritis mutilans.
Rheumatoid arthritis
With severe disease, there may be subluxation at the
atlantoaxial joint, possibility of neurological symptoms from
compression of the spinal cord by the odontoid process
A widespread erosive arthropathy isalmost diagnostic of
rheumatoid arthritis.
Osteoarthritis
commonest form of arthritis.
The hip and the knee are frequently involved, the ankle elbow
are infrequently affected.
The wrist, joints of the hand and the metatarsophalangeal joint
of the big toe are also frequently involved.
Radiological features:
 Joint space narrowing.
 Osteophytes
 Subchondral sclerosis
 Subchondral cysts
 Loose bodies
Osteoarthritis
Comparison of osteoarthritis and rheumatoid arthritis
Radiological feature Osteoarthritis Rheumatoid arthritis
Joint space narrowing Maximal at
weight- bearing
site
Uniform
Erosions Not occur Is a characteristic feature
Subchondral sclerosis
and cysts
Seen Not a feature
Sclerosis Prominent feature Not a feature
Osteoporosis Not occur Often present
Joint involved Knee, hip
Metacarpophalangeal
Distal interphalangeal
Metacarpophalangeal
Proximal
interphalangeal
Gouty Arthritis
Most commonly affects the metatarsophalangeal joint of
the big toe.
The earliest change issoft tissue swelling
Erosions have a well-defined, often sclerotic overhanging
edge
Usually no osteoporosis
Localized soft tissue lumps, known as tophi, may occur in
the periarticular and occasionally show calcification.
Gouty Arthritis
T
ophi
Joint Infection
Pyogenic bacterial infection or tuberculosis
In pyogenic arthritis, Staphylococcus aureus, there is rapid
destruction of the articular cartilage & subchondral bone
and a soft tissue swelling. A joint effusion isthe earliest
finding
TBarthritis, The hip and knee are the most commonly
affected. Joint space narrowing and erosions, articular
cortex destruction, and striking osteoporosis,
pyogenic arthritis TBarthritis
Neuropathic joint (Charcot joint)
6 Ds of Charcot joint:
 Increased Density (subchondral sclerosis)
 Destruction
 Debris (intra-articular loose bodies)
 Dislocation
 Distention
 Disorganisation
Charcot joint
Avascular (aseptic) necrosis
Causes:
 steroid therapy
 Collagen vascular diseases
 Radiation therapy
 Sickle cell anemia
 Exposure to high pressure environments
 Fractures.
T
he radiographic features:
 Increased density of the subchondral bone with irregularity of the
articular contour or fragmentation of the bone
 A characteristic crescentic lucent line just beneath the articular
cortex.
 The cartilage space is preserved until secondary degenerative
changes supervene.
Osteochondrosis
A group of disorders that affect the progress of bone
growth by bone necrosis. It is only seen in children and
adolescents who are still growing.
 Perthe's disease:femora head
 Freiberg’s disease:metatarsal heads
 Kohler’s disease:navicular bone of the foot
 Osgood–Schlatter’s disease:tibial tuberosity
 Kienböck’s disease: lunate bone in the wrist
Perthe's disease
Perthe's disease
Kienböck’sdisease
Developmental dysplasia of the hip (DDH)
Abnormal development of the hip joint resulting from an
abnormal relationship of the femoral head to the
acetabulum. There isa clear female predominance, and it
usually occurs from ligamentous laxity and abnormal position
in utero. Therefore, it ismore common with oligohydramnios
pregnancies.
Risk factors include:
• female gender (M: F ratio ~1:8)
• firstborn baby
• family history
• breech presentation
• oligohydramnios
Investigation used for diagnosis:
 Ultrasound at early infancy
 X ray later in life
 The features:lateral and upper displacement
of the head of the femur. Increased slope to
the acetabular roof
BestRegards

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  • 2. Aims of our lecture: 🠶Radiological signs of joint disease 🠶Diagnosis of arthritis 🠶Different types of arthritis 🠶Other joint pathology 🠶MRI of knee and shoulder
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  • 4. Plain film signs of joint disease Joint space narrowing: due to destruction of articular cartilage. Itoccurs in practically all forms of joint disease, except avascular necrosis. Soft tissue swelling: a feature of inflammatory, and particularly infective arthritis. Also can be seen in gouty tophi. Osteoporosis: painful conditions and underuse of the bones. E.g. rheumatoid and tuberculous arthritis.
  • 5. Articular erosions: destruction of the articular cortex and the adjacent trabecular bone Causes: 1 Inflammatory overgrowth of the synovium (pannus) • Rheumatoid arthritis , commonest • Juvenile rheumatoid arthritis (Still’s disease) • Psoriasis • Reiter’sdisease • Ankylosing spondylitis • Tuberculosis. 2 Deposition of urate crystalsin gout. 3 Infection: pyogenic arthritis and tuberculosis. 4Repeated hemorrhage in hemophilia 5- Neoplastic, e.g. synovial sarcoma.
  • 6. Osteophytes, subchondral sclerosis and cysts: Features of osteoarthritis. A characteristic increase in the density of subchondral bone isseen in avascular necrosis Alteration in the shape of the joint:slipped epiphysis, developmental dysplasia of the hip, osteochondritis dissecans and avascular necrosis in its later stages.
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  • 10. Diagnosis of arthritis 1. Whether one or more than one joint involved? e.g. rheumatoid arthritis, infections and synovial tumours. 2. Which joints are involved?  Rheumatoid arthritis  Psoriatic arthritis  Gout characteristically  Osteoarthritis  Neuropathic arthritis 3. Isa known disease present? e.g. haemophilia or diabetes.
  • 11. Rheumatoid arthritis A polyarthritis caused by inflammatory overgrowth of synovium known as pannus. The earliest change is periarticular soft tissue swelling and osteoporosis. Joint space narrowing. Initially small bony erosions, at the joint margins. Later, extensive erosions Metatarso-or metacarpophalangeal joints, proximal interphalangeal joints and on the styloid process of the ulna. Advance changes: Ulnar deviation. Arthritis mutilans.
  • 12. Rheumatoid arthritis With severe disease, there may be subluxation at the atlantoaxial joint, possibility of neurological symptoms from compression of the spinal cord by the odontoid process A widespread erosive arthropathy isalmost diagnostic of rheumatoid arthritis.
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  • 14. Osteoarthritis commonest form of arthritis. The hip and the knee are frequently involved, the ankle elbow are infrequently affected. The wrist, joints of the hand and the metatarsophalangeal joint of the big toe are also frequently involved. Radiological features:  Joint space narrowing.  Osteophytes  Subchondral sclerosis  Subchondral cysts  Loose bodies
  • 16. Comparison of osteoarthritis and rheumatoid arthritis Radiological feature Osteoarthritis Rheumatoid arthritis Joint space narrowing Maximal at weight- bearing site Uniform Erosions Not occur Is a characteristic feature Subchondral sclerosis and cysts Seen Not a feature Sclerosis Prominent feature Not a feature Osteoporosis Not occur Often present Joint involved Knee, hip Metacarpophalangeal Distal interphalangeal Metacarpophalangeal Proximal interphalangeal
  • 17. Gouty Arthritis Most commonly affects the metatarsophalangeal joint of the big toe. The earliest change issoft tissue swelling Erosions have a well-defined, often sclerotic overhanging edge Usually no osteoporosis Localized soft tissue lumps, known as tophi, may occur in the periarticular and occasionally show calcification.
  • 19. Joint Infection Pyogenic bacterial infection or tuberculosis In pyogenic arthritis, Staphylococcus aureus, there is rapid destruction of the articular cartilage & subchondral bone and a soft tissue swelling. A joint effusion isthe earliest finding TBarthritis, The hip and knee are the most commonly affected. Joint space narrowing and erosions, articular cortex destruction, and striking osteoporosis,
  • 21. Neuropathic joint (Charcot joint) 6 Ds of Charcot joint:  Increased Density (subchondral sclerosis)  Destruction  Debris (intra-articular loose bodies)  Dislocation  Distention  Disorganisation
  • 23. Avascular (aseptic) necrosis Causes:  steroid therapy  Collagen vascular diseases  Radiation therapy  Sickle cell anemia  Exposure to high pressure environments  Fractures. T he radiographic features:  Increased density of the subchondral bone with irregularity of the articular contour or fragmentation of the bone  A characteristic crescentic lucent line just beneath the articular cortex.  The cartilage space is preserved until secondary degenerative changes supervene.
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  • 26. Osteochondrosis A group of disorders that affect the progress of bone growth by bone necrosis. It is only seen in children and adolescents who are still growing.  Perthe's disease:femora head  Freiberg’s disease:metatarsal heads  Kohler’s disease:navicular bone of the foot  Osgood–Schlatter’s disease:tibial tuberosity  Kienböck’s disease: lunate bone in the wrist
  • 29. Developmental dysplasia of the hip (DDH) Abnormal development of the hip joint resulting from an abnormal relationship of the femoral head to the acetabulum. There isa clear female predominance, and it usually occurs from ligamentous laxity and abnormal position in utero. Therefore, it ismore common with oligohydramnios pregnancies. Risk factors include: • female gender (M: F ratio ~1:8) • firstborn baby • family history • breech presentation • oligohydramnios
  • 30. Investigation used for diagnosis:  Ultrasound at early infancy  X ray later in life  The features:lateral and upper displacement of the head of the femur. Increased slope to the acetabular roof
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