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BOWEL OBSTRUCTION
By:- Dr Yuvraj(M.S.)
Associate professor
General Surgery
BOWEL OBSTRUCTION OVERVIEW
🠶 CLASSI
FICATI
ON
🠶 COMMON CAUSESOF OBSTRUCTION
🠶 CLINICAL FEATURES
🠶 I
NVES
TI
GATI
ON
🠶 TREATMENT
INTRODUCTION
🠶 Accountsfor 5%of all acute surgical admissions
🠶 Patients are often extremely ill requiring prompt assessment, resuscitation
and intensive monitoring
🠶 Types:
🠶 Obstruction:
🠶 A mechanical blockage arising from a structural abnormality that presents
a physical barrier to the progression of gut contents.
🠶 Ileus:
🠶 is a paralytic or functional variety of obstruction
🠶 Obstruction is:
🠶 Partial or complete
🠶 Simple or strangulated
CLASSIFICATION
R
R
e
e
s
s
u
u
l
l
t
tf
f
r
r
o
o
m
m
a
a
t
t
o
o
n
n
y
y
o
o
f
ft
t
h
h
e
e
i
i
n
n
t
t
e
e
s
s
t
t
i
i
n
n
e
ew
w
i
i
t
t
h
hl
l
o
o
s
s
s
so
o
f
f
normal ppeerriistalsis, in the
absence of a mecchaniccaall
cause.
o
or
r i
it
t m
ma
ay
y b
be
e p
pr
re
es
se
en
nt
t
in a non-
propulsive f
fo
or
rm
m (
(e
e.
.g
g.
. m
me
es
se
en
nt
te
er
ri
ic
c
p
vascular occlusion or
A
A
D
D
Y
Y
N
N
A
A
M
M
I
I
C
C
(FUNCTIONAL)
TYPESOF BOWEL OBSTRUCTION
TYPES AND CAUSESOF DYNAMIC
OBSTRUCTION
Intraluminal
•Impaction
•Foreign bodies
•Bezoars
•Gallstone
Intramural
•Congenital atresia
•Stricture
•Malignancy(15%)
Extramural
•Bands/
adhesion(40%)
•Hernia (12%)
•Volvulus
•Intussusception
•Tumor-
benign/malignant
🠶 Peritoneal irritation local fibrin production produces adhesions between
apposed surfaces
🠶 As early as 4 weeks post laparotomy. The majority of patients present
between 1-5 years
Colorectal Surgery 25%
Gynaecological 20%
Appendectomy 14%
🠶 Prevention: good surgical technique, washing of the peritoneal cavity with
saline to remove clots, etc, minimizing contact w/ gauze, covering
anastomosis& raw peritoneal surfaces
TREATMENT OF
ADHESIVE OBSTRUCTION
Initially treat conservatively provided there is no signs of
strangulation; should rarely continue conservative
treatment for longer than 72 hours
At operation, divide only the causative adhesion and
limit dissection
Laparoscopic adhesiolysis in cases of chronic
subacute obstruction
Hernia
🠶 Accounts for 20%of SBO
🠶 Commonest1. Femoral hernia
2. IDinguinal
3. Umbilical
4. Others: incisional
🠶 The site of obstruction is the neck of hernia
🠶 The compromised viscus is with in the sac.
🠶 Ischaemia occurs initially by venous occlusion, followed by oedema and
arterial compromise.
🠶 Attempt to distinguish the difference between:
🠶 Incarceration
🠶 Sliding
🠶 Obstruction
🠶 Strangulation is noted by:
🠶 Persistent pain
🠶 Discolouration
🠶 Tenderness
🠶 Constitutional symptoms
Volvulus
A twisting or axial rotation of
a portion of bowel about its
mesentery. When complete it
formsa closed loop
obstruction ischemia
Commonest spontaneous
type in adult issigmoid, can
be relieved by
decompression per anum
Surgery isrequired to prevent
or relieve ischaemia
Features: palpable tympanic lump
(sausage shape) in the midline or
left side of abdomen.
Constipation, abdominal
distension (early & progressive)
ACUTE INTUSSUSEPTION
Occurs when one portion of the gut becomes invaginated within an
immediately adjacent segment.
Common in 1st yearof life
Common after viral illness enlargement of Peyer’s patches
Ileocolic isthe commonest variety in child.
Colocolic intussusception commonest in adult
🠶 An intussusception iscomposed of three parts:
🠶 the entering or innertube;
🠶 the returning or middle tube;
🠶 the sheath or outer tube (intussuscipiens).
🠶 Classically, a previously healthy infant presents
with colicky pain and vomiting (milk then bile).
🠶 Between episodes the child initially appears well.
🠶 Later
, they may passa ‘redcurrant jelly’ stool.
LARGE BOWEL OBSTRUCTION
🠶 Distinguishing ileus from mechanical obstruction is challenging
🠶 Caecum is at the greatest risk of perforation
🠶 Perforation results in the release of formed feaces with heavy bacterial
contamination
Aetiology:
1. Carcinoma:
The commonest cause, 18%of colonic ca. present with obstruction
2. Benign stricture:
Due to Diverticular disease, Ischemia, Inflammatory bowel disease.
3. Volvulus:
-Sigmoid Volvulus/ Caecal Volvulus
4. Hernia.
5. Congenital : HirschPrung, anal stenosis and agenesis
CLINICAL FEATURES
Large bowel obstruction
distension is early and pronounced.
Pain is mild and vomiting and dehydration are late.
The proximal colon and caecum are distended on abdominal radiography
CARDINAL FEATURES:
Colicky pain
Vomiting
Abd distention
Constipation
OTHER FEATURES:
Dehydration
Hypokalaemia
Pyrexia
Abd tenderness
PHYSICAL EXAMINATION
INSPECTION
Abdominal distention, scars, visible peristalsis.
PALPATION
Mass, tenderness, guarding
PERCUSSION
T
ymphanic, dullness
AUSCULTATION
Bowel sound are high pitch and increase in frequency
INVESTIGATIONS:
🠶 Lab:
🠶 FBC (leukocytosis, anaemia, hematocrit, platelets)
🠶 Clotting profile
🠶 Arterial blood gasses
🠶 U& Crt, Na, K, Amylase, LFTand glucose, LDH
🠶 Group and save (x-match if needed)
🠶 Optional (ESR, CRP, Hepatitis profile)
🠶 RadiOlogical:
🠶 Plain ABDOMINAL xrays
🠶 USS ( free fluid, masses, mucosal folds, pattern of paristalsis, Doppler of mesenteric
vasulature, solid organs)
🠶 Other advanced studies (CT, MRI, Contrast studieS)
Fluid levels with gas above;
‘stepladder pattern’. Ileal
obstruction by adhesions;
patient erect.
Supine radiograph from a patient with
complete small bowel obstruction
shows distended small bowel loops in
the central abdomen with prominent
valvulae conniventes (small white
arrow)
Figure 3. Lateral decubitus
view of the abdomen, showing
air-fluid levels consistent with
intestinal obstruction (arrows).
🠶 In small bowel
🠶 Central 3cm thick
diameter
🠶 Vulvulae coniventae
🠶 Ileum may occur
tubeless
🠶 In large bowel
🠶 Peripheral diameter6cm
🠶 Presence of haustration
ROLE OF CT
🠶
🠶
Used with iv contrast, oral and rectal
contrast (triple contrast).
Able to demonstrate abnormality in the
bowel wall, mesentery, mesenteric vessels
and peritoneum.
🠶 It can define:
🠶 the level of obstruction
🠶 The degree of obstruction
🠶 The cause: volvulus, hernia, luminal and
mural causes
🠶 The degree of ischaemia
🠶 Free fluid and gas
🠶 Ensure: patient vitally stable with no renal
failure and no previous alergy to iodine
• FIGURE: AXIAL COMPUTED TOMOGRAPHY SCAN SHOWING
DILATED, CONTRAST-FILLED LOOPS OF BOWEL ON THE
PATIENT’S LEFT (YELLOW ARROWS), WITH DECOMPRESSED
DISTAL SMALL BOWEL ON THE PATIENT’S RIGHT (RED
ARROWS). THE CAUSE OF OBSTRUCTION, AN
INCARCERATED UMBILICAL HERNIA, CAN ALSO BE SEEN
(GREEN ARROW), WITH PROXIMALLY DILATED BOWEL
ENTERING THE HERNIA AND DECOMPRESSED BOWEL
EXITING THE HERNIA.
CONTRAST STUDIES
Barium should not be used in
a patient with peritonitis
🠶 As: follow through, enema
🠶 Limited use in the acute setting
🠶 Gastrografin is used in acute
abdomen but is diluted
🠶 Useful in recurrent and chronic
obstruction
🠶 May able to define the level and
mural causes.
🠶 Can be used to distinguish
adynamic and mechanical
obstruction
bowelobstruction1-150701160238-lva1-app6891 copy.pdf

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bowelobstruction1-150701160238-lva1-app6891 copy.pdf

  • 1. BOWEL OBSTRUCTION By:- Dr Yuvraj(M.S.) Associate professor General Surgery
  • 2. BOWEL OBSTRUCTION OVERVIEW 🠶 CLASSI FICATI ON 🠶 COMMON CAUSESOF OBSTRUCTION 🠶 CLINICAL FEATURES 🠶 I NVES TI GATI ON 🠶 TREATMENT
  • 3. INTRODUCTION 🠶 Accountsfor 5%of all acute surgical admissions 🠶 Patients are often extremely ill requiring prompt assessment, resuscitation and intensive monitoring 🠶 Types: 🠶 Obstruction: 🠶 A mechanical blockage arising from a structural abnormality that presents a physical barrier to the progression of gut contents. 🠶 Ileus: 🠶 is a paralytic or functional variety of obstruction 🠶 Obstruction is: 🠶 Partial or complete 🠶 Simple or strangulated
  • 4. CLASSIFICATION R R e e s s u u l l t tf f r r o o m m a a t t o o n n y y o o f ft t h h e e i i n n t t e e s s t t i i n n e ew w i i t t h hl l o o s s s so o f f normal ppeerriistalsis, in the absence of a mecchaniccaall cause. o or r i it t m ma ay y b be e p pr re es se en nt t in a non- propulsive f fo or rm m ( (e e. .g g. . m me es se en nt te er ri ic c p vascular occlusion or A A D D Y Y N N A A M M I I C C (FUNCTIONAL)
  • 6. TYPES AND CAUSESOF DYNAMIC OBSTRUCTION Intraluminal •Impaction •Foreign bodies •Bezoars •Gallstone Intramural •Congenital atresia •Stricture •Malignancy(15%) Extramural •Bands/ adhesion(40%) •Hernia (12%) •Volvulus •Intussusception •Tumor- benign/malignant
  • 7.
  • 8. 🠶 Peritoneal irritation local fibrin production produces adhesions between apposed surfaces 🠶 As early as 4 weeks post laparotomy. The majority of patients present between 1-5 years Colorectal Surgery 25% Gynaecological 20% Appendectomy 14% 🠶 Prevention: good surgical technique, washing of the peritoneal cavity with saline to remove clots, etc, minimizing contact w/ gauze, covering anastomosis& raw peritoneal surfaces
  • 9. TREATMENT OF ADHESIVE OBSTRUCTION Initially treat conservatively provided there is no signs of strangulation; should rarely continue conservative treatment for longer than 72 hours At operation, divide only the causative adhesion and limit dissection Laparoscopic adhesiolysis in cases of chronic subacute obstruction
  • 10. Hernia 🠶 Accounts for 20%of SBO 🠶 Commonest1. Femoral hernia 2. IDinguinal 3. Umbilical 4. Others: incisional 🠶 The site of obstruction is the neck of hernia 🠶 The compromised viscus is with in the sac. 🠶 Ischaemia occurs initially by venous occlusion, followed by oedema and arterial compromise. 🠶 Attempt to distinguish the difference between: 🠶 Incarceration 🠶 Sliding 🠶 Obstruction 🠶 Strangulation is noted by: 🠶 Persistent pain 🠶 Discolouration 🠶 Tenderness 🠶 Constitutional symptoms
  • 11. Volvulus A twisting or axial rotation of a portion of bowel about its mesentery. When complete it formsa closed loop obstruction ischemia Commonest spontaneous type in adult issigmoid, can be relieved by decompression per anum Surgery isrequired to prevent or relieve ischaemia Features: palpable tympanic lump (sausage shape) in the midline or left side of abdomen. Constipation, abdominal distension (early & progressive)
  • 12.
  • 13. ACUTE INTUSSUSEPTION Occurs when one portion of the gut becomes invaginated within an immediately adjacent segment. Common in 1st yearof life Common after viral illness enlargement of Peyer’s patches Ileocolic isthe commonest variety in child. Colocolic intussusception commonest in adult 🠶 An intussusception iscomposed of three parts: 🠶 the entering or innertube; 🠶 the returning or middle tube; 🠶 the sheath or outer tube (intussuscipiens).
  • 14. 🠶 Classically, a previously healthy infant presents with colicky pain and vomiting (milk then bile). 🠶 Between episodes the child initially appears well. 🠶 Later , they may passa ‘redcurrant jelly’ stool.
  • 15.
  • 16. LARGE BOWEL OBSTRUCTION 🠶 Distinguishing ileus from mechanical obstruction is challenging 🠶 Caecum is at the greatest risk of perforation 🠶 Perforation results in the release of formed feaces with heavy bacterial contamination Aetiology: 1. Carcinoma: The commonest cause, 18%of colonic ca. present with obstruction 2. Benign stricture: Due to Diverticular disease, Ischemia, Inflammatory bowel disease. 3. Volvulus: -Sigmoid Volvulus/ Caecal Volvulus 4. Hernia. 5. Congenital : HirschPrung, anal stenosis and agenesis
  • 17. CLINICAL FEATURES Large bowel obstruction distension is early and pronounced. Pain is mild and vomiting and dehydration are late. The proximal colon and caecum are distended on abdominal radiography CARDINAL FEATURES: Colicky pain Vomiting Abd distention Constipation OTHER FEATURES: Dehydration Hypokalaemia Pyrexia Abd tenderness
  • 18. PHYSICAL EXAMINATION INSPECTION Abdominal distention, scars, visible peristalsis. PALPATION Mass, tenderness, guarding PERCUSSION T ymphanic, dullness AUSCULTATION Bowel sound are high pitch and increase in frequency
  • 19. INVESTIGATIONS: 🠶 Lab: 🠶 FBC (leukocytosis, anaemia, hematocrit, platelets) 🠶 Clotting profile 🠶 Arterial blood gasses 🠶 U& Crt, Na, K, Amylase, LFTand glucose, LDH 🠶 Group and save (x-match if needed) 🠶 Optional (ESR, CRP, Hepatitis profile) 🠶 RadiOlogical: 🠶 Plain ABDOMINAL xrays 🠶 USS ( free fluid, masses, mucosal folds, pattern of paristalsis, Doppler of mesenteric vasulature, solid organs) 🠶 Other advanced studies (CT, MRI, Contrast studieS)
  • 20. Fluid levels with gas above; ‘stepladder pattern’. Ileal obstruction by adhesions; patient erect. Supine radiograph from a patient with complete small bowel obstruction shows distended small bowel loops in the central abdomen with prominent valvulae conniventes (small white arrow) Figure 3. Lateral decubitus view of the abdomen, showing air-fluid levels consistent with intestinal obstruction (arrows).
  • 21. 🠶 In small bowel 🠶 Central 3cm thick diameter 🠶 Vulvulae coniventae 🠶 Ileum may occur tubeless 🠶 In large bowel 🠶 Peripheral diameter6cm 🠶 Presence of haustration
  • 22. ROLE OF CT 🠶 🠶 Used with iv contrast, oral and rectal contrast (triple contrast). Able to demonstrate abnormality in the bowel wall, mesentery, mesenteric vessels and peritoneum. 🠶 It can define: 🠶 the level of obstruction 🠶 The degree of obstruction 🠶 The cause: volvulus, hernia, luminal and mural causes 🠶 The degree of ischaemia 🠶 Free fluid and gas 🠶 Ensure: patient vitally stable with no renal failure and no previous alergy to iodine • FIGURE: AXIAL COMPUTED TOMOGRAPHY SCAN SHOWING DILATED, CONTRAST-FILLED LOOPS OF BOWEL ON THE PATIENT’S LEFT (YELLOW ARROWS), WITH DECOMPRESSED DISTAL SMALL BOWEL ON THE PATIENT’S RIGHT (RED ARROWS). THE CAUSE OF OBSTRUCTION, AN INCARCERATED UMBILICAL HERNIA, CAN ALSO BE SEEN (GREEN ARROW), WITH PROXIMALLY DILATED BOWEL ENTERING THE HERNIA AND DECOMPRESSED BOWEL EXITING THE HERNIA.
  • 23. CONTRAST STUDIES Barium should not be used in a patient with peritonitis 🠶 As: follow through, enema 🠶 Limited use in the acute setting 🠶 Gastrografin is used in acute abdomen but is diluted 🠶 Useful in recurrent and chronic obstruction 🠶 May able to define the level and mural causes. 🠶 Can be used to distinguish adynamic and mechanical obstruction