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NUR FADZLINA ZABRI
082013100006
Definition
• Burn is a type of coagulative necrosis caused
by heat, transferred from the source to the
body
• Types :
1. Thermal : flame and scald
2. Electrical
3. Chemical
4. Radiation
• Frostbite is also a coagulative necrosis but it
is caused by extreme degrees of cold
electrical BURNS
CHEMICAL
BURNS
Injury to the
airway and
lung
Inflammation
and circulatory
changes
Other life
threatening
events
Pathophysiology of burns
Injury to the airway and lung
Physical burn injury to the
airway above larynx
– Hot gases bur the nose,
mouth, tongue, palate
– Lining of these structures
started to burn and swell
– Obstruct the airway
Metabolic poisoning
Carbon monoxide
binds to Hb
Blocks oxygen
transport
Hydrogen cyanide
Interfere mitochondrial
respiration
Metabolic acidosis
Inhalational injury
Minute particles not filtered by airways
Stick to moist lining, causing intense reaction in
alveoli
Chemical pneumonitis causing edema within
alveolar sac
Bacterial pneumonitis
• Immune system and infection
• Changes to intestine
• Danger to peripheral circulation
Inflammation and circulatory changes
Other life threatening events
Pre hospital care
– Stop burning
process
– Check other
injuries
– Cool the burn
wound
– Give oxygen
– Maintain airway
Care of burn patient
Hospital care
– Airway
maintenance
– Breathing
– Circulation
– Disability
– Exposure in a
controlled
environment
– Fluid resuscitation
Airway maintenance
Assessment of burn wound
 First degree – injury localized to the epidermis
 Second degree
 Superficial second degree – injury to the epidermis and
superficial papillary dermis
 Deep second degree – injury through the epidermis and
deep up to reticular dermis
 Third degree – full-thickness injury through the
epidermis and dermis into subcutaneous fat
 Fourth degree – injury through the skin and
subcutaneous fat into underlying muscle and
bone
Grading of burns
Heals in 3-4
days by peeling
of epidermis
Forms blister.Heals in
3-9 weeks with
scarring, sensitive on
pressure
All elements are
destroyed no
potential to heal
IV fluid with IV narcotics analgesic, antibiotics
– Children : 10% per cent
– Adult : 15% per cent
– Fluid use – ringer lactate’s/Hartmann’s solution,
fresh frozen plasma, hypertonic saline
Treatment of burn shock
• Monitoring :
– Urine output should be 0.5-1.0ml/kg body weight
– If less , infusion rate should be increased by 50 per
cent
– Signs of hypoperfusion : bolus of 10ml/kg body
weight
– Acid-base balance
– Hematocrit level
Treatment of burn wound
Escharatomy
– Incising the whole length of full thickness
– Large amount of blood may loss
Full-thickness burns and obvious deep dermal
wounds
– Dressing with nanocrystalline silver
Superficial partial thickness wounds and mixed
depth wounds
– Vaseline impregnated gauze or fenestrated
silicone sheet
– Prevent swabs adhere to the wound, reduces the
stiffness of dry eschar
– Early debridement and grafting
Additional treatment
• Analgesics
• Energy balance and nutrition
• Monitoring and control of infecrion
• Nursing care
• Physiotherapy
• Psychological
Surgery for the acute burn wound
• Any deep partial thickness and full thickness
burn requires surgery
Delayed reconstruction and scar management
– Common for large full-thickness burns
Minor burns
• Local burn wound care
• Topical agents
• Dressing wound
• Debridement or grafting if wound not heal
• Itching :antihistamines , endopeptides, aloe
vera
Non thermal burn injury
Electrical injury
• Results from source of electrical power that makes
contact with patient’s body
• Treatment : ABC,IV access, ECG, urine output
monitoring
• Intravenous fluid administration should be increased to
ensure urine output of 100ml/hr in adult
• If pigment is not clear with increased fluid, 25g mannitol
given and 12.5g mannitol should be added in
subsequent liters of fluid to maintain diuresis
Chemical injury
– Results from alkali, acid or petroleum products
– Burn is influenced by duration of contact,
concentration and amount
– Burn site should be immediately flushed with copious
amount of water for at least 20-30 minutes
– Alkali burns require longer irrigation
– If dry powder present, brush it away before irrigation
References
• Bailey and Love
• Internet

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Burn

  • 2. Definition • Burn is a type of coagulative necrosis caused by heat, transferred from the source to the body • Types : 1. Thermal : flame and scald 2. Electrical 3. Chemical 4. Radiation • Frostbite is also a coagulative necrosis but it is caused by extreme degrees of cold
  • 3.
  • 5.
  • 7. Injury to the airway and lung Inflammation and circulatory changes Other life threatening events Pathophysiology of burns
  • 8. Injury to the airway and lung Physical burn injury to the airway above larynx – Hot gases bur the nose, mouth, tongue, palate – Lining of these structures started to burn and swell – Obstruct the airway
  • 9. Metabolic poisoning Carbon monoxide binds to Hb Blocks oxygen transport Hydrogen cyanide Interfere mitochondrial respiration Metabolic acidosis
  • 10. Inhalational injury Minute particles not filtered by airways Stick to moist lining, causing intense reaction in alveoli Chemical pneumonitis causing edema within alveolar sac Bacterial pneumonitis
  • 11. • Immune system and infection • Changes to intestine • Danger to peripheral circulation Inflammation and circulatory changes Other life threatening events
  • 12. Pre hospital care – Stop burning process – Check other injuries – Cool the burn wound – Give oxygen – Maintain airway Care of burn patient Hospital care – Airway maintenance – Breathing – Circulation – Disability – Exposure in a controlled environment – Fluid resuscitation
  • 15.
  • 16.  First degree – injury localized to the epidermis  Second degree  Superficial second degree – injury to the epidermis and superficial papillary dermis  Deep second degree – injury through the epidermis and deep up to reticular dermis  Third degree – full-thickness injury through the epidermis and dermis into subcutaneous fat  Fourth degree – injury through the skin and subcutaneous fat into underlying muscle and bone Grading of burns
  • 17.
  • 18. Heals in 3-4 days by peeling of epidermis Forms blister.Heals in 3-9 weeks with scarring, sensitive on pressure All elements are destroyed no potential to heal
  • 19.
  • 20.
  • 21. IV fluid with IV narcotics analgesic, antibiotics – Children : 10% per cent – Adult : 15% per cent – Fluid use – ringer lactate’s/Hartmann’s solution, fresh frozen plasma, hypertonic saline Treatment of burn shock
  • 22. • Monitoring : – Urine output should be 0.5-1.0ml/kg body weight – If less , infusion rate should be increased by 50 per cent – Signs of hypoperfusion : bolus of 10ml/kg body weight – Acid-base balance – Hematocrit level
  • 23. Treatment of burn wound Escharatomy – Incising the whole length of full thickness – Large amount of blood may loss
  • 24. Full-thickness burns and obvious deep dermal wounds – Dressing with nanocrystalline silver
  • 25. Superficial partial thickness wounds and mixed depth wounds – Vaseline impregnated gauze or fenestrated silicone sheet – Prevent swabs adhere to the wound, reduces the stiffness of dry eschar – Early debridement and grafting
  • 26. Additional treatment • Analgesics • Energy balance and nutrition • Monitoring and control of infecrion • Nursing care • Physiotherapy • Psychological
  • 27. Surgery for the acute burn wound • Any deep partial thickness and full thickness burn requires surgery
  • 28. Delayed reconstruction and scar management – Common for large full-thickness burns
  • 29.
  • 30.
  • 31. Minor burns • Local burn wound care • Topical agents • Dressing wound • Debridement or grafting if wound not heal • Itching :antihistamines , endopeptides, aloe vera
  • 32. Non thermal burn injury Electrical injury • Results from source of electrical power that makes contact with patient’s body • Treatment : ABC,IV access, ECG, urine output monitoring • Intravenous fluid administration should be increased to ensure urine output of 100ml/hr in adult • If pigment is not clear with increased fluid, 25g mannitol given and 12.5g mannitol should be added in subsequent liters of fluid to maintain diuresis
  • 33.
  • 34. Chemical injury – Results from alkali, acid or petroleum products – Burn is influenced by duration of contact, concentration and amount – Burn site should be immediately flushed with copious amount of water for at least 20-30 minutes – Alkali burns require longer irrigation – If dry powder present, brush it away before irrigation
  • 35. References • Bailey and Love • Internet

Editor's Notes

  1. To explain RL, hypertonic saline, colloid resuscitation more in details
  2. Signs of hypoperfusion- restlessness with tachycardia, cold peripheries, high hematocrit
  3. Frequently more serious than they appear on body surface