Ride the Storm: Navigating Through Unstable Periods / Katerina Rudko (Belka G...
Current pediatrics
1. Images
Chapter 1
Figure 1.1
Figure 1.2
Figure 1.3
Figure 1.4
Figure 1.5
Figure 1.6
Figure 1.7
Chapter 7
Figure 7.1
Figure 7.2
Figure 7.3A
Figure 7.3B
Figure 7.4
Figure 7.5
Figure 7.6
Figure 7.7
Figure 7.8
Figure 7.9
Chapter 8
Figure 8.1
Figure 8.2
Figure 8.3
Figure 8.4
Figure 8.5
Chapter 15
Figure 15.1
Figure 15.2
Figure 15.3
Figure 15.4
Figure 15.5
Chapter 16
Figure 16.1
Figure 16.2
Figure 16.3
Figure 16.4
Figure 16.5
Figure 16.6
Chapter 20
Figure 20.1
Figure 20.2
Figure 20.3
Figure 20.4
Figure 20.5
Figure 20.6
Figure 20.7
Figure 20.8
Figure 20.9
Chapter 21
Figure 21.1
Fig 21.2
Fig 21.3
Fig 21.4
Figure 21.5
Figure 21.6
Figure 21.7
Chapter 24
Figure 24.1
Figure 24.2
Figure 24.3A
Figure 24.3B
Figure 24.3C
Figure 24.4
Figure 24.5A
Figure 24.5B
Figure 24.6A
Figure 24.6B
Figure 24.7
Figure 24.8
Figure 24.9
Figure 24.10A
Figure 24.10B
Figure 24.10C
Figure 24.11A
Figure 24.11B
Figure 24.11C
Figure 24.12A
Figure 24.12B
Figure 24.13
Figure 24.14
Figure 24.15A
Figure 24.15B
Figure 24.16A
Figure 24.16B
Figure 24.17A
Figure 24.17B
Figure 24.17C
Figure 24.17D
Figure 24.18
Figure 24.19A
Figure 24.19B
Figure 24.19C
Chapter 28
Figure 28.1
Figure 28.2
The twentieth edition of Current Diagnosis & Treatment Pediatrics by
William W. Hay, Jr., Myron J. Levin, Judith M. Sondheimer, and Robin R.
Deterding provides practical, up-to-date, well-referenced coverage of the
care of children from birth through adolescence. The additional full color
images on this CD complement chapters on ophthalmology, child abuse
and neglect, oral medicine and dentistry, orthopedics, and endocrinology.
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10. Chapter 7
Figure 7.1
Head CT of infant with recent abusive head injury: acute subdural hemorrhage (SDH) and poor
grey-white matter differentiation indicative of global parenchymal injury.
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11. Chapter 7
Figure 7.2
Six month follow up head CT of infant with severe abusive head injury: chronic subdural
effusions and hydrocephalus ex vacuo from cerebral atrophy
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36. Chapter 20
Figure 20.1
Peptic stricture of the esophagus: Narrowing of the distal esophagus seen on endoscopic
evaluation, with associated friability and irregular contour of the esophageal mucosa.
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37. Chapter 20
Figure 20.2
Eosinophilic Esophagitis: Whitish exudates coat the esophageal mucosa. Esophageal edema is demonstrated by
the linear furrows created by the thickened adjacent mucosa
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38. Chapter 20
Figure 20.3
Esophageal foreign body: Endoscopic view of a penny lodged in the proximal esophagus in a
toddler with acute onset of drooling and dysphagia.
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39. Chapter 20
Figure 20.4
Gastric ulcer: Endoscopic view of a superficial gastric ulcer in the antrum, with a white
base and surrounding erythema. The patient presented with chronic epigastric pain,
melena and anemia.
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40. Chapter 20
Figure 20.5
Clostridium difficile: Characteristic white plaques in the sigmoid colon with relatively normal
intervening mucosa, indicative of pseudomembranous colitis with Clostridium difficile infection.
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41. Chapter 20
Figure 20.6
Juvenile polyp: A single, pedunculated juvenile polyp in the sigmoid colon, with hyperemia but no
active bleeding.
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42. Chapter 20
Figure 20.7
Esophageal Varices: Endoscopic view of a grade 3 esophageal varix, demonstrating
engorgement and tortuosity, without any cherry-red spots or evidence of active bleeding.
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43. Chapter 20
Figure 20.8
Celiac Disease: White, patchy exudates and blunted villous architecture in the duodenum of a
child with untreated celiac disease.
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44. Chapter 20
Figure 20.9
Crohn’s disease: Deep, discreet ulcers in the transverse colon in an adolescent with Crohn’s disease.
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45. Chapter 21
Figure 21.1
Liver biopsy from a patient with biliary atresia demonstrating Bile duct proliferation (black arrows), bile duct plugs
(yellow arrow) and expansion of the portal region.
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46. Chapter 21
Fig 21.2
Liver Biopsy from a patient with type 1 autoimmune hepatitis that demonstrates intense inflammation (black
circle), piecemeal necrosis (yellow arrow) and increased fibrosis (black arrow).
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47. Chapter 21
Fig 21.3
Liver Pancreas. ERCP demonstrating a massively dilated pancreatic duct (solid arrow) and multiple pancreatic
duct stones (dashed arrow) from hereditary pancreatitis
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48. Chapter 21
Fig 21.4
Liver Pancreas Abdominal US from a patient with a type 1 choledochal cyst demonstrating a large cyst (arrow)
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49. Chapter 21
Figure 21.5
Liver Pancreas. Abdominal MRI demonstrating a large type 1 choledochal cyst (arrow)
from the same patient shown in figure 4.
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50. Chapter 21
Figure 21.6
Liver Pancreas. Abdominal US demonstrating 2 stones in the gallbladder (arrows).
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51. Chapter 21
Figure 21.7
Liver Pancreas. Abdominal US demonstrating a stone in a dilated common bile duct in a patient who presented
with pancreatitis.
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52. Chapter 24
Figure 24.1
Unicameral Bone Cyst- the proximal femoral lesion is well circumscribed and lytic, both benign features. However, large
lesions in weightbearing bones are at risk for pathologic fracture.
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54. Chapter 24
Figure 24.3A
Aneurysmal Bone Cyst- similar looking to unicameral bone cyst, but more expansile. Fluid-
fluid levels on MRI are characteristic [arrows]
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55. Chapter 24
Figure 24.3B
Aneurysmal Bone Cyst- similar looking to unicameral bone cyst, but more expansile. Fluid-fluid levels
on MRI are characteristic [arrows]
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56. Chapter 24
Figure 24.3C
Aneurysmal Bone Cyst- similar looking to unicameral bone cyst, but more expansile. Fluid-fluid levels
on MRI are characteristic [arrows]
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57. Chapter 24
Figure 24.4
Non-ossifying fibroma/Fibrous cortical defect-benign appearing,
well circumscribed. Often incidental findings on radiographs.
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58. Chapter 24
Figure 24.5A
Osteochondroma- Common sites: proximal tibia, distal femur. May be painful due to soft-tissue irritation.
Benign. Surgical excision is indicated for asymptomatic lesions.
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59. Chapter 24
Figure 24.5B
Osteochondroma- Common sites: proximal tibia, distal femur. May be painful due to soft-tissue irritation.
Benign. Surgical excision is indicated for asymptomatic lesions.
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60. Chapter 24
Figure 24.6A
Osteosarcoma-note the permeative, destructive pattern. The lesion is not well
circumscribed and forming pathologic bone.
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61. Chapter 24
Figure 24.6B
Osteosarcoma-note the permeative, destructive pattern. The lesion is not well
circumscribed and forming pathologic bone.
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71. Chapter 24
Figure 24.12A
Slipped Capital Femoral Epiphysis-note the widening of the physis. The displacement is best seen on the lateral
x-ray of the right hip.
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72. Chapter 24
Figure 24.12B
Slipped Capital Femoral Epiphysis-note the widening of the physis. The displacement is best seen on the lateral
x-ray of the right hip.
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73. Chapter 24
Figure 24.13
Developmental dysplasia of the hip- the femoral head [red arrow] is dislocated proximally from the acetabulum
[yellow arrow].
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87. Chapter 28
Figure 28.1
Compound heterozygous protein C deficiency with undetectable protein C activity. Purpura fulminans in severe protein C
deficiency often presents within hours of birth at points of minimal pressure.
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88. Chapter 28
Figure 28.2
Symptomatic heterozygous protein C deficiency. Individuals with recurrent venous thromboembolism secondary to
heterozygous protein C deficiency are at risk for post-thrombotic syndrome. This patient manifests venous stasis
ulcers.
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