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CARDIO PULMONARY
NAME: NEELAM SHARMA
YEAR BPT: 4TH
SUBMITTED TO: Dr. Jamal Moiz1
HEART FAILURE
2
INTRODUCTION
• Heart failure (HF) is often referred as congestive
heart failure (CHF)
• Occurs when heart is unable to pump sufficiently to
maintained blood flow to meets the body needs
This condition results from -
• SYSTOLIC DYSFUNCTION
• DIASTOLIC DYSFUNCTION
3
Heart Failure: Key Concepts
•
Cardiac output (CO) = Stroke Volume (SV) x Heart Rate
(HR)
– Becomes insufficient to meet metabolic needs of body
•
•
Ejection Fraction (EF) (proportion of blood pumped out)
Normal EF = 55 to 75 percent
•
Classifications HF–
–
 Systolic failure – decrease contractility
 Diastolic failure – decrease filling
 Mixed
HeartFailure
6
CLASSIFICATION
• Left sided v/s right sided heart failure
• Acute v/s chronic failure
7
LHF
• left ventricle cardiac output is decreased.
• blood accumulates in the left ventricle, left atrium.
• Result is pulmonary congestion
• pulmonary interstitial edema and impaired gas
exchange.
8
RHF
• Right ventricle cardiac output is less then volume
received from the peripheral venous circulation
• blood accumulates in RA, RV and peripheral venous
system
• Increased venous pressure lead increased
capillary hydrostatic and peripheral Edima.
9
CLINICAL PICTURE:
LVF
10
RVF
11
ACUTE V/S CHRONIC
• Acute failure occurs in response to a sudden
decrease in cardiac output which results in rapid
decrease in tissue perfusion. Ex: drug toxicity
• Chronic failure occurs slowly, giving time to the
body to adjust to decrease in cardiac output
through compensatory mechanisms which
results in systemic congestion.
12
INCIDENCE
• More than 20 million people have heart failure
worldwide
• Prevalence of heart failure in India due to
• Coronary heart disease
• Hypertension
• Obesity
• Diabetes
• Rheumatic heart disease
• Ranges from 1.3 to 4.6 million, with an annual
incidence of 491 600-1.8 million.
• Heart failure is the leading cause of
hospitalization > 65 years age 13
ETIOLOGY
The incidence of heart failure increases with
advancing age and coronary artery disease
• Diabetes
• Cigarette Smoking
• Obesity
• Elevated Total Cholesterol
• Abnormally High or Low Hematocrit Level
• Chronic Kidney Disease and Proteinuria
 Common precipitating causes of Heart Failure are as
follows
• Anaemia
• Infection
14
DIAGNOSTIC ASESSMENT
• History
• Physical examination
• ABG analysis
• Chest X ray
• 12 leads ECG
15
PHARMACOLOGICAL MANAGMENT
• DIGOXIN
• BETA BLOCKERS
• INOTROPES
• ANGIOTENSIN RECEPTOR
BLOCKERS (ARBs)
• ANGIOTENSIN CONCERTING
ENZYME (ACE) INHIBITORS
• DIURETICS
16
SURGICAL MANAGEMENT
HEART TRANSPLANTATION:
 When the heart is irreversibly managed and no
longer functions adequately and when the client is
at risk of dying
 cardiac transplantation and use of an artificial
heart to assist or replace the failing heart are
soluitions
 A heart transplant, or a cardiac transplant, is a
surgical transplant procedure performed on
patients with end-stage heart failure or severe
coronary artery disease when other medical or
surgical treatments have failed as a last resort.
17
CARDIOMYOPLASTY
• Cardiomyoplasty is a surgical procedure in which
healthy muscle from another part of the body is
wrapped around the heart to provide support for
the failing heart.
• VENTRICULAR ASSIST DEVICE(VAD):
• It is a Electromechanical device for assisting
cardiac circulation, which is used either to
partially or to completely replace the function of
a failing heart.
18
MANAGEMENT
• Cardiac rehabilitation
• should be offered to all cardiac patients who would
benefit:
• Recent myocardial infarction
• Acute coronary artery syndrome
• Chronic stable angina
• Congestive heart failure
• After coronary artery bypass surgery
• After a percutaneous coronary intervention
• Valvular surgery
• Cardiac transplantation
• CR begins as soon as possible in ICU (only if the patient
is in stablemedical condition).
19
Goals of Cardiac Rehabilitation
• Comprehensive cardiac rehabilitation program should contain
specific corecomponents.
• These components should optimize cardiovascular risk
reduction, reduce disability, encourage active and healthy lifestyle
changes, and help maintain those healthy habits after rehabilitation is
complete..
• Reduce the risk of sudden death or reinfarction,
• Control cardiac symptom,
• Stabilize and reverse the atherosclerotic process and
• Enhance the psychological and vocational status of selective patients
20
21
Phase I Cardiac Rehabilitation
• Involves immediate inpatient exercise rehabilitation that emphasizes:
a) Patient education (informal discussions with nurses and physicians) and
b) Counselling.
• Exercise therapy-
a) Musculoskeletal ROM activities.
b) ADLs (sitting, standing, and walking).
• Purpose:
a) Counter the deconditioning effects of prolonged bed rest,
b) Prepare patient for a return to normal daily activities.
Phase II: Outpatient cardiac rehab
• Once a patient is stable and cleared by cardiology, outpatient cardiac
rehabilitation may begin.
• Phase II typically lasts three to six weeks though some may last up to
up to twelve weeks. Initially, patients have an assessment with a focus
on identifying limitations in physical function, restrictions of
participation secondary to comorbidities, and limitations toactivities.
• A more rigorous patient-centered therapy plan is designed, comprising
three modalities: information/advice, tailored training program, and a
relaxation program. The treatment phase intends to promote
independence and lifestyle changes to prepare patients to return to
their lives at home.
Phase III: Post-cardiac rehab. Maintenance
• This phase involves more independence and self-monitoring. Phase III
centers on increasing flexibility, strengthening, and aerobic
conditioning.
• Goal: facilitate long term maintenance of lifestyle changes,
monitoring risk factor changes and secondaryprevention.
• FIIT PRINCIPLE:
Frequency
3-5 times/week
Intensity
60-80 % HRmax
Duration
30-60 min
Activity
ROM, biking, jogging, swimming,
Patient shouldn’t exercise if they are generally unwell,
symptomatic or clinically unstable on arrival;
• Fever/acute systemic illness
• Unresolved/unstable angina
• Resting BP systolic >200mmHg and diastolic >110mmHg
• Significant drop in BP
• Symptomatic hypotension
• Resting/uncontrolled tachycardia (>100bpm)
• Uncontrolled atrial or ventricular arrhythmias
• New/recurrent symptoms of breathlessness, lethargy, palpitations,
dizziness
• Unstable heart failure
• Unstable/uncontrolled diabetes

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heart failure bpt

  • 1. CARDIO PULMONARY NAME: NEELAM SHARMA YEAR BPT: 4TH SUBMITTED TO: Dr. Jamal Moiz1
  • 3. INTRODUCTION • Heart failure (HF) is often referred as congestive heart failure (CHF) • Occurs when heart is unable to pump sufficiently to maintained blood flow to meets the body needs This condition results from - • SYSTOLIC DYSFUNCTION • DIASTOLIC DYSFUNCTION 3
  • 4. Heart Failure: Key Concepts • Cardiac output (CO) = Stroke Volume (SV) x Heart Rate (HR) – Becomes insufficient to meet metabolic needs of body • • Ejection Fraction (EF) (proportion of blood pumped out) Normal EF = 55 to 75 percent • Classifications HF– –  Systolic failure – decrease contractility  Diastolic failure – decrease filling  Mixed
  • 6. 6
  • 7. CLASSIFICATION • Left sided v/s right sided heart failure • Acute v/s chronic failure 7
  • 8. LHF • left ventricle cardiac output is decreased. • blood accumulates in the left ventricle, left atrium. • Result is pulmonary congestion • pulmonary interstitial edema and impaired gas exchange. 8
  • 9. RHF • Right ventricle cardiac output is less then volume received from the peripheral venous circulation • blood accumulates in RA, RV and peripheral venous system • Increased venous pressure lead increased capillary hydrostatic and peripheral Edima. 9
  • 12. ACUTE V/S CHRONIC • Acute failure occurs in response to a sudden decrease in cardiac output which results in rapid decrease in tissue perfusion. Ex: drug toxicity • Chronic failure occurs slowly, giving time to the body to adjust to decrease in cardiac output through compensatory mechanisms which results in systemic congestion. 12
  • 13. INCIDENCE • More than 20 million people have heart failure worldwide • Prevalence of heart failure in India due to • Coronary heart disease • Hypertension • Obesity • Diabetes • Rheumatic heart disease • Ranges from 1.3 to 4.6 million, with an annual incidence of 491 600-1.8 million. • Heart failure is the leading cause of hospitalization > 65 years age 13
  • 14. ETIOLOGY The incidence of heart failure increases with advancing age and coronary artery disease • Diabetes • Cigarette Smoking • Obesity • Elevated Total Cholesterol • Abnormally High or Low Hematocrit Level • Chronic Kidney Disease and Proteinuria  Common precipitating causes of Heart Failure are as follows • Anaemia • Infection 14
  • 15. DIAGNOSTIC ASESSMENT • History • Physical examination • ABG analysis • Chest X ray • 12 leads ECG 15
  • 16. PHARMACOLOGICAL MANAGMENT • DIGOXIN • BETA BLOCKERS • INOTROPES • ANGIOTENSIN RECEPTOR BLOCKERS (ARBs) • ANGIOTENSIN CONCERTING ENZYME (ACE) INHIBITORS • DIURETICS 16
  • 17. SURGICAL MANAGEMENT HEART TRANSPLANTATION:  When the heart is irreversibly managed and no longer functions adequately and when the client is at risk of dying  cardiac transplantation and use of an artificial heart to assist or replace the failing heart are soluitions  A heart transplant, or a cardiac transplant, is a surgical transplant procedure performed on patients with end-stage heart failure or severe coronary artery disease when other medical or surgical treatments have failed as a last resort. 17
  • 18. CARDIOMYOPLASTY • Cardiomyoplasty is a surgical procedure in which healthy muscle from another part of the body is wrapped around the heart to provide support for the failing heart. • VENTRICULAR ASSIST DEVICE(VAD): • It is a Electromechanical device for assisting cardiac circulation, which is used either to partially or to completely replace the function of a failing heart. 18
  • 19. MANAGEMENT • Cardiac rehabilitation • should be offered to all cardiac patients who would benefit: • Recent myocardial infarction • Acute coronary artery syndrome • Chronic stable angina • Congestive heart failure • After coronary artery bypass surgery • After a percutaneous coronary intervention • Valvular surgery • Cardiac transplantation • CR begins as soon as possible in ICU (only if the patient is in stablemedical condition). 19
  • 20. Goals of Cardiac Rehabilitation • Comprehensive cardiac rehabilitation program should contain specific corecomponents. • These components should optimize cardiovascular risk reduction, reduce disability, encourage active and healthy lifestyle changes, and help maintain those healthy habits after rehabilitation is complete.. • Reduce the risk of sudden death or reinfarction, • Control cardiac symptom, • Stabilize and reverse the atherosclerotic process and • Enhance the psychological and vocational status of selective patients 20
  • 21. 21
  • 22. Phase I Cardiac Rehabilitation • Involves immediate inpatient exercise rehabilitation that emphasizes: a) Patient education (informal discussions with nurses and physicians) and b) Counselling. • Exercise therapy- a) Musculoskeletal ROM activities. b) ADLs (sitting, standing, and walking). • Purpose: a) Counter the deconditioning effects of prolonged bed rest, b) Prepare patient for a return to normal daily activities.
  • 23. Phase II: Outpatient cardiac rehab • Once a patient is stable and cleared by cardiology, outpatient cardiac rehabilitation may begin. • Phase II typically lasts three to six weeks though some may last up to up to twelve weeks. Initially, patients have an assessment with a focus on identifying limitations in physical function, restrictions of participation secondary to comorbidities, and limitations toactivities. • A more rigorous patient-centered therapy plan is designed, comprising three modalities: information/advice, tailored training program, and a relaxation program. The treatment phase intends to promote independence and lifestyle changes to prepare patients to return to their lives at home.
  • 24. Phase III: Post-cardiac rehab. Maintenance • This phase involves more independence and self-monitoring. Phase III centers on increasing flexibility, strengthening, and aerobic conditioning. • Goal: facilitate long term maintenance of lifestyle changes, monitoring risk factor changes and secondaryprevention. • FIIT PRINCIPLE: Frequency 3-5 times/week Intensity 60-80 % HRmax Duration 30-60 min Activity ROM, biking, jogging, swimming,
  • 25. Patient shouldn’t exercise if they are generally unwell, symptomatic or clinically unstable on arrival; • Fever/acute systemic illness • Unresolved/unstable angina • Resting BP systolic >200mmHg and diastolic >110mmHg • Significant drop in BP • Symptomatic hypotension • Resting/uncontrolled tachycardia (>100bpm) • Uncontrolled atrial or ventricular arrhythmias • New/recurrent symptoms of breathlessness, lethargy, palpitations, dizziness • Unstable heart failure • Unstable/uncontrolled diabetes