5. • Definition
Pyloric stenosis is is hypertrophy of
musculature of pyloric Antrim,especially the
circular muscle fibers causing primary failure
of pylorus to relax
6. Epidermiology
1. 2-3.5 live birth
2 .Male:Female 4:1
3 . 3-5 weeks of life
4. Preterm baby
5. First born child for 30%
7. Risk factor and etiology
Risk factor /etiology
1.Maternal smoking or alcholism
2.Bottle feeding
3.Genetical history/family history of IHPS
4.Microide like erythromycin/azithromycin
13. inverstigation
1 .Abdominal x rays; dilated stomach
2. Barium meal series ;string sign and rail roads
sign
3 USG show dilated and elongated pyloric
stenosis I.E doughnut appearance
4; serum electrolyte ( hypo Na,K,cl)
5 RFT
14.
15.
16. Diagnostic criteria of IHPS
Rohrscchneider
1 .Pyloric channel length; 1.5 to 2cm
(normal 1cm)
2. pyloric channel diameter ;1.3 to 1.5cm
(normal 1 cm)
3. Circular muscle thickness ;4 to 5 mms
(normal < 2mms
17. Diffential diagnosis
1 .duodenal atresia( bilious vomiting present
too)
2 High intesine obstruction( volvulus
neonstorum)
3 Intercranial haemorrhage
4 GERD
5 Congenital like pyloris atresia or membrane
and antral atresia or membrane
18. Complications of pyloric stenosis
1. Hypovolemic shock due to severe vomiting
that lead severe dehydration
2. Acute kidney injury due to hypovolemic
shock
3. Electrolyte imbalance
4. failure to thrieve
5. metabolic disturbance due to metabolic
alkalosis
21. complication
Occasional complications are — (i)
postoperative vomiting — this may be
treated by repeated
aspiration and intravenous feeding, (ii)
postoperative pyrexia, (iii) gastroenteritis —
all these can be
treated by antibiotic and tepid sponging with
repeated aspirations, and (iv) disruption of
wound
NB the patient is not textbook can happen by any unkown reason
3.Condition due to difficiency of (NO2 SI) which is most common muscle relaxant. 4 oral erythromycin due to diffent condition increase the folds of IHSP so it must prescibe only for child below 2-3 weeks when there is no other alternative
From the diagram above there is hypertrophoid of circulsr muscle the lead G.O.O ,so whenthat happen cause postprandial projective non billious ,so wheen that happen result loose of gastric fluid and this condition id malor cause of hypochloric hypokalemic metabolic alkalosis
VGP visible gastric peristalis .occasional jaundice due bilirubin levels may increase and because of fasting or too little food intake.olive mass below the liver ( good after or during vommitin or at the ends of feedin)
For preterm baby pyloric muscle wall thicknees/pyloric diameter>0.27 ids diagnostic
We correct alkalosis because will post operative apnea that will cause of postoperative death due to postoperative respiratory complicattion due to vomiting resalt apnea