Svalbard
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Svalbard is a group of islands located between the Arctic Ocean and northern seas of Europe. The climate is extremely cold, with temperatures ranging from -46 to 21 degrees Celsius. Proper equipment like warm clothing, sleeping bags, food, and first aid is essential for withstanding the harsh conditions. The integumentary, circulatory, and respiratory systems are most affected by the frigid temperatures and wind. Hypothermia is a major health risk due to the cold, wet, and windy environment.
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![[object Object],[object Object],A group of islands located between the Arctic Ocean, Barents Sea, Greenland Sea, and the Norwegian Sea. The islands are directly North of (and part of) Norway.](https://image.slidesharecdn.com/svalbard-1229059862330375-1/85/Svalbard-1-320.jpg)
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- As air masses move towards low pressure regions, they are modified by changes in surface properties like heating/cooling and by adding or removing moisture through processes like evaporation and precipitation.
- Air masses are classified and named based on their region of origin and associated surface type, and bring general weather conditions to areas over which they pass.
Biome Tundra
Tundra is the coldest of all the biomes. Tundra comes from the Finnish word tunturia, meaning treeless plain. It is noted for its frost-molded landscapes, extremely low temperatures, little precipitation, poor nutrients, and short growing seasons. Dead organic material functions as a nutrient pool. The two major nutrients are nitrogen and phosphorus. Nitrogen is created by biological fixation, and phosphorus is created by precipitation. Tundra is separated into two types: arctic tundra and alpine tundra.
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Simple vegetation structure
Limitation of drainage
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Cell Therapy Expansion and Challenges in Autoimmune Disease
There is increasing confidence that cell therapies will soon play a role in the treatment of autoimmune disorders, but the extent of this impact remains to be seen. Early readouts on autologous CAR-Ts in lupus are encouraging, but manufacturing and cost limitations are likely to restrict access to highly refractory patients. Allogeneic CAR-Ts have the potential to broaden access to earlier lines of treatment due to their inherent cost benefits, however they will need to demonstrate comparable or improved efficacy to established modalities.
In addition to infrastructure and capacity constraints, CAR-Ts face a very different risk-benefit dynamic in autoimmune compared to oncology, highlighting the need for tolerable therapies with low adverse event risk. CAR-NK and Treg-based therapies are also being developed in certain autoimmune disorders and may demonstrate favorable safety profiles. Several novel non-cell therapies such as bispecific antibodies, nanobodies, and RNAi drugs, may also offer future alternative competitive solutions with variable value propositions.
Widespread adoption of cell therapies will not only require strong efficacy and safety data, but also adapted pricing and access strategies. At oncology-based price points, CAR-Ts are unlikely to achieve broad market access in autoimmune disorders, with eligible patient populations that are potentially orders of magnitude greater than the number of currently addressable cancer patients. Developers have made strides towards reducing cell therapy COGS while improving manufacturing efficiency, but payors will inevitably restrict access until more sustainable pricing is achieved.
Despite these headwinds, industry leaders and investors remain confident that cell therapies are poised to address significant unmet need in patients suffering from autoimmune disorders. However, the extent of this impact on the treatment landscape remains to be seen, as the industry rapidly approaches an inflection point.
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Overall life span (LS) was 1671.7±1721.6 days and cumulative 5YS reached 62.4%, 10 years – 50.4%, 20 years – 44.6%. 94 LCP lived more than 5 years without cancer (LS=2958.6±1723.6 days), 22 – more than 10 years (LS=5571±1841.8 days). 67 LCP died because of LC (LS=471.9±344 days). AT significantly improved 5YS (68% vs. 53.7%) (P=0.028 by log-rank test). Cox modeling displayed that 5YS of LCP significantly depended on: N0-N12, T3-4, blood cell circuit, cell ratio factors (ratio between cancer cells-CC and blood cells subpopulations), LC cell dynamics, recalcification time, heparin tolerance, prothrombin index, protein, AT, procedure type (P=0.000-0.031). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and N0-12 (rank=1), thrombocytes/CC (rank=2), segmented neutrophils/CC (3), eosinophils/CC (4), erythrocytes/CC (5), healthy cells/CC (6), lymphocytes/CC (7), stick neutrophils/CC (8), leucocytes/CC (9), monocytes/CC (10). Correct prediction of 5YS was 100% by neural networks computing (error=0.000; area under ROC curve=1.0).
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