SlideShare a Scribd company logo
1 of 64
1
Fundamental of Nursing
Vital Signs
Respiratory rate & Blood Pressure
Dr. mosa alfageh
After completing this lecture, each student will be able to:
 Define respiration
 Describe the mechanics of breathing and the mechanisms that control respirations
 List the factors that influence the body respiration
 List the characteristics that should be included when assessing respiration
 Define blood pressure
 Understand the alteration in blood pressure
 List the factors that influence the blood pressure
 Identify the sites used to assess the blood pressure
2
3
 Respiration is the act of breathing. (is the process by which the lungs bring oxygen
into the body and remove carbon dioxide)
Inhalation or inspiration refers to the intake of air into the lungs.
Exhalation or expiration refers to breathing out or the movement of gases from the
lungs to the atmosphere.
Ventilation is also used to refer to the movement of air in and out of the lungs.
4
There are basically two types of breathing:
 Costal (thoracic)breathing Costal breathing involves the external intercostal muscles
and other accessory muscles, such as the sternocleidomastoid muscles. It can be
observed by the movement of the chest upward and outward.
 Diaphragmatic (abdominal) breathing Diaphragmatic breathing involves the
contraction and relaxation of the diaphragm, and it is observed by the movement of
the abdomen, which occurs as a result of the diaphragm’s contraction and downward
movement.
Mechanics and regulation of
breathing
During inhalation, the
following processes normally
occur :
The diaphragm contracts
(flattens), the ribs move
upward and outward, and the
sternum moves outward, thus
enlarging the thorax and
permitting the lungs to
expand.
5
During exhalation: the
diaphragm relaxes, the ribs
move downward and inward,
and the sternum moves inward,
thus decreasing the size of the
thorax as the lungs are
compressed.
6
Mechanics and regulation of
breathing
Normal breath inspiration animation, awake
Diaghram contracts
Chest volume
Pleural pressure
Air moves down
pressure gradient
to fill lungs
-2cm H20
-7cm H20
Alveolar
pressure falls
Normal breath
Normal breath expiration animation, awake
Diaghram relaxes
Pleural /
Chest volume 
Pleural pressure
rises
Normal breath
Alveolar
pressure rises
Air moves down
pressure gradient
out of lungs
Time
volume
0
-1
-2
-5
Pressure
Expiration
Inspiration
+3
+2
+1
Normal breath
 Normal breathing is automatic and effortless. A normal adult inspiration lasts 1 to
1.5 seconds, and an expiration lasts 2 to 3 seconds.
 Respiration is controlled by Respiratory centers in the medulla oblongata and the
pons of the brain
 Chemoreceptors located centrally in the medulla and peripherally in the carotid and
aortic bodies. These centers and receptors respond to changes in the concentrations
of oxygen (O2), carbon dioxide (CO2), and hydrogen (H+) in the arterial blood
11
Mechanics and regulation of breathing
Assessing respirations
 Resting respirations should be assessed when the client is relaxed because exercise
affects respirations, increasing their rate and depth.
 Anxiety is likely to affect respiratory rate and depth as well.
12
Before assessing a client’s respirations, a nurse should be aware of the following :
• The client’s normal breathing pattern
• The influence of the client’s health problems on respirations
• Any medications or therapies that might affect respirations
• The relationship of the client’s respirations to cardiovascular function.
Assessing respirations
 The rate, depth, rhythm, quality, and effectiveness of respirations should be
assessed.
 The respiratory rate is normally described in breaths per minute.
 Breathing that is normal in rate and depth is called eupnea.
 Abnormally slow respirations are referred to as bradypnea
 Abnormally fast respirations are called tachypnea
 Apnea is the absence of breathing.
13
The depth
 The depth of a person’s respirations can be established by watching the movement
of the chest.
 Respiratory depth is generally described as normal, deep, or shallow.
Deep respirations are those in which a large volume of air is inhaled and exhaled,
inflating most of the lungs.
Shallow respirations involve the exchange of a small volume of air and often the
minimal use of lung tissue.
 During a normal inspiration and expiration, an adult takes in about 500 mL of air.
This volume is called the tidal volume.
 Hyperventilation refers to very deep, rapid respirations.
 Hypoventilation refers to very shallow respirations.
Assessing respirations
Respiratory rhythm
Respiratory rhythm refers to the regularity of the expirations and the inspirations.
Normally, respirations are evenly spaced.
 Respiratory rhythm can be described as regular or irregular.
 An infant’s respiratory rhythm may be less regular than an adult’s
14
Respiratory quality or character
refers to those aspects of breathing that are different from normal, effortless breathing.
 Two of these aspects are The amount of effort a client must exert to breathe
 The sound of breathing.
 Usually, breathing does not require noticeable effort.
 Sometimes, however, clients can breathe only with substantial effort—this is
referred to as labored breathing.
 The sound of breathing is also significant. Normal breathing is silent, but a number
of abnormal sounds such as a wheeze are obvious to the nurse’s ear.
 Many sounds occur as a result of the presence of fluid in the lungs and are most
clearly heard with a stethoscope.
Definitions
 Eupnea ; breathing normal in rate and depth (12-20 breath /min)
 Bradypnea ; rate of breathing is regular but abnormally slow (less than
10 breath/min)
 Apnea respirations cease for several seconds . Persistent cessation
results in cardiac arrest
 Kussmaul’s respirations ;respirations that are regular but abnormally
deep and increase in rate . It is associated with DKA(DIABETIC
KETOACIDOSIS)
 Dyspnea; the term for difficult or painful breathing
 Orthopnea ; when the difficulty is so marked that the client can
breathe only when in an upright position
 Cyanosis; (bluish tinge); especially in the lips (circumoral cyanosis)
and mucus membranes of the mouth . In severe conditions , cyanosis
spreads to the nails and extremities . An excess of carbon dioxide
causes the bluish tinge
15
16
17
Factors affecting respirations
 Exercise (increases metabolism),
Exercise increases rate and depth to meet the body's need for additional oxygen and to
rid the co2
 Acute pain
Pain alters rate and rhythm of respirations ; breathing becomes shallow
Patient inhibits or splints chest wall movement when pain is in area of chest or
abdomen
 Anxiety
Anxiety increases respiration rate and depth
18
 Smoking
Chronic smoking changes pulmonary airways, resulting in increased rate of respirations
at rest when not smoking
Body position
 A straight , erect posture promotes full chest expansion
 Lying flat prevents full chest expansion
Factors affecting respirations
19
 Medications
For example, narcotics such as morphine and large doses of barbiturates such as
pentobarbital depress the respiratory centers in the brain, thereby depressing the
respiratory rate and depth.
 Neurological injury
Injury to brainstem impairs respiratory center and inhibits respiratory rate and rhythm
 Hemoglobin functions
Decrease hemoglobin levels (anemia) reduce oxygen carrying capacity of the blood ,
which increase respiratory rate
Abnormal blood cell function (e.g sickle cell anemia) reduce ability of hemoglobin to
carry oxygen , which increase respiratory rate and depth
20
Measurement of oxygen saturation (pulse oximetry)
 Assessment of Diffusion and Perfusion
 Evaluate the respiratory processes of diffusion and perfusion by measuring the
oxygen saturation of the blood.
 The percent of hemoglobin that is bound with oxygen in the arteries is the percent of
saturation of hemoglobin (or SaO2). It is usually between 95% and 100%.
21
 The saturation of venous blood (SVO2) is lower because the tissues have removed
some of the oxygen from the hemoglobin molecules. The usual value for Sv02,
which is 70%.
 Measurement of Arterial Oxygen Saturation.
 A pulse oximeter is a noninvasive device that estimates a client's arterial blood
oxygen saturation (SaO2) by means of a sensor attached to the client's finger, toe,
nose, earlobe, or forehead (or around the hand or foot of a neonate).
 The pulse oximeter can detect hypoxemia (low oxygen saturation) before clinical
signs and symptoms, such as a dusky color to skin and nail beds develop.
 Normal oxygen saturation is 95% to 100%, and below 70% is life threatening.
22
(pulse oximetry)
(pulse oximetry)
23
Blood pressure
Arterial blood pressure is a measure of the pressure exerted by the blood as it flows
through the arteries.
Because the blood moves in waves, there are two blood pressure measurements.
The systolic pressure is the pressure of the blood as a result of contraction of the
ventricles, that is, the pressure of the height of the blood wave.
The diastolic pressure is the pressure when the ventricles are at rest.
Diastolic pressure ;is the lower pressure, present at all times within the arteries.
 The difference between the diastolic and the systolic pressures is called the pulse
pressure.
 A normal pulse pressure is about 40 mmHg but can be as high as 100 mmHg
during exercise
24
 Blood pressure is measured in millimeters of mercury (mmHg) and recorded as a
fraction: systolic pressure over the diastolic pressure.
 A typical blood pressure for a healthy adult is 120/80 mmHg (pulse pressure of 40).
 Sometimes, it is useful to determine the mean arterial pressure (MAP) because this
represents the pressure actually delivered to the body’s organs.
 A normal MAP is 70 to 110 mmHg.
Determinants of blood pressure
Arterial blood pressure is the result of several factors:
1. The pumping action of the heart
2. The peripheral vascular resistance (the resistance supplied by the blood vessels through which the
blood flows)
3. The blood volume and viscosity.
PUMPING ACTION OF THE HEART
 When the pumping action of the heart is weak, less blood is pumped into arteries (lower cardiac
output), and the blood pressure decreases.
 When the heart’s pumping action is strong and the volume of blood pumped into the circulation
increases (higher cardiac output), the blood pressure increases.
25
PERIPHERAL VASCULAR RESISTANCE
 Peripheral resistance can increase blood pressure. The diastolic pressure especially is affected.
 Some factors that create resistance in the arterial system are the capacity of the arterioles and
capillaries, the compliance of the arteries, and the viscosity of the blood.
 The internal diameter or capacity of the arterioles and the capillaries determines in great part the
peripheral resistance to the blood in the body.
The smaller the space within a vessel, the greater the resistance.
Normally, the arterioles are in a state of partial constriction.
Increased vasoconstriction, such as occurs with smoking, raises the blood pressure, whereas
decreased vasoconstriction lowers the blood pressure.
Determinants of blood pressure
BLOOD VOLUME
When the blood volume decreases (for example, as a result of a hemorrhage or
dehydration), the blood pressure decreases because of decreased fluid in the arteries.
 Conversely, when the volume increases (for example, as a result of a rapid
intravenous infusion), the blood pressure increases because of the greater fluid
volume within the circulatory system.
BLOOD VISCOSITY
 Blood pressure is higher when the blood is highly viscous (thick), that is, when the
proportion of red blood cells to the blood plasma is high.
 This proportion is referred to as the hematocrit. The viscosity increases markedly
when the hematocrit is more than 60% to 65%.
26
Factors affecting blood pressure
1. Age
2. Exercise
3. Stress
4. Race
5. Gender
6. Medications
7. Obesity
8. Diurnal variations
9. Medical conditions
10. Temperature
27
1.Age
Newborns have a systolic pressure of about 75 mmHg. The pressure rises with age,
2.Exercise.
Physical activity increases the cardiac output and hence the blood pressure. For reliable assessment of
resting blood pressure, wait 20 to 30 minutes following exercise.
3.Stress.
Stimulation of the sympathetic nervous system increases cardiac output and vasoconstriction of the
arterioles, thus increasing the blood pressure reading; however, severe pain can decrease blood
pressure greatly by inhibiting the vasomotor center and producing vasodilation.
4. Race.
African Americans older than 35 years tend to have higher blood pressures than European Americans
of the same age although the exact reasons for these differences are unclear
5.Sex.
After puberty, females usually have lower blood pressures than males of the same age; this difference
is thought to be due to hormonal variations. After menopause, women generally have higher blood
pressures than before. 28
Factors affecting blood pressure
6.Medications.
Many medications, including caffeine, may increase or decrease the blood pressure.
7.Obesity.
Both childhood and adult obesity predispose to hypertension.
8.Diurnal variations.
Pressure is usually lowest early in the morning, when the metabolic rate is lowest, then rises
throughout the day and peaks in the late afternoon or early evening.
9.Medical conditions.
Any condition affecting the cardiac output, blood volume, blood viscosity, and/or compliance of
the arteries has a direct effect on the blood pressure.
10.Temperature.
Because of increased metabolic rate, fever can increase blood pressure. However, external heat
causes vasodilation and decreased blood pressure. Cold causes vasoconstriction and elevates
blood pressure.
29
Factors affecting blood pressure
Hypertension
A blood pressure that is persistently above normal is called hypertension.
 A single elevated blood pressure reading indicates the need for reassessment.
 Hypertension cannot be diagnosed unless an elevated blood pressure is found when
measured twice at different times.
 An elevated blood pressure of unknown cause is called primary hypertension.
 An elevated blood pressure of known cause is called secondary hypertension
Individuals with diastolic blood pressures of 80 to 89 mmHg or systolic blood pressures
of 120 to 139 mmHg should be considered prehypertensive and, without intervention,
may develop cardiac disease.
 Hypertension is when either the systolic BP is higher than 140 mmHg or when the
diastolic blood pressure (BP) is 90 mmHg or higher.
30
31
hypotension
 Hypotension is a blood pressure that is below normal
 Orthostatic hypotension is a blood pressure that decreases
when the client sits or stands.
 Hypotension can also be caused by analgesics ,bleeding, severe
burns, and dehydration.
32
Method of measure B.P
Blood pressure can be assessed directly or indirectly..
 Direct (invasive monitoring) measurement involves the insertion of a catheter into the
brachial, radial, or femoral artery. Arterial pressure is represented as wavelike forms
displayed on a monitor. With correct placement, this pressure reading is highly accurate.
 Noninvasive indirect methods of measuring blood pressure are the auscultatory and
palpatory methods)
33
Noninvasive indirect
Manual sphygmomanometers :
- Mercury and aneroid sphygmomanometer
need to listen for the sounds of the client’s systolic and diastolic blood pressures through a
stethoscope.
Automated sphygmomanometers : Electronic blood pressure devices should be calibrated
periodically to check accuracy.
- Use in hospitals
- Self measurement
- AMBP (ambulatory blood pressure)measurement
- Measurement in community settings
THE PROPER NAME FOR A BLOOD PRESSURE CUFF IS
SPHYGMOMANOMETER
MERCURY
ANEROID
The ambulatory blood pressure monitoring
(ABPM)
Assessing B.P
BLOOD PRESSURE ASSESSMENT
SITES
 The blood pressure is usually assessed in
the client’s upper arm using the brachial
artery and a standard stethoscope.
 Assessing the blood pressure on a client’s
thigh is indicated in these situations :
The blood pressure cannot be measured on
either arm (e.g., because of burns or other
trauma).
37
 Brachial – taken on the upper arm; most common site.
 Radial – taken on the lower arm; possible site for infants or
clients who have very large upper arms.
 Popliteal – taken on the thigh.
 Dorsalis pedis and posterior tibial – taken on the lower leg.
BLOOD PRESSURE ASSESSMENT SITES
 Systolic pressure in the legs is usually higher by 10 to 40 mm Hg (20mmhg) than in the brachial artery, but the
diastolic pressure is the same.
Contraindications to assesse in the client’s upper arm using the brachial artery
1. The brachial artery should not be used to measure blood pressure in those with
arteriovenous fistulas (e.g., for renal dialysis).,
2. Patients who have had trauma to the upper arm, previous mastectomy or a
forearm amputation should not have blood pressure measured on the affected side
at the brachial artery.
3. Blood pressure should not be measured on an arm that has had brachial artery
surgery or is at risk of lymphoedema or burn.
4. The client has an intravenous infusion or blood transfusion in that limb.
38
Accurate blood pressure measurement
I. Patient
II. Equipment
III. Technique
I. Patient - Posture
¤ Patient seated , back
supported, arm bared
at heart level
¤ Five minutes rest
Blood pressure measurement
• Sitting position
• Arm and back are supported.
• Feet should be resting firmly on the
floor
• Feet not dangling.
Position of the arm
• Raise patient arm so that the brachial artery
is roughly at the same height as the heart. If
the arm is held too high, the reading will be
artifactually lowered, and vice versa.
I. Patient - Posture
I. Patient - Posture
I. Patient - Arm
I. Patient - Arm
Popliteal BP +20 mmHg
I. Patient - Circumstances
¤ Quiet , warm room
¤ No caffeine, smoking, 30 minutes.
¤ No talking
Blood pressure measurement
II. Equipment
Blood pressure measurement
Blood pressure is measured with
• a blood pressure cuff,
• a sphygmomanometer, and
• a stethoscope.
The blood pressure cuff consists of
bag, called a bladder, that can be
inflated with air
 Blood pressure cuffs come in various
sizes because the bladder must be the
correct width and length for the client’s
arm
 The arm circumference, not the age of
the client, should always be used to
determine bladder size.
 If the bladder is too narrow, the blood
pressure reading will be erroneously
elevated; if it is too wide, the reading
will be erroneously low
49
II. Equipment
 The width should be 40% of the circumference,
or 20% wider than the diameter of the midpoint,
of the limb on which it is used.
 The length of the bladder also affects the
accuracy of measurement.
 The bladder should be sufficiently long to cover
at least two-thirds of the limb’s circumference.
50
• Non - invasive
• Cuff size
• Cuff size
II. Equipment
Blood pressure measurement
Calibration
Calibration
III. Technique
• Korotkoff Sound no 5 (disappear of sound=DBP)
• Both arms: (if peripheral arterial disease)
• Standing BP: in elderly & diabetic(orthostatic HTN.)
• Cuff at heart level (whatever patient’s position)
Blood pressure measurement
 When taking a blood pressure using a stethoscope,
the nurse identifies phases in the series of sounds
called Korotkoff’s sounds
 Five phases occur but may not always be audible .
The systolic pressure is the point where the first tapping
sound is heard (phase 1).
the diastolic pressure is the point where the sounds
become inaudible (phase 5).
57
Assessing B.P
200
180
160
140
120
100
80
60
40
20
0
No sound
Clear sound
Clear sound
Muffled sound
No sound
Phase 1
Phase 3
Phase 4
Phase 5
Muffling Phase 2
Auscultatory
gap
No sound
mmHg
Korotkoff sounds
Systolic BP
Phase 3
Phase 4
Diastolic BP
BP Measurement Definitions
BP Measurement Definition
SBP First Korotkoff sound*
DBP Fifth Korotkoff sound*
Pulse pressure SBP minus DBP
Mean arterial pressure
BP (blood pressure)
DBP, (diastolic blood pressure)
SBP,( systolic blood pressure).
What is
auscultatory
gap?
61
62
Recording vital signs
 Special electronic and paper graphic flow sheets exist for recording vital
signs.
 Record vital sign and site assessed on vital sign.
 Record in the nurses' notes any accompanying or precipitating symptoms
such as chest pain and dizziness with abnormal BP, shortness of breath with
abnormal respirations, cyanosis with hypoxemia, or flushing and diaphoresis
with elevated temperature.
 Document any interventions initiated as a result of vital sign measurement
such as administration of oxygen therapy, hydration, or an antihypertensive
63
V.S chart
64

More Related Content

Similar to respiratory & B.P.ppt

Lecture 9 Respiratory System.pptx
Lecture 9 Respiratory System.pptxLecture 9 Respiratory System.pptx
Lecture 9 Respiratory System.pptxMesfinShifara
 
Lecture 9 Respiratory System.pptx
Lecture 9 Respiratory System.pptxLecture 9 Respiratory System.pptx
Lecture 9 Respiratory System.pptxMesfinShifara
 
Pressure changes during Respiration
Pressure changes during RespirationPressure changes during Respiration
Pressure changes during RespirationSRILATHA BASHETTI
 
BA&P Respiratory
BA&P RespiratoryBA&P Respiratory
BA&P Respiratorynatjkeen
 
hyperventilation.pptx
hyperventilation.pptxhyperventilation.pptx
hyperventilation.pptxDrkAnwerAli
 
Evaluation of Dyspnea PP.pptx
Evaluation of Dyspnea PP.pptxEvaluation of Dyspnea PP.pptx
Evaluation of Dyspnea PP.pptxVraj99
 
Respiratory physiology and respiratory disorders
Respiratory physiology and respiratory disordersRespiratory physiology and respiratory disorders
Respiratory physiology and respiratory disordersMarvin Gonzaga
 
Effect of exercise on respiration.pdf
Effect of exercise on respiration.pdfEffect of exercise on respiration.pdf
Effect of exercise on respiration.pdfMostafaGouda8
 
Resp Physio and PFTmade by me welcome to
Resp Physio and PFTmade by me welcome toResp Physio and PFTmade by me welcome to
Resp Physio and PFTmade by me welcome tokaqib1234789
 
5. RESPIRATORY physiology.pptx
5. RESPIRATORY physiology.pptx5. RESPIRATORY physiology.pptx
5. RESPIRATORY physiology.pptxAklilu26
 
Pulmophysiology
PulmophysiologyPulmophysiology
PulmophysiologyAyub Abdi
 
Respiratory anatomy ppt2020 vo
Respiratory anatomy ppt2020 voRespiratory anatomy ppt2020 vo
Respiratory anatomy ppt2020 vocsullivan0220
 
Respiration (Tetyana ma'am) Physiology.ppt
Respiration (Tetyana ma'am) Physiology.pptRespiration (Tetyana ma'am) Physiology.ppt
Respiration (Tetyana ma'am) Physiology.pptGauravPrakashGaurav
 
FN 513 SIMARPREET KAUR,RESPIRATION MECHANISM AND REGULATION.pptx
FN 513 SIMARPREET KAUR,RESPIRATION MECHANISM AND REGULATION.pptxFN 513 SIMARPREET KAUR,RESPIRATION MECHANISM AND REGULATION.pptx
FN 513 SIMARPREET KAUR,RESPIRATION MECHANISM AND REGULATION.pptxSimarpreetKaur311857
 
Mechanical Ventilation
Mechanical VentilationMechanical Ventilation
Mechanical VentilationShalini Garg
 
Control of breathing
Control of breathingControl of breathing
Control of breathingAnindya Jana
 
Physiology of Respiratory System
Physiology of Respiratory SystemPhysiology of Respiratory System
Physiology of Respiratory SystemMegha Jayan
 

Similar to respiratory & B.P.ppt (20)

Lecture 9 Respiratory System.pptx
Lecture 9 Respiratory System.pptxLecture 9 Respiratory System.pptx
Lecture 9 Respiratory System.pptx
 
Lecture 9 Respiratory System.pptx
Lecture 9 Respiratory System.pptxLecture 9 Respiratory System.pptx
Lecture 9 Respiratory System.pptx
 
Pressure changes during Respiration
Pressure changes during RespirationPressure changes during Respiration
Pressure changes during Respiration
 
Pressure changes
Pressure changesPressure changes
Pressure changes
 
BA&P Respiratory
BA&P RespiratoryBA&P Respiratory
BA&P Respiratory
 
hyperventilation.pptx
hyperventilation.pptxhyperventilation.pptx
hyperventilation.pptx
 
Evaluation of Dyspnea PP.pptx
Evaluation of Dyspnea PP.pptxEvaluation of Dyspnea PP.pptx
Evaluation of Dyspnea PP.pptx
 
Respiratory physiology and respiratory disorders
Respiratory physiology and respiratory disordersRespiratory physiology and respiratory disorders
Respiratory physiology and respiratory disorders
 
Effect of exercise on respiration.pdf
Effect of exercise on respiration.pdfEffect of exercise on respiration.pdf
Effect of exercise on respiration.pdf
 
Respiratory..ppt
Respiratory..pptRespiratory..ppt
Respiratory..ppt
 
Resp Physio and PFTmade by me welcome to
Resp Physio and PFTmade by me welcome toResp Physio and PFTmade by me welcome to
Resp Physio and PFTmade by me welcome to
 
5. RESPIRATORY physiology.pptx
5. RESPIRATORY physiology.pptx5. RESPIRATORY physiology.pptx
5. RESPIRATORY physiology.pptx
 
Pulmophysiology
PulmophysiologyPulmophysiology
Pulmophysiology
 
Respiratory anatomy ppt2020 vo
Respiratory anatomy ppt2020 voRespiratory anatomy ppt2020 vo
Respiratory anatomy ppt2020 vo
 
Respiration (Tetyana ma'am) Physiology.ppt
Respiration (Tetyana ma'am) Physiology.pptRespiration (Tetyana ma'am) Physiology.ppt
Respiration (Tetyana ma'am) Physiology.ppt
 
FN 513 SIMARPREET KAUR,RESPIRATION MECHANISM AND REGULATION.pptx
FN 513 SIMARPREET KAUR,RESPIRATION MECHANISM AND REGULATION.pptxFN 513 SIMARPREET KAUR,RESPIRATION MECHANISM AND REGULATION.pptx
FN 513 SIMARPREET KAUR,RESPIRATION MECHANISM AND REGULATION.pptx
 
Mechanical Ventilation
Mechanical VentilationMechanical Ventilation
Mechanical Ventilation
 
upper and lower of respiratory system
upper and lower of respiratory system upper and lower of respiratory system
upper and lower of respiratory system
 
Control of breathing
Control of breathingControl of breathing
Control of breathing
 
Physiology of Respiratory System
Physiology of Respiratory SystemPhysiology of Respiratory System
Physiology of Respiratory System
 

More from MosaHasen

7- HssssssssssssssssssTN-2020ثثثثثث.pptx
7- HssssssssssssssssssTN-2020ثثثثثث.pptx7- HssssssssssssssssssTN-2020ثثثثثث.pptx
7- HssssssssssssssssssTN-2020ثثثثثث.pptxMosaHasen
 
Vital_signs-BcccccccccccccvvvvvvvvP-1.ppt
Vital_signs-BcccccccccccccvvvvvvvvP-1.pptVital_signs-BcccccccccccccvvvvvvvvP-1.ppt
Vital_signs-BcccccccccccccvvvvvvvvP-1.pptMosaHasen
 
Lecture 4-Valvularcccccccc Heart Diseases.ppt
Lecture 4-Valvularcccccccc Heart Diseases.pptLecture 4-Valvularcccccccc Heart Diseases.ppt
Lecture 4-Valvularcccccccc Heart Diseases.pptMosaHasen
 
Vital_signdddddddddddddddddddds-BP-1.ppt
Vital_signdddddddddddddddddddds-BP-1.pptVital_signdddddddddddddddddddds-BP-1.ppt
Vital_signdddddddddddddddddddds-BP-1.pptMosaHasen
 
Lecture 4-Valvuddddlar Heart Diseases.ppt
Lecture 4-Valvuddddlar Heart Diseases.pptLecture 4-Valvuddddlar Heart Diseases.ppt
Lecture 4-Valvuddddlar Heart Diseases.pptMosaHasen
 
autonoddddddddddddddddddddddddmic 1 .ppt
autonoddddddddddddddddddddddddmic 1 .pptautonoddddddddddddddddddddddddmic 1 .ppt
autonoddddddddddddddddddddddddmic 1 .pptMosaHasen
 
autdddddddddddddddddddddddddonomic 1 .ppt
autdddddddddddddddddddddddddonomic 1 .pptautdddddddddddddddddddddddddonomic 1 .ppt
autdddddddddddddddddddddddddonomic 1 .pptMosaHasen
 
Vital_sfffffffffffffffffffffigns-BP-1.ppt
Vital_sfffffffffffffffffffffigns-BP-1.pptVital_sfffffffffffffffffffffigns-BP-1.ppt
Vital_sfffffffffffffffffffffigns-BP-1.pptMosaHasen
 
Lecture 4-Valvulfffar Heart Diseases.ppt
Lecture 4-Valvulfffar Heart Diseases.pptLecture 4-Valvulfffar Heart Diseases.ppt
Lecture 4-Valvulfffar Heart Diseases.pptMosaHasen
 
Lecture 4-Valvular Heddddart Diseases.ppt
Lecture 4-Valvular Heddddart Diseases.pptLecture 4-Valvular Heddddart Diseases.ppt
Lecture 4-Valvular Heddddart Diseases.pptMosaHasen
 
Vital_signs-BPdddddddddddddddddddd-1.ppt
Vital_signs-BPdddddddddddddddddddd-1.pptVital_signs-BPdddddddddddddddddddd-1.ppt
Vital_signs-BPdddddddddddddddddddd-1.pptMosaHasen
 
autonomddddddddddddddddddddddddic 1 .ppt
autonomddddddddddddddddddddddddic 1 .pptautonomddddddddddddddddddddddddic 1 .ppt
autonomddddddddddddddddddddddddic 1 .pptMosaHasen
 
Lecture 4-Valvular Hikkeart Diseases.ppt
Lecture 4-Valvular Hikkeart Diseases.pptLecture 4-Valvular Hikkeart Diseases.ppt
Lecture 4-Valvular Hikkeart Diseases.pptMosaHasen
 
Lecture 4-Vajjjlvular Heart Diseases.ppt
Lecture 4-Vajjjlvular Heart Diseases.pptLecture 4-Vajjjlvular Heart Diseases.ppt
Lecture 4-Vajjjlvular Heart Diseases.pptMosaHasen
 
Traudddddddddddddddddddddddddddddma .ppt
Traudddddddddddddddddddddddddddddma .pptTraudddddddddddddddddddddddddddddma .ppt
Traudddddddddddddddddddddddddddddma .pptMosaHasen
 
Traumaaaaaaaaaaaaaaaaaaaaaaaaaaaaaa .ppt
Traumaaaaaaaaaaaaaaaaaaaaaaaaaaaaaa .pptTraumaaaaaaaaaaaaaaaaaaaaaaaaaaaaaa .ppt
Traumaaaaaaaaaaaaaaaaaaaaaaaaaaaaaa .pptMosaHasen
 
parhhhhhhhhhhhhhhhhhhhhhhhhhhathyriod.ppt
parhhhhhhhhhhhhhhhhhhhhhhhhhhathyriod.pptparhhhhhhhhhhhhhhhhhhhhhhhhhhathyriod.ppt
parhhhhhhhhhhhhhhhhhhhhhhhhhhathyriod.pptMosaHasen
 
Vital_signs-BddddddddddddddddddddP-1.ppt
Vital_signs-BddddddddddddddddddddP-1.pptVital_signs-BddddddddddddddddddddP-1.ppt
Vital_signs-BddddddddddddddddddddP-1.pptMosaHasen
 
hydrdddddddddddddddddddddddddddddo..pptx
hydrdddddddddddddddddddddddddddddo..pptxhydrdddddddddddddddddddddddddddddo..pptx
hydrdddddddddddddddddddddddddddddo..pptxMosaHasen
 
autonomicfdfdddddddddddddddddddddf 1 .ppt
autonomicfdfdddddddddddddddddddddf 1 .pptautonomicfdfdddddddddddddddddddddf 1 .ppt
autonomicfdfdddddddddddddddddddddf 1 .pptMosaHasen
 

More from MosaHasen (20)

7- HssssssssssssssssssTN-2020ثثثثثث.pptx
7- HssssssssssssssssssTN-2020ثثثثثث.pptx7- HssssssssssssssssssTN-2020ثثثثثث.pptx
7- HssssssssssssssssssTN-2020ثثثثثث.pptx
 
Vital_signs-BcccccccccccccvvvvvvvvP-1.ppt
Vital_signs-BcccccccccccccvvvvvvvvP-1.pptVital_signs-BcccccccccccccvvvvvvvvP-1.ppt
Vital_signs-BcccccccccccccvvvvvvvvP-1.ppt
 
Lecture 4-Valvularcccccccc Heart Diseases.ppt
Lecture 4-Valvularcccccccc Heart Diseases.pptLecture 4-Valvularcccccccc Heart Diseases.ppt
Lecture 4-Valvularcccccccc Heart Diseases.ppt
 
Vital_signdddddddddddddddddddds-BP-1.ppt
Vital_signdddddddddddddddddddds-BP-1.pptVital_signdddddddddddddddddddds-BP-1.ppt
Vital_signdddddddddddddddddddds-BP-1.ppt
 
Lecture 4-Valvuddddlar Heart Diseases.ppt
Lecture 4-Valvuddddlar Heart Diseases.pptLecture 4-Valvuddddlar Heart Diseases.ppt
Lecture 4-Valvuddddlar Heart Diseases.ppt
 
autonoddddddddddddddddddddddddmic 1 .ppt
autonoddddddddddddddddddddddddmic 1 .pptautonoddddddddddddddddddddddddmic 1 .ppt
autonoddddddddddddddddddddddddmic 1 .ppt
 
autdddddddddddddddddddddddddonomic 1 .ppt
autdddddddddddddddddddddddddonomic 1 .pptautdddddddddddddddddddddddddonomic 1 .ppt
autdddddddddddddddddddddddddonomic 1 .ppt
 
Vital_sfffffffffffffffffffffigns-BP-1.ppt
Vital_sfffffffffffffffffffffigns-BP-1.pptVital_sfffffffffffffffffffffigns-BP-1.ppt
Vital_sfffffffffffffffffffffigns-BP-1.ppt
 
Lecture 4-Valvulfffar Heart Diseases.ppt
Lecture 4-Valvulfffar Heart Diseases.pptLecture 4-Valvulfffar Heart Diseases.ppt
Lecture 4-Valvulfffar Heart Diseases.ppt
 
Lecture 4-Valvular Heddddart Diseases.ppt
Lecture 4-Valvular Heddddart Diseases.pptLecture 4-Valvular Heddddart Diseases.ppt
Lecture 4-Valvular Heddddart Diseases.ppt
 
Vital_signs-BPdddddddddddddddddddd-1.ppt
Vital_signs-BPdddddddddddddddddddd-1.pptVital_signs-BPdddddddddddddddddddd-1.ppt
Vital_signs-BPdddddddddddddddddddd-1.ppt
 
autonomddddddddddddddddddddddddic 1 .ppt
autonomddddddddddddddddddddddddic 1 .pptautonomddddddddddddddddddddddddic 1 .ppt
autonomddddddddddddddddddddddddic 1 .ppt
 
Lecture 4-Valvular Hikkeart Diseases.ppt
Lecture 4-Valvular Hikkeart Diseases.pptLecture 4-Valvular Hikkeart Diseases.ppt
Lecture 4-Valvular Hikkeart Diseases.ppt
 
Lecture 4-Vajjjlvular Heart Diseases.ppt
Lecture 4-Vajjjlvular Heart Diseases.pptLecture 4-Vajjjlvular Heart Diseases.ppt
Lecture 4-Vajjjlvular Heart Diseases.ppt
 
Traudddddddddddddddddddddddddddddma .ppt
Traudddddddddddddddddddddddddddddma .pptTraudddddddddddddddddddddddddddddma .ppt
Traudddddddddddddddddddddddddddddma .ppt
 
Traumaaaaaaaaaaaaaaaaaaaaaaaaaaaaaa .ppt
Traumaaaaaaaaaaaaaaaaaaaaaaaaaaaaaa .pptTraumaaaaaaaaaaaaaaaaaaaaaaaaaaaaaa .ppt
Traumaaaaaaaaaaaaaaaaaaaaaaaaaaaaaa .ppt
 
parhhhhhhhhhhhhhhhhhhhhhhhhhhathyriod.ppt
parhhhhhhhhhhhhhhhhhhhhhhhhhhathyriod.pptparhhhhhhhhhhhhhhhhhhhhhhhhhhathyriod.ppt
parhhhhhhhhhhhhhhhhhhhhhhhhhhathyriod.ppt
 
Vital_signs-BddddddddddddddddddddP-1.ppt
Vital_signs-BddddddddddddddddddddP-1.pptVital_signs-BddddddddddddddddddddP-1.ppt
Vital_signs-BddddddddddddddddddddP-1.ppt
 
hydrdddddddddddddddddddddddddddddo..pptx
hydrdddddddddddddddddddddddddddddo..pptxhydrdddddddddddddddddddddddddddddo..pptx
hydrdddddddddddddddddddddddddddddo..pptx
 
autonomicfdfdddddddddddddddddddddf 1 .ppt
autonomicfdfdddddddddddddddddddddf 1 .pptautonomicfdfdddddddddddddddddddddf 1 .ppt
autonomicfdfdddddddddddddddddddddf 1 .ppt
 

Recently uploaded

Russian Escorts Aishbagh Road * 9548273370 Naughty Call Girls Service in Lucknow
Russian Escorts Aishbagh Road * 9548273370 Naughty Call Girls Service in LucknowRussian Escorts Aishbagh Road * 9548273370 Naughty Call Girls Service in Lucknow
Russian Escorts Aishbagh Road * 9548273370 Naughty Call Girls Service in Lucknowgragteena
 
Call Girls Hyderabad Just Call 9907093804 Top Class Call Girl Service Available
Call Girls Hyderabad Just Call 9907093804 Top Class Call Girl Service AvailableCall Girls Hyderabad Just Call 9907093804 Top Class Call Girl Service Available
Call Girls Hyderabad Just Call 9907093804 Top Class Call Girl Service AvailableDipal Arora
 
Dehradun Call Girls Service 7017441440 Real Russian Girls Looking Models
Dehradun Call Girls Service 7017441440 Real Russian Girls Looking ModelsDehradun Call Girls Service 7017441440 Real Russian Girls Looking Models
Dehradun Call Girls Service 7017441440 Real Russian Girls Looking Modelsindiancallgirl4rent
 
VIP Call Girl Sector 25 Gurgaon Just Call Me 9899900591
VIP Call Girl Sector 25 Gurgaon Just Call Me 9899900591VIP Call Girl Sector 25 Gurgaon Just Call Me 9899900591
VIP Call Girl Sector 25 Gurgaon Just Call Me 9899900591adityaroy0215
 
Hot Call Girl In Ludhiana 👅🥵 9053'900678 Call Girls Service In Ludhiana
Hot  Call Girl In Ludhiana 👅🥵 9053'900678 Call Girls Service In LudhianaHot  Call Girl In Ludhiana 👅🥵 9053'900678 Call Girls Service In Ludhiana
Hot Call Girl In Ludhiana 👅🥵 9053'900678 Call Girls Service In LudhianaRussian Call Girls in Ludhiana
 
Call Girl In Zirakpur ❤️♀️@ 9988299661 Zirakpur Call Girls Near Me ❤️♀️@ Sexy...
Call Girl In Zirakpur ❤️♀️@ 9988299661 Zirakpur Call Girls Near Me ❤️♀️@ Sexy...Call Girl In Zirakpur ❤️♀️@ 9988299661 Zirakpur Call Girls Near Me ❤️♀️@ Sexy...
Call Girl In Zirakpur ❤️♀️@ 9988299661 Zirakpur Call Girls Near Me ❤️♀️@ Sexy...Sheetaleventcompany
 
(Sonam Bajaj) Call Girl in Jaipur- 09257276172 Escorts Service 50% Off with C...
(Sonam Bajaj) Call Girl in Jaipur- 09257276172 Escorts Service 50% Off with C...(Sonam Bajaj) Call Girl in Jaipur- 09257276172 Escorts Service 50% Off with C...
(Sonam Bajaj) Call Girl in Jaipur- 09257276172 Escorts Service 50% Off with C...indiancallgirl4rent
 
❤️♀️@ Jaipur Call Girl Agency ❤️♀️@ Manjeet Russian Call Girls Service in Jai...
❤️♀️@ Jaipur Call Girl Agency ❤️♀️@ Manjeet Russian Call Girls Service in Jai...❤️♀️@ Jaipur Call Girl Agency ❤️♀️@ Manjeet Russian Call Girls Service in Jai...
❤️♀️@ Jaipur Call Girl Agency ❤️♀️@ Manjeet Russian Call Girls Service in Jai...Gfnyt.com
 
Vip Kolkata Call Girls Cossipore 👉 8250192130 ❣️💯 Available With Room 24×7
Vip Kolkata Call Girls Cossipore 👉 8250192130 ❣️💯 Available With Room 24×7Vip Kolkata Call Girls Cossipore 👉 8250192130 ❣️💯 Available With Room 24×7
Vip Kolkata Call Girls Cossipore 👉 8250192130 ❣️💯 Available With Room 24×7Miss joya
 
VIP Call Girls Noida Sia 9711199171 High Class Call Girl Near Me
VIP Call Girls Noida Sia 9711199171 High Class Call Girl Near MeVIP Call Girls Noida Sia 9711199171 High Class Call Girl Near Me
VIP Call Girls Noida Sia 9711199171 High Class Call Girl Near Memriyagarg453
 
💚😋Chandigarh Escort Service Call Girls, ₹5000 To 25K With AC💚😋
💚😋Chandigarh Escort Service Call Girls, ₹5000 To 25K With AC💚😋💚😋Chandigarh Escort Service Call Girls, ₹5000 To 25K With AC💚😋
💚😋Chandigarh Escort Service Call Girls, ₹5000 To 25K With AC💚😋Sheetaleventcompany
 
Leading transformational change: inner and outer skills
Leading transformational change: inner and outer skillsLeading transformational change: inner and outer skills
Leading transformational change: inner and outer skillsHelenBevan4
 
indian Call Girl Panchkula ❤️🍑 9907093804 Low Rate Call Girls Ludhiana Tulsi
indian Call Girl Panchkula ❤️🍑 9907093804 Low Rate Call Girls Ludhiana Tulsiindian Call Girl Panchkula ❤️🍑 9907093804 Low Rate Call Girls Ludhiana Tulsi
indian Call Girl Panchkula ❤️🍑 9907093804 Low Rate Call Girls Ludhiana TulsiHigh Profile Call Girls Chandigarh Aarushi
 
Vip sexy Call Girls Service In Sector 137,9999965857 Young Female Escorts Ser...
Vip sexy Call Girls Service In Sector 137,9999965857 Young Female Escorts Ser...Vip sexy Call Girls Service In Sector 137,9999965857 Young Female Escorts Ser...
Vip sexy Call Girls Service In Sector 137,9999965857 Young Female Escorts Ser...Call Girls Noida
 
💚😋Kolkata Escort Service Call Girls, ₹5000 To 25K With AC💚😋
💚😋Kolkata Escort Service Call Girls, ₹5000 To 25K With AC💚😋💚😋Kolkata Escort Service Call Girls, ₹5000 To 25K With AC💚😋
💚😋Kolkata Escort Service Call Girls, ₹5000 To 25K With AC💚😋Sheetaleventcompany
 

Recently uploaded (20)

Russian Escorts Aishbagh Road * 9548273370 Naughty Call Girls Service in Lucknow
Russian Escorts Aishbagh Road * 9548273370 Naughty Call Girls Service in LucknowRussian Escorts Aishbagh Road * 9548273370 Naughty Call Girls Service in Lucknow
Russian Escorts Aishbagh Road * 9548273370 Naughty Call Girls Service in Lucknow
 
Call Girls Hyderabad Just Call 9907093804 Top Class Call Girl Service Available
Call Girls Hyderabad Just Call 9907093804 Top Class Call Girl Service AvailableCall Girls Hyderabad Just Call 9907093804 Top Class Call Girl Service Available
Call Girls Hyderabad Just Call 9907093804 Top Class Call Girl Service Available
 
Dehradun Call Girls Service 7017441440 Real Russian Girls Looking Models
Dehradun Call Girls Service 7017441440 Real Russian Girls Looking ModelsDehradun Call Girls Service 7017441440 Real Russian Girls Looking Models
Dehradun Call Girls Service 7017441440 Real Russian Girls Looking Models
 
VIP Call Girl Sector 25 Gurgaon Just Call Me 9899900591
VIP Call Girl Sector 25 Gurgaon Just Call Me 9899900591VIP Call Girl Sector 25 Gurgaon Just Call Me 9899900591
VIP Call Girl Sector 25 Gurgaon Just Call Me 9899900591
 
Hot Call Girl In Ludhiana 👅🥵 9053'900678 Call Girls Service In Ludhiana
Hot  Call Girl In Ludhiana 👅🥵 9053'900678 Call Girls Service In LudhianaHot  Call Girl In Ludhiana 👅🥵 9053'900678 Call Girls Service In Ludhiana
Hot Call Girl In Ludhiana 👅🥵 9053'900678 Call Girls Service In Ludhiana
 
Call Girl In Zirakpur ❤️♀️@ 9988299661 Zirakpur Call Girls Near Me ❤️♀️@ Sexy...
Call Girl In Zirakpur ❤️♀️@ 9988299661 Zirakpur Call Girls Near Me ❤️♀️@ Sexy...Call Girl In Zirakpur ❤️♀️@ 9988299661 Zirakpur Call Girls Near Me ❤️♀️@ Sexy...
Call Girl In Zirakpur ❤️♀️@ 9988299661 Zirakpur Call Girls Near Me ❤️♀️@ Sexy...
 
(Sonam Bajaj) Call Girl in Jaipur- 09257276172 Escorts Service 50% Off with C...
(Sonam Bajaj) Call Girl in Jaipur- 09257276172 Escorts Service 50% Off with C...(Sonam Bajaj) Call Girl in Jaipur- 09257276172 Escorts Service 50% Off with C...
(Sonam Bajaj) Call Girl in Jaipur- 09257276172 Escorts Service 50% Off with C...
 
❤️♀️@ Jaipur Call Girl Agency ❤️♀️@ Manjeet Russian Call Girls Service in Jai...
❤️♀️@ Jaipur Call Girl Agency ❤️♀️@ Manjeet Russian Call Girls Service in Jai...❤️♀️@ Jaipur Call Girl Agency ❤️♀️@ Manjeet Russian Call Girls Service in Jai...
❤️♀️@ Jaipur Call Girl Agency ❤️♀️@ Manjeet Russian Call Girls Service in Jai...
 
Vip Kolkata Call Girls Cossipore 👉 8250192130 ❣️💯 Available With Room 24×7
Vip Kolkata Call Girls Cossipore 👉 8250192130 ❣️💯 Available With Room 24×7Vip Kolkata Call Girls Cossipore 👉 8250192130 ❣️💯 Available With Room 24×7
Vip Kolkata Call Girls Cossipore 👉 8250192130 ❣️💯 Available With Room 24×7
 
VIP Call Girls Noida Sia 9711199171 High Class Call Girl Near Me
VIP Call Girls Noida Sia 9711199171 High Class Call Girl Near MeVIP Call Girls Noida Sia 9711199171 High Class Call Girl Near Me
VIP Call Girls Noida Sia 9711199171 High Class Call Girl Near Me
 
Russian Call Girls in Dehradun Komal 🔝 7001305949 🔝 📍 Independent Escort Serv...
Russian Call Girls in Dehradun Komal 🔝 7001305949 🔝 📍 Independent Escort Serv...Russian Call Girls in Dehradun Komal 🔝 7001305949 🔝 📍 Independent Escort Serv...
Russian Call Girls in Dehradun Komal 🔝 7001305949 🔝 📍 Independent Escort Serv...
 
#9711199012# African Student Escorts in Delhi 😘 Call Girls Delhi
#9711199012# African Student Escorts in Delhi 😘 Call Girls Delhi#9711199012# African Student Escorts in Delhi 😘 Call Girls Delhi
#9711199012# African Student Escorts in Delhi 😘 Call Girls Delhi
 
💚😋Chandigarh Escort Service Call Girls, ₹5000 To 25K With AC💚😋
💚😋Chandigarh Escort Service Call Girls, ₹5000 To 25K With AC💚😋💚😋Chandigarh Escort Service Call Girls, ₹5000 To 25K With AC💚😋
💚😋Chandigarh Escort Service Call Girls, ₹5000 To 25K With AC💚😋
 
VIP Call Girls Lucknow Isha 🔝 9719455033 🔝 🎶 Independent Escort Service Lucknow
VIP Call Girls Lucknow Isha 🔝 9719455033 🔝 🎶 Independent Escort Service LucknowVIP Call Girls Lucknow Isha 🔝 9719455033 🔝 🎶 Independent Escort Service Lucknow
VIP Call Girls Lucknow Isha 🔝 9719455033 🔝 🎶 Independent Escort Service Lucknow
 
Leading transformational change: inner and outer skills
Leading transformational change: inner and outer skillsLeading transformational change: inner and outer skills
Leading transformational change: inner and outer skills
 
indian Call Girl Panchkula ❤️🍑 9907093804 Low Rate Call Girls Ludhiana Tulsi
indian Call Girl Panchkula ❤️🍑 9907093804 Low Rate Call Girls Ludhiana Tulsiindian Call Girl Panchkula ❤️🍑 9907093804 Low Rate Call Girls Ludhiana Tulsi
indian Call Girl Panchkula ❤️🍑 9907093804 Low Rate Call Girls Ludhiana Tulsi
 
Call Girls in Lucknow Esha 🔝 8923113531 🔝 🎶 Independent Escort Service Lucknow
Call Girls in Lucknow Esha 🔝 8923113531  🔝 🎶 Independent Escort Service LucknowCall Girls in Lucknow Esha 🔝 8923113531  🔝 🎶 Independent Escort Service Lucknow
Call Girls in Lucknow Esha 🔝 8923113531 🔝 🎶 Independent Escort Service Lucknow
 
Call Girl Guwahati Aashi 👉 7001305949 👈 🔝 Independent Escort Service Guwahati
Call Girl Guwahati Aashi 👉 7001305949 👈 🔝 Independent Escort Service GuwahatiCall Girl Guwahati Aashi 👉 7001305949 👈 🔝 Independent Escort Service Guwahati
Call Girl Guwahati Aashi 👉 7001305949 👈 🔝 Independent Escort Service Guwahati
 
Vip sexy Call Girls Service In Sector 137,9999965857 Young Female Escorts Ser...
Vip sexy Call Girls Service In Sector 137,9999965857 Young Female Escorts Ser...Vip sexy Call Girls Service In Sector 137,9999965857 Young Female Escorts Ser...
Vip sexy Call Girls Service In Sector 137,9999965857 Young Female Escorts Ser...
 
💚😋Kolkata Escort Service Call Girls, ₹5000 To 25K With AC💚😋
💚😋Kolkata Escort Service Call Girls, ₹5000 To 25K With AC💚😋💚😋Kolkata Escort Service Call Girls, ₹5000 To 25K With AC💚😋
💚😋Kolkata Escort Service Call Girls, ₹5000 To 25K With AC💚😋
 

respiratory & B.P.ppt

  • 1. 1 Fundamental of Nursing Vital Signs Respiratory rate & Blood Pressure Dr. mosa alfageh
  • 2. After completing this lecture, each student will be able to:  Define respiration  Describe the mechanics of breathing and the mechanisms that control respirations  List the factors that influence the body respiration  List the characteristics that should be included when assessing respiration  Define blood pressure  Understand the alteration in blood pressure  List the factors that influence the blood pressure  Identify the sites used to assess the blood pressure 2
  • 3. 3
  • 4.  Respiration is the act of breathing. (is the process by which the lungs bring oxygen into the body and remove carbon dioxide) Inhalation or inspiration refers to the intake of air into the lungs. Exhalation or expiration refers to breathing out or the movement of gases from the lungs to the atmosphere. Ventilation is also used to refer to the movement of air in and out of the lungs. 4 There are basically two types of breathing:  Costal (thoracic)breathing Costal breathing involves the external intercostal muscles and other accessory muscles, such as the sternocleidomastoid muscles. It can be observed by the movement of the chest upward and outward.  Diaphragmatic (abdominal) breathing Diaphragmatic breathing involves the contraction and relaxation of the diaphragm, and it is observed by the movement of the abdomen, which occurs as a result of the diaphragm’s contraction and downward movement.
  • 5. Mechanics and regulation of breathing During inhalation, the following processes normally occur : The diaphragm contracts (flattens), the ribs move upward and outward, and the sternum moves outward, thus enlarging the thorax and permitting the lungs to expand. 5
  • 6. During exhalation: the diaphragm relaxes, the ribs move downward and inward, and the sternum moves inward, thus decreasing the size of the thorax as the lungs are compressed. 6 Mechanics and regulation of breathing
  • 7. Normal breath inspiration animation, awake Diaghram contracts Chest volume Pleural pressure Air moves down pressure gradient to fill lungs -2cm H20 -7cm H20 Alveolar pressure falls Normal breath
  • 8. Normal breath expiration animation, awake Diaghram relaxes Pleural / Chest volume  Pleural pressure rises Normal breath Alveolar pressure rises Air moves down pressure gradient out of lungs
  • 9.
  • 11.  Normal breathing is automatic and effortless. A normal adult inspiration lasts 1 to 1.5 seconds, and an expiration lasts 2 to 3 seconds.  Respiration is controlled by Respiratory centers in the medulla oblongata and the pons of the brain  Chemoreceptors located centrally in the medulla and peripherally in the carotid and aortic bodies. These centers and receptors respond to changes in the concentrations of oxygen (O2), carbon dioxide (CO2), and hydrogen (H+) in the arterial blood 11 Mechanics and regulation of breathing
  • 12. Assessing respirations  Resting respirations should be assessed when the client is relaxed because exercise affects respirations, increasing their rate and depth.  Anxiety is likely to affect respiratory rate and depth as well. 12 Before assessing a client’s respirations, a nurse should be aware of the following : • The client’s normal breathing pattern • The influence of the client’s health problems on respirations • Any medications or therapies that might affect respirations • The relationship of the client’s respirations to cardiovascular function.
  • 13. Assessing respirations  The rate, depth, rhythm, quality, and effectiveness of respirations should be assessed.  The respiratory rate is normally described in breaths per minute.  Breathing that is normal in rate and depth is called eupnea.  Abnormally slow respirations are referred to as bradypnea  Abnormally fast respirations are called tachypnea  Apnea is the absence of breathing. 13 The depth  The depth of a person’s respirations can be established by watching the movement of the chest.  Respiratory depth is generally described as normal, deep, or shallow. Deep respirations are those in which a large volume of air is inhaled and exhaled, inflating most of the lungs. Shallow respirations involve the exchange of a small volume of air and often the minimal use of lung tissue.  During a normal inspiration and expiration, an adult takes in about 500 mL of air. This volume is called the tidal volume.  Hyperventilation refers to very deep, rapid respirations.  Hypoventilation refers to very shallow respirations.
  • 14. Assessing respirations Respiratory rhythm Respiratory rhythm refers to the regularity of the expirations and the inspirations. Normally, respirations are evenly spaced.  Respiratory rhythm can be described as regular or irregular.  An infant’s respiratory rhythm may be less regular than an adult’s 14 Respiratory quality or character refers to those aspects of breathing that are different from normal, effortless breathing.  Two of these aspects are The amount of effort a client must exert to breathe  The sound of breathing.  Usually, breathing does not require noticeable effort.  Sometimes, however, clients can breathe only with substantial effort—this is referred to as labored breathing.  The sound of breathing is also significant. Normal breathing is silent, but a number of abnormal sounds such as a wheeze are obvious to the nurse’s ear.  Many sounds occur as a result of the presence of fluid in the lungs and are most clearly heard with a stethoscope.
  • 15. Definitions  Eupnea ; breathing normal in rate and depth (12-20 breath /min)  Bradypnea ; rate of breathing is regular but abnormally slow (less than 10 breath/min)  Apnea respirations cease for several seconds . Persistent cessation results in cardiac arrest  Kussmaul’s respirations ;respirations that are regular but abnormally deep and increase in rate . It is associated with DKA(DIABETIC KETOACIDOSIS)  Dyspnea; the term for difficult or painful breathing  Orthopnea ; when the difficulty is so marked that the client can breathe only when in an upright position  Cyanosis; (bluish tinge); especially in the lips (circumoral cyanosis) and mucus membranes of the mouth . In severe conditions , cyanosis spreads to the nails and extremities . An excess of carbon dioxide causes the bluish tinge 15
  • 16. 16
  • 17. 17
  • 18. Factors affecting respirations  Exercise (increases metabolism), Exercise increases rate and depth to meet the body's need for additional oxygen and to rid the co2  Acute pain Pain alters rate and rhythm of respirations ; breathing becomes shallow Patient inhibits or splints chest wall movement when pain is in area of chest or abdomen  Anxiety Anxiety increases respiration rate and depth 18  Smoking Chronic smoking changes pulmonary airways, resulting in increased rate of respirations at rest when not smoking Body position  A straight , erect posture promotes full chest expansion  Lying flat prevents full chest expansion
  • 19. Factors affecting respirations 19  Medications For example, narcotics such as morphine and large doses of barbiturates such as pentobarbital depress the respiratory centers in the brain, thereby depressing the respiratory rate and depth.  Neurological injury Injury to brainstem impairs respiratory center and inhibits respiratory rate and rhythm  Hemoglobin functions Decrease hemoglobin levels (anemia) reduce oxygen carrying capacity of the blood , which increase respiratory rate Abnormal blood cell function (e.g sickle cell anemia) reduce ability of hemoglobin to carry oxygen , which increase respiratory rate and depth
  • 20. 20
  • 21. Measurement of oxygen saturation (pulse oximetry)  Assessment of Diffusion and Perfusion  Evaluate the respiratory processes of diffusion and perfusion by measuring the oxygen saturation of the blood.  The percent of hemoglobin that is bound with oxygen in the arteries is the percent of saturation of hemoglobin (or SaO2). It is usually between 95% and 100%. 21  The saturation of venous blood (SVO2) is lower because the tissues have removed some of the oxygen from the hemoglobin molecules. The usual value for Sv02, which is 70%.  Measurement of Arterial Oxygen Saturation.  A pulse oximeter is a noninvasive device that estimates a client's arterial blood oxygen saturation (SaO2) by means of a sensor attached to the client's finger, toe, nose, earlobe, or forehead (or around the hand or foot of a neonate).  The pulse oximeter can detect hypoxemia (low oxygen saturation) before clinical signs and symptoms, such as a dusky color to skin and nail beds develop.  Normal oxygen saturation is 95% to 100%, and below 70% is life threatening.
  • 23. 23
  • 24. Blood pressure Arterial blood pressure is a measure of the pressure exerted by the blood as it flows through the arteries. Because the blood moves in waves, there are two blood pressure measurements. The systolic pressure is the pressure of the blood as a result of contraction of the ventricles, that is, the pressure of the height of the blood wave. The diastolic pressure is the pressure when the ventricles are at rest. Diastolic pressure ;is the lower pressure, present at all times within the arteries.  The difference between the diastolic and the systolic pressures is called the pulse pressure.  A normal pulse pressure is about 40 mmHg but can be as high as 100 mmHg during exercise 24  Blood pressure is measured in millimeters of mercury (mmHg) and recorded as a fraction: systolic pressure over the diastolic pressure.  A typical blood pressure for a healthy adult is 120/80 mmHg (pulse pressure of 40).  Sometimes, it is useful to determine the mean arterial pressure (MAP) because this represents the pressure actually delivered to the body’s organs.  A normal MAP is 70 to 110 mmHg.
  • 25. Determinants of blood pressure Arterial blood pressure is the result of several factors: 1. The pumping action of the heart 2. The peripheral vascular resistance (the resistance supplied by the blood vessels through which the blood flows) 3. The blood volume and viscosity. PUMPING ACTION OF THE HEART  When the pumping action of the heart is weak, less blood is pumped into arteries (lower cardiac output), and the blood pressure decreases.  When the heart’s pumping action is strong and the volume of blood pumped into the circulation increases (higher cardiac output), the blood pressure increases. 25 PERIPHERAL VASCULAR RESISTANCE  Peripheral resistance can increase blood pressure. The diastolic pressure especially is affected.  Some factors that create resistance in the arterial system are the capacity of the arterioles and capillaries, the compliance of the arteries, and the viscosity of the blood.  The internal diameter or capacity of the arterioles and the capillaries determines in great part the peripheral resistance to the blood in the body. The smaller the space within a vessel, the greater the resistance. Normally, the arterioles are in a state of partial constriction. Increased vasoconstriction, such as occurs with smoking, raises the blood pressure, whereas decreased vasoconstriction lowers the blood pressure.
  • 26. Determinants of blood pressure BLOOD VOLUME When the blood volume decreases (for example, as a result of a hemorrhage or dehydration), the blood pressure decreases because of decreased fluid in the arteries.  Conversely, when the volume increases (for example, as a result of a rapid intravenous infusion), the blood pressure increases because of the greater fluid volume within the circulatory system. BLOOD VISCOSITY  Blood pressure is higher when the blood is highly viscous (thick), that is, when the proportion of red blood cells to the blood plasma is high.  This proportion is referred to as the hematocrit. The viscosity increases markedly when the hematocrit is more than 60% to 65%. 26
  • 27. Factors affecting blood pressure 1. Age 2. Exercise 3. Stress 4. Race 5. Gender 6. Medications 7. Obesity 8. Diurnal variations 9. Medical conditions 10. Temperature 27
  • 28. 1.Age Newborns have a systolic pressure of about 75 mmHg. The pressure rises with age, 2.Exercise. Physical activity increases the cardiac output and hence the blood pressure. For reliable assessment of resting blood pressure, wait 20 to 30 minutes following exercise. 3.Stress. Stimulation of the sympathetic nervous system increases cardiac output and vasoconstriction of the arterioles, thus increasing the blood pressure reading; however, severe pain can decrease blood pressure greatly by inhibiting the vasomotor center and producing vasodilation. 4. Race. African Americans older than 35 years tend to have higher blood pressures than European Americans of the same age although the exact reasons for these differences are unclear 5.Sex. After puberty, females usually have lower blood pressures than males of the same age; this difference is thought to be due to hormonal variations. After menopause, women generally have higher blood pressures than before. 28 Factors affecting blood pressure
  • 29. 6.Medications. Many medications, including caffeine, may increase or decrease the blood pressure. 7.Obesity. Both childhood and adult obesity predispose to hypertension. 8.Diurnal variations. Pressure is usually lowest early in the morning, when the metabolic rate is lowest, then rises throughout the day and peaks in the late afternoon or early evening. 9.Medical conditions. Any condition affecting the cardiac output, blood volume, blood viscosity, and/or compliance of the arteries has a direct effect on the blood pressure. 10.Temperature. Because of increased metabolic rate, fever can increase blood pressure. However, external heat causes vasodilation and decreased blood pressure. Cold causes vasoconstriction and elevates blood pressure. 29 Factors affecting blood pressure
  • 30. Hypertension A blood pressure that is persistently above normal is called hypertension.  A single elevated blood pressure reading indicates the need for reassessment.  Hypertension cannot be diagnosed unless an elevated blood pressure is found when measured twice at different times.  An elevated blood pressure of unknown cause is called primary hypertension.  An elevated blood pressure of known cause is called secondary hypertension Individuals with diastolic blood pressures of 80 to 89 mmHg or systolic blood pressures of 120 to 139 mmHg should be considered prehypertensive and, without intervention, may develop cardiac disease.  Hypertension is when either the systolic BP is higher than 140 mmHg or when the diastolic blood pressure (BP) is 90 mmHg or higher. 30
  • 31. 31
  • 32. hypotension  Hypotension is a blood pressure that is below normal  Orthostatic hypotension is a blood pressure that decreases when the client sits or stands.  Hypotension can also be caused by analgesics ,bleeding, severe burns, and dehydration. 32
  • 33. Method of measure B.P Blood pressure can be assessed directly or indirectly..  Direct (invasive monitoring) measurement involves the insertion of a catheter into the brachial, radial, or femoral artery. Arterial pressure is represented as wavelike forms displayed on a monitor. With correct placement, this pressure reading is highly accurate.  Noninvasive indirect methods of measuring blood pressure are the auscultatory and palpatory methods) 33 Noninvasive indirect Manual sphygmomanometers : - Mercury and aneroid sphygmomanometer need to listen for the sounds of the client’s systolic and diastolic blood pressures through a stethoscope. Automated sphygmomanometers : Electronic blood pressure devices should be calibrated periodically to check accuracy. - Use in hospitals - Self measurement - AMBP (ambulatory blood pressure)measurement - Measurement in community settings
  • 34. THE PROPER NAME FOR A BLOOD PRESSURE CUFF IS SPHYGMOMANOMETER MERCURY ANEROID
  • 35.
  • 36. The ambulatory blood pressure monitoring (ABPM)
  • 37. Assessing B.P BLOOD PRESSURE ASSESSMENT SITES  The blood pressure is usually assessed in the client’s upper arm using the brachial artery and a standard stethoscope.  Assessing the blood pressure on a client’s thigh is indicated in these situations : The blood pressure cannot be measured on either arm (e.g., because of burns or other trauma). 37  Brachial – taken on the upper arm; most common site.  Radial – taken on the lower arm; possible site for infants or clients who have very large upper arms.  Popliteal – taken on the thigh.  Dorsalis pedis and posterior tibial – taken on the lower leg.
  • 38. BLOOD PRESSURE ASSESSMENT SITES  Systolic pressure in the legs is usually higher by 10 to 40 mm Hg (20mmhg) than in the brachial artery, but the diastolic pressure is the same. Contraindications to assesse in the client’s upper arm using the brachial artery 1. The brachial artery should not be used to measure blood pressure in those with arteriovenous fistulas (e.g., for renal dialysis)., 2. Patients who have had trauma to the upper arm, previous mastectomy or a forearm amputation should not have blood pressure measured on the affected side at the brachial artery. 3. Blood pressure should not be measured on an arm that has had brachial artery surgery or is at risk of lymphoedema or burn. 4. The client has an intravenous infusion or blood transfusion in that limb. 38
  • 39. Accurate blood pressure measurement I. Patient II. Equipment III. Technique
  • 40. I. Patient - Posture ¤ Patient seated , back supported, arm bared at heart level ¤ Five minutes rest Blood pressure measurement • Sitting position • Arm and back are supported. • Feet should be resting firmly on the floor • Feet not dangling. Position of the arm • Raise patient arm so that the brachial artery is roughly at the same height as the heart. If the arm is held too high, the reading will be artifactually lowered, and vice versa.
  • 41. I. Patient - Posture
  • 42.
  • 43. I. Patient - Posture
  • 47. I. Patient - Circumstances ¤ Quiet , warm room ¤ No caffeine, smoking, 30 minutes. ¤ No talking Blood pressure measurement
  • 48. II. Equipment Blood pressure measurement Blood pressure is measured with • a blood pressure cuff, • a sphygmomanometer, and • a stethoscope. The blood pressure cuff consists of bag, called a bladder, that can be inflated with air
  • 49.  Blood pressure cuffs come in various sizes because the bladder must be the correct width and length for the client’s arm  The arm circumference, not the age of the client, should always be used to determine bladder size.  If the bladder is too narrow, the blood pressure reading will be erroneously elevated; if it is too wide, the reading will be erroneously low 49 II. Equipment
  • 50.  The width should be 40% of the circumference, or 20% wider than the diameter of the midpoint, of the limb on which it is used.  The length of the bladder also affects the accuracy of measurement.  The bladder should be sufficiently long to cover at least two-thirds of the limb’s circumference. 50
  • 51.
  • 52.
  • 53. • Non - invasive
  • 54. • Cuff size • Cuff size
  • 55. II. Equipment Blood pressure measurement Calibration Calibration
  • 56. III. Technique • Korotkoff Sound no 5 (disappear of sound=DBP) • Both arms: (if peripheral arterial disease) • Standing BP: in elderly & diabetic(orthostatic HTN.) • Cuff at heart level (whatever patient’s position) Blood pressure measurement
  • 57.  When taking a blood pressure using a stethoscope, the nurse identifies phases in the series of sounds called Korotkoff’s sounds  Five phases occur but may not always be audible . The systolic pressure is the point where the first tapping sound is heard (phase 1). the diastolic pressure is the point where the sounds become inaudible (phase 5). 57 Assessing B.P
  • 58. 200 180 160 140 120 100 80 60 40 20 0 No sound Clear sound Clear sound Muffled sound No sound Phase 1 Phase 3 Phase 4 Phase 5 Muffling Phase 2 Auscultatory gap No sound mmHg Korotkoff sounds Systolic BP Phase 3 Phase 4 Diastolic BP
  • 59.
  • 60. BP Measurement Definitions BP Measurement Definition SBP First Korotkoff sound* DBP Fifth Korotkoff sound* Pulse pressure SBP minus DBP Mean arterial pressure BP (blood pressure) DBP, (diastolic blood pressure) SBP,( systolic blood pressure).
  • 62. 62
  • 63. Recording vital signs  Special electronic and paper graphic flow sheets exist for recording vital signs.  Record vital sign and site assessed on vital sign.  Record in the nurses' notes any accompanying or precipitating symptoms such as chest pain and dizziness with abnormal BP, shortness of breath with abnormal respirations, cyanosis with hypoxemia, or flushing and diaphoresis with elevated temperature.  Document any interventions initiated as a result of vital sign measurement such as administration of oxygen therapy, hydration, or an antihypertensive 63

Editor's Notes

  1. ميكانيكا وتنظيم التنفس التنفس الطبيعي بيكون بدون مجهود المستقبلات الكيمائية
  2. The respiratory rate ; observe a full inspiration and expiration per minute Ventilatory depth ; assess the depth of respiration by observing the movements in the chest wall يجب تقييم التنفس عنما يكون المريض مرتاح الان التمرين يوثر ع القلق ممكن يوثر نمط التنفس المشاكل الصحية ادوية او علاج قد يوثر ع التنفس علاقة التنفس ب القلب و الاوعية
  3. Dep استنشاق كمية كبيرة مما ادي الي تضخم معظم الرئتين
  4. انتظام الشهيق والزفير نوعية او طبيعه الجهاز التنفسي بيذل مجهود عادة لا يتطلب التنفس جهدا ملحوضا ومع ذلك في بعض الأحيان لا يستطيع المرضى التنفس الا بجهد كبير وهذا م يشار اليه ب صعوبة التنفس
  5. Infants can be considered children anywhere from birth to 1 year old Infants can be considered children anywhere from birth to 1 year old toddler around 1 year (12 months) of age and is typically considered one until around 3-years-old Preschool 3-6 School age child (6 year through 12 years) Adolescents (13 years through 17 years.
  6. General anesthesia
  7. he key difference between perfusion and diffusion is, perfusion is the blood flow through a certain mass of the tissue in a unit time whereas, diffusion is the passive movement of particles along a concentration gradient (gas exchange in alveoli
  8.  help your care team predict your risk of heart and blood vessel events, such as heart attacks and strokes. A pulse pressure greater than 60 is a risk factor for heart disease
  9. Age Newborns have a systolic pressure of about 75 mmHg. The pressure rises with age, In older adults, elasticity of the arteries is decreased—the arteries are more rigid and less yielding to the pressure of the blood. This produces an elevated systolic pressure. Because the walls no longer retract as flexibly with decreased pressure, the diastolic pressure may also be high.
  10. Cuff size : 12-13 cm x 35 cm Sphygmomanometer : types
  11.  orthostatic HTN an increase in systolic blood pressure of 20 mmHg when changing position from supine to standing