Refreshing our memory
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The human genome project was a large, international scientific research project that began in 1990 with the goal of mapping the entire human genome and determining the sequence of chemical base pairs that make up human DNA. It was coordinated by the US Department of Energy and National Institutes of Health. By 2003, the project had generated a rough draft of the human genome sequence and identified most human genes. The goals of the project included developing chromosome maps, sequencing the human genome, analyzing genetic variation, and identifying all human genes. The project has led to many medical and other applications such as improved disease diagnosis, drug development, and agriculture.
Genetic Memory
This document summarizes genetic research into the proteins involved in memory formation. Key findings include:
- mBDNF (mature brain-derived neurotrophic factor) helps neurons communicate and form long-term memories.
- Experiments showed mBDNF is essential for long-term potential (LTP) and memory formation.
- The enzyme TPA (tissue plasminogen activator) and plasminogen are needed to convert proBDNF into active mBDNF.
- The enzyme CPE (carboxypeptidase E) delivers proBDNF to neurons where it is converted to mBDNF and released to form memories.
- Future applications could allow extracting and storing memories in
Human genome
The document discusses the Human Genome Project, an international research effort begun in 1990 with the goals of mapping all of the genes in human DNA and determining the sequences of chemical base pairs that make up human DNA. It aimed to identify the approximately 30,000 genes in humans, determine the sequences of the 3 billion base pairs of human DNA, and address ethical issues. The project was originally expected to last 15 years but was completed earlier than expected, in 2003, due to rapid technological advances.
Human genome project ()
This document is not about ways to keep cool in Kathmandu. It discusses the Human Genome Project.
Human genome project
The Human Genome Project was an international scientific research project that aimed to determine the complete sequence of nucleotide base pairs that make up human DNA. It was launched in 1990, formally began in 1990, and was declared complete in 2003. The goals were to map all human genes, determine the sequences of the 3 billion chemical base pairs in human DNA, and address related ethical issues. Key outcomes included identifying approximately 22,300 protein-coding genes in humans and applications in molecular medicine, cancer research, and forensics.
Human genome project 1
The document discusses the history and goals of the Human Genome Project, which began in 1990 with the aim of identifying all human genes and determining the sequence of DNA base pairs. Some key goals were to map all genes, determine DNA sequences, and address ethical issues. The project was completed earlier than expected in 2003 and has led to over 1,800 disease genes being discovered and many medical benefits like new diagnostic tests.
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Mutations Powerpoint
The document discusses different types of mutations that can occur in DNA, including point mutations, frameshift mutations, and chromosomal mutations. Point mutations involve a substitution, insertion, or deletion of a single nucleotide and can change one amino acid. Frameshift mutations are insertions or deletions that shift the reading frame and change all subsequent amino acids. Chromosomal mutations involve changes in chromosome number or structure. The document also describes mutagens like chemicals and radiation that can cause mutations, and notes mutations can have harmful, beneficial, or no effects on organisms.
Human genome project[1]
The document summarizes basic principles of genetics including alleles, dominant and recessive traits, and how traits are inherited from parents. It then discusses the goals and accomplishments of the Human Genome Project which was completed in 2000, two years ahead of schedule. The document notes some social, ethical, and legal implications of the project, including issues around genetic discrimination and patenting of DNA sequences. It also describes different types of genetic disorders like single gene disorders, chromosomal abnormalities, and multifactorial disorders.
Human genome project
The human genome project was a large, international scientific research project that began in 1990 with the goal of mapping the entire human genome and determining the sequence of chemical base pairs that make up human DNA. It was coordinated by the US Department of Energy and National Institutes of Health. By 2003, the project had generated a rough draft of the human genome sequence and identified most human genes. The goals of the project included developing chromosome maps, sequencing the human genome, analyzing genetic variation, and identifying all human genes. The project has led to many medical and other applications such as improved disease diagnosis, drug development, and agriculture.
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This document summarizes genetic research into the proteins involved in memory formation. Key findings include:
- mBDNF (mature brain-derived neurotrophic factor) helps neurons communicate and form long-term memories.
- Experiments showed mBDNF is essential for long-term potential (LTP) and memory formation.
- The enzyme TPA (tissue plasminogen activator) and plasminogen are needed to convert proBDNF into active mBDNF.
- The enzyme CPE (carboxypeptidase E) delivers proBDNF to neurons where it is converted to mBDNF and released to form memories.
- Future applications could allow extracting and storing memories in
Human genome
The document discusses the Human Genome Project, an international research effort begun in 1990 with the goals of mapping all of the genes in human DNA and determining the sequences of chemical base pairs that make up human DNA. It aimed to identify the approximately 30,000 genes in humans, determine the sequences of the 3 billion base pairs of human DNA, and address ethical issues. The project was originally expected to last 15 years but was completed earlier than expected, in 2003, due to rapid technological advances.
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This document is not about ways to keep cool in Kathmandu. It discusses the Human Genome Project.
Human genome project
The Human Genome Project was an international scientific research project that aimed to determine the complete sequence of nucleotide base pairs that make up human DNA. It was launched in 1990, formally began in 1990, and was declared complete in 2003. The goals were to map all human genes, determine the sequences of the 3 billion chemical base pairs in human DNA, and address related ethical issues. Key outcomes included identifying approximately 22,300 protein-coding genes in humans and applications in molecular medicine, cancer research, and forensics.
Human genome project 1
The document discusses the history and goals of the Human Genome Project, which began in 1990 with the aim of identifying all human genes and determining the sequence of DNA base pairs. Some key goals were to map all genes, determine DNA sequences, and address ethical issues. The project was completed earlier than expected in 2003 and has led to over 1,800 disease genes being discovered and many medical benefits like new diagnostic tests.
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The document summarizes key aspects of the human genome and genome projects. It discusses that a genome contains an organism's complete DNA including all genes. It describes the physical structure of human DNA including nuclear DNA, mitochondrial DNA, and RNA. It provides details on the goals and completion of the Human Genome Project in 2003, two years ahead of schedule. The project aimed to identify all human genes and map the 3 billion base pairs of human DNA.
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This document discusses different types of mutations that can occur in DNA, including base changes, insertions/deletions of bases, frameshift mutations, and trinucleotide repeats. It provides examples of different mutations that cause conditions like sickle cell anemia, beta-thalassemia, Huntington's disease, and retinoblastoma. The key points are that mutations can be random changes to DNA that provide raw material for evolution, and they can occur in germ cells or somatic cells with different effects on inheritance and disease.
COMMON GENETIC DISORDERS
This document discusses several genetic disorders and diseases:
1. It lists 13 genetic diseases including Gaucher Disease, Huntington's Disease, Hemophilia, Parkinson's Disease, and Cystic Fibrosis.
2. It categorizes genetic disorders into four types - single gene disorders, multifactorial disorders caused by multiple genes and environment, chromosomal abnormalities, and mitochondrial disorders.
3. It provides brief descriptions of some specific genetic disorders like Gaucher Disease caused by enzyme deficiency, Huntington's Disease causing uncontrolled movements, and Hemophilia being a bleeding disorder linked to the X chromosome.
Genetic disorder
This document discusses genetic disorders, which are illnesses caused by abnormalities in a person's genome. It begins by introducing genetic disorders and explaining that most are rare. It then discusses the history of genetics research. The document classifies genetic disorders into three main types: single gene disorders, chromosomal disorders, and multifactorial disorders. For each type, it provides examples and descriptions of specific disorders like cystic fibrosis, Down syndrome, and Alzheimer's disease. It concludes by listing references used.
mutation ppt
This document summarizes DNA mutation and repair mechanisms. It discusses Lamarck and Darwin's theories of heredity and adaptation. It describes different types of mutations like substitutions, deletions, and frameshifts. Experiments by Luria and Delbrück tested Lamarck and Darwin's theories. The document also discusses DNA repair mechanisms in cells, spontaneous mutations from replication errors, and induced mutations from radiation, chemicals and intercalating agents. The Ames test is described to detect mutagens and carcinogens. Site-specific mutagenesis techniques like PCR can introduce mutations into genes.
The human genome project
The Human Genome Project was a 15-year scientific effort that mapped the entire human genome. It was primarily funded by governments in the US, UK, Japan, and other countries and cost $3 billion total. The project successfully identified the locations of all genes within human DNA and provided insights that enable genetically modifying crops, locating cancer cells, and diagnosing genetic diseases prenatally. Key techniques included genetic mapping to locate gene pairs on chromosomes and linkage analysis to determine the distance between disease-causing genes. The project's outcomes include further enabling gene therapy and precisely locating genes responsible for diseases.
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Mutations are heritable changes in genetic material that can occur spontaneously or be induced. There are several types of mutations including point mutations, frameshift mutations, and translocations. Point mutations include transitions, transversions, insertions, and deletions. Frameshift mutations disrupt the reading frame and can result in abnormal or nonfunctional proteins. Mutations can be harmful, beneficial, or neutral depending on their effects. They provide the raw material for evolution by creating genetic variation.
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The document discusses the human genome project, which aimed to sequence the entire human genome and identify all human genes. It provides background on the human genome, describing its size, number of genes, and chromosomes. It details the goals and milestones of the human genome project from 1986 to 2003. Vectors like yeast artificial chromosomes and bacterial artificial chromosomes were used to clone large fragments of DNA for sequencing.
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Genetic disorders
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The document discusses different types of mutations that can occur in DNA, including changes in the nucleotide sequence that may happen in somatic or germ cells. Mutations can be caused spontaneously during DNA replication or DNA damage from environmental factors like UV radiation. Types of mutations include point mutations, deletions, inversions, translocations, duplications, and aneuploidy. These genetic changes can have varying effects from being neutral to causing genetic disorders or cancer.
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- Mutations can be caused by natural DNA replication errors or external mutagens like radiation, chemicals, and viruses. These mutagens can directly damage DNA or produce reactive molecules that cause mutations.
- Several genetic disorders in humans are caused by chromosomal mutations, such as Down syndrome from trisomy 21, Edward's syndrome from trisomy 18, and Patau syndrome from trisomy 13. Other disorders involve the loss or gain of whole chromosomes or chromosome segments.
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This document outlines a lecture on infant nutrition. It discusses assessing newborn health, including birthweight and factors related to infant mortality. It covers infant development, including motor, cognitive, and digestive system development. It also addresses energy and nutrient needs of infants, including calories, protein, fat and other nutrients. The document discusses physical growth assessment of infants and common feeding practices in early infancy, including breastfeeding and formula. It concludes by covering the development of infant feeding skills.
Human Genome Project
The document summarizes key aspects of the human genome and genome projects. It discusses that a genome contains an organism's complete DNA including all genes. It describes the physical structure of human DNA including nuclear DNA, mitochondrial DNA, and RNA. It provides details on the goals and completion of the Human Genome Project in 2003, two years ahead of schedule. The project aimed to identify all human genes and map the 3 billion base pairs of human DNA.
Mutation
This document discusses different types of mutations that can occur in DNA, including base changes, insertions/deletions of bases, frameshift mutations, and trinucleotide repeats. It provides examples of different mutations that cause conditions like sickle cell anemia, beta-thalassemia, Huntington's disease, and retinoblastoma. The key points are that mutations can be random changes to DNA that provide raw material for evolution, and they can occur in germ cells or somatic cells with different effects on inheritance and disease.
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This document discusses several genetic disorders and diseases:
1. It lists 13 genetic diseases including Gaucher Disease, Huntington's Disease, Hemophilia, Parkinson's Disease, and Cystic Fibrosis.
2. It categorizes genetic disorders into four types - single gene disorders, multifactorial disorders caused by multiple genes and environment, chromosomal abnormalities, and mitochondrial disorders.
3. It provides brief descriptions of some specific genetic disorders like Gaucher Disease caused by enzyme deficiency, Huntington's Disease causing uncontrolled movements, and Hemophilia being a bleeding disorder linked to the X chromosome.
Genetic disorder
This document discusses genetic disorders, which are illnesses caused by abnormalities in a person's genome. It begins by introducing genetic disorders and explaining that most are rare. It then discusses the history of genetics research. The document classifies genetic disorders into three main types: single gene disorders, chromosomal disorders, and multifactorial disorders. For each type, it provides examples and descriptions of specific disorders like cystic fibrosis, Down syndrome, and Alzheimer's disease. It concludes by listing references used.
mutation ppt
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The human genome project
The Human Genome Project was a 15-year scientific effort that mapped the entire human genome. It was primarily funded by governments in the US, UK, Japan, and other countries and cost $3 billion total. The project successfully identified the locations of all genes within human DNA and provided insights that enable genetically modifying crops, locating cancer cells, and diagnosing genetic diseases prenatally. Key techniques included genetic mapping to locate gene pairs on chromosomes and linkage analysis to determine the distance between disease-causing genes. The project's outcomes include further enabling gene therapy and precisely locating genes responsible for diseases.
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Gene mutations can occur when there is a change in the DNA code, such as a substitution, insertion, or deletion of nucleotide bases. Substitution mutations, where one base is swapped for another, typically have the smallest effect since only one amino acid may change. Insertion and deletion mutations, which add or remove bases, can have larger effects by disrupting the reading frame of the entire DNA sequence. An example is sickle cell anemia, a substitution mutation that causes red blood cells to take on a sickle shape.
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Genetic disorders
Gregor Mendel's work in the 1860s laid the foundation for understanding inheritance of traits. Later, Francis Crick, James Watson, and Rosalind Franklin discovered the structure of DNA. This led to understanding how genetic information is passed from parents to offspring. Many genetic disorders result from mutations in DNA, including Down syndrome from an extra chromosome 21, Klinefelter syndrome from an extra X chromosome, and Turner syndrome from a missing X chromosome. Other genetic disorders discussed include sickle cell anemia, cystic fibrosis, Tay-Sachs disease, muscular dystrophy, hemophilia, Huntington's disease, phenylketonuria (PKU), and ALS. Both dominant and recessive inheritance patterns were
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The document discusses nutrition and healthy eating, covering the five main food groups, importance of daily exercise, recommended daily servings of fruits and vegetables, benefits of dairy products, and how proper nutrition provides energy, reduces stress and prevents health issues. Key recommendations include eating a variety of fruits and vegetables each day, drinking milk or eating yogurt, getting 60 minutes of moderate exercise daily, and choosing whole grains over refined grains.
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The document discusses nutrition, diet, and healthy eating. It defines nutrition and diet, and explains why eating healthy is important. It outlines the major food groups from the food pyramid, including grains, fruits and vegetables, dairy, meat, and drinks. It provides examples of common foods from each group and recommendations for daily servings. The document emphasizes eating a variety of foods, drinking water, and limiting high fat, sugar, and caffeine intake to support a healthy lifestyle.
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The document discusses different types of mutations that can occur in DNA, including changes in the nucleotide sequence that may happen in somatic or germ cells. Mutations can be caused spontaneously during DNA replication or DNA damage from environmental factors like UV radiation. Types of mutations include point mutations, deletions, inversions, translocations, duplications, and aneuploidy. These genetic changes can have varying effects from being neutral to causing genetic disorders or cancer.
Mutation, Types and Causes, Chromosomal Variation in Number, Gene Mutation
- Mutation is a change in the nucleotide sequence of an organism's genome. There are several types of mutations including substitution, insertion, deletion, and frameshift.
- Mutations can be caused by natural DNA replication errors or external mutagens like radiation, chemicals, and viruses. These mutagens can directly damage DNA or produce reactive molecules that cause mutations.
- Several genetic disorders in humans are caused by chromosomal mutations, such as Down syndrome from trisomy 21, Edward's syndrome from trisomy 18, and Patau syndrome from trisomy 13. Other disorders involve the loss or gain of whole chromosomes or chromosome segments.
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Human genome project
The document discusses the principal aims and applications of the Human Genome Project. The three main aims are to improve human genetics research infrastructure, establish DNA sequence as the interface between human and model organism biology, and improve DNA analytical biochemistry. Key applications discussed are molecular medicine like disease diagnosis, and using microbial genomes to research waste control and environmental cleanup. It provides examples of how genome sequencing is advancing fields like medicine, biotechnology, and environmental applications.
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Denis is a dynamic and results-driven Chief Information Officer (CIO) with a distinguished career spanning information systems analysis and technical project management. With a proven track record of spearheading the design and delivery of cutting-edge Information Management solutions, he has consistently elevated business operations, streamlined reporting functions, and maximized process efficiency.
Certified as an ISO/IEC 27001: Information Security Management Systems (ISMS) Lead Implementer, Data Protection Officer, and Cyber Risks Analyst, Denis brings a heightened focus on data security, privacy, and cyber resilience to every endeavor.
His expertise extends across a diverse spectrum of reporting, database, and web development applications, underpinned by an exceptional grasp of data storage and virtualization technologies. His proficiency in application testing, database administration, and data cleansing ensures seamless execution of complex projects.
What sets Denis apart is his comprehensive understanding of Business and Systems Analysis technologies, honed through involvement in all phases of the Software Development Lifecycle (SDLC). From meticulous requirements gathering to precise analysis, innovative design, rigorous development, thorough testing, and successful implementation, he has consistently delivered exceptional results.
Throughout his career, he has taken on multifaceted roles, from leading technical project management teams to owning solutions that drive operational excellence. His conscientious and proactive approach is unwavering, whether he is working independently or collaboratively within a team. His ability to connect with colleagues on a personal level underscores his commitment to fostering a harmonious and productive workplace environment.
Date: May 29, 2024
Tags: Information Security, ISO/IEC 27001, ISO/IEC 42001, Artificial Intelligence, GDPR
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