2. ADHESIVE SEALERS
e.g: adhesive bonding systems-resin luting
cements-GI luting cement
Provide chemical bond
Varnishes reduce but don’t eliminate microleakage
around amalgam
High copper amalgam
Comparable in short term(24hrs to 14days)
Leakage increase: margin on dentin/cementum-
1month to year (long term=caries)
Drawbacks: pooling of resin(x-ray artifact/perio
prob)-incorporation to amalgam/loss of strength-
more tech sensitive than varnish
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3. PHYSIOLOGIC CONSIDERATIONS
1-RDT:
No material provides better protection
Role :Buffering-insulation
Single most important factor inprotecting pulp
Conservation better than replacement
2-Causes of pulpal inflammation:
Bacteria or toxins (dental materials : mild &transitory)
Early enamel caries ¼ DEJ causes slight reaction
(enamel permeability)
Outward flow doesn’t prevent bacteria
Acid etch: tolerable if bacterial invasion in prevented
Rotary instruments : friction/dessication
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0.5mm=75%
1mm=90%
2mm
0.25-0.3
4. INSTRUMENTATION
Least effect: high speed-light force(1-3oz)-new bur-
air coolant-water spray-least prep.
Frictional heat: burn lesions-abscess
Dessication: tolerable in limited areas- loss of
dentinal fluid
Temperature rise: enamel vs dentine?/ pressure vs
speed (low speeds)/ diamond vs carbide bur/ depth
of prep.> full coverage:3-22%
New methods: laser(co2-Er:YAG-Nd:YAG-FEL)/ air
abrasion (water coolant necessary)
Electrosurgery: intact enamel/ 0.4sec
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5. CAUSES OF PULPAL PAIN
inflammation>pressure on nerve ends
No inflammation> changes in outward flow
speed>deformation of nerve ends
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6. CAUSES OF THERMAL SENSITIVITY
Theory of thermal shock (diffusivity) >base fore
metallic restorations-thickness :0.5-0.75mm-E.M)
Theory of pulpal hydrodynamics (gap-volume and
flow of outward fluid due to
density/diameter/permeability) > effective sealing-
integrity of interface
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6months max even with no base
50% 24hrs/78% mild
almost always disappears within 30 days
7. CAVITY SEALERS,LINERS AND BASES
Sealer
• prevention of leakage at interface- coating all walls :
a-varnish (natural/synthetic) b-adhesive sealer
(seal&bond)
Liner
• minimal thickness- only near pulp wall- physical
barrier and/or therapeutic effect(fluoride-antibacter)
Base
• replace missing dentin or block out undercuts
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8. CAVITY SEALERS
Kidd : microleakage= passage of bacteria-fluids-
molecules-ions along the interface
Leads to : secondary caries-marginal discoloration-
pulpal pathosis
Causes of clinical failures of restorations : sec.
caries- marginal gap/fracture- discoloration
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9. VARNISHES
Natural gum (copal) , a rosin or synthetic resin
dissolved in acetone,chloroform,ether
A thin protective film of 2-5μ
No thermal insulation
2applications
Reducing dentin permeability by 69%
Reducing microleakage 4-6months
Under ZPC crown cementation
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10. LINERS
Ca(OH)2
Not all formulations have stimulatory effect on
pulpoblasts
Reparative dentin assisted rather than stimulated
Antibacterial/anti inflammatory action- release G.F
from dentin>healing
Conventional: poor physical properties-high
solubility-lower E.M
Light activated : better properties
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11. LINERS
Glass ionomer
o Chemical bond-fluoride release
o Decreases interfacial bacterial penetration (fluoride-
low pH- metal cation)
o Acidic for 24hrs
o Conventional : reduced gap-higher E.M-lower
resistance to acid etch
o Bonded base technique (open sandwich provides
better seal due to increased strain capacity
because of delayed set)
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12. BASES
ZOE-ZPC: excellent thermal insulation-physical
properties but not adhesive
GI (conventional)
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13. GUIDELINES FOR BASING-LINING-SEALING
Don’t remove sound tooth structure to provide
space for base
Bases for buildup materials but for if for amalgam or
composite restorations: minimal extent
Min. thickness of liner
Adhesive sealer under amalgam?
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14. PULP CAPPING
Endodontic treatment designed to maintain the vitality of
the endodontium
1-vital pulp/no spontaneous pain 2-no lingering pain after
hot/cold stimulus 3-no PA lesion 4-bacteria excluded
Indirect P.C prefered
Monitoring for several months
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15. DIRECT PULP CAP
Ideal condition= bacteria free
Aged pulps : increased fibrosis and decreased
blood supply
Type/extent of exposure-bleeding amount
Only when: small mechanical exposure of healthy
pulp-rubber dam isolation and adequate
hemostasis
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16. INDIRECT PULP CAP
X-ray deep caries-no spontaneous pain-normal
vitality test
Spoon excavator or large round bur,low speed
handpiece
Wet(soft,amorphus) dentin removed- dry, fibrous
demineralized dentin leaved(moderate resistance to
gentle scraping)
Caries-disclosing dyes: caution for deep dentin
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Delay indirect restorations 4-6months
Do not remove IPC after removing
temporary restoration
17. DENTIN BONDING VS CA(OH)2 AS DPC
After smear layer removal pulpal tissue and adhesive
resin are compatible for 90days
Faster dentin bridge with Ca(OH)2
Toxic components rapid release
In bacteria free environment>successful capping with
bonding
Imperfect seal (nanoleakage in hybrid layer-collagen
hydrolysis)
Lower bond strength to carious dentin
Less intertubular dentin in deep lesions
Bond degredation
Acid etch: increase dentinal fluid flow-foreign body
reaction- increased bleeding
Resin components inhibit T lymphocytes
QTH temperature(20sec=25.2˚c)/11.2 irreversible damage
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18. CALCIUM HYDROXIDE SHORTCOMINGS
Break down in acid etching
Dissolution under leaking restoration
Interfacial failure in amalgam condensation
Tunnel defect in reparative dentin
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19. FUTURE OF DIRECT PULP CAPPING MATERIALS
Hydroxyapatite as scaffold for dentin formation
BMP-BSP
MTA(tricalcium silicate/aluminate/oxide-silicate
oxide) : high pH- compressive strength comparable
to reinforced ZOE-radiopaque-antibacterial-
biocompatible
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20. ANTIBACTERIAL EFFICACY OF RESTORATIVE
MATERIALS
Amalgam: copper-mercury-zinc-silver-chloride
components/effective against: S mutans, A viscous,
lactobacillus spp
Marginal seal improves with time(acidic
environment/ low oxygen concentration/corrosion
products)
GI
Resin composites (some resin
components/Glutaraldehyde)
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