Lecture by-
Dr . K . Sowmya Reddy
Assst.Prof
Department of OBG
OMC
Maternal injuries following
childbirth process are quite
common.
VULVA
Lacerations
Paraurethral tear
PERINEUM
 Incidence - 1 % .
CAUSES:
i. Over stretching.
ii. Rapid stretching.
PREVENTION:
1) Proper conduct in the second stage.
2) Taking due care of perineum.
RISK FACTORS FOR THIRD DEGREE
PERINEL TEAR:
 Big baby(weight >= 3kg)
 Nulliparity
 Outlet contraction with
narrow pubic arc
 Shoulder dystocia
 Forceps delivery
 Scar in the perineum
(perineorrhaphy,
episiotomy)
 Face to pubis delivery
 Midline episiotomy
 Precipitate labor
MANAGEMENT
Recent tear : Repaired immediately following the
delivery of the placenta.
Delay beyond 24 hours: Antibiotics.
 The complete tear should be repaired after 3 months if
delayed beyond 24hours.
REPAIR OF COMPLETE PERINEAL
TEAR
Step 1:
 Lithotomy position –repair done with local infiltration
of lignocaine hydrochloride
Step 2:
 Rectal and anal mucosa
 The rectal muscles including the pararectal facia
 The torn ends of sphincter ani externus(EAS)
 Overlapping or end to end approximation method for
repair
Step3:
 Repair of perineal muscle done by interrupted sutures
Step 4:
 The Vaginal wall and the perineal skin are apposed by
interrupted sutures
Aftercare:
 Similar to that following episiotomy.
 Special care following repair of complete tear.
 A low residual diet
 Lactulose 8 ml
 Bread-spectrum antibiotics
 Metronidazole 400 mg thrice daily ,continued for 5-7
days
 Physiotherapy and pelvic floor exercises
 Reviewed again 6-12 weeks postpartum
Plan for Future Delivery:
 Need to have institutional delivery.
 Women having symptoms or with abnormal endoanal
USG and/or manometry should be delivered by
elective cesarean birth.
VAGINA
 Isolated vaginal tears
 Lacerations without involvement of the perineum or
cervix
 Following instrumental or manipulative delivery
 Associated with brisk hemorrhage
Treatment:
 Exploration under general anesthesia with good light.
 Tears are repaired by interrupted or continuous sutures.
 In addition to sutures, hemostasis may be achieved by
intravaginal plugging by roller gauze.
 Plug should be removed after 24 hours.
 Arterial embolization
Colporrhexis:
 Rupture of the vault of the vagina is called colporrhexis
 Primary
 Secondary
 Complete
 Treatment:
 Limited to the vault close to the cervix , repair is done
from below.
 Tear extends high up , laparotomy is to be done
simultaneously with resuscitative measures.
CERVIX
 Commonest cause of traumatic postpartum haemorrhage
 Causes:
 Iatrogenic: Attempted forceps or Breech extraction
 Rigid cervix: congenital or scar from previous operations
amputation, conization lesion like carcinoma cervix
 Strong uterine contractions
 Detachment:
 ANNULAR : Detachment of the cervix may be annular which involved
the entire circumference of the cervix.
 Following prolonged labor in primary cervical dystocia.
 ANTERIOR LIP : May involve only anterior lip when it is nipped
between the head and symphysispubis.
DIAGNOSIS:
 Excessive vaginal bleeding immediately following delivery
in presence of a hard and contracted uterus , raises the
suspicion of a traumatic bleeding
DANGERS:
 Early:
 Deep cervical tears- Severe postpartum haemorrhage
 Broad ligament hematoma
 Pelvic cellulitis
 Thrombophlebitis
 Late:
 Ectropion
 Cervical incompetence
TREATMENT:
 Repaired soon after delivery
 Repair should be done under general anesthesia , in
lithotomy position with a good light.
 The prerequisites are :
 Speculum
 Retractors
 Two sponge holding forceps
 Assistant.
PELVIC HEMATOMA
 DEFINITION: Collection of blood anywhere in the
area between the pelvic peritoneum and the peineal
skin is called pelvic hematoma.
 ANATOMICAL TYPES:
 Infralevator hematoma
 Supralevator hematoma
 Commonest one is vulval hematoma
PELVIC HEMATOMA
Infralevator Hematoma:
ETIOLOGY:
 Improper hemostasis
 Failure to obliterate the dead space
 Rupture of para vaginal venous plexus
Symptoms:
 Persistent , severe pain on the perineal region
 There may be rectal tenesmus or bearing down efforts
when extension occurs to the ischiorectal fossa
Signs:
 Variable degrees of shock
 Local examination : tense swelling at the vulva
Treatment:
 Small hematoma conservatively with cold compress
 Larger hematomas explored in theater under general
anesthesia
 Blood clots are to be scooped out and the bleeding
points are to be secured
 The dead space is to be obliterated
 A foley catheter is inserted
Supralevator hematoma
Causes:
 Extension of cervical laceration
 Lower uterine segment rupture
 Spontaneous rupture of paravaginal venous plexus
Diagnosis:
 Unexplained shock with features of internal haemorrhage
following delivery raises the suspicion
 Swelling above the inguinal ligament pushing uterus to the
contralateralside
 Vaginal examination:
 Occlusion of the vaginal canal by a bulge (or)
 Boggy swelling felt through the fornix
 Rectal examination:
 Boggy mass
 Ultrasonography
Management:
 Treatment of shock and Laparotomy
 Anterior leaf of the broad ligament peritoneum is
incised and the blood clot is scooped out.
 Random blind sutures should not be placed to prevent
ureteric damage
 Tie the anterior division of the internal iliac artery
RUPTURE OF THE UTERUS
Definition : Disruption in the continuity of the all uterine
layers(endometrium , myometrium and serosa) any time
beyond 28 weeks of pregnancy is called rupture of the uterus
 Perforation
 Rupture of a rudimentary pregnant horn
Incidence : 1 in 2000 to 1 in 200 deliveries
 Scar rupture following increased incidence of cesarean
section over the years.
ETIOLOGY
SPONTANEOUS:
During pregnancy : rare
 Causes:
1. Previous damage to the uterine walls, dilatation and
curettage or manual removal of placenta
2. Grand multiparae
3. Congenital malformation
4. Couvelaire uterus
 Usually complete
 Involves upper segment
 Later months of pregnancy
During Labor:
 Obstructive rupture: end result of an obstructed labor
 Nonobstructive rupture : grand multiparae are usually
in early labor
 Involves the fundal area and is complete
SCAR RUPTURE:
 The incidence of lower segment scar rupture is about
1-2%,while that following classical one is 5-10 times
higher.
 Uterine scar following hysterotomy
During Pregnancy:
 Classical cesarean or hystrotomy scar is likely to give
way during later months of pregnancy
During labour:
 The classical or hysterotomy scar or cornual resection
for ectopic pregnancy is more vulnerable to rupture
during labor
IATROGENIC or TRAUMATIC:
During pregnancy:
 Use of oxytocin
 Prostaglandins
 Forcible external version
 Fall or blow on the abdomen
During labor:
 Internal podalic version
 Destructive operation
 Manual removal of placenta
 Forceps or breech extraction in incompletely dilated
cervix
 Injudicious administration of oxytocin
PATHOLOGY
Types:
 Complete and Incomplete rupture depending whether the
peritoneal coat is involved or not
Incomplete Rupture :
 Rupture of lower segment scar
 Extension of a cervical tear into the lower segment
Complete Rupture :
 disruption of the scar in in upper segment
 spontaneous rupture of both obstructive and nonobstructive type
Sites:
 Spontaneous nonobstructive rupture: Involves the upper
segment and often involves the fundus
 Obstructive type : involves the anterior lower segment
transversely
 May involve the cervix and Vaginal wall(colporrhexis). The
bladder may be involved, at times.
 Rupture over the previous scar
 Rent over the lower segment scar may extend to one or
both the sides to involve the major branches of uterine
vessels.
 Traumatic rupture after destructive operations is similar to
that met in spontaneous obstructive variety.
Dehiscence and scar rupture :
Scar dehiscence:
 Disruption of part of scar
 Fetal membranes intact
 Bleeding minimal
Scar rupture :
 Disruption of the entire length
 Complete separation of all layers
 Rupture of the membranes
 Bleeding
 The uterine cavity and peritoneal cavity become
continuous.
FETUS AND PLACENTA :
 In incomplete rupture
 In complete rupture
PROGNOSIS :
 Depends on
 Manner in which labour is managed ,
 Type of rupture,
 Morbid pathological changes ,
 Effective management.
 20% or more maternal death rate
The major causes of death:
 Haemorrhage,
 Shock and sepsis.
Late sequelae:
 Intestinal obstruction
 Scar rupture in subsequent pregnancies
DIAGNOSIS OF RUPTURE UTERUS
 That one should be conscious of the entity for an early
diagnosis
During pregnancy :
 Scar Rupture
 Spontaneous
 Iatrogenic
Scar Rupture:
Classical or hysterotomy:
 Dull abdominal pain , slight vaginal bleeding
 Tenderness on uterine palpation.
 FHS may be irregular or absent (silent phase).
 There is a sense of something giving way accompanied
by acute abdominal pain and collapse.
Spontaneous rupture in uninjured uterus :
 High parous women
 Acute but insidious
 In acute types diagnosis is established by the presence of features
of shock, acute tenderness on abdominal examination, palpation
of superficial fetal parts, absence of fetal heart rate
 Confused with concealed accidental haemorrhage or rectus
sheath hematoma
Rupture following fall, blow or extranal version or use of
oxytocics
 There is history of such anaccident followed by acute
pain abdomen and slight vaginal bleeding
 The confirmation is done by laparotomy
 Too often confused with accidental haemorrhage
During Labor:
 Scar Rupture
 Spontaneous Obstructive
 Spontaneous NonObstructive
 Iatrogenic
During Labor:
Scar Rupture:
 Classical or hysterotomy scar rupture
 Lower segment scar rupture
 Insidious
 Confirmation is by laparotomy
 Features of scar rupture are not as dramatic
 “Silent Rupture”
Spontaneaous obstructive rupture :
 Distinct premonitory phase prior to rupture
Premonitory phase
 Patient is usually a multipara who is in labour with features of
obstruction
 On examination dehydrated exhausted pulse rate and
temperature rise
 Abdominal examination reveals tender lower segment bandl’s
ring may be visible evidence of fetal distress FHS may be
absent
 On vaginal examination, the presenting part is found jammed
in the pelvis and vagina becomes dry and edematous
Phase of rupture
 Sense of something giving way at the height of uterine
contraction
 The constant pain changes to dull aching pain with
cessation of uterine contractions
General examination :
 Features of exhaustion and shock
 Abdominal examination reveals
 Superficial fetal parts
 Absence of FHS
 Absence uterine contour
 Two separate swellings one contracted fetal ovoid
 Vaginal examination reveals
 Recession of the presenting part
 Varying degrees of bleeding.
Spontaneous nonobstructive rupture
 To high parous women
 The height of uterine contraction is suddenly seized
with an agonizing bursting pain followed by a relief,
with cessation of contractions
features of the catastrophe :
 Presence of shock
 Internal haemorrhage
 Tenderness the uterus
 Varying amount of vaginal bleeding.
Rupture following manipulative or instrumental delivery :
 Sudden deterioration of the general condition
 Exploration of uterus to feel the rent confirms the
diagnosis
 Shortening of the cord immediately following a
difficult vaginal delivery is pathognomonic of uterine
rupture
MANAGEMENT OF RUPTURE
UTERUS
PROPHYLAXIS:
At risk mothers should have mandatory hospital delivery these are
 Contracted pelvis
 Previous history of caesarean section
 Uncorrected transverse lie
 Grand multiparity
 Known case of hydrocephalus
 General anesthesia should not be used give undue force in
external version
 Undue delay in the progress of labour
 Judicious selection of cases for vaginal delivery (VBAC) ,
oxytocin infusion either for induction or augmantaion of
labour.
 Internal podalic version never be done in obstructed
labor
 Attempted forceps delivery or breech extraction through
incompletely dilated cervix be avoided
 Destructive vaginal operations should be performed by
skilled personnel
 Manual removal in morbid adherent placenta should be
done by senior person
TREATMENT:
 RESUSCITATION
 LAPAROTOMY
Hysterectomy :
 The surgery for rupture uterus
 Preferable to perform a quick subtotal hysterectomy
rather than total hysterectomy
Repair :
 Applicable to a scar rupture where the margines or
clean.
 Repair is done by excision of the margins
Repair and sterilization :
Broad ligament hematoma:
Open the anterior leaf
Scoop out the blood clot
Secure the bleeding points
VISCERAL INJURIES
BLADDER:
Causes:
 Traumatic:
 Instrumental vaginal delivery
 Destructive operations
 Forceps delivery
 Abdominal operation
 Sloughing fistula
Diagnosis:
 Traumatic:
 Urine dribbles
 Blood stained urine
 Margins are clear
 Sloughing fistula:
 Margins devitalized and necrosed
 Missing if a chunk of tissue
MANAGEMENT:
Traumatic fistula:
 Immediate local repair is preferable
 In unfavorable conditions catheter for 10-14 days
 Antiseptics
 Favorable condition spontaneous closure
 Done after 3 months
Sloughing fistula:
 Repair is to be done after 3 months
RECTUM
 Is rare in obstetrics
 Repair postponed for at least 3 months
URETHRA
 Traumatic resulting from instrumental delivery
 Principles in management –similar to those of bladder
injury.

Injuries to the birth canal

  • 1.
    Lecture by- Dr .K . Sowmya Reddy Assst.Prof Department of OBG OMC
  • 2.
    Maternal injuries following childbirthprocess are quite common.
  • 3.
  • 4.
    PERINEUM  Incidence -1 % . CAUSES: i. Over stretching. ii. Rapid stretching. PREVENTION: 1) Proper conduct in the second stage. 2) Taking due care of perineum.
  • 5.
    RISK FACTORS FORTHIRD DEGREE PERINEL TEAR:  Big baby(weight >= 3kg)  Nulliparity  Outlet contraction with narrow pubic arc  Shoulder dystocia  Forceps delivery  Scar in the perineum (perineorrhaphy, episiotomy)  Face to pubis delivery  Midline episiotomy  Precipitate labor
  • 6.
    MANAGEMENT Recent tear :Repaired immediately following the delivery of the placenta. Delay beyond 24 hours: Antibiotics.  The complete tear should be repaired after 3 months if delayed beyond 24hours.
  • 7.
    REPAIR OF COMPLETEPERINEAL TEAR Step 1:  Lithotomy position –repair done with local infiltration of lignocaine hydrochloride Step 2:  Rectal and anal mucosa  The rectal muscles including the pararectal facia  The torn ends of sphincter ani externus(EAS)  Overlapping or end to end approximation method for repair
  • 8.
    Step3:  Repair ofperineal muscle done by interrupted sutures Step 4:  The Vaginal wall and the perineal skin are apposed by interrupted sutures
  • 9.
    Aftercare:  Similar tothat following episiotomy.  Special care following repair of complete tear.  A low residual diet  Lactulose 8 ml  Bread-spectrum antibiotics  Metronidazole 400 mg thrice daily ,continued for 5-7 days  Physiotherapy and pelvic floor exercises  Reviewed again 6-12 weeks postpartum
  • 10.
    Plan for FutureDelivery:  Need to have institutional delivery.  Women having symptoms or with abnormal endoanal USG and/or manometry should be delivered by elective cesarean birth.
  • 11.
    VAGINA  Isolated vaginaltears  Lacerations without involvement of the perineum or cervix  Following instrumental or manipulative delivery  Associated with brisk hemorrhage
  • 12.
    Treatment:  Exploration undergeneral anesthesia with good light.  Tears are repaired by interrupted or continuous sutures.  In addition to sutures, hemostasis may be achieved by intravaginal plugging by roller gauze.  Plug should be removed after 24 hours.  Arterial embolization
  • 13.
    Colporrhexis:  Rupture ofthe vault of the vagina is called colporrhexis  Primary  Secondary  Complete  Treatment:  Limited to the vault close to the cervix , repair is done from below.  Tear extends high up , laparotomy is to be done simultaneously with resuscitative measures.
  • 14.
    CERVIX  Commonest causeof traumatic postpartum haemorrhage  Causes:  Iatrogenic: Attempted forceps or Breech extraction  Rigid cervix: congenital or scar from previous operations amputation, conization lesion like carcinoma cervix  Strong uterine contractions  Detachment:  ANNULAR : Detachment of the cervix may be annular which involved the entire circumference of the cervix.  Following prolonged labor in primary cervical dystocia.  ANTERIOR LIP : May involve only anterior lip when it is nipped between the head and symphysispubis.
  • 15.
    DIAGNOSIS:  Excessive vaginalbleeding immediately following delivery in presence of a hard and contracted uterus , raises the suspicion of a traumatic bleeding DANGERS:  Early:  Deep cervical tears- Severe postpartum haemorrhage  Broad ligament hematoma  Pelvic cellulitis  Thrombophlebitis  Late:  Ectropion  Cervical incompetence
  • 16.
    TREATMENT:  Repaired soonafter delivery  Repair should be done under general anesthesia , in lithotomy position with a good light.  The prerequisites are :  Speculum  Retractors  Two sponge holding forceps  Assistant.
  • 17.
    PELVIC HEMATOMA  DEFINITION:Collection of blood anywhere in the area between the pelvic peritoneum and the peineal skin is called pelvic hematoma.  ANATOMICAL TYPES:  Infralevator hematoma  Supralevator hematoma  Commonest one is vulval hematoma
  • 18.
    PELVIC HEMATOMA Infralevator Hematoma: ETIOLOGY: Improper hemostasis  Failure to obliterate the dead space  Rupture of para vaginal venous plexus Symptoms:  Persistent , severe pain on the perineal region  There may be rectal tenesmus or bearing down efforts when extension occurs to the ischiorectal fossa
  • 19.
    Signs:  Variable degreesof shock  Local examination : tense swelling at the vulva Treatment:  Small hematoma conservatively with cold compress  Larger hematomas explored in theater under general anesthesia  Blood clots are to be scooped out and the bleeding points are to be secured  The dead space is to be obliterated  A foley catheter is inserted
  • 20.
    Supralevator hematoma Causes:  Extensionof cervical laceration  Lower uterine segment rupture  Spontaneous rupture of paravaginal venous plexus
  • 21.
    Diagnosis:  Unexplained shockwith features of internal haemorrhage following delivery raises the suspicion  Swelling above the inguinal ligament pushing uterus to the contralateralside  Vaginal examination:  Occlusion of the vaginal canal by a bulge (or)  Boggy swelling felt through the fornix  Rectal examination:  Boggy mass  Ultrasonography
  • 22.
    Management:  Treatment ofshock and Laparotomy  Anterior leaf of the broad ligament peritoneum is incised and the blood clot is scooped out.  Random blind sutures should not be placed to prevent ureteric damage  Tie the anterior division of the internal iliac artery
  • 23.
    RUPTURE OF THEUTERUS Definition : Disruption in the continuity of the all uterine layers(endometrium , myometrium and serosa) any time beyond 28 weeks of pregnancy is called rupture of the uterus  Perforation  Rupture of a rudimentary pregnant horn Incidence : 1 in 2000 to 1 in 200 deliveries  Scar rupture following increased incidence of cesarean section over the years.
  • 24.
    ETIOLOGY SPONTANEOUS: During pregnancy :rare  Causes: 1. Previous damage to the uterine walls, dilatation and curettage or manual removal of placenta 2. Grand multiparae 3. Congenital malformation 4. Couvelaire uterus  Usually complete  Involves upper segment  Later months of pregnancy
  • 25.
    During Labor:  Obstructiverupture: end result of an obstructed labor  Nonobstructive rupture : grand multiparae are usually in early labor  Involves the fundal area and is complete
  • 26.
    SCAR RUPTURE:  Theincidence of lower segment scar rupture is about 1-2%,while that following classical one is 5-10 times higher.  Uterine scar following hysterotomy During Pregnancy:  Classical cesarean or hystrotomy scar is likely to give way during later months of pregnancy During labour:  The classical or hysterotomy scar or cornual resection for ectopic pregnancy is more vulnerable to rupture during labor
  • 27.
    IATROGENIC or TRAUMATIC: Duringpregnancy:  Use of oxytocin  Prostaglandins  Forcible external version  Fall or blow on the abdomen
  • 28.
    During labor:  Internalpodalic version  Destructive operation  Manual removal of placenta  Forceps or breech extraction in incompletely dilated cervix  Injudicious administration of oxytocin
  • 29.
    PATHOLOGY Types:  Complete andIncomplete rupture depending whether the peritoneal coat is involved or not Incomplete Rupture :  Rupture of lower segment scar  Extension of a cervical tear into the lower segment Complete Rupture :  disruption of the scar in in upper segment  spontaneous rupture of both obstructive and nonobstructive type
  • 30.
    Sites:  Spontaneous nonobstructiverupture: Involves the upper segment and often involves the fundus  Obstructive type : involves the anterior lower segment transversely  May involve the cervix and Vaginal wall(colporrhexis). The bladder may be involved, at times.  Rupture over the previous scar  Rent over the lower segment scar may extend to one or both the sides to involve the major branches of uterine vessels.  Traumatic rupture after destructive operations is similar to that met in spontaneous obstructive variety.
  • 31.
    Dehiscence and scarrupture : Scar dehiscence:  Disruption of part of scar  Fetal membranes intact  Bleeding minimal Scar rupture :  Disruption of the entire length  Complete separation of all layers  Rupture of the membranes  Bleeding  The uterine cavity and peritoneal cavity become continuous.
  • 32.
    FETUS AND PLACENTA:  In incomplete rupture  In complete rupture PROGNOSIS :  Depends on  Manner in which labour is managed ,  Type of rupture,  Morbid pathological changes ,  Effective management.  20% or more maternal death rate
  • 33.
    The major causesof death:  Haemorrhage,  Shock and sepsis. Late sequelae:  Intestinal obstruction  Scar rupture in subsequent pregnancies
  • 34.
    DIAGNOSIS OF RUPTUREUTERUS  That one should be conscious of the entity for an early diagnosis During pregnancy :  Scar Rupture  Spontaneous  Iatrogenic
  • 35.
    Scar Rupture: Classical orhysterotomy:  Dull abdominal pain , slight vaginal bleeding  Tenderness on uterine palpation.  FHS may be irregular or absent (silent phase).  There is a sense of something giving way accompanied by acute abdominal pain and collapse.
  • 36.
    Spontaneous rupture inuninjured uterus :  High parous women  Acute but insidious  In acute types diagnosis is established by the presence of features of shock, acute tenderness on abdominal examination, palpation of superficial fetal parts, absence of fetal heart rate  Confused with concealed accidental haemorrhage or rectus sheath hematoma
  • 37.
    Rupture following fall,blow or extranal version or use of oxytocics  There is history of such anaccident followed by acute pain abdomen and slight vaginal bleeding  The confirmation is done by laparotomy  Too often confused with accidental haemorrhage
  • 38.
    During Labor:  ScarRupture  Spontaneous Obstructive  Spontaneous NonObstructive  Iatrogenic
  • 39.
    During Labor: Scar Rupture: Classical or hysterotomy scar rupture  Lower segment scar rupture  Insidious  Confirmation is by laparotomy  Features of scar rupture are not as dramatic  “Silent Rupture”
  • 40.
    Spontaneaous obstructive rupture:  Distinct premonitory phase prior to rupture Premonitory phase  Patient is usually a multipara who is in labour with features of obstruction  On examination dehydrated exhausted pulse rate and temperature rise  Abdominal examination reveals tender lower segment bandl’s ring may be visible evidence of fetal distress FHS may be absent  On vaginal examination, the presenting part is found jammed in the pelvis and vagina becomes dry and edematous
  • 41.
    Phase of rupture Sense of something giving way at the height of uterine contraction  The constant pain changes to dull aching pain with cessation of uterine contractions General examination :  Features of exhaustion and shock
  • 42.
     Abdominal examinationreveals  Superficial fetal parts  Absence of FHS  Absence uterine contour  Two separate swellings one contracted fetal ovoid  Vaginal examination reveals  Recession of the presenting part  Varying degrees of bleeding.
  • 43.
    Spontaneous nonobstructive rupture To high parous women  The height of uterine contraction is suddenly seized with an agonizing bursting pain followed by a relief, with cessation of contractions features of the catastrophe :  Presence of shock  Internal haemorrhage  Tenderness the uterus  Varying amount of vaginal bleeding.
  • 44.
    Rupture following manipulativeor instrumental delivery :  Sudden deterioration of the general condition  Exploration of uterus to feel the rent confirms the diagnosis  Shortening of the cord immediately following a difficult vaginal delivery is pathognomonic of uterine rupture
  • 45.
    MANAGEMENT OF RUPTURE UTERUS PROPHYLAXIS: Atrisk mothers should have mandatory hospital delivery these are  Contracted pelvis  Previous history of caesarean section  Uncorrected transverse lie  Grand multiparity  Known case of hydrocephalus  General anesthesia should not be used give undue force in external version  Undue delay in the progress of labour
  • 46.
     Judicious selectionof cases for vaginal delivery (VBAC) , oxytocin infusion either for induction or augmantaion of labour.  Internal podalic version never be done in obstructed labor  Attempted forceps delivery or breech extraction through incompletely dilated cervix be avoided  Destructive vaginal operations should be performed by skilled personnel  Manual removal in morbid adherent placenta should be done by senior person
  • 47.
    TREATMENT:  RESUSCITATION  LAPAROTOMY Hysterectomy:  The surgery for rupture uterus  Preferable to perform a quick subtotal hysterectomy rather than total hysterectomy
  • 48.
    Repair :  Applicableto a scar rupture where the margines or clean.  Repair is done by excision of the margins Repair and sterilization :
  • 49.
    Broad ligament hematoma: Openthe anterior leaf Scoop out the blood clot Secure the bleeding points
  • 50.
    VISCERAL INJURIES BLADDER: Causes:  Traumatic: Instrumental vaginal delivery  Destructive operations  Forceps delivery  Abdominal operation  Sloughing fistula
  • 51.
    Diagnosis:  Traumatic:  Urinedribbles  Blood stained urine  Margins are clear  Sloughing fistula:  Margins devitalized and necrosed  Missing if a chunk of tissue
  • 52.
    MANAGEMENT: Traumatic fistula:  Immediatelocal repair is preferable  In unfavorable conditions catheter for 10-14 days  Antiseptics  Favorable condition spontaneous closure  Done after 3 months Sloughing fistula:  Repair is to be done after 3 months
  • 53.
    RECTUM  Is rarein obstetrics  Repair postponed for at least 3 months URETHRA  Traumatic resulting from instrumental delivery  Principles in management –similar to those of bladder injury.