Osteoporosis is a disease where bone density decreases and bones become more fragile and prone to fractures. It affects over 10 million Americans, especially post-menopausal women. To prevent and slow bone loss, one should get enough calcium and vitamin D through foods or supplements, do weight-bearing exercise, and protect themselves from falls. Bone density tests can detect osteoporosis early before fractures occur. Medications may be needed if bone loss has progressed to osteopenia or osteoporosis.
Global Medical Cures™ | Bone health & Osteoporosis
DISCLAIMER-
Global Medical Cures™ does not offer any medical advice, diagnosis, treatment or recommendations. Only your healthcare provider/physician can offer you information and recommendations for you to decide about your healthcare choices.
Global Medical Cures™ | Bone health & Osteoporosis
DISCLAIMER-
Global Medical Cures™ does not offer any medical advice, diagnosis, treatment or recommendations. Only your healthcare provider/physician can offer you information and recommendations for you to decide about your healthcare choices.
Slide set that accompanied a public lecture on Nutrients and Bone Health held in London September 2011. The lecture focused on the importance of diet and lifestyle in maintaining healthy bone and preventing osteoporosis. Aspects of diagnosis are reviewed and the use of bone markers to indicate bone turnover are discussed in relation to monitoring the response to treatment.
This presentation was Shown on a community gathering in Gulshan Club Dhaka on the eve of the World Osteoporosis Day, 2010.
Prof. Shahiduzzaman was the key note speaker.
New zeland Dairy Milk was the organiser of this Seminar.
Osteoporosis is a condition characterized by a decrease in the density of bone, decreasing its strength and resulting in fragile bones. Know the Risk Factors for Osteoporotic Fracture, Preventive Measures and exercise for osteoporosis. For more health Tips, Visit at http://gisurgery.info
Slide set that accompanied a public lecture on Nutrients and Bone Health held in London September 2011. The lecture focused on the importance of diet and lifestyle in maintaining healthy bone and preventing osteoporosis. Aspects of diagnosis are reviewed and the use of bone markers to indicate bone turnover are discussed in relation to monitoring the response to treatment.
This presentation was Shown on a community gathering in Gulshan Club Dhaka on the eve of the World Osteoporosis Day, 2010.
Prof. Shahiduzzaman was the key note speaker.
New zeland Dairy Milk was the organiser of this Seminar.
Osteoporosis is a condition characterized by a decrease in the density of bone, decreasing its strength and resulting in fragile bones. Know the Risk Factors for Osteoporotic Fracture, Preventive Measures and exercise for osteoporosis. For more health Tips, Visit at http://gisurgery.info
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Osteoporosis weakens bones, making them more susceptible to sudden and unexpected fractures.
The disease often progresses without any symptoms or pain, and is not found until bones fracture. You can take steps to prevent this disease, and treatments do exist.
The word ‘osteoporosis’ means ‘porous bone.’ It is a disease that weakens bones, and if you have it, you are at a greater risk for sudden and unexpected bone fractures.
Osteoporosis means that you have less bone mass and strength. The disease often develops without any symptoms or pain, and it is usually not discovered until the weakened bones cause painful fractures.
Most of these are fractures of the hip, wrist and spine.
Prevent and Treat Osteoporosis 9769352147Bhupesh Kode
Osteoporosis means "porous bones." Our bones are strongest at about age 30, then begin to lose density. More than 300 million Indians have osteoporosis, which is significant bone loss that increases the risk of fracture. About half of women 50 and older will have an osteoporosis-related fracture in their lifetime.
Before & After Press Release for a Los Angeles HospitalTia Dobi
I knew nothing about osteoporosis when suddenly the phone rang.
BRRRRRRRING.
“Hello, Tia? This is Jane. I own Jane Doe Communications Group. We’re a public relations and marketing council here in Los Angeles. I heard your writing is exciting. Can you re-write a press release for us today?”
“Sure. What should I keep in mind?”
“Work magic.”
“Okay.”
Global Medical Cures™ | Lactose Intolerance & Osteoporosis
DISCLAIMER-
Global Medical Cures™ does not offer any medical advice, diagnosis, treatment or recommendations. Only your healthcare provider/physician can offer you information and recommendations for you to decide about your healthcare choices.
Osteoporosis is a disease in which bones become fragile and can easily break. It has no symptoms in its early stages and is a public health threat to more than 44 million Americans. In this community lecture given live on our Berkeley Heights, NJ campus, Dr. Toscano-Zukor, explains how to identify your risk factors for osteoporosis as well as prevent and treat this disease.
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
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June 20, 2024, Prix Galien International and Jerusalem Ethics Forum in ROME. Detailed agenda including panels:
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RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
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Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
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Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
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7 Ways to Save Your Bones
By Edward C. Geehr, M.D., Lifescript Chief Medical Officer
Published September 02, 2009
If you’re over 30, your skeleton’s already rebelling, tossing out bone faster than you can
replace it. Doctors deliberate over when to begin osteoporosis treatments, but your best bet
is to get serious about diet and exercise. Bone up on how to hang onto your main frame with
these tips. Plus, test your supplement IQ...
Did your hump-backed Grandma fall and break a hip or has a friend in her 50s cracked a rib
moving furniture?
Blame osteoporosis, a loss of bone that leads to debilitating fractures.
The disease is a major health threat for aging females, affecting about 8 million of the 10
million osteoporosis sufferers in the U.S. One in three women over 50 get fractures resulting
from osteoporosis.
Another 34 million have osteopenia, a precursor to the disease.
What Bone Loss Looks Like
Bone is not just a solid hunk of calcium; it’s living, growing tissue with a soft core and a
hardened framework of calcium phosphate.
The inner core, or marrow, produces our blood cells. And bones (along with teeth) act as a
storage tank for more than 99% of the body’s calcium.
As a living organ, bone is constantly breaking down its older framework and replacing it.
Formation outpaces destruction until about age 30. After that, the process slowly reverses,
causing a net bone loss.
2. As the loss becomes severe, bones lose density, becoming more porous and fragile.
In fact, under a microscope, osteoporotic bone looks like a sponge. The weakened bone, like a
dry twig, becomes more vulnerable to fractures, even under normal stresses.
That’s one reason Grandma hunches over like she’s perennially searching for a dropped penny.
The hump on the back of the elderly – called the dowager’s hump because it occurs mostly in
women – results from small bone fractures on the front of the vertebrae, usually upper ones.
As the fractured edge of a vertebra compresses, the vertebrae above it shift forward, curving
the spine. The forward tilt results in a hump, or kyphosis (which means "bent over").
As more vertebrae crack or collapse, the hump becomes more pronounced and painful, limiting
activities as well.
A woman may have to crane her neck to look someone in the face and breathing becomes more
difficult because the new spinal position makes it harder for the lungs to expand.
Other common results of osteoporosis are fractures of the hip and wrists in a fall.
Hip fractures – 300,000 per year – are the most serious.
About 24% of hip-fracture patients and a third of elderly men with hip fractures die within a
year, often because they can’t regain mobility.
Women are particularly vulnerable: Their bone loss accelerates in the first few years after
menopause as estrogen, which stimulates bone growth, declines.
Small, thin-boned women are at greatest risk. Other factors include:
Caucasian or Asian ancestry
Family history of osteoporosis
Anorexia
Low calcium and vitamin D intake
Long-term use of steroids
Cigarette smoking
Alcoholism
Inactivity
When Bone Loss Becomes a Problem
As with many trends in medicine, the prevailing wisdom on bone loss is shifting. This has
affected recommendations on when bone-loss treatments should begin.
Normal bone mass is defined as the average bone mineral density of a white woman 20-29
years old.
Based on that, researchers developed a T-score: A zero score was baseline (ideal), anything
between zero and negative 1 was normal; negative 2.5 or lower indicated osteoporosis.
But scores between negative 1 and negative 2.5 fell into a grey area called osteopenia, which
involves low bone density and mass. It became a catch phrase for bone density scores falling
outside the "normal" range.
3. In 2004, the National Osteoporosis Foundation (NOF) and the American College of Obstetrics
and Gynecology (ACOG) advised women who scored negative 2.0 or lower (or negative 1.5 or
lower if they had certain risk factors such as family history, smoking, etc.) to get osteoporosis
treatment.
That shift increased the number of aged 65-plus women recommended for treatment from 6.5
million to 11 million.
For women 50 to 64 years old, the treatment group expanded from 1.6 million to 4 million.
Now women’s health experts question whether those diagnosed with osteopenia need medical
treatment.
Drugs are expensive and can have significant side effects, such as chest pain, severe joint,
bone or muscle pain or heartburn.
The new standard raised questions: How much would the extension of treatment reduce serious
fractures? At what point along the bone loss continuum – from osteopenia to osteoporosis –
should treatment begin? When do the benefits outweigh the cost and risks?
Your Bone-Saving Options
Fortunately, you don’t have to wait for doctors to weigh in to protect yourself. Here are seven
ways to strengthen your bones:
1. Eat for better bones. Bulk up on foods high in calcium and vitamin D. Calcium is the major
bone builder, but it needs vitamin D to do its job. Vitamin D helps the body absorb calcium
that would otherwise flush out in our urine.
Foods high in calcium include dairy products, tofu, sardines, salmon, turnips and leafy greens.
Foods high in vitamin D: salmon, tuna and other saltwater fish, fortified milk, egg yolks, liver
and fish oils.
2. Get some daily sun. Sunlight stimulates the production of vitamin D in our skin. So get
about 5-30 minutes of sun – without sunscreen – at least twice a week. (But don’t overdo it
because too much sun raises the risk of skin cancer.)
3. Exercise. Just like muscles, bones need exercise to stay healthy. Strength training with
weight-bearing exercises (such as walking, jogging and dancing) helps prevent or slow
progression of osteoporosis.
Strength-training increases the tug of muscles on the bones and weight-bearing exercise also
stresses bones, which keep them strong.
For 4 bone-building exercises, check out our slideshow.
4. Consider supplements. If you can't get enough calcium from food or sunshine, take a daily
supplement that includes 1,000 milligrams calcium and 400 I.U. (international units) of vitamin
D. But don't take it all at once: The body can only absorb 500 milligrams of calcium at a time.
5. Get a bone density test. A bone mineral density test – a DEXA scan, or dual-energy X-ray
absorptiometry – will show how your bone mass is holding up.
The NOF advises women older than 65, and those with risk factors (like thinness, family history,