‫محمد‬ ‫كامل‬ ‫وداد‬ ‫الدكتورة‬ ‫المساعد‬ ‫االستاذ‬
Refers to a group of ocular conditions
characterized by optic nerve damage.
A condition of elevated IOP .
The optic nerve damage is related to the IOP
caused by congestion of aqueous humor in the
eye.
Is the 2nd leading cause of blindness in adult
in the US.
Is more prevalent in people older than 40 yrs.
of age.
There is no cure for glaucoma, but this
disease can be control.
Family history
Thin cornea
Older age
D.M.
Cardiovascular disease
Eye trauma
Prolong use of corticosteroids
Myopia
There are 2 theories regarding how increase
IOP damage the optic nerve in glaucoma.
The direct mechanical theory suggested :
High IOP damages the retinal layer as it
passes through the optic nerve head.
The indirect ischemic theory suggested that
high IOP compresses the microcirculation in
the optic nerve head, resulting in cell injury
and death.
Open angle glaucoma
Angle-closure glaucoma
Congenital glaucoma and
Glaucoma associated with other condition,
such as developmental anomalies or
corticosteroid use .
“Silent thief of sight”
Most pts. Are unaware that they have the
disease until they have experienced visual
changes and vision loss.
Blurred vision or “halos” around lights,
difficulty focusing , loss of peripheral vision,
aching or discomfort around the eye and
headache.
Ocular and medical history must be detailed to
investigate the history of predisposing
factors.
Tonometery to measure the IOP.
Ophthalmoscope to inspect the optic nerve.
Gonioscopy to examine the filtration angle of
the anterior chamber
Perimetry to assess the visual fields.
The aim of all glaucoma treat.
Is prevention of optic nerve damage.
Pharmacological therapy (miotics).
laser trabeculoplasty)
)
Laser procedures
surgery or a combination of these approaches
Is a lens opacity or cloudiness.
Cataract affecting 40 yrs old of age.
Cataract is the leading cause of blindness in
the world.
Pathophysiology :
Cataract can develop in one or both eyes at any
age.
Three common type of cataract are define by their
location in the lens:
1. Nuclear
2. Cortical
3. Posterior subcapsular
Visual impairment depends on their :
Size
Density
Location in the lens
More than one type can be present in the eye.
A nuclear cataract :tends to have a substantial
genetic component that causes a central
opacity in the lens. It is associated with
myopia.
A cortical cataract :involves the anterior,
posterior, or equatorial cortex of the lens.
Vision is worse in very bright light.
Posterior subcapsular cataract :occur in front
of the posterior or equatorial cortex of the
lens.
Aging
Loss of lens transparency
Decreased oxygen uptake
Decrease in level of vitamin C, protein
Increase in sodium and calcium
Accumulation of a yellow- brown pigment due
to the break-down of lens protein
Associated ocular conditions
Retinitis
Myopia
Retinal detachment and surgery
Infection
Toxic Factors
Ionizing radiation
Aspirin use
Corticosteroids
Alkaline chemical eye burns, poisoning
Cigarette smoking
Calcium, copper , iron, gold, sliver, and
mercury
Nutritional factors:
Reduced level of antioxidants
Poor nutrition
Obesity
Physical factors
Dehydration
Blunt trauma
Electrical shock
Perforation of the lens with sharp object or
foreign body,
Ultraviolent radiation in sunlight and x-ray
Systemic Diseases and Syndrome
Diabetes
Down Syndrome
Renal disorder
Disorder related to lipid metabolism
Musculoskeletal disorder
Painless
Blurred vision
Diplopia
Reduce visual acuity
Astigmatism: refractive error due to an
irregularity in the curvature of the cornea.
The Snellen visual acuity test.
Ophthalmoscope
Slit lump examination.
Medications, eye drops, eye glasses
treatment cures cataract or prevents age-
related cataract.
In the early stage of cataract development,
glasses, contact lenses, may improve vision.
Intracapsular cataract Extraction:
Extracapsular cataract Extraction:
Phacomulisification:
Lens Replacement:
Providing preoperative care:
Withhold any anticoagulation(e.g. aspirin,
warfarin) to reduce the risk of hemorrhage.
Dilating drops are administer every 10 minutes
for 4 doses at least one hour before surgery.
Providing postoperative care:
The patient receive verbal and written
Instruction about how protect the
eye Administer medication
Recognizes the signs of complications and
obtain emergency care.
Instruct the patient to take a mild analgesia
agent, as needed.
Anti-inflammatory and corticosteroid eye
drops or ointment.
Teaching patient self care:
Eye patch for 24 hrs. after surgery.
followed by eye glasses worn during the
day,.
Sunglasses should be worn.
A clean , damp wash cloth may be used to
remove eye discharge.
Eye patch remove after the first follow up
appointment .
Vision is stabilized when the eye healed,
usually within 6-12 weeks.

Glaucoma.pptx

  • 1.
    ‫محمد‬ ‫كامل‬ ‫وداد‬‫الدكتورة‬ ‫المساعد‬ ‫االستاذ‬
  • 2.
    Refers to agroup of ocular conditions characterized by optic nerve damage. A condition of elevated IOP . The optic nerve damage is related to the IOP caused by congestion of aqueous humor in the eye.
  • 3.
    Is the 2ndleading cause of blindness in adult in the US. Is more prevalent in people older than 40 yrs. of age. There is no cure for glaucoma, but this disease can be control.
  • 4.
    Family history Thin cornea Olderage D.M. Cardiovascular disease Eye trauma Prolong use of corticosteroids Myopia
  • 5.
    There are 2theories regarding how increase IOP damage the optic nerve in glaucoma. The direct mechanical theory suggested : High IOP damages the retinal layer as it passes through the optic nerve head.
  • 6.
    The indirect ischemictheory suggested that high IOP compresses the microcirculation in the optic nerve head, resulting in cell injury and death.
  • 7.
    Open angle glaucoma Angle-closureglaucoma Congenital glaucoma and Glaucoma associated with other condition, such as developmental anomalies or corticosteroid use .
  • 8.
    “Silent thief ofsight” Most pts. Are unaware that they have the disease until they have experienced visual changes and vision loss. Blurred vision or “halos” around lights, difficulty focusing , loss of peripheral vision, aching or discomfort around the eye and headache.
  • 9.
    Ocular and medicalhistory must be detailed to investigate the history of predisposing factors. Tonometery to measure the IOP. Ophthalmoscope to inspect the optic nerve. Gonioscopy to examine the filtration angle of the anterior chamber Perimetry to assess the visual fields.
  • 10.
    The aim ofall glaucoma treat. Is prevention of optic nerve damage. Pharmacological therapy (miotics). laser trabeculoplasty) ) Laser procedures surgery or a combination of these approaches
  • 11.
    Is a lensopacity or cloudiness. Cataract affecting 40 yrs old of age. Cataract is the leading cause of blindness in the world.
  • 12.
    Pathophysiology : Cataract candevelop in one or both eyes at any age. Three common type of cataract are define by their location in the lens: 1. Nuclear 2. Cortical 3. Posterior subcapsular
  • 13.
    Visual impairment dependson their : Size Density Location in the lens More than one type can be present in the eye.
  • 14.
    A nuclear cataract:tends to have a substantial genetic component that causes a central opacity in the lens. It is associated with myopia. A cortical cataract :involves the anterior, posterior, or equatorial cortex of the lens. Vision is worse in very bright light. Posterior subcapsular cataract :occur in front of the posterior or equatorial cortex of the lens.
  • 15.
    Aging Loss of lenstransparency Decreased oxygen uptake Decrease in level of vitamin C, protein Increase in sodium and calcium Accumulation of a yellow- brown pigment due to the break-down of lens protein
  • 16.
  • 17.
    Toxic Factors Ionizing radiation Aspirinuse Corticosteroids Alkaline chemical eye burns, poisoning Cigarette smoking Calcium, copper , iron, gold, sliver, and mercury
  • 18.
    Nutritional factors: Reduced levelof antioxidants Poor nutrition Obesity
  • 19.
    Physical factors Dehydration Blunt trauma Electricalshock Perforation of the lens with sharp object or foreign body, Ultraviolent radiation in sunlight and x-ray
  • 20.
    Systemic Diseases andSyndrome Diabetes Down Syndrome Renal disorder Disorder related to lipid metabolism Musculoskeletal disorder
  • 21.
    Painless Blurred vision Diplopia Reduce visualacuity Astigmatism: refractive error due to an irregularity in the curvature of the cornea.
  • 22.
    The Snellen visualacuity test. Ophthalmoscope Slit lump examination.
  • 23.
    Medications, eye drops,eye glasses treatment cures cataract or prevents age- related cataract. In the early stage of cataract development, glasses, contact lenses, may improve vision.
  • 24.
    Intracapsular cataract Extraction: Extracapsularcataract Extraction: Phacomulisification: Lens Replacement:
  • 25.
    Providing preoperative care: Withholdany anticoagulation(e.g. aspirin, warfarin) to reduce the risk of hemorrhage. Dilating drops are administer every 10 minutes for 4 doses at least one hour before surgery.
  • 26.
    Providing postoperative care: Thepatient receive verbal and written Instruction about how protect the eye Administer medication Recognizes the signs of complications and obtain emergency care. Instruct the patient to take a mild analgesia agent, as needed. Anti-inflammatory and corticosteroid eye drops or ointment.
  • 27.
    Teaching patient selfcare: Eye patch for 24 hrs. after surgery. followed by eye glasses worn during the day,. Sunglasses should be worn. A clean , damp wash cloth may be used to remove eye discharge.
  • 28.
    Eye patch removeafter the first follow up appointment . Vision is stabilized when the eye healed, usually within 6-12 weeks.