Department of Surgery
North East Medical College
Presentation on Fluid
and
Electrolytes Management
Presented By:
Dr. Nusrat Farhin Ahmed
&
Dr. Katha Acharjee
Contents
● Total body water:
● Fluid Compartment
● Composition Of Fluid
Compartment
● Normal intake- output chart
● Disorder Of fluid Balance
● Assessment
● Monitoring Of fluid Balance
● Hypervolemia
Total body water:
Total water daily requirement:
Depends upon age,size,temperature and body surface-
1.Most accurate guide is surface area 1.5 L/m2
area daily.
2.Practical guide is 30-40 ml/kg of total body
weight Adult Male: 60%
Adult Female : 50%
Infant:80%
Fluid Compartment
Composition Of Fluid Compartment
●ECF :Na+ is the principal cation and
Chloride and HCo3 are principal
anions .
●ICF: K+ and Mg2+ are principal
cation and phosphate and
Sulphate are principal anion
Normal intake- output chart:
Fluids used in ward
Crystalloid Isotonic 0.9%Nacl(Normal saline)
5%Dextrose in Aqua
Electrolytes rich: Ringers lactate, Hartman solution,
Cholera saline
Hypotonic 0.45% Nacl
Hypertonic 5% DNS
Colloid Plasma albumin
Dextran
Haemaccel
Disorder Of fluid Balance
Hypovolemia ( volume depletion)
- ECF volume deficit is most common fluid loss in the surgical
patients,and aggravated by General Anesthesia.
Cause:
Non hemorrhagic:
Vomiting
Diarrhoe
a
Intestinal
obstruction Burn
Fistula drainage
Stomas
Diuresis
Soft-tissue injuries and
infections Inflammation
Hemorrhagic:
-Chest:
aortic disruption, pulmonary
parenchyma trauma, pulmonary vascular injury,
hemoptysis
-Abdomen/pelvis/peritoneum:
GI hemorrhage(varices/ulcers),solid organ
injuries, vascular( trauma, rupture)
-Fractures (pelvic, long
bones)
-Gynaecological causes
-External bleeding from tissue skin
Assessment
C/F-
Dry tongue
Patient is thirsty and
restless Rapid, low volume
pulse Cold clammy
extremities Sunken eye
Oliguria
History:
-Poor intake ,GI bleeding ,
excessive
vomiting ,symptoms of
underlying disease
.
Hypervolemia
Signs
•CNS: Confusion , seizure
• CVS: Elevated JVP, venous distension - pulmonary edema
• Respiratory: shortness of breath even in rest , basal
crepitation.
• Tissue: pitting edema - anasarca, ascites, weight gain.
Management of Hypervolemia
Electrolytes disorders:
1)Hyponatraemia: Na+ level below normal.
2)Hypernatraemia: ↑ Na+level above
normal.
3)Hypokalaemia: K + level below normal.
4)Hyperkalaemia: ↑ K⁺ level above normal.
5)Hypocalcaemia: Ca++ level below normal.
6)Hypercalcaemia: Ca++ level above normal
Sodium Balance
Distribution of Sodium
1. Total body sodium is 58 mmol/kg body weight
2. Distribution: ECF = 50%, ICF = 10%, Bone = 40%.
3. Exchangeable sodium is about 73%
C/F:
Mild form patient is asymptomatic.
Na<120 mmol/L :patient become confused. There are sign-symptoms of water excess
1. Puffy face, JVP raised, High bounding pulse.
2.S/S of cerebral oedema like headache, vomiting, muscle twitching, poor co-ordination, then weakness,
apathy and confusion and finally coma and convulsion.
3. S/S of pulmonary oedema like cough, frothy sputum, basal crepitation
4. Low plasma Na. If plasma sodium falls below 110 mmol/L :Convulsion and coma may occur.
Hyponatraemia
Measurement of Na deficit in mmol
Total deficit = Body weight * (140-plasma
Na) * TBW in percentage
Sodium deficit if body weight is 60 kg and S
sodium is 130 mmol.
Total deficit = 60 X (140-130) X 60 /100 =
360 mmol.
Treatment
1. Treatment of underlying cause.
2.Loss of Na can be replaced by sodium
intake or by giving I/V 0.9% saline.
3. Water retention should be treated by fluid
restriction.
1. Clinical features are like that of water depletion.
2. Clinically patient is thirsty and drowsy.
3. Urine scanty with high specific gravity.
4. BP below normal
5. Serum sodium increased.
6. If serum Na rises above 160 mmol/L hypernatremic encephalopathy
Hypernatre
mia
Clinical
features
Treatment
High serum sodium should be corrected by slow intravenous
injection of hypotonic saline or by giving 5% glucose solution.
Potassium Balance
Total body potassium is 3500 mmol/L or 50 mmol/kg body
weight.
ECF = 2%
ICF = 98%
Daily Requirement : 1 mmol/L
Control of Potassium balance by Aldosterone
 Clinical Features
1. Mainly asymptomatic and may present with cardiac arrhythmia.
2. In severe cases there is abdominal distension, listlessness, slurred speech, muscular
hypotonia, and depressed reflex.
3. Weakness of respiratory muscle may result in rapid, shallow and gasping respiration.
 ECG findings of hypokalamia are
● ST depression.
● T wave inversion.
● Prominent U waves.
Hypokalamia
Treatment of hypokalemia
Treatment of hypokalemia usually focuses on loss control, replacement,
and loss prevention.
The first step is to make sure that the primary problem causing the
hypokalemia is dealt with i.e changing a diuretic.
The second step is to replace the missing potassium. For mild hypokalemia,
oral
supplements are often enough to replace the missing potassium. Potassium
levels
of less than 2.5 mEq/L usually are treated with intravenous potassium. This
could be anywhere from two to six doses of the medication.
Finally, you need to take steps to help the patient prevent potassium losses
in the
future. This may mean nutritional or medication education to ensure that
the loss doesn't occur again
Hyperkalemia
Hyperkalemia, is an elevated level of potassium (K+) in the blood
serum. Normal potassium levels are between 3.5 and 5.0 mmol/L
(3.5 and 5.0 mEq/L) with levels above 5mmol/L defined as
hyperkalemia.
Clinical Features: Typically this results in no symptoms.
Occasionally when severe it results in palpitations, muscle pain, muscle
weakness, or numbness. An abnormal heart rate can occur which can
result in cardiac arrest and death. This is preceded by ECG Changes-a
peaked T wave, prolonged P-R interval and widens QRS complex and
small P wave.
More serious symptoms of hyperkalemia
include slow heart rate and weak pulse.
The severity is divided into mild (5.5-5.9
mmol/L), moderate (6.0-6.4 mmol/L),
and severe (>6.5 mmol/L).
Common causes include kidney failure,
hypoaldosteronism, and
rhabdomyolysis. A
number of medications can also cause
high
blood potassium including
spironolactone,
NSAIDS, and angiotensin
converting enzyme inhibitors.
Treatme
nt
1. Infuse intravenous hypertonic saline (50%) dextrose infusion together
with
insulin (1 unit of insulin per 2 gm of carbohydrate) to transport
potassium from extracellular to intracellular fluid.
2. Calcium gluconate, Sodium bicarbonate.
3. Ion exchange resin by oral or enema.
Thank you……

Fluid and electrolyte management in surgery.pptx

  • 1.
    Department of Surgery NorthEast Medical College Presentation on Fluid and Electrolytes Management Presented By: Dr. Nusrat Farhin Ahmed & Dr. Katha Acharjee
  • 2.
    Contents ● Total bodywater: ● Fluid Compartment ● Composition Of Fluid Compartment ● Normal intake- output chart ● Disorder Of fluid Balance ● Assessment ● Monitoring Of fluid Balance ● Hypervolemia
  • 3.
    Total body water: Totalwater daily requirement: Depends upon age,size,temperature and body surface- 1.Most accurate guide is surface area 1.5 L/m2 area daily. 2.Practical guide is 30-40 ml/kg of total body weight Adult Male: 60% Adult Female : 50% Infant:80%
  • 4.
  • 5.
    Composition Of FluidCompartment ●ECF :Na+ is the principal cation and Chloride and HCo3 are principal anions . ●ICF: K+ and Mg2+ are principal cation and phosphate and Sulphate are principal anion
  • 6.
  • 7.
    Fluids used inward Crystalloid Isotonic 0.9%Nacl(Normal saline) 5%Dextrose in Aqua Electrolytes rich: Ringers lactate, Hartman solution, Cholera saline Hypotonic 0.45% Nacl Hypertonic 5% DNS Colloid Plasma albumin Dextran Haemaccel
  • 8.
    Disorder Of fluidBalance Hypovolemia ( volume depletion) - ECF volume deficit is most common fluid loss in the surgical patients,and aggravated by General Anesthesia. Cause: Non hemorrhagic: Vomiting Diarrhoe a Intestinal obstruction Burn Fistula drainage Stomas Diuresis Soft-tissue injuries and infections Inflammation Hemorrhagic: -Chest: aortic disruption, pulmonary parenchyma trauma, pulmonary vascular injury, hemoptysis -Abdomen/pelvis/peritoneum: GI hemorrhage(varices/ulcers),solid organ injuries, vascular( trauma, rupture) -Fractures (pelvic, long bones) -Gynaecological causes -External bleeding from tissue skin
  • 9.
    Assessment C/F- Dry tongue Patient isthirsty and restless Rapid, low volume pulse Cold clammy extremities Sunken eye Oliguria History: -Poor intake ,GI bleeding , excessive vomiting ,symptoms of underlying disease .
  • 10.
    Hypervolemia Signs •CNS: Confusion ,seizure • CVS: Elevated JVP, venous distension - pulmonary edema • Respiratory: shortness of breath even in rest , basal crepitation. • Tissue: pitting edema - anasarca, ascites, weight gain.
  • 11.
  • 12.
    Electrolytes disorders: 1)Hyponatraemia: Na+level below normal. 2)Hypernatraemia: ↑ Na+level above normal. 3)Hypokalaemia: K + level below normal. 4)Hyperkalaemia: ↑ K⁺ level above normal. 5)Hypocalcaemia: Ca++ level below normal. 6)Hypercalcaemia: Ca++ level above normal
  • 13.
    Sodium Balance Distribution ofSodium 1. Total body sodium is 58 mmol/kg body weight 2. Distribution: ECF = 50%, ICF = 10%, Bone = 40%. 3. Exchangeable sodium is about 73%
  • 14.
    C/F: Mild form patientis asymptomatic. Na<120 mmol/L :patient become confused. There are sign-symptoms of water excess 1. Puffy face, JVP raised, High bounding pulse. 2.S/S of cerebral oedema like headache, vomiting, muscle twitching, poor co-ordination, then weakness, apathy and confusion and finally coma and convulsion. 3. S/S of pulmonary oedema like cough, frothy sputum, basal crepitation 4. Low plasma Na. If plasma sodium falls below 110 mmol/L :Convulsion and coma may occur. Hyponatraemia
  • 15.
    Measurement of Nadeficit in mmol Total deficit = Body weight * (140-plasma Na) * TBW in percentage Sodium deficit if body weight is 60 kg and S sodium is 130 mmol. Total deficit = 60 X (140-130) X 60 /100 = 360 mmol.
  • 16.
    Treatment 1. Treatment ofunderlying cause. 2.Loss of Na can be replaced by sodium intake or by giving I/V 0.9% saline. 3. Water retention should be treated by fluid restriction.
  • 17.
    1. Clinical featuresare like that of water depletion. 2. Clinically patient is thirsty and drowsy. 3. Urine scanty with high specific gravity. 4. BP below normal 5. Serum sodium increased. 6. If serum Na rises above 160 mmol/L hypernatremic encephalopathy Hypernatre mia Clinical features
  • 18.
    Treatment High serum sodiumshould be corrected by slow intravenous injection of hypotonic saline or by giving 5% glucose solution.
  • 19.
    Potassium Balance Total bodypotassium is 3500 mmol/L or 50 mmol/kg body weight. ECF = 2% ICF = 98% Daily Requirement : 1 mmol/L Control of Potassium balance by Aldosterone
  • 20.
     Clinical Features 1.Mainly asymptomatic and may present with cardiac arrhythmia. 2. In severe cases there is abdominal distension, listlessness, slurred speech, muscular hypotonia, and depressed reflex. 3. Weakness of respiratory muscle may result in rapid, shallow and gasping respiration.  ECG findings of hypokalamia are ● ST depression. ● T wave inversion. ● Prominent U waves. Hypokalamia
  • 21.
    Treatment of hypokalemia Treatmentof hypokalemia usually focuses on loss control, replacement, and loss prevention. The first step is to make sure that the primary problem causing the hypokalemia is dealt with i.e changing a diuretic. The second step is to replace the missing potassium. For mild hypokalemia, oral supplements are often enough to replace the missing potassium. Potassium levels of less than 2.5 mEq/L usually are treated with intravenous potassium. This could be anywhere from two to six doses of the medication. Finally, you need to take steps to help the patient prevent potassium losses in the future. This may mean nutritional or medication education to ensure that the loss doesn't occur again
  • 22.
    Hyperkalemia Hyperkalemia, is anelevated level of potassium (K+) in the blood serum. Normal potassium levels are between 3.5 and 5.0 mmol/L (3.5 and 5.0 mEq/L) with levels above 5mmol/L defined as hyperkalemia. Clinical Features: Typically this results in no symptoms. Occasionally when severe it results in palpitations, muscle pain, muscle weakness, or numbness. An abnormal heart rate can occur which can result in cardiac arrest and death. This is preceded by ECG Changes-a peaked T wave, prolonged P-R interval and widens QRS complex and small P wave.
  • 23.
    More serious symptomsof hyperkalemia include slow heart rate and weak pulse. The severity is divided into mild (5.5-5.9 mmol/L), moderate (6.0-6.4 mmol/L), and severe (>6.5 mmol/L). Common causes include kidney failure, hypoaldosteronism, and rhabdomyolysis. A number of medications can also cause high blood potassium including spironolactone, NSAIDS, and angiotensin converting enzyme inhibitors.
  • 24.
    Treatme nt 1. Infuse intravenoushypertonic saline (50%) dextrose infusion together with insulin (1 unit of insulin per 2 gm of carbohydrate) to transport potassium from extracellular to intracellular fluid. 2. Calcium gluconate, Sodium bicarbonate. 3. Ion exchange resin by oral or enema.
  • 25.