Cardiogenic Shock
Med-Surg - I
Professor:
Dr. Hector Barroso. RN, BSN, MS-FNP
Team
1. Luis Alvarez
2. Lilian Santana
3. Diana Soler
4. Favier Pastrana
5. Ian Guevara
Objectives
1. Concept of Cardiogenic Shock (CS).
2. Understand your Pathophysiology.
3. Know the statistics, incidence and its causes.
4. Recognize its clinical manifestations, the assessment and.
diagnostic finding.
5. Medical and surgical management.
6. Learn to elaborate an adequate nursing management.
Definition of CS
● Inadequate circulation due to primary failure of the heart to
function effectively (↓ ventricular contraction).
● Diminished cardiac output (CO).
● Systemic hypoperfusion and maladaptive cycles of ischemia,
inflammation, vasoconstriction, and volume overload.
Signs of tissue hypoperfusion:
● low urine production, cool extremities and altered mental of
consciousness.
Multi-organ failure and death.
Pathophysiology
Epidemiology (Incidence)
1. CS complicate 5 to 12 % of AMIs, > 40% loss of LV.
2. Most frequently:
a. > 75 year, Man (+ freq CAD), Woman (> 55 year).
b. Mechanical complications post AMI (free wall,
papillary muscle and IV septal rupture).
c. Patients with more associated comorbidities.
Epidemiology (Mortality)
3. CS: 80 to 90 % - historically
4. Post to the advent: ↓ 55 - 60 %
a. Thrombolytic, Graft coronary, PCI
b. Early diagnostic, medical treatment
and haemodynamic support (IABP)
a. MCS (mechanical circulatory support) = IMPELLA
(~50%)
Clinical manifestations
● Pale, cool and clammy skin.
● Systolic blood pressure (< 90 mmHg).
● Tachycardia (> 100 bpm).
● Dyspnea or shortness of breath.
● Oliguria (< 20 - 30 ml/h).
● Mental confusion.
● Cyanosis.
Assessment and Diagnostic Findings
Myocardial Damage:
● Laboratory Biomarkers for Ventricular Dysfunction
● Cardiac Enzyme levels and Biomarkers
● Serum Lactate
At BedSide
● Transthoracic Echocardiography
● Serial 12-lead Electrocardiograms
Assessment and Diagnostic Findings
● Auscultation
● Arterial Pressure Monitoring
● ABG Analysis
● Enzyme Levels
Surgical Management
Last option when drug therapy and medical
procedures do not work.
● Intra-Aortic Balloon Pump (IABP).
Medical Management
● Oxygen.
● Fluid therapy.
● Pain control.
● Balloon pump.
● Hemodynamic monitoring.
● Angioplasty and stenting.
Pharmacology Therapy
● Norepinephrine.
● IV dobutamine.
● IV dopamine.
● IV nitroprusside.
Nursing Management
Nursing Assessment
● Fluid overload
● Vital signs
Nursing Diagnosis
● Acute Pain
● Activity intolerance
● Excess fluid volume
● Decreased cardiac output
● impaired gas exchange
● ineffective tissue perfusion
Nursing Care Planning & Goals
● Maintain intra-aortic balloon counterpulsation.
● Monitor hemodynamic status.
● Administer intravenous fluids.
● Prevent recurrence of cardiogenic shock.
Nursing Interventions
● Fluids.
● Arterial blood gas.
● Prevent recurrence.
● Positioning.
● Enhance safety and comfort.
● Intra-aortic balloon counterpulsation.
● Hemodynamic status.
Evaluation
Expected outcomes include:
● Prevented recurrence of cardiogenic shock.
● Monitored hemodynamic status.
● Administered medications and intravenous fluids.
● Maintained intra-aortic balloon counterpulsation.
Discharge and Home Care
Lifestyle changes must be made to avoid the recurrence of
cardiogenic shock:
1. Control hypertension.
2. Avoid smoking.
3. Maintain a healthy weight.
4. Diet.
5. Exercise.
Documentation Guidelines
1. Baseline and subsequent findings and individual hemodynamic
parameters.
2. Prior medication use.
3. Plan of care.
4. Teaching plan.
5. Client’s responses to interventions, teaching, and actions
performed.
6. Attainment or progress toward desired outcomes.
7. Modifications to plan of care.
Discharge home
Thanks

Cardiogenic Shock.pptx

  • 1.
    Cardiogenic Shock Med-Surg -I Professor: Dr. Hector Barroso. RN, BSN, MS-FNP
  • 2.
    Team 1. Luis Alvarez 2.Lilian Santana 3. Diana Soler 4. Favier Pastrana 5. Ian Guevara
  • 3.
    Objectives 1. Concept ofCardiogenic Shock (CS). 2. Understand your Pathophysiology. 3. Know the statistics, incidence and its causes. 4. Recognize its clinical manifestations, the assessment and. diagnostic finding. 5. Medical and surgical management. 6. Learn to elaborate an adequate nursing management.
  • 4.
    Definition of CS ●Inadequate circulation due to primary failure of the heart to function effectively (↓ ventricular contraction). ● Diminished cardiac output (CO). ● Systemic hypoperfusion and maladaptive cycles of ischemia, inflammation, vasoconstriction, and volume overload. Signs of tissue hypoperfusion: ● low urine production, cool extremities and altered mental of consciousness. Multi-organ failure and death.
  • 5.
  • 6.
    Epidemiology (Incidence) 1. CScomplicate 5 to 12 % of AMIs, > 40% loss of LV. 2. Most frequently: a. > 75 year, Man (+ freq CAD), Woman (> 55 year). b. Mechanical complications post AMI (free wall, papillary muscle and IV septal rupture). c. Patients with more associated comorbidities.
  • 7.
    Epidemiology (Mortality) 3. CS:80 to 90 % - historically 4. Post to the advent: ↓ 55 - 60 % a. Thrombolytic, Graft coronary, PCI b. Early diagnostic, medical treatment and haemodynamic support (IABP) a. MCS (mechanical circulatory support) = IMPELLA (~50%)
  • 10.
    Clinical manifestations ● Pale,cool and clammy skin. ● Systolic blood pressure (< 90 mmHg). ● Tachycardia (> 100 bpm). ● Dyspnea or shortness of breath. ● Oliguria (< 20 - 30 ml/h). ● Mental confusion. ● Cyanosis.
  • 11.
    Assessment and DiagnosticFindings Myocardial Damage: ● Laboratory Biomarkers for Ventricular Dysfunction ● Cardiac Enzyme levels and Biomarkers ● Serum Lactate At BedSide ● Transthoracic Echocardiography ● Serial 12-lead Electrocardiograms
  • 12.
    Assessment and DiagnosticFindings ● Auscultation ● Arterial Pressure Monitoring ● ABG Analysis ● Enzyme Levels
  • 13.
    Surgical Management Last optionwhen drug therapy and medical procedures do not work. ● Intra-Aortic Balloon Pump (IABP).
  • 14.
    Medical Management ● Oxygen. ●Fluid therapy. ● Pain control. ● Balloon pump. ● Hemodynamic monitoring. ● Angioplasty and stenting.
  • 15.
    Pharmacology Therapy ● Norepinephrine. ●IV dobutamine. ● IV dopamine. ● IV nitroprusside.
  • 16.
    Nursing Management Nursing Assessment ●Fluid overload ● Vital signs
  • 17.
    Nursing Diagnosis ● AcutePain ● Activity intolerance ● Excess fluid volume ● Decreased cardiac output ● impaired gas exchange ● ineffective tissue perfusion
  • 18.
    Nursing Care Planning& Goals ● Maintain intra-aortic balloon counterpulsation. ● Monitor hemodynamic status. ● Administer intravenous fluids. ● Prevent recurrence of cardiogenic shock.
  • 19.
    Nursing Interventions ● Fluids. ●Arterial blood gas. ● Prevent recurrence. ● Positioning. ● Enhance safety and comfort. ● Intra-aortic balloon counterpulsation. ● Hemodynamic status.
  • 20.
    Evaluation Expected outcomes include: ●Prevented recurrence of cardiogenic shock. ● Monitored hemodynamic status. ● Administered medications and intravenous fluids. ● Maintained intra-aortic balloon counterpulsation.
  • 21.
    Discharge and HomeCare Lifestyle changes must be made to avoid the recurrence of cardiogenic shock: 1. Control hypertension. 2. Avoid smoking. 3. Maintain a healthy weight. 4. Diet. 5. Exercise.
  • 22.
    Documentation Guidelines 1. Baselineand subsequent findings and individual hemodynamic parameters. 2. Prior medication use. 3. Plan of care. 4. Teaching plan. 5. Client’s responses to interventions, teaching, and actions performed. 6. Attainment or progress toward desired outcomes. 7. Modifications to plan of care.
  • 23.

Editor's Notes

  • #2 My group is number 3, the theme of the presentation is Cardiogenic Shock.
  • #5 A life-threatening medical condition resulting from an inadequate circulation of blood due to primary failure of the ventricles of the heart to function effectively. Signs of tissue hypoperfusion: low urine production, cool extremities, and altered mental of consciousness. Multi-organ failure and death.
  • #6 Cardiogenic shock is a low-output state stemming from primary cardiac dysfunction, resulting in hypotension and systemic hypoperfusion. This maladaptive syndrome is perpetuated by physiologic cycles of inflammation, ischemia, vasoconstriction, and volume overload.
  • #11 Cardiogenic shock produces symptoms of poor tissue perfusion. Clammy skin. The patient experiences cool, clammy skin as the blood could not circulate properly to the peripheries. Decreased systolic blood pressure.The systolic blood pressure decreases to 30 mmHg below baseline. Tachycardia. Tachycardia occurs because the heart pumps faster than normal to compensate for the decreased output all over the body. Rapid respirations. The patient experiences rapid, shallow respirations because there is not enough oxygen circulating in the body. Oliguria. An output of less than 20ml/hour is indicative of oliguria. Mental confusion. Insufficient oxygenated blood in the brain could gradually cause mental confusion and obtundation. Cyanosis. Cyanosis occurs because there is insufficient oxygenated blood that is being distributed to all body systems.