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ASTHMA
1
INTRODUCTION
ī‚ĸ Asthma is a chronic inflammatory disease of
the airways that causes airway hyper-
responsiveness, mucosal edema, and mucus
production
Asthma is characterized by chronic airway
inflammation and increased airway hyper-
responsiveness leading to symptoms of wheeze,
cough, chest tightness and dyspnoea.
4
ī‚ĸ Asthma is a disorder of the bronchial airways
characterized by period of reversible bronchospam.
5
INTRODUCTION CONTDâ€Ļ..
ī‚ĸAsthma differs from the other obstructive lung
diseases in that it is largely reversible, either
spontaneously or with treatment.
ī‚ĸPatients with asthma may experience
symptom-free periods alternating with acute
exacerbations, which last from minutes to
hours or days.
6
INTRODUCTION CONTDâ€Ļ
ī‚ĸThe most frequent form has its onset
in childhood between the ages of 3
and 5 years and may either worsen or
improve during adolescence.
7
8
EPIDEMIOLOGY
ī‚ĸIn many countries the prevalence of
asthma is increasing.
ī‚ĸThis increase, with its accompanying
allergy, is particularly in children and
young adults where this disease may
affect up to 15% of the population.
ī‚ĸAsthma being commoner in more
developed countries. 9
EPIDEMIOLOGY CONTDâ€Ļ.
ī‚ĸWorld-wide ,approximately 300 million
people have asthma and this is ,expected to
rise to 400 million by 2025.
ī‚ĸIn childhood, asthma is more common in
boys, but following puberty females are
more frequently affected.
10
ETIOLOGY AND RISK FACTOR
īƒ˜ Asthma occurs in families which suggest that it
is an inherited disorder.
ī‚ĸAllergy is the strongest predisposing factor for
asthma.
ī‚ĸChronic exposure to airway irritants or
allergens also increases the risk for developing
asthma.
ī‚ĸCommon allergens can be seasonal (eg, grass,
tree, and weed pollens, mold, dust, or animal
dander).
11
CONTDâ€Ļ
īƒ˜ Excitatory state (stress ,cry )
īƒ˜ Occupational environment
īƒ˜ factor such as cold air, air pollution, drug
infection,
īƒ˜ Occupational environment
īƒ˜ Other factor such as cold air ,air pollution,
infection, diet
12
CONTDâ€Ļ
Triggers
ī‚ĸ Allergens
ī‚ĸ Upper respiratory tract viral infections
ī‚ĸExercise
ī‚ĸ Cold air
ī‚ĸ Sulfur dioxide Drugs ( BETA blockers,
aspirin)
ī‚ĸ Stress
ī‚ĸ Irritants (household sprays, paint fumes)13
CLASSIFICATION
Asthma is a complex disorder of the
conducting airways that most simply can be
classified as:
ī‚ĸ extrinsic – implying a definite external cause
ī‚ĸ intrinsic – when no causative agent can be
identified.
14
Physiology/Pathophysiology
15
16
Exposure to allergens & irritants
IGE stimulation
Mast cells degradation
Histamine Prostaglandins Bradikinins Leukotrienes
Air way hyperresponsiveness
Mucus secretion
Bronchospasm
Inflammation
Non productive cough
Shortness of breath
Wheezing, chest tightness,
Peakflow variability
CLINICAL MANIFESTATIONS
ī‚ĸ The principal symptoms of asthma are wheezing
attacks and episodic shortness of breath.
ī‚ĸ Typical symptoms include recurrent episodes of
wheezing, chest tightness, breathlessness and
cough.
ī‚ĸ In some instances, cough may be the only
symptom
ī‚ĸ cough, with or without mucus production
ī‚ĸ Expiration requires effort and becomes
prolonged.
17
Contdâ€Ļ.
ī‚ĸAs the exacerbation progresses,
diaphoresis, Tachycardia and a widened
pulse pressure may occur along with
hypoxemia and central cyanosis .
18
DIAGNOSIS
History taking
ī‚ĸA complete family, environmental, and
occupational history is essential.
ī‚ĸ Family history : History of asthma in family
ī‚ĸEnvironmental history : seasonal changes,
high pollen counts, mold, climate changes
(particularly cold air), and air pollution,
19
CONTDâ€Ļ
ī‚ĸ Occupational history : occupation-related
chemicals and compounds, including metal
salts, wood and vegetable dust
ī‚ĸ Medications (eg, aspirin)
ī‚ĸ Industrial chemicals and plastics, biologic
enzymes (eg, laundry detergents), animal and
insect dusts, sera, and secretions.
20
CONTD..
Physical examination
ī‚ĸ wheezing all over the lung
ī‚ĸbreathlessness and cough.
ī‚ĸCyanosis
21
INVESTIGATIONS
ī‚ĸLung function tests/ pulmonary function
test : Shows variable airflow limitation
ī‚ĸBlood tests :shows increase in the number
of eosinophils in peripheral blood (> 0.4 ×
109/L).
ī‚ĸ Sputum tests The presence of large
numbers of eosinophils in the sputum is a
more useful diagnostic tool. 22
CONTDâ€Ļ
ī‚ĸChest X-ray : There are no diagnostic
features of asthma on the chest X-ray
ī‚ĸA chest X-ray may be helpful in excluding
a pneumothorax, which can occur as a
complication of asthma
ī‚ĸSkin tests Skin-prick tests (SPT) should
be performed in all cases of asthma to
help identify allergic causes.
23
24
ESSENTIALS OF DIAGNOSIS
ī‚ĸ Episodic or chronic symptoms of airflow
obstruction: breathlessness, cough, wheezing, and
chest tightness.
ī‚ĸ Symptoms frequently worse at night or in the early
morning.
ī‚ĸ Prolonged expiration and diffuse wheezes on
physical examination.
ī‚ĸ Limitation of airflow on pulmonary function testing.
ī‚ĸ Complete or partial reversibility of airflow
obstruction, either spontaneously or following
bronchodilator therapy.
25
MEDICAL MANAGEMENT
THE GOALS OF ASTHMA MANAGEMENT
ī‚ĸ Achieve and maintain control of symptoms
ī‚ĸPrevent asthma exacerbations
ī‚ĸMaintain pulmonary function as close to normal
as possible
ī‚ĸAvoid adverse effects from asthma medications
ī‚ĸPrevent development of irreversible airflow
limitation
ī‚ĸPrevent asthma mortality
27
MEDICAL MANAGEMENT
ī‚ĸ Reassure the patient , as anxiety worsen respiratory
distress
ī‚ĸ Keep the patient in upright position
ī‚ĸ Start oxygen 50-60 % initially, continue till the patient
is better and not dyspnoeic
ī‚ĸ Nebulized with salbutamol or Terbutalin for
immediate relief.
Salbutamol 5 mg (1 ml with 1 ml normal saline) stat.
Repeat dose at 15 mins if required during the first
hour.
Hourly for next few hours till bronchospasm is
controlled.
28
MEDICAL MANAGEMENT CONTDâ€Ļ.
ī‚ĸSecure I/V line
ī‚ĸInj. Hydrocotisone 200mg I/V stat routinely
given to all severe cases than 6 hourly
ī‚ĸAntibiotic if there is evidence of infection
ī‚ĸAdequate hydration and mucolytics.
29
NURSING MANAGEMENT
NURSING PROCESS:
Assessment
ī‚ĸAssessment involves obtaining information
about current symptoms as well as previous
disease manifestations
ī‚ĸA complete family, environmental, and
occupational history is essential.
ī‚ĸ family history : History of asthma in family
31
Contdâ€Ļ
ī‚ĸEnvironmental history : seasonal changes,
high pollen counts, mold, climate changes
(particularly cold air), and air pollution
ī‚ĸoccupational history : occupation-related
chemicals and compounds, including metal
salts, wood and vegetable dust,
ī‚ĸMedications taken (eg, aspirin)
32
CONTDâ€Ļ
Industrial chemicals and plastics, biologic
enzymes (eg, laundry detergents), animal
and insect dusts, sera, and secretions
Physical examination
ī‚ĸ wheezing all over the lung
ī‚ĸbreathlessness and cough.
ī‚ĸCyanosis
33
NURSING DIAGNOSIS
ī‚ĸIneffective airway clearance related to
increased production of secretions and
bronchospasm.
ī‚ĸIneffective breathing pattern related to
shortness of breath, mucus,
bronchoconstriction, and airway
irritants
34
Contdâ€Ļ
ī‚ĸActivity intolerance due to fatigue, ineffective
breathing patterns, and hypoxemia
ī‚ĸ Deficient knowledge of self-care strategies to
be performed at home.
ī‚ĸIneffective coping related to reduced
socialization, anxiety, depression, lower
activity level, and the inability to work
35
PLANNING AND GOALS
The major goals for the patient may include
ī‚ĸsmoking cessation,
ī‚ĸImproved gas exchange, & Airway
clearance,
ī‚ĸimproved breathing pattern,
ī‚ĸ Improved activity tolerance ,
ī‚ĸMaximal self-management,
ī‚ĸImproved coping ability,
ī‚ĸAdherence to the therapeutic program ,Home
care & Absence of complications.
36
DIAGNOSIS: INEFFECTIVE BREATHING
PATTERN
Nursing diagnosis: Ineffective breathing
pattern related to shortness of breath, mucus,
bronchoconstriction, and airway irritants
Outcomes
ī‚ĸA decreasing respiratory rate to within normal
limits.
ī‚ĸDecreased dyspnoea, less nasal flaring, and
reduced use of accessory muscles.
ī‚ĸDecreased manifestations of anxiety.
ī‚ĸOxygen saturation greater than 95%.
37
INTERVENTIONS
ī‚ĸAssess the client frequently, observing
respiratory rate and depth.
ī‚ĸAssess the breathing pattern for shortness
of breath, pursed-lip breathing, nasal
flaring,
ī‚ĸAssess of Sternal and intercostals
retractions, or a prolonged expiratory,
phase.
38
INTERVENTIONS CONTDâ€Ļ
ī‚ĸPlace the client in the fowler position
ī‚ĸGive oxygen by face mask as ordered.
ī‚ĸ Give Nebulization using asthalin and impravent
solution.
ī‚ĸMonitor ABGs and oxygen saturation level to
determine the effectiveness of treatments.
ī‚ĸReassure the patient .
39
DIAGNOSIS: INEFFECTIVE AIRWAY
CLEARANCE:
ī‚ĸThe nursing diagnosis: Ineffective
Airway Clearance related to increased
production of secretions and
bronchospasm.
Outcomes
ī‚ĸDecreased inspiratory and expiratory
wheezing.
ī‚ĸDecreased rhonchi.
ī‚ĸDecreasing dry, nonproductive cough. 40
INTERVENTIONS
ī‚ĸMonitor the color and consistency of the
sputum.
ī‚ĸAssist the client to cough effectively.
ī‚ĸEncourage oral fluids to thin the secretions
and replace fluids lost through rapid
respiration.
ī‚ĸ perform Postural drainage, lung percussion
and vibration.
ī‚ĸ Give Expectorants as prescribe
ī‚ĸ Do frequent position changes.
41
DIAGNOSIS: IMPAIRED GAS EXCHANGE.
ī‚ĸNursing diagnosis: Impaired Gas
Exchange related to air trapping.
Outcomes
ī‚ĸDecreased inspiratory and expiratory
wheezing.
ī‚ĸOxygen saturation >90%.
ī‚ĸpH of 7.35 to 7.45.
ī‚ĸUsual skin color (no cyanosis).
ī‚ĸDecreasing dry, nonproductive cough. 42
INTERVENTIONS
ī‚ĸAsses lung sounds every hour during acute
episodes to determine the adequacy of gas
exchange.
ī‚ĸAsses skin and mucous membrane color of
cyanosis.(Cyanosis is a late manifestation of
hypoxia and an indication of serious gas-
exchange problems.)
ī‚ĸMonitor pulse oximetry for oxygen saturation
levels.
ī‚ĸAdminister oxygen as ordered to maintain
optimal oxygen saturation.
43
INTERVENTIONS CONTDâ€Ļ
ī‚ĸAssist the client to cough effectively.
ī‚ĸEncourage oral fluids to thin the secretions
and replace fluids lost through rapid
respiration.
ī‚ĸ perform Postural drainage, lung
percussion and vibration.
ī‚ĸ Give Expectorants as prescribe
ī‚ĸ Do frequent position changes.
44
KNOWLEDGE DEFICIT.
ī‚ĸ Nursing Diagnosis: Knowledge Deficit
related to use of inhaler and /nebulizer
mechine.
Outcomes
ī‚ĸThe client will have increased knowledge
about how to use nebulizer /inhaler
evidenced by explaining or demonstrating
the correct technique
45
INTERVENTIONS
ī‚ĸShow the each part of device and explain
the function of inhaler device.
ī‚ĸDemonstrate/ explain the patient about
correct use of device and how to care of
the device .
ī‚ĸCertain the client knows prober use of
device (post test)
ī‚ĸ Observe the client’s use of the inhaled to
ascertain whether the medication is
entering the airway. 46
47
RISK FOR DECISIONAL CONFLICT
Outcomes
ī‚ĸThe client will express an understanding of
the conflict involved with maintain the
present life style and its effect asthma.
48
INTERVENTIONS
ī‚ĸEncourage clients to stop smoking.
ī‚ĸElimination of irritants as much as possible .
ī‚ĸ Encourage for use of mask to protect from
dust .
ī‚ĸAdvice patient not to directly expose in cold .
ī‚ĸAdvice to keep the emergency medicine at
any time with him/her
ī‚ĸTake special precaution while caring the pets.
49
ACTIVITY INTOLERANCE
ī‚ĸ Nursing Diagnosis: Activity intolerance
due to fatigue, ineffective breathing patterns,
and hypoxemia
Out comes
ī‚ĸMaintained respiratory depth and rate while
activity
ī‚ĸ Decreased dyspnoea
ī‚ĸDecreased manifestations of anxiety
ī‚ĸIncreased participation in activity of daily
living 50
INTERVENTION
ī‚ĸ Assess level of activity tolerance and degree of
fatigue, lethargy, and malaise when performing
routine ADLs.
ī‚ĸ Assist with activities and hygiene when fatigued.
ī‚ĸ Encourage for rest when fatigued
ī‚ĸ finish the procedure with in a short period of
time to avoid extra exertion
ī‚ĸ Give rest between the procedure
ī‚ĸ monitor the oxygen saturation frequently &
Provide oxygen when necessary during the
activity .
51
PREVENTIVE MEASURES FOR ASTHMA
Asthma is not a totally preventable disease
however the person should take
precaution from allergen related to
occupational exposure and household
exposure .
īƒ˜ Use of face mask
īƒ˜ Avoid upper respiratory tract infection as
much as possible.
īƒ˜ Avoid smoking and smoky environment
īƒ˜ Avoid passive smoking
52
PREVENTION CONTDâ€Ļ
You can reduce asthma symptoms by avoiding
known triggers and substances that irritate the
airways.
ī‚ĸCover bedding with "allergy-proof" casings to
reduce exposure to dust mites.
ī‚ĸRemove carpets from bedrooms and vacuum
regularly.
ī‚ĸUse only odorless detergents and cleaning
materials in the home.
ī‚ĸKeep humidity levels low and fix leaks to
reduce the growth of organisms such as mold.
53
PREVENTION CONTDâ€Ļ
ī‚ĸKeep humidity levels low and fix leaks to
reduce the growth of organisms such as
mold.
ī‚ĸKeep the house clean and keep food in
containers and out of bedrooms -- this helps
reduce the possibility of cockroaches, which
can trigger asthma attacks in some people.
ī‚ĸIf a person is allergic to an animal that
cannot be removed from the home, the
animal should be kept out of the bedroom..
54
PREVENTION CONTDâ€Ļ
ī‚ĸ Eliminate tobacco smoke from the home. This is
the single most important thing a family can do to
help a child with asthma. Family members and
visitors who smoke outside carry smoke residue
inside on their clothes and hair -- this can trigger
asthma symptoms.
ī‚ĸ Persons with asthma should also avoid air
pollution, industrial dusts, and other irritating fumes
as much as possible.
55
REFERENCES
ī‚ĸJoyce M. BLACK., Jane Hokanson
Hawk.(2009).Medical-Surgical Nursing ,
Clinical Management for Positive
Outcomes(8th ed.).Elsevier, a division of
Reed Elsevier India private Limited.(page
1570-1577)
ī‚ĸvidyasagar Suda., & Acharya V.
Raviraj.(2000). Manipal Medical Manual (4th
ed.) CBS Publisher and Distributors, Delhi
(Page 53-54)
ī‚ĸBruner& Siddhartha’s (2004). Medical
Surgical Nursing .(12th Ed.) 56
REFERENCES CONTD..
ī‚ĸDavidson’s Principle and Practice of
Medicine (20th Ed)
ī‚ĸ kumar & Clark’s., Clinical Medicine(7th
Ed)
ī‚ĸHarison’s .,Principles of Internal
medicine(17th )
ī‚ĸhttp://www.ncbi.nlm.nih.gov/pubmedhealt
h/PMH0001196/#adam_000141.disease.
prevention retrieve on 2068/10/17
57
THANK YOU
58

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asthma-160424141552.pdf

  • 2. INTRODUCTION ī‚ĸ Asthma is a chronic inflammatory disease of the airways that causes airway hyper- responsiveness, mucosal edema, and mucus production Asthma is characterized by chronic airway inflammation and increased airway hyper- responsiveness leading to symptoms of wheeze, cough, chest tightness and dyspnoea. 4
  • 3. ī‚ĸ Asthma is a disorder of the bronchial airways characterized by period of reversible bronchospam. 5
  • 4. INTRODUCTION CONTDâ€Ļ.. ī‚ĸAsthma differs from the other obstructive lung diseases in that it is largely reversible, either spontaneously or with treatment. ī‚ĸPatients with asthma may experience symptom-free periods alternating with acute exacerbations, which last from minutes to hours or days. 6
  • 5. INTRODUCTION CONTDâ€Ļ ī‚ĸThe most frequent form has its onset in childhood between the ages of 3 and 5 years and may either worsen or improve during adolescence. 7
  • 6. 8
  • 7. EPIDEMIOLOGY ī‚ĸIn many countries the prevalence of asthma is increasing. ī‚ĸThis increase, with its accompanying allergy, is particularly in children and young adults where this disease may affect up to 15% of the population. ī‚ĸAsthma being commoner in more developed countries. 9
  • 8. EPIDEMIOLOGY CONTDâ€Ļ. ī‚ĸWorld-wide ,approximately 300 million people have asthma and this is ,expected to rise to 400 million by 2025. ī‚ĸIn childhood, asthma is more common in boys, but following puberty females are more frequently affected. 10
  • 9. ETIOLOGY AND RISK FACTOR īƒ˜ Asthma occurs in families which suggest that it is an inherited disorder. ī‚ĸAllergy is the strongest predisposing factor for asthma. ī‚ĸChronic exposure to airway irritants or allergens also increases the risk for developing asthma. ī‚ĸCommon allergens can be seasonal (eg, grass, tree, and weed pollens, mold, dust, or animal dander). 11
  • 10. CONTDâ€Ļ īƒ˜ Excitatory state (stress ,cry ) īƒ˜ Occupational environment īƒ˜ factor such as cold air, air pollution, drug infection, īƒ˜ Occupational environment īƒ˜ Other factor such as cold air ,air pollution, infection, diet 12
  • 11. CONTDâ€Ļ Triggers ī‚ĸ Allergens ī‚ĸ Upper respiratory tract viral infections ī‚ĸExercise ī‚ĸ Cold air ī‚ĸ Sulfur dioxide Drugs ( BETA blockers, aspirin) ī‚ĸ Stress ī‚ĸ Irritants (household sprays, paint fumes)13
  • 12. CLASSIFICATION Asthma is a complex disorder of the conducting airways that most simply can be classified as: ī‚ĸ extrinsic – implying a definite external cause ī‚ĸ intrinsic – when no causative agent can be identified. 14
  • 14. 16 Exposure to allergens & irritants IGE stimulation Mast cells degradation Histamine Prostaglandins Bradikinins Leukotrienes Air way hyperresponsiveness Mucus secretion Bronchospasm Inflammation Non productive cough Shortness of breath Wheezing, chest tightness, Peakflow variability
  • 15. CLINICAL MANIFESTATIONS ī‚ĸ The principal symptoms of asthma are wheezing attacks and episodic shortness of breath. ī‚ĸ Typical symptoms include recurrent episodes of wheezing, chest tightness, breathlessness and cough. ī‚ĸ In some instances, cough may be the only symptom ī‚ĸ cough, with or without mucus production ī‚ĸ Expiration requires effort and becomes prolonged. 17
  • 16. Contdâ€Ļ. ī‚ĸAs the exacerbation progresses, diaphoresis, Tachycardia and a widened pulse pressure may occur along with hypoxemia and central cyanosis . 18
  • 17. DIAGNOSIS History taking ī‚ĸA complete family, environmental, and occupational history is essential. ī‚ĸ Family history : History of asthma in family ī‚ĸEnvironmental history : seasonal changes, high pollen counts, mold, climate changes (particularly cold air), and air pollution, 19
  • 18. CONTDâ€Ļ ī‚ĸ Occupational history : occupation-related chemicals and compounds, including metal salts, wood and vegetable dust ī‚ĸ Medications (eg, aspirin) ī‚ĸ Industrial chemicals and plastics, biologic enzymes (eg, laundry detergents), animal and insect dusts, sera, and secretions. 20
  • 19. CONTD.. Physical examination ī‚ĸ wheezing all over the lung ī‚ĸbreathlessness and cough. ī‚ĸCyanosis 21
  • 20. INVESTIGATIONS ī‚ĸLung function tests/ pulmonary function test : Shows variable airflow limitation ī‚ĸBlood tests :shows increase in the number of eosinophils in peripheral blood (> 0.4 × 109/L). ī‚ĸ Sputum tests The presence of large numbers of eosinophils in the sputum is a more useful diagnostic tool. 22
  • 21. CONTDâ€Ļ ī‚ĸChest X-ray : There are no diagnostic features of asthma on the chest X-ray ī‚ĸA chest X-ray may be helpful in excluding a pneumothorax, which can occur as a complication of asthma ī‚ĸSkin tests Skin-prick tests (SPT) should be performed in all cases of asthma to help identify allergic causes. 23
  • 22. 24
  • 23. ESSENTIALS OF DIAGNOSIS ī‚ĸ Episodic or chronic symptoms of airflow obstruction: breathlessness, cough, wheezing, and chest tightness. ī‚ĸ Symptoms frequently worse at night or in the early morning. ī‚ĸ Prolonged expiration and diffuse wheezes on physical examination. ī‚ĸ Limitation of airflow on pulmonary function testing. ī‚ĸ Complete or partial reversibility of airflow obstruction, either spontaneously or following bronchodilator therapy. 25
  • 24. MEDICAL MANAGEMENT THE GOALS OF ASTHMA MANAGEMENT ī‚ĸ Achieve and maintain control of symptoms ī‚ĸPrevent asthma exacerbations ī‚ĸMaintain pulmonary function as close to normal as possible ī‚ĸAvoid adverse effects from asthma medications ī‚ĸPrevent development of irreversible airflow limitation ī‚ĸPrevent asthma mortality 27
  • 25. MEDICAL MANAGEMENT ī‚ĸ Reassure the patient , as anxiety worsen respiratory distress ī‚ĸ Keep the patient in upright position ī‚ĸ Start oxygen 50-60 % initially, continue till the patient is better and not dyspnoeic ī‚ĸ Nebulized with salbutamol or Terbutalin for immediate relief. Salbutamol 5 mg (1 ml with 1 ml normal saline) stat. Repeat dose at 15 mins if required during the first hour. Hourly for next few hours till bronchospasm is controlled. 28
  • 26. MEDICAL MANAGEMENT CONTDâ€Ļ. ī‚ĸSecure I/V line ī‚ĸInj. Hydrocotisone 200mg I/V stat routinely given to all severe cases than 6 hourly ī‚ĸAntibiotic if there is evidence of infection ī‚ĸAdequate hydration and mucolytics. 29
  • 27. NURSING MANAGEMENT NURSING PROCESS: Assessment ī‚ĸAssessment involves obtaining information about current symptoms as well as previous disease manifestations ī‚ĸA complete family, environmental, and occupational history is essential. ī‚ĸ family history : History of asthma in family 31
  • 28. Contdâ€Ļ ī‚ĸEnvironmental history : seasonal changes, high pollen counts, mold, climate changes (particularly cold air), and air pollution ī‚ĸoccupational history : occupation-related chemicals and compounds, including metal salts, wood and vegetable dust, ī‚ĸMedications taken (eg, aspirin) 32
  • 29. CONTDâ€Ļ Industrial chemicals and plastics, biologic enzymes (eg, laundry detergents), animal and insect dusts, sera, and secretions Physical examination ī‚ĸ wheezing all over the lung ī‚ĸbreathlessness and cough. ī‚ĸCyanosis 33
  • 30. NURSING DIAGNOSIS ī‚ĸIneffective airway clearance related to increased production of secretions and bronchospasm. ī‚ĸIneffective breathing pattern related to shortness of breath, mucus, bronchoconstriction, and airway irritants 34
  • 31. Contdâ€Ļ ī‚ĸActivity intolerance due to fatigue, ineffective breathing patterns, and hypoxemia ī‚ĸ Deficient knowledge of self-care strategies to be performed at home. ī‚ĸIneffective coping related to reduced socialization, anxiety, depression, lower activity level, and the inability to work 35
  • 32. PLANNING AND GOALS The major goals for the patient may include ī‚ĸsmoking cessation, ī‚ĸImproved gas exchange, & Airway clearance, ī‚ĸimproved breathing pattern, ī‚ĸ Improved activity tolerance , ī‚ĸMaximal self-management, ī‚ĸImproved coping ability, ī‚ĸAdherence to the therapeutic program ,Home care & Absence of complications. 36
  • 33. DIAGNOSIS: INEFFECTIVE BREATHING PATTERN Nursing diagnosis: Ineffective breathing pattern related to shortness of breath, mucus, bronchoconstriction, and airway irritants Outcomes ī‚ĸA decreasing respiratory rate to within normal limits. ī‚ĸDecreased dyspnoea, less nasal flaring, and reduced use of accessory muscles. ī‚ĸDecreased manifestations of anxiety. ī‚ĸOxygen saturation greater than 95%. 37
  • 34. INTERVENTIONS ī‚ĸAssess the client frequently, observing respiratory rate and depth. ī‚ĸAssess the breathing pattern for shortness of breath, pursed-lip breathing, nasal flaring, ī‚ĸAssess of Sternal and intercostals retractions, or a prolonged expiratory, phase. 38
  • 35. INTERVENTIONS CONTDâ€Ļ ī‚ĸPlace the client in the fowler position ī‚ĸGive oxygen by face mask as ordered. ī‚ĸ Give Nebulization using asthalin and impravent solution. ī‚ĸMonitor ABGs and oxygen saturation level to determine the effectiveness of treatments. ī‚ĸReassure the patient . 39
  • 36. DIAGNOSIS: INEFFECTIVE AIRWAY CLEARANCE: ī‚ĸThe nursing diagnosis: Ineffective Airway Clearance related to increased production of secretions and bronchospasm. Outcomes ī‚ĸDecreased inspiratory and expiratory wheezing. ī‚ĸDecreased rhonchi. ī‚ĸDecreasing dry, nonproductive cough. 40
  • 37. INTERVENTIONS ī‚ĸMonitor the color and consistency of the sputum. ī‚ĸAssist the client to cough effectively. ī‚ĸEncourage oral fluids to thin the secretions and replace fluids lost through rapid respiration. ī‚ĸ perform Postural drainage, lung percussion and vibration. ī‚ĸ Give Expectorants as prescribe ī‚ĸ Do frequent position changes. 41
  • 38. DIAGNOSIS: IMPAIRED GAS EXCHANGE. ī‚ĸNursing diagnosis: Impaired Gas Exchange related to air trapping. Outcomes ī‚ĸDecreased inspiratory and expiratory wheezing. ī‚ĸOxygen saturation >90%. ī‚ĸpH of 7.35 to 7.45. ī‚ĸUsual skin color (no cyanosis). ī‚ĸDecreasing dry, nonproductive cough. 42
  • 39. INTERVENTIONS ī‚ĸAsses lung sounds every hour during acute episodes to determine the adequacy of gas exchange. ī‚ĸAsses skin and mucous membrane color of cyanosis.(Cyanosis is a late manifestation of hypoxia and an indication of serious gas- exchange problems.) ī‚ĸMonitor pulse oximetry for oxygen saturation levels. ī‚ĸAdminister oxygen as ordered to maintain optimal oxygen saturation. 43
  • 40. INTERVENTIONS CONTDâ€Ļ ī‚ĸAssist the client to cough effectively. ī‚ĸEncourage oral fluids to thin the secretions and replace fluids lost through rapid respiration. ī‚ĸ perform Postural drainage, lung percussion and vibration. ī‚ĸ Give Expectorants as prescribe ī‚ĸ Do frequent position changes. 44
  • 41. KNOWLEDGE DEFICIT. ī‚ĸ Nursing Diagnosis: Knowledge Deficit related to use of inhaler and /nebulizer mechine. Outcomes ī‚ĸThe client will have increased knowledge about how to use nebulizer /inhaler evidenced by explaining or demonstrating the correct technique 45
  • 42. INTERVENTIONS ī‚ĸShow the each part of device and explain the function of inhaler device. ī‚ĸDemonstrate/ explain the patient about correct use of device and how to care of the device . ī‚ĸCertain the client knows prober use of device (post test) ī‚ĸ Observe the client’s use of the inhaled to ascertain whether the medication is entering the airway. 46
  • 43. 47
  • 44. RISK FOR DECISIONAL CONFLICT Outcomes ī‚ĸThe client will express an understanding of the conflict involved with maintain the present life style and its effect asthma. 48
  • 45. INTERVENTIONS ī‚ĸEncourage clients to stop smoking. ī‚ĸElimination of irritants as much as possible . ī‚ĸ Encourage for use of mask to protect from dust . ī‚ĸAdvice patient not to directly expose in cold . ī‚ĸAdvice to keep the emergency medicine at any time with him/her ī‚ĸTake special precaution while caring the pets. 49
  • 46. ACTIVITY INTOLERANCE ī‚ĸ Nursing Diagnosis: Activity intolerance due to fatigue, ineffective breathing patterns, and hypoxemia Out comes ī‚ĸMaintained respiratory depth and rate while activity ī‚ĸ Decreased dyspnoea ī‚ĸDecreased manifestations of anxiety ī‚ĸIncreased participation in activity of daily living 50
  • 47. INTERVENTION ī‚ĸ Assess level of activity tolerance and degree of fatigue, lethargy, and malaise when performing routine ADLs. ī‚ĸ Assist with activities and hygiene when fatigued. ī‚ĸ Encourage for rest when fatigued ī‚ĸ finish the procedure with in a short period of time to avoid extra exertion ī‚ĸ Give rest between the procedure ī‚ĸ monitor the oxygen saturation frequently & Provide oxygen when necessary during the activity . 51
  • 48. PREVENTIVE MEASURES FOR ASTHMA Asthma is not a totally preventable disease however the person should take precaution from allergen related to occupational exposure and household exposure . īƒ˜ Use of face mask īƒ˜ Avoid upper respiratory tract infection as much as possible. īƒ˜ Avoid smoking and smoky environment īƒ˜ Avoid passive smoking 52
  • 49. PREVENTION CONTDâ€Ļ You can reduce asthma symptoms by avoiding known triggers and substances that irritate the airways. ī‚ĸCover bedding with "allergy-proof" casings to reduce exposure to dust mites. ī‚ĸRemove carpets from bedrooms and vacuum regularly. ī‚ĸUse only odorless detergents and cleaning materials in the home. ī‚ĸKeep humidity levels low and fix leaks to reduce the growth of organisms such as mold. 53
  • 50. PREVENTION CONTDâ€Ļ ī‚ĸKeep humidity levels low and fix leaks to reduce the growth of organisms such as mold. ī‚ĸKeep the house clean and keep food in containers and out of bedrooms -- this helps reduce the possibility of cockroaches, which can trigger asthma attacks in some people. ī‚ĸIf a person is allergic to an animal that cannot be removed from the home, the animal should be kept out of the bedroom.. 54
  • 51. PREVENTION CONTDâ€Ļ ī‚ĸ Eliminate tobacco smoke from the home. This is the single most important thing a family can do to help a child with asthma. Family members and visitors who smoke outside carry smoke residue inside on their clothes and hair -- this can trigger asthma symptoms. ī‚ĸ Persons with asthma should also avoid air pollution, industrial dusts, and other irritating fumes as much as possible. 55
  • 52. REFERENCES ī‚ĸJoyce M. BLACK., Jane Hokanson Hawk.(2009).Medical-Surgical Nursing , Clinical Management for Positive Outcomes(8th ed.).Elsevier, a division of Reed Elsevier India private Limited.(page 1570-1577) ī‚ĸvidyasagar Suda., & Acharya V. Raviraj.(2000). Manipal Medical Manual (4th ed.) CBS Publisher and Distributors, Delhi (Page 53-54) ī‚ĸBruner& Siddhartha’s (2004). Medical Surgical Nursing .(12th Ed.) 56
  • 53. REFERENCES CONTD.. ī‚ĸDavidson’s Principle and Practice of Medicine (20th Ed) ī‚ĸ kumar & Clark’s., Clinical Medicine(7th Ed) ī‚ĸHarison’s .,Principles of Internal medicine(17th ) ī‚ĸhttp://www.ncbi.nlm.nih.gov/pubmedhealt h/PMH0001196/#adam_000141.disease. prevention retrieve on 2068/10/17 57