Prepared by MD, PhD., Associate Professor, Marta R. Gerasymchyk, pathophysiology department of Ivano-Frankivsk National Medical University, Ukraine.
For medical students
The document discusses the conduction system of the heart and various cardiac arrhythmias or dysrhythmias. It describes the normal conduction pathway beginning with the sinoatrial node and discusses how arrhythmias can result from disturbances to automaticity, conduction or reentry of impulses. Several specific types of arrhythmias are described including sinus node dysrhythmias, atrial arrhythmias, AV junctional dysrhythmias and ventricular dysrhythmias. Causes, characteristics, clinical manifestations and treatment approaches are provided for select arrhythmias such as sinus tachycardia, sinus bradycardia and sick sinus syndrome.
The conducting system of the heart consists of specialized cardiac muscle tissue that generates and transmits electrical impulses to initiate and coordinate heart muscle contraction. It includes the sinoatrial node, atrioventricular node, bundle of His, Purkinje fibers and their left and right branches. These structures work together to conduct electrical signals from the upper to lower chambers and allow synchronized, rhythmic pumping of blood throughout the body. Damage to parts of this system can lead to arrhythmias or require treatment like artificial pacemakers.
The electrocardiogram (ECG) records the electrical activity of the heart during each cardiac cycle. It detects the P wave, QRS complex, and T wave which represent atrial depolarization, ventricular depolarization, and ventricular repolarization, respectively. The ECG is used to analyze the heart rate, rhythm, and intervals between waves to diagnose cardiac conditions. Common arrhythmias include premature beats, supraventricular tachycardias like atrial flutter, and atrial fibrillation which is characterized by disorganized atrial activity and an irregular ventricular response.
This document provides information about heart block, including its definition, types, causes, characteristics, and significance. It begins with an introduction to heart block and the electrical conduction system of the heart. It then defines and describes the three types of heart block - first, second, and third degree heart block - and provides details about their characteristics, causes, and clinical significance. Mobitz types I and II are discussed as subtypes of second degree heart block. The document aims to explain heart block and its different classifications to nursing students.
The conduction system of the heart consists of specialized cardiac muscle fibers that generate and conduct electrical impulses through the heart to coordinate the cardiac cycle. The key components are the sinoatrial node, atrioventricular node, bundle of His, and Purkinje fibers. The sinoatrial node initiates the heartbeat and stimulates the atria to contract. The impulse then travels to the atrioventricular node and through the bundle of His to stimulate simultaneous contraction of the ventricles. The autonomic nervous system also regulates heart rate in response to physiological demands on the body.
Pacemakers provide electrical stimulation to cause cardiac contraction when intrinsic cardiac activity is slow or absent. Pacing systems consist of a pulse generator and leads placed in the heart. Permanent systems use endocardial leads inserted transvenously into the right atrium and/or ventricle. Temporary systems use external pulse generators connected to transvenous or transcutaneous leads. Pacemakers can operate in asynchronous or synchronous modes and are used to treat symptomatic bradycardia. Complications include failure to output or capture pacing stimuli, oversensing, undersensing, and infection.
Preclinical Screening of Antiarrhythmatic Agents- Mpharm-edited.pptxAshish Kumar Jha
This document provides an overview of preclinical screening methods for antiarrhythmic agents. It discusses various in vivo and in vitro models used to study cardiac arrhythmias and evaluate potential antiarrhythmic drugs. In vivo models include chemically-induced arrhythmias using drugs like aconitine or digoxin in rats or guinea pigs. Electrically-induced models use techniques like ventricular fibrillation threshold testing in dogs. Mechanically-induced models involve coronary artery ligation and reperfusion in rats. In vitro preparations include isolated tissues like guinea pig papillary muscle to study action potentials. The goal is to find safe and effective therapies for treating abnormal heart rhythms.
Prepared by MD, PhD., Associate Professor, Marta R. Gerasymchyk, pathophysiology department of Ivano-Frankivsk National Medical University, Ukraine.
For medical students
The document discusses the conduction system of the heart and various cardiac arrhythmias or dysrhythmias. It describes the normal conduction pathway beginning with the sinoatrial node and discusses how arrhythmias can result from disturbances to automaticity, conduction or reentry of impulses. Several specific types of arrhythmias are described including sinus node dysrhythmias, atrial arrhythmias, AV junctional dysrhythmias and ventricular dysrhythmias. Causes, characteristics, clinical manifestations and treatment approaches are provided for select arrhythmias such as sinus tachycardia, sinus bradycardia and sick sinus syndrome.
The conducting system of the heart consists of specialized cardiac muscle tissue that generates and transmits electrical impulses to initiate and coordinate heart muscle contraction. It includes the sinoatrial node, atrioventricular node, bundle of His, Purkinje fibers and their left and right branches. These structures work together to conduct electrical signals from the upper to lower chambers and allow synchronized, rhythmic pumping of blood throughout the body. Damage to parts of this system can lead to arrhythmias or require treatment like artificial pacemakers.
The electrocardiogram (ECG) records the electrical activity of the heart during each cardiac cycle. It detects the P wave, QRS complex, and T wave which represent atrial depolarization, ventricular depolarization, and ventricular repolarization, respectively. The ECG is used to analyze the heart rate, rhythm, and intervals between waves to diagnose cardiac conditions. Common arrhythmias include premature beats, supraventricular tachycardias like atrial flutter, and atrial fibrillation which is characterized by disorganized atrial activity and an irregular ventricular response.
This document provides information about heart block, including its definition, types, causes, characteristics, and significance. It begins with an introduction to heart block and the electrical conduction system of the heart. It then defines and describes the three types of heart block - first, second, and third degree heart block - and provides details about their characteristics, causes, and clinical significance. Mobitz types I and II are discussed as subtypes of second degree heart block. The document aims to explain heart block and its different classifications to nursing students.
The conduction system of the heart consists of specialized cardiac muscle fibers that generate and conduct electrical impulses through the heart to coordinate the cardiac cycle. The key components are the sinoatrial node, atrioventricular node, bundle of His, and Purkinje fibers. The sinoatrial node initiates the heartbeat and stimulates the atria to contract. The impulse then travels to the atrioventricular node and through the bundle of His to stimulate simultaneous contraction of the ventricles. The autonomic nervous system also regulates heart rate in response to physiological demands on the body.
Pacemakers provide electrical stimulation to cause cardiac contraction when intrinsic cardiac activity is slow or absent. Pacing systems consist of a pulse generator and leads placed in the heart. Permanent systems use endocardial leads inserted transvenously into the right atrium and/or ventricle. Temporary systems use external pulse generators connected to transvenous or transcutaneous leads. Pacemakers can operate in asynchronous or synchronous modes and are used to treat symptomatic bradycardia. Complications include failure to output or capture pacing stimuli, oversensing, undersensing, and infection.
Preclinical Screening of Antiarrhythmatic Agents- Mpharm-edited.pptxAshish Kumar Jha
This document provides an overview of preclinical screening methods for antiarrhythmic agents. It discusses various in vivo and in vitro models used to study cardiac arrhythmias and evaluate potential antiarrhythmic drugs. In vivo models include chemically-induced arrhythmias using drugs like aconitine or digoxin in rats or guinea pigs. Electrically-induced models use techniques like ventricular fibrillation threshold testing in dogs. Mechanically-induced models involve coronary artery ligation and reperfusion in rats. In vitro preparations include isolated tissues like guinea pig papillary muscle to study action potentials. The goal is to find safe and effective therapies for treating abnormal heart rhythms.
The document discusses arrhythmias and their management. It begins by describing the normal electrical conduction system of the heart. It then defines arrhythmias as disorders of heart rhythm or rate caused by issues with impulse formation or conduction. Various types of arrhythmias are classified based on the site of abnormal impulse formation or conduction, including sinus node arrhythmias, atrial arrhythmias, junctional arrhythmias, and ventricular arrhythmias. Causes and treatments of several specific arrhythmias are described in detail.
The document discusses arrhythmias and their management. It begins by describing the normal electrical conduction system of the heart. It then defines arrhythmias as disorders of heart rhythm or rate caused by issues with electrical impulse formation or conduction. Various types of arrhythmias are classified based on the site of abnormal impulse formation or conduction, including sinus node arrhythmias, atrial arrhythmias, junctional arrhythmias, and ventricular arrhythmias. Treatment depends on restoring normal rhythm and addressing any underlying causes.
The SA node acts as the pacemaker of the heart by spontaneously generating action potentials that spread through the atria. The atrioventricular node then slows conduction to allow time for blood to fill the ventricles before they contract. The excitation wave travels through specialized conduction pathways in the ventricles before activating the purkinje fibers and ventricular muscle. The autonomic nervous system controls heart rate and conduction through the atrioventricular node. An ECG records the electrical activity of the heart by detecting the P, QRS, and T waves which represent atrial depolarization, ventricular depolarization, and ventricular repolarization, respectively.
This document provides information on the regulation of circulation. It discusses three major types of regulation: neural, humoral, and local. For neural regulation, it describes the innervation of the heart and blood vessels by the sympathetic and parasympathetic nervous systems. It also discusses cardiovascular centers in the brainstem that control blood pressure. For humoral regulation, it outlines vasoconstrictors like epinephrine and angiotensin II, as well as the vasodilator nitric oxide. Finally, it briefly introduces local autoregulation mediated by myogenic activity and chemical factors.
Cardiovascular assessment and diagnostic proceduresANILKUMAR BR
The document provides an overview of cardiovascular anatomy and physiology, including the structure of the heart, chambers of the heart, heart valves, cardiac cycle, and coronary arteries. It also discusses the cardiac conduction system, including the sinoatrial node, atrioventricular node, bundle of His, and Purkinje fibers. Common diagnostic tests and manifestations of cardiovascular disease are also mentioned.
This document provides an overview of tachyarrhythmias and their mechanisms. It discusses the normal cardiac conduction system and describes how abnormalities can lead to arrhythmias via mechanisms like accelerated automaticity, triggered activity, and reentry. It then focuses on atrial fibrillation, describing its classification, causes, diagnosis, and treatment approaches like rate control and anticoagulation based on stroke risk scores. The document emphasizes the importance of evaluating hemodynamic stability and controlling heart rate for arrhythmia patients.
Anatomy, physiology & patophysiology of the cardiovascularCarlos Galiano
This document provides an overview of the anatomy, physiology, and pathophysiology of the cardiovascular system as it relates to anesthesia. It discusses the structure and function of the heart and coronary circulation. It then covers topics such as cardiac cycle, hemodynamics, effects of the autonomic nervous system, and cardiovascular disorders including heart failure, hypertension, ischemic heart disease, and cardiac tamponade. For each topic, it provides details on pathophysiology and considerations for anesthesia management.
This document discusses the electrophysiology of the heart and how cardiovascular drugs can act on heart tissue. It outlines several key properties of cardiac electrophysiology that are important for understanding drug action, including impulse generation, conduction, excitability, and refractory period. Automatic fibers located in the sinoatrial and atrioventricular nodes generate impulses, while nonautomatic fibers cannot. The document also notes that autonomic influences like the sympathetic and parasympathetic nervous systems impact cardiac electrophysiology and contractility, and that many cardiovascular drugs have indirect autonomic effects.
Cardiac arrhythmia refers to any abnormal heart rhythm and can cause the heart rate to be too fast, too slow, or irregular. Common types include sinus tachycardia, ventricular tachycardia, sinus bradycardia, paroxysmal supraventricular tachycardia, atrial flutter, atrial fibrillation, premature atrial contractions, premature ventricular contractions, and nodal rhythm. Treatment depends on the specific arrhythmia but may include medications, cardioversion, pacemakers, ablation, or defibrillation.
The document provides an overview of the cardiovascular system, including the anatomy and physiology of the heart and blood vessels. It discusses the chambers of the heart, valves, coronary circulation, conduction system, and nerve supply. It also covers measurements of various pressures like mean arterial pressure, central venous pressure, and pulmonary artery pressure. Other topics include cardiac output, stroke volume, contractility, and the determinants of cardiac performance. Graphs of the cardiac cycle and Wigger's diagram are presented showing the mechanical events in systole and diastole.
ORIGIN OF THE HEARTBEAT & THE ELECTRICAL ACTIVITY OF THE HEART.pptxshreya730959
The heartbeat originates in the sinoatrial (SA) node, which acts as the heart's natural pacemaker. Impulses from the SA node spread through the conduction system to the atria and ventricles. The SA node discharges spontaneously at the fastest rate, setting the heartbeat. Impulses pass from the SA node through the atria to the atrioventricular (AV) node and bundle of His, then via Purkinje fibers to ventricular muscle. Vagal stimulation slows the heartbeat by inhibiting the SA and AV nodes, while sympathetic stimulation increases the heart rate by facilitating impulse propagation. Digitalis depresses the conduction system like vagal stimulation and is used clinically to improve heart function and control
Basic science and forensic pathology aspects of cardiac conduction system dis...Luchengam
This document provides an overview of cardiac conduction system disorders from both a basic science and forensic pathology perspective. It discusses arrhythmias, their causes, types and potential symptoms. Sudden cardiac death is also examined, with the most common cause being coronary artery disease. Non-atherosclerotic causes are more prevalent in younger victims and can include channelopathies, cardiomyopathies and other structural abnormalities. The morphology of sudden cardiac death is outlined, with most cases associated with significant coronary atherosclerosis but acute myocardial infarction only present in a minority of cases. Forensic examination of the cardiac conduction system can provide insights into underlying causes of death in select circumstances.
CARDIAC AUTONOMIC SYSTEM CLINICAL SIGNIFICANCE.pptxaamirrashid39
This document discusses the anatomy and physiology of the cardiac autonomic nervous system. It covers the extrinsic and intrinsic innervation of the heart, including the sympathetic and parasympathetic fibers. Evaluation methods for the autonomic system are described, such as orthostatic tests, heart rate variability, and tilt-table testing. Conditions related to autonomic dysfunction like postural orthostatic tachycardia syndrome and neurally mediated syncope are explained. The role of the autonomic system in arrhythmias and other cardiac conditions is also summarized.
The intrinsic conduction system consists of specialized noncontractile cardiac cells that initiate and distribute impulses throughout the heart in a sequential manner. Impulses travel through autorhythmic cells like the sinoatrial node, then through the atrioventricular node, bundle of His, and Purkinje fibers to trigger rhythmic contractions. Defects can cause arrhythmias like fibrillation. The cardiac cycle involves systole, diastole, and the flow of blood through the heart, regulated by the medulla oblongata and monitored by electrocardiograms.
Term paper on ECG and cardiac arrhythmiasRomena Begum
The document provides information about ECGs and cardiac arrhythmias. It discusses the history and development of ECGs, the anatomy and conduction system of the heart, common arrhythmias like sinus tachycardia and sinus bradycardia, and how to diagnose and manage various types of cardiac arrhythmias using ECGs. The document contains diagrams of ECG readings and the heart to illustrate different arrhythmias and the heart's structure and function.
Term paper on ecg and cardiac arrhythmiasROMENABEGUM
The document provides information on ECGs and cardiac arrhythmias. It begins with an introduction to ECGs and what they measure. It then discusses the history of ECGs, the anatomy and conduction system of the heart, common indications for ECGs, how ECGs are arranged and interpreted, and definitions of heart rate and rhythm. The majority of the document categorizes and describes different types of cardiac arrhythmias like sinus tachycardia, sinus bradycardia, premature atrial contractions, atrial flutter, atrial fibrillation, junctional rhythm, junctional tachycardia, premature junctional contractions, and supraventricular tachycardia. For each type it discusses causes
The document summarizes the intrinsic cardiac conduction system and how it regulates heart rate. It describes the roles of the sinoatrial node, atrioventricular node, bundle of His, bundle branches and Purkinje fibers in initiating and distributing electrical impulses throughout the heart. It also discusses how the sympathetic and parasympathetic nervous systems can respectively increase or decrease heart rate through their effects on these conduction pathways. Various types of bradyarrhythmias and atrioventricular conduction blocks are then outlined.
External manifestations of cardiac activity. Regulation of heart workEneutron
The document summarizes the external manifestations and regulation of cardiac activity. It describes the phases of the cardiac cycle including atrial systole, ventricular systole, and common pause. It discusses mechanical, sound, and electrical external manifestations observed on sphygmograms, heart tones, and ECGs. Regulation occurs through intracardiac mechanisms like the Frank-Starling law and peripheral heart reflexes, as well as extracardiac neural influences of the sympathetic and parasympathetic nervous systems and hormonal regulation.
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The document discusses arrhythmias and their management. It begins by describing the normal electrical conduction system of the heart. It then defines arrhythmias as disorders of heart rhythm or rate caused by issues with impulse formation or conduction. Various types of arrhythmias are classified based on the site of abnormal impulse formation or conduction, including sinus node arrhythmias, atrial arrhythmias, junctional arrhythmias, and ventricular arrhythmias. Causes and treatments of several specific arrhythmias are described in detail.
The document discusses arrhythmias and their management. It begins by describing the normal electrical conduction system of the heart. It then defines arrhythmias as disorders of heart rhythm or rate caused by issues with electrical impulse formation or conduction. Various types of arrhythmias are classified based on the site of abnormal impulse formation or conduction, including sinus node arrhythmias, atrial arrhythmias, junctional arrhythmias, and ventricular arrhythmias. Treatment depends on restoring normal rhythm and addressing any underlying causes.
The SA node acts as the pacemaker of the heart by spontaneously generating action potentials that spread through the atria. The atrioventricular node then slows conduction to allow time for blood to fill the ventricles before they contract. The excitation wave travels through specialized conduction pathways in the ventricles before activating the purkinje fibers and ventricular muscle. The autonomic nervous system controls heart rate and conduction through the atrioventricular node. An ECG records the electrical activity of the heart by detecting the P, QRS, and T waves which represent atrial depolarization, ventricular depolarization, and ventricular repolarization, respectively.
This document provides information on the regulation of circulation. It discusses three major types of regulation: neural, humoral, and local. For neural regulation, it describes the innervation of the heart and blood vessels by the sympathetic and parasympathetic nervous systems. It also discusses cardiovascular centers in the brainstem that control blood pressure. For humoral regulation, it outlines vasoconstrictors like epinephrine and angiotensin II, as well as the vasodilator nitric oxide. Finally, it briefly introduces local autoregulation mediated by myogenic activity and chemical factors.
Cardiovascular assessment and diagnostic proceduresANILKUMAR BR
The document provides an overview of cardiovascular anatomy and physiology, including the structure of the heart, chambers of the heart, heart valves, cardiac cycle, and coronary arteries. It also discusses the cardiac conduction system, including the sinoatrial node, atrioventricular node, bundle of His, and Purkinje fibers. Common diagnostic tests and manifestations of cardiovascular disease are also mentioned.
This document provides an overview of tachyarrhythmias and their mechanisms. It discusses the normal cardiac conduction system and describes how abnormalities can lead to arrhythmias via mechanisms like accelerated automaticity, triggered activity, and reentry. It then focuses on atrial fibrillation, describing its classification, causes, diagnosis, and treatment approaches like rate control and anticoagulation based on stroke risk scores. The document emphasizes the importance of evaluating hemodynamic stability and controlling heart rate for arrhythmia patients.
Anatomy, physiology & patophysiology of the cardiovascularCarlos Galiano
This document provides an overview of the anatomy, physiology, and pathophysiology of the cardiovascular system as it relates to anesthesia. It discusses the structure and function of the heart and coronary circulation. It then covers topics such as cardiac cycle, hemodynamics, effects of the autonomic nervous system, and cardiovascular disorders including heart failure, hypertension, ischemic heart disease, and cardiac tamponade. For each topic, it provides details on pathophysiology and considerations for anesthesia management.
This document discusses the electrophysiology of the heart and how cardiovascular drugs can act on heart tissue. It outlines several key properties of cardiac electrophysiology that are important for understanding drug action, including impulse generation, conduction, excitability, and refractory period. Automatic fibers located in the sinoatrial and atrioventricular nodes generate impulses, while nonautomatic fibers cannot. The document also notes that autonomic influences like the sympathetic and parasympathetic nervous systems impact cardiac electrophysiology and contractility, and that many cardiovascular drugs have indirect autonomic effects.
Cardiac arrhythmia refers to any abnormal heart rhythm and can cause the heart rate to be too fast, too slow, or irregular. Common types include sinus tachycardia, ventricular tachycardia, sinus bradycardia, paroxysmal supraventricular tachycardia, atrial flutter, atrial fibrillation, premature atrial contractions, premature ventricular contractions, and nodal rhythm. Treatment depends on the specific arrhythmia but may include medications, cardioversion, pacemakers, ablation, or defibrillation.
The document provides an overview of the cardiovascular system, including the anatomy and physiology of the heart and blood vessels. It discusses the chambers of the heart, valves, coronary circulation, conduction system, and nerve supply. It also covers measurements of various pressures like mean arterial pressure, central venous pressure, and pulmonary artery pressure. Other topics include cardiac output, stroke volume, contractility, and the determinants of cardiac performance. Graphs of the cardiac cycle and Wigger's diagram are presented showing the mechanical events in systole and diastole.
ORIGIN OF THE HEARTBEAT & THE ELECTRICAL ACTIVITY OF THE HEART.pptxshreya730959
The heartbeat originates in the sinoatrial (SA) node, which acts as the heart's natural pacemaker. Impulses from the SA node spread through the conduction system to the atria and ventricles. The SA node discharges spontaneously at the fastest rate, setting the heartbeat. Impulses pass from the SA node through the atria to the atrioventricular (AV) node and bundle of His, then via Purkinje fibers to ventricular muscle. Vagal stimulation slows the heartbeat by inhibiting the SA and AV nodes, while sympathetic stimulation increases the heart rate by facilitating impulse propagation. Digitalis depresses the conduction system like vagal stimulation and is used clinically to improve heart function and control
Basic science and forensic pathology aspects of cardiac conduction system dis...Luchengam
This document provides an overview of cardiac conduction system disorders from both a basic science and forensic pathology perspective. It discusses arrhythmias, their causes, types and potential symptoms. Sudden cardiac death is also examined, with the most common cause being coronary artery disease. Non-atherosclerotic causes are more prevalent in younger victims and can include channelopathies, cardiomyopathies and other structural abnormalities. The morphology of sudden cardiac death is outlined, with most cases associated with significant coronary atherosclerosis but acute myocardial infarction only present in a minority of cases. Forensic examination of the cardiac conduction system can provide insights into underlying causes of death in select circumstances.
CARDIAC AUTONOMIC SYSTEM CLINICAL SIGNIFICANCE.pptxaamirrashid39
This document discusses the anatomy and physiology of the cardiac autonomic nervous system. It covers the extrinsic and intrinsic innervation of the heart, including the sympathetic and parasympathetic fibers. Evaluation methods for the autonomic system are described, such as orthostatic tests, heart rate variability, and tilt-table testing. Conditions related to autonomic dysfunction like postural orthostatic tachycardia syndrome and neurally mediated syncope are explained. The role of the autonomic system in arrhythmias and other cardiac conditions is also summarized.
The intrinsic conduction system consists of specialized noncontractile cardiac cells that initiate and distribute impulses throughout the heart in a sequential manner. Impulses travel through autorhythmic cells like the sinoatrial node, then through the atrioventricular node, bundle of His, and Purkinje fibers to trigger rhythmic contractions. Defects can cause arrhythmias like fibrillation. The cardiac cycle involves systole, diastole, and the flow of blood through the heart, regulated by the medulla oblongata and monitored by electrocardiograms.
Term paper on ECG and cardiac arrhythmiasRomena Begum
The document provides information about ECGs and cardiac arrhythmias. It discusses the history and development of ECGs, the anatomy and conduction system of the heart, common arrhythmias like sinus tachycardia and sinus bradycardia, and how to diagnose and manage various types of cardiac arrhythmias using ECGs. The document contains diagrams of ECG readings and the heart to illustrate different arrhythmias and the heart's structure and function.
Term paper on ecg and cardiac arrhythmiasROMENABEGUM
The document provides information on ECGs and cardiac arrhythmias. It begins with an introduction to ECGs and what they measure. It then discusses the history of ECGs, the anatomy and conduction system of the heart, common indications for ECGs, how ECGs are arranged and interpreted, and definitions of heart rate and rhythm. The majority of the document categorizes and describes different types of cardiac arrhythmias like sinus tachycardia, sinus bradycardia, premature atrial contractions, atrial flutter, atrial fibrillation, junctional rhythm, junctional tachycardia, premature junctional contractions, and supraventricular tachycardia. For each type it discusses causes
The document summarizes the intrinsic cardiac conduction system and how it regulates heart rate. It describes the roles of the sinoatrial node, atrioventricular node, bundle of His, bundle branches and Purkinje fibers in initiating and distributing electrical impulses throughout the heart. It also discusses how the sympathetic and parasympathetic nervous systems can respectively increase or decrease heart rate through their effects on these conduction pathways. Various types of bradyarrhythmias and atrioventricular conduction blocks are then outlined.
External manifestations of cardiac activity. Regulation of heart workEneutron
The document summarizes the external manifestations and regulation of cardiac activity. It describes the phases of the cardiac cycle including atrial systole, ventricular systole, and common pause. It discusses mechanical, sound, and electrical external manifestations observed on sphygmograms, heart tones, and ECGs. Regulation occurs through intracardiac mechanisms like the Frank-Starling law and peripheral heart reflexes, as well as extracardiac neural influences of the sympathetic and parasympathetic nervous systems and hormonal regulation.
Similar to arrythemias abnormal heart rate aaaaaaaa (20)
Kosmoderma Academy, a leading institution in the field of dermatology and aesthetics, offers comprehensive courses in cosmetology and trichology. Our specialized courses on PRP (Hair), DR+Growth Factor, GFC, and Qr678 are designed to equip practitioners with advanced skills and knowledge to excel in hair restoration and growth treatments.
These lecture slides, by Dr Sidra Arshad, offer a simplified look into the mechanisms involved in the regulation of respiration:
Learning objectives:
1. Describe the organisation of respiratory center
2. Describe the nervous control of inspiration and respiratory rhythm
3. Describe the functions of the dorsal and respiratory groups of neurons
4. Describe the influences of the Pneumotaxic and Apneustic centers
5. Explain the role of Hering-Breur inflation reflex in regulation of inspiration
6. Explain the role of central chemoreceptors in regulation of respiration
7. Explain the role of peripheral chemoreceptors in regulation of respiration
8. Explain the regulation of respiration during exercise
9. Integrate the respiratory regulatory mechanisms
10. Describe the Cheyne-Stokes breathing
Study Resources:
1. Chapter 42, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 36, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 13, Human Physiology by Lauralee Sherwood, 9th edition
NAVIGATING THE HORIZONS OF TIME LAPSE EMBRYO MONITORING.pdfRahul Sen
Time-lapse embryo monitoring is an advanced imaging technique used in IVF to continuously observe embryo development. It captures high-resolution images at regular intervals, allowing embryologists to select the most viable embryos for transfer based on detailed growth patterns. This technology enhances embryo selection, potentially increasing pregnancy success rates.
Histololgy of Female Reproductive System.pptxAyeshaZaid1
Dive into an in-depth exploration of the histological structure of female reproductive system with this comprehensive lecture. Presented by Dr. Ayesha Irfan, Assistant Professor of Anatomy, this presentation covers the Gross anatomy and functional histology of the female reproductive organs. Ideal for students, educators, and anyone interested in medical science, this lecture provides clear explanations, detailed diagrams, and valuable insights into female reproductive system. Enhance your knowledge and understanding of this essential aspect of human biology.
Osteoporosis - Definition , Evaluation and Management .pdfJim Jacob Roy
Osteoporosis is an increasing cause of morbidity among the elderly.
In this document , a brief outline of osteoporosis is given , including the risk factors of osteoporosis fractures , the indications for testing bone mineral density and the management of osteoporosis
Travel Clinic Cardiff: Health Advice for International TravelersNX Healthcare
Travel Clinic Cardiff offers comprehensive travel health services, including vaccinations, travel advice, and preventive care for international travelers. Our expert team ensures you are well-prepared and protected for your journey, providing personalized consultations tailored to your destination. Conveniently located in Cardiff, we help you travel with confidence and peace of mind. Visit us: www.nxhealthcare.co.uk
DECLARATION OF HELSINKI - History and principlesanaghabharat01
This SlideShare presentation provides a comprehensive overview of the Declaration of Helsinki, a foundational document outlining ethical guidelines for conducting medical research involving human subjects.
- Video recording of this lecture in English language: https://youtu.be/Pt1nA32sdHQ
- Video recording of this lecture in Arabic language: https://youtu.be/uFdc9F0rlP0
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
8 Surprising Reasons To Meditate 40 Minutes A Day That Can Change Your Life.pptxHolistified Wellness
We’re talking about Vedic Meditation, a form of meditation that has been around for at least 5,000 years. Back then, the people who lived in the Indus Valley, now known as India and Pakistan, practised meditation as a fundamental part of daily life. This knowledge that has given us yoga and Ayurveda, was known as Veda, hence the name Vedic. And though there are some written records, the practice has been passed down verbally from generation to generation.
2. • As is well known, the heart contains specialized conduction
system consisting of excitatory myocytes that regulate the
rate and rhythm of cardiac contraction and are essential for
normal cardiac function. This system is influenced by direct
neural inputs (e.g., vagal stimulation), adrenergic agents
(e.g., epinephrine [adrenaline]), hypoxia, and potassium
concentrations (i.e., hyperkalemia can block signal
transmission altogether).
3. • The components of the conduction system include (1) the
sinoatrial (SA) node pacemaker (located at the junction of
the right atrial appendage and superior vena cava), (2) the
atrioventricular (AV) node (located in the right atrium along
the atrial septum), (3) the bundle of His, connecting the right
atrium to the ventricular septum, and the subsequent
divisions into (4) the right and left bundle branches that
stimulate their respective ventricles.
4. • Abnormalities in myocardial conduction can be sustained or
sporadic (paroxysmal). Aberrant rhythms can be initiated
anywhere in the conduction system, from the SA node down
to the level of an individual myocyte; they are typically
designated as originating from the atrium (supraventricular)
or within the ventricular myocardium.
5. • Arrhythmias can manifest as tachycardia (fast heart rate),
bradycardia (slow heart rate), an irregular rhythm with
normal ventricular contraction, chaotic depolarization
without functional ventricular contraction (ventricular
fibrillation), or no electrical activity at all (asystole). Patients
may be unaware of a rhythm disorder or may note a “racing
heart” or palpitations; loss of adequate cardiac output due
to sustained arrhythmia can produce lightheadedness (near
syncope), loss of consciousness (syncope), or sudden cardiac
death .
6. • Ischemic injury is the most common cause of rhythm
disorders, because of direct damage or due to the dilation of
heart chambers with consequent alteration in conduction
system firing.