anti epileptics drugs is a part of pharmacology. the topic contain information regarding epilepsy and their drug. ppt is short and simple and have correct information
Anticonvulsants are a diverse group of pharmacological agents used in the treatment of epileptic seizures. Charles Locock commented in the Lancet on his use of potassium bromide in 15 cases of "hysterical" epilepsy in young women. The next development was the serendipitous discovery of the anticonvulsant properties of phenobarbital by Alfred Hauptmann in 1912. This predated by more than 20 years the screening of potential therapeutic agents against "electrical seizures" in cats by Houston Merritt and Tracy Putnam. The result was the launching of phenytoin in 1938. Next came primidone, ethosuximide, carbamazepine and valproic acid, all of which can be regarded as first generation antiepileptic drugs (AEDs). Shortly after their synthesis, the benzodiazepines were rapidly recognised as having anticonvulsant activity. The modern era focused on the systematic screening of many thousands of compounds against rodent seizure models under the Anticonvulsant Drug Development Program in the US. This resulted in the global licensing, in chronological order, of vigabatrin, zonisamide, oxcarbazepine, lamotrigine, felbamate, gabapentin, topiramate, tiagabine, levetiracetam, pregabalin and lacosamide.
anti epileptics drugs is a part of pharmacology. the topic contain information regarding epilepsy and their drug. ppt is short and simple and have correct information
Anticonvulsants are a diverse group of pharmacological agents used in the treatment of epileptic seizures. Charles Locock commented in the Lancet on his use of potassium bromide in 15 cases of "hysterical" epilepsy in young women. The next development was the serendipitous discovery of the anticonvulsant properties of phenobarbital by Alfred Hauptmann in 1912. This predated by more than 20 years the screening of potential therapeutic agents against "electrical seizures" in cats by Houston Merritt and Tracy Putnam. The result was the launching of phenytoin in 1938. Next came primidone, ethosuximide, carbamazepine and valproic acid, all of which can be regarded as first generation antiepileptic drugs (AEDs). Shortly after their synthesis, the benzodiazepines were rapidly recognised as having anticonvulsant activity. The modern era focused on the systematic screening of many thousands of compounds against rodent seizure models under the Anticonvulsant Drug Development Program in the US. This resulted in the global licensing, in chronological order, of vigabatrin, zonisamide, oxcarbazepine, lamotrigine, felbamate, gabapentin, topiramate, tiagabine, levetiracetam, pregabalin and lacosamide.
This interesting ppt deals with the Pharmacology of Antiepileptic drugs and the treatment of different types of seizures with beautiful illustrations....
Epilepsy and antiepileptics. Dr.Ashok Kumar Batham,M.D.,DrAshok Batham
This presentation provides relevant description and classification of epilepsy with easy-to-remember mechanism-based and chemistry-based classifications of Anti-epileptic Drugs (AEDs). General features and salient details of all the Anti-epileptic Drugs (AEDs) are provided that can be used as short-notes. Hopefully, this presentation would be useful to students of medicine, pharmacology, pharmacy, clinical pharmacy, and representatives of pharmaceutical companies.
Please find the power point on Choice of Antiepileptic drugs. I tried to present it on understandable way and all the contents are reviewed by experts and from very reliable references. Thank you
Epilepsy is the disease which has prevalence in India more than 1 %
Of population. This is topic of research for young medicine practioner and pharmacist.
1. Epilepsy, Seizure, Convulsion
2. Causes & Pathophysiology of Epilepsy
3. Classification and Choice of antiepileptics
4. Antiepileptics Mechanism of action of , Adverse effects, Drug interactions, General guidelines for use.
5. Recommendation to Antiepileptics and pregnancy according to RCOG 2016, SIGN 2017 guidelines
6. Treatment of status epilepticus according to American Epilepsy Society 2016 guidelines
This interesting ppt deals with the Pharmacology of Antiepileptic drugs and the treatment of different types of seizures with beautiful illustrations....
Epilepsy and antiepileptics. Dr.Ashok Kumar Batham,M.D.,DrAshok Batham
This presentation provides relevant description and classification of epilepsy with easy-to-remember mechanism-based and chemistry-based classifications of Anti-epileptic Drugs (AEDs). General features and salient details of all the Anti-epileptic Drugs (AEDs) are provided that can be used as short-notes. Hopefully, this presentation would be useful to students of medicine, pharmacology, pharmacy, clinical pharmacy, and representatives of pharmaceutical companies.
Please find the power point on Choice of Antiepileptic drugs. I tried to present it on understandable way and all the contents are reviewed by experts and from very reliable references. Thank you
Epilepsy is the disease which has prevalence in India more than 1 %
Of population. This is topic of research for young medicine practioner and pharmacist.
1. Epilepsy, Seizure, Convulsion
2. Causes & Pathophysiology of Epilepsy
3. Classification and Choice of antiepileptics
4. Antiepileptics Mechanism of action of , Adverse effects, Drug interactions, General guidelines for use.
5. Recommendation to Antiepileptics and pregnancy according to RCOG 2016, SIGN 2017 guidelines
6. Treatment of status epilepticus according to American Epilepsy Society 2016 guidelines
Apa workshop 05.20.15 what every psychiatrist needs to know about epilepsygbaslet
Dear colleagues,
Thank you for attending our workshop "What Every Psychiatrist Needs to Know About Epilepsy" on May 20, 2015 during the American Psychiatric Association meeting in Toronto.
As promised, we are sending here a copy of our slides. Please do not hesitate to contact the authors of each presentation should you have any questions. Here are our emails:
Dr. Elia Pestana Knight: pestane@ccf.org
Dr. Jana Jones: jejones@neurology.wisc.edu
Dr. Tatiana Falcone: falcont1@ccf.org
Dr. Gaston Baslet: gbaslet@partners.org
Hope you all made it home safe.
Sincerely,
Gaston Baslet, M.D.
New York Times bestselling author and Cleveland Clinic Pyshologist, Dr Susan Albers specializes in eating issues, weight loss, body image concerns and mindfulness.
Susan Albers-Bowling, Psy.D., is a clinical psychologist at the Cleveland Clinic and a New York Times bestselling author who specializes in eating issues, weight loss, body image concerns and mindfulness.
Why Choose Cleveland Clinic's Epilepsy Center?
Cleveland Clinic has one of the largest, most comprehensive programs in the world for the evaluation, medical and surgical treatment of epilepsy in children and adults. Our goal is to help you or your loved one manage this disease in order to enjoy a fuller, more productive life.
Team Approach
Our team of dedicated physicians, healthcare professionals and support staff participate in the evaluation and treatment of our epilepsy patients who come here from across the country and around the world.
Pediatric and adult neurologists; neurosurgeons; neuroradiologists; nuclear medicine physicians; nurse specialists; pharmacologists; physical, occupational and speech therapists; dietitians; neuropsychologists and psychiatrists; educational counselors and social workers; and an array of scientists and technologists all work together to offer individualized care to adults and children.
Thank you for joining this video chat about Children with Seizures and Modern Treatments with Elaine Wyllie, MD
Patients with epilepsy may experience seizures that are difficult to control despite diligent treatment with medications. When medications are not keeping the symptoms at bay, or when they are causing undesirable side effects, then sometimes more advanced solutions are the answer. At Cleveland Clinic’s Epilepsy Center, we strongly believe the burden of epilepsy extends beyond a “seizure count”. Join Cleveland Clinic pediatric epileptologist Elaine Wyllie, MD, to explore the most modern treatments for children with seizures.
These lecture slides, by Dr Sidra Arshad, offer a quick overview of the physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar lead (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
6. Describe the flow of current around the heart during the cardiac cycle
7. Discuss the placement and polarity of the leads of electrocardiograph
8. Describe the normal electrocardiograms recorded from the limb leads and explain the physiological basis of the different records that are obtained
9. Define mean electrical vector (axis) of the heart and give the normal range
10. Define the mean QRS vector
11. Describe the axes of leads (hexagonal reference system)
12. Comprehend the vectorial analysis of the normal ECG
13. Determine the mean electrical axis of the ventricular QRS and appreciate the mean axis deviation
14. Explain the concepts of current of injury, J point, and their significance
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. Chapter 3, Cardiology Explained, https://www.ncbi.nlm.nih.gov/books/NBK2214/
7. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Integrating Ayurveda into Parkinson’s Management: A Holistic ApproachAyurveda ForAll
Explore the benefits of combining Ayurveda with conventional Parkinson's treatments. Learn how a holistic approach can manage symptoms, enhance well-being, and balance body energies. Discover the steps to safely integrate Ayurvedic practices into your Parkinson’s care plan, including expert guidance on diet, herbal remedies, and lifestyle modifications.
Adv. biopharm. APPLICATION OF PHARMACOKINETICS : TARGETED DRUG DELIVERY SYSTEMSAkankshaAshtankar
MIP 201T & MPH 202T
ADVANCED BIOPHARMACEUTICS & PHARMACOKINETICS : UNIT 5
APPLICATION OF PHARMACOKINETICS : TARGETED DRUG DELIVERY SYSTEMS By - AKANKSHA ASHTANKAR
share - Lions, tigers, AI and health misinformation, oh my!.pptxTina Purnat
• Pitfalls and pivots needed to use AI effectively in public health
• Evidence-based strategies to address health misinformation effectively
• Building trust with communities online and offline
• Equipping health professionals to address questions, concerns and health misinformation
• Assessing risk and mitigating harm from adverse health narratives in communities, health workforce and health system
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
Basavarajeeyam is a Sreshta Sangraha grantha (Compiled book ), written by Neelkanta kotturu Basavaraja Virachita. It contains 25 Prakaranas, First 24 Chapters related to Rogas& 25th to Rasadravyas.
Muktapishti is a traditional Ayurvedic preparation made from Shoditha Mukta (Purified Pearl), is believed to help regulate thyroid function and reduce symptoms of hyperthyroidism due to its cooling and balancing properties. Clinical evidence on its efficacy remains limited, necessitating further research to validate its therapeutic benefits.
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists Saeid Safari
Preoperative Management of Patients on GLP-1 Receptor Agonists like Ozempic and Semiglutide
ASA GUIDELINE
NYSORA Guideline
2 Case Reports of Gastric Ultrasound
Ozempic: Preoperative Management of Patients on GLP-1 Receptor Agonists
Anti seizure and rescue medications.updated 8.7.2014
1. Anti-Seizure and RescueAnti-Seizure and Rescue
MedicationsMedications
Elia M Pestana Knight, MDElia M Pestana Knight, MD
Cleveland ClinicCleveland Clinic
Epilepsy CenterEpilepsy Center
2. Topics for this webinarTopics for this webinar
• What are antiepileptic drugs?What are antiepileptic drugs?
• Historical development of the drugsHistorical development of the drugs
• How doctors select the drugs to treatHow doctors select the drugs to treat
the seizures?the seizures?
• Common side effectCommon side effect
• Considerations in special populationsConsiderations in special populations
• Drug interactionsDrug interactions
• Seizure rescue medicationsSeizure rescue medications
5. Anti-seizure medicationsAnti-seizure medications
• Drugs that decrease the frequencyDrugs that decrease the frequency
and/or severity of seizures in peopleand/or severity of seizures in people
with epilepsywith epilepsy
• Treat the symptom of seizures, not theTreat the symptom of seizures, not the
underlying epileptic conditionunderlying epileptic condition
• They don’t change the course of theThey don’t change the course of the
diseasedisease
6. Antiepileptic drugsAntiepileptic drugs
• Drugs that prevent the development of recurrentDrugs that prevent the development of recurrent
seizures in people at riskseizures in people at risk
• Drugs that reduce seizure frequency and severityDrugs that reduce seizure frequency and severity
outlasting the treatment periodoutlasting the treatment period
- Example: after head trauma, stroke, brainExample: after head trauma, stroke, brain
tumors, etctumors, etc
• Animal studies suggest that Levetiracetam andAnimal studies suggest that Levetiracetam and
Ethosuximide are potential antiepileptic drugs BUTEthosuximide are potential antiepileptic drugs BUT
there are no studies in humans to support thisthere are no studies in humans to support this
7. Use of anti-seizureUse of anti-seizure
medicationsmedications
• EpilepsyEpilepsy
• Pain treatmentPain treatment
- Neuralgias, neuropathic pain, etcNeuralgias, neuropathic pain, etc
• Migraine prophylaxisMigraine prophylaxis
• Treatment of bipolar disorder, moodTreatment of bipolar disorder, mood
disorder and anxietydisorder and anxiety
9. How anti-seizure medicationsHow anti-seizure medications
work?work?
• Suppressing the rapid and excessiveSuppressing the rapid and excessive
firing of neurons during seizuresfiring of neurons during seizures
• Preventing the spread of seizuresPreventing the spread of seizures
within the brainwithin the brain
14. Other treatments, diet andOther treatments, diet and
devicesdevices
• Sex hormones: ProgesteroneSex hormones: Progesterone
• Diets: Ketogenic diet, Low glycemicDiets: Ketogenic diet, Low glycemic
diet, modified Atkins dietdiet, modified Atkins diet
• Devices: Vagus Nerve Stimulator (VNS)Devices: Vagus Nerve Stimulator (VNS)
and Neuropaceand Neuropace
• Epilepsy SurgeryEpilepsy Surgery
15. HOW DOCTORS SELECT THEHOW DOCTORS SELECT THE
DRUGS TO TREAT THEDRUGS TO TREAT THE
SEIZURES?SEIZURES?
16. Goals of treatmentGoals of treatment
• Minimal number of seizuresMinimal number of seizures
• Minimal adverse events or side effectsMinimal adverse events or side effects
• Best quality of life possibleBest quality of life possible
17. Selecting the AEDsSelecting the AEDs
• Seizure types or syndromeSeizure types or syndrome
• Age of the patient (oral suspension,Age of the patient (oral suspension,
chewable, sprinkles for children)chewable, sprinkles for children)
• Dosing scheduleDosing schedule
• Comorbidities and other medicalComorbidities and other medical
conditionsconditions
• Potential side effects and toxicityPotential side effects and toxicity
• Interaction with other drugsInteraction with other drugs
19. Adverse EffectsAdverse Effects
Acute dose-related:Acute dose-related: reversiblereversible
IdiosyncraticIdiosyncratic
- Uncommon - rareUncommon - rare
- Potentially serious or life threateningPotentially serious or life threatening
Chronic:Chronic: reversibility and seriousnessreversibility and seriousness
varyvary
20. Acute, Dose-Related AdverseAcute, Dose-Related Adverse
Effects of AEDsEffects of AEDs
Neurologic/psychiatric:Neurologic/psychiatric: most commonmost common
• Sedation, fatigueSedation, fatigue
− All AEDs, except unusual with LTG and FBMAll AEDs, except unusual with LTG and FBM
• Unsteadiness, uncoordination, dizzinessUnsteadiness, uncoordination, dizziness
− Mainly traditional AEDsMainly traditional AEDs
− May be sign of toxicity with many AEDsMay be sign of toxicity with many AEDs
• Tremor - valproic acidTremor - valproic acid
21. Acute, Dose-Related AdverseAcute, Dose-Related Adverse
Effects of AEDsEffects of AEDs (cont.)(cont.)
• Paresthesia (topiramate, zonisamide)Paresthesia (topiramate, zonisamide)
• Double vision , blurred vision, visual distortionDouble vision , blurred vision, visual distortion
(carbamazepine, lamotrigine)(carbamazepine, lamotrigine)
• Mental/motor slowing or impairment (topiramate at higherMental/motor slowing or impairment (topiramate at higher
doses)doses)
• Mood or behavioral changes (levetiracetam)Mood or behavioral changes (levetiracetam)
• Changes in libido or sexual function (carbamazepine,Changes in libido or sexual function (carbamazepine,
phenytoin, phenobarbital)phenytoin, phenobarbital)
22. Acute, Dose-Related AdverseAcute, Dose-Related Adverse
Effects of AEDs (cont.)Effects of AEDs (cont.)
GastrointestinalGastrointestinal (nausea, heartburn)(nausea, heartburn)
Mild to moderate laboratoryMild to moderate laboratory
changeschanges
• Hyponatremia: carbamazepine, oxcarbazepineHyponatremia: carbamazepine, oxcarbazepine
• Increases in ALT or ASTIncreases in ALT or AST
• LeukopeniaLeukopenia
• ThrombocytopeniaThrombocytopenia
P-Slide 22
29. The ChildrenThe Children
NeonateNeonate - often lower per kg doses- often lower per kg doses
- Low protein bindingLow protein binding
- Low metabolic rateLow metabolic rate
ChildrenChildren - higher, more frequent doses- higher, more frequent doses
- Faster metabolismFaster metabolism
- Better renal clearanceBetter renal clearance
30. The TeenagersThe Teenagers
• FemaleFemale
- Avoid some medications with potentialAvoid some medications with potential
teratogenesis (damage to the fetus)teratogenesis (damage to the fetus)
- Some AEDs reduce the efficacy of oralSome AEDs reduce the efficacy of oral
contraceptive so other contraceptive methodscontraceptive so other contraceptive methods
should be consideredshould be considered
- Some drugs could have undesirable physicalSome drugs could have undesirable physical
effectseffects
• Both gendersBoth genders
- AEDs have a black label for depression andAEDs have a black label for depression and
suicidal ideation, so screening is importantsuicidal ideation, so screening is important
31. PregnancyPregnancy
Increased volume of distributionIncreased volume of distribution
Lower serum albuminLower serum albumin
Faster metabolismFaster metabolism
Higher dose, but probably less than predicted by totalHigher dose, but probably less than predicted by total
level (measure free level)level (measure free level)
Consider more frequent dosingConsider more frequent dosing
Return to pre-pregnancy conditions rapidly (within 2Return to pre-pregnancy conditions rapidly (within 2
weeks) after deliveryweeks) after delivery
Teratogenic effect of some medicationsTeratogenic effect of some medications
32. Metabolic Changes of AEDsMetabolic Changes of AEDs
Febrile IllnessesFebrile Illnesses
- ↑↑ metabolic rate and ↓ serum concentrationsmetabolic rate and ↓ serum concentrations
- ↑↑ serum proteins that can bind AEDs and ↓ free levels ofserum proteins that can bind AEDs and ↓ free levels of
AED serum concentrationsAED serum concentrations
Severe Hepatic DiseaseSevere Hepatic Disease
- Impairs metabolism and ↑ serum levels of AEDsImpairs metabolism and ↑ serum levels of AEDs
- ↓↓ serum proteins and ↑ free levels of AED serumserum proteins and ↑ free levels of AED serum
concentrationsconcentrations
- Often serum levels can be harder to predict in this situationOften serum levels can be harder to predict in this situation
33. Metabolic Changes of AEDsMetabolic Changes of AEDs
Renal DiseaseRenal Disease
- ↓↓ the elimination of some AEDsthe elimination of some AEDs
- Gabapentin, pregabalin, levetiracetamGabapentin, pregabalin, levetiracetam
Chronic Renal DiseaseChronic Renal Disease
- ↑↑ protein loss and ↑ free fraction of highly protein boundprotein loss and ↑ free fraction of highly protein bound
AEDsAEDs
- It may be helpful to give smaller doses more frequently to ↓It may be helpful to give smaller doses more frequently to ↓
adverse effectsadverse effects
- Phenytoin, valproic acid, tiagabinePhenytoin, valproic acid, tiagabine
P-Slide 33
35. Pharmacokinetic InteractionsPharmacokinetic Interactions
Be aware that drug interactions mayBe aware that drug interactions may
occur when there is the:occur when there is the:
- Addition of a new medication when you are taking aAddition of a new medication when you are taking a
medication that can change the liver metabolism of othermedication that can change the liver metabolism of other
medicationsmedications
- AdditionAddition of another medication that can change theof another medication that can change the
liver metabolism of other medicationsliver metabolism of other medications to an existingto an existing
medication regimen.medication regimen.
- Removal ofRemoval of a medication that can change the livera medication that can change the liver
metabolism of other medicationsmetabolism of other medications from chronicfrom chronic
medication regimen.medication regimen.
36. Hepatic Drug Metabolizing EnzymesHepatic Drug Metabolizing Enzymes
and Specific AED Interactionsand Specific AED Interactions
Phenytoin:Phenytoin: CYP2C9/CYP2C19CYP2C9/CYP2C19
- Inhibitors: valproate, ticlopidine, fluoxetine, topiramate,Inhibitors: valproate, ticlopidine, fluoxetine, topiramate,
fluconazolefluconazole
Carbamazepine:Carbamazepine: CYP3A4/CYP2C8/CYP1A2CYP3A4/CYP2C8/CYP1A2
- Inhibitors: ketoconazole, fluconazole, erythromycin,Inhibitors: ketoconazole, fluconazole, erythromycin,
diltiazemdiltiazem
Lamotrigine:Lamotrigine: UGT 1A4UGT 1A4
- Inhibitor: valproateInhibitor: valproate
• Important note about oral contraceptives (OCPs):Important note about oral contraceptives (OCPs):
- OCP efficacy is decreased by inducers, including:OCP efficacy is decreased by inducers, including:
phenytoin, phenobarbital, primidone, carbamazepine; andphenytoin, phenobarbital, primidone, carbamazepine; and
higher doses of topiramate and oxcarbazepinehigher doses of topiramate and oxcarbazepine
- OCPs and pregnancy significantly decrease serum levels ofOCPs and pregnancy significantly decrease serum levels of
lamotrigine.lamotrigine.
37. AEDs and Drug InteractionsAEDs and Drug Interactions
AEDs that do not appear to be eitherAEDs that do not appear to be either
inducers or inhibitors of the CYPinducers or inhibitors of the CYP
system include:system include:
GabapentinGabapentin
LamotrigineLamotrigine
PregabalinPregabalin
TiagabineTiagabine
LevetiracetamLevetiracetam
ZonisamideZonisamide
P-Slide 37
40. Medicines for the treatment ofMedicines for the treatment of
acute seizuresacute seizures
• Intravenous (in hospital or paramediceIntravenous (in hospital or paramedice
use)use)
• Transmucosal (at home use)Transmucosal (at home use)
42. Take home pointsTake home points
• Anti-seizure medications are prescribedAnti-seizure medications are prescribed
taking into account a number of factorstaking into account a number of factors
• Always disclose to your physician whatAlways disclose to your physician what
other medications or supplements areother medications or supplements are
you taking as these can cause drugyou taking as these can cause drug
interactionsinteractions
• Seizure rescue plan is very importantSeizure rescue plan is very important
for all people with epilepsyfor all people with epilepsy