This document provides an overview of the anatomy of the nose, including both the external nose and nasal cavity. It describes the skin, muscles, bones, and cartilages that make up the external nose. It then details the structures within the nasal cavity, including the nasal septum, lateral wall with its turbinates and meatuses, roof and floor. Throughout, it emphasizes the applied clinical significance of various anatomical structures and relationships as they relate to nasal function, trauma, and surgery.
surgical anatomy of nose is a humble attempt to make the anatomy of nose simpler and easy for medical students and fellow physicians. at the end of the presentation the students will be able to identify all the structures.
surgical anatomy of nose is a humble attempt to make the anatomy of nose simpler and easy for medical students and fellow physicians. at the end of the presentation the students will be able to identify all the structures.
Functions of Nose
Respiration
Heat exchange
Humidification
Filtration
Nasal resistance
Nasal fluids & cilliary function
Nasal neurovascular reflexes
Voice modification
Olfaction
Respiration
Inspiration
Air current passes along mid-portion of nasal cavity in lamellar flow
Expiration
Resistance of nasal valve & turbinates leads to formation of eddy current in expired air
Results in awareness of breathing and ventilation of paranasal sinus
Air Conditioning
Filtration : Particles > 5 μm in inspired air are trapped by nasal vibrissae
Temperature control
Heat exchange between blood in cavernous venous sinusoids of turbinates and inspired air (radiation)
Humidification
Secretions of nasal & PNS mucosa; for better ciliary function
Muco-ciliary blanket : traps pathogens in inspired air > 0.5 μm & transports them to nasopharynx for swallowing
Sneezing : protects against irritants
Lysozyme : kills bacteria & viruses
Immunoglobulins A & E : protection against bacteria
Interferon : for protection against virus
Preface
If not coincidence, at least it was in the same decade when endoscopic sinus surgery and computed tomography were introduced to Otolaryngologists, which have changed the approach to sinonasal problems dramatically. Probably, there are no such coincidences in the history of medicine where two new modalities of approaches appeared at the same time, to deal with the same problem and complement each other while doing so. The asset of improved visualization and magnification, available through endoscopes, has revolutionized the understanding of the pathophysiology of sinusitis, and resulted in better appreciation of the anatomy of the paranasal sinuses. However, non-invasive diagnostic endoscopy has its limits, and the deeper structures cannot be evaluated by endoscopy alone . Computed tomography, which has an ability to optimally display bone, soft tissue and air simultaneously, can not only complement endoscopic examination, it can provide a surgical road map delineating the anatomy, defining the obstructing lesions, and noting anatomic variations that may predispose to operative complications.
Computed tomography has scored over plain radiographs and polytomographs as an imaging modality in this area. Even though surpassing CT's capacity to image soft tissue, MRI is less suitable as an imaging modality for evaluation of this area because of the similar signal intensities for bone and air.
CT scanning has become imaging modality of choice and the cooperation required between the Radiologist and the Surgeon is mandatory for both evaluation and treatment of paranasal disorders. It is of paramount importance on the part of the Otolaryngologists to understand interpretation of CT films, Radiological anatomy of the paranasal sinuses, Anatomical variations and the pathology to complement the endoscopy findings for initial screening, surgical planning, reduce postoperative complications and to provide better results. This Presentations is prepared to help Otolaryngology colleagues to learn the Imaging/radiological aspects required for endoscopic sinus surgery.
Functions of Nose
Respiration
Heat exchange
Humidification
Filtration
Nasal resistance
Nasal fluids & cilliary function
Nasal neurovascular reflexes
Voice modification
Olfaction
Respiration
Inspiration
Air current passes along mid-portion of nasal cavity in lamellar flow
Expiration
Resistance of nasal valve & turbinates leads to formation of eddy current in expired air
Results in awareness of breathing and ventilation of paranasal sinus
Air Conditioning
Filtration : Particles > 5 μm in inspired air are trapped by nasal vibrissae
Temperature control
Heat exchange between blood in cavernous venous sinusoids of turbinates and inspired air (radiation)
Humidification
Secretions of nasal & PNS mucosa; for better ciliary function
Muco-ciliary blanket : traps pathogens in inspired air > 0.5 μm & transports them to nasopharynx for swallowing
Sneezing : protects against irritants
Lysozyme : kills bacteria & viruses
Immunoglobulins A & E : protection against bacteria
Interferon : for protection against virus
Preface
If not coincidence, at least it was in the same decade when endoscopic sinus surgery and computed tomography were introduced to Otolaryngologists, which have changed the approach to sinonasal problems dramatically. Probably, there are no such coincidences in the history of medicine where two new modalities of approaches appeared at the same time, to deal with the same problem and complement each other while doing so. The asset of improved visualization and magnification, available through endoscopes, has revolutionized the understanding of the pathophysiology of sinusitis, and resulted in better appreciation of the anatomy of the paranasal sinuses. However, non-invasive diagnostic endoscopy has its limits, and the deeper structures cannot be evaluated by endoscopy alone . Computed tomography, which has an ability to optimally display bone, soft tissue and air simultaneously, can not only complement endoscopic examination, it can provide a surgical road map delineating the anatomy, defining the obstructing lesions, and noting anatomic variations that may predispose to operative complications.
Computed tomography has scored over plain radiographs and polytomographs as an imaging modality in this area. Even though surpassing CT's capacity to image soft tissue, MRI is less suitable as an imaging modality for evaluation of this area because of the similar signal intensities for bone and air.
CT scanning has become imaging modality of choice and the cooperation required between the Radiologist and the Surgeon is mandatory for both evaluation and treatment of paranasal disorders. It is of paramount importance on the part of the Otolaryngologists to understand interpretation of CT films, Radiological anatomy of the paranasal sinuses, Anatomical variations and the pathology to complement the endoscopy findings for initial screening, surgical planning, reduce postoperative complications and to provide better results. This Presentations is prepared to help Otolaryngology colleagues to learn the Imaging/radiological aspects required for endoscopic sinus surgery.
1. Classification of Bones of the Head & Neck
2. Bones of the Viscerocranium
3. Bones of the Neurocranium
4. The Auditory Ossicles
5. The Hyoid Bone
6. The Cervical Vertebrae
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Ve...kevinkariuki227
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
TEST BANK for Operations Management, 14th Edition by William J. Stevenson, Verified Chapters 1 - 19, Complete Newest Version.pdf
MANAGEMENT OF ATRIOVENTRICULAR CONDUCTION BLOCK.pdfJim Jacob Roy
Cardiac conduction defects can occur due to various causes.
Atrioventricular conduction blocks ( AV blocks ) are classified into 3 types.
This document describes the acute management of AV block.
Explore natural remedies for syphilis treatment in Singapore. Discover alternative therapies, herbal remedies, and lifestyle changes that may complement conventional treatments. Learn about holistic approaches to managing syphilis symptoms and supporting overall health.
Lung Cancer: Artificial Intelligence, Synergetics, Complex System Analysis, S...Oleg Kshivets
RESULTS: Overall life span (LS) was 2252.1±1742.5 days and cumulative 5-year survival (5YS) reached 73.2%, 10 years – 64.8%, 20 years – 42.5%. 513 LCP lived more than 5 years (LS=3124.6±1525.6 days), 148 LCP – more than 10 years (LS=5054.4±1504.1 days).199 LCP died because of LC (LS=562.7±374.5 days). 5YS of LCP after bi/lobectomies was significantly superior in comparison with LCP after pneumonectomies (78.1% vs.63.7%, P=0.00001 by log-rank test). AT significantly improved 5YS (66.3% vs. 34.8%) (P=0.00000 by log-rank test) only for LCP with N1-2. Cox modeling displayed that 5YS of LCP significantly depended on: phase transition (PT) early-invasive LC in terms of synergetics, PT N0—N12, cell ratio factors (ratio between cancer cells- CC and blood cells subpopulations), G1-3, histology, glucose, AT, blood cell circuit, prothrombin index, heparin tolerance, recalcification time (P=0.000-0.038). Neural networks, genetic algorithm selection and bootstrap simulation revealed relationships between 5YS and PT early-invasive LC (rank=1), PT N0—N12 (rank=2), thrombocytes/CC (3), erythrocytes/CC (4), eosinophils/CC (5), healthy cells/CC (6), lymphocytes/CC (7), segmented neutrophils/CC (8), stick neutrophils/CC (9), monocytes/CC (10); leucocytes/CC (11). Correct prediction of 5YS was 100% by neural networks computing (area under ROC curve=1.0; error=0.0).
CONCLUSIONS: 5YS of LCP after radical procedures significantly depended on: 1) PT early-invasive cancer; 2) PT N0--N12; 3) cell ratio factors; 4) blood cell circuit; 5) biochemical factors; 6) hemostasis system; 7) AT; 8) LC characteristics; 9) LC cell dynamics; 10) surgery type: lobectomy/pneumonectomy; 11) anthropometric data. Optimal diagnosis and treatment strategies for LC are: 1) screening and early detection of LC; 2) availability of experienced thoracic surgeons because of complexity of radical procedures; 3) aggressive en block surgery and adequate lymph node dissection for completeness; 4) precise prediction; 5) adjuvant chemoimmunoradiotherapy for LCP with unfavorable prognosis.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
New Directions in Targeted Therapeutic Approaches for Older Adults With Mantl...i3 Health
i3 Health is pleased to make the speaker slides from this activity available for use as a non-accredited self-study or teaching resource.
This slide deck presented by Dr. Kami Maddocks, Professor-Clinical in the Division of Hematology and
Associate Division Director for Ambulatory Operations
The Ohio State University Comprehensive Cancer Center, will provide insight into new directions in targeted therapeutic approaches for older adults with mantle cell lymphoma.
STATEMENT OF NEED
Mantle cell lymphoma (MCL) is a rare, aggressive B-cell non-Hodgkin lymphoma (NHL) accounting for 5% to 7% of all lymphomas. Its prognosis ranges from indolent disease that does not require treatment for years to very aggressive disease, which is associated with poor survival (Silkenstedt et al, 2021). Typically, MCL is diagnosed at advanced stage and in older patients who cannot tolerate intensive therapy (NCCN, 2022). Although recent advances have slightly increased remission rates, recurrence and relapse remain very common, leading to a median overall survival between 3 and 6 years (LLS, 2021). Though there are several effective options, progress is still needed towards establishing an accepted frontline approach for MCL (Castellino et al, 2022). Treatment selection and management of MCL are complicated by the heterogeneity of prognosis, advanced age and comorbidities of patients, and lack of an established standard approach for treatment, making it vital that clinicians be familiar with the latest research and advances in this area. In this activity chaired by Michael Wang, MD, Professor in the Department of Lymphoma & Myeloma at MD Anderson Cancer Center, expert faculty will discuss prognostic factors informing treatment, the promising results of recent trials in new therapeutic approaches, and the implications of treatment resistance in therapeutic selection for MCL.
Target Audience
Hematology/oncology fellows, attending faculty, and other health care professionals involved in the treatment of patients with mantle cell lymphoma (MCL).
Learning Objectives
1.) Identify clinical and biological prognostic factors that can guide treatment decision making for older adults with MCL
2.) Evaluate emerging data on targeted therapeutic approaches for treatment-naive and relapsed/refractory MCL and their applicability to older adults
3.) Assess mechanisms of resistance to targeted therapies for MCL and their implications for treatment selection
Pulmonary Thromboembolism - etilogy, types, medical- Surgical and nursing man...VarunMahajani
Disruption of blood supply to lung alveoli due to blockage of one or more pulmonary blood vessels is called as Pulmonary thromboembolism. In this presentation we will discuss its causes, types and its management in depth.
The prostate is an exocrine gland of the male mammalian reproductive system
It is a walnut-sized gland that forms part of the male reproductive system and is located in front of the rectum and just below the urinary bladder
Function is to store and secrete a clear, slightly alkaline fluid that constitutes 10-30% of the volume of the seminal fluid that along with the spermatozoa, constitutes semen
A healthy human prostate measures (4cm-vertical, by 3cm-horizontal, 2cm ant-post ).
It surrounds the urethra just below the urinary bladder. It has anterior, median, posterior and two lateral lobes
It’s work is regulated by androgens which are responsible for male sex characteristics
Generalised disease of the prostate due to hormonal derangement which leads to non malignant enlargement of the gland (increase in the number of epithelial cells and stromal tissue)to cause compression of the urethra leading to symptoms (LUTS
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
6. Skin and Soft Tissue
Upper/bony part Thin and loosely adherent
Tip / cartilaginous part Thick and strongly adherent
contains numerous sebaceous glands
7. Subcutaneous Tissue
The subcutaneous tissue of the
nose is made up of four layers:
1. Superficial fatty layer,
2. Fibromuscular layer (comprises
the nasal SMAS)
3. Deep fatty layer (contains the
neurovascular system)
4. Periosteum / perichondrium.
8. Applied !
During external approach
rhinoplasty dissection deep to the
third layer of the nose (deep fatty
layer) minimizes post-operative
scarring and retraction because the
neurovascular and SMAS structures
are preserved.
15. Upper Lateral Cartilage (ULC)
Triangular
Superiorly, the upper lateral
cartilages are overlapped by nasal
bones
Caudally, the upper lateral cartilage
curves under and articulates with the
lower lateral cartilages in the scroll
region.
Medially, the upper lateral cartilages
fuse with the septal cartilage
16. Upper Lateral Cartilage (ULC)
Inferiorly, the medial edge of the
cartilage is often free.
The lateral aspect of the upper
lateral cartilage is adherent to
fibrous tissue that approaches the
piriform aperture and, if present,
the sesamoid cartilages.
17. Upper Cartilaginous Vault
Keystone area: centre of support
of nasal roof
Scroll area: nasal tip support.
18. Upper Cartilaginous Vault
• Internal nasal valve
• The narrowest portion of the nasal
cavity
• The apex of the internal nasal valve
is approximately 10–15 degrees in
Caucasians and wider in non-
Caucasian populations.
Septum medially (1). Caudal edge of the upper
lateral cartilage (2) and head of the inferior
turbinate (3) laterally. Nasal floor inferiorly
Applied!
Changes in the relationship of any
structure within this space can cause
symptoms of nasal obstruction.
19. • Applied !
Violation of the support for the upper
lateral cartilages can cause cosmetic
and functional problems, including nasal
obstruction and the inverted-V
deformity.
20. Lower Cartilaginous Vault
Paired alar cartilages (lower lateral
cartilage)
Lower lateral cartilage
C-shaped cartilage
Parts…..medial, intermediate and
lateral crura
Angulations or ‘domes’ between
the medial and lateral crurae of the
LLCs are separated by a notch
palpable at the tip of the nose.
21. Lower Cartilaginous Vault
Tripod theory
Anderson has compared Lower Lateral
Cartilage to a tripod
1. Two lateral crura forming the cephalic
lateral legs,
2. The adjoining medial crura as the third,
caudal-medial leg.
25. Factors Affecting Shape & Position of
Nasal Tip
1. Lower lateral cartilages (major
support)
2. Ligaments and fibrous
attachments of nasal tip
structures
3. Thickness of overlying skin
26. Features of Nasal Tip
A pleasing appearing nasal tip will
usually have the following
components:
1. The basal width of the nose
should be approximately the same
as the intercanthal distance.
27. Features of Nasal Tip
2. Four landmarks should be highlighted
on the frontal view:
Supra-tip break,
Paired tip defining points (most anterior
projection of the tip cartilages)
The most inferior portion of the infra-tip
lobule.
These points should divide the tip
complex into two equal and opposite
equilateral triangles
28. Features of Nasal Tip
3. From the basal view, the base
should form an equilateral triangle
with rounded corners, side walls and
a lobule to nostril ratio of 1 : 2
29. Features of Nasal Tip
4. On lateral view, a straight line drawn from the naso-
frontal angle to the TDP should be 2–3 mm anterior
to the nasal dorsum in females. This should result in
a slight supra-tip break.
Generally, this is not necessary in males and one
should err on the side of a straight nasal dorsum to
prevent the nose from appearing feminine.
30. Columella
Extends between upper lip and tip
of the nose
Anteriorly- diverging crura form
an angle of 30 degrees for tip
formation.
Posteriorly - also diverge to
receive septal cartilage.
Shape of columella depends on size
and shape of medial crura.
32. Blood supply of external nose
1. Dorsal and external nasal branch
of ophthalmic artery
2. Infraorbital br. of maxillary artery
3. Angular and superior labial
branches of facial artery.
The venous networks do not parallel
the arterial supply. Drain into facial
and ophthalmic veins.
33. Applied!
The upper lip and nose are
considered the danger area of the
face because infections in this
region may be transmitted through
a valve-less venous system to the
cavernous sinus.
35. NASAL CAVITY
Communicates with the exterior
through naris or nostril
and with the nasopharynx through
posterior nasal aperture or the
choana.
Each nasal cavity consists of a
skin-lined portion—the vestibule
and a mucosa-lined portion, the
nasal cavity proper.
36. A. Vestibule
Anterior and inferior part of nasal cavity is
called vestibule.
Lined by skin and contains sebaceous
glands, hair follicles and the hair called
vibrissae.
Its upper limit on the lateral wall is marked
by limen nasi, located at the caudal border
of the lower lateral cartilage.
37. B. Nasal Cavity Proper
Each nasal cavity has a
• Lateral wall,
• Medial wall,
• Roof and
• Floor.
40. Medial Wall (Nasal Septum)
A. Bony septum
The posterosuperior vomer
The anterosuperior perpendicular
plate of the ethmoid .
Minor contributions
1. Nasal bones + the nasal spine of the
frontal bones.
2. The rostrum and crest of the sphenoid.
3. The nasal crests of the maxilla and
palatine bones.
41. Medial Wall (Nasal Septum)
B. Cartilaginous septum
Septal/quadrilateral cartilage.
Anterosuperior margin is connected
to internasal suture,
Distal end of its superior portion is
continuous with the ULC
The anteroinferior border is
connected to the medial crurae of the
major alar cartilage.
42. Medial Wall (Nasal Septum)
The posterosuperior border
joins the perpendicular plate of
the ethmoid,
Posteroinferior border is
attached to the vomer and
anterior to that to the nasal
crest and anterior nasal spine of
the maxilla.
43. Applied!
The inferior attachment sits within the
nasal crest of the maxilla and is
bounded by looser connective tissue.
This joint allows mobility of the septal
cartilage base during flexion thereby
reducing the risk of fracture or
dislocation with trauma.
44. Medial Wall (Nasal Septum)
Septal tail
A projection of the septal cartilage
called the sphenoidal process or
septal tail extends posteriorly between
the vomer and perpendicular plate of
the ethmoid.
The septal tail can serve as an
additional source of cartilage to
harvest especially during revision
rhinoplasty.
45. Applied!
Different studies* showed that a long SP, a
remnant cartilaginous tail of the nasal septum
resulting from delayed ossification of the nasal
septum, contributes to the worsening of septal
deviation.
*
1. Kim J, Kim SW, Kim SW, Cho JH, Park YJ. Role of the sphenoidal process of the septal cartilage in the development of septal deviation. Otolaryngol Head Neck Surg.
2012;146(1):151‐155. doi:10.1177/0194599811425000
2. Kim, J, Han, SH, Kim, SW. Clinical significance of the sphenoidal process of the cartilaginous nasal septum: a preliminary morphological evaluation. Clin Anat. 2010;23:265-
269.
46. Medial Wall (Nasal Septum)
C. Membranous septum
Lies b/w septal cartilage &
columella
Consists of vestibular skin with
intervening areolar tissue.
It acts as a shock absorber between the
columella and the cartilaginous septum,
softening blows to the lower nose.
47. Medial Wall (Nasal Septum)
D. Columellar septum.
It is formed of columella containing
the medial crura of alar cartilages
united together by fibrous tissue and
covered on either side by skin.
48. Medial Wall (Nasal Septum)
The nasal septal swell body
A widened region of the anterior nasal
septum located anterior to the middle
turbinate at the internal nasal valve.
Histological analysis of this tissue
demonstrates an increased amount of venous
sinusoids and fewer glandular elements
compared to adjacent septal mucosa.
49. Applied!
The high proportion of venous
sinusoids suggests the capacity to
alter nasal airflow in a similar
manner to the inferior turbinates.
50. Lateral Wall
Lateral wall (osteology)
Maxilla
Perpendicular plate of the
palatine bone;
Labyrinth of the ethmoid bone.
Turbinates
Three projections
Meatus
The space below the turbinate
51. Lateral Wall
Inferior turbinate and inferior
meatus
Independent bone
The nasolacrimal duct drains
into the inferior meatus
approximately 1 cm posterior to
the head of the inferior turbinate.
Guarded by mucosal valve
(Hasner’s valve)
52. Lateral Wall
Middle Turbinate.
A part of ethmoid bone.
Its attachment is not straight but in
an S-shaped manner.
53. LATERAL WALL
(Middle Meatus)
Middle meatus
Uncinate process
Hook-like structure
Running from AS to PI direction.
PS border runs parallel to anterior
border of bulla ethmoidalis; the gap
between the two is called hiatus
semilunaris
54. LATERAL WALL
(Middle Meatus)
The anteroinferior border is attached to
the lateral wall.
Posteroinferior end of uncinate process
is attached to inferior turbinate dividing
the membranous part of lower middle
meatus into anterior and posterior
fontanelle. The fontanel area is devoid of
bone and consists of membrane only and
leads into maxillary sinus when
perforated.
58. LATERAL WALL
(Middle Meatus)
Agger Nasi.
Anterior ethmoid air cells
Anterior to the attachment of
middle turbinate.
59. Applied!
An enlarged agger nasi cell may encroach on
frontal recess area, and cause mechanical
obstruction to frontal sinus drainage.
60. LATERAL WALL
(Middle Meatus)
Concha Bullosa
Pneumatization of middle turbinate
leads to an enlarged ballooned out
middle turbinate. (30%)
61. LATERAL WALL
(Middle Meatus)
Haller cells
Air cells situated in the roof of
maxillary sinus.
Applied!
Enlargement of Haller cells
encroaches on ethmoid infundibulum,
impeding draining of maxillary sinus.
62. LATERAL WALL
(Superior Meatus)
Superior Turbinate. (Part of
ethmoid bone)
Superior Meatus.
It is a space below the superior
turbinate.
Posterior ethmoid cells open into
it.
It is a pyramidal structure.
Its root is continuous with the forehead, and its free tip forms the apex.
Its base contains two ellipsoidal apertures, the external nares or nostrils, which open onto its inferior surface, separated by the nasal septum and columella.
Anterior sloping part of the roof is formed by nasal bones and the nasal spine of frontal bone.
posterior sloping part is formed by the body of sphenoid bone. here you can see the opening of sphenoid sinus
middle horizontal part is formed by the cribriform plate of ethmoid through which the olfactory nerves enter the nasal cavity and there is a separate anterior foramen which transmits the anterior ethmoidal nerve and vessels..
It is formed by palatine process of the maxilla in its anterior three-fourths and
horizontal part of the palatine bone in its posterior one-fourth.
Approximately 12 mm behind the anterior aspect of the nasal floor is a slight depression which corresponds to the incisive canal.
The incisive canal contains terminal branches of the nasopalatine nerve and greater palatine artery.
The space limited medially by the uncinate process and frontal process of maxilla and sometimes lacrimal bone, and laterally by the lamina papyracea is called infundibulum.
Natural ostium of the maxillary sinus is situated in the lower part of infundibulum.
It can hinder the drainage of adjacent sinus leading to recurrent sinusitis.