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Pain 
Not a disease or curse 
Protective mechanism
Dr. Shrikant Gore 
M.S. Orthopaedics 
• Professor and Head 
• Department of Orthopaedics 
• Government Medical College 
• Latur – 413512 
• E-mail – laturmedical@gmail,com
• Unpleasant sensation felt, by a person, to any injury 
to the body 
• Mechanical - pinch, prick, cut, stretch, crush etc. 
• Chemical - external & internal (Bradykinines, 
Serotonins, prostaglandins, Bacterial toxins) 
• Thermal, electrical 
• Psychological 
Definition
• It initiates body’s protective response to injury 
• Minimizing the damage and helps in repairs by 
initiating healing process 
• Pain is essential to maintain health and repair 
damage 
• Pain needs to be treated as an emergency to 
protect body from damage by removing the cause 
of pain 
Protective
• Stimulus 
• Sensory receptors at nerve endings 
• Receptors are type specific and their 
concentration is area specific, depending on the 
the need of the area, e.g. maximum 
concentration of touch receptors at finger tips 
minimum touch receptors on visceral organs 
Physiology
• On stimulation of receptor biochemical reaction 
sets in generating electrical impulse with injury 
specific modality, Carried through nerves, spinal 
cord. 
• Spinal reflex is set in and the part of body 
subjected to injury is withdrawn away from the 
injury source to avoid damage 
Pathway
• The impulse is carried further to Sensory cortex 
representing the area of stimulus 
• The sensory cortex analyses this modality to 
recognize the specific injury type resulting in 
perfect location of the area of injury and the type 
of injury 
• It sends suppressive signals to spinal cord to 
reduce the severity of further similar impulse 
Perception
• Nerve ending 
• Peripheral Nerves 
• Nerves 
• Nerve root 
• Spinal cord 
• Basal Ganglion 
• Neuropathies 
• Stretching 
• Compression, 
• Girdle pain 
• Involuntary movements 
Pathological Sites
• Cerebellum 
• Cerebrum 
• Psyche 
• Autonomous n. 
• Ataxia, Balance 
• Altered, lost, 
• Pain without stimulus 
• Causalgia 
Pathological Sites
• Carry electrical current generated at sensory 
receptor 
• If the nerve is affected by inflammation or 
pressure the modality(frequency) of electrical 
current changes on its way to brain changing the 
perception of sensation (Altered sensation) 
• Touch is perceived as Tingling, Burning, 
Numbness, pricking etc. due to alteration of the 
electrical impulse 
Peripheral Nerves
• Nutritional - B-complex Vitamins 
• Metabolic - Diabetes mellitus 
• Toxicity - Lead 
• Infections - Leprosy, viral 
• Drugs - Alcohol, 
Peripheral neuropathies
• Throbbing 
• Radiating 
• Referred 
• Shifting 
• Phantom 
• Causalgia 
Abnormal Types
• Pulsatile pain 
• Spike of pain coming at regular intervals 
• Aneurisms Aortic - stretching of outer layer of vascular 
wall 
Berry’s – increased intra cranial pressure with 
each pulsation 
Migraine dilated intracranial vessels increased blood with each 
pulsation increases intracranial pressure 
• Abscess Inflamed abscess wall and surrounding tissue 
with hyperactive nerve endings getting stretched and 
stimulated with every pulsation 
Throbbing pain
• Nerves, Nerve roots - Compression, Stretching 
• In addition to altered sensation 
• Affected nerve becomes painful 
• The pain radiating along the course of the nerve 
Radiating Pain
• Pathology at one site, pain felt at other site 
• Cerebral cortex unable to locate the site of pain. 
• Commonly seen in pathologies affecting deeper 
structures Heart, appendicitis, Hip joint 
• Deeper structures less likely to get injured by 
physical trauma have very low number of pain 
receptors. 
Referred Pain
• One single nerve supplies multiple organs 
• The pain is felt at the organ having better number 
of sensory receptors , supplied by same nerve 
root 
• It gets distributed to larger area of brain making 
it difficult for pin point location 
Referred Pain
• Pain initially felt at one site disappears and is felt 
at other site 
• Referred pain with progression of damage is 
better located by the brain and felt at the site of 
pathology. The confusion of site getting cleared 
with increase in the injury at the site 
Shifting pain
• Phantom sensation is felt in amputated part The 
person feels presence of limb with every type of 
sensation in it. 
• The cut nerve at the end of amputation stump 
heals by fibrosis forming sensitive neuro-fibromatous 
mass which on stimulation sends 
impulses to brain which are analyzed by brain as 
coming from the limb more the fibrosis more the 
chance of phantom sensation 
Phantom
• Condition where partial peripheral nerve injury in 
associated with reflex sympathetic dystrophy 
• Injured peripheral nerve with sympathetic 
dysfunction 
• Commonly seen in Brachial plexus injury 
Causalgia 
Complex regional pain syndrome
• Peripheral vasodilatation – Burning, swelling, 
edema, increased sweating, hair & nail growth 
initially but loss of hair, dry cracked nails with 
progression 
• Three times more Common in female patients 
• Average age being 42 years 
Clinical features
• Pain initiated with any stimulus hypersensitivity 
• gradually worsens 
• Red, hot, shiny skin with excessive or less 
sweating 
• Wasting of muscles 
• Stiffness of joints 
• Regional Osteo porosis 
Clinical features
Attitude 
Position taken by the part at the time of 
examination
• Attitude of joint to be described in every 
direction in which normal physiological 
movements are possible 
• Any position not possible physiologically will be 
called as deformity 
• Any loss of physiological position will be called 
as deformity 
Attitude / Deformity
Deformity 
Loss of form 
Any deviation from normal anatomy
• Change in size or shape &loss of range of 
movement 
• Size -The length 
-The width 
-The Depth 
-The circumference 
with its exact location, extent & site of 
maximum deviation 
The Size
• The extremity Change in 
-The angle 
-The shift or step 
-The rotation 
-The curvature –concavity or 
convexity – reduced obliterated or reversed 
- The outline -any local bump or 
depression 
The shape
• Change at a site 
Lump or swelling 
• Presence of 
Scar, Sinus, ulcer, 
abrasion, wound, blebs 
abnormal skin patch, 
Local changes
• The Skin 
-Appearance – Normal, stretched, wrinkled 
-Color - pale, red, blue, black 
-Dilated veins 
-Hydration – Normal, dry, wet, oily, glossy 
-Texture – normal, smooth, roughened, 
puckered 
-The hair distribution & appearance 
-The skin thickness- thinned, thickened, 
edematous 
The look
• Loss of range of movement will be called as 
fixed deformity 
• In fixed flection deformity of 10 degrees neutral 
position will not be possible as will be extension 
• From fixed flection deformity of 10 degrees 
further flection up to certain degrees will be 
possible 
• To confirm fixed deformity in a joint the joint 
above and below must be in normal anatomical 
position as the deformity can be masked by 
change in the position of joint above or below 
Loss of range of movements

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Pain - Protective tool not a curse - deformity , the medical concept

  • 1. Pain Not a disease or curse Protective mechanism
  • 2. Dr. Shrikant Gore M.S. Orthopaedics • Professor and Head • Department of Orthopaedics • Government Medical College • Latur – 413512 • E-mail – laturmedical@gmail,com
  • 3. • Unpleasant sensation felt, by a person, to any injury to the body • Mechanical - pinch, prick, cut, stretch, crush etc. • Chemical - external & internal (Bradykinines, Serotonins, prostaglandins, Bacterial toxins) • Thermal, electrical • Psychological Definition
  • 4. • It initiates body’s protective response to injury • Minimizing the damage and helps in repairs by initiating healing process • Pain is essential to maintain health and repair damage • Pain needs to be treated as an emergency to protect body from damage by removing the cause of pain Protective
  • 5. • Stimulus • Sensory receptors at nerve endings • Receptors are type specific and their concentration is area specific, depending on the the need of the area, e.g. maximum concentration of touch receptors at finger tips minimum touch receptors on visceral organs Physiology
  • 6. • On stimulation of receptor biochemical reaction sets in generating electrical impulse with injury specific modality, Carried through nerves, spinal cord. • Spinal reflex is set in and the part of body subjected to injury is withdrawn away from the injury source to avoid damage Pathway
  • 7. • The impulse is carried further to Sensory cortex representing the area of stimulus • The sensory cortex analyses this modality to recognize the specific injury type resulting in perfect location of the area of injury and the type of injury • It sends suppressive signals to spinal cord to reduce the severity of further similar impulse Perception
  • 8. • Nerve ending • Peripheral Nerves • Nerves • Nerve root • Spinal cord • Basal Ganglion • Neuropathies • Stretching • Compression, • Girdle pain • Involuntary movements Pathological Sites
  • 9. • Cerebellum • Cerebrum • Psyche • Autonomous n. • Ataxia, Balance • Altered, lost, • Pain without stimulus • Causalgia Pathological Sites
  • 10. • Carry electrical current generated at sensory receptor • If the nerve is affected by inflammation or pressure the modality(frequency) of electrical current changes on its way to brain changing the perception of sensation (Altered sensation) • Touch is perceived as Tingling, Burning, Numbness, pricking etc. due to alteration of the electrical impulse Peripheral Nerves
  • 11. • Nutritional - B-complex Vitamins • Metabolic - Diabetes mellitus • Toxicity - Lead • Infections - Leprosy, viral • Drugs - Alcohol, Peripheral neuropathies
  • 12. • Throbbing • Radiating • Referred • Shifting • Phantom • Causalgia Abnormal Types
  • 13. • Pulsatile pain • Spike of pain coming at regular intervals • Aneurisms Aortic - stretching of outer layer of vascular wall Berry’s – increased intra cranial pressure with each pulsation Migraine dilated intracranial vessels increased blood with each pulsation increases intracranial pressure • Abscess Inflamed abscess wall and surrounding tissue with hyperactive nerve endings getting stretched and stimulated with every pulsation Throbbing pain
  • 14. • Nerves, Nerve roots - Compression, Stretching • In addition to altered sensation • Affected nerve becomes painful • The pain radiating along the course of the nerve Radiating Pain
  • 15. • Pathology at one site, pain felt at other site • Cerebral cortex unable to locate the site of pain. • Commonly seen in pathologies affecting deeper structures Heart, appendicitis, Hip joint • Deeper structures less likely to get injured by physical trauma have very low number of pain receptors. Referred Pain
  • 16. • One single nerve supplies multiple organs • The pain is felt at the organ having better number of sensory receptors , supplied by same nerve root • It gets distributed to larger area of brain making it difficult for pin point location Referred Pain
  • 17. • Pain initially felt at one site disappears and is felt at other site • Referred pain with progression of damage is better located by the brain and felt at the site of pathology. The confusion of site getting cleared with increase in the injury at the site Shifting pain
  • 18. • Phantom sensation is felt in amputated part The person feels presence of limb with every type of sensation in it. • The cut nerve at the end of amputation stump heals by fibrosis forming sensitive neuro-fibromatous mass which on stimulation sends impulses to brain which are analyzed by brain as coming from the limb more the fibrosis more the chance of phantom sensation Phantom
  • 19. • Condition where partial peripheral nerve injury in associated with reflex sympathetic dystrophy • Injured peripheral nerve with sympathetic dysfunction • Commonly seen in Brachial plexus injury Causalgia Complex regional pain syndrome
  • 20. • Peripheral vasodilatation – Burning, swelling, edema, increased sweating, hair & nail growth initially but loss of hair, dry cracked nails with progression • Three times more Common in female patients • Average age being 42 years Clinical features
  • 21. • Pain initiated with any stimulus hypersensitivity • gradually worsens • Red, hot, shiny skin with excessive or less sweating • Wasting of muscles • Stiffness of joints • Regional Osteo porosis Clinical features
  • 22. Attitude Position taken by the part at the time of examination
  • 23. • Attitude of joint to be described in every direction in which normal physiological movements are possible • Any position not possible physiologically will be called as deformity • Any loss of physiological position will be called as deformity Attitude / Deformity
  • 24. Deformity Loss of form Any deviation from normal anatomy
  • 25. • Change in size or shape &loss of range of movement • Size -The length -The width -The Depth -The circumference with its exact location, extent & site of maximum deviation The Size
  • 26. • The extremity Change in -The angle -The shift or step -The rotation -The curvature –concavity or convexity – reduced obliterated or reversed - The outline -any local bump or depression The shape
  • 27. • Change at a site Lump or swelling • Presence of Scar, Sinus, ulcer, abrasion, wound, blebs abnormal skin patch, Local changes
  • 28. • The Skin -Appearance – Normal, stretched, wrinkled -Color - pale, red, blue, black -Dilated veins -Hydration – Normal, dry, wet, oily, glossy -Texture – normal, smooth, roughened, puckered -The hair distribution & appearance -The skin thickness- thinned, thickened, edematous The look
  • 29. • Loss of range of movement will be called as fixed deformity • In fixed flection deformity of 10 degrees neutral position will not be possible as will be extension • From fixed flection deformity of 10 degrees further flection up to certain degrees will be possible • To confirm fixed deformity in a joint the joint above and below must be in normal anatomical position as the deformity can be masked by change in the position of joint above or below Loss of range of movements